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Article ; Online: Exploration of Resonant Modes for Circular and Polygonal Chladni Plates.

Val Baker, Amira / Csanad, Mate / Fellas, Nicolas / Atassi, Nour / Mgvdliashvili, Ia / Oomen, Paul

2024 Volume 26, Issue 3

Abstract: In general, sound waves propagate radially outwards from a point source. These waves will continue in the same direction, decreasing in intensity, unless a boundary condition is met. To arrive at a universal understanding of the relation between ... ...

Abstract	In general, sound waves propagate radially outwards from a point source. These waves will continue in the same direction, decreasing in intensity, unless a boundary condition is met. To arrive at a universal understanding of the relation between frequency and wave propagation within spatial boundaries, we explore the maximum entropy states that are realized as resonant modes. For both circular and polygonal Chladni plates, a model is presented that successfully recreates the nodal line patterns to a first approximation. We discuss the benefits of such a model and the future work necessary to develop the model to its full predictive ability.
Language	English
Publishing date	2024-03-15
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2014734-X
ISSN	1099-4300 ; 1099-4300
ISSN (online)	1099-4300
ISSN	1099-4300
DOI	10.3390/e26030264
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Article ; Online: Time evolution of the sQGP with hydrodynamic models

Csanád M.

EPJ Web of Conferences, Vol 70, p

2014 Volume 00011

Abstract: The strongly interacting Quark-Gluon-Plasma (sQGP) created in relativistic nucleus-nucleus collisions, can be described by hydrodynamic models. Low energy hadrons are created after the so called freeze-out of this medium, thus their distributions reveal ... ...

Abstract	The strongly interacting Quark-Gluon-Plasma (sQGP) created in relativistic nucleus-nucleus collisions, can be described by hydrodynamic models. Low energy hadrons are created after the so called freeze-out of this medium, thus their distributions reveal information about the final state of the sQGP. Photons are created throughout the evolution, so their distributions carry information on the whole expansion and cool down. We show results from an analytic, 1+3 dimensional perfect relativistic hydrodynamic solution, and compare hadron and photon observables to RHIC data. We extract an average equation of state of the expanding quark matter from this comparison. In the second part of this paper, we generalize the before mentioned analytic solution of relativistic perfect fluid hydrodynamics to arbitrary temperature-dependent Equation of State. We investigate special cases of this class of solutions, in particular, we present hydrodynamical solutions with an Equation of State determined from lattice QCD calculations.
Keywords	Physics ; QC1-999 ; Science ; Q
Language	English
Publishing date	2014-04-01T00:00:00Z
Publisher	EDP Sciences
Document type	Article ; Online
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Article ; Online: Event-by-Event Investigation of the Two-Particle Source Function in Heavy-Ion Collisions with EPOS.

Kincses, Dániel / Stefaniak, Maria / Csanád, Máté

Entropy (Basel, Switzerland)

2022 Volume 24, Issue 3

Abstract: Exploring the shape of the pair-source function for particles such as pions or kaons has been an important goal of heavy-ion physics, and substantial effort has been made in order to understand the underlying physics behind the experimental observations ... ...

Abstract	Exploring the shape of the pair-source function for particles such as pions or kaons has been an important goal of heavy-ion physics, and substantial effort has been made in order to understand the underlying physics behind the experimental observations of non-Gaussian behavior. In experiments, since no direct measurement of the source function is possible, quantum-statistical momentum correlations are utilized to gain information about the space-time geometry of the particle emitting source. Event generators, such as EPOS, however, provide direct access to the freeze-out coordinates of final state particles, and thus the source function can be constructed and investigated. The EPOS model is a sophisticated hybrid model where the initial stage evolution of the system is governed by Parton-Based Gribov-Regge theory, and subsequently a hydrodynamic evolution is utilized, followed by hadronization and hadron dynamics. EPOS has already proven to be successful in describing several different experimental observations for systems characterized by baryon chemical potential close to zero, but so far the source shape has not been explored in detail. In this paper we discuss an event-by-event analysis of the two-particle source function in sNN = 200 GeV Au+Au collisions generated by the EPOS model. We find that when utilizing all stages of the model, Lévy-shaped distributions (unlike Gaussian distributions) provide a good description of the source shape in the individual events. Hence it is clear that it is not the event averaging that creates the non-Gaussian features in the pair distributions. Based on this observation, we determine Lévy-parameters of the source as a function of event centrality and particle momentum.
Language	English
Publishing date	2022-02-22
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2014734-X
ISSN	1099-4300 ; 1099-4300
ISSN (online)	1099-4300
ISSN	1099-4300
DOI	10.3390/e24030308
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Article: Az ismétlődési kockázat becslése családi halmozódást mutató emlőrák esetén.

Joó, József Gábor / Csanád, Mónika / Tóth, Katalin / Máté, Szabolcs / Nagy, Zsolt

Orvosi hetilap

2011 Volume 152, Issue 19, Page(s) 758–762

Abstract: Women with a history of breast cancer are common at centers for cancer genetic risk all over Europe. Given limited health care resources, managing this demand, while achieving good value for money coming from health services, is generally a major ... ...

