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Article ; Online: Paediatric trigger fingers: a 47-year experience.

Wan, Rou / Sarcon, Aida K / Pommer, Paula Pino / Mundell, Benjamin F / Zhao, Chunfeng / Moran, Steven L

The Journal of hand surgery, European volume

2023 , Page(s) 17531934231214103

Abstract: Paediatric trigger finger is rare compared to adult trigger finger or paediatric trigger thumb, and the aetiology is unclear. Proposed causes include local trauma, anatomical anomalies and systemic conditions. The aim of the present study was to detail ... ...

Abstract	Paediatric trigger finger is rare compared to adult trigger finger or paediatric trigger thumb, and the aetiology is unclear. Proposed causes include local trauma, anatomical anomalies and systemic conditions. The aim of the present study was to detail the anatomical causes of surgically treated paediatric trigger fingers and provide an operative algorithm based on the anatomical findings. A total of 76 trigger fingers in 38 patients were identified retrospectively at our institution between 1975 and 2022. In total, 41 fingers in 26 patients had anatomical variations. A nodular thickening on the tendon, similar to Notta's nodule in trigger thumbs, was the most common anatomical cause. Abnormal decussation of the flexor digitorum superficialis tendon was the second most common variation. The recurrence rate was significantly lower after resection of one slip of the flexor digitorum superficialis tendon compared to other surgical techniques in these patients. We recommend that surgeons assess for possible anatomical variation during surgery for the trigger finger.
Language	English
Publishing date	2023-11-21
Publishing country	England
Document type	Journal Article
ZDB-ID	2272801-6
ISSN	2043-6289 ; 1753-1934
ISSN (online)	2043-6289
ISSN	1753-1934
DOI	10.1177/17531934231214103
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Article ; Online: Dural puncture during lumbar epidural access in the setting of degenerative spondylolisthesis: case series and risk mitigation strategies.

Sindhi, Vivek / Lim, Christine G / Khan, Anver / Pino, Carlos / Cohen, Steven P

Regional anesthesia and pain medicine

2021 Volume 46, Issue 11, Page(s) 992–996

Abstract: Degenerative spondylolisthesis is a common back pathology in the general adult population. Patients with this condition may present for epidural steroid injection, epidural blood patch, or epidural analgesia. We report five patients with degenerative ... ...

Abstract	Degenerative spondylolisthesis is a common back pathology in the general adult population. Patients with this condition may present for epidural steroid injection, epidural blood patch, or epidural analgesia. We report five patients with degenerative spondylolisthesis who experienced inadvertent dural puncture during interlaminar epidural steroid injection attempts: four with intrathecal contrast spread or cerebrospinal backflow into the epidural needle and one with subdural contrast spread. Patients with degenerative spondylolisthesis may be at higher risk for dural puncture due to stretching of the dura and contraction of the epidural space at the translated spinal level. In the following report, we summarize the cases and suggest risk mitigation strategies for both chronic and acute pain physicians.
MeSH term(s)	Adult ; Analgesia, Epidural/adverse effects ; Blood Patch, Epidural ; Dura Mater/diagnostic imaging ; Humans ; Spinal Puncture ; Spondylolisthesis/diagnostic imaging ; Spondylolisthesis/surgery
Language	English
Publishing date	2021-07-15
Publishing country	England
Document type	Journal Article
ZDB-ID	1425299-5
ISSN	1532-8651 ; 1098-7339 ; 0146-521X
ISSN (online)	1532-8651
ISSN	1098-7339 ; 0146-521X
DOI	10.1136/rapm-2021-102963
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Article ; Online: Non-Abelian topological order and anyons on a trapped-ion processor.

Iqbal, Mohsin / Tantivasadakarn, Nathanan / Verresen, Ruben / Campbell, Sara L / Dreiling, Joan M / Figgatt, Caroline / Gaebler, John P / Johansen, Jacob / Mills, Michael / Moses, Steven A / Pino, Juan M / Ransford, Anthony / Rowe, Mary / Siegfried, Peter / Stutz, Russell P / Foss-Feig, Michael / Vishwanath, Ashvin / Dreyer, Henrik

Nature

2024 Volume 626, Issue 7999, Page(s) 505–511

Abstract: Non-Abelian topological order is a coveted state of matter with remarkable properties, including quasiparticles that can remember the sequence in which they are ... ...

