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  1. Article ; Online: Acute diarrhea in hospitalized horses.

    Chapman, Ann M

    The Veterinary clinics of North America. Equine practice

    2009  Volume 25, Issue 2, Page(s) 363–380

    Abstract: The development of diarrhea among hospitalized horses is a major concern for equine veterinary hospitals and referral centers. It is a potential complication of hospitalization for surgical or medical procedures and can contribute to the morbidity and ... ...

    Abstract The development of diarrhea among hospitalized horses is a major concern for equine veterinary hospitals and referral centers. It is a potential complication of hospitalization for surgical or medical procedures and can contribute to the morbidity and mortality of horses with gastrointestinal and non-gastrointestinal diseases. Unfortunately, it can be difficult to pinpoint the exact cause of acute diarrhea or colitis, and in most cases, the specific etiologic agent is presumptive or undetermined. This article discusses the major etiologic agents of diarrhea in hospitalized horses, considers factors that place hospitalized horses at special risk for diarrhea, and examines several infectious colitis outbreaks that have occurred at veterinary referral centers.
    MeSH term(s) Animals ; Anti-Inflammatory Agents, Non-Steroidal/adverse effects ; Diarrhea/complications ; Diarrhea/veterinary ; Diet/adverse effects ; Diet/veterinary ; Dietary Carbohydrates ; Enterocolitis, Pseudomembranous/veterinary ; Feces ; Horse Diseases/etiology ; Horses ; Salmonella Infections, Animal/diagnosis ; Salmonella Infections, Animal/etiology ; Surgical Procedures, Operative/adverse effects
    Chemical Substances Anti-Inflammatory Agents, Non-Steroidal ; Dietary Carbohydrates
    Language English
    Publishing date 2009-08
    Publishing country United States
    Document type Journal Article
    ZDB-ID 286049-1
    ISSN 1558-4224 ; 0749-0739
    ISSN (online) 1558-4224
    ISSN 0749-0739
    DOI 10.1016/j.cveq.2009.05.001
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Se 1109: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
    bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
    ab Jg. 2022: Lesesaal (EG)

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  2. Article: Effects of Epinephrine, Detomidine, and Butorphanol on Assessments of Insulin Sensitivity in Mares.

    Kerrigan, Lauren E / Thompson, Donald L / Chapman, Ann M / Oberhaus, Erin L

    Journal of equine veterinary science

    2019  Volume 85, Page(s) 102842

    Abstract: Sympathoadrenal stimulation may perturb results of endocrine tests performed on fractious horses. Sedation may be beneficial; however, perturbation of results may preclude useful information. Four experiments were designed to 1) determine the effects of ... ...

    Abstract Sympathoadrenal stimulation may perturb results of endocrine tests performed on fractious horses. Sedation may be beneficial; however, perturbation of results may preclude useful information. Four experiments were designed to 1) determine the effects of epinephrine on insulin response to glucose (IR2G), 2) assess the effects of detomidine (DET), alone or combined with butorphanol (DET/BUT), on IR2G and glucose response to insulin (GR2I), and 3) assess the effects of BUT alone on IR2G. In Experiment 1, mares were administered saline or epinephrine (5 μg/kg BW) immediately before infusion of glucose (100 mg/kg BW). Glucose stimulated (P < .05) insulin release in controls at 5 minutes that persisted through 30 minutes; insulin was suppressed (P < .05) by epinephrine from 5 to 15 minutes, rising gradually through 30 minutes. Experiments 2 (IR2G) and 3 (GR2I) were conducted as triplicated 3 × 3 Latin squares with the following treatments: saline (SAL), DET, and DET/BUT (all administered at .01 mg/kg BW). Glucose stimulated (P < .05) insulin release that persisted through 30 minutes in SAL mares; DET and DET/BUT severely suppressed (P < .0001) the IR2G. Sedation did not affect resting glucose and had inconsistent effects on the GR2I when mares were treated with 50 mIU/kg BW recombinant human insulin. Butorphanol had no effect on IR2G. In conclusion, adrenergic agonists severely suppress the IR2G and cannot be used for sedation for this test. The use of DET did not alter the GR2I, and therefore may be useful for conducting this test in fractious horses.
    MeSH term(s) Animals ; Butorphanol ; Cross-Over Studies ; Epinephrine ; Female ; Horse Diseases ; Horses ; Imidazoles ; Insulin Resistance
    Chemical Substances Imidazoles ; detomidine (7N8K34P2XH) ; Butorphanol (QV897JC36D) ; Epinephrine (YKH834O4BH)
    Language English
    Publishing date 2019-12-03
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 2102631-2
    ISSN 1542-7412 ; 0737-0806
    ISSN (online) 1542-7412
    ISSN 0737-0806
    DOI 10.1016/j.jevs.2019.102842
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  3. Article: Effects of Epinephrine, Detomidine, and Butorphanol on Assessments of Insulin Sensitivity in Mares

    Kerrigan, Lauren E / Thompson, Donald L / Chapman, Ann M / Oberhaus, Erin L

    Journal of equine veterinary science. 2020 Feb., v. 85

    2020  

    Abstract: Sympathoadrenal stimulation may perturb results of endocrine tests performed on fractious horses. Sedation may be beneficial; however, perturbation of results may preclude useful information. Four experiments were designed to 1) determine the effects of ... ...

