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  1. Book ; Thesis: Untersuchungen über die Effektivität von Strategien zur Vermeidung der Rötelnembryopathie

    Bartram, Ulrike

    1991  

    Author's details vorgelegt von: Ulrike Bartram
    Language German
    Size II, 225, [13] Bl. : graph. Darst.
    Document type Book ; Thesis
    Thesis / German Habilitation thesis Hannover, Med. Hochsch., Diss., 1993
    HBZ-ID HT006342694
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

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  2. Book: Individuelle Pädiatrie

    Soldner, Georg / Stellmann, Hermann Michael / Bartram, Ulrike / Huber, Benedikt / Madeleyn, René Antoine

    leibliche, seelische und geistige Aspekte in Diagnostik und Beratung : anthroposophisch-homöopathische Therapie

    2018  

    Author's details Soldner/Stellmann ; Georg Soldner (München), H. Michael Stellmann ; unter Mitarbeit von Ulrike Bartram, Benedikt Huber, René Madeleyn
    Keywords Kind ; Krankheit ; Homöopathie ; Anthroposophische Medizin
    Subject Intuitive Medizin ; Homöotherapie ; Erkrankung ; Krankheitszustand ; Krankheiten ; Morbus ; Nosos ; Pathos ; Kindheit ; Kindesalter ; Kindschaft ; Kinder
    Language German
    Size XXXI, 1202 Seiten, 3 Illustrationen, 25 cm
    Edition 5., völlig neu bearbeitete und erweiterte Auflage
    Publisher Wissenschaftliche Verlagsgesellschaft
    Publishing place Stuttgart
    Publishing country Germany
    Document type Book
    HBZ-ID HT019848350
    ISBN 978-3-8047-3303-9 ; 9783804738812 ; 3-8047-3303-4 ; 3804738818
    Database Catalogue ZB MED Medicine, Health

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    2018 A 1789 available for loan (reading room) Lesesaal EG

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  3. Book ; Audio / Video ; Online: Photobiosense

    Unruh, Michael / Ostendorf, Ralf / / Bartram, Markus / Sacher, Joachim / Honsberg, Martin / Willer, Ulrike / Sepp, Susanne / Müller, Ulf

    Dual getriebener photonischer Sensor zur Überwachung von Biogasanlagen

    2018  

    Keywords Text ; ddc:333.7
    Language German
    Publishing country de
    Document type Book ; Audio / Video ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  4. Article: Harmonic versus conventional ultrasound imaging of the urinary tract in children.

    Bartram, Ulrike / Darge, Kassa

    Pediatric radiology

    2005  Volume 35, Issue 7, Page(s) 655–660

    Abstract: Background: Harmonic imaging (HI) is a relatively new US method. Its usefulness in children has yet to be determined.: Objective: To evaluate the value of HI in urinary tract imaging of infants and children.: Materials and methods: The bladder and ...

    Abstract Background: Harmonic imaging (HI) is a relatively new US method. Its usefulness in children has yet to be determined.
    Objective: To evaluate the value of HI in urinary tract imaging of infants and children.
    Materials and methods: The bladder and kidneys of 29 unselected patients, aged 2 months to 12.8 years (mean, 6.8 years), were examined from ventral and dorsal approaches using conventional (= fundamental) imaging (FI) (6.5 and 3.4 MHz) and HI (3.3 MHz). In addition to global image quality, visualization of bladder wall, retrovesical space, renal contrast to liver/spleen, the pelvicalyceal systems, and the difference between cortex and medulla were assessed. Parameters were rated by two independent examiners and statistical analysis was performed.
    Results: There were significant differences between the three imaging settings for all parameters studied (P<0.003). With the dorsal approach, HI was superior to FI for all parameters analysed (P<0.05). Using the ventral approach, a significant improvement of imaging with HI was found for the bladder and the renal pelvis (P<0.02).
    Conclusions: HI is a useful additional tool for imaging the urinary tract in children of all ages. The method particularly improves visualization of the bladder and the kidney from a dorsal approach.
    MeSH term(s) Body Weight ; Child ; Child, Preschool ; Female ; Humans ; Image Enhancement/methods ; Image Processing, Computer-Assisted ; Infant ; Kidney/diagnostic imaging ; Kidney Calices/diagnostic imaging ; Kidney Cortex/diagnostic imaging ; Kidney Medulla/diagnostic imaging ; Kidney Pelvis/diagnostic imaging ; Liver/diagnostic imaging ; Male ; Observer Variation ; Spleen/diagnostic imaging ; Ultrasonography/methods ; Urinary Bladder/diagnostic imaging
    Language English
    Publishing date 2005-07
    Publishing country Germany
    Document type Journal Article
    ZDB-ID 124459-0
    ISSN 1432-1998 ; 0301-0449
    ISSN (online) 1432-1998
    ISSN 0301-0449
    DOI 10.1007/s00247-005-1415-9
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 927: Show issues Location:
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    Jg. 1995 - 2021: Lesesall (1.OG)
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  5. Article: The role of transforming growth factor beta in lung development and disease.