Abstract	Non-Abelian topological order is a coveted state of matter with remarkable properties, including quasiparticles that can remember the sequence in which they are exchanged
Language	English
Publishing date	2024-02-14
Publishing country	England
Document type	Journal Article
ZDB-ID	120714-3
ISSN	1476-4687 ; 0028-0836
ISSN (online)	1476-4687
ISSN	0028-0836
DOI	10.1038/s41586-023-06934-4
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Article ; Online: Randomised trial of genetic testing and targeted intervention to prevent the development and progression of Paget's disease of bone.

Phillips, Jonathan / Subedi, Deepak / Lewis, Steff C / Keerie, Catriona / Cronin, Owen / Porteous, Mary / Moore, David / Cetnarskyj, Roseanne / Ranganath, Lakshminarayan / Selby, Peter L / Turgut, Tolga / Hampson, Geeta / Chandra, Rama / Ho, Shu / Tobias, Jon / Young-Min, Steven / McKenna, Malachi J / Crowley, Rachel K / Fraser, William D /

Annals of the rheumatic diseases

2024 Volume 83, Issue 4, Page(s) 529–536

Abstract: Introduction: Paget's disease of bone (PDB) frequently presents at an advanced stage with irreversible skeletal damage. Clinical outcomes might be improved by earlier diagnosis and prophylactic treatment.: Methods: We randomised 222 individuals at ... ...

Abstract	Introduction: Paget's disease of bone (PDB) frequently presents at an advanced stage with irreversible skeletal damage. Clinical outcomes might be improved by earlier diagnosis and prophylactic treatment. Methods: We randomised 222 individuals at increased risk of PDB because of pathogenic Results: The median duration of follow-up was 84 months (range 0-127) and 180 participants (81%) completed the study. At baseline, 9 (8.1%) of the ZA group had PDB lesions vs 12 (10.8%) of the placebo group. Two of the placebo group developed new lesions versus none in the ZA group (OR 0.41, 95% CI 0.00 to 3.43, p=0.25). Eight of the placebo group had a poor outcome (lesions which were new, unchanged or progressing) compared with none of the ZA group (OR 0.08, 95% CI 0.00 to 0.42, p=0.003). At the study end, 1 participant in the ZA group had lesions compared with 11 in the placebo group. Biochemical markers of bone turnover were significantly reduced in the ZA group. One participant allocated to placebo required rescue therapy with ZA because of symptomatic disease. The number and severity of adverse events did not differ between groups. Conclusions: Genetic testing for pathogenic Trial registration number: ISRCTN11616770.
MeSH term(s)	Humans ; Diphosphonates/adverse effects ; Osteitis Deformans/complications ; Osteitis Deformans/drug therapy ; Osteitis Deformans/genetics ; Sequestosome-1 Protein/genetics ; Zoledronic Acid/therapeutic use ; Genetic Testing ; Biomarkers
Chemical Substances	Diphosphonates ; Sequestosome-1 Protein ; Zoledronic Acid (6XC1PAD3KF) ; Biomarkers
Language	English
Publishing date	2024-03-12
Publishing country	England
Document type	Randomized Controlled Trial ; Journal Article
ZDB-ID	7090-7
ISSN	1468-2060 ; 0003-4967
ISSN (online)	1468-2060
ISSN	0003-4967
DOI	10.1136/ard-2023-224990
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Article ; Online: The role of innate immune pathways in type 1 diabetes pathogenesis.

Pino, Steven C / Kruger, Annie J / Bortell, Rita

Current opinion in endocrinology, diabetes, and obesity

2010 Volume 17, Issue 2, Page(s) 126–130

Abstract: Purpose of review: Type 1 diabetes (T1D) is an autoimmune disease typically believed to result from malfunctions in adaptive immune response signaling which result in activation of self-reactive T cells. However, recent research has indicated components ...

