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Article ; Online: Intoxication pédiatrique sévère avec une faible dose de clonidine : à propos d'un cas.

Schmitt, C / Kervégant, M / Ajaltouni, Z / Tauber, M / Tichadou, L / de Haro, L

Archives de pediatrie : organe officiel de la Societe francaise de pediatrie

2014 Volume 21, Issue 11, Page(s) 1213–1215

Abstract: Clonidine poisoning's clinical feature is well documented in the medical literature, but the minimal toxic dose has not yet been established. The effectiveness of naloxone is also controversial. The authors describe a clonidine overdose in a 9-year-old ... ...

Title translation	Clonidine poisoning in a child: a case report.
Abstract	Clonidine poisoning's clinical feature is well documented in the medical literature, but the minimal toxic dose has not yet been established. The effectiveness of naloxone is also controversial. The authors describe a clonidine overdose in a 9-year-old boy (25 kg) during a growth hormone test: he received tenfold the prescribed clonidine dose (0.23 mg instead of 0.023 mg) with 6.2 mg betaxolol. About 40 min later, he became drowsy and then complained of low blood pressure, bradycardia, and myosis. By maintaining the Trendelenburg position, administering fluids as well as salbutamol and naloxone (three doses of 0.2 mg were required), he recovered and was discharged from the hospital on day 2. The minimal clonidine toxic dose, the clinical picture, and the effectiveness of naloxone administration are discussed in this paper.
MeSH term(s)	Adrenergic alpha-2 Receptor Agonists/poisoning ; Albuterol/administration & dosage ; Antihypertensive Agents/poisoning ; Betaxolol/administration & dosage ; Betaxolol/poisoning ; Blood Pressure/drug effects ; Child ; Clonidine/administration & dosage ; Clonidine/poisoning ; Combined Modality Therapy ; Dose-Response Relationship, Drug ; Drug Overdose/diagnosis ; Drug Overdose/drug therapy ; Drug Therapy, Combination ; Heart Rate/drug effects ; Humans ; Male ; Medication Errors ; Naloxone/administration & dosage
Chemical Substances	Adrenergic alpha-2 Receptor Agonists ; Antihypertensive Agents ; Naloxone (36B82AMQ7N) ; Clonidine (MN3L5RMN02) ; Betaxolol (O0ZR1R6RZ2) ; Albuterol (QF8SVZ843E)
Language	French
Publishing date	2014-11
Publishing country	France
Document type	Case Reports ; English Abstract ; Journal Article
ZDB-ID	1181947-9
ISSN	1769-664X ; 0929-693X
ISSN (online)	1769-664X
ISSN	0929-693X
DOI	10.1016/j.arcped.2014.07.014
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Article ; Online: Syndrome de Klinefelter et syndrome de Turner : pour une meilleure prise en charge.

Pienkowski, C / Cartault, A / Caula-Legriel, S / Ajaltouni, Z / Daudin, M / Tauber, M

Gynecologie, obstetrique & fertilite

2011 Volume 39, Issue 9, Page(s) 521–524

Abstract: Klinefelter's syndrome (KS) affects one in 600 men and Turner's syndrome (TS), one in 2500 women. These 2 diseases are the most sex chromosome disorders characterized by one extra X in the SK male (47XXY) and the loss of an X in the girls with ST (45 X). ...

