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  1. AU="Alger, Bradley E"
  2. AU="Frederik Boe Hüttel"
  3. AU="Dhar, Sandipan"
  4. AU=Pan Tianhong
  5. AU="Horvat, Elena"
  6. AU=Alany Raid G
  7. AU="Jiang, Cui"
  8. AU="Suresh, Vinod"
  9. AU="Monferrer, Dominique"
  10. AU="John Tam"
  11. AU="Yi, Rong"
  12. AU="Bhandary, R P"
  13. AU="Merkebu, Jerusalem"
  14. AU="Han, Junhyek"
  15. AU="Muneoka, Yusuke"
  16. AU="Griggs, Lisa"
  17. AU="Klauck, Sabine M"
  18. AU="Turton, James A"
  19. AU="Patel, Abhijit A"
  20. AU="Shankowsky, Heather A"
  21. AU="Płóciennik, Przemysław"
  22. AU="Marchesi, Pietro"
  23. AU="Kim Je Hyoung"
  24. AU="Huber, Ingrid"
  25. AU="Hasuko, K."
  26. AU="Yao, Weigen"
  27. AU="Huang, Xiao-Fan"
  28. AU=Zuo Chuantian
  29. AU="Varchetta, Veronica"
  30. AU="Zhang, Lingye"
  31. AU="Venko, Katja"
  32. AU="Kasthuri, Thirupathi"
  33. AU="Pirtskhalava, Tamar"
  34. AU="Saridakis, E N"
  35. AU="Vithana, Eranga N"
  36. AU="Suárez-Lledó, M"
  37. AU="Olivo-Marston, Susan"
  38. AU="Denise P Momesso"
  39. AU="Obrecht-Sturm, Denise"

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Treffer 1 - 10 von insgesamt 49

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  1. Artikel ; Online: Neuroscience Needs to Test Both Statistical and Scientific Hypotheses.

    Alger, Bradley E

    The Journal of neuroscience : the official journal of the Society for Neuroscience

    2022  Band 42, Heft 45, Seite(n) 8432–8438

    Abstract: Experimental neuroscience typically uses " ...

    Abstract Experimental neuroscience typically uses "
    Mesh-Begriff(e) Humans ; Data Interpretation, Statistical ; Research Design ; Neurosciences
    Sprache Englisch
    Erscheinungsdatum 2022-11-09
    Erscheinungsland United States
    Dokumenttyp Journal Article
    ZDB-ID 604637-x
    ISSN 1529-2401 ; 0270-6474
    ISSN (online) 1529-2401
    ISSN 0270-6474
    DOI 10.1523/JNEUROSCI.1134-22.2022
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  2. Artikel ; Online: Scientific Hypothesis-Testing Strengthens Neuroscience Research.

    Alger, Bradley E

    eNeuro

    2020  Band 7, Heft 4

    Abstract: Science needs to understand the strength of its findings. This essay considers the evaluation of studies that test scientific (not statistical) hypotheses. A scientific hypothesis is a putative explanation for an observation or phenomenon; it makes (or " ... ...

    Abstract Science needs to understand the strength of its findings. This essay considers the evaluation of studies that test scientific (not statistical) hypotheses. A scientific hypothesis is a putative explanation for an observation or phenomenon; it makes (or "entails") testable predictions that must be true if the hypothesis is true and that lead to its rejection if they are false. The question is, "how should we judge the strength of a hypothesis that passes a series of experimental tests?" This question is especially relevant in view of the "reproducibility crisis" that is the cause of great unease. Reproducibility is said to be a dire problem because major neuroscience conclusions supposedly rest entirely on the outcomes of single,
    Mesh-Begriff(e) Mental Processes ; Neurosciences ; Reproducibility of Results ; Research Design
    Sprache Englisch
    Erscheinungsdatum 2020-07-23
    Erscheinungsland United States
    Dokumenttyp Journal Article
    ZDB-ID 2800598-3
    ISSN 2373-2822 ; 2373-2822
    ISSN (online) 2373-2822
    ISSN 2373-2822
    DOI 10.1523/ENEURO.0357-19.2020
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  3. Artikel: David Casarett's Stoned: A Doctor's Case for Medical Marijuana.

    Alger, Bradley E

    Cerebrum : the Dana forum on brain science

    2016  Band 2016

    Abstract: With legal cannabis sales at $5.4 billion in 2015 and expected to rise by another billion this year in the United States, legalization and marijuana's impact on health is a hot topic of national debate. Casarett, a physician at the University of ... ...

