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  1. Article ; Online: Reference values of 25-hydroxyvitamin D revisited: a position statement from the Brazilian Society of Endocrinology and Metabolism (SBEM) and the Brazilian Society of Clinical Pathology/Laboratory Medicine (SBPC).

    Moreira, Carolina Aguiar / Ferreira, Carlos Eduardo Dos S / Madeira, Miguel / Silva, Barbara Campolina Carvalho / Maeda, Sergio Setsuo / Batista, Marcelo Cidade / Bandeira, Francisco / Borba, Victória Z Cochenski / Lazaretti-Castro, Marise

    Archives of endocrinology and metabolism

    2020  Volume 64, Issue 4, Page(s) 462–478

    Abstract: Hypovitaminosis D is a common condition with a negative impact on health. This statement, prepared by experts from the Brazilian Society of Endocrinology and Metabolism and the Brazilian Society of Clinical Pathology/Laboratory Medicine, includes ... ...

    Abstract Hypovitaminosis D is a common condition with a negative impact on health. This statement, prepared by experts from the Brazilian Society of Endocrinology and Metabolism and the Brazilian Society of Clinical Pathology/Laboratory Medicine, includes methodological aspects and limitations of the measurement of 25-hydroxyvitamin D [25(OH)D] for identification of vitamin D status, and identifies individuals at increased risk for deficiency of this vitamin in whom 25(OH)D measurement is recommended. For the general population, 25(OH)D levels between 20 and 60 ng/mL are considered normal, while individuals with levels below 20 ng/mL are considered to be vitamin D deficient. This statement identifies potential benefits of maintaining 25(OH)D levels > 30 ng/mL in specific conditions, including patients aged > 65 years or pregnant, those with recurrent falls, fragility fractures, osteoporosis, secondary hyperparathyroidism, chronic kidney disease, or cancer, and individuals using drugs with the potential to affect the vitamin D metabolism. This statement also calls attention to the risk of vitamin D intoxication, a life-threatening condition that occurs at 25(OH)D levels above 100 ng/mL.
    MeSH term(s) Aged ; Brazil ; Humans ; Pathology, Clinical ; Reference Values ; Vitamin D/analogs & derivatives ; Vitamin D Deficiency
    Chemical Substances Vitamin D (1406-16-2) ; 25-hydroxyvitamin D (A288AR3C9H)
    Language English
    Publishing date 2020-06-05
    Publishing country Brazil
    Document type Journal Article
    ISSN 2359-4292
    ISSN (online) 2359-4292
    DOI 10.20945/2359-3997000000258
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  2. Article ; Online: The use of PTH in the treatment of osteoporosis.

    Borba, Victória Z Cochenski / Mañas, Nádila Cecyn Pietszkowski

    Arquivos brasileiros de endocrinologia e metabologia

    2010  Volume 54, Issue 2, Page(s) 213–219

    Abstract: Anabolic drugs have recently widened therapeutic options in osteoporosis treatment, as they influence processes associated with bone formation to a greater extent and earlier than bone reabsortion. They positively affect a number of skeletal properties ... ...