Title translation	Klinefelter's syndrome and Turner's syndrome. For a better management.
Abstract	Klinefelter's syndrome (KS) affects one in 600 men and Turner's syndrome (TS), one in 2500 women. These 2 diseases are the most sex chromosome disorders characterized by one extra X in the SK male (47XXY) and the loss of an X in the girls with ST (45 X). Their common characteristic is the gonadal dysgenesis, which is the main cause of male or female infertility. Called "the forgotten syndrome", KS is under-diagnosed because apart from the large size, there are no dysmorphic features, along with a great ignorance of cognitive and language disorders in children. There are often comorbidities that lead to diagnosis such as autoimmune diseases or metabolic syndrome. TS is often diagnosed by the short stature. Management of Turner's girls has profoundly changed with Growth hormone therapy. There is an international consensus for a better management of associated diseases such as ORL, cardiac, renal, hepatic, autoimmune and metabolic diseases. Prenatal diagnosis allows early detection and management of cognitive deficiencies and of associated diseases.
MeSH term(s)	Cognition Disorders/etiology ; Cognition Disorders/genetics ; Cognition Disorders/therapy ; Female ; Gonadal Dysgenesis/etiology ; Gonadal Dysgenesis/genetics ; Humans ; Infertility/etiology ; Infertility/genetics ; Klinefelter Syndrome/complications ; Klinefelter Syndrome/diagnosis ; Klinefelter Syndrome/therapy ; Language Disorders/etiology ; Language Disorders/genetics ; Language Disorders/therapy ; Male ; Pregnancy ; Prenatal Diagnosis ; Turner Syndrome/complications ; Turner Syndrome/diagnosis ; Turner Syndrome/therapy
Language	French
Publishing date	2011-09
Publishing country	France
Document type	English Abstract ; Journal Article
ZDB-ID	2008222-8
ISSN	1769-6682 ; 1297-9589
ISSN (online)	1769-6682
ISSN	1297-9589
DOI	10.1016/j.gyobfe.2011.07.009
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Article ; Online: Unexpected high frequency of skeletal dysplasia in idiopathic short stature and small for gestational age patients.

Flechtner, I / Lambot-Juhan, K / Teissier, R / Colmenares, A / Baujat, G / Beltrand, J / Ajaltouni, Z / Pauwels, C / Pinto, G / Samara-Boustani, D / Simon, A / Thalassinos, C / Le Merrer, M / Cormier-Daire, V / Polak, M

European journal of endocrinology

2014 Volume 170, Issue 5, Page(s) 677–684

Abstract: Objective: To assess the prevalence of skeletal dysplasias (SDs) in patients with idiopathic short stature (ISS) or small for gestational age (SGA) status.: Setting: Rare Endocrine/Growth Diseases Center in Paris, France.: Design: A prospective ... ...

Abstract	Objective: To assess the prevalence of skeletal dysplasias (SDs) in patients with idiopathic short stature (ISS) or small for gestational age (SGA) status. Setting: Rare Endocrine/Growth Diseases Center in Paris, France. Design: A prospective study on consecutive patients with ISS and SGA enrolled from 2004 to 2009. Method: We used a standardized workup to classify patients into well-established diagnostic categories. Of 713 patients with ISS (n=417) or SGA status (n=296), 50.9% underwent a skeletal survey. We chose patients labeled normal or with a prepubertal slowdown of growth as a comparison group. Results: Diagnoses were ISS (16.9%), SGA (13.5%), normal growth (24.5%), transient growth rate slowing (17.3%), endocrine dysfunction (12%), genetic syndrome (8.9%), chronic disease (5.1%), and known SD (1.8%). SD was found in 20.9% of SGA and 21.8% ISS patients and in only 13.2% in our comparison group. SD prevalence was significantly higher in the ISS group than in the comparison group, especially (50%) for patients having at least one parent whose height was <-2 SDS. Dyschondrosteosis and hypochondroplasia were the most frequently identified SD, and genetic anomaly was found in 61.5 and 30% respectively. Subtle SD was found equally in the three groups and require long-term growth follow-up to evaluate the impact on final height. Conclusion: SD may explain more than 20% of cases of growth retardation ascribed to ISS or SGA, and this proportion is higher when parental height is <-2 SDS. A skeletal survey should be obtained in patients with delayed growth in a context of ISS or SGA.
MeSH term(s)	Adolescent ; Bone Diseases, Developmental/epidemiology ; Bone Diseases, Developmental/genetics ; Bone Diseases, Developmental/physiopathology ; Bone and Bones/abnormalities ; Bone and Bones/physiopathology ; Child ; Child, Preschool ; Cohort Studies ; Dwarfism/epidemiology ; Dwarfism/genetics ; Dwarfism/physiopathology ; Family Health ; Female ; Fetal Growth Retardation/epidemiology ; Fetal Growth Retardation/genetics ; Fetal Growth Retardation/physiopathology ; France/epidemiology ; Genetic Variation ; Growth Disorders/epidemiology ; Growth Disorders/etiology ; Growth Disorders/genetics ; Growth Disorders/physiopathology ; Hospitals, Pediatric ; Hospitals, Teaching ; Humans ; Infant ; Infant, Small for Gestational Age ; Limb Deformities, Congenital/epidemiology ; Limb Deformities, Congenital/genetics ; Limb Deformities, Congenital/physiopathology ; Lordosis/epidemiology ; Lordosis/genetics ; Lordosis/physiopathology ; Male ; Osteochondrodysplasias/epidemiology ; Osteochondrodysplasias/genetics ; Osteochondrodysplasias/physiopathology ; Prevalence ; Prospective Studies ; Referral and Consultation
Language	English
Publishing date	2014-05
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1183856-5
ISSN	1479-683X ; 0804-4643
ISSN (online)	1479-683X
ISSN	0804-4643
DOI	10.1530/EJE-13-0864
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Book ; Online: Search for the decay $B^0\to\phi\mu^+\mu^-$