Title translation	Risk assessment in familial breast cancer.
Abstract	Women with a history of breast cancer are common at centers for cancer genetic risk all over Europe. Given limited health care resources, managing this demand, while achieving good value for money coming from health services, is generally a major challenge. This paper recapitulates and summarizes the available methods of the risk assessment of familial breast cancer. After a systematic review of the literature Gail-model, Claus-model and BOADICEA-model were selected, as well as softwares (LINKAGE software; MENDEL v3.3 software) available in the application of these algorhythms are also summarized. Comparisons were made between the models concerning their advantages and disadvantages. The really reliable methods of risk estimation of familial breast cancer are always based on the analysis of the pedigree structure and allow the estimation of the patient's probability of carrying a susceptibility gene under a particular genetic model, given her family history. For this method the knowledge of BRCA mutation status is absolutely indispensable. The methods of BRCA mutation analysis as well as the main characteristics of the occurrence of BRCA mutation carrier condition are discussed in details.
MeSH term(s)	Adult ; Age Factors ; Aged ; Breast Neoplasms/genetics ; DNA Mutational Analysis ; Female ; Genes, BRCA1 ; Genes, BRCA2 ; Genetic Predisposition to Disease ; Genetic Testing ; Humans ; Middle Aged ; Penetrance ; Risk Assessment ; Risk Factors
Language	Hungarian
Publishing date	2011-05-08
Publishing country	Hungary
Document type	English Abstract ; Journal Article
ZDB-ID	123879-6
ISSN	1788-6120 ; 0030-6002
ISSN (online)	1788-6120
ISSN	0030-6002
DOI	10.1556/OH.2011.29110
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Article: A BRCA-génmutációt hordozó nők klinikogenetikai ellátása: szűrés, diagnosztika, terápia.

Nagy, Zsolt / Csanád, Mónika / Tóth, Katalin / Máté, Szabolcs / Joó, József Gábor

Orvosi hetilap

2011 Volume 152, Issue 23, Page(s) 913–918

Abstract: Predictive genetics opens a considerable perspective in the diagnostics as well as the treatment of breast and ovarian cancer. Current recommendations and guidelines for the management of BRCA 1 and BRCA 2 mutation carriers are not based on controlled ... ...

Title translation	Clinical-genetic care of BRCA-mutation carrier women: prevention, diagnosis and therapy.
Abstract	Predictive genetics opens a considerable perspective in the diagnostics as well as the treatment of breast and ovarian cancer. Current recommendations and guidelines for the management of BRCA 1 and BRCA 2 mutation carriers are not based on controlled randomized trials, but on expert opinions. The existing options of prevention, early diagnosis and treatment must be clearly interpreted to the patient. In the context of a dedicated genetic counseling the participation of all involved professionals (geneticist, oncologist, surgeon, gynecologist) is required. The decision-making process concerning the possibilities of prevention, diagnosis and treatment is always deeply influenced by the patient's own experience with the cancer occurred in the family, as well as by her values and expectations of life. The focused multidisciplinary approach, with the application of results from prospective studies in cohorts of BRCA mutation carriers allow the concerned individuals to benefit from this kind of approach of medical treatment.
MeSH term(s)	Adult ; Aged ; Breast Neoplasms/diagnosis ; Breast Neoplasms/prevention & control ; Breast Neoplasms/therapy ; Case Management/organization & administration ; Case Management/standards ; Early Detection of Cancer ; Female ; Genes, BRCA1 ; Genes, BRCA2 ; Genetic Counseling ; Genetic Predisposition to Disease ; Heterozygote ; Humans ; Middle Aged ; Mutation ; Ovarian Neoplasms/diagnosis ; Ovarian Neoplasms/prevention & control ; Ovarian Neoplasms/therapy ; Primary Prevention/methods
Language	Hungarian
Publishing date	2011-06-05
Publishing country	Hungary
Document type	English Abstract ; Journal Article
ZDB-ID	123879-6
ISSN	1788-6120 ; 0030-6002
ISSN (online)	1788-6120
ISSN	0030-6002
DOI	10.1556/OH.2011.29131
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Book ; Online: Measurement of $\psi(2S)$ nuclear modification at backward and forward rapidity in $p$$+$$p$, $p$$+$Al, and $p$$+$Au collisions at $\sqrt{s_{_{NN}}}=200$ GeV

Acharya, U. A. / Aidala, C. / Akiba, Y. / Alfred, M. / Andrieux, V. / Apadula, N. / Asano, H. / Azmoun, B. / Babintsev, V. / Bandara, N. S. / Barish, K. N. / Bathe, S. / Bazilevsky, A. / Beaumier, M. / Belmont, R. / Berdnikov, A. / Berdnikov, Y. / Bichon, L. / Blankenship, B. /

Blau, D. S. / Bok, J. S. / Borisov, V. / Brooks, M. L. / Bryslawskyj, J. / Bumazhnov, V. / Campbell, S. / Roman, V. Canoa / Cervantes, R. / Chiu, M. / Chi, C. Y. / Choi, I. J. / Choi, J. B. / Citron, Z. / Connors, M. / Morales, Y. Corrales / Corliss, R. / Cronin, N. / Csörgő, T. / Csanád, M. / Danley, T. W. / Daugherity, M. S. / David, G. / Dