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  1. Article ; Online: Fishing CHIPs to Predict Postoperative Atrial Fibrillation.

    Mallat, Ziad

    Journal of the American College of Cardiology

    2023  Volume 81, Issue 13, Page(s) 1279–1282

    MeSH term(s) Humans ; Aortic Valve/surgery ; Aortic Valve Stenosis/surgery ; Atrial Fibrillation/diagnosis ; Atrial Fibrillation/surgery ; Heart Valve Prosthesis Implantation ; Postoperative Complications/diagnosis ; Postoperative Complications/surgery ; Risk Factors
    Language English
    Publishing date 2023-03-07
    Publishing country United States
    Document type Editorial ; Research Support, Non-U.S. Gov't ; Comment
    ZDB-ID 605507-2
    ISSN 1558-3597 ; 0735-1097
    ISSN (online) 1558-3597
    ISSN 0735-1097
    DOI 10.1016/j.jacc.2023.01.037
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    Zs.A 1846: Show issues Location:
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    ab Jg. 2022: Lesesaal (EG)
    Zs.MO 196: Show issues

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  2. Article ; Online: Century of Milestones and Breakthroughs Related to the Immune Mechanisms of Atherosclerosis.

    Mallat, Ziad / Tedgui, Alain

    Arteriosclerosis, thrombosis, and vascular biology

    2024  Volume 44, Issue 5, Page(s) 1002–1006

    MeSH term(s) Atherosclerosis/immunology ; Atherosclerosis/history ; Humans ; History, 20th Century ; Animals ; History, 21st Century ; Biomedical Research/history ; Biomedical Research/trends
    Language English
    Publishing date 2024-04-24
    Publishing country United States
    Document type Historical Article ; Journal Article ; Review ; Research Support, Non-U.S. Gov't ; Research Support, N.I.H., Extramural ; Editorial
    ZDB-ID 1221433-4
    ISSN 1524-4636 ; 1079-5642
    ISSN (online) 1524-4636
    ISSN 1079-5642
    DOI 10.1161/ATVBAHA.124.319397
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 1705: Show issues Location:
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  3. Article ; Online: Dietary sucrose induces more atherosclerosis than dietary fat in LDLr

    Mallat, Ziad

    Atherosclerosis

    2021  Volume 325, Page(s) 117

    MeSH term(s) Animals ; Apolipoprotein B-100 ; Atherosclerosis ; Cardiovascular Diseases ; Cholesterol ; Dietary Fats ; Dietary Sucrose ; Inflammation ; Mice ; Receptors, LDL/genetics
    Chemical Substances Apolipoprotein B-100 ; Dietary Fats ; Dietary Sucrose ; Receptors, LDL ; Cholesterol (97C5T2UQ7J)
    Language English
    Publishing date 2021-02-23
    Publishing country Ireland
    Document type Letter ; Research Support, Non-U.S. Gov't ; Comment
    ZDB-ID 80061-2
    ISSN 1879-1484 ; 0021-9150
    ISSN (online) 1879-1484
    ISSN 0021-9150
    DOI 10.1016/j.atherosclerosis.2021.02.015
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    Zs.A 226: Show issues Location:
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  4. Article ; Online: Adapting treatments for adaptive immunity in ischaemic heart disease.

    Zhao, Tian / Mallat, Ziad

    Cardiovascular research

    2022  Volume 118, Issue 9, Page(s) e66–e68

    MeSH term(s) Adaptive Immunity ; Atherosclerosis ; Humans ; Immunity, Innate ; Inflammation ; Myocardial Ischemia/therapy
    Language English
    Publishing date 2022-06-02
    Publishing country England
    Document type Journal Article
    ZDB-ID 80340-6
    ISSN 1755-3245 ; 0008-6363
    ISSN (online) 1755-3245
    ISSN 0008-6363
    DOI 10.1093/cvr/cvac074
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 565: Show issues Location:
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  5. Article ; Online: Regulating heart repair with cardiac-specific T lymphocytes.

    Mallat, Ziad

    The Journal of clinical investigation

    2019  Volume 129, Issue 11, Page(s) 4587–4589

    Abstract: Cardiac tissue necrosis secondary to coronary artery occlusion is one of the most common and deadly sterile injuries in developed countries. In this issue of the JCI, Rieckmann et al. identified and characterized antigen-specific CD4+ T helper (Th) cells ...

    Abstract Cardiac tissue necrosis secondary to coronary artery occlusion is one of the most common and deadly sterile injuries in developed countries. In this issue of the JCI, Rieckmann et al. identified and characterized antigen-specific CD4+ T helper (Th) cells that developed in the context of myocardial infarction (MI) in mice. They showed that myosin heavy chain α (MYHCA) is a dominant cardiac autoantigen and that T cells with T cell receptor (TCR) specificity to MYHCA acquired a Treg phenotype when adoptively transferred into infarcted mice, which mediated a cardioprotective healing response. Thus, Rieckmann et al. showed that an acute ischemic insult to the heart, which induces sterile inflammation, promoted, rather than limited, protective T cell autoimmunity. Notably, strategies that support an antigen-specific Treg response may limit the immune-inflammatory response and promote cardiac repair after acute MI.
    MeSH term(s) Animals ; Heart ; Mice ; Myocardial Infarction ; Myocardium ; T-Lymphocytes, Helper-Inducer ; T-Lymphocytes, Regulatory
    Language English
    Publishing date 2019-10-14
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Comment
    ZDB-ID 3067-3
    ISSN 1558-8238 ; 0021-9738
    ISSN (online) 1558-8238
    ISSN 0021-9738
    DOI 10.1172/JCI132441
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  6. Article ; Online: "Plaque erosion" or the danger of eerily quiet appearance.