LHCb collaboration / Aaij, R. / Abdelmotteleb, A. S. W. / Beteta, C. Abellán / Abudinén, F. / Ackernley, T. / Adeva, B. / Adinolfi, M. / Afsharnia, H. / Agapopoulou, C. / Aidala, C. A. / Aiola, S. / Ajaltouni, Z. / Akar, S. / Albrecht, J. / Alessio, F. / Alexander, M. / Albero, A. Alfonso / Aliouche, Z. /

Alkhazov, G. / Cartelle, P. Alvarez / Amato, S. / Amey, J. L. / Amhis, Y. / An, L. / Anderlini, L. / Andersson, M. / Andreianov, A. / Andreotti, M. / Archilli, F. / Artamonov, A. / Artuso, M. / Arzymatov, K. / Aslanides, E. / Atzeni, M. / Audurier, B. / Bachmann, S. / Bachmayer, M. / Back, J. J. / Rodriguez, P. Baladron / Balagura, V. / Baldini, W. / Leite, J. Baptista de Souza / Barbetti, M. / Barlow, R. J. / Barsuk, S. / Barter, W. / Bartolini, M. / Baryshnikov, F. / Basels, J. M.

2022

Abstract: A search for the decay $B^0\to\phi\mu^+\mu^-$ is performed using proton-proton collisions at centre-of-mass energies of 7, 8, and 13 TeV collected by the LHCb experiment and corresponding to an integrated luminosity of 9 fb$^{-1}$. No evidence for the $B^ ...

Abstract	A search for the decay $B^0\to\phi\mu^+\mu^-$ is performed using proton-proton collisions at centre-of-mass energies of 7, 8, and 13 TeV collected by the LHCb experiment and corresponding to an integrated luminosity of 9 fb$^{-1}$. No evidence for the $B^0\to \phi \mu^+ \mu^-$ decay is found and an upper limit on the branching fraction, excluding the $\phi$ and charmonium regions in the dimuon spectrum, of $4.4 \times 10^{-3}$ at a 90$\%$ credibility level, relative to that of the $B^0_s \to \phi \mu^+ \mu^-$ decay, is established. Using the measured $B^0_s\to\phi\mu^+\mu^-$ branching fraction and assuming a phase-space model, the absolute branching fraction of the decay $B^0\to \phi \mu^+ \mu^-$ in the full $q^2$ range is determined to be less than $3.2 \times 10^{-9}$ at a 90$\%$ credibility level. Comment: All figures and tables, along with machine-readable versions and any supplementary material and additional information, are available at https://cern.ch/lhcbproject/Publications/p/LHCb-PAPER-2021-042.html (LHCb public pages)
Keywords	High Energy Physics - Experiment
Subject code	612
Publishing date	2022-01-25
Publishing country	us
Document type	Book ; Online
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Article ; Online: Pituitary Stalk Interruption Syndrome from Infancy to Adulthood: Clinical, Hormonal, and Radiological Assessment According to the Initial Presentation.

