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  1. Book: Mulliken & Young's vascular anomalies

    Mulliken, John B. / Young, Anthony E. / Burrows, Patricia E. / Fishman, Steven J.

    hemangiomas and malformations

    2013  

    Title variant Vascular anomalies
    Author's details ed. by John B. Mulliken ; Patricia E. Burrows ; Steven J. Fishman
    Keywords Vascular Malformations / etiology ; Vascular Malformations / therapy ; Hemangioma / etiology ; Hemangioma / therapy ; Vascular Neoplasms
    Language English
    Size XVIII, 1118 S. : zahlr. Ill.
    Edition 2. ed.
    Publisher Oxford Univ. Press
    Publishing place Oxford
    Publishing country Great Britain
    Document type Book
    Note Includes bibliographical references and index
    HBZ-ID HT017839679
    ISBN 978-0-19-514505-2 ; 978-0-19-972254-9 ; 978-0-19-998209-7 ; 0-19-514505-4 ; 0-19-972254-4 ; 0-19-998209-0
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2013 A 3128 available for loan (reading room) Lesesaal EG

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  2. Article: Angioarchitecture of Hereditary Arteriovenous Malformations.

    Burrows, Patricia E

    Seminars in interventional radiology

    2017  Volume 34, Issue 3, Page(s) 250–257

    Abstract: This article describes three hereditary conditions known to be associated with arteriovenous malformation (AVM), along with their clinical and imaging features and angiographic angioarchitecture. Hereditary hemorrhagic telangiectasia, capillary ... ...

    Abstract This article describes three hereditary conditions known to be associated with arteriovenous malformation (AVM), along with their clinical and imaging features and angiographic angioarchitecture. Hereditary hemorrhagic telangiectasia, capillary malformation-AVM (CM-AVM), and PTEN tumor hamartoma syndrome are conditions with autosomal dominant inheritance, caused by mutations in different molecular pathways, which frequently present with symptomatic AVMs. Imaging biomarkers, including sites of predilection, angioarchitecture, and tissue overgrowth patterns, are helpful in identifying these patients and selecting appropriate treatment.
    Language English
    Publishing date 2017-09-11
    Publishing country United States
    Document type Journal Article ; Review
    ZDB-ID 848341-3
    ISSN 1098-8963 ; 0739-9529
    ISSN (online) 1098-8963
    ISSN 0739-9529
    DOI 10.1055/s-0037-1604298
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

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  3. Article: Endothelial cilia dysfunction in pathogenesis of hereditary hemorrhagic telangiectasia.

    Eisa-Beygi, Shahram / Burrows, Patricia E / Link, Brian A

    Frontiers in cell and developmental biology

    2022  Volume 10, Page(s) 1037453

    Abstract: Hereditary hemorrhagic telangiectasia (HHT) is associated with defective capillary network, leading to dilated superficial vessels and arteriovenous malformations (AVMs) in which arteries connect directly to the veins. Loss or haploinsufficiency of ... ...

    Abstract Hereditary hemorrhagic telangiectasia (HHT) is associated with defective capillary network, leading to dilated superficial vessels and arteriovenous malformations (AVMs) in which arteries connect directly to the veins. Loss or haploinsufficiency of components of TGF-β signaling, ALK1, ENG, SMAD4, and BMP9, have been implicated in the pathogenesis AVMs. Emerging evidence suggests that the inability of endothelial cells to detect, transduce and respond to blood flow, during early development, is an underpinning of AVM pathogenesis. Therefore, components of endothelial flow detection may be instrumental in potentiating TGF-β signaling in perfused blood vessels. Here, we argue that endothelial cilium, a microtubule-based and flow-sensitive organelle, serves as a signaling hub by coupling early flow detection with potentiation of the canonical TGF-β signaling in nascent endothelial cells. Emerging evidence from animal models suggest a role for primary cilia in mediating vascular development. We reason, on recent observations, that endothelial cilia are crucial for vascular development and that embryonic loss of endothelial cilia will curtail TGF-β signaling, leading to associated defects in arteriovenous development and impaired vascular stability. Loss or dysfunction of endothelial primary cilia may be implicated in the genesis of AVMs due, in part, to inhibition of ALK1/SMAD4 signaling. We speculate that AVMs constitute part of the increasing spectrum of ciliopathy-associated vascular defects.
    Language English
    Publishing date 2022-11-10
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 2737824-X
    ISSN 2296-634X
    ISSN 2296-634X
    DOI 10.3389/fcell.2022.1037453
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article ; Online: Endothelial cilia dysfunction in pathogenesis of hereditary hemorrhagic telangiectasia

