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Article: Homenaje Dr Alejandro S Oría (1943-2014).

Frider, Bernardo

2015 Volume 45, Issue 1, Page(s) 97

Title translation	Homage Dr Alejandro S Oria (1943-2014).
MeSH term(s)	Argentina ; General Surgery/history ; History, 20th Century ; History, 21st Century
Language	Spanish
Publishing date	2015-03
Publishing country	Argentina
Document type	Biography ; Historical Article ; Journal Article
ZDB-ID	730001-3
ISSN	0300-9033
ISSN	0300-9033
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Article ; Online: Homenaje Dr Alejandro S Oría (1943 – 2014)

Bernardo Frider

Acta Gastroenterológica Latinoamericana, Vol 45, Iss 1, p

2015 Volume 97

Keywords	Diseases of the digestive system. Gastroenterology ; RC799-869 ; Specialties of internal medicine ; RC581-951 ; Internal medicine ; RC31-1245 ; Medicine ; R
Language	Spanish
Publishing date	2015-01-01T00:00:00Z
Publisher	Sociedad Argentina de Gastroenterología
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article ; Online: Treatment of liver hydatidosis

Bernardo Frider, Edmundo Larrieu

World Journal of Gastroenterology, Vol 16, Iss 33, Pp 4123-

How to treat an asymptomatic carrier?

2010 Volume 4129

Abstract: Liver hydatidosis is the most common clinical presentation of cystic echinococcosis (CE). Ultrasonographic mass surveys have demonstrated the true prevalence, including the asymptomatic characteristic of the majority of cases, providing new insight into ... ...

Abstract	Liver hydatidosis is the most common clinical presentation of cystic echinococcosis (CE). Ultrasonographic mass surveys have demonstrated the true prevalence, including the asymptomatic characteristic of the majority of cases, providing new insight into the natural history of the disease. This raises the question of whether to treat or not to treat these patients, due to the high and unsuspected prevalence of CE. The high rate of liver/lung frequencies of cyst localization, the autopsy findings, and the involution of cysts demonstrated in long time follow-up of asymptomatic carriers contribute to this discussion. The decision to treat an asymptomatic patient by surgery, albendazole, or puncture aspiration injection and reaspiration or to wait and watch, is based on conflicting reports in the literature, the lack of complications in untreated patients over time, and the spontaneous disappearance and involution of cysts. All these points contribute to difficulties of individual clinical decisions. The patients should be informed of the reasons and the risks of watchful/waiting without treatment, the possibility of complications, and the risks of the other options. As more information on the natural history of liver hydatidosis is acquired, selection of the best treatment will be come easier. Without this knowledge it would be very difficult to establish definitive rules of treatment. At present, it is possible to manage these patients over time and to wait for the best moment for treatment. Follow-up studies must be conducted to achieve this objective.
Keywords	Hydatid cyst ; Liver ; Hepatic cystic echinococcosis ; Albendazole ; Liver ultrasonography ; Puncture aspiration injection and reaspiration ; Ultrasonography screening ; Asymptomatic liver hydatidosis ; Diseases of the digestive system. Gastroenterology ; RC799-869 ; Specialties of internal medicine ; RC581-951 ; Internal medicine ; RC31-1245 ; Medicine ; R ; DOAJ:Gastroenterology ; DOAJ:Medicine (General) ; DOAJ:Health Sciences
Subject code	610
Language	English
Publishing date	2010-09-01T00:00:00Z
Publisher	Baishideng Publishing Group Co. Limited
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article ; Online: Treatment of liver hydatidosis: how to treat an asymptomatic carrier?

