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Article: Propiedades cardiometabólicas del manidipino: ¿más allá de la reducción de la presión arterial?

Buset Ríos, N / Rodríguez Esparragón, F / Rodríguez Pérez, Jc

Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia

2009 Volume 29, Issue 3, Page(s) 203–207

Abstract: From its introduction in the decade of the 70's the evolution of the calcium channel blockers has allowed to resolve the uncertainty initially generated by those first generation drugs. These, are characterized by a smaller oral availability, a fast ... ...

Title translation	Cardio-metabolic properties of manidipine: beyond lowering arterial pressure?.
Abstract	From its introduction in the decade of the 70's the evolution of the calcium channel blockers has allowed to resolve the uncertainty initially generated by those first generation drugs. These, are characterized by a smaller oral availability, a fast vasodilator action and a short duration of action. Manidipine arises as a dihydropyridine calcium antagonist of third generation with real additional advantages regarding to previous generations. They show high lipophilia, a more prolonged action and as well as a prolonged average life at the level of his receptor and, in addition, some theoretical advantages among others calcium antagonists, improvements on the renal function by reducing the intraglomerular pressure and microalbuminuria. Nevertheless, the clinical evaluation of these last properties still depends on the results derived from clinical trials. Besides to go deep in its role in their antihypertensive effect, we presented a brief review on new cardiometabolic aspects of these dihydropyridines calcium antagonists focusing in manidipine.
MeSH term(s)	Blood Pressure/drug effects ; Calcium Channel Blockers/metabolism ; Calcium Channel Blockers/pharmacology ; Dihydropyridines/metabolism ; Dihydropyridines/pharmacology ; Heart/drug effects ; Humans
Chemical Substances	Calcium Channel Blockers ; Dihydropyridines ; manidipine (6O4754US88)
Language	Spanish
Publishing date	2009
Publishing country	Spain
Document type	English Abstract ; Journal Article ; Review
ZDB-ID	632512-9
ISSN	1989-2284 ; 0211-6995
ISSN (online)	1989-2284
ISSN	0211-6995
DOI	10.3265/Nefrologia.2009.29.3.5078.en.full
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Article ; Online: Vascular and metabolic properties of manidipine.

Buset Ríos, N / Rodríguez Esparragón, F / Fernández-Andrade Rodríguez, C / Rodríguez Pérez, J C

Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia

2011 Volume 31, Issue 3, Page(s) 268–274

Abstract: The combination of renin-angiotensin system blockers with calcium channel blockers appears to be one of the most effective options for treating hypertension and diabetes.Nevertheless, not all calcium blockers behave in the same manner. Manidipine, unlike ...

Abstract	The combination of renin-angiotensin system blockers with calcium channel blockers appears to be one of the most effective options for treating hypertension and diabetes.Nevertheless, not all calcium blockers behave in the same manner. Manidipine, unlike other third-generation dihydropyridine derived drugs, blocks T-type calcium channels present in the efferent glomerular arterioles, reducing intraglomerular pressure and microalbuminuria. In addition,T-type channels are related to proliferation, inflammation,fibrosis, vasoconstriction and activation of the renin-angiotensin system. The inhibition of these factors could explain the non-haemodynamic effects of manidipine as compared to other blockers.
MeSH term(s)	Albuminuria/drug therapy ; Calcium Channel Blockers/metabolism ; Calcium Channel Blockers/pharmacology ; Cardiovascular Diseases ; Dihydropyridines/pharmacology ; Humans ; Insulin Resistance ; Oxidative Stress/drug effects
Chemical Substances	Calcium Channel Blockers ; Dihydropyridines ; manidipine (6O4754US88)
Language	Spanish
Publishing date	2011
Publishing country	Spain
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	632512-9
ISSN	1989-2284 ; 0211-6995
ISSN (online)	1989-2284
ISSN	0211-6995
DOI	10.3265/Nefrologia.pre2010.Nov.10643
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Article ; Online: Actualización en la genética del ictus.

López Fernández, Juan Carlos / Rodríguez Esparragón, Francisco / Buset Ríos, Nisa

Medicina clinica

2014 Volume 143, Issue 4, Page(s) 176–179

Abstract: Stroke is a disease with significant morbidity, mortality, and economic and social impacts. It is a complex entity whose pathogenesis involves multiple environmental and genetic factors, with the latter having a role in up to 50% of strokes. The ... ...