    Abstract With legal cannabis sales at $5.4 billion in 2015 and expected to rise by another billion this year in the United States, legalization and marijuana's impact on health is a hot topic of national debate. Casarett, a physician at the University of Pennsylvania, immerses himself in the culture, science, and smoke of medical marijuana in order to sort out the truth behind the buzz. Our reviewer, who has authored more than 120 research papers and reviews on the regulation of synaptic inhibition and endocannabinoids, tell us what the author got right, but also overlooked on his journey to learn more about a complex and controversial subject.
    Sprache Englisch
    Erscheinungsdatum 2016-03
    Erscheinungsland United States
    Dokumenttyp Journal Article
    ZDB-ID 2251230-5
    ISSN 1524-6205
    ISSN 1524-6205
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  4. Artikel: Seizing an opportunity for the endocannabinoid system.

    Alger, Bradley E

    Epilepsy currents

    2014  Band 14, Heft 5, Seite(n) 272–276

    Abstract: Exogenous cannabinoids can limit seizures and neurodegeneration, and their actions are largely mimicked by endogenous cannabinoids (endocannabinoids). Endocannabinoids are mobilized by epileptiform activity and in turn influence this activity by ... ...

    Abstract Exogenous cannabinoids can limit seizures and neurodegeneration, and their actions are largely mimicked by endogenous cannabinoids (endocannabinoids). Endocannabinoids are mobilized by epileptiform activity and in turn influence this activity by inhibiting synaptic transmission; both excitatory and some inhibitory synapses can be suppressed, leading to potentially complex outcomes. Moreover, the endocannabinoid system is not a fixed entity, and its strength can be enhanced or reduced. Endocannabinoids and their receptors are altered by epileptic seizures in ways that can reduce the efficacy of both exogenous and endogenous cannabinoids in sometimes unexpected ways.
    Sprache Englisch
    Erscheinungsdatum 2014-09
    Erscheinungsland United States
    Dokumenttyp Journal Article
    ZDB-ID 2270080-8
    ISSN 1535-7597
    ISSN 1535-7597
    DOI 10.5698/1535-7597-14.5.272
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  5. Artikel: Getting high on the endocannabinoid system.

    Alger, Bradley E

    Cerebrum : the Dana forum on brain science

    2013  Band 2013, Seite(n) 14

    Abstract: The endogenous cannabinoid system-named for the plant that led to its discovery-is one of the most important physiologic systems involved in establishing and maintaining human health. Endocannabinoids and their receptors are found throughout the body: in ...

    Abstract The endogenous cannabinoid system-named for the plant that led to its discovery-is one of the most important physiologic systems involved in establishing and maintaining human health. Endocannabinoids and their receptors are found throughout the body: in the brain, organs, connective tissues, glands, and immune cells. With its complex actions in our immune system, nervous system, and virtually all of the body's organs, the endocannabinoids are literally a bridge between body and mind. By understanding this system, we begin to see a mechanism that could connect brain activity and states of physical health and disease.
    Sprache Englisch
    Erscheinungsdatum 2013-11-01
    Erscheinungsland United States
    Dokumenttyp Journal Article
    ZDB-ID 2251230-5
    ISSN 1524-6205
    ISSN 1524-6205
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  6. Artikel ; Online: Endocannabinoids at the synapse a decade after the dies mirabilis (29 March 2001): what we still do not know.

    Alger, Bradley E

    The Journal of physiology

    2012  Band 590, Heft 10, Seite(n) 2203–2212

    Abstract: Endogenous cannabinoids (endocannabinoids, eCBs) are ubiquitous regulators of synaptic transmission in the brain, mediating numerous forms of short- and long-term plasticity, and having strong influences on synapse formation and neurogenesis. Their roles ...