Bar, Céline / Zadro, Charline / Diene, Gwenaelle / Oliver, Isabelle / Pienkowski, Catherine / Jouret, Béatrice / Cartault, Audrey / Ajaltouni, Zeina / Salles, Jean-Pierre / Sevely, Annick / Tauber, Maithé / Edouard, Thomas

PloS one

2015 Volume 10, Issue 11, Page(s) e0142354

Abstract: Background: Patients with pituitary stalk interruption syndrome (PSIS) are initially referred for hypoglycemia during the neonatal period or growth retardation during childhood. PSIS is either isolated (nonsyndromic) or associated with extra-pituitary ... ...

Abstract	Background: Patients with pituitary stalk interruption syndrome (PSIS) are initially referred for hypoglycemia during the neonatal period or growth retardation during childhood. PSIS is either isolated (nonsyndromic) or associated with extra-pituitary malformations (syndromic). Objective: To compare baseline characteristics and long-term evolution in patients with PSIS according to the initial presentation. Study design: Sixty-seven patients with PSIS were included. Data from subgroups were compared: neonates (n = 10) versus growth retardation patients (n = 47), and syndromic (n = 32) versus nonsyndromic patients (n = 35). Results: Neonates displayed a more severe hormonal and radiological phenotype than children referred for growth retardation, with a higher incidence of multiple hormonal deficiencies (100% versus 34%; P = 0.0005) and a nonvisible anterior pituitary lobe (33% versus 2%; P = 0.0017). Regular follow-up of growth might have allowed earlier diagnosis in the children with growth retardation, as decreased growth velocity and growth retardation were present respectively 3 and 2 years before referral. We documented a progressive worsening of endocrine impairment throughout childhood in these patients. Presence of extra-pituitary malformations (found in 48%) was not associated with more severe hormonal and radiological characteristics. Growth under GH treatment was similar in the patient groups and did not vary according to the pituitary MRI findings. Conclusions: PSIS diagnosed in the neonatal period has a particularly severe hormonal and radiological phenotype. The progressive worsening of endocrine impairment throughout childhood justifies periodic follow-up to check for additional hormonal deficiencies.
MeSH term(s)	Adolescent ; Adult ; Child ; Child, Preschool ; Female ; Growth Disorders/blood ; Growth Disorders/drug therapy ; Hormone Replacement Therapy ; Hormones/blood ; Hormones/deficiency ; Hormones/therapeutic use ; Humans ; Infant ; Infant, Newborn ; Longitudinal Studies ; Magnetic Resonance Imaging ; Male ; Pituitary Diseases/blood ; Pituitary Diseases/diagnosis ; Pituitary Diseases/drug therapy ; Pituitary Gland/abnormalities ; Pituitary Gland/diagnostic imaging ; Pituitary Gland, Anterior/abnormalities ; Pituitary Gland, Anterior/diagnostic imaging ; Radiography ; Regression Analysis ; Retrospective Studies ; Syndrome ; Treatment Outcome
Chemical Substances	Hormones
Language	English
Publishing date	2015
Publishing country	United States
Document type	Journal Article
ISSN	1932-6203
ISSN (online)	1932-6203
DOI	10.1371/journal.pone.0142354
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Book ; Online: Measurement of the nuclear modification factor and prompt charged particle production in $p\mathrm{Pb}$ and $pp$ collisions at $\sqrt{s_{\scriptscriptstyle\mathrm{NN}}}=5\,\mathrm{TeV}$

LHCb collaboration / Aaij, R. / Beteta, C. Abellán / Ackernley, T. / Adeva, B. / Adinolfi, M. / Afsharnia, H. / Aidala, C. A. / Aiola, S. / Ajaltouni, Z. / Akar, S. / Albrecht, J. / Alessio, F. / Alexander, M. / Albero, A. Alfonso / Aliouche, Z. / Alkhazov, G. / Cartelle, P. Alvarez / Amato, S. /

Amey, J. L. / Amhis, Y. / An, L. / Anderlini, L. / Andreianov, A. / Andreotti, M. / Archilli, F. / Artamonov, A. / Artuso, M. / Arzymatov, K. / Aslanides, E. / Atzeni, M. / Audurier, B. / Bachmann, S. / Bachmayer, M. / Back, J. J. / Rodriguez, P. Baladron / Balagura, V. / Baldini, W. / Leite, J. Baptista / Barlow, R. J. / Barsuk, S. / Barter, W. / Bartolini, M. / Baryshnikov, F. / Basels, J. M. / Bassi, G. / Batsukh, B. / Battig, A. / Bay, A. / Becker, M.