    Abstract Anabolic drugs have recently widened therapeutic options in osteoporosis treatment, as they influence processes associated with bone formation to a greater extent and earlier than bone reabsortion. They positively affect a number of skeletal properties besides bone density, as intermittent administration of parathyroid hormone (PTH) results in an increase in the number and activity of osteoblasts leading to an increase in bone mass and improvement in skeletal architecture at both the trabecular and cortical bone. Human recombinant parathyroid hormone (hrPTH 1-84) and human recombinant PTH peptide 1-34 (teriparatide) belong to this group. The objective of this paper is to review PTH actions, benefits and adverse effects, action on biochemical markers, combination therapy with antiresorptive agents, impact of antiresorptive therapy prior to anabolic treatment, sequential treatment, and effect on glucocorticoid-induced osteoporosis.
    MeSH term(s) Anabolic Agents/adverse effects ; Anabolic Agents/therapeutic use ; Biomarkers/metabolism ; Bone Density/drug effects ; Bone Density Conservation Agents/adverse effects ; Bone Density Conservation Agents/therapeutic use ; Bone Resorption/metabolism ; Humans ; Lumbar Vertebrae/drug effects ; Osteoporosis/drug therapy ; Parathyroid Hormone/adverse effects ; Parathyroid Hormone/therapeutic use ; Spinal Fractures/prevention & control ; Teriparatide/adverse effects ; Teriparatide/therapeutic use
    Chemical Substances Anabolic Agents ; Biomarkers ; Bone Density Conservation Agents ; Parathyroid Hormone ; Teriparatide (10T9CSU89I)
    Language English
    Publishing date 2010-05-19
    Publishing country Brazil
    Document type Journal Article ; Review
    ZDB-ID 603919-4
    ISSN 1677-9487 ; 0004-2730
    ISSN (online) 1677-9487
    ISSN 0004-2730
    DOI 10.1590/s0004-27302010000200018
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  3. Article ; Online: Osteoporosis after transplantation.

    Kulak, Carolina A Moreira / Borba, Victoria Z Cochenski / Kulak, Jaime / Custódio, Melani Ribeiro

    Current osteoporosis reports

    2011  Volume 10, Issue 1, Page(s) 48–55

    Abstract: Transplantation is an established therapy for end-stage diseases of kidney, lung, liver, and heart among others. Osteoporosis and fragility fractures are serious complications of organ transplantation, particularly in the first post-transplant year. Many ...

    Abstract Transplantation is an established therapy for end-stage diseases of kidney, lung, liver, and heart among others. Osteoporosis and fragility fractures are serious complications of organ transplantation, particularly in the first post-transplant year. Many factors contribute to the pathogenesis of osteoporosis following organ transplantation. This review addresses the mechanisms of bone loss that occurs both in the early and late post-transplant periods, including the contribution of the immunosuppressive agents as well as the specific features to bone loss after kidney, lung, liver, cardiac, and bone marrow transplantation. Prevention and treatment for osteoporosis in the transplant recipient are also discussed.
    MeSH term(s) Animals ; Bone Marrow Transplantation/adverse effects ; Glucocorticoids/adverse effects ; Glucocorticoids/therapeutic use ; Heart Transplantation/adverse effects ; Humans ; Hyperparathyroidism, Secondary/complications ; Immunosuppressive Agents/administration & dosage ; Kidney Transplantation/adverse effects ; Liver Transplantation/adverse effects ; Lung Transplantation/adverse effects ; Organ Transplantation/adverse effects ; Osteoporosis/complications ; Osteoporosis/etiology ; Osteoporotic Fractures/etiology ; Tacrolimus/pharmacology ; Vitamin D Deficiency/etiology
    Chemical Substances Glucocorticoids ; Immunosuppressive Agents ; Tacrolimus (WM0HAQ4WNM)
    Language English
    Publishing date 2011-12-14
    Publishing country United States
    Document type Journal Article ; Review
    ZDB-ID 2186581-4
    ISSN 1544-2241 ; 1544-1873
    ISSN (online) 1544-2241
    ISSN 1544-1873
    DOI 10.1007/s11914-011-0083-y
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article ; Online: Post-transplantation osteoporosis.

    Kulak, Carolina A Moreira / Borba, Victória Z Cochenski / Kulak Júnior, Jaime / Campos, Denise Jonhsson / Shane, Elizabeth

    Arquivos brasileiros de endocrinologia e metabologia

    2010  Volume 54, Issue 2, Page(s) 143–149

    Abstract: Transplantation is an established therapy for many hematologic disorders as well as for end-stage diseases of the kidney, lung, liver, heart among others. Osteoporosis and a high incidence of fragility fractures have emerged as a complication of organ ... ...