    Abstract Sympathoadrenal stimulation may perturb results of endocrine tests performed on fractious horses. Sedation may be beneficial; however, perturbation of results may preclude useful information. Four experiments were designed to 1) determine the effects of epinephrine on insulin response to glucose (IR2G), 2) assess the effects of detomidine (DET), alone or combined with butorphanol (DET/BUT), on IR2G and glucose response to insulin (GR2I), and 3) assess the effects of BUT alone on IR2G. In Experiment 1, mares were administered saline or epinephrine (5 μg/kg BW) immediately before infusion of glucose (100 mg/kg BW). Glucose stimulated (P < .05) insulin release in controls at 5 minutes that persisted through 30 minutes; insulin was suppressed (P < .05) by epinephrine from 5 to 15 minutes, rising gradually through 30 minutes. Experiments 2 (IR2G) and 3 (GR2I) were conducted as triplicated 3 × 3 Latin squares with the following treatments: saline (SAL), DET, and DET/BUT (all administered at .01 mg/kg BW). Glucose stimulated (P < .05) insulin release that persisted through 30 minutes in SAL mares; DET and DET/BUT severely suppressed (P < .0001) the IR2G. Sedation did not affect resting glucose and had inconsistent effects on the GR2I when mares were treated with 50 mIU/kg BW recombinant human insulin. Butorphanol had no effect on IR2G. In conclusion, adrenergic agonists severely suppress the IR2G and cannot be used for sedation for this test. The use of DET did not alter the GR2I, and therefore may be useful for conducting this test in fractious horses.
    Keywords adrenergic agonists ; butorphanol ; detomidine ; epinephrine ; glucose ; humans ; insulin ; insulin resistance ; mares ; sedation
    Language English
    Dates of publication 2020-02
    Publishing place Elsevier Inc.
    Document type Article
    ZDB-ID 2102631-2
    ISSN 1542-7412 ; 0737-0806
    ISSN (online) 1542-7412
    ISSN 0737-0806
    DOI 10.1016/j.jevs.2019.102842
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  4. Article ; Online: Effects of clenbuterol administration on serum biochemical, histologic, and echocardiographic measurements of muscle injury in exercising horses.

    Thompson, Jessica A / Eades, Susan C / Chapman, Ann M / Paulsen, Daniel B / Barker, Steven A / McConnico, Rebecca S

    American journal of veterinary research

    2012  Volume 73, Issue 6, Page(s) 875–883

    Abstract: Objective: To determine the effects of clenbuterol, at a dosage of up to 3.2 μg/kg for 14 days, PO, on skeletal and cardiac muscle in healthy horses undergoing treadmill exercise.: Animals: 12 healthy horses from 3 to 10 years old.: Procedures: ... ...

    Abstract Objective: To determine the effects of clenbuterol, at a dosage of up to 3.2 μg/kg for 14 days, PO, on skeletal and cardiac muscle in healthy horses undergoing treadmill exercise.
    Animals: 12 healthy horses from 3 to 10 years old.
    Procedures: Horses were randomly assigned to a control group (n = 6) or clenbuterol group (6) and received either saline (0.9% NaCl) solution or clenbuterol, PO, every 12 hours for 14 days. Horses were subjected to submaximal treadmill exercise daily during treatment. Muscle biopsy specimens were collected before and after treatment for determination of apoptosis. Echocardiographic measurements, serum clenbuterol and cardiac troponin I concentrations, and serum activities of creatine kinase and aspartate aminotransferase were measured before, during, and after treatment. Jugular venous blood samples were collected every 3 days during treatment. Echocardiography was repeated every 7 days after beginning treatment. Response variables were compared between treatment groups and across time periods.
    Results: No significant effect of clenbuterol or exercise on response variables was found between treatment and control groups at any time point or within groups over time.
    Conclusions and clinical relevance: Results did not reveal any adverse effects of treatment with an approved dose of clenbuterol on equine cardiac or skeletal muscle in the small number of horses tested.
    MeSH term(s) Analysis of Variance ; Animals ; Apoptosis/drug effects ; Apoptosis/physiology ; Aspartate Aminotransferases/blood ; Biopsy/veterinary ; Clenbuterol/administration & dosage ; Clenbuterol/blood ; Clenbuterol/pharmacology ; Creatine Kinase/blood ; Echocardiography/veterinary ; Horses/injuries ; Immunohistochemistry/veterinary ; Muscle, Skeletal/drug effects ; Muscle, Skeletal/injuries ; Physical Conditioning, Animal/adverse effects ; Troponin I/metabolism
    Chemical Substances Troponin I ; Aspartate Aminotransferases (EC 2.6.1.1) ; Creatine Kinase (EC 2.7.3.2) ; Clenbuterol (XTZ6AXU7KN)
    Language English
    Publishing date 2012-06
    Publishing country United States
    Document type Journal Article ; Randomized Controlled Trial ; Research Support, Non-U.S. Gov't
    ZDB-ID 390796-x
    ISSN 1943-5681 ; 0002-9645
    ISSN (online) 1943-5681
    ISSN 0002-9645
    DOI 10.2460/ajvr.73.6.875
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 422: Show issues Location:
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  5. Article: Effects of clenbuterol administration on serum biochemical, histologic, and echocardiographic measurements of muscle injury in exercising horses