2021

Abstract: The production of prompt charged particles in proton-lead collisions and in proton-proton collisions at the nucleon-nucleon centre-of-mass energy ${\sqrt{s_{\scriptscriptstyle\mathrm{NN}}}=5\,\mathrm{TeV}}$ is studied at LHCb as a function of ... ...

Abstract	The production of prompt charged particles in proton-lead collisions and in proton-proton collisions at the nucleon-nucleon centre-of-mass energy ${\sqrt{s_{\scriptscriptstyle\mathrm{NN}}}=5\,\mathrm{TeV}}$ is studied at LHCb as a function of pseudorapidity ($\eta$) and transverse momentum ($p_{\mathrm{T}}$) with respect to the proton beam direction. The nuclear modification factor for charged particles is determined as a function of $\eta$ between ${-4.8<\eta<-2.5}$ (backward region) and ${2.0<\eta<4.8}$ (forward region), and $p_{\mathrm{T}}$ between ${0.2<p_{\mathrm{T}}<8.0\,\mathrm{GeV}/c}$. The results show a suppression of charged particle production in proton-lead collisions relative to proton-proton collisions in the forward region and an enhancement in the backward region for $p_{\mathrm{T}}$ larger than $1.5\,\mathrm{GeV}/c$. This measurement constrains nuclear PDFs and saturation models at previously unexplored values of the parton momentum fraction down to $10^{-6}$.<br /> Comment: All figures and tables, along with any supplementary material and additional information, are available at https://cern.ch/lhcbproject/Publications/p/LHCb-PAPER-2021-015.html (LHCb public pages)
Keywords	High Energy Physics - Experiment ; Nuclear Experiment
Subject code	306
Publishing date	2021-08-30
Publishing country	us
Document type	Book ; Online
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Article: Unusual phenotypic features in a patient with a novel splice mutation in the GHRHR gene.

Hilal, Latifa / Hajaji, Yassir / Vie-Luton, Marie-Pierre / Ajaltouni, Zeina / Benazzouz, Bouchra / Chana, Maha / Chraïbi, Adelmajid / Kadiri, Abdelkrim / Amselem, Serge / Sobrier, Marie-Laure

Molecular medicine (Cambridge, Mass.)

2008 Volume 14, Issue 5-6, Page(s) 286–292

Abstract: Isolated growth hormone deficiency (IGHD) may be of genetic origin. One of the few genes involved in that condition encodes the growth hormone releasing hormone receptor (GHRHR) that, through its ligand (GHRH), plays a pivotal role in the GH synthesis ... ...