Title translation	Update on the genetics of stroke.
Abstract	Stroke is a disease with significant morbidity, mortality, and economic and social impacts. It is a complex entity whose pathogenesis involves multiple environmental and genetic factors, with the latter having a role in up to 50% of strokes. The objective of the review is to analyze the available methods for the genetic diagnosis including linkage studies of variation in copy number, gene - candidate approximations, or whole genome (GWAS) and polymorphisms associated with its pathogenesis. We describe several single nucleotide polymorphisms (SNPs) associated with stroke in association studies and GWAS such as SNPs of angiotensin, the aldosterone system, paraoxonases, nitric oxide, coagulation, and fibrinolysis system, among others. We also analyze the role of certain polymorphisms in the phenotype of the carotid plaque, intracranial aneurysms and lobar hemorrhages. Pharmacogenomic aspects in which SNPs affect the response and safety regarding the use of different drugs are also described. Several SNPs that significantly contribute to the risk of stroke are also described. The advent of techniques like GWAS has contributed to the understanding of genetics and pharmacogenomics of stroke.
MeSH term(s)	Biotransformation/genetics ; Cardiovascular Agents/pharmacokinetics ; Cardiovascular Agents/therapeutic use ; Cardiovascular Diseases/diagnosis ; Cardiovascular Diseases/drug therapy ; Cardiovascular Diseases/genetics ; Gene Dosage ; Genetic Linkage ; Genetic Predisposition to Disease ; Genome-Wide Association Study ; Humans ; Molecular Diagnostic Techniques ; Pharmacogenetics ; Polymorphism, Single Nucleotide ; Stroke/genetics
Chemical Substances	Cardiovascular Agents
Language	Spanish
Publishing date	2014-08-19
Publishing country	Spain
Document type	English Abstract ; Journal Article ; Review
ZDB-ID	411607-0
ISSN	1578-8989 ; 0025-7753
ISSN (online)	1578-8989
ISSN	0025-7753
DOI	10.1016/j.medcli.2014.02.009
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Article: Peritoneal dialysis fluid biocompatibility impact on human peritoneal membrane permeability.

Rodríguez-Esparragón, Francisco / Marrero-Robayna, Silvia / González-Cabrera, Fayna / Hernández-Trujillo, Yaridé / Buset-Ríos, Nisa / Carlos Rodríguez-Pérez, José / Vega-Díaz, Nicanor

Clinical kidney journal

2018 Volume 11, Issue 6, Page(s) 881–888

Abstract: Background: We have compared the effects of conventional lactate-based peritoneal dialysis fluid (CPDF) with respect to bicarbonate/lactate-based fluid on peritoneal ultrafiltration (UF) and peritoneal permeability, and on variations on gene expression ... ...

Abstract	Background: We have compared the effects of conventional lactate-based peritoneal dialysis fluid (CPDF) with respect to bicarbonate/lactate-based fluid on peritoneal ultrafiltration (UF) and peritoneal permeability, and on variations on gene expression in cells isolated from effluents of patients' peritoneal bags. Methods: This was a non-randomized sequential prospective study including all incident peritoneal dialysis (PD) patients ( Results: In the overall study, the use of BPDF was associated with significantly lower mass transfer area coefficient for urea and creatinine, longer accelerated peritoneal examination test times for urea and creatinine, lower total pore area available for exchange over diffusion distance and lower UF. There were no differences in the gene expression of Conclusion: This study shows that those patients who started PD treatment with BPDF were characterized by a better biocompatibility profile. BPDF associates with lower peritoneal permeability to small molecules and lower UF.
Language	English
Publishing date	2018-06-25
Publishing country	England
Document type	Journal Article
ZDB-ID	2655800-2
ISSN	2048-8513 ; 2048-8505
ISSN (online)	2048-8513
ISSN	2048-8505
DOI	10.1093/ckj/sfy043
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Article: Diagnóstico genético de poliquistosis renal autosómica dominante mediante PCR múltiple.

Buset Ríos, N / Rodríguez Pérez, J C / Sánchez, J J / Hernández, C R / Hernández, E / Torres Galván, M J

Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia

2009 Volume 29, Issue 4, Page(s) 327–330

Abstract: Background: Autosomal Dominant Polycystic Kidney Disease (ADPKD) is the most common life-threatening hereditary disease. Molecular analysis with highly polymorphic short tandem repeats, located in the vicinity of the two genes responsible for the ... ...

