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Book ; Online ; E-Book: Kala Azar in South Asia

Noiri, Eisei / Jha, T.K.

Current status and sustainable challenges

2017

Author's details	Eisei Noiri, T.K. Jha editors
Keywords	Medicine ; Laboratory medicine ; Drug resistance ; Parasitology ; Nephrology
Subject code	616.96
Language	English
Size	1 Online-Ressource (XX, 309 Seiten)
Edition	Second edition
Publisher	Springer
Publishing place	Cham
Publishing country	Switzerland
Document type	Book ; Online ; E-Book
Remark	Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
HBZ-ID	HT019260164
ISBN	978-3-319-47101-3 ; 9783319436111 ; 3-319-47101-5 ; 3319436112
DOI	10.1007/978-3-319-47101-3
Database	ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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Book: The concise manual of apheresis therapy

Noiri, Eisei / Hanafusa, Norio

2014

Author's details	Eisei Noiri ; Norio Hanafusa ed
Language	English
Size	XXVII, 418 S. : Ill., graph. Darst.
Publisher	Springer
Publishing place	Tokyo u.a.
Publishing country	Japan
Document type	Book
HBZ-ID	HT018126031
ISBN	978-4-431-54411-1 ; 4-431-54411-9 ; 9784431544128 ; 4431544127
Database	Catalogue ZB MED Medicine, Health

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2014 A 109	available for loan (reading room)	Lesesaal EG

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Book: Kala Azar in South Asia

Jha, T. K. / Noiri, Eisei

current status and challenges ahead

2011

Author's details	T. K. Jha ; E. Noiri, ed
Language	English
Size	XI, 148 S. : Ill., graph. Darst., Kt.
Publisher	Springer
Publishing place	Dordrecht u.a.
Publishing country	Great Britain
Document type	Book
HBZ-ID	HT016681627
ISBN	978-94-007-0276-9 ; 94-007-0276-0 ; 9789400702776 ; 9400702779
Database	Catalogue ZB MED Medicine, Health

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2011 A 442	order for loan	Magazin

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Article ; Online: Urine oxygenation predicts COVID-19 risk.

Noiri, Eisei / Katagiri, Daisuke / Asai, Yusuke / Sugaya, Takeshi / Tokunaga, Katsushi

Clinical and experimental nephrology

2024

Abstract: Since February, 2023, the omicron variant has accounted for essentially all new coronavirus infections in Japan. If future infections involve mutant strains with the same level of infectivity and virulence as omicron, the government's basic policy will ... ...

Abstract	Since February, 2023, the omicron variant has accounted for essentially all new coronavirus infections in Japan. If future infections involve mutant strains with the same level of infectivity and virulence as omicron, the government's basic policy will be to prevent the spread of infection, without compromising socioeconomic activities. Objectives include protecting pregnant women and elderly persons, and focusing on citizens requiring hospitalization and those at risk of serious illness, without imposing new social restrictions. Although the government tries to raise public awareness through education, most people affected by COVID-19 stay at home, and by the time patients become aware of the seriousness of their disease, it has often reached moderate or higher severity. In this review, we discuss why this situation persists even though the disease seems to have become milder with the shift from the delta variant to omicron. We also propose a pathophysiological method to determine the risk of severe illness. This assessment can be made at home in the early stages of COVID-19 infection, using urine analysis. Applicability of this method to drug discovery and development is also discussed.
Language	English
Publishing date	2024-02-24
Publishing country	Japan
Document type	Journal Article ; Review
ZDB-ID	1338768-6
ISSN	1437-7799 ; 1342-1751
ISSN (online)	1437-7799
ISSN	1342-1751
DOI	10.1007/s10157-023-02456-5
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Zs.A 4922: Show issues

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Article ; Online: HMGB1 Is a Prognostic Factor for Mortality in Acute Kidney Injury Requiring Renal Replacement Therapy.

Matsuura, Ryo / Komaru, Yohei / Miyamoto, Yoshihisa / Yoshida, Teruhiko / Yoshimoto, Kohei / Yamashita, Tetsushi / Hamasaki, Yoshifumi / Noiri, Eisei / Nangaku, Masaomi / Doi, Kent

Blood purification

2023 Volume 52, Issue 7-8, Page(s) 660–667

Abstract: Instruction: High mobility group box 1 (HMGB1) is a pro-inflammatory cytokine that reportedly causes kidney injury and other organ damage in rodent acute kidney injury (AKI) models. However, it remains unclear whether HMGB1 is associated with clinical ... ...