    Bartram, Ulrike / Speer, Christian P

    Chest

    2004  Volume 125, Issue 2, Page(s) 754–765

    Abstract: Transforming growth factor (TGF) beta plays an important role in normal pulmonary morphogenesis and function and in the pathogenesis of lung disease. The effect of TGFbeta is regulated via a selective pathway of TGFbeta synthesis and signaling that ... ...

    Abstract Transforming growth factor (TGF) beta plays an important role in normal pulmonary morphogenesis and function and in the pathogenesis of lung disease. The effect of TGFbeta is regulated via a selective pathway of TGFbeta synthesis and signaling that involves activation of latent TGFbeta, specific TGFbeta receptors, and intracellular signaling via Smad molecules. All three isoforms of TGFbeta are expressed at high levels during normal lung development, being particularly important for branching morphogenesis and epithelial cell differentiation with maturation of surfactant synthesis. Small amounts of TGFbeta are still present in the adult lung, and TGFbeta is involved in normal tissue repair following lung injury. However, in a variety of forms of pulmonary pathology, the expression of TGFbeta is increased. These include chronic lung disease of prematurity as well as several forms of acute and chronic adult lung disease. While TGFbeta1 appears to be the predominant isoform involved, elevated levels of all three isoforms have been demonstrated. The increase in TGFbeta precedes abnormalities in lung function and detectable lung pathology, but correlates with the severity of the disease. TGFbeta plays a key role in mediating fibrotic tissue remodeling by increasing the production and decreasing the degradation of connective tissue via several mechanisms.
    MeSH term(s) Animals ; Female ; Humans ; Lung/embryology ; Lung/growth & development ; Lung Diseases/physiopathology ; Male ; Morphogenesis ; Pulmonary Fibrosis/physiopathology ; Risk Factors ; Sensitivity and Specificity ; Transforming Growth Factor beta/analysis ; Transforming Growth Factor beta/metabolism ; Tumor Necrosis Factor-alpha/metabolism
    Chemical Substances Transforming Growth Factor beta ; Tumor Necrosis Factor-alpha
    Language English
    Publishing date 2004-02
    Publishing country United States
    Document type Comparative Study ; Journal Article ; Review
    ZDB-ID 1032552-9
    ISSN 1931-3543 ; 0012-3692
    ISSN (online) 1931-3543
    ISSN 0012-3692
    DOI 10.1378/chest.125.2.754
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.MO 349: Show issues

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  6. Article: Congenitally interrupted inferior vena cava without other features of the heterotaxy syndrome: report of five cases and characterization of a rare entity.

    Bartram, Ulrike / Fischer, Gunther / Kramer, Hans H

    Pediatric and developmental pathology : the official journal of the Society for Pediatric Pathology and the Paediatric Pathology Society

    2008  Volume 11, Issue 4, Page(s) 266–273

    Abstract: Congenital interruption of the inferior vena cava (IVC) due to absence of its hepatic segment is usually found in patients with polysplenia. The occurrence without other features of the heterotaxy syndrome is rare, and the frequency of associated ... ...