    Shahram Eisa-Beygi / Patricia E. Burrows / Brian A. Link

    Frontiers in Cell and Developmental Biology, Vol

    2022  Volume 10

    Abstract: Hereditary hemorrhagic telangiectasia (HHT) is associated with defective capillary network, leading to dilated superficial vessels and arteriovenous malformations (AVMs) in which arteries connect directly to the veins. Loss or haploinsufficiency of ... ...

    Abstract Hereditary hemorrhagic telangiectasia (HHT) is associated with defective capillary network, leading to dilated superficial vessels and arteriovenous malformations (AVMs) in which arteries connect directly to the veins. Loss or haploinsufficiency of components of TGF-β signaling, ALK1, ENG, SMAD4, and BMP9, have been implicated in the pathogenesis AVMs. Emerging evidence suggests that the inability of endothelial cells to detect, transduce and respond to blood flow, during early development, is an underpinning of AVM pathogenesis. Therefore, components of endothelial flow detection may be instrumental in potentiating TGF-β signaling in perfused blood vessels. Here, we argue that endothelial cilium, a microtubule-based and flow-sensitive organelle, serves as a signaling hub by coupling early flow detection with potentiation of the canonical TGF-β signaling in nascent endothelial cells. Emerging evidence from animal models suggest a role for primary cilia in mediating vascular development. We reason, on recent observations, that endothelial cilia are crucial for vascular development and that embryonic loss of endothelial cilia will curtail TGF-β signaling, leading to associated defects in arteriovenous development and impaired vascular stability. Loss or dysfunction of endothelial primary cilia may be implicated in the genesis of AVMs due, in part, to inhibition of ALK1/SMAD4 signaling. We speculate that AVMs constitute part of the increasing spectrum of ciliopathy-associated vascular defects.
    Keywords Vascular disease ; Zebrafish ; TGF-β ; Endothelial cilia ; BMP signaling ; Biology (General) ; QH301-705.5
    Language English
    Publishing date 2022-11-01T00:00:00Z
    Publisher Frontiers Media S.A.
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

    Full text online

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  5. Article: Angioarchitecture of Hereditary Arteriovenous Malformations

    Burrows, Patricia E.

    Seminars in Interventional Radiology

    (Vascular Anomalies)

    2017  Volume 34, Issue 03, Page(s) 250–257

    Abstract: This article describes three hereditary conditions known to be associated with arteriovenous malformation (AVM), along with their clinical and imaging features and angiographic angioarchitecture. Hereditary hemorrhagic telangiectasia, capillary ... ...

    Series title Vascular Anomalies
    Abstract This article describes three hereditary conditions known to be associated with arteriovenous malformation (AVM), along with their clinical and imaging features and angiographic angioarchitecture. Hereditary hemorrhagic telangiectasia, capillary malformation–AVM (CM–AVM), and PTEN tumor hamartoma syndrome are conditions with autosomal dominant inheritance, caused by mutations in different molecular pathways, which frequently present with symptomatic AVMs. Imaging biomarkers, including sites of predilection, angioarchitecture, and tissue overgrowth patterns, are helpful in identifying these patients and selecting appropriate treatment.
    Keywords arteriovenous malformation ; capillary malformation–arteriovenous malformation ; hereditary hemorrhagic telangiectasia ; PTEN tumor hamartoma syndrome ; genetic mutations
    Language English
    Publishing date 2017-09-01
    Publisher Thieme Medical Publishers
    Publishing place Stuttgart ; New York
    Document type Article
    ZDB-ID 848341-3
    ISSN 1098-8963 ; 0739-9529
    ISSN (online) 1098-8963
    ISSN 0739-9529
    DOI 10.1055/s-0037-1604298
    Database Thieme publisher's database

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    In stock of ZB MED Cologne/Königswinter

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  6. Article ; Online: Endovascular treatment of slow-flow vascular malformations.