Frider, Bernardo / Larrieu, Edmundo

World journal of gastroenterology

2010 Volume 16, Issue 33, Page(s) 4123–4129

Abstract	Liver hydatidosis is the most common clinical presentation of cystic echinococcosis (CE). Ultrasonographic mass surveys have demonstrated the true prevalence, including the asymptomatic characteristic of the majority of cases, providing new insight into the natural history of the disease. This raises the question of whether to treat or not to treat these patients, due to the high and unsuspected prevalence of CE. The high rate of liver/lung frequencies of cyst localization, the autopsy findings, and the involution of cysts demonstrated in long time follow-up of asymptomatic carriers contribute to this discussion. The decision to treat an asymptomatic patient by surgery, albendazole, or puncture aspiration injection and re-aspiration or to wait and watch, is based on conflicting reports in the literature, the lack of complications in untreated patients over time, and the spontaneous disappearance and involution of cysts. All these points contribute to difficulties of individual clinical decisions. The patients should be informed of the reasons and the risks of watchful/waiting without treatment, the possibility of complications, and the risks of the other options. As more information on the natural history of liver hydatidosis is acquired, selection of the best treatment will be come easier. Without this knowledge it would be very difficult to establish definitive rules of treatment. At present, it is possible to manage these patients over time and to wait for the best moment for treatment. Follow-up studies must be conducted to achieve this objective.
MeSH term(s)	Albendazole/therapeutic use ; Animals ; Anthelmintics/therapeutic use ; Asymptomatic Diseases/therapy ; Biopsy, Fine-Needle ; Echinococcosis, Hepatic/drug therapy ; Echinococcosis, Hepatic/surgery ; Echinococcosis, Hepatic/therapy ; Echinococcus/isolation & purification ; Humans ; Liver/parasitology ; Liver/surgery
Chemical Substances	Anthelmintics ; Albendazole (F4216019LN)
Language	English
Publishing date	2010-10-07
Publishing country	United States
Document type	Editorial
ZDB-ID	2185929-2
ISSN	2219-2840 ; 1007-9327
ISSN (online)	2219-2840
ISSN	1007-9327
DOI	10.3748/wjg.v16.i33.4123
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Article: Drug-induced liver injury caused by adalimumab: a case report and review of the bibliography.

Frider, Bernardo / Bruno, Andres / Ponte, Marcelo / Amante, Marcelo

Case reports in hepatology

2013 Volume 2013, Page(s) 406901

Abstract: The most serious adverse drug reaction of adalimumab (ADR) is tuberculosis reactivation. We describe a case of a 35-year-old man, with rheumatoid arthritis (RA) and hepatitis C virus genotype 1a with a liver biopsy in 2001 with a METAVIR score pattern A1 ...

Abstract	The most serious adverse drug reaction of adalimumab (ADR) is tuberculosis reactivation. We describe a case of a 35-year-old man, with rheumatoid arthritis (RA) and hepatitis C virus genotype 1a with a liver biopsy in 2001 with a METAVIR score pattern A1 F0; he received interferon alpha 2b for six months, but treatment was suspended because of reactivation of RA. Liver function tests after treatment were similar to previous ones showing a minimal cholestatic pattern. In 2008, methotrexate was prescribed, but the drug was withdrawn at the third month because of the appearance of pruritus and Ggt rise. Viral load at that moment was 9300000 UI/mL, log 6,9. The liver biopsy showed a Metavir Score A2 F1. Adalimumab was started in 2010, and at the third month of treatment, Ggt showed a rise of 23 times normal value (NV), alkaline phosphatase 2,5 times NV with AST and ALT with no change. A new liver biopsy showed portal inflammation with eosinophils and a METAVIR A1 F2. We think that adalimumab appears to be responsible for the liver injury, because of temporal relationship, liver biopsy findings, other clinical conditions being discarded, and the improvement of clinical symptoms and biochemical abnormalities when adalimumab was suspended.
Language	English
Publishing date	2013-05-14
Publishing country	United States
Document type	Journal Article
ISSN	2090-6587
ISSN	2090-6587
DOI	10.1155/2013/406901
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Article ; Online: Drug-Induced Liver Injury Caused by Adalimumab

Bernardo Frider / Andres Bruno / Marcelo Ponte / Marcelo Amante

Case Reports in Hepatology, Vol

A Case Report and Review of the Bibliography

2013 Volume 2013

Keywords	Diseases of the digestive system. Gastroenterology ; RC799-869 ; Specialties of internal medicine ; RC581-951 ; Internal medicine ; RC31-1245 ; Medicine ; R ; DOAJ:Gastroenterology ; DOAJ:Medicine (General) ; DOAJ:Health Sciences
Language	English
Publishing date	2013-01-01T00:00:00Z
Publisher	Hindawi Publishing Corporation
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article: Autoimmune cholangitis associated to IgG4 related sclerosing disease.

Frider, Bernardo / Bruno, Andrés / Zylberman, Marcelo / Oría, Alejandro / Amante, Marcelo

Acta gastroenterologica Latinoamericana

2011 Volume 41, Issue 1, Page(s) 55–59

Abstract: The IgG4-related sclerosing disease is characterized by the presence of plasmatic IgG4 positive cells and T-lymphocytes infiltration in different organs. We herein report a case of cholestasis due to autoimmune cholangitis associated to IgG4 disease. A ... ...