    Abstract Endogenous cannabinoids (endocannabinoids, eCBs) are ubiquitous regulators of synaptic transmission in the brain, mediating numerous forms of short- and long-term plasticity, and having strong influences on synapse formation and neurogenesis. Their roles as retrograde messengers that suppress both excitatory and inhibitory transmission are well-established. Yet, despite intensive investigation, many basic aspects of the eCB system are not understood. This brief review highlights recent advances, problems that remain unresolved, and avenues for future exploration. While 2-arachidonoylglycerol (2-AG) is probably the major eCB for intercellular CB1R-dependent signalling, anandamide (AEA) has come to the forefront in several novel contexts, both as a dual endovanilloid/endocannabinoid that regulates synaptic transmission acutely and as the source of a steady eCB tone in hippocampus. Complexities in the cellular processing of 2-AG are receiving renewed attention, as they are increasingly recognized as major determinants of how 2-AG affects cells. Long-standing fundamental issues such as the synthesis pathway for AEA and the molecular mechanism(s) underlying cellular uptake and release of eCBs remain problematical.
    Mesh-Begriff(e) Animals ; Arachidonic Acids/physiology ; Cannabinoid Receptor Modulators/physiology ; Endocannabinoids ; Glycerides/physiology ; Polyunsaturated Alkamides ; Synapses/physiology
    Chemische Substanzen Arachidonic Acids ; Cannabinoid Receptor Modulators ; Endocannabinoids ; Glycerides ; Polyunsaturated Alkamides ; glyceryl 2-arachidonate (8D239QDW64) ; anandamide (UR5G69TJKH)
    Sprache Englisch
    Erscheinungsdatum 2012-01-30
    Erscheinungsland England
    Dokumenttyp Journal Article ; Research Support, N.I.H., Extramural ; Review
    ZDB-ID 3115-x
    ISSN 1469-7793 ; 0022-3751
    ISSN (online) 1469-7793
    ISSN 0022-3751
    DOI 10.1113/jphysiol.2011.220855
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  7. Artikel ; Online: Homer protein-metabotropic glutamate receptor binding regulates endocannabinoid signaling and affects hyperexcitability in a mouse model of fragile X syndrome.

    Tang, Ai-Hui / Alger, Bradley E

    The Journal of neuroscience : the official journal of the Society for Neuroscience

    2015  Band 35, Heft 9, Seite(n) 3938–3945

    Abstract: The Fmr1 knock-out mouse model of fragile X syndrome (Fmr1(-/y)) has an epileptogenic phenotype that is triggered by group I metabotropic glutamate receptor (mGluR) activation. We found that a membrane-permeable peptide that disrupts mGluR5 interactions ... ...

    Abstract The Fmr1 knock-out mouse model of fragile X syndrome (Fmr1(-/y)) has an epileptogenic phenotype that is triggered by group I metabotropic glutamate receptor (mGluR) activation. We found that a membrane-permeable peptide that disrupts mGluR5 interactions with long-form Homers enhanced mGluR-induced epileptiform burst firing in wild-type (WT) animals, replicating the early stages of hyperexcitability in Fmr1(-/y). The peptide enhanced mGluR-evoked endocannabinoid (eCB)-mediated suppression of inhibitory synapses, decreased it at excitatory synapses in WTs, but had no effect on eCB actions in Fmr1(-/y). At a low concentration, the mGluR agonist did not generate eCBs at excitatory synapses but nevertheless induced burst firing in both Fmr1(-/y) and peptide-treated WT slices. This burst firing was suppressed by a cannabinoid receptor antagonist. We suggest that integrity of Homer scaffolds is essential for normal mGluR-eCB functioning and that aberrant eCB signaling resulting from disturbances of this molecular structure contributes to the epileptic phenotype of Fmr1(-/y).
    Mesh-Begriff(e) Animals ; Carrier Proteins/metabolism ; Endocannabinoids/metabolism ; Fragile X Mental Retardation Protein/genetics ; Fragile X Mental Retardation Protein/physiology ; Fragile X Syndrome/metabolism ; Hippocampus/metabolism ; Homer Scaffolding Proteins ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Protein Binding ; Receptor, Cannabinoid, CB1/antagonists & inhibitors ; Receptor, Metabotropic Glutamate 5/metabolism ; Signal Transduction/genetics ; Signal Transduction/physiology
    Chemische Substanzen Carrier Proteins ; Endocannabinoids ; Fmr1 protein, mouse ; Homer Scaffolding Proteins ; Receptor, Cannabinoid, CB1 ; Receptor, Metabotropic Glutamate 5 ; Fragile X Mental Retardation Protein (139135-51-6)
    Sprache Englisch
    Erscheinungsdatum 2015-03-04
    Erscheinungsland United States
    Dokumenttyp Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 604637-x
    ISSN 1529-2401 ; 0270-6474
    ISSN (online) 1529-2401
    ISSN 0270-6474
    DOI 10.1523/JNEUROSCI.4499-14.2015
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  8. Artikel: Endocannabinoid signaling in neural plasticity.

    Alger, Bradley E

    Current topics in behavioral neurosciences

    2009  Band 1, Seite(n) 141–172

    Abstract: Plasticity refers to a physiologically measured change that may last for short or long periods of time. Endocannabinoids (ECBs) are prevalent throughout most of the brain, and modulate synaptic transmission in many ways. This chapter will focus on the ... ...