Abstract	Instruction: High mobility group box 1 (HMGB1) is a pro-inflammatory cytokine that reportedly causes kidney injury and other organ damage in rodent acute kidney injury (AKI) models. However, it remains unclear whether HMGB1 is associated with clinical AKI and related outcomes. This study aimed to evaluate the association with HMGB1 and prognosis of AKI requiring continuous renal replacement therapy (CRRT). Methods: AKI patients treated with CRRT in our intensive care unit were enrolled consecutively during 2013-2016. Plasma HMGB1 was measured on initiation. Classic initiation was defined as presenting at least one of the following conventional indications: hyperkalemia (K ≥6.5 mEq/L), severe acidosis (pH <7.15), uremia (UN >100 mg/dL), and diuretics-resistant pulmonary edema. Early initiation was defined as presenting no conventional indications. The primary outcome was defined as 90-day mortality. Results: A total of 177 AKI patients were enrolled in this study. HMGB1 was significantly associated with the primary outcome (hazard ratio, 1.06; 95% CI, 1.04-1.08). When the patients were divided into two-by-two groups by the timing of CRRT initiation and the HMBG1 cutoff value obtained by receiver operating curve (ROC) analysis, the high HMGB1 group (>10 ng/mL) with classic initiation was significantly associated with the primary outcome compared with the others, even after adjusting for other factors including the nonrenal serial organ failure assessment (SOFA) score. Conclusion: HMGB1 was associated with 90-day mortality in AKI patients requiring CRRT. Notably, the highest mortality was observed in the high HMGB1 group with classic initiation. These findings suggest that CRRT should be considered for AKI patients with high HMGB1, regardless of the conventional indications.
MeSH term(s)	Humans ; Continuous Renal Replacement Therapy ; Prognosis ; HMGB1 Protein ; Renal Replacement Therapy ; Intensive Care Units ; Acute Kidney Injury ; Retrospective Studies
Chemical Substances	HMGB1 Protein
Language	English
Publishing date	2023-06-19
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	605548-5
ISSN	1421-9735 ; 0253-5068
ISSN (online)	1421-9735
ISSN	0253-5068
DOI	10.1159/000530774
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Zs.A 1872: Show issues

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Article ; Online: Different Roles of Functional and Structural Renal Markers Measured at Discontinuation of Renal Replacement Therapy for Acute Kidney Injury.

Yoshida, Teruhiko / Matsuura, Ryo / Komaru, Yohei / Miyamoto, Yoshihisa / Yoshimoto, Kohei / Hamasaki, Yoshifumi / Noiri, Eisei / Nangaku, Masaomi / Doi, Kent

Blood purification

2023 Volume 52, Issue 9-10, Page(s) 786–792

Abstract: Introduction: Severe acute kidney injury (AKI) requiring renal replacement therapy (RRT) has been associated with an unacceptably high mortality of 50% or more. Successful discontinuation of RRT is thought to be linked to better outcomes. Although ... ...