    Burrows, Patricia E

    Techniques in vascular and interventional radiology

    2013  Volume 16, Issue 1, Page(s) 12–21

    Abstract: Symptomatic slow-flow vascular malformations include venous malformations and lymphatic malformations, as well as combined anomalies. Endovascular therapy, consisting mainly of intralesional sclerosant injection, is now accepted as the primary treatment ... ...

    Abstract Symptomatic slow-flow vascular malformations include venous malformations and lymphatic malformations, as well as combined anomalies. Endovascular therapy, consisting mainly of intralesional sclerosant injection, is now accepted as the primary treatment for most of these lesions. Magnetic resonance imaging and ultrasonography supplement physical examination for diagnosis and assessment of the extent of malformation. Endovascular treatment is usually carried out under general anesthesia. Sclerosants for venous malformations include ethanol, 3% sodium tetradecyl sulfate, and bleomycin. Lymphatic malformations can be injected with doxycycline, bleomycin, OK-432, or other sclerosants. Complications of sclerotherapy include tissue necrosis, peripheral nerve injury, hemoglobinuria, deep vein thrombosis, and pulmonary embolism. Although most vascular malformations are not cured, the majority of patients benefit from endovascular treatment.
    MeSH term(s) Humans ; Injections, Intralesional ; Sclerosing Solutions/therapeutic use ; Sclerotherapy/methods ; Vascular Malformations/drug therapy
    Chemical Substances Sclerosing Solutions
    Language English
    Publishing date 2013-03
    Publishing country United States
    Document type Journal Article ; Review
    ZDB-ID 2170922-1
    ISSN 1557-9808 ; 1089-2516
    ISSN (online) 1557-9808
    ISSN 1089-2516
    DOI 10.1053/j.tvir.2013.01.003
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  7. Article: Associations between Adolescent Alcohol Use and Neurocognitive Functioning in Young Adulthood.

    East, Patricia L / Delva, Jorge / Blanco, Estela / Correa-Burrows, Paulina / Burrows, Raquel / Gahagan, Sheila

    Applied developmental science

    2022  Volume 28, Issue 1, Page(s) 46–57

    Abstract: This study examined the associations between excessive alcohol intake during adolescence and neurocognitive functioning in young adulthood and whether these relations varied by sex. Participants were working-class Chilean adolescents ( ...

    Abstract This study examined the associations between excessive alcohol intake during adolescence and neurocognitive functioning in young adulthood and whether these relations varied by sex. Participants were working-class Chilean adolescents (
    Language English
    Publishing date 2022-11-11
    Publishing country United States
    Document type Journal Article
    ZDB-ID 2031678-1
    ISSN 1532-480X ; 1088-8691
    ISSN (online) 1532-480X
    ISSN 1088-8691
    DOI 10.1080/10888691.2022.2138886
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  8. Article ; Online: Socioeconomic position and executive functioning from childhood to young adulthood: Evidence from Santiago, Chile.

    Delker, Erin / Gahagan, Sheila / Burrows, Raquel / Burrows-Correa, Paulina / East, Patricia / Lozoff, Betsy / Blanco, Estela

    Advances in life course research

    2023  Volume 56, Page(s) 100546

    Abstract: Background: Optimizing cognitive development through early adulthood has implications for population health. This study aims to understand how socioeconomic position (SEP) across development relates to executive functioning. We evaluate three frameworks ...