Abstract	Purpose of review: Type 1 diabetes (T1D) is an autoimmune disease typically believed to result from malfunctions in adaptive immune response signaling which result in activation of self-reactive T cells. However, recent research has indicated components of the innate immune response as having a key role in the initiation of the autoimmune process of T1D. This review will highlight recent studies which examined the role of innate immune response signaling and the connections to T1D pathogenesis. Recent findings: Investigations indicate that components of innate immunity, including inflammation and Toll-like receptor signaling, are involved in pancreatic islet infiltration and insulitis. Recent studies examining the role of viral infections in T1D development also implicate innate immune response signaling in disease pathogenesis. Summary: Current research indicates that components of innate immune response signaling are involved in the initiation of the autoimmune process which results in the eventual destruction of beta cells during T1D pathogenesis. Continuing efforts by researchers to uncover the molecular pathways of innate immunity linked to T1D development could potentially lead to therapeutics capable of preventing and curing the autoimmune disease.
MeSH term(s)	Animals ; Diabetes Mellitus, Type 1/etiology ; Diabetes Mellitus, Type 1/immunology ; Humans ; Immunity, Innate/physiology ; Inflammation/immunology ; Signal Transduction/immunology ; Signal Transduction/physiology ; Toll-Like Receptors/physiology ; Virus Diseases/etiology ; Virus Diseases/immunology
Chemical Substances	Toll-Like Receptors
Language	English
Publishing date	2010-01-27
Publishing country	England
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	2272017-0
ISSN	1752-2978 ; 1752-296X
ISSN (online)	1752-2978
ISSN	1752-296X
DOI	10.1097/MED.0b013e3283372819
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Article ; Online: Entanglement from Tensor Networks on a Trapped-Ion Quantum Computer.

Foss-Feig, Michael / Ragole, Stephen / Potter, Andrew / Dreiling, Joan / Figgatt, Caroline / Gaebler, John / Hall, Alex / Moses, Steven / Pino, Juan / Spaun, Ben / Neyenhuis, Brian / Hayes, David

Physical review letters

2022 Volume 128, Issue 15, Page(s) 150504

Abstract: The ability to selectively measure, initialize, and reuse qubits during a quantum circuit enables a mapping of the spatial structure of certain tensor-network states onto the dynamics of quantum circuits, thereby achieving dramatic resource savings when ... ...

Abstract	The ability to selectively measure, initialize, and reuse qubits during a quantum circuit enables a mapping of the spatial structure of certain tensor-network states onto the dynamics of quantum circuits, thereby achieving dramatic resource savings when simulating quantum systems with limited entanglement. We experimentally demonstrate a significant benefit of this approach to quantum simulation: the entanglement structure of an infinite system-specifically the half-chain entanglement spectrum-is conveniently encoded within a small register of "bond qubits" and can be extracted with relative ease. Using Honeywell's model H0 quantum computer equipped with selective midcircuit measurement and reset, we quantitatively determine the near-critical entanglement entropy of a correlated spin chain directly in the thermodynamic limit and show that its phase transition becomes quickly resolved upon expanding the bond-qubit register.
Language	English
Publishing date	2022-05-02
Publishing country	United States
Document type	Journal Article
ZDB-ID	208853-8
ISSN	1079-7114 ; 0031-9007
ISSN (online)	1079-7114
ISSN	0031-9007
DOI	10.1103/PhysRevLett.128.150504
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Article ; Online: Modulation of type I interferon responses potently inhibits SARS-CoV-2 replication and inflammation in rhesus macaques.

Viox, Elise G / Hoang, Timothy N / Upadhyay, Amit A / Nchioua, Rayhane / Hirschenberger, Maximilian / Strongin, Zachary / Tharp, Gregory K / Pino, Maria / Nguyen, Kevin / Harper, Justin L / Gagne, Matthew / Marciano, Shir / Boddapati, Arun K / Pellegrini, Kathryn L / Pradhan, Arpan / Tisoncik-Go, Jennifer / Whitmore, Leanne S / Karunakaran, Kirti A / Roy, Melissa /

Science immunology

2023 Volume 8, Issue 85, Page(s) eadg0033

Abstract: Type I interferons (IFN-I) are critical mediators of innate control of viral infections but also drive the recruitment of inflammatory cells to sites of infection, a key feature of severe coronavirus disease 2019. Here, IFN-I signaling was modulated in ... ...