    Abstract Congenital interruption of the inferior vena cava (IVC) due to absence of its hepatic segment is usually found in patients with polysplenia. The occurrence without other features of the heterotaxy syndrome is rare, and the frequency of associated additional vascular and cardiac anomalies in these patients is not well known. We reviewed the literature and present 5 of our own patients to characterize this entity. All but 1 of the 67 known cases had visceroatrial situs solitus. There was 1 patient with interruption of the IVC in complete situs inversus. The interrupted IVC typically continued via the azygos system into the superior vena cava, but there were 4 cases without a direct connection between the infrahepatic IVC and the azygos system. Additional venous abnormalities or cardiac malformations were present in 47.8% and 31.4%, respectively. In contrast to the classical heterotaxy syndrome, heart defects with interrupted IVC and normal situs did not involve inversion of cardiac segments. The diagnosis can readily be made by echocardiography or magnetic resonance imaging. Failure to recognize this anomaly can result in various problems during invasive medical or surgical procedures. In our own patients, interventional procedures failed, necessitating an operation in all 5 cases. Careful analysis of systemic venous drainage should be performed in all patients prior to cardiac catheterization, pacemaker implantation, or surgery, even if the visceroatrial situs is normal.
    MeSH term(s) Abnormalities, Multiple/pathology ; Abnormalities, Multiple/surgery ; Adolescent ; Angiography ; Child, Preschool ; Female ; Heart Defects, Congenital/pathology ; Heart Defects, Congenital/surgery ; Humans ; Infant, Newborn ; Male ; Situs Inversus/pathology ; Vena Cava, Inferior/abnormalities ; Vena Cava, Inferior/surgery
    Language English
    Publishing date 2008-07
    Publishing country United States
    Document type Case Reports ; Journal Article
    ZDB-ID 1463498-3
    ISSN 1615-5742 ; 1093-5266
    ISSN (online) 1615-5742
    ISSN 1093-5266
    DOI 10.2350/07-01-0207.1
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 5063: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
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    Jg. 1995 - 2021: Lesesall (2.OG)
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  7. Article ; Online: Inhibition of IGF1-R overcomes IGFBP7-induced chemotherapy resistance in T-ALL.

    Bartram, Isabelle / Erben, Ulrike / Ortiz-Tanchez, Jutta / Blunert, Katja / Schlee, Cornelia / Neumann, Martin / Heesch, Sandra / Baldus, Claudia D

    BMC cancer

    2015  Volume 15, Page(s) 663

    Abstract: Background: T-cell acute lymphoblastic leukemia (T-ALL) is a genetically heterogeneous disease with the need for treatment optimization. Previously, high expression of Insulin-like growth factor binding protein 7 (IGFBP7), a member of the IGF system, ... ...

    Abstract Background: T-cell acute lymphoblastic leukemia (T-ALL) is a genetically heterogeneous disease with the need for treatment optimization. Previously, high expression of Insulin-like growth factor binding protein 7 (IGFBP7), a member of the IGF system, was identified as negative prognostic factor in adult T-ALL patients. Since aberrant IGFBP7 expression was observed in a variety of neoplasia and was relevant for prognosis in T-ALL, we investigated the functional role of IGFBP7 in Jurkat and Molt-4 cells as in vitro models for T-ALL.
    Methods: Jurkat and Molt-4 cells were stably transfected with an IGFBP7 over-expression vector or the empty vector as control. Proliferation of the cells was assessed by WST-1 assays and cell cycle status was measured by flow-cytometry after BrDU/7-AAD staining. The effect of IGFBP7 over-expression on sensitivity to cytostatic drugs was determined in AnnexinV/7-AAD assays. IGF1-R protein expression was measured by Western Blot and flow-cytometric analysis. IGF1-R associated gene expression profiles were generated from microarray gene expression data of 86 T-ALL patients from the Microarrays Innovations in Leukemia (MILE) multicenter study.
    Results: IGFBP7-transfected Jurkat cells proliferated less, leading to a longer survival in a nutrient-limited environment. Both IGFBP7-transfected Jurkat and Molt-4 cells showed an arrest in the G0/G1 cell cycle phase. Furthermore, Jurkat IGFBP7-transfected cells were resistant to vincristine and asparaginase treatment. Surface expression and whole protein measurement of IGF1-R protein expression showed a reduced abundance of the receptor after IGFBP7 transfection in Jurkat cells. Interestingly, combination of the IGF1-R inhibitor NPV-AEW541 restored sensitivity to vincristine in IGFBP7-transfected cells. Additionally, IGF1-R associated GEP revealed an up-regulation of important drivers of T-ALL pathogenesis and regulators of chemo-resistance and apoptosis such as NOTCH1, BCL-2, PRKCI, and TP53.
    Conclusion: This study revealed a proliferation inhibiting effect of IGFBP7 by G0/G1 arrest and a drug resistance-inducing effect of IGFBP7 against vincristine and asparaginase in T-ALL. These results provide a model for the previously observed association between high IGFBP7 expression and chemotherapy failure in T-ALL patients. Since the resistance against vincristine was abolished by IGF1-R inhibition, IGFBP7 could serve as biomarker for patients who may benefit from therapies including IGF1-R inhibitors in combination with chemotherapy.
    MeSH term(s) Antineoplastic Agents/pharmacology ; Cell Cycle Checkpoints/drug effects ; Cell Cycle Checkpoints/genetics ; Cell Line, Tumor ; Cell Proliferation ; Drug Resistance, Neoplasm/genetics ; Gene Expression ; Gene Expression Profiling ; Humans ; Insulin-Like Growth Factor Binding Proteins/genetics ; Jurkat Cells ; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma/genetics ; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma/metabolism ; Receptors, Somatomedin/antagonists & inhibitors ; Receptors, Somatomedin/metabolism ; Transcriptome
    Chemical Substances Antineoplastic Agents ; IGF1R protein, human ; Insulin-Like Growth Factor Binding Proteins ; Receptors, Somatomedin ; insulin-like growth factor binding protein-related protein 1
    Language English
    Publishing date 2015-10-08
    Publishing country England
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ISSN 1471-2407
    ISSN (online) 1471-2407
    DOI 10.1186/s12885-015-1677-z
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  8. Article: Heterotaxy syndrome -- asplenia and polysplenia as indicators of visceral malposition and complex congenital heart disease.

