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Article ; Online: Role of G-Proteins and GPCR-Mediated Signalling in Neuropathophysiology.

Rahman, Md Mominur / Mim, Sadia Afsana / Islam, Md Rezaul / Sultana, Nasrin / Ahmed, Muniruddin / Kamal, Mohammad Amjad

CNS & neurological disorders drug targets

2022 Volume 22, Issue 1, Page(s) 2–5

Abstract: G-protein-coupled receptors (GPCRs) are activated by manifold neurotransmitters, and their activation, in turn, evokes slow synaptic transmission. They are profoundly related to numerous psychiatric and neurological disorders such as schizophrenia and ... ...

Abstract	G-protein-coupled receptors (GPCRs) are activated by manifold neurotransmitters, and their activation, in turn, evokes slow synaptic transmission. They are profoundly related to numerous psychiatric and neurological disorders such as schizophrenia and Parkinson's disease. The significant malady indications for GPCR modulators demonstrate a change towards obesity, diabetes, and Alzheimer's disease, while other central nervous system disorders persist highly represented. GPR52, GPR6, and GPR8 are recognised as orphan GPCRs, co-exist either with both the dopamine D2 and D1 receptors in neurons of the basal ganglia or with the dopamine D2 receptor alone, and recommend that between these orphan receptors, GPR52 has the maximum potential of being a therapeutic psychiatric receptor. Genetically modified creature models and molecular biological investigations have suggested that these improved GPCRs could be potential therapeutic psychiatric receptors. In this perspective, the role of molecular targets in GPCR-mediated signalling has been discussed that would be novel drug design and discovery options for a scientist to elaborate previous knowledge with modern techniques.
MeSH term(s)	GTP-Binding Proteins ; Nervous System Diseases ; Humans
Chemical Substances	GTP-Binding Proteins (EC 3.6.1.-)
Language	English
Publishing date	2022-05-04
Publishing country	United Arab Emirates
Document type	Journal Article
ZDB-ID	2228394-8
ISSN	1996-3181 ; 1871-5273
ISSN (online)	1996-3181
ISSN	1871-5273
DOI	10.2174/1871527321666220430142722
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Article: Citrus limon

Rahman, Md Mominur / Islam, Fahadul / Parvez, Anwar / Azad, Md A K / Ashraf, Ghulam Md / Ullah, Mohammad Fahad / Ahmed, Muniruddin

Journal of integrative neuroscience

2022 Volume 21, Issue 1, Page(s) 42

Abstract: ... Citrus ... ...

Abstract	Citrus limon
MeSH term(s)	Animals ; Behavior, Animal/drug effects ; Citrus ; Dose-Response Relationship, Drug ; Hypnotics and Sedatives/pharmacology ; Locomotion/drug effects ; Models, Animal ; Neurotransmitter Agents/pharmacology ; Phytochemicals/pharmacology ; Plant Extracts/pharmacology ; Seeds ; Sleep/drug effects ; Thiopental/pharmacology ; Time Factors
Chemical Substances	Hypnotics and Sedatives ; Neurotransmitter Agents ; Phytochemicals ; Plant Extracts ; Thiopental (JI8Z5M7NA3)
Language	English
Publishing date	2022-02-14
Publishing country	Singapore
Document type	Journal Article
ZDB-ID	2136427-8
ISSN	0219-6352
ISSN	0219-6352
DOI	10.31083/j.jin2101042
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Article ; Online: Retraction Note: Multifaceted role of natural sources for COVID-19 pandemic as marine drugs.

Rahman, Md Mominur / Islam, Md Rezaul / Shohag, Sheikh / Hossain, Md Emon / Shah, Muddaser / Shuvo, Shakil Khan / Khan, Hosneara / Chowdhury, Md Arifur Rahman / Bulbul, Israt Jahan / Hossain, Md Sarowar / Sultana, Sharifa / Ahmed, Muniruddin / Akhtar, Muhammad Furqan / Saleem, Ammara / Rahman, Md Habibur

Environmental science and pollution research international

2024 Volume 31, Issue 15, Page(s) 23299

Language	English
Publishing date	2024-03-14
Publishing country	Germany
Document type	Retraction of Publication
ZDB-ID	1178791-0
ISSN	1614-7499 ; 0944-1344
ISSN (online)	1614-7499
ISSN	0944-1344
DOI	10.1007/s11356-024-32891-0
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Article ; Online: Food Color Additives in Hazardous Consequences of Human Health: An Overview.

