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  1. Article: [Independent action of the Japanese Society of Neurology is a driving force behind the development of Japanese neurology].

    Araki, Shukuro

    Rinsho shinkeigaku = Clinical neurology

    2009  Volume 49, Issue 11, Page(s) 731–736

    Abstract: The Japanese Society of Neurology was founded in 1960. The Department of Neurology was established at The Kyushu University Faculty of Medicine in 1963, and then The Japanese Neurology has advanced. ...

    Abstract The Japanese Society of Neurology was founded in 1960. The Department of Neurology was established at The Kyushu University Faculty of Medicine in 1963, and then The Japanese Neurology has advanced.
    MeSH term(s) History, 20th Century ; Humans ; Japan ; Neurology/history ; Neurology/trends ; Societies, Medical/history
    Language Japanese
    Publishing date 2009-10-14
    Publishing country Japan
    Document type English Abstract ; Historical Article ; Journal Article
    ZDB-ID 604200-4
    ISSN 1882-0654 ; 0009-918X
    ISSN (online) 1882-0654
    ISSN 0009-918X
    DOI 10.5692/clinicalneurol.49.731
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  2. Article: [Contribution of Japanese researchers to progress in the field of neurology in the last 100 years: Minamata disease].

    Araki, Shukuro

    Nihon Naika Gakkai zasshi. The Journal of the Japanese Society of Internal Medicine

    2002  Volume 91, Issue 8, Page(s) 2304–2307

    MeSH term(s) History, 20th Century ; Humans ; Japan ; Mercury Poisoning, Nervous System/diagnosis ; Mercury Poisoning, Nervous System/etiology ; Mercury Poisoning, Nervous System/history ; Methylmercury Compounds/adverse effects ; Methylmercury Compounds/history ; Water Pollution, Chemical/adverse effects ; Water Pollution, Chemical/history
    Chemical Substances Methylmercury Compounds
    Language Japanese
    Publishing date 2002-08-10
    Publishing country Japan
    Document type Historical Article ; Journal Article
    ZDB-ID 952816-7
    ISSN 1883-2083 ; 0021-5384
    ISSN (online) 1883-2083
    ISSN 0021-5384
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  3. Article ; Online: Transthyretin-related familial amyloidotic polyneuropathy-Progress in Kumamoto, Japan (1967-2010)-.

    Araki, Shukuro / Ando, Yukio

    Proceedings of the Japan Academy. Series B, Physical and biological sciences

    2010  Volume 86, Issue 7, Page(s) 694–706

    Abstract: ... to 2009). Andrade (1952) first described a large group of patients with FAP in Portugal and Araki et al ...

    Abstract The authors reviewed contribution of Kumamoto University group to the progress of the studies on transthyretin (TTR)-related familial amyloidotic polyneuropathy (TTR-related FAP) for 42 years (from 1967 to 2009). Andrade (1952) first described a large group of patients with FAP in Portugal and Araki et al. (1967) in second discovered similar FAP patients in Arao, Kumamoto, Japan. Owing to progress in biochemical and molecular genetic analyses, FAP is now believed to occur worldwide. As of today, reports of about 100 different points of single or two mutations, or a deletion in the transthyretin (TTR) gene, have been published. The authors' group has made pioneer works for study of FAP in the world. The focus on therapy in amylodosis will increase sharply as an impetus in near future, and successful treatments are expected.
    MeSH term(s) Amyloid Neuropathies, Familial/epidemiology ; Amyloid Neuropathies, Familial/genetics ; Amyloid Neuropathies, Familial/physiopathology ; Amyloid Neuropathies, Familial/therapy ; Cities ; Humans ; Japan ; Liver Transplantation ; Prealbumin/chemistry ; Prealbumin/genetics ; Prealbumin/metabolism ; Prognosis
    Chemical Substances Prealbumin
    Language English
    Publishing date 2010-08-05
    Publishing country Japan
    Document type Journal Article ; Review
    ZDB-ID 161781-3
    ISSN 1349-2896 ; 0386-2208
    ISSN (online) 1349-2896
    ISSN 0386-2208
    DOI 10.2183/pjab.86.694
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article: [Astasia in a patient with unilateral thalamic infarction: clinical and MRI study].