Abstract	Introduction: Severe acute kidney injury (AKI) requiring renal replacement therapy (RRT) has been associated with an unacceptably high mortality of 50% or more. Successful discontinuation of RRT is thought to be linked to better outcomes. Although functional and structural renal markers have been evaluated in AKI, little is known about their roles in predicting outcomes at the time of RRT discontinuation. Methods: In this prospective single-center cohort study, we analyzed patients who received continuous RRT (CRRT) for AKI between August 2016 and March 2018 in the intensive care unit of the University of Tokyo Hospital (Tokyo, Japan). Clinical parameters and urine samples were obtained at CRRT discontinuation. Successful CRRT discontinuation was defined as neither resuming CRRT for 48 h nor receiving intermittent hemodialysis for 7 days from the CRRT termination. Major adverse kidney events (MAKEs) were defined as death, requirement for dialysis, or a decrease in the estimated glomerular filtration rate (eGFR) of more than 25% from the baseline at day 90. Results: Of 73 patients, who received CRRT for AKI, 59 successfully discontinued CRRT and 14 could not. Kinetic eGFR, urine volume, urinary neutrophil gelatinase-associated lipocalin (NGAL), and urinary L-type fatty acid binding protein were predictive for CRRT discontinuation. Of these factors, urine volume had the highest area under the curve (AUC) 0.91 with 95% confidence interval [0.80-0.96] for successful CRRT discontinuation. For predicting MAKEs at day 90, the urinary NGAL showed the highest AUC 0.76 [0.62-0.86], whereas kinetic eGFR and urine volume failed to show statistical significance (AUC 0.49 [0.35-0.63] and AUC 0.59 [0.44-0.73], respectively). Conclusions: Our prospective study confirmed that urine volume, a functional renal marker, predicted successful discontinuation of RRT and that urinary NGAL, a structural renal marker, predicted long-term renal outcomes. These observations suggest that the functional and structural renal makers play different roles in predicting the outcomes of severe AKI requiring RRT.
MeSH term(s)	Humans ; Continuous Renal Replacement Therapy/adverse effects ; Lipocalin-2/urine ; Prospective Studies ; Cohort Studies ; Renal Dialysis ; Biomarkers/urine ; Renal Replacement Therapy/adverse effects ; Acute Kidney Injury ; Kidney/metabolism
Chemical Substances	Lipocalin-2 ; Biomarkers
Language	English
Publishing date	2023-09-27
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	605548-5
ISSN	1421-9735 ; 0253-5068
ISSN (online)	1421-9735
ISSN	0253-5068
DOI	10.1159/000532034
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Zs.A 1872: Show issues

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Article ; Online: Mitochondrial Dysfunction in Cardiorenal Syndrome.

Doi, Kent / Noiri, Eisei

Antioxidants & redox signaling

2016 Volume 25, Issue 4, Page(s) 200–207

Abstract: Significance: Acute kidney injury (AKI) has a significant impact on the outcomes of critically ill patients, although no effective and specific treatment against AKI is currently available in the clinical setting. It is assumed that reactive oxygen ... ...

Abstract	Significance: Acute kidney injury (AKI) has a significant impact on the outcomes of critically ill patients, although no effective and specific treatment against AKI is currently available in the clinical setting. It is assumed that reactive oxygen species production by the mitochondria plays a crucial role in renal damage especially caused by cellular apoptosis. Mitochondrial injury in the heart is reported as an important determinant of myocardial contractility. Clinical epidemiological data indicate that remote organ effects induced by AKI, especially organ cross talk between the kidney and heart, might contribute to the poor outcome of AKI patients. Recent advances: Cardiorenal syndrome (CRS) has recently been defined based on clinical observations that acute and chronic heart failure causes kidney injury and AKI and that chronic kidney disease worsens heart diseases. Possible pathways that connect these two organs have been suggested; however, the precise mechanisms are still unclarified. Mitochondrial injury in the kidney and heart has been shown as a crucial pathway of AKI and acute heart failure by several animal studies. Critical issues: Clinical evidence clearly shows cardiorenal interactions in clinically ill patients, but evidence for distant organ effects of AKI on the heart is lacking. We recently found dysregulation of mitochondrial dynamics caused by increased Drp1 expression and cellular apoptosis of the heart in an experimental AKI animal model of renal ischemia-reperfusion. Future directions: Precise mechanisms that induce cardiac mitochondrial injury in AKI remain unclarified. A recently suggested concept of mitochondrial hormesis may need to be considered in chronic cardiorenal interaction. Identifying the role of mitochondrial injury for CRS will enable the development of novel interventional approaches to reduce mortality associated with AKI. Antioxid. Redox Signal. 25, 200-207.
MeSH term(s)	Acute Kidney Injury/diagnosis ; Acute Kidney Injury/etiology ; Acute Kidney Injury/metabolism ; Animals ; Cardio-Renal Syndrome/diagnosis ; Cardio-Renal Syndrome/etiology ; Cardio-Renal Syndrome/metabolism ; Heart Diseases/diagnosis ; Heart Diseases/etiology ; Heart Diseases/metabolism ; Humans ; Mitochondria/genetics ; Mitochondria/metabolism ; Mitochondrial Dynamics
Language	English
Publishing date	2016-08-01
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	1483836-9
ISSN	1557-7716 ; 1523-0864
ISSN (online)	1557-7716
ISSN	1523-0864
DOI	10.1089/ars.2016.6654
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Zs.A 5488: Show issues

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Article ; Online: Clinical Utility of a Biomarker to Detect Contrast-Induced Acute Kidney Injury during Percutaneous Cardiovascular Procedures.

