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  1. Book ; Online ; E-Book: Woher Kommt Gewalt?

    Bogerts, Bernhard

    Erklärungen Aus Neurowissenschaften, Psychologie, Soziologie and Co

    2021  

    Language German
    Size 1 online resource (283 pages)
    Publisher Springer Berlin / Heidelberg
    Publishing place Berlin, Heidelberg
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    ISBN 3-662-63338-8 ; 3-662-63337-X ; 978-3-662-63338-0 ; 978-3-662-63337-3
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  2. Book: Where does violence come from? A multidimensional approach to its causes and manifestations

    Bogerts, Bernhard

    2021  

    Abstract: Where does violence come from? How can people do such things? These are often the first questions that arise when we witness violence in the in the media or in real life. This book provides comprehensive answers by combining the explanatory approaches ... ...

    Title translation Woher kommt die Gewalt? Ein multidimensionaler Ansatz für ihre Ursachen und Erscheinungsformen (DeepL)
    Abstract Where does violence come from? How can people do such things? These are often the first questions that arise when we witness violence in the in the media or in real life. This book provides comprehensive answers by combining the explanatory approaches from criminology, sociology, psychology, psychiatry, brain research, genetics, pedagogy, historical sciences, and justice into a big, exciting, and comprehensible picture - in an entertaining way with current, state-of-the art science(s). Multiple case studies are presented that show us the frightening diversity of human violence: acts of violence by individual perpetrators; violence between groups; riots and tumults by gangs and hooligans; violent ethnic and religious conflicts; extreme violence in the form of amok and terror; and up to armed conflicts, pogroms, and genocide. Last but not least, the knowledge gained from this book can help answer another big question: how can violence be contained or even prevented? (c) The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Switzerland AG - Contents: (1) Manifestations of violence. (2) Incidence, frequency and consequences of violence. (3) Why ist he tendency to violence a human trait? (4) Heritability of aggressive behavior. (5) Neurobiology of violence. (6) Brain pathology in violent offenders. (7) The role of hormones and messenger substances in the brain. (8) Gender differences in the propensity for violence. (9) Mental disorders and violence. (10) Alcohol, drugs and violence. (11) Psychology of violence. (12) Violence as an end in itself and lust gain. (13) Social causes of violence. (14) Violence in children and adolescents: Early risk factors. (15) Rampage and school shooting. (16) Terror. (17) Collective violence, xenophobia, pogroms, genocide. (18) Sexual violence. (19) Religion and violence. (20) Conclusions for the prediction and prevention of violence. (c) ZPID
    Keywords Aggressionsverhalten ; Aggressive Behavior ; Cognitive Neuroscience ; Criminal Behavior ; Gewalt ; Gewaltverbrechen ; Kognitive Neurowissenschaft ; Kriminelles Verhalten ; Neurobiologie ; Neurobiology ; Neuropsychologie ; Neuropsychology ; Personality ; Persönlichkeit ; Social Influences ; Soziale Einflüsse ; Theorien ; Theories ; Violence ; Violent Crime
    Language English
    Size 222 pp.
    Publisher Springer International Publishing
    Publishing place Cham
    Document type Book
    ISBN 978-3-030-81792-3 ; 3-030-81792-X
    DOI 10.1007/978-3-030-81792-3
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  3. Book: Woher kommt Gewalt? Von Neurowissenschaft bis Soziologie - Eine mehrdimensionale Betrachtung

    Bogerts, Bernhard

    2021  

    Abstract: Warum tun Menschen so etwas!? Dies ist oft die erste Frage, die sich aufdrängt, wenn wir in den Medien oder im echten Leben Zeugen von Gewalt werden. Dieses Buch gibt umfassende Antworten: Es erklärt die Ursachen von Gewalt nicht aus der eingeschränkten ... ...