Abstract	The IgG4-related sclerosing disease is characterized by the presence of plasmatic IgG4 positive cells and T-lymphocytes infiltration in different organs. We herein report a case of cholestasis due to autoimmune cholangitis associated to IgG4 disease. A 40-year-old woman with a history of pruritus, anosmia, Sjögren's syndrome and diabetes, was referred for a pancreatic tumor. Alkaline phosphatase was 24-fold upper limit of normal (ULN), gamma-glutamyl transpeptidase 21-fold ULN, aspartate aminotransferase 3-fold ULN, alanine aminotransferase 2-fold ULN, cholesterol 408 mg/dL, bilirubin normal, gamma-globulin 3.92 g/dL, IgG4 4.6 g/L, antinuclear antibody positive (1/320), and antimitochondrial antibodies negative. Ultrasound scan (US) showed a mass in the pancreatic head and thickening of the gallbladder and the bile duct walls. Dilation and strictures of the main pancreatic duct and intrahepatic bile ducts were detected by MR cholangiopancreatography. Liver biopsy showed chronic inflammatory lesions, ductal damage (autoimmune cholangitis) (METAVIRA2, F2) and IgG4 bearing plasmatic cells. A cervical lymph node showed IgG4 bearing plasmatic cells. After 2 weeks of treatment with meprednisone, ursodeoxycholic acid and insulin, pruritus and anosmia disappeared. After eleven months of treatment imaging studies showed disappearance of the pancreatic tumor, atrophy of the body and the pancreatic tail and normal biochemical parameters, except for alkaline phosphatase 2-fold ULN. The final diagnosis of our patient was autoimmune hepatitis with cholangitis associated to IgG4 systemic diseases.
MeSH term(s)	Adult ; Autoimmune Diseases/complications ; Autoimmune Diseases/diagnosis ; Cholangitis, Sclerosing/complications ; Cholangitis, Sclerosing/diagnosis ; Cholestasis/diagnosis ; Cholestasis/etiology ; Female ; Humans ; Immunoglobulin G/blood ; Immunoglobulin G/immunology
Chemical Substances	Immunoglobulin G
Language	English
Publishing date	2011-03
Publishing country	Argentina
Document type	Case Reports ; Journal Article
ZDB-ID	730001-3
ISSN	0300-9033
ISSN	0300-9033
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Article: Tratamiento de las hepatitis virales en coinfectados HIV-HBV-HCV.

Manero, Estela / Frider, Bernardo / Landeira, Graciela

Acta gastroenterologica Latinoamericana

2006 Volume 36 Suppl 1, Page(s) S59–60; discussion S74–82

Title translation	Treatment of viral hepatitis in HIV-HBV-HCV co-infected patients.
MeSH term(s)	Antiviral Agents/therapeutic use ; HIV Infections/complications ; HIV Infections/drug therapy ; Hepatitis B/complications ; Hepatitis B/drug therapy ; Hepatitis C/complications ; Hepatitis C/drug therapy ; Humans
Chemical Substances	Antiviral Agents
Language	Spanish
Publishing date	2006-06
Publishing country	Argentina
Document type	Journal Article
ZDB-ID	730001-3
ISSN	0300-9033
ISSN	0300-9033
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Article: Reversal of advanced fibrosis after long-term ursodeoxycholic acid therapy in a patient with residual expression of MDR3.

Frider, Bernardo / Castillo, Amalia / Gordo-Gilart, Raquel / Bruno, Andrés / Amante, Marcelo / Alvarez, Luis / Mathet, Verónica

Annals of hepatology

2015 Volume 14, Issue 5, Page(s) 745–751

Abstract: Introduction: Progressive familial intrahepatic cholestasis type 3 (PFIC-3) is a severe liver disorder associated with inherited dysfunction of multidrug resistance protein 3 (MDR3/ABCB4), which functions as a phospholipid floppase, translocating ... ...

