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  1. Article: Atrial flutter with multilevel atrioventricular block and cyclical occurrence of varying QRS morphologies.

    Abedin, Zainul / Conner, Robert P / Ali, Sania / Dahal, Ranjan / Abedin, Moeen

    Journal of arrhythmia

    2022  Volume 39, Issue 1, Page(s) 78–81

    Abstract: An electrocardiogram showing atrial flutter in which varying ratios of AV conduction and the cyclical recurrence of varying QRS morphologies are observed is presented. ...

    Abstract An electrocardiogram showing atrial flutter in which varying ratios of AV conduction and the cyclical recurrence of varying QRS morphologies are observed is presented.
    Language English
    Publishing date 2022-12-04
    Publishing country Japan
    Document type Journal Article
    ZDB-ID 2696593-8
    ISSN 1883-2148 ; 1880-4276
    ISSN (online) 1883-2148
    ISSN 1880-4276
    DOI 10.1002/joa3.12798
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  2. Article ; Online: Failure to Treat Life-Threatening Ventricular Tachyarrhythmias in Contemporary Implantable Cardioverter-Defibrillators: Implications for Strategic Programming.

    Thøgersen, Anna Margrethe / Larsen, Jacob Moesgaard / Johansen, Jens Brock / Abedin, Moeen / Swerdlow, Charles D

    Circulation. Arrhythmia and electrophysiology

    2017  Volume 10, Issue 9

    Abstract: Background: In clinical trials, manufacturer-specific, strategic programming of implantable cardioverter-defibrillators (ICDs), including faster detection rates, reduces unnecessary therapy but permits therapy for ventricular tachycardia/ventricular ... ...

    Abstract Background: In clinical trials, manufacturer-specific, strategic programming of implantable cardioverter-defibrillators (ICDs), including faster detection rates, reduces unnecessary therapy but permits therapy for ventricular tachycardia/ventricular fibrillation (VF). Present consensus recommends a generic rate threshold between 185 and 200 beats per minute, which exceeds the rate tested in clinical trials for some manufacturers. In a case series, we sought to determine the relationship between programmed parameters and failure of modern ICDs to treat VF.
    Methods and results: We reviewed cases in which normally functioning ICDs failed to deliver timely therapy for VF from April 2015 to January 2017 at 4 institutions. Of 10 ambulatory patients, 5 died from untreated VF, 4 had cardiac arrests requiring external shocks, and 1 was rescued by a delayed ICD shock. VF did not satisfy programmed detection criteria in 9 patients (90%). Seven of these patients had slowest detection rates that were consistent with generic recommendations but not tested in a peer-reviewed trial for their manufacturer's ICDs. Manufacturer-specific factors interacted with fast detection rates to withhold therapy, including strict VF episode termination rules, enhancements to minimize T-wave oversensing, and features that restrict therapy to regular rhythms in ventricular tachycardia zones. Untreated VF despite recommended programming accounted for 56% of sudden deaths and 11% of all deaths during the study period.
    Conclusions: Complex and unanticipated interactions between manufacturer-specific features and generic programming can prevent therapy for VF. More data are needed to assess the risks and benefits of translating evidence-based detection parameters from one manufacturer to another.
    MeSH term(s) Adolescent ; Adult ; Aged ; Aged, 80 and over ; Defibrillators, Implantable/adverse effects ; Electric Countershock/statistics & numerical data ; Humans ; Male ; Middle Aged ; Prosthesis Failure ; Risk Factors ; Ventricular Fibrillation/mortality ; Ventricular Fibrillation/physiopathology ; Ventricular Fibrillation/prevention & control
    Language English
    Publishing date 2017-05-11
    Publishing country United States
    Document type Journal Article
    ZDB-ID 2426129-4
    ISSN 1941-3084 ; 1941-3149
    ISSN (online) 1941-3084
    ISSN 1941-3149
    DOI 10.1161/CIRCEP.117.005305
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 6667: Show issues Location:
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  3. Article ; Online: Advances in the management of atrial fibrillation with a special focus on non-pharmacological approaches to prevent thromboembolism: a review of current recommendations.

    Riddle, Malini / McCallum, Richard / Ojha, Chandra Prakash / Paul, Timir Kumar / Gupta, Vineet / Baran, David Alan / Prakash, Bharat Ved / Misra, Amogh / Mares, Adriana Camila / Abedin, Moeen / Kedar, Archana / Mulukutla, Venkatachalam / Ibrahim, Ahmed / Nagarajarao, Harsha

    Journal of investigative medicine : the official publication of the American Federation for Clinical Research

    2020  Volume 68, Issue 8, Page(s) 1317–1333

    Abstract: Atrial fibrillation (AFIB) is the most common heart rhythm abnormality and is associated with significant morbidity and mortality. While the treatment of AFIB involves strategies of rate with or without rhythm control, it is also essential to strategize ... ...