    Title translation Where does violence come from?. From neuroscience to sociology - A multidimensional view (DeepL)
    Abstract Warum tun Menschen so etwas!? Dies ist oft die erste Frage, die sich aufdrängt, wenn wir in den Medien oder im echten Leben Zeugen von Gewalt werden. Dieses Buch gibt umfassende Antworten: Es erklärt die Ursachen von Gewalt nicht aus der eingeschränkten Sicht einer einzelnen Fachdisziplin, sondern verbindet die Erklärungsansätze aus Hirnforschung, Kriminologie, Soziologie, Psychologie, Psychiatrie, Genetik, Pädagogik, Geschichtswissenschaften und Justiz zu einem großen, spannenden und verständlichen Bild - auf unterhaltsame Weise und auf dem aktuellen Stand der Wissenschaft(en). Und immer nah an Fallbeispielen, die uns die erschreckende Vielfalt menschlicher Gewalt vor Augen führen: Gewalthandlungen einzelner Täter, Gewalt zwischen Gruppen, Randale und Krawalle durch Gangs und Hooligans, gewaltsame ethnische und religiöse Konflikte, Extremgewalt in Form von Amok und Terror bis hin zu kriegerischen Auseinandersetzungen und Völkermord. Nicht zuletzt kann das Wissen aus diesem Buch dazu beitragen, eine andere große Frage zu beantworten: Wie kann Gewalt eingedämmt oder gar verhindert werden? (c) Der/die Herausgeber bzw. der/die Autor(en), exklusiv lizenziert durch Springer-Verlag GmbH - Inhalt: (1) Formen von Gewalt. (2) Vorkommen, Häufigkeit und Folgen von Gewalt. (3) Warum gehört Gewaltneigung zu den menschlichen Eigenschaften? (4) Erblichkeit aggressiven Verhaltens. (5) Neurobiologie der Gewalt. (6) Hirnstruktur und Hirnfunktion von Gewalttätern. (7) Bedeutung von Hormonen und Botenstoffen des Gehirns. (8) Geschlechterdifferenz der Gewaltneigung. (9) Psychische Störungen und Gewaltneigung. (10) Alkohol, Drogen und Gewalt. (11) Psychologie der Gewalt. (12) Gewalt als Selbstzweck und Lustgewinn. (13) Soziale Ursachen von Gewalt. (14) Gewalt bei Kindern und Jugendlichen - frühe Risikofaktoren. (15) Amok und School Shooting. (16) Terror. (17) Kollektive Gewalt, Fremdenfeindlichkeit, Pogrome, Völkermord. (18) Sexuelle Gewalt. (19) Religion und Gewalt. (20) Schlussfolgerungen für die Vorhersage und Prävention von Gewalt. (c) ZPID
    Keywords Epidemiologie ; Epidemiology ; Etiology ; Genetics ; Genetik ; Gewalt ; Gewaltprävention ; Neuro-Wissenschaften ; Neurosciences ; Psychologie ; Psychology ; Sociology ; Soziologie ; Theorien ; Theories ; Violence ; Violence Prevention ; Ätiologie
    Language German
    Size 293 S.
    Publisher Springer
    Publishing place Berlin, Heidelberg
    Document type Book
    ISBN 978-3-662-63338-0 ; 3-662-63338-8
    DOI 10.1007/978-3-662-63338-0
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  4. Book ; Conference proceedings: Mehrdimensionale Psychiatrie

    Bogerts, Bernhard / Wiedemann, Georg

    [Tagung ... 4. und 5. November 1994 in Tübung]

    1997  

    Author's details hrsg. von Georg Wiedemann ... Unter Mitarb. von Bernhard Bogerts
    Keywords Psychotherapy ; Psychotherapie
    Subject Behandlung ; Psychologische Behandlung
    Language German
    Size XIII, 142 S. : Ill., graph. Darst.
    Edition 1. Aufl.
    Publisher Fischer
    Publishing place Stuttgart u.a.
    Publishing country Germany
    Document type Book ; Conference proceedings
    HBZ-ID HT007402501
    ISBN 3-437-21118-8 ; 978-3-437-21118-8
    Database Catalogue ZB MED Medicine, Health

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    Shelf markLoan statusLocation
    1998 A 5537 order for loan Magazin

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  5. Book: Die Hirnstruktur Schizophrener und ihre Bedeutung für die Pathophysiologie und Psychopathologie der Erkrankung