Abstract	Isolated growth hormone deficiency (IGHD) may be of genetic origin. One of the few genes involved in that condition encodes the growth hormone releasing hormone receptor (GHRHR) that, through its ligand (GHRH), plays a pivotal role in the GH synthesis and secretion by the pituitary. Our objective is to describe the phenotype of two siblings born to a consanguineous union presenting with short stature (IGHD) and Magnetic Resonance Imaging (MRI) abnormalities, and to identify the molecular basis of this condition. Our main outcome measures were clinical and endocrinological investigations, MRI of the pituitary region, study of the GHRHR gene sequence and transcripts. In both patients, the severe growth retardation (-5SD) was combined with anterior pituitary hypoplasia. In addition to these classical phenotypic features for IGHD, one of the patients had a Chiari I malformation, an arachnoid cyst, and a dysmorphic anterior pituitary. A homozygous sequence variation in the consensus donor splice site of intron 1 (IVS1 + 2T > G) of the GHRHR gene was identified in both patients. Using in vitro transcription assay, we showed that this mutation results in abnormal splicing of GHRHR transcripts. In this report, which broadens the phenotype associated with GHRHR defects, we discuss the possible role of the GHRHR in the proper development of extrapituitary structures, through a mechanism that could be direct or secondary to severe GH deficiency.
MeSH term(s)	Alternative Splicing ; Child ; DNA Mutational Analysis ; Dwarfism, Pituitary/drug therapy ; Dwarfism, Pituitary/genetics ; Dwarfism, Pituitary/pathology ; Female ; Growth Hormone/therapeutic use ; Humans ; Magnetic Resonance Imaging ; Male ; Mutation ; Pedigree ; Phenotype ; RNA Splice Sites/genetics ; Receptors, Neuropeptide/genetics ; Receptors, Neuropeptide/physiology ; Receptors, Pituitary Hormone-Regulating Hormone/genetics ; Receptors, Pituitary Hormone-Regulating Hormone/physiology ; Reverse Transcriptase Polymerase Chain Reaction
Chemical Substances	RNA Splice Sites ; Receptors, Neuropeptide ; Receptors, Pituitary Hormone-Regulating Hormone ; Growth Hormone (9002-72-6) ; somatotropin releasing hormone receptor (F8L0ODC9D7)
Language	English
Publishing date	2008-02-20
Publishing country	England
Document type	Case Reports ; Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1283676-x
ISSN	1076-1551
ISSN	1076-1551
DOI	10.2119/2007-00128.Hilal
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Article ; Online: Measurement of CP Violation in B^{0}→ψ(→ℓ^{+}ℓ^{-})K_{S}^{0}(→π^{+}π^{-}) Decays.

Aaij, R / Abdelmotteleb, A S W / Abellan Beteta, C / Abudinén, F / Ackernley, T / Adeva, B / Adinolfi, M / Adlarson, P / Afsharnia, H / Agapopoulou, C / Aidala, C A / Ajaltouni, Z / Akar, S / Akiba, K / Albicocco, P / Albrecht, J / Alessio, F / Alexander, M / Alfonso Albero, A /

Aliouche, Z / Alvarez Cartelle, P / Amalric, R / Amato, S / Amey, J L / Amhis, Y / An, L / Anderlini, L / Andersson, M / Andreianov, A / Andreola, P / Andreotti, M / Andreou, D / Ao, D / Archilli, F / Artamonov, A / Artuso, M / Aslanides, E / Atzeni, M / Audurier, B / Bacher, D / Bachiller Perea, I / Bachmann, S / Bachmayer, M / Back, J J / Bailly-Reyre, A / Baladron Rodriguez, P / Balagura, V / Baldini, W / Baptista de Souza Leite, J / Barbetti, M / Barbosa, I R / Barlow, R J / Barsuk, S / Barter, W / Bartolini, M / Baryshnikov, F / Basels, J M / Bassi, G / Batsukh, B / Battig, A / Bay, A / Beck, A / Becker, M / Bedeschi, F / Bediaga, I B / Beiter, A / Belin, S / Bellee, V / Belous, K / Belov, I / Belyaev, I / Benane, G / Bencivenni, G / Ben-Haim, E / Berezhnoy, A / Bernet, R / Bernet Andres, S / Berninghoff, D / Bernstein, H C / Bertella, C / Bertolin, A / Betancourt, C / Betti, F / Bex, J / Bezshyiko, Ia / Bhom, J / Bian, L / Bieker, M S / Biesuz, N V / Billoir, P / Biolchini, A / Birch, M / Bishop, F C R / Bitadze, A / Bizzeti, A / Blago, M P / Blake, T / Blanc, F / Blank, J E / Blusk, S / Bobulska, D / Bocharnikov, V / Boelhauve, J A / Boente Garcia, O / Boettcher, T / Bohare, A / Boldyrev, A / Bolognani, C S / Bolzonella, R / Bondar, N / Borgato, F / Borghi, S / Borsato, M / Borsuk, J T / Bouchiba, S A / Bowcock, T J V / Boyer, A / Bozzi, C / Bradley, M J / Braun, S / Brea Rodriguez, A / Breer, N / Brodzicka, J / Brossa Gonzalo, A / Brown, J / Brundu, D / Buonaura, A / Buonincontri, L / Burke, A T / Burr, C / Bursche, A / Butkevich, A / Butter, J S / Buytaert, J / Byczynski, W / Cadeddu, S / Cai, H / Calabrese, R / Calefice, L / Cali, S / Calvi, M / Calvo Gomez, M / Cambon Bouzas, J / Campana, P / Campora Perez, D H / Campoverde Quezada, A F / Capelli, S / Capriotti, L / Carbone, A / Carcedo Salgado, L / Cardinale, R / Cardini, A / Carniti, P / Carus, L / Casais Vidal, A / Caspary, R / Casse, G / Cattaneo, M / Cavallero, G / Cavallini, V / Celani, S / Cerasoli, J / Cervenkov, D / Chadwick, A J / Chahrour, I / Chapman, M G / Charles, M / Charpentier, Ph / Chavez Barajas, C A / Chefdeville, M / Chen, C / Chen, S / Chernov, A / Chernyshenko, S / Chobanova, V / Cholak, S / Chrzaszcz, M / Chubykin, A / Chulikov, V / Ciambrone, P / Cicala, M F / Cid Vidal, X / Ciezarek, G / Cifra, P / Clarke, P E L / Clemencic, M / Cliff, H V / Closier, J / Cobbledick, J L / Cocha Toapaxi, C / Coco, V / Cogan, J / Cogneras, E / Cojocariu, L / Collins, P / Colombo, T / Comerma-Montells, A / Congedo, L / Contu, A / Cooke, N / Corredoira, I / Correia, A / Corti, G / Cottee Meldrum, J J / Couturier, B / Craik, D C / Cruz Torres, M / Currie, R / Da Silva, C L / Dadabaev, S / Dai, L / Dai, X / Dall'Occo, E / Dalseno, J / D'Ambrosio, C / Daniel, J / Danilina, A / d'Argent, P / Davidson, A / Davies, J E / Davis, A / De Aguiar Francisco, O / de Boer, J / De Bruyn, K / De Capua, S / De Cian, M / De Freitas Carneiro Da Graca, U / De Lucia, E / De Miranda, J M / De 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Physical review letters