    Bartram, Ulrike / Wirbelauer, Johannes / Speer, Christian P

    Biology of the neonate

    2005  Volume 88, Issue 4, Page(s) 278–290

    Abstract: Heterotaxy results from failure of the developing embryo to establish normal left-right asymmetry. Typical manifestations include abnormal symmetry and malposition of the thoraco-abdominal organs and vessels, complex congenital heart disease and ... ...

    Abstract Heterotaxy results from failure of the developing embryo to establish normal left-right asymmetry. Typical manifestations include abnormal symmetry and malposition of the thoraco-abdominal organs and vessels, complex congenital heart disease and extracardiac defects involving midline-associated structures. The spleen is almost always affected, and there is syndromic clustering of the malformations corresponding to the type of splenic abnormality present. This review outlines the embryologic and genetic background of the heterotaxy syndrome as well as the characteristic anatomic features, clinical manifestations, and diagnostic clues of its two main presentations with asplenia or polysplenia.
    MeSH term(s) Activin Receptors, Type II/genetics ; Animals ; Electrocardiography ; Heart Defects, Congenital/diagnosis ; Heart Defects, Congenital/genetics ; Humans ; Infant, Newborn ; Lung/abnormalities ; Mice ; Situs Inversus/diagnosis ; Situs Inversus/genetics ; Spleen/abnormalities ; Syndrome ; Viscera/abnormalities
    Chemical Substances Activin Receptors, Type II (EC 2.7.11.30) ; activin receptor type II-B (EC 2.7.11.30)
    Language English
    Publishing date 2005
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 213537-1
    ISSN 0006-3126 ; 0523-6525
    ISSN 0006-3126 ; 0523-6525
    DOI 10.1159/000087625
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 391: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
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  9. Article ; Online: Pulmonary artery banding for idiopathic dilative cardiomyopathy: a novel therapeutic strategy using an old surgical procedure.

    Schranz, Dietmar / Veldman, Alex / Bartram, Ulrike / Michel-Behnke, Ina / Bauer, Jürgen / Akintürk, Hakan

    The Journal of thoracic and cardiovascular surgery

    2007  Volume 134, Issue 3, Page(s) 796–797

    MeSH term(s) Cardiac Surgical Procedures/methods ; Cardiomyopathy, Dilated/surgery ; Humans ; Infant ; Male ; Pulmonary Artery
    Language English
    Publishing date 2007-09
    Publishing country United States
    Document type Case Reports ; Journal Article
    ZDB-ID 3104-5
    ISSN 1097-685X ; 0022-5223
    ISSN (online) 1097-685X
    ISSN 0022-5223
    DOI 10.1016/j.jtcvs.2007.04.044
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    Uk I Zs.104: Show issues Location:
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  10. Article ; Online: Breast cancer risk in BRCA1/2 mutation carriers and noncarriers under prospective intensified surveillance.