    Misumi, Youhei / Nishida, Yasuto / Araki, Shukuro

    Rinsho shinkeigaku = Clinical neurology

    2006  Volume 46, Issue 9, Page(s) 649–651

    Abstract: We report a 82-year-old woman who developed difficulty in standing and sitting in the morning. She had no other complaints and stayed in the bed. The next day, she was admitted to the hospital and neurological examination revealed that she was alert, ... ...

    Abstract We report a 82-year-old woman who developed difficulty in standing and sitting in the morning. She had no other complaints and stayed in the bed. The next day, she was admitted to the hospital and neurological examination revealed that she was alert, with no other motor or sensory abnormalities. Finger to nose test, and knee to heel test were normal. No dysdiadochokinesia was seen. Astasia was the only observed abnormal finding. MRI showed a small infarction (14 x 8mm) in the posterolateral portion of the left thalamus (VPL-LP nucleus). During the following 15 days, her imbalance has gradually improved and then disappeared. We diagnosed the patient as astasia occurring from a small unilateral infarction in the thalamus. It is thought that thalamic astasia is caused by the disruption of afferent pathway from the vestibulocerebellum; however, this case is based on just clinical and MRI study, so physiological and pathological studies will be necessary in the future.
    MeSH term(s) Aged, 80 and over ; Cerebral Infarction/complications ; Cerebral Infarction/diagnosis ; Cerebral Infarction/pathology ; Female ; Gait Ataxia/etiology ; Humans ; Magnetic Resonance Imaging ; Thalamus/blood supply ; Thalamus/pathology
    Language Japanese
    Publishing date 2006-09
    Publishing country Japan
    Document type Case Reports ; English Abstract ; Journal Article
    ZDB-ID 604200-4
    ISSN 1882-0654 ; 0009-918X
    ISSN (online) 1882-0654
    ISSN 0009-918X
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  5. Article: [Patient with Gerstmann-Striussler-Scheinker syndrome (GSS P102L) presenting high intensity lesions in the cerebral cortex on diffusion weighted MRI].

    Misumi, Misumi / Nishida, Yasuto / Araki, Shukuro

    Rinsho shinkeigaku = Clinical neurology

    2006  Volume 46, Issue 4, Page(s) 291–293

    Abstract: A 59-year-old woman with Gerstmann-Sträussler-Scheinker syndrome (GSS P102L) was reported. She slowly developed progressive gait disturbance and limb ataxia by the age of 58, subsequently followed by dementia and myoclonus. EEG showed periodic ... ...

    Abstract A 59-year-old woman with Gerstmann-Sträussler-Scheinker syndrome (GSS P102L) was reported. She slowly developed progressive gait disturbance and limb ataxia by the age of 58, subsequently followed by dementia and myoclonus. EEG showed periodic synchronous discharges, and MRI by diffusion weighted imaging revealed abnormal high signal intensity lesions in the bilateral cerebral cortex and basal ganglia. A prorin-for-leution substitution at codon 102 of the prion protein gene was demonstrated; and thus, she was diagnosed as GSS (P102L). This is a case of GSS presenting high intensity lesions in the cerebral cortex on diffusion weighted MRI; it suggests that MRI findings disease stages in GSS.
    MeSH term(s) Atrophy ; Cerebral Cortex/pathology ; Codon/genetics ; Female ; Gerstmann-Straussler-Scheinker Disease/diagnosis ; Gerstmann-Straussler-Scheinker Disease/genetics ; Gerstmann-Straussler-Scheinker Disease/pathology ; Humans ; Magnetic Resonance Imaging ; Middle Aged ; Mutation ; Prions/genetics
    Chemical Substances Codon ; Prions
    Language Japanese
    Publishing date 2006-04
    Publishing country Japan
    Document type Case Reports ; English Abstract ; Journal Article
    ZDB-ID 604200-4
    ISSN 1882-0654 ; 0009-918X
    ISSN (online) 1882-0654
    ISSN 0009-918X
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  6. Article: Transthyretin-related familial amyloidotic polyneuropathy.

    Ando, Yukio / Nakamura, Masaaki / Araki, Shukuro

    Archives of neurology

    2005  Volume 62, Issue 7, Page(s) 1057–1062

    Abstract: Transthyretin-related familial amyloidotic polyneuropathy (FAP) is a fatal hereditary amyloidosis. Until 20 years ago, FAP was thought to be restricted to endemic occurrence in certain areas. However, owing to progress in biochemical and molecular ... ...