Title translation	Genetic diagnosis of autosomal dominant polycystic kidney disease using multiplex-PCR.
Abstract	Background: Autosomal Dominant Polycystic Kidney Disease (ADPKD) is the most common life-threatening hereditary disease. Molecular analysis with highly polymorphic short tandem repeats, located in the vicinity of the two genes responsible for the disease (PKD1 and PKD2), is used to confirm diagnosis and give genetic counseling to members of affected families. Methods: We have developed a new assay to genotype five PKD1 and four PKD2 markers, based on two multiplex PCR reactions, and capillary electrophoresis analysis. A total of 110 subjects, belonging to 14 affected families, were genotyped to confirm the concordance with the singleplex method used previously. Results: The amplicons ranged from 95 to 154 bp in length, and complete STR profiles were obtained from 1-5 ng DNA. The specificity of the multiplex PCR system was 88,5% (95%CI= 75,9-95,2), and the sensitivity, 87,9 (95%CI= 76,1-94,6). Conclusions: This is a useful strategy that, together with automated computer-based allele detection, allows reliable, simple, faster, and cheaper genetic analysis than the previous singleplex method.
MeSH term(s)	Female ; Humans ; Male ; Microsatellite Repeats ; Polycystic Kidney, Autosomal Dominant/diagnosis ; Polycystic Kidney, Autosomal Dominant/genetics ; Polymerase Chain Reaction/methods ; TRPP Cation Channels/analysis ; TRPP Cation Channels/genetics
Chemical Substances	TRPP Cation Channels ; polycystic kidney disease 1 protein ; polycystic kidney disease 2 protein
Language	Spanish
Publishing date	2009
Publishing country	Spain
Document type	English Abstract ; Journal Article
ZDB-ID	632512-9
ISSN	1989-2284 ; 0211-6995
ISSN (online)	1989-2284
ISSN	0211-6995
DOI	10.3265/Nefrologia.2009.29.4.5360.en.full
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Article ; Online: Paraoxonase 1 and 2 gene variants and the ischemic stroke risk in Gran Canaria population: an association study and meta-analysis.

Rodríguez-Esparragón, Francisco / López-Fernández, Juan Carlos / Buset-Ríos, Nisa / García-Bello, Miguel A / Hernández-Velazquez, Erika / Cappiello, Laura / Rodríguez-Pérez, José Carlos

The International journal of neuroscience

2017 Volume 127, Issue 3, Page(s) 191–198

Abstract: Purpose of the study: The present study aims to evaluate the relationship between rs662 (Gln(Q)192Arg(R)) and rs854560 (L55M) and the rs7493 (S311C) in the paraoxonase genes and ischemic stroke (IS) in the population of Gran Canaria (Canary Islands). ... ...

Abstract	Purpose of the study: The present study aims to evaluate the relationship between rs662 (Gln(Q)192Arg(R)) and rs854560 (L55M) and the rs7493 (S311C) in the paraoxonase genes and ischemic stroke (IS) in the population of Gran Canaria (Canary Islands). The association with stroke was also evaluated using systematic review and meta-analysis. Methods: A total of 129 IS patients and 176 age and gender matched controls were enrolled. For meta-analysis, eligible studies were identified through search in public databases. Results: In multivariate regression analysis only the PON2 S311C variant showed to be an independent predictor of IS (OR = 0.093, 95% CI: 0.014-0.627). Overall, no significant association was found between L55M and IS when all studies were pooled nor by subgroup analysis by ethnicity. Gln192Arg showed a modest risk for IS in the global and in Asian population but with high heterogeneity among studies. A modest risk under a dominant inheritance model was found for the S311C variant with an overall random effect OR of 1.004 (95% CI: 1.00-1.35). There was strong evidence of heterogeneity among studies ( p = 0.0097, I Conclusions: The overall analysis shows a significant contribution of the rs662 variant to IS risk. We found that the CC genotype of the PON2 S311C polymorphism is a risk factor for IS. Results of the meta-analysis partially support this conclusion.
MeSH term(s)	Acetylcholinesterase/blood ; Adult ; Aged ; Aryldialkylphosphatase/genetics ; Brain Ischemia/complications ; Community Health Planning ; Female ; Genetic Association Studies ; Genetic Predisposition to Disease/genetics ; Genotype ; Humans ; Male ; Middle Aged ; Polymorphism, Single Nucleotide/genetics ; Prospective Studies ; Spain/epidemiology ; Statistics, Nonparametric ; Stroke/blood ; Stroke/enzymology ; Stroke/etiology ; Stroke/genetics
Chemical Substances	Acetylcholinesterase (EC 3.1.1.7) ; Aryldialkylphosphatase (EC 3.1.8.1) ; PON1 protein, human (EC 3.1.8.1) ; PON2 protein, human (EC 3.1.8.1)
Language	English
Publishing date	2017-03
Publishing country	England
Document type	Journal Article ; Meta-Analysis
ZDB-ID	3061-2
ISSN	1563-5279 ; 1543-5245 ; 0020-7454
ISSN (online)	1563-5279 ; 1543-5245
ISSN	0020-7454
DOI	10.3109/00207454.2016.1165675
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Article ; Online: Homocysteinylated protein levels in internal mammary artery (IMA) fragments and its genotype-dependence. S-homocysteine-induced methylation modifications in IMA and aortic fragments.