    Thompson, Jessica A / Eades, Susan C / Chapman, Ann M / Paulsen, Daniel B / Barker, Steven A / McConnico, Rebecca S

    American journal of veterinary research. 2012 June, v. 73, no. 6

    2012  

    Abstract: Objective: To determine the effects of clenbuterol, at a dosage of up to 3.2 μg/kg for 14 days, PO, on skeletal and cardiac muscle in healthy horses undergoing treadmill exercise. Animals: 12 healthy horses from 3 to 10 years old. Procedures: Horses were ...

    Abstract Objective: To determine the effects of clenbuterol, at a dosage of up to 3.2 μg/kg for 14 days, PO, on skeletal and cardiac muscle in healthy horses undergoing treadmill exercise. Animals: 12 healthy horses from 3 to 10 years old. Procedures: Horses were randomly assigned to a control group (n = 6) or clenbuterol group (6) and received either saline (0.9% NaCl) solution or clenbuterol, PO, every 12 hours for 14 days. Horses were subjected to submaximal treadmill exercise daily during treatment. Muscle biopsy specimens were collected before and after treatment for determination of apoptosis. Echocardiographic measurements, serum clenbuterol and cardiac troponin I concentrations, and serum activities of creatine kinase and aspartate aminotransferase were measured before, during, and after treatment. Jugular venous blood samples were collected every 3 days during treatment. Echocardiography was repeated every 7 days after beginning treatment. Response variables were compared between treatment groups and across time periods. Results: No significant effect of clenbuterol or exercise on response variables was found between treatment and control groups at any time point or within groups over time. Conclusions and Clinical Relevance: Results did not reveal any adverse effects of treatment with an approved dose of clenbuterol on equine cardiac or skeletal muscle in the small number of horses tested.
    Keywords adverse effects ; apoptosis ; aspartate transaminase ; biopsy ; blood serum ; clenbuterol ; creatine kinase ; echocardiography ; exercise ; horses ; muscles ; myocardium ; skeletal muscle ; sodium chloride
    Language English
    Size p. 875-883.
    Document type Article
    ZDB-ID 390796-x
    ISSN 1943-5681 ; 0002-9645
    ISSN (online) 1943-5681
    ISSN 0002-9645
    DOI 10.2460/ajvr.73.6.875
    Database NAL-Catalogue (AGRICOLA)

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    In stock of ZB MED Bonn / Germany

    Z 3312: Show issues

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  6. Article: Effects of clenbuterol administration on serum biochemical, histologic, and echocardiographic measurements of muscle injury in exercising horses

    Thompson, Jessica A. / Eades, Susan C. / Chapman, Ann M. / Paulsen, Daniel B. / Barker, Steven A. / McConnico, Rebecca S.

    American journal of veterinary research

    Volume v. 73,, Issue no. 6

    Abstract: Objective: To determine the effects of clenbuterol, at a dosage of up to 3.2 μg/kg for 14 days, PO, on skeletal and cardiac muscle in healthy horses undergoing treadmill exercise. Animals: 12 healthy horses from 3 to 10 years old. Procedures: Horses were ...

    Abstract Objective: To determine the effects of clenbuterol, at a dosage of up to 3.2 μg/kg for 14 days, PO, on skeletal and cardiac muscle in healthy horses undergoing treadmill exercise. Animals: 12 healthy horses from 3 to 10 years old. Procedures: Horses were randomly assigned to a control group (n = 6) or clenbuterol group (6) and received either saline (0.9% NaCl) solution or clenbuterol, PO, every 12 hours for 14 days. Horses were subjected to submaximal treadmill exercise daily during treatment. Muscle biopsy specimens were collected before and after treatment for determination of apoptosis. Echocardiographic measurements, serum clenbuterol and cardiac troponin I concentrations, and serum activities of creatine kinase and aspartate aminotransferase were measured before, during, and after treatment. Jugular venous blood samples were collected every 3 days during treatment. Echocardiography was repeated every 7 days after beginning treatment. Response variables were compared between treatment groups and across time periods. Results: No significant effect of clenbuterol or exercise on response variables was found between treatment and control groups at any time point or within groups over time. Conclusions and Clinical Relevance: Results did not reveal any adverse effects of treatment with an approved dose of clenbuterol on equine cardiac or skeletal muscle in the small number of horses tested.
    Keywords blood serum ; myocardium ; clenbuterol ; sodium chloride ; biopsy ; horses ; apoptosis ; exercise ; aspartate transaminase ; skeletal muscle ; creatine kinase ; adverse effects ; muscles ; echocardiography
    Language English
    Document type Article
    ISSN 0002-9645
    Database AGRIS - International Information System for the Agricultural Sciences and Technology

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