    Abstract Atrial fibrillation (AFIB) is the most common heart rhythm abnormality and is associated with significant morbidity and mortality. While the treatment of AFIB involves strategies of rate with or without rhythm control, it is also essential to strategize appropriate therapies to prevent thromboembolic complications arising from AFIB. Previously, anticoagulation was the main treatment option which exposed patients to higher than usual risk of bleeding. However, with the advent of new technology, novel therapeutic options aimed at surgical or percutaneous exclusion or occlusion of the left atrial appendage in preventing thromboembolic complications from AFIB have evolved. This review evaluates recent advances and therapeutic options in treating AFIB with a special focus on both surgical and percutaneous interventions which can reduce and/or eliminate thromboembolic complications of AFIB.
    MeSH term(s) Atrial Fibrillation/economics ; Atrial Fibrillation/therapy ; Catheter Ablation ; Cost-Benefit Analysis ; Health Planning Guidelines ; Humans ; Randomized Controlled Trials as Topic ; Thromboembolism/economics ; Thromboembolism/etiology ; Thromboembolism/prevention & control
    Language English
    Publishing date 2020-11-17
    Publishing country England
    Document type Journal Article ; Review
    ZDB-ID 1217870-6
    ISSN 1708-8267 ; 0009-9279 ; 1081-5589
    ISSN (online) 1708-8267
    ISSN 0009-9279 ; 1081-5589
    DOI 10.1136/jim-2020-001500
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    In stock of ZB MED Cologne/Königswinter

    Zs.A 307: Show issues Location:
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  4. Article: Skeleton key to vascular disease.

    Demer, Linda L / Abedin, Moeen

    Journal of the American College of Cardiology

    2004  Volume 44, Issue 10, Page(s) 1977–1979

    MeSH term(s) Coronary Artery Disease/blood ; Glycoproteins/blood ; Humans ; Osteoprotegerin ; Receptors, Cytoplasmic and Nuclear/blood ; Receptors, Tumor Necrosis Factor/blood
    Chemical Substances Glycoproteins ; Osteoprotegerin ; Receptors, Cytoplasmic and Nuclear ; Receptors, Tumor Necrosis Factor ; TNFRSF11B protein, human
    Language English
    Publishing date 2004-11-16
    Publishing country United States
    Document type Comment ; Editorial
    ZDB-ID 605507-2
    ISSN 1558-3597 ; 0735-1097
    ISSN (online) 1558-3597
    ISSN 0735-1097
    DOI 10.1016/j.jacc.2004.08.032
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  5. Article ; Online: Inappropriate ICD shocks for inappropriate reasons.

    Pai, Rakesh K / Abedin, Moeen / Rawling, David A

    Indian pacing and electrophysiology journal

    2008  Volume 8, Issue 1, Page(s) 69–71

    Abstract: ICD shocks can result from a variety of etiologies; determining the proper etiology of the inappropriate shock is essential for correction of the problem. Electromagnetic interference (EMI) can mimic cardiac signals and cause inappropriate defibrillator ... ...

    Abstract ICD shocks can result from a variety of etiologies; determining the proper etiology of the inappropriate shock is essential for correction of the problem. Electromagnetic interference (EMI) can mimic cardiac signals and cause inappropriate defibrillator shocks. We present two cases of inappropriate ICD shocks due to EMI and reversal of the proximal and distal DF-1 lead terminals of the ICD lead. These are two unusual etiologies for inappropriate defibrillator shocks.
    Language English
    Publishing date 2008-02-01
    Publishing country Netherlands
    Document type Journal Article
    ZDB-ID 2123606-9
    ISSN 0972-6292 ; 0972-6292
    ISSN (online) 0972-6292
    ISSN 0972-6292
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  6. Article ; Online: Inappropriate ICD Shocks for Inappropriate Reasons

    Rakesh K Pai / Moeen Abedin / David A Rawling

    Indian Pacing and Electrophysiology Journal, Vol 8, Iss 1, Pp 69-

    2008  Volume 71

    Abstract: ICD shocks can result from a variety of etiologies; determining the proper etiology of the inappropriate shock is essential for correction of the problem. Electromagnetic interference (EMI) can mimic cardiac signals and cause inappropriate defibrillator ... ...