Rodríguez-Esparragón, Francisco / Serna-Gómez, Jaime Alberto / Hernández-Velázquez, Erika / Buset-Ríos, Nisa / Hernández-Trujillo, Yaridé / García-Bello, Miguel A / Rodríguez-Pérez, José C

Molecular and cellular biochemistry

2012 Volume 369, Issue 1-2, Page(s) 235–246

Abstract: ... of proteins as a result of N- or S-homocysteinylation. We evaluated S/N-homocysteinylated protein levels ...

Abstract	The resistance of internal mammary artery (IMA) toward atherosclerosis is not well understood. In plasma, homocysteine (Hcy) occurs in reduced, oxidized, homocysteine thiolactone and a component of proteins as a result of N- or S-homocysteinylation. We evaluated S/N-homocysteinylated protein levels in IMA fragments of patients undergoing coronary artery bypass grafting, and whether they were affected by genetic common variants. We tested whether tHcy, Hcy-S-protein levels, genotypes or Hcy-induced methylation modifications were related to differences in iNOS, Ddah2, and eNOS gene expression between territories. A small percentage of Hcy-S-proteins were found in IMA fragments. The Mthfr C677T (rs1801133) and Pon-1 Leu55Met (rs854560) variants were associated with Hcy-S-proteins. We observed a gradual difference according to Hcy-S-protein levels in the methylation degree of the Ddah2 gene promoter in aortic, but not in IMA, fragments. No correlation between the degree of methylation and the Ddah2 gene expression levels was found in both types of analyzed fragments. Total Hcy but not Hcy-S-proteins correlated with iNOS promoter methylation. Analyzed variants seem to contribute to the in vivo Hcy binding properties to IMA. The contribution of the Hcy-derived methylation modifications to Ddah2 and eNOS gene expression seems to be tissue-specific and independent of the Ddah2/ADMA/eNOS pathway. Hcy-derived methylation modifications to the iNOS gene promoter contribute to a lesser extent to iNOS gene expression.
MeSH term(s)	Aged ; Amidohydrolases/genetics ; Amidohydrolases/metabolism ; Aorta/enzymology ; Atherosclerosis/enzymology ; Atherosclerosis/genetics ; Coronary Artery Bypass ; Female ; Gene Expression ; Genotype ; Homocysteine/genetics ; Homocysteine/metabolism ; Humans ; Male ; Mammary Arteries/enzymology ; Methylation ; Middle Aged ; Nitric Oxide Synthase Type II/genetics ; Nitric Oxide Synthase Type II/metabolism ; Nitric Oxide Synthase Type III/genetics ; Nitric Oxide Synthase Type III/metabolism ; Polymorphism, Genetic ; Promoter Regions, Genetic
Chemical Substances	Homocysteine (0LVT1QZ0BA) ; NOS2 protein, human (EC 1.14.13.39) ; NOS3 protein, human (EC 1.14.13.39) ; Nitric Oxide Synthase Type II (EC 1.14.13.39) ; Nitric Oxide Synthase Type III (EC 1.14.13.39) ; Amidohydrolases (EC 3.5.-) ; dimethylargininase (EC 3.5.3.18)
Language	English
Publishing date	2012-07-14
Publishing country	Netherlands
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	184833-1
ISSN	1573-4919 ; 0300-8177
ISSN (online)	1573-4919
ISSN	0300-8177
DOI	10.1007/s11010-012-1387-7
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Article ; Online: Psychonephrology: psychological aspects in autosomal dominant polycystic kidney disease.