Abstract	Introduction: Progressive familial intrahepatic cholestasis type 3 (PFIC-3) is a severe liver disorder associated with inherited dysfunction of multidrug resistance protein 3 (MDR3/ABCB4), which functions as a phospholipid floppase, translocating phosphatidylcholine from the inner to the outer hemileaflet of the canalicular membrane of hepatocytes. MDR3 deficiency results in a disbalanced bile which may damage the luminal membrane of cells of the hepatobiliary system. We evaluated clinical, biochemical and histological improvement in a genetically proven PFIC-3 patient after long-term ursodeoxycholic acid (UDCA) administration. Material and methods: A PFIC-3 patient and a relative with cholestatic liver disease were studied. Hepatic MDR3 expression was analyzed by immunohistochemistry and ABCB4 mutations were identified. The effect of the mutations on MDR3 expression and subcellular localization was studied in vitro. Results: A 23-year-old man presented cholestasis with severe fibrosis and incomplete cirrhosis. Canalicular staining for MDR3 was faint. Sequence analysis of ABCB4 revealed two missense mutations that reduce drastically protein expression levels. After 9 years of treatment with UDCA disappearance of fibrosis and cirrhosis was achieved. Conclusion: These data indicate that fibrosis associated with MDR3 deficiency can be reversed by long-term treatment with UDCA, at least when there is residual expression of the protein.
MeSH term(s)	ATP Binding Cassette Transporter, Subfamily B/deficiency ; ATP Binding Cassette Transporter, Subfamily B/genetics ; ATP Binding Cassette Transporter, Subfamily B/metabolism ; Animals ; Cholagogues and Choleretics/therapeutic use ; Cholestasis, Intrahepatic/diagnosis ; Cholestasis, Intrahepatic/drug therapy ; Cholestasis, Intrahepatic/genetics ; DNA Mutational Analysis ; Dogs ; Elasticity Imaging Techniques ; Genetic Predisposition to Disease ; HEK293 Cells ; Humans ; Immunohistochemistry ; Liver Cirrhosis/diagnosis ; Liver Cirrhosis/drug therapy ; Liver Cirrhosis/genetics ; Madin Darby Canine Kidney Cells ; Male ; Mutation, Missense ; Phenotype ; Remission Induction ; Severity of Illness Index ; Time Factors ; Transfection ; Treatment Outcome ; Ursodeoxycholic Acid/therapeutic use ; Young Adult
Chemical Substances	ATP Binding Cassette Transporter, Subfamily B ; Cholagogues and Choleretics ; Ursodeoxycholic Acid (724L30Y2QR) ; multidrug resistance protein 3 (9EI49ZU76O)
Language	English
Publishing date	2015-08-07
Publishing country	Mexico
Document type	Case Reports ; Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2188733-0
ISSN	1665-2681
ISSN	1665-2681
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Article: Hidatidosis: Catastro ecográfico en la Provincia de Río Negro 25 anos después del primer catastro.

Salviti, Juan Carlos / Sobrino, Mariano / Del Carpio, Mario / Mercapide, Carlos / Uchiumi, Leonardo / Moguilensky, Jorge / Moguilansky, Sergio / Frider, Bernardo / Larrieu, Edmundo

Acta gastroenterologica Latinoamericana

2015 Volume 45, Issue 1, Page(s) 51–55

Abstract: ... In 1980 started a control program against the disease. In 1984 Frider et al performed the first ultrasound ...

Title translation	Hydatidosis: Ultrasonographyc screening in the Río Negro Province 25 years after the first screening.
Abstract	Introduction: Hydatidosis (cystic echinococcosis) is endemic in Rio Negro Province, Argentina. In 1980 started a control program against the disease. In 1984 Frider et al performed the first ultrasound screening in the world at Pilcaniyeu city, later extended to other localities of Rio Negro province. The initial prevalence in asymptomatic people was 7.1% in Pilcaniyeu and 10.1% in Comallo, being all new cases and with surgical indication. Objective: The aim of this investigation was to determine the current prevalence and analyze the evolution of the disease across 25 years of the control program. Materials and methods: In 2009 and 2010 ultrasound screening studies were conducted in both locations in all age groups. Results: In 512 ultrasound studies at Pilcaniyeu, the prevalence was 1.5% in children (6 to 14years old) and 4.2% in adults (total 2.5%). In 770 ultrasound studies at Comallo, the prevalence was 1.1 %in children and 6.6% in adults (total 3.0%). The overall reduction in the prevalence reached 67.2%. Regarding the age distribution, rates of 1.6%-1.9% were observed in Pilcaniyeu and of 1.0-1.9% in Comallo between 0 and 30 years old, increasing significantly above 10% after 60 years old in Pilcaniyeu and after 40 years old in Comallo. Conclusions: The implementation of the program actions reduced the prevalence of the disease but there are still new cases, and that indicates that some bias persists in the control of the epidemiology of the disease and levels of transmission to humans as a consequence of this failures. So the search of asymptomatic cases is still important and also their management based on the follow-up by ultrasound (watch and wait) or in the treatment with albendazol.
MeSH term(s)	Adult ; Aged ; Argentina/epidemiology ; Child ; Child, Preschool ; Echinococcosis/diagnostic imaging ; Echinococcosis/epidemiology ; Echinococcosis/prevention & control ; Female ; Humans ; Infant ; Male ; Middle Aged ; Prevalence ; Ultrasonography ; Young Adult
Language	Spanish
Publishing date	2015-03
Publishing country	Argentina
Document type	Journal Article
ZDB-ID	730001-3
ISSN	0300-9033
ISSN	0300-9033
Database	MEDical Literature Analysis and Retrieval System OnLINE

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