    Abstract ICD shocks can result from a variety of etiologies; determining the proper etiology of the inappropriate shock is essential for correction of the problem. Electromagnetic interference (EMI) can mimic cardiac signals and cause inappropriate defibrillator shocks. We present two cases of inappropriate ICD shocks due to EMI and reversal of the proximal and distal DF-1 lead terminals of the ICD lead. These are two unusual etiologies for inappropriate defibrillator shocks.
    Keywords Inappropriate ICD Shock ; DF-1 Lead Terminal Reversal ; Integrated Bipolar ICD lead ; Complication ; External Alternating Current ; Diseases of the circulatory (Cardiovascular) system ; RC666-701 ; Specialties of internal medicine ; RC581-951 ; Internal medicine ; RC31-1245 ; Medicine ; R ; DOAJ:Cardiovascular ; DOAJ:Medicine (General) ; DOAJ:Health Sciences ; Physiology ; QP1-981 ; Science ; Q ; DOAJ:Physiology ; DOAJ:Biology ; DOAJ:Biology and Life Sciences
    Language English
    Publishing date 2008-02-01T00:00:00Z
    Publisher Elsevier
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  7. Article ; Online: Don't miss this opportunity: Diagnosing diabetes.

    Abedin, Moeen / McGuire, Darren K

    American heart journal

    2003  Volume 145, Issue 2, Page(s) 195–197

    MeSH term(s) Cardiology ; Diabetes Mellitus/diagnosis ; Humans ; Practice Guidelines as Topic ; Primary Health Care
    Language English
    Publishing date 2003-02
    Publishing country United States
    Document type Comment ; Editorial
    ZDB-ID 80026-0
    ISSN 1097-6744 ; 0002-8703
    ISSN (online) 1097-6744
    ISSN 0002-8703
    DOI 10.1067/mhj.2003.135
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    Ui IV Zs.15: Show issues Location:
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  8. Article ; Online: Mesenchymal stem cells and the artery wall.

    Abedin, Moeen / Tintut, Yin / Demer, Linda L

    Circulation research

    2004  Volume 95, Issue 7, Page(s) 671–676

    Abstract: The presence of ectopic tissue in the diseased artery wall is evidence for the presence of multipotential stem cells in the vasculature. Mesenchymal stem cells were first identified in the marrow stroma, and they differentiate along multiple lineages ... ...

    Abstract The presence of ectopic tissue in the diseased artery wall is evidence for the presence of multipotential stem cells in the vasculature. Mesenchymal stem cells were first identified in the marrow stroma, and they differentiate along multiple lineages giving rise to cartilage, bone, fat, muscle, and vascular tissue in vitro and in vivo. Transplantation studies show that marrow-derived mesenchymal stem cells appear to enter the circulation and engraft other tissues, including the artery wall, at sites of injury. Recent evidence indicates that mesenchymal stem cells are also present in normal artery wall and microvessels and that they also may enter the circulation, contributing to the population of circulating progenitor cells and engrafting other tissues. Thus, the artery wall is not only a destination but also a source of progenitor cells that have regenerative potential. Although potential artifacts, such as fusion, need to be taken into consideration, these new developments in vascular biology open important therapeutic avenues. A greater understanding of how mesenchymal stem cells from the bone marrow or artery wall bring about vascular regeneration and repair may lead to novel cell-based treatments for cardiovascular disease.
    MeSH term(s) Animals ; Arteries/cytology ; Arteries/pathology ; Arteries/physiology ; Arteriosclerosis/pathology ; Arteriosclerosis/therapy ; Bone Marrow Cells/physiology ; Cell Differentiation ; Cell Lineage ; Cells, Cultured/cytology ; Chondrogenesis/physiology ; Choristoma/pathology ; Endothelium, Vascular/cytology ; Graft Survival ; Heart Transplantation ; Humans ; Inflammation ; Mesenchymal Stem Cell Transplantation ; Mesenchymal Stromal Cells/physiology ; Metaplasia ; Mice ; Osteogenesis/physiology ; Regeneration ; Sheep ; Transplantation, Heterologous ; Tunica Media/cytology ; Tunica Media/pathology
    Language English
    Publishing date 2004-10-01
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, P.H.S. ; Review
    ZDB-ID 80100-8
    ISSN 1524-4571 ; 0009-7330 ; 0931-6876
    ISSN (online) 1524-4571
    ISSN 0009-7330 ; 0931-6876
    DOI 10.1161/01.RES.0000143421.27684.12
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    Ui IV Zs.70: Show issues Location:
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  9. Article ; Online: Vascular calcification: mechanisms and clinical ramifications.