    Abstract Transthyretin-related familial amyloidotic polyneuropathy (FAP) is a fatal hereditary amyloidosis. Until 20 years ago, FAP was thought to be restricted to endemic occurrence in certain areas. However, owing to progress in biochemical and molecular genetic analyses, FAP is now believed to occur worldwide. As of today, reports of about 100 different points of single or double mutations, or a deletion in the transthyretin gene, have been published, and several different phenotypes of FAP have been documented, even for the same mutation in the transthyretin gene. We present herein the current clinicopathological, biochemical, molecular genetic, and epidemiological aspects of transthyretin-related FAP, and we introduce a new diagnostic procedure for the disease.
    MeSH term(s) Amyloid Neuropathies, Familial/epidemiology ; Amyloid Neuropathies, Familial/genetics ; Amyloid Neuropathies, Familial/physiopathology ; Genetic Testing ; Humans ; Incidence ; Liver Transplantation ; Prealbumin/genetics ; Prognosis
    Chemical Substances Prealbumin
    Language English
    Publishing date 2005-07
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 80049-1
    ISSN 1538-3687 ; 0003-9942
    ISSN (online) 1538-3687
    ISSN 0003-9942
    DOI 10.1001/archneur.62.7.1057
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  7. Article ; Online: Transforming growth factor beta induces IgA production and acts additively with interleukin 5 for IgA production. J. Exp. Med. 1989. 170: 1415-1420.

    Sonoda, Eiichiro / Matsumoto, Ryoji / Hitoshi, Yasumichi / Ishii, Takehisa / Sugimoto, Mineharu / Araki, Shukuro / Tominaga, Akira / Yamaguchi, Naoto / Takatsu, Kiyoshi

    Journal of immunology (Baltimore, Md. : 1950)

    2009  Volume 182, Issue 1, Page(s) 14–19

    MeSH term(s) Adjuvants, Immunologic/history ; Adjuvants, Immunologic/physiology ; Animals ; B-Lymphocytes/immunology ; Cell Line ; Cricetinae ; Cricetulus ; History, 20th Century ; Humans ; Immunoglobulin A/biosynthesis ; Immunoglobulin A/history ; Interleukin-5/history ; Interleukin-5/physiology ; Lipopolysaccharides/history ; Lipopolysaccharides/physiology ; Mice ; Mice, Inbred BALB C ; Transforming Growth Factor beta/history ; Transforming Growth Factor beta/physiology
    Chemical Substances Adjuvants, Immunologic ; Immunoglobulin A ; Interleukin-5 ; Lipopolysaccharides ; Transforming Growth Factor beta
    Language English
    Publishing date 2009-01-01
    Publishing country United States
    Document type Biography ; Classical Article ; Historical Article ; Journal Article
    ZDB-ID 3056-9
    ISSN 1550-6606 ; 0022-1767 ; 1048-3233 ; 1047-7381
    ISSN (online) 1550-6606
    ISSN 0022-1767 ; 1048-3233 ; 1047-7381
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  8. Book: Rinshōi no tame no byōtai seirigaku kōza

    Araki, Shukurō

    tadashii shindan to chiryō no tame ni = New integrated pathophysiology. Shinkei

    1986  

    Author's details Araki Shukurō henshū
    MeSH term(s) Nervous System/physiopathology
    Language Japanese
    Size 473 p. :, ill.
    Publisher Mejikarubyūsha
    Publishing place Tōkyō
    Document type Book
    Database Catalogue of the US National Library of Medicine (NLM)

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  9. Book: Shinkei

    Araki, Shukurō

    (Essential lecture)

    1985  

    Author's details henshū Araki Shukurō
    Series title Essential lecture
    MeSH term(s) Nervous System Diseases
    Language Japanese
    Size ix, 229 p. :, ill.
    Publisher Mejikarubyūsha : hatsubai Gurobyūsha
    Publishing place Tōkyō
    Document type Book
    Database Catalogue of the US National Library of Medicine (NLM)

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  10. Book: Shinkeibyō kēsu sutadi

    Araki, Shukurō

    71-rei no PO kenshū

    1980  

    Author's details henshū Araki Shukurō, Ōtomo Eiichi
    MeSH term(s) Nervous System Diseases
    Language Japanese
    Size vii, 326 p. :, ill.
    Edition Dai 1-han.
    Publisher Igaku Shoin
    Publishing place Tōkyō
    Document type Book
    Database Catalogue of the US National Library of Medicine (NLM)

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