Abstract	Type I interferons (IFN-I) are critical mediators of innate control of viral infections but also drive the recruitment of inflammatory cells to sites of infection, a key feature of severe coronavirus disease 2019. Here, IFN-I signaling was modulated in rhesus macaques (RMs) before and during acute SARS-CoV-2 (severe acute respiratory syndrome coronavirus 2) infection using a mutated IFN-α2 (IFN-modulator; IFNmod), which has previously been shown to reduce the binding and signaling of endogenous IFN-I. IFNmod treatment in uninfected RMs was observed to induce a modest up-regulation of only antiviral IFN-stimulated genes (ISGs); however, in SARS-CoV-2-infected RMs, IFNmod reduced both antiviral and inflammatory ISGs. IFNmod treatment resulted in a potent reduction in SARS-CoV-2 viral loads both in vitro in Calu-3 cells and in vivo in bronchoalveolar lavage (BAL), upper airways, lung, and hilar lymph nodes of RMs. Furthermore, in SARS-CoV-2-infected RMs, IFNmod treatment potently reduced inflammatory cytokines, chemokines, and CD163
MeSH term(s)	Animals ; Interferon Type I/pharmacology ; SARS-CoV-2 ; Macaca mulatta ; COVID-19 ; Virus Replication ; Antiviral Agents/pharmacology ; Antiviral Agents/therapeutic use ; Inflammation/drug therapy
Chemical Substances	Interferon Type I ; Antiviral Agents
Language	English
Publishing date	2023-07-28
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ISSN	2470-9468
ISSN (online)	2470-9468
DOI	10.1126/sciimmunol.adg0033
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Article ; Online: Hindgut microbiota in laboratory-reared and wild Triatoma infestans.

Waltmann, Andreea / Willcox, Alexandra C / Balasubramanian, Sujata / Borrini Mayori, Katty / Mendoza Guerrero, Sandra / Salazar Sanchez, Renzo S / Roach, Jeffrey / Condori Pino, Carlos / Gilman, Robert H / Bern, Caryn / Juliano, Jonathan J / Levy, Michael Z / Meshnick, Steven R / Bowman, Natalie M

PLoS neglected tropical diseases

2019 Volume 13, Issue 5, Page(s) e0007383

Abstract: Triatomine vectors transmit Trypanosoma cruzi, the etiological agent of Chagas disease in humans. Transmission to humans typically occurs when contaminated triatomine feces come in contact with the bite site or mucosal membranes. In the Southern Cone of ... ...

Abstract	Triatomine vectors transmit Trypanosoma cruzi, the etiological agent of Chagas disease in humans. Transmission to humans typically occurs when contaminated triatomine feces come in contact with the bite site or mucosal membranes. In the Southern Cone of South America, where the highest burden of disease exists, Triatoma infestans is the principal vector for T. cruzi. Recent studies of other vector-borne illnesses have shown that arthropod microbiota influences the ability of infectious agents to colonize the insect vector and transmit to the human host. This has garnered attention as a potential control strategy against T. cruzi, as vector control is the main tool of Chagas disease prevention. Here we characterized the microbiota in T. infestans feces of both wild-caught and laboratory-reared insects and examined the relationship between microbial composition and T. cruzi infection using highly sensitive high-throughput sequencing technology to sequence the V3-V4 region of the 16S ribosomal RNA gene on the MiSeq Illumina platform. We collected 59 wild (9 with T. cruzi infection) and 10 lab-reared T. infestans (4 with T. cruzi infection) from the endemic area of Arequipa, Perú. Wild T. infestans had greater hindgut bacterial diversity than laboratory-reared bugs. Microbiota of lab insects comprised a subset of those identified in their wild counterparts, with 96 of the total 124 genera also observed in laboratory-reared insects. Among wild insects, variation in bacterial composition was observed, but time and location of collection and development stage did not explain this variation. T. cruzi infection in lab insects did not affect α- or β-diversity; however, we did find that the β-diversity of wild insects differed if they were infected with T. cruzi and identified 10 specific taxa that had significantly different relative abundances in infected vs. uninfected wild T. infestans (Bosea, Mesorhizobium, Dietzia, and Cupriavidus were underrepresented in infected bugs; Sporosarcina, an unclassified genus of Porphyromonadaceae, Nestenrenkonia, Alkalibacterium, Peptoniphilus, Marinilactibacillus were overrepresented in infected bugs). Our findings suggest that T. cruzi infection is associated with the microbiota of T. infestans and that inferring the microbiota of wild T. infestans may not be possible through sampling of T. infestans reared in the insectary.
MeSH term(s)	Animals ; Bacteria/classification ; Bacteria/genetics ; Bacteria/isolation & purification ; Chagas Disease/parasitology ; Chagas Disease/transmission ; DNA, Bacterial/genetics ; Feces/microbiology ; Gastrointestinal Tract/microbiology ; Humans ; Insect Vectors/microbiology ; Insect Vectors/parasitology ; Insect Vectors/physiology ; Laboratories ; Microbiota ; Phylogeny ; RNA, Ribosomal, 16S/genetics ; Triatoma/microbiology ; Triatoma/parasitology ; Triatoma/physiology ; Trypanosoma cruzi/physiology
Chemical Substances	DNA, Bacterial ; RNA, Ribosomal, 16S
Language	English
Publishing date	2019-05-06
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural
ZDB-ID	2429704-5
ISSN	1935-2735 ; 1935-2727
ISSN (online)	1935-2735
ISSN	1935-2727
DOI	10.1371/journal.pntd.0007383
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Article ; Online: Embedding of Precision-Cut Lung Slices in Engineered Hydrogel Biomaterials Supports Extended