Sultana, Sharifa / Rahman, Md Mominur / Aovi, Farjana Islam / Jahan, Farhana Israt / Hossain, Md Sakhawat / Brishti, Shadia Afrin / Yamin, Md / Ahmed, Muniruddin / Rauf, Abdur / Sharma, Rohit

Current topics in medicinal chemistry

2023 Volume 23, Issue 14, Page(s) 1380–1393

Abstract: Food color additives are used to make food more appetizing. The United States Food and Drug Administration (FDA) permitted nine artificial colorings in foods, drugs, and cosmetics, whereas the European Union (EU) approved five artificial colors (E-104, ... ...

Abstract	Food color additives are used to make food more appetizing. The United States Food and Drug Administration (FDA) permitted nine artificial colorings in foods, drugs, and cosmetics, whereas the European Union (EU) approved five artificial colors (E-104, 122, 124, 131, and 142) for food. However, these synthetic coloring materials raise various health hazards. The present review aimed to summarize the toxic effects of these coloring food additives on the brain, liver, kidney, lungs, urinary bladder, and thyroid gland. In this respect, we aimed to highlight the scientific evidence and the crucial need to assess potential health hazards of all colors used in food on human and nonhuman biota for better scrutiny. Blue 1 causes kidney tumor in mice, and there is evidence of death due to ingestion through a feeding tube. Blue 2 and Citrus Red 2 cause brain and urinary bladder tumors, respectively, whereas other coloring additives may cause different types of cancers and numerous adverse health effects. In light of this, this review focuses on the different possible adverse health effects caused by these food coloring additives, and possible ways to mitigate or avoid the damage they may cause. We hope that the data collected from in vitro or in vivo studies and from clinical investigations related to the possible health hazards of food color additives will be helpful to both researchers and the food industry in the future.
MeSH term(s)	Animals ; Humans ; Mice ; Food Additives/adverse effects ; Food Coloring Agents/adverse effects ; Liver ; United States ; United States Food and Drug Administration
Chemical Substances	Food Additives ; Food Coloring Agents
Language	English
Publishing date	2023-02-14
Publishing country	United Arab Emirates
Document type	Review ; Journal Article
ZDB-ID	2064823-6
ISSN	1873-4294 ; 1568-0266
ISSN (online)	1873-4294
ISSN	1568-0266
DOI	10.2174/1568026623666230117122433
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Article ; Online: Emerging Promise of Therapeutic Approaches Targeting Mitochondria in Neurodegenerative Disorders.

Rahman, Md Mominur / Tumpa, Mst Afroza Alam / Rahaman, Md Saidur / Islam, Fahadul / Sutradhar, Popy Rani / Ahmed, Muniruddin / Alghamdi, Badrah S / Hafeez, Abdul / Alexiou, Athanasios / Perveen, Asma / Ashraf, Ghulam Md

Current neuropharmacology

2023 Volume 21, Issue 5, Page(s) 1081–1099

Abstract: Mitochondria are critical for homeostasis and metabolism in all cellular eukaryotes. Brain mitochondria are the primary source of fuel that supports many brain functions, including intracellular energy supply, cellular calcium regulation, regulation of ... ...