    Abstract Transplantation is an established therapy for many hematologic disorders as well as for end-stage diseases of the kidney, lung, liver, heart among others. Osteoporosis and a high incidence of fragility fractures have emerged as a complication of organ transplantation. Many factors contribute to the pathogenesis of osteoporosis following organ transplantation. In addition, most patients have some form of bone disease prior to transplantation, which is usually related to adverse effects of end-stage organ failure on the skeleton. This chapter reviews the mechanisms of bone loss that occur both in the early and late post-transplant periods including the contribution of immunosuppressive agents as well as the specific features of bone loss after kidney, lung, liver, cardiac and bone marrow transplantation. Prevention and treatment for osteoporosis in the transplant recipient will also be addressed.
    MeSH term(s) Bone Density ; Bone Marrow Transplantation/adverse effects ; Bone Resorption/metabolism ; Humans ; Osteoporosis/etiology ; Osteoporosis/prevention & control
    Language English
    Publishing date 2010-05-19
    Publishing country Brazil
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 603919-4
    ISSN 1677-9487 ; 0004-2730
    ISSN (online) 1677-9487
    ISSN 0004-2730
    DOI 10.1590/s0004-27302010000200009
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  5. Article ; Online: Níveis baixos de 25-hidroxivitamina D (25OHD) em pacientes com doença inflamatória intestinal e sua correlação com a densidade mineral óssea.

    Souza, Hevelyn Noemberg de / Lora, Fabiana Lígia / Kulak, Carolina A Moreira / Mañas, Nádila Cecyn Pietszkowski / Amarante, Heda M B / Borba, Victória Z Cochenski

    Arquivos brasileiros de endocrinologia e metabologia

    2008  Volume 52, Issue 4, Page(s) 684–691

    Abstract: Unlabelled: Patients with inflammatory bowel disease (IBD) are at risk of having vitamin D deficiency (25-OHD) and low bone mineral density (BMD).: Objectives: To measure 25OHD in a young group of IBD patients submitted to a clinical evaluation, ... ...

    Title translation Low levels of 25-hydroxyvitamin D (25OHD) in patients with inflammatory bowel disease and its correlation with bone mineral density.
    Abstract Unlabelled: Patients with inflammatory bowel disease (IBD) are at risk of having vitamin D deficiency (25-OHD) and low bone mineral density (BMD).
    Objectives: To measure 25OHD in a young group of IBD patients submitted to a clinical evaluation, routine biochemistry and BMD measurement (lumbar spine and proximal femur).
    Results: 39 Crohn disease (CD) and 37 ulcerative colitis (UC) patients had lower serum levels of 25OHD compared to the control group (CD p = 0.003; UC p < 0.001), and 48.5% of the UC patients were 25OHD deficient. Lumbar spine BMD was lower in patients than controls (CD p = 0.001; UC p = 0.008). In CD patients, serum levels of 25OHD were significantly correlated with total femur (r = 0.391; p = 0.027) and femoral neck (r = 0.384; p = 0.03) BMD.
    Conclusion: It was found lower levels of 25OHD and BMD in young IBD patients compared to normal controls, suggesting an important role of 25OHD deficiency in the pathogenesis of the IBD bone disease.
    MeSH term(s) Adult ; Bone Density ; Case-Control Studies ; Colitis, Ulcerative/blood ; Colitis, Ulcerative/complications ; Crohn Disease/blood ; Crohn Disease/complications ; Female ; Humans ; Male ; Vitamin D/analogs & derivatives ; Vitamin D/blood ; Vitamin D Deficiency/diagnosis ; Vitamin D Deficiency/etiology ; Vitamin D Deficiency/physiopathology
    Chemical Substances Vitamin D (1406-16-2) ; 25-hydroxyvitamin D (A288AR3C9H)
    Language Portuguese
    Publishing date 2008-06-11
    Publishing country Brazil
    Document type Journal Article
    ZDB-ID 603919-4
    ISSN 1677-9487 ; 0004-2730
    ISSN (online) 1677-9487
    ISSN 0004-2730
    DOI 10.1590/s0004-27302008000400015
    Database MEDical Literature Analysis and Retrieval System OnLINE

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