    Engel, Christoph / Fischer, Christine / Zachariae, Silke / Bucksch, Karolin / Rhiem, Kerstin / Giesecke, Jutta / Herold, Natalie / Wappenschmidt, Barbara / Hübbel, Verena / Maringa, Monika / Reichstein-Gnielinski, Simone / Hahnen, Eric / Bartram, Claus R / Dikow, Nicola / Schott, Sarah / Speiser, Dorothee / Horn, Denise / Fallenberg, Eva M / Kiechle, Marion /
    Quante, Anne S / Vesper, Anne-Sophie / Fehm, Tanja / Mundhenke, Christoph / Arnold, Norbert / Leinert, Elena / Just, Walter / Siebers-Renelt, Ulrike / Weigel, Stefanie / Gehrig, Andrea / Wöckel, Achim / Schlegelberger, Brigitte / Pertschy, Stefanie / Kast, Karin / Wimberger, Pauline / Briest, Susanne / Loeffler, Markus / Bick, Ulrich / Schmutzler, Rita K

    International journal of cancer

    2019  Volume 146, Issue 4, Page(s) 999–1009

    Abstract: Comparably little is known about breast cancer (BC) risks in women from families tested negative for BRCA1/2 mutations despite an indicative family history, as opposed to BRCA1/2 mutation carriers. We determined the age-dependent risks of first and ... ...

    Abstract Comparably little is known about breast cancer (BC) risks in women from families tested negative for BRCA1/2 mutations despite an indicative family history, as opposed to BRCA1/2 mutation carriers. We determined the age-dependent risks of first and contralateral breast cancer (FBC, CBC) both in noncarriers and carriers of BRCA1/2 mutations, who participated in an intensified breast imaging surveillance program. The study was conducted between January 1, 2005, and September 30, 2017, at 12 university centers of the German Consortium for Hereditary Breast and Ovarian Cancer. Two cohorts were prospectively followed up for incident FBC (n = 4,380; 16,398 person-years [PY], median baseline age: 39 years) and CBC (n = 2,993; 10,090 PY, median baseline age: 42 years). Cumulative FBC risk at age 60 was 61.8% (95% CI 52.8-70.9%) for BRCA1 mutation carriers, 43.2% (95% CI 32.1-56.3%) for BRCA2 mutation carriers and 15.7% (95% CI 11.9-20.4%) for noncarriers. FBC risks were significantly higher than in the general population, with incidence rate ratios of 23.9 (95% CI 18.9-29.8) for BRCA1 mutation carriers, 13.5 (95% CI 9.2-19.1) for BRCA2 mutation carriers and 4.9 (95% CI 3.8-6.3) for BRCA1/2 noncarriers. Cumulative CBC risk 10 years after FBC was 25.1% (95% CI 19.6-31.9%) for BRCA1 mutation carriers, 6.6% (95% CI 3.4-12.5%) for BRCA2 mutation carriers and 3.6% (95% CI 2.2-5.7%) for noncarriers. CBC risk in noncarriers was similar to women with unilateral BC from the general population. Further studies are needed to confirm whether less intensified surveillance is justified in women from BRCA1/2 negative families with elevated risk.
    MeSH term(s) Adult ; Age Factors ; BRCA1 Protein/genetics ; BRCA2 Protein/genetics ; Breast Neoplasms/epidemiology ; Breast Neoplasms/genetics ; Epidemiological Monitoring ; Female ; Follow-Up Studies ; Genetic Predisposition to Disease ; Germany/epidemiology ; Heterozygote ; Humans ; Incidence ; Medical History Taking ; Middle Aged ; Mutation ; Prospective Studies ; Risk Assessment ; Risk Factors
    Chemical Substances BRCA1 Protein ; BRCA1 protein, human ; BRCA2 Protein ; BRCA2 protein, human
    Language English
    Publishing date 2019-05-22
    Publishing country United States
    Document type Journal Article ; Multicenter Study ; Research Support, Non-U.S. Gov't
    ZDB-ID 218257-9
    ISSN 1097-0215 ; 0020-7136
    ISSN (online) 1097-0215
    ISSN 0020-7136
    DOI 10.1002/ijc.32396
    Database MEDical Literature Analysis and Retrieval System OnLINE

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