Abstract	Mitochondria are critical for homeostasis and metabolism in all cellular eukaryotes. Brain mitochondria are the primary source of fuel that supports many brain functions, including intracellular energy supply, cellular calcium regulation, regulation of limited cellular oxidative capacity, and control of cell death. Much evidence suggests that mitochondria play a central role in neurodegenerative disorders (NDDs) such as Parkinson's disease, Alzheimer's disease, Huntington's disease, and amyotrophic lateral sclerosis. Ongoing studies of NDDs have revealed that mitochondrial pathology is mainly found in inherited or irregular NDDs and is thought to be associated with the pathophysiological cycle of these disorders. Typical mitochondrial disturbances in NDDs include increased free radical production, decreased ATP synthesis, alterations in mitochondrial permeability, and mitochondrial DNA damage. The main objective of this review is to highlight the basic mitochondrial problems that occur in NDDs and discuss the use mitochondrial drugs, especially mitochondrial antioxidants, mitochondrial permeability transition blockade, and mitochondrial gene therapy, for the treatment and control of NDDs.
MeSH term(s)	Humans ; Oxidative Stress/physiology ; Mitochondrial Diseases/drug therapy ; Mitochondrial Diseases/metabolism ; Mitochondria/metabolism ; Neurodegenerative Diseases/drug therapy ; Neurodegenerative Diseases/genetics ; DNA, Mitochondrial/metabolism ; DNA, Mitochondrial/therapeutic use
Chemical Substances	DNA, Mitochondrial
Language	English
Publishing date	2023-03-17
Publishing country	United Arab Emirates
Document type	Review ; Journal Article
ZDB-ID	2192352-8
ISSN	1875-6190 ; 1570-159X
ISSN (online)	1875-6190
ISSN	1570-159X
DOI	10.2174/1570159X21666230316150559
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Article ; Online: A clinical study and future prospects for bioactive compounds and semi-synthetic molecules in the therapies for Huntington's disease.

Islam, Md Rezaul / Jony, Maruf Hossain / Thufa, Gazi Kaifeara / Akash, Shopnil / Dhar, Puja Sutra / Rahman, Md Mominur / Afroz, Tahmina / Ahmed, Muniruddin / Hemeg, Hassan A / Rauf, Abdur / Thiruvengadam, Muthu / Venkidasamy, Baskar

Molecular neurobiology

2023 Volume 61, Issue 3, Page(s) 1237–1270

Abstract: A neurodegenerative disorder (ND) refers to Huntington's disease (HD) which affects memory loss, weight loss, and movement dysfunctions such as chorea and dystonia. In the striatum and brain, HD most typically impacts medium-spiny neurons. Molecular ... ...

Abstract	A neurodegenerative disorder (ND) refers to Huntington's disease (HD) which affects memory loss, weight loss, and movement dysfunctions such as chorea and dystonia. In the striatum and brain, HD most typically impacts medium-spiny neurons. Molecular genetics, excitotoxicity, oxidative stress (OS), mitochondrial, and metabolic dysfunction are a few of the theories advanced to explicit the pathophysiology of neuronal damage and cell death. Numerous in-depth studies of the literature have supported the therapeutic advantages of natural products in HD experimental models and other treatment approaches. This article briefly discusses the neuroprotective impacts of natural compounds against HD models. The ability of the discovered natural compounds to suppress HD was tested using either in vitro or in vivo models. Many bioactive compounds considerably lessened the memory loss and motor coordination brought on by 3-nitropropionic acid (3-NP). Reduced lipid peroxidation, increased endogenous enzymatic antioxidants, reduced acetylcholinesterase activity, and enhanced mitochondrial energy generation have profoundly decreased the biochemical change. It is significant since histology showed that therapy with particular natural compounds lessened damage to the striatum caused by 3-NP. Moreover, natural products displayed varying degrees of neuroprotection in preclinical HD studies because of their antioxidant and anti-inflammatory properties, maintenance of mitochondrial function, activation of autophagy, and inhibition of apoptosis. This study highlighted about the importance of bioactive compounds and their semi-synthetic molecules in the treatment and prevention of HD.
MeSH term(s)	Rats ; Animals ; Huntington Disease/metabolism ; Rats, Wistar ; Acetylcholinesterase ; Antioxidants/pharmacology ; Antioxidants/therapeutic use ; Biological Products/therapeutic use ; Nitro Compounds/pharmacology ; Propionates/pharmacology ; Propionates/therapeutic use ; Neuroprotective Agents/pharmacology ; Neuroprotective Agents/therapeutic use ; Disease Models, Animal
Chemical Substances	Acetylcholinesterase (EC 3.1.1.7) ; Antioxidants ; Biological Products ; Nitro Compounds ; Propionates ; Neuroprotective Agents
Language	English
Publishing date	2023-09-12
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	645020-9
ISSN	1559-1182 ; 0893-7648
ISSN (online)	1559-1182
ISSN	0893-7648
DOI	10.1007/s12035-023-03604-4
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Zs.A 2411: Show issues

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Article ; Online: Hutchinson-Gilford Progeria Syndrome: An Overview of the Molecular Mechanism, Pathophysiology and Therapeutic Approach.