Peabody, John / Paculdo, David / Valdenor, Czarlota / McCullough, Peter A / Noiri, Eisei / Sugaya, Takeshi / Dahlen, Jeffrey R

Cardiorenal medicine

2022 Volume 12, Issue 1, Page(s) 11–19

Abstract: Introduction: Contrast-induced acute kidney injury (CI-AKI) is a major clinical complication of percutaneous cardiovascular procedures requiring iodinated contrast. Despite its relative frequency, practicing physicians are unlikely to identify or treat ... ...

Abstract	Introduction: Contrast-induced acute kidney injury (CI-AKI) is a major clinical complication of percutaneous cardiovascular procedures requiring iodinated contrast. Despite its relative frequency, practicing physicians are unlikely to identify or treat this condition. Methods: In a 2-round clinical trial of simulated patients, we examined the clinical utility of a urine-based assay that measures liver-type fatty acid-binding protein (L-FABP), a novel marker of CI-AKI. We sought to determine if interventional cardiologists' ability to diagnose and treat potential CI-AKI improved using the biomarker assay for 3 different patient types: pre-procedure, peri-procedure, and post-procedure patients. Results: 154 participating cardiologists were randomly divided into either control or intervention. At baseline, we found no difference in the demographics or how they identified and treated potential complications of AKI, with both groups providing less than half the necessary care to their patients (46.4% for control vs. 47.6% for intervention, p = 0.250). The introduction of L-FABP into patient care resulted in a statistically significant improvement of 4.6% (p = 0.001). Compared to controls, physicians receiving L-FABP results were 2.9 times more likely to correctly identify their patients' risk for AKI (95% CI 2.1-4.0) and were more than twice as likely to treat for AKI by providing volume expansion and withholding nephrotoxic medications. We found the greatest clinical utility in the pre-procedure and peri-procedure settings but limited value in the post-procedure setting. Conclusion: This study suggests L-FABP as a clinical marker for assessing the risk of potential CI-AKI, has clinical utility, and can lead to more accurate diagnosis and treatment.
MeSH term(s)	Acute Kidney Injury/chemically induced ; Acute Kidney Injury/diagnosis ; Biomarkers ; Cardiologists ; Contrast Media/adverse effects ; Humans
Chemical Substances	Biomarkers ; Contrast Media
Language	English
Publishing date	2022-01-14
Publishing country	Switzerland
Document type	Journal Article ; Randomized Controlled Trial ; Research Support, Non-U.S. Gov't
ZDB-ID	2595659-0
ISSN	1664-5502 ; 1664-3828
ISSN (online)	1664-5502
ISSN	1664-3828
DOI	10.1159/000520820
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Article ; Online: Urinary liver-type fatty acid binding protein is increased in the early stages of the disease with a risk of acute kidney injury induced by histone.

Ohata, Keiichi / Sugaya, Takeshi / Nguyen, Hanh Nhung / Hatanaka, Yuri / Uno, Kinuko / Tohma, Marika / Oikawa, Tsuyoshi / Nagabukuro, Hiroshi / Kuniyeda, Kanako / Kamijo-Ikemori, Atsuko / Suzuki-Kemuriyama, Noriko / Nakae, Dai / Noiri, Eisei / Miyajima, Katsuhiro

Nephrology (Carlton, Vic.)

2023 Volume 28, Issue 6, Page(s) 345–355

Abstract: Aim: Urinary liver-type fatty acid binding protein (L-FABP) has potential utility as an early prognostic biomarker ahead of traditional severity scores in coronavirus disease 2019 and sepsis, however, the mechanism of elevated urinary L-FABP in the ... ...