2024 Volume 132, Issue 2, Page(s) 21801

Abstract: A measurement of time-dependent CP violation in the decays of B^{0} and B[over ¯]^{0} mesons to the final states J/ψ(→μ^{+}μ^{-})K_{S}^{0}, ψ(2S)(→μ^{+}μ^{-})K_{S}^{0} and J/ψ(→e^{+}e^{-})K_{S}^{0} with K_{S}^{0}→π^{+}π^{-} is presented. The data ... ...

Abstract	A measurement of time-dependent CP violation in the decays of B^{0} and B[over ¯]^{0} mesons to the final states J/ψ(→μ^{+}μ^{-})K_{S}^{0}, ψ(2S)(→μ^{+}μ^{-})K_{S}^{0} and J/ψ(→e^{+}e^{-})K_{S}^{0} with K_{S}^{0}→π^{+}π^{-} is presented. The data correspond to an integrated luminosity of 6 fb^{-1} collected at a center-of-mass energy of sqrt[s]=13 TeV with the LHCb detector. The CP-violation parameters are measured to be S_{ψK_{S}^{0}}=0.717±0.013(stat)±0.008(syst) and C_{ψK_{S}^{0}}=0.008±0.012(stat)±0.003(syst). This measurement of S_{ψK_{S}^{0}} represents the most precise single measurement of the CKM angle β to date and is more precise than the current world average. In addition, measurements of the CP-violation parameters of the individual channels are reported and a combination with the LHCb Run 1 measurements is performed.
Language	English
Publishing date	2024-01-26
Publishing country	United States
Document type	Journal Article
ZDB-ID	208853-8
ISSN	1079-7114 ; 0031-9007
ISSN (online)	1079-7114
ISSN	0031-9007
DOI	10.1103/PhysRevLett.132.021801
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article ; Online: Improved Measurement of CP Violation Parameters in B_{s}^{0}→J/ψK^{+}K^{-} Decays in the Vicinity of the ϕ(1020) Resonance.