    Bogerts, Bernhard

    18 Tabellen

    (Sammlung psychiatrischer und neurologischer Einzeldarstellungen)

    1990  

    Series title Sammlung psychiatrischer und neurologischer Einzeldarstellungen
    Keywords Brain / pathology ; Schizophrenia / physiopathology ; Schizophrenia / pathology ; Schizophrenie ; Gehirn ; Pathophysiologie ; Psychopathologie
    Subject Cerebrum ; Hirn ; Encephalon ; Enzephalon ; Hirngewebe ; Hirnmasse ; Gehirnmasse ; Spaltungsirresein ; Schizophrene Psychose ; Pathopsychologie ; Pathologische Physiologie ; Physiologische Pathologie ; Physiopathologie
    Size 66 S. : Ill., graph. Darst.
    Edition 1. Aufl.
    Publisher Thieme
    Publishing place Stuttgart u.a.
    Document type Book
    HBZ-ID HT003508920
    ISBN 3-13-735801-9 ; 978-3-13-735801-5
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    1990 A 4751 order for loan Magazin

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  6. Article ; Online: Neuroanatomical correlates of aggressiveness: a case-control voxel- and surface-based morphometric study.

    Seidenbecher, Stephanie / Schöne, Maria / Kaufmann, Jörn / Schiltz, Kolja / Bogerts, Bernhard / Frodl, Thomas

    Brain structure & function

    2023  Volume 229, Issue 1, Page(s) 31–46

    Abstract: Aggression occurs across the population ranging on a symptom continuum. Most previous studies have used magnetic resonance imaging in clinical/forensic samples, which is associated with several confounding factors. The present study examined structural ... ...

    Abstract Aggression occurs across the population ranging on a symptom continuum. Most previous studies have used magnetic resonance imaging in clinical/forensic samples, which is associated with several confounding factors. The present study examined structural brain characteristics in two healthy samples differing only in their propensity for aggressive behavior. Voxel- and surface-based morphometry (SBM) analyses were performed on 29 male martial artists and 32 age-matched male controls. Martial artists had significantly increased mean gray matter volume in two frontal (left superior frontal gyrus and bilateral anterior cingulate cortex) and one parietal (bilateral posterior cingulate gyrus and precuneus) brain clusters compared to controls (whole brain: p < 0.001, cluster level: family-wise error (FWE)-corrected). SBM analyses revealed a trend for greater gyrification indices in martial artists compared to controls in the left lateral orbital frontal cortex and the left pars orbitalis (whole brain: p < 0.001, cluster level: FWE-corrected). The results indicate brain structural differences between martial artists and controls in frontal and parietal brain areas critical for emotion processing/inhibition of emotions as well as empathic processes. The present study highlights the importance of studying healthy subjects with a propensity for aggressive behavior in future structural MRI research on aggression.
    MeSH term(s) Humans ; Male ; Aggression/psychology ; Brain/diagnostic imaging ; Brain/pathology ; Gray Matter/diagnostic imaging ; Gray Matter/pathology ; Cerebral Cortex/pathology ; Case-Control Studies ; Magnetic Resonance Imaging/methods
    Language English
    Publishing date 2023-10-11
    Publishing country Germany
    Document type Journal Article
    ZDB-ID 2273162-3
    ISSN 1863-2661 ; 1863-2653
    ISSN (online) 1863-2661
    ISSN 1863-2653
    DOI 10.1007/s00429-023-02715-x
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  7. Article ; Online: Altered expression of costimulatory molecules in dementias.

    Busse, Stefan / von Hoff, Franz / Michler, Enrico / Hartig, Roland / Bogerts, Bernhard / Busse, Mandy

    European archives of psychiatry and clinical neuroscience

    2021  Volume 272, Issue 5, Page(s) 807–815

    Abstract: Although the expression of co-stimulatory molecules plays an important role in the immune system, only little is known about their regulation in dementias. Therefore, we determined the expression of CD28, ICOS (CD278) and CTLA-4 (CD152) by CD4 + and CD8 + ...