Rahman, Md Mominur / Ferdous, Kazi Sayma / Ahmed, Muniruddin / Islam, Mohammad Touhidul / Khan, Md Robin / Perveen, Asma / Ashraf, Ghulam Md / Uddin, Md Sahab

Current gene therapy

2021 Volume 21, Issue 3, Page(s) 216–229

Abstract: Lamin A/C encoded by the LMNA gene is an essential component for maintaining the nuclear structure. Mutation in the lamin A/C leads to a group of inherited disorders is known as laminopathies. In the human body, there are several mutations in the LMNA ... ...

Abstract	Lamin A/C encoded by the LMNA gene is an essential component for maintaining the nuclear structure. Mutation in the lamin A/C leads to a group of inherited disorders is known as laminopathies. In the human body, there are several mutations in the LMNA gene that have been identified. It can affect diverse organs or tissues or can be systemic, causing different diseases. In this review, we mainly focused on one of the most severe laminopathies, Hutchinson-Gilford progeria syndrome (HGPS). HGPS is an immensely uncommon, deadly, metameric ill-timed laminopathies caused by the abnormal splicing of the LMNA gene and production of an aberrant protein known as progerin. Here, we also presented the currently available data on the molecular mechanism, pathophysiology, available treatment, and future approaches to this deadly disease. Due to the production of progerin, an abnormal protein leads to an abnormality in nuclear structure, defects in DNA repair, shortening of telomere, and impairment in gene regulation which ultimately results in aging in the early stage of life. Now some treatment options are available for this disease, but a proper understanding of the molecular mechanism of this disease will help to develop a more appropriate treatment which makes it an emerging area of research.
MeSH term(s)	Cell Nucleus/metabolism ; DNA Repair ; Gene Expression Regulation ; Gene Silencing ; Humans ; Lamin Type A/physiology ; Mutation ; Phenotype ; Progeria/physiopathology ; Progeria/therapy ; Telomere/physiology
Chemical Substances	Lamin Type A ; prelamin A
Language	English
Publishing date	2021-03-02
Publishing country	United Arab Emirates
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	2146187-9
ISSN	1875-5631 ; 1566-5232
ISSN (online)	1875-5631
ISSN	1566-5232
DOI	10.2174/1566523221666210303100805
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Article ; Online: Bacterial Resistance in Pneumonia in Developing Countries-A Role for Iron Chelation.

Islam, Sufia / Chisti, Mohammod Jobayer / Ahmed, Muniruddin / Anwar, Nafiza / Lehmann, Christian

Tropical medicine and infectious disease

2019 Volume 4, Issue 2

Abstract: Pneumonia represents one of the major infectious diseases in developing countries and is associated with high mortality, in particular in children under the age of five. The main causative bacterial agents ... ...

Abstract	Pneumonia represents one of the major infectious diseases in developing countries and is associated with high mortality, in particular in children under the age of five. The main causative bacterial agents are
Language	English
Publishing date	2019-04-10
Publishing country	Switzerland
Document type	Journal Article ; Review
ISSN	2414-6366
ISSN (online)	2414-6366
DOI	10.3390/tropicalmed4020059
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Article ; Online: Exploring the management approaches of cytokines including viral infection and neuroinflammation for neurological disorders.

Mominur Rahman, Md / Afsana Mim, Sadia / Afroza Alam Tumpa, Mst / Taslim Sarker, Md / Ahmed, Muniruddin / Alghamdi, Badrah S / Hafeez, Abdul / Alexiou, Athanasios / Perveen, Asma / Md Ashraf, Ghulam

Cytokine

2022 Volume 157, Page(s) 155962

Abstract: Considerable evidence supports that cytokines are important mediators of pathophysiologic processes within the central nervous system (CNS). Numerous studies have documented the increased production of various cytokines in the human CNS in various ... ...