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Physical review letters

2024 Volume 132, Issue 5, Page(s) 51802

Abstract: The decay-time-dependent CP asymmetry in B_{s}^{0}→J/ψ(→μ^{+}μ^{-})K^{+}K^{-} decays is measured using proton-proton collision data, corresponding to an integrated luminosity of 6 fb^{-1}, collected with the LHCb detector at a center-of-mass energy of ... ...

Abstract	The decay-time-dependent CP asymmetry in B_{s}^{0}→J/ψ(→μ^{+}μ^{-})K^{+}K^{-} decays is measured using proton-proton collision data, corresponding to an integrated luminosity of 6 fb^{-1}, collected with the LHCb detector at a center-of-mass energy of 13 TeV. Using a sample of approximately 349 000 B_{s}^{0} signal decays with an invariant K^{+}K^{-} mass in the vicinity of the ϕ(1020) resonance, the CP-violating phase ϕ_{s} is measured, along with the difference in decay widths of the light and heavy mass eigenstates of the B_{s}^{0}-B[over ¯]_{s}^{0} system, ΔΓ_{s}, and the difference of the average B_{s}^{0} and B^{0} meson decay widths, Γ_{s}-Γ_{d}. The values obtained are ϕ_{s}=-0.039±0.022±0.006 rad, ΔΓ_{s}=0.0845±0.0044±0.0024 ps^{-1}, and Γ_{s}-Γ_{d}=-0.0056_{-0.0015}^{+0.0013}±0.0014 ps^{-1}, where the first uncertainty is statistical and the second systematic. These are the most precise single measurements to date and are consistent with expectations based on the Standard Model and with the previous LHCb analyses of this decay. These results are combined with previous independent LHCb measurements. The phase ϕ_{s} is also measured independently for each polarization state of the K^{+}K^{-} system and shows no evidence for polarization dependence.
Language	English
Publishing date	2024-02-16
Publishing country	United States
Document type	Journal Article
ZDB-ID	208853-8
ISSN	1079-7114 ; 0031-9007
ISSN (online)	1079-7114
ISSN	0031-9007
DOI	10.1103/PhysRevLett.132.051802
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article ; Online: Observation of Cabibbo-Suppressed Two-Body Hadronic Decays and Precision Mass Measurement of the Ω_{c}^{0} Baryon.

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Physical review letters

2024 Volume 132, Issue 8, Page(s) 81802

Abstract: The first observation of the singly Cabibbo-suppressed Ω_{c}^{0}→Ω^{-}K^{+} and Ω_{c}^{0}→Ξ^{-}π^{+} decays is reported, using proton-proton collision data at a center-of-mass energy of 13 TeV, corresponding to an integrated luminosity of 5.4 fb^{-1}, ... ...

Abstract	The first observation of the singly Cabibbo-suppressed Ω_{c}^{0}→Ω^{-}K^{+} and Ω_{c}^{0}→Ξ^{-}π^{+} decays is reported, using proton-proton collision data at a center-of-mass energy of 13 TeV, corresponding to an integrated luminosity of 5.4 fb^{-1}, collected with the LHCb detector between 2016 and 2018. The branching fraction ratios are measured to be B(Ω_{c}^{0}→Ω^{-}K^{+})/B(Ω_{c}^{0}→Ω^{-}π^{+})=[6.08±0.51(stat)±0.40(syst)]%,B(Ω_{c}^{0}→Ξ^{-}π^{+})/B(Ω_{c}^{0}→Ω^{-}π^{+})=[15.81±0.87(stat)±0.44(syst)±0.16(ext)]%. In addition, using the Ω_{c}^{0}→Ω^{-}π^{+} decay channel, the Ω_{c}^{0} baryon mass is measured to be M(Ω_{c}^{0})=2695.28±0.07(stat)±0.27(syst)±0.30(ext) MeV, improving the precision of the previous world average by a factor of 4.
Language	English
Publishing date	2024-02-28
Publishing country	United States
Document type	Journal Article
ZDB-ID	208853-8
ISSN	1079-7114 ; 0031-9007
ISSN (online)	1079-7114
ISSN	0031-9007
DOI	10.1103/PhysRevLett.132.081802
Database	MEDical Literature Analysis and Retrieval System OnLINE

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