    Abstract Although the expression of co-stimulatory molecules plays an important role in the immune system, only little is known about their regulation in dementias. Therefore, we determined the expression of CD28, ICOS (CD278) and CTLA-4 (CD152) by CD4 + and CD8 + T cells in the peripheral blood of patients with mild cognitive impairment (MCI; N = 19), Alzheimer's disease (AD; N = 51), vascular dementia (VD; N = 21) and frontotemporal dementia (FTD; N = 6) at the point in time of diagnosis compared to 19 non-demented elderly persons. The expression of CD28 and ICOS by CD4 + and CD8 + T cells was not changed in AD, FTD or VD patients. The expression of the negative regulator CTLA-4 was increased by CD4 + T cells from AD and FTD patients and by CD8 + T cells from VD patients. The classification of the AD patients according to the severity of the disorder showed stage-dependent alterations of CD28, ICOS and CTLA-4 expression. In AD patients, the correlation analysis showed an association between the decline in CD28 + T cells and the increase in CTLA-4 + T cells with cognitive decline, measured by the mini-mental state examination (MMSE), tau proteins and Amyloid-β, important AD biomarkers in cerebrospinal fluid (CSF). In FTD patients, a positive association between Q Albumin, a marker for blood-CSF-barrier function, and CD28 and a negative correlation between Q Albumin and ICOS expression were determined. Our data suggest a dysregulated balance between the expression of negative and positive co-stimulatory molecules by T cells in AD patients, which might contribute to chronic inflammation observed in dementia.
    MeSH term(s) Aged ; Albumins ; Alzheimer Disease/diagnosis ; Amyloid beta-Peptides/cerebrospinal fluid ; Biomarkers/cerebrospinal fluid ; CD28 Antigens ; CTLA-4 Antigen ; Frontotemporal Dementia/cerebrospinal fluid ; Frontotemporal Dementia/diagnosis ; Humans ; Peptide Fragments/cerebrospinal fluid ; tau Proteins/cerebrospinal fluid
    Chemical Substances Albumins ; Amyloid beta-Peptides ; Biomarkers ; CD28 Antigens ; CTLA-4 Antigen ; Peptide Fragments ; tau Proteins
    Language English
    Publishing date 2021-08-24
    Publishing country Germany
    Document type Journal Article
    ZDB-ID 1045583-8
    ISSN 1433-8491 ; 0175-758X ; 0940-1334
    ISSN (online) 1433-8491
    ISSN 0175-758X ; 0940-1334
    DOI 10.1007/s00406-021-01297-1
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  8. Article ; Online: N-Methyl-d-aspartate receptor autoantibodies in schizophrenia and affective disorders.

    Steiner, Johann / Bogerts, Bernhard

    Schizophrenia research

    2015  Volume 162, Issue 1-3, Page(s) 291

    MeSH term(s) Autoantibodies/blood ; Bipolar Disorder/immunology ; Depressive Disorder, Major/blood ; Humans ; Psychotic Disorders/immunology ; Receptors, N-Methyl-D-Aspartate/immunology ; Schizophrenia/blood
    Chemical Substances Autoantibodies ; Receptors, N-Methyl-D-Aspartate
    Language English
    Publishing date 2015-03
    Publishing country Netherlands
    Document type Comment ; Letter
    ZDB-ID 639422-x
    ISSN 1573-2509 ; 0920-9964
    ISSN (online) 1573-2509
    ISSN 0920-9964
    DOI 10.1016/j.schres.2015.01.009
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    Zs.MO 543: Show issues

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  9. Article: VGF expression by monocytes in patients with Alzheimer's disease and vascular dementia

    Busse, Stefan / Meyer, Eva / Dobrowolny, Henrik / Mawrin, Christian / Hartig, Roland / Bogerts, Bernhard / Busse, Mandy

    GeroPsych

    2022  Volume 35, Issue 3, Page(s) 149–155

    Abstract: Because its secretion is changed in cerebrospinal fluid and peripheral blood, the neuronal polypeptide VGF (nonacronymic) has been discussed as a biomarker for neuropsychiatric disorders. We have shown an enhanced VGF expression by T-cells from Alzheimer' ...