    Caligiuri, Giuseppina / Mallat, Ziad

    Atherosclerosis

    2021  Volume 318, Page(s) 43–44

    MeSH term(s) Acute Coronary Syndrome ; Humans ; Plaque, Atherosclerotic
    Language English
    Publishing date 2021-01-05
    Publishing country Ireland
    Document type Editorial ; Comment
    ZDB-ID 80061-2
    ISSN 1879-1484 ; 0021-9150
    ISSN (online) 1879-1484
    ISSN 0021-9150
    DOI 10.1016/j.atherosclerosis.2020.12.022
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 226: Show issues Location:
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  7. Article ; Online: The why and how of adaptive immune responses in ischemic cardiovascular disease.

    Mallat, Ziad / Binder, Christoph J

    Nature cardiovascular research

    2022  Volume 1, Page(s) 431–444

    Abstract: Atherosclerotic cardiovascular disease is a major cause of disability and death worldwide. Most therapeutic approaches target traditional risk factors but ignore the fundamental role of the immune system. This is a huge unmet need. Recent evidence ... ...

    Abstract Atherosclerotic cardiovascular disease is a major cause of disability and death worldwide. Most therapeutic approaches target traditional risk factors but ignore the fundamental role of the immune system. This is a huge unmet need. Recent evidence indicates that reducing inflammation may limit cardiovascular events. However, the concomitant increase in the risk of lifethreatening infections is a major drawback. In this context, targeting adaptive immunity could constitute a highly effective and safer approach. In this Review, we address the why and how of the immuno-cardiovascular unit, in health and in atherosclerotic disease. We review and discuss fundamental mechanisms that ensure immune tolerance to cardiovascular tissue, and examine how their disruption promotes disease progression. We identify promising strategies to manipulate the adaptive immune system for patient benefit, including novel biologics and RNA-based vaccination strategies. Finally, we advocate for establishing a molecular classification of atherosclerosis as an important milestone in our quest to radically change the understanding and treatment of atherosclerotic disease.
    Language English
    Publishing date 2022-04-21
    Publishing country England
    Document type Journal Article
    ISSN 2731-0590
    ISSN (online) 2731-0590
    DOI 10.1038/s44161-022-00049-1
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  8. Article ; Online: Macrophages.

    Mallat, Ziad

    Arteriosclerosis, thrombosis, and vascular biology

    2017  Volume 37, Issue 8, Page(s) e92–e98

    MeSH term(s) Animals ; Cardiovascular Diseases/immunology ; Cardiovascular Diseases/metabolism ; Cardiovascular Diseases/pathology ; Cardiovascular System/immunology ; Cardiovascular System/metabolism ; Cardiovascular System/pathology ; Humans ; Immunity, Innate ; Macrophages/immunology ; Macrophages/metabolism ; Macrophages/pathology ; Phenotype ; Signal Transduction
    Language English
    Publishing date 2017-07-26
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 1221433-4
    ISSN 1524-4636 ; 1079-5642
    ISSN (online) 1524-4636
    ISSN 1079-5642
    DOI 10.1161/ATVBAHA.117.309730
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 1705: Show issues Location:
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  9. Article ; Online: Suppression of Hematopoiesis in Recurrent Myocardial Infarction: A Deadly Silence.

    Mallat, Ziad / Hulot, Jean-Sébastien

    Journal of the American College of Cardiology

    2020  Volume 75, Issue 8, Page(s) 916–918

    MeSH term(s) Animals ; Bone Marrow ; Hematopoiesis ; Inflammation ; Mice ; Myocardial Infarction
    Language English
    Publishing date 2020-02-06
    Publishing country United States
    Document type Editorial ; Research Support, Non-U.S. Gov't ; Comment
    ZDB-ID 605507-2
    ISSN 1558-3597 ; 0735-1097
    ISSN (online) 1558-3597
    ISSN 0735-1097
    DOI 10.1016/j.jacc.2020.01.008
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  10. Article: Editorial: Inflammation and Reparative Process After Cardiac Injury.

    Silvestre, Jean-Sébastien / Mallat, Ziad

    Frontiers in cardiovascular medicine

    2019  Volume 6, Page(s) 162

    Language English
    Publishing date 2019-11-08
    Publishing country Switzerland
    Document type Editorial
    ZDB-ID 2781496-8
    ISSN 2297-055X
    ISSN 2297-055X
    DOI 10.3389/fcvm.2019.00162
    Database MEDical Literature Analysis and Retrieval System OnLINE

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