Abstract	Considerable evidence supports that cytokines are important mediators of pathophysiologic processes within the central nervous system (CNS). Numerous studies have documented the increased production of various cytokines in the human CNS in various neurological and neuropsychiatric disorders. Deciphering cytokine actions in the intact CNS has important implications for our understanding of the pathogenesis and treatment of these disorders. The purpose of this study is to discuss the recent research on treating cytokine storm and amyloids, including stroke, Parkinson's disease (PD), Alzheimer's disease (AD), Huntington's condition, Multi-sclerosis (MS), Amyotrophic Lateral Sclerosis (ALS). Neuroinflammation observed in neurological disorders has a pivotal role in exacerbating Aβ burden and tau hyperphosphorylation, suggesting that stimulating cytokines in response to an undesirable external response could be a checkpoint for treating neurological disorders. Furthermore, the pro-inflammatory cytokines help our immune system through a neuroprotective mechanism in clearing viral infection by recruiting mononuclear cells. This study reveals that cytokine applications may play a vital role in providing novel regulation and methods for the therapeutic approach to neurological disorders and the causes of the deregulation, which is responsible for neuroinflammation and viral infection. However, it needs to be further investigated to clarify better the mechanisms of cytokine release in response to various stimuli, which could be the central point for treating neurological disorders.
MeSH term(s)	Alzheimer Disease ; Cytokines/physiology ; Humans ; Nervous System Diseases/therapy ; Neuroinflammatory Diseases ; Virus Diseases
Chemical Substances	Cytokines
Language	English
Publishing date	2022-07-16
Publishing country	England
Document type	Journal Article ; Review ; Research Support, Non-U.S. Gov't
ZDB-ID	1018055-2
ISSN	1096-0023 ; 1043-4666
ISSN (online)	1096-0023
ISSN	1043-4666
DOI	10.1016/j.cyto.2022.155962
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Article ; Online: Bioactive Compounds and Diabetes Mellitus: Prospects and Future Challenges.

Rahman, Md Mominur / Islam, Md Rezaul / Rabbi, Fazle / Islam, Mohammad Touhidul / Sultana, Sharifa / Ahmed, Muniruddin / Sehgal, Aayush / Singh, Sukhbir / Sharma, Neelam / Behl, Tapan

Current pharmaceutical design

2022 Volume 28, Issue 16, Page(s) 1304–1320

Abstract: Diabetes mellitus is a metabolic condition that influences the endocrine framework. Hyperglycemia and hyperlipidemia are two of the most widely recognized metabolic irregularities in diabetes and two of the most well-known reasons for diabetic ... ...

Abstract	Diabetes mellitus is a metabolic condition that influences the endocrine framework. Hyperglycemia and hyperlipidemia are two of the most widely recognized metabolic irregularities in diabetes and two of the most well-known reasons for diabetic intricacies. Diabetes mellitus is a persistent illness brought about by metabolic irregularities in hyperglycemic pancreatic cells. Hyperglycemia can be brought about by an absence of insulin-producing beta cells in the pancreas (Type 1 diabetes mellitus) or inadequate insulin creation that does not work effectively (Type 2 diabetes mellitus). Present diabetes medication directs blood glucose levels in the systemic circulation to the typical levels. Numerous advanced prescription medicines have many negative results that can bring about unexpected severe issues during treatment of the bioactive compound from a different source that is beneficially affected by controlling and adjusting metabolic pathways or cycles. Moreover, a few new bioactive medications disengaged from plants have shown antidiabetic action with more noteworthy adequacy than the oral hypoglycemic agent that specialists have utilized in clinical treatment lately. Since bioactive mixtures are collected from familiar sources, they have a great activity in controlling diabetes mellitus. This study discusses bioactive compounds, their activity in managing diabetes mellitus, and their prospects. Though bioactive compounds have many health-beneficial properties, adequate clinical studies still need to acknowledge that they effectively manage diabetes mellitus.
MeSH term(s)	Diabetes Mellitus/drug therapy ; Diabetes Mellitus, Type 2/drug therapy ; Diabetes Mellitus, Type 2/metabolism ; Humans ; Hyperglycemia/drug therapy ; Hypoglycemic Agents/pharmacology ; Hypoglycemic Agents/therapeutic use ; Insulin ; Insulin-Secreting Cells
Chemical Substances	Hypoglycemic Agents ; Insulin
Language	English
Publishing date	2022-04-13
Publishing country	United Arab Emirates
Document type	Journal Article
ZDB-ID	1304236-1
ISSN	1873-4286 ; 1381-6128
ISSN (online)	1873-4286
ISSN	1381-6128
DOI	10.2174/1381612828666220412090808
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