    Abedin, Moeen / Tintut, Yin / Demer, Linda L

    Arteriosclerosis, thrombosis, and vascular biology

    2004  Volume 24, Issue 7, Page(s) 1161–1170

    Abstract: Vascular calcification, long thought to result from passive degeneration, involves a complex, regulated process of biomineralization resembling osteogenesis. Evidence indicates that proteins controlling bone mineralization are also involved in the ... ...

    Abstract Vascular calcification, long thought to result from passive degeneration, involves a complex, regulated process of biomineralization resembling osteogenesis. Evidence indicates that proteins controlling bone mineralization are also involved in the regulation of vascular calcification. Artery wall cells grown in culture are induced to become osteogenic by inflammatory and atherogenic stimuli. Furthermore, osteoclast-like cells are found in calcified atherosclerotic plaques, and active resorption of ectopic vascular calcification has been demonstrated. In general, soft tissue calcification arises in areas of chronic inflammation, possibly functioning as a barrier limiting the spread of the inflammatory stimulus. Atherosclerotic calcification may be one example of this process, in which oxidized lipids are the inflammatory stimulus. Calcification is widely used as a clinical indicator of atherosclerosis. It progresses nonlinearly with time, following a sigmoid-shaped curve. The relationship between calcification and clinical events likely relates to mechanical instability introduced by calcified plaque at its interface with softer, noncalcified plaque. In general, as calcification proceeds, interface surface area increases initially, but eventually decreases as plaques coalesce. This phenomenon may account for reports of less calcification in unstable plaque. Vascular calcification is exacerbated in certain clinical entities, including diabetes, menopause, and osteoporosis. Mechanisms linking them must be considered in clinical decisions. For example, treatments for osteoporosis may have unanticipated effects on vascular calcification; the converse also applies. Further understanding of processes governing vascular calcification may yield new therapeutic options for vascular disease.
    MeSH term(s) Animals ; Arteriosclerosis/metabolism ; Arteriosclerosis/pathology ; Calcinosis/metabolism ; Calcinosis/pathology ; Calcium-Binding Proteins/metabolism ; Diabetes Mellitus/metabolism ; Endpoint Determination ; Extracellular Matrix Proteins/metabolism ; Glycoproteins/metabolism ; Humans ; Menopause ; Mice ; Mice, Knockout ; Minerals/metabolism ; Osteogenesis ; Osteopontin ; Osteoporosis/metabolism ; Osteoprotegerin ; Phosphates/metabolism ; Receptors, Cytoplasmic and Nuclear/metabolism ; Receptors, Tumor Necrosis Factor ; Sialoglycoproteins/metabolism ; Matrix Gla Protein
    Chemical Substances Calcium-Binding Proteins ; Extracellular Matrix Proteins ; Glycoproteins ; Minerals ; Osteoprotegerin ; Phosphates ; Receptors, Cytoplasmic and Nuclear ; Receptors, Tumor Necrosis Factor ; SPP1 protein, human ; Sialoglycoproteins ; Spp1 protein, mouse ; TNFRSF11B protein, human ; Tnfrsf11b protein, mouse ; Osteopontin (106441-73-0)
    Language English
    Publishing date 2004-05-20
    Publishing country United States
    Document type Comparative Study ; Journal Article ; Review
    ZDB-ID 1221433-4
    ISSN 1524-4636 ; 1079-5642
    ISSN (online) 1524-4636
    ISSN 1079-5642
    DOI 10.1161/01.ATV.0000133194.94939.42
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    Zs.A 1705: Show issues Location:
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  10. Article: Biventricular pacing reduces ventricular arrhythmic burden and defibrillator therapies in patients with heart failure.

    Daccarett, Marcos / Pai, Rakesh K / Abedin, Moeen / Segerson, Nathan M / Hamdan, Mohamed H

    Clinical cardiology

    2006  Volume 29, Issue 10, Page(s) 474–5; author reply 475

    MeSH term(s) Arrhythmias, Cardiac/physiopathology ; Arrhythmias, Cardiac/therapy ; Cardiac Pacing, Artificial/methods ; Defibrillators, Implantable ; Heart Failure/therapy ; Humans
    Language English
    Publishing date 2006-10-19
    Publishing country United States
    Document type Comment ; Letter
    ZDB-ID 391935-3
    ISSN 1932-8737 ; 0160-9289
    ISSN (online) 1932-8737
    ISSN 0160-9289
    DOI 10.1002/clc.4960291015
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