    Abstract Background: Optimizing cognitive development through early adulthood has implications for population health. This study aims to understand how socioeconomic position (SEP) across development relates to executive functioning. We evaluate three frameworks in life-course epidemiology - the sensitive period, accumulation, and social mobility hypotheses.
    Methods: Participants were young adults from Santiago, Chile who were studied from 6 months to 21 years. Family SEP was measured at ages 1 y, 10 y, and 16 y with the modified Graffar Index. Executive functioning was assessed at ages 16 y and 21 y by the Trail Making Test Part B (Trails B). Analyses estimating 16 y and 21 y executive function involved 581 and 469 participants, respectively. Trails B scores were modeled as a function of SEP at 1 y, 10 y, and 16 y, as the total accumulation of disadvantage, and as change in SEP between 1 y and 10 y and between 10 y and 16 y.
    Results: Participants were low- to middle-income in infancy and, on average, experienced upwards mobility across childhood. Half of participants (58%) improved Trails B scores from 16 y and 21 y. Most (68%) experienced upward social mobility between infancy and 16 y. When examined independently, worse SEP measured at 10 y and 16 y related to worse (longer time to complete) Trails B scores at Age 21 but did not relate to the other outcomes. After mutual adjustment as a test of the sensitivity hypothesis, no SEP measure was independently related to any outcome. Testing the accumulation hypothesis, cumulative low SEP was associated with worse cognitive performance at 21 y (β = 3.6, p = 0.04). Results for the social mobility hypothesis showed no relation to cognitive scores or to change in cognitive scores. Comparing all hypotheses, SEP at 16 y explained the most variability in executive functioning at 21 y, providing support for the sensitive period hypothesis.
    Conclusions: Results indicate that experiencing cumulatively low socioeconomic position from infancy to adolescence can have a negative impact on cognitive functioning in young adulthood. Findings also provide evidence in support of adolescence as a key developmental period during which SEP can most strongly impact cognitive functioning.
    MeSH term(s) Child ; Adolescent ; Young Adult ; Humans ; Adult ; Executive Function ; Chile ; Cognition ; Income ; Social Mobility
    Language English
    Publishing date 2023-04-17
    Publishing country Netherlands
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Research Support, N.I.H., Extramural
    ZDB-ID 2474504-2
    ISSN 1879-6974 ; 1569-4909
    ISSN (online) 1879-6974
    ISSN 1569-4909
    DOI 10.1016/j.alcr.2023.100546
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  9. Article ; Online: Breastfeeding Duration and Timing of Bottle Supplementation: Associations with Body Mass Index from Childhood to Young-Adulthood.

    Blanco, Estela / Martinez, Suzanna M / East, Patricia / Burrows, Raquel / Correa-Burrows, Paulina / Lozoff, Betsy / Gahagan, Sheila

    Nutrients

    2023  Volume 15, Issue 14

    Abstract: Evidence for the association between breastfeeding (BF) duration and later body mass index (BMI) is inconsistent. We explored how BF duration and BF type (exclusive or partial) related to BMI from childhood to young adulthood in a Chilean cohort. Infants ...

    Abstract Evidence for the association between breastfeeding (BF) duration and later body mass index (BMI) is inconsistent. We explored how BF duration and BF type (exclusive or partial) related to BMI from childhood to young adulthood in a Chilean cohort. Infants were recruited at 6 months between 1994 and 1996 in Santiago, Chile (
    MeSH term(s) Infant ; Female ; Humans ; Child ; Young Adult ; Adult ; Breast Feeding ; Body Mass Index ; Milk, Human ; Mothers ; Dietary Supplements
    Language English
    Publishing date 2023-07-13
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 2518386-2
    ISSN 2072-6643 ; 2072-6643
    ISSN (online) 2072-6643
    ISSN 2072-6643
    DOI 10.3390/nu15143121
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  10. Article: Vascular malformations involving the female pelvis.

    Burrows, Patricia E

    Seminars in interventional radiology

    2011  Volume 25, Issue 4, Page(s) 347–360

    Abstract: Vascular malformations (VM) are classified by flow characteristics and channel content. They can involve any organ and tissue plane, and occur in focal and diffuse forms. Slow-flow vascular malformations (venous and lymphatic malformations) are typically ...

    Abstract Vascular malformations (VM) are classified by flow characteristics and channel content. They can involve any organ and tissue plane, and occur in focal and diffuse forms. Slow-flow vascular malformations (venous and lymphatic malformations) are typically treated by sclerotherapy, whereas fast-flow lesions (arteriovenous malformations) are managed with embolizations. Some VMs, such as VMs of the rectum or uterus, are best dealt with surgically. This review will present a summary of the conditions, their imaging features, and some useful endovascular therapeutic techniques.
    Language English
    Publishing date 2011-02-17
    Publishing country United States
    Document type Journal Article
    ZDB-ID 848341-3
    ISSN 1098-8963 ; 0739-9529
    ISSN (online) 1098-8963
    ISSN 0739-9529
    DOI 10.1055/s-0028-1102993
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

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