    Title translation VGF-Expression durch Monozyten bei Patienten mit Alzheimer-Krankheit und vaskulärer Demenz
    Abstract Because its secretion is changed in cerebrospinal fluid and peripheral blood, the neuronal polypeptide VGF (nonacronymic) has been discussed as a biomarker for neuropsychiatric disorders. We have shown an enhanced VGF expression by T-cells from Alzheimer's disease (AD) patients. In this study, we investigated the VGF expression by peripheral monocytes in 38 AD patients, 5 patients with vascular dementia (VD), and 20 neuropsychiatrically healthy individuals using flow cytometry. We determined an enhanced number of VGF-expressing monocytes in VD patients compared to AD patients. VGF+CD14+ monocytes were not correlated with age, body mass index, Mini-Mental State Examination (MMSE), or Q albumin. These preliminary data support findings indicating that VGF might play a role as a peripheral biomarker in VD.
    Keywords Aging ; Altern ; Alzheimer's Disease ; Alzheimersche Krankheit ; Biological Markers ; Biologische Marker ; Brain ; Gehirn ; Leucocytes ; Leukozyten ; Neuropeptide ; Neuropeptides ; Vascular Dementia ; Vaskuläre Demenz
    Language English
    Document type Article
    ZDB-ID 2536939-8
    ISSN 1662-971X ; 1662-9647
    ISSN (online) 1662-971X
    ISSN 1662-9647
    DOI 10.1024/1662-9647/a000263
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  10. Article ; Online: Increased densities of T and B lymphocytes indicate neuroinflammation in subgroups of schizophrenia and mood disorder patients.

    Schlaaff, Konstantin / Dobrowolny, Henrik / Frodl, Thomas / Mawrin, Christian / Gos, Tomasz / Steiner, Johann / Bogerts, Bernhard

    Brain, behavior, and immunity

    2020  Volume 88, Page(s) 497–506

    Abstract: An increasing number of clinical, epidemiological and genetic studies as well as investigations of CSF and blood suggests that neuroinflammation plays an essential role in the etiology of schizophrenia and mood disorders. However, direct ... ...

    Abstract An increasing number of clinical, epidemiological and genetic studies as well as investigations of CSF and blood suggests that neuroinflammation plays an essential role in the etiology of schizophrenia and mood disorders. However, direct neuropathological evidence of inflammation within the brain tissue remains sparse and the regional distribution of lymphocytes as surrogate markers of blood-brain barrier (BBB) impairment has not yet been investigated in this context. Densities of T and B lymphocytes were assessed in coronal whole brain sections of 22 patients with schizophrenia and 20 patients suffering from major depression or bipolar disorder, compared to 20 individuals without neuropsychiatric disorders from the Magdeburg Brain Collection. Cell densities were determined by immunohistochemical staining (anti-CD3 for T cells, anti-CD20 for B cells), followed by automated microscopic image acquisition and analysis. Hierarchical clustering and detailed cluster analysis were performed to detect possible subgroups of patients. Regional distribution was assessed by analysis of color coded mappings based on microsopic scans. Elevated lymphocyte density was found in 7 out of 20 mood disorder patients (adj. p = 0.022; Fisher's exact test, FET), 9 out of 22 schizophrenic patients (adj. p = 0.014; FET) and in 1 of 20 controls (p < 0.005; FET). Several cases showed different patterns of infiltration affecting cortical regions or subcortical white matter, while some presented diffuse infiltration. In two thirds of patients, no increased lymphocyte density could be found. The current findings indicate that lymphocyte infiltration occurs in a greater proportion of schizophrenia and mood disorder patients as compared to healthy controls. Under healthy conditions lymphocytes rarely cross the BBB. Thus, higher densities are considered indicators of neuroinflammation associated with an impairment of the BBB.
    MeSH term(s) B-Lymphocytes ; Bipolar Disorder ; Brain ; Humans ; Mood Disorders ; Schizophrenia ; T-Lymphocytes
    Language English
    Publishing date 2020-04-10
    Publishing country Netherlands
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 639219-2
    ISSN 1090-2139 ; 0889-1591
    ISSN (online) 1090-2139
    ISSN 0889-1591
    DOI 10.1016/j.bbi.2020.04.021
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