    Abstract Plasticity refers to a physiologically measured change that may last for short or long periods of time. Endocannabinoids (ECBs) are prevalent throughout most of the brain, and modulate synaptic transmission in many ways. This chapter will focus on the roles of ECBs in neural plasticity in the mammalian brain. The topics covered can be divided loosely into two themes: how ECBs regulate synaptic plasticity, and how ECBs' actions themselves are regulated by neuronal activity. Because ECBs regulate synaptic plasticity, the modifiability of ECB mobilization constitutes a form of "metaplasticity" (as reported by Abraham and Bear (Trends Neurosci 19:126-130, 1996)), i.e., an upstream process that determines the nature and extent of synaptic plasticity. Many of their basic functions are still being discovered, and while there is consensus on large issues, many points of divergence exist as well. This chapter concentrates on developments in the roles of ECBs in synaptic plasticity that have come to light since the major review by Chevaleyre et al. (Annu Rev Neurosci 29:37-76, 2006).
    Mesh-Begriff(e) Animals ; Brain/cytology ; Cannabinoid Receptor Modulators/metabolism ; Cannabinoid Receptor Modulators/pharmacology ; Endocannabinoids ; Humans ; Neuronal Plasticity/drug effects ; Neuronal Plasticity/physiology ; Neurons/drug effects ; Neurons/physiology ; Receptors, Cannabinoid/physiology ; Signal Transduction/drug effects ; Signal Transduction/physiology
    Chemische Substanzen Cannabinoid Receptor Modulators ; Endocannabinoids ; Receptors, Cannabinoid
    Sprache Englisch
    Erscheinungsdatum 2009
    Erscheinungsland Germany
    Dokumenttyp Journal Article ; Review
    ISSN 1866-3370
    ISSN 1866-3370
    DOI 10.1007/978-3-540-88955-7_6
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  9. Artikel: Not too excited? Thank your endocannabinoids.

    Alger, Bradley E

    Neuron

    2006  Band 51, Heft 4, Seite(n) 393–395

    Abstract: Endocannabinoids can mediate neuroprotection, but it is not known how. In this issue of Neuron, Monory et al. use mutant mice and localized viral targeting to produce conditional knockouts of the cannabinoid CB1 receptor. They show that protection ... ...

    Abstract Endocannabinoids can mediate neuroprotection, but it is not known how. In this issue of Neuron, Monory et al. use mutant mice and localized viral targeting to produce conditional knockouts of the cannabinoid CB1 receptor. They show that protection against kainic acid-induced seizures and cell death is conferred by CB1Rs on hippocampal glutamatergic nerve terminals.
    Mesh-Begriff(e) Animals ; Cannabinoid Receptor Modulators/physiology ; Cell Death/drug effects ; Endocannabinoids ; Epilepsy/chemically induced ; Epilepsy/pathology ; Epilepsy/prevention & control ; Excitatory Amino Acid Agonists/toxicity ; Kainic Acid/toxicity ; Mice ; Mice, Knockout ; Neurons/drug effects ; Neurons/pathology ; Neuroprotective Agents ; Receptor, Cannabinoid, CB1/deficiency ; Receptor, Cannabinoid, CB1/genetics
    Chemische Substanzen Cannabinoid Receptor Modulators ; Endocannabinoids ; Excitatory Amino Acid Agonists ; Neuroprotective Agents ; Receptor, Cannabinoid, CB1 ; Kainic Acid (SIV03811UC)
    Sprache Englisch
    Erscheinungsdatum 2006-08-17
    Erscheinungsland United States
    Dokumenttyp Comment ; Journal Article
    ZDB-ID 808167-0
    ISSN 1097-4199 ; 0896-6273
    ISSN (online) 1097-4199
    ISSN 0896-6273
    DOI 10.1016/j.neuron.2006.08.003
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  10. Artikel ; Online: Do cannabinoids reduce brain power?

    Alger, Bradley E / Tang, Ai-Hui

    Nature neuroscience

    2012  Band 15, Heft 4, Seite(n) 499–501

    Mesh-Begriff(e) Animals ; Energy Metabolism/physiology ; Female ; Male ; Mitochondria/physiology ; Mitochondrial Membranes/physiology ; Neurons/metabolism ; Receptor, Cannabinoid, CB1/physiology
    Chemische Substanzen Receptor, Cannabinoid, CB1
    Sprache Englisch
    Erscheinungsdatum 2012-03-27
    Erscheinungsland United States
    Dokumenttyp News ; Research Support, N.I.H., Extramural ; Comment
    ZDB-ID 1420596-8
    ISSN 1546-1726 ; 1097-6256
    ISSN (online) 1546-1726
    ISSN 1097-6256
    DOI 10.1038/nn.3072
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