Abstract	Aim: Urinary liver-type fatty acid binding protein (L-FABP) has potential utility as an early prognostic biomarker ahead of traditional severity scores in coronavirus disease 2019 and sepsis, however, the mechanism of elevated urinary L-FABP in the disease has not been clearly elucidated. We investigated the background mechanisms of urinary L-FABP excretion through non-clinical animal model focusing on histone, which is one of the aggravating factors in these infectious diseases. Methods: Male Sprague-Dawley rats were placed in central intravenous catheters, and these rats were given a continuous intravenous infusion of 0.25 or 0.5 mg/kg/min calf thymus histones for 240 min from caudal vena cava. Results: After the administration of histone, urinary L-FABP and gene expression of an oxidative stress marker in the kidney increased in a histone dose-dependent manner before increased serum creatinine. Upon further investigation, fibrin deposition in the glomerulus was observed and it tended to be remarkable in the high dose administrated groups. The levels of coagulation factor were significantly changed after the administration of histone, and these were significantly correlated with the levels of urinary L-FABP. Conclusions: Firstly, it was suggested that histone is one of the causative agents for the urinary L-FABP increase at an early stage of the disease with a risk of acute kidney injury. Secondly, urinary L-FABP could be a marker reflecting the changes of coagulation system and microthrombus caused by histone in the early stage of acute kidney injury before becoming severely ill and maybe a guide to early treatment initiation.
MeSH term(s)	Male ; Animals ; Rats ; Histones ; Rats, Sprague-Dawley ; Biomarkers ; COVID-19/complications ; Acute Kidney Injury/diagnosis ; Acute Kidney Injury/etiology ; Fatty Acid-Binding Proteins ; Liver
Chemical Substances	Histones ; Biomarkers ; Fatty Acid-Binding Proteins
Language	English
Publishing date	2023-04-19
Publishing country	Australia
Document type	Journal Article
ZDB-ID	1303661-0
ISSN	1440-1797 ; 1320-5358
ISSN (online)	1440-1797
ISSN	1320-5358
DOI	10.1111/nep.14162
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Zs.A 4822: Show issues

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Article ; Online: PpSP32-like protein as a marker of human exposure to Phlebotomus argentipes in Leishmania donovani foci in Bangladesh.

Sumova, Petra / Sanjoba, Chizu / Willen, Laura / Polanska, Nikola / Matsumoto, Yoshitsugu / Noiri, Eisei / Paul, Shyamal Kumar / Ozbel, Yusuf / Volf, Petr

International journal for parasitology

2021 Volume 51, Issue 12, Page(s) 1059–1068

Abstract: Phlebotomus argentipes is a predominant vector of Leishmania donovani, the protozoan parasite causing visceral leishmaniasis in the Indian subcontinent. In hosts bitten by P. argentipes, sand fly saliva elicits the production of specific anti-salivary ... ...

Abstract	Phlebotomus argentipes is a predominant vector of Leishmania donovani, the protozoan parasite causing visceral leishmaniasis in the Indian subcontinent. In hosts bitten by P. argentipes, sand fly saliva elicits the production of specific anti-salivary protein antibodies. Here, we have utilised these antibodies as markers of human exposure to P. argentipes in a visceral leishmaniasis endemic area in Pabna district, Bangladesh. The use of whole salivary gland homogenate as an antigen to detect these antibodies has several limitations, therefore it is being superseded by the use of specific recombinant salivary proteins. We have identified three major P. argentipes salivary antigenic proteins recognised by sera of bitten humans, expressed them in a recombinant form (rPagSP04, rPagSP05 and rPagSP06) and tested their applicability in ELISA and immunoblot. One of them, PpSP32-like protein rPagSP06, was identified as the most promising antigen, showing highest resemblance and correlation with the IgG response to P. argentipes salivary gland homogenate. Furthermore, we have validated the applicability of rPagSP06 in a large cohort of 585 individuals and obtained a high correlation coefficient for anti-rPagSP06 and anti-P. argentipes saliva IgG responses. The anti-rPagSP06 and anti-P. argentipes salivary gland homogenate IgG responses followed a similar right-skewed distribution. This is the first report of screening human sera for anti-P. argentipes saliva antibodies using recombinant salivary protein. The rPagSP06 was proven to be a valid antigen for screening human sera for exposure to P. argentipes bites in a visceral leishmaniasis endemic area.
MeSH term(s)	Animals ; Bangladesh/epidemiology ; Bites and Stings/epidemiology ; Humans ; Insect Proteins/immunology ; Leishmania donovani ; Phlebotomus ; Saliva ; Salivary Proteins and Peptides/immunology
Chemical Substances	Insect Proteins ; Salivary Proteins and Peptides
Language	English
Publishing date	2021-07-15
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	120518-3
ISSN	1879-0135 ; 0020-7519
ISSN (online)	1879-0135
ISSN	0020-7519
DOI	10.1016/j.ijpara.2021.05.006
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