Bailey, Kolene E / Pino, Christopher / Lennon, Mallory L / Lyons, Anne / Jacot, Jeffrey G / Lammers, Steven R / Königshoff, Melanie / Magin, Chelsea M

American journal of respiratory cell and molecular biology

2019 Volume 62, Issue 1, Page(s) 14–22

Abstract: Maintaining the three-dimensional architecture and cellular complexity of lung ... ...

Abstract	Maintaining the three-dimensional architecture and cellular complexity of lung tissue
MeSH term(s)	Animals ; Biocompatible Materials/administration & dosage ; Humans ; Hydrogels/administration & dosage ; Lung/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Organ Culture Techniques/methods ; Pulmonary Disease, Chronic Obstructive/pathology
Chemical Substances	Biocompatible Materials ; Hydrogels
Language	English
Publishing date	2019-09-09
Publishing country	United States
Document type	Journal Article
ZDB-ID	1025960-0
ISSN	1535-4989 ; 1044-1549
ISSN (online)	1535-4989
ISSN	1044-1549
DOI	10.1165/rcmb.2019-0232MA
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Article ; Online: Activated Oncogenic Pathway Modifies Iron Network in Breast Epithelial Cells: A Dynamic Modeling Perspective.

Chifman, Julia / Arat, Seda / Deng, Zhiyong / Lemler, Erica / Pino, James C / Harris, Leonard A / Kochen, Michael A / Lopez, Carlos F / Akman, Steven A / Torti, Frank M / Torti, Suzy V / Laubenbacher, Reinhard

PLoS computational biology

2017 Volume 13, Issue 2, Page(s) e1005352

Abstract: Dysregulation of iron metabolism in cancer is well documented and it has been suggested that there is interdependence between excess iron and increased cancer incidence and progression. In an effort to better understand the linkages between iron ... ...

Abstract	Dysregulation of iron metabolism in cancer is well documented and it has been suggested that there is interdependence between excess iron and increased cancer incidence and progression. In an effort to better understand the linkages between iron metabolism and breast cancer, a predictive mathematical model of an expanded iron homeostasis pathway was constructed that includes species involved in iron utilization, oxidative stress response and oncogenic pathways. The model leads to three predictions. The first is that overexpression of iron regulatory protein 2 (IRP2) recapitulates many aspects of the alterations in free iron and iron-related proteins in cancer cells without affecting the oxidative stress response or the oncogenic pathways included in the model. This prediction was validated by experimentation. The second prediction is that iron-related proteins are dramatically affected by mitochondrial ferritin overexpression. This prediction was validated by results in the pertinent literature not used for model construction. The third prediction is that oncogenic Ras pathways contribute to altered iron homeostasis in cancer cells. This prediction was validated by a combination of simulation experiments of Ras overexpression and catalase knockout in conjunction with the literature. The model successfully captures key aspects of iron metabolism in breast cancer cells and provides a framework upon which more detailed models can be built.
MeSH term(s)	Adaptation, Physiological ; Animals ; Breast/metabolism ; Breast/pathology ; Cell Transformation, Neoplastic/metabolism ; Computer Simulation ; Epithelial Cells/metabolism ; Epithelial Cells/pathology ; Female ; Humans ; Iron/metabolism ; Iron Regulatory Protein 2/metabolism ; Models, Biological ; Signal Transduction ; Tumor Cells, Cultured ; ras Proteins/metabolism
Chemical Substances	Iron (E1UOL152H7) ; ras Proteins (EC 3.6.5.2) ; Iron Regulatory Protein 2 (EC 4.2.1.3)
Language	English
Publishing date	2017-02-06
Publishing country	United States
Document type	Evaluation Studies ; Journal Article ; Validation Studies ; Research Support, U.S. Gov't, Non-P.H.S. ; Research Support, Non-U.S. Gov't ; Research Support, N.I.H., Extramural
ZDB-ID	2193340-6
ISSN	1553-7358 ; 1553-734X
ISSN (online)	1553-7358
ISSN	1553-734X
DOI	10.1371/journal.pcbi.1005352
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