Pérez Domínguez, T S / Rodríguez Pérez, A / Buset Ríos, N / Rodríguez Esparragón, F / García Bello, M A / Pérez Borges, P / Parodis López, Y / Rodríguez Pérez, J C

Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia

2011 Volume 31, Issue 6, Page(s) 716–722

Abstract: The biological, physical and psychological burden of a chronic disease has an impact on the quality of life of people who suffer from it. The perception of quality of life is affected by psychological disorders such as anxiety and depression that have a ... ...

Abstract	The biological, physical and psychological burden of a chronic disease has an impact on the quality of life of people who suffer from it. The perception of quality of life is affected by psychological disorders such as anxiety and depression that have a high prevalence in people with chronic kidney disease (CKD). These factors are also linked to lower life expectancy. It is therefore surprising that the psychological aspects of people with autosomal dominant polycystic kidney disease (ADPKD) have received so little attention in the medical literature, despite their importance for the overall health of these patients. The relatively new discipline called psychonephrology provides a broader view of the impact that these aspects have on individuals with chronic kidney disease, with a consequent practical application. In this article, we examine the consequences and prevalence of psychological problems that can be related to CKD and ADPKD. Firstly, we will focus on the field of CKD and ADPKD within the scope of psychonephrology. Secondly, the article introduces the concept of quality of life as a basic pillar of health that is affected when a person is diagnosed with CKD. Thirdly, we will present a summary of the main research related to anxiety and depression disorders in CKD and ADPKD. The article will conclude by synthesising findings from the different lines of research undertaken.
MeSH term(s)	Anxiety/epidemiology ; Anxiety/etiology ; Depression/epidemiology ; Depression/etiology ; Family Relations ; Fear ; Humans ; Inpatients/psychology ; Kidney Failure, Chronic/etiology ; Kidney Failure, Chronic/psychology ; Kidney Transplantation/psychology ; Life Expectancy ; Life Style ; Outpatients/psychology ; Polycystic Kidney, Autosomal Dominant/complications ; Polycystic Kidney, Autosomal Dominant/psychology ; Polycystic Kidney, Autosomal Dominant/surgery ; Quality of Life ; Renal Dialysis/psychology ; Social Support ; Spain/epidemiology ; Stress, Psychological/epidemiology ; Stress, Psychological/etiology ; Waiting Lists
Language	Spanish
Publishing date	2011
Publishing country	Spain
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	632512-9
ISSN	1989-2284 ; 0211-6995
ISSN (online)	1989-2284
ISSN	0211-6995
DOI	10.3265/Nefrologia.pre2011.Jul.10847
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Article ; Online: Progression of chronic kidney disease. Prevalence of anxiety and depression in autosomal dominant polycystic kidney disease.

Pérez-Dominguez, Tais / Rodríguez-Pérez, Armando / García-Bello, Miguel A / Buset-Ríos, Nisa / Rodríguez-Esparragón, Francisco / Parodis-López, Yanet / Rodríguez-Pérez, José C

Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia

2012 Volume 32, Issue 3, Page(s) 397–399

MeSH term(s)	Adaptation, Psychological ; Anxiety/epidemiology ; Anxiety/etiology ; Anxiety Disorders/diagnosis ; Anxiety Disorders/epidemiology ; Anxiety Disorders/etiology ; Depression/epidemiology ; Depression/etiology ; Depressive Disorder/diagnosis ; Depressive Disorder/epidemiology ; Depressive Disorder/etiology ; Humans ; Polycystic Kidney, Autosomal Dominant/psychology ; Polycystic Kidney, Autosomal Dominant/therapy ; Prevalence ; Quality of Life ; Renal Dialysis/psychology ; Severity of Illness Index ; Surveys and Questionnaires ; Time Factors
Language	Spanish
Publishing date	2012-05-14
Publishing country	Spain
Document type	Letter ; Research Support, Non-U.S. Gov't
ZDB-ID	632512-9
ISSN	1989-2284 ; 0211-6995
ISSN (online)	1989-2284
ISSN	0211-6995
DOI	10.3265/Nefrologia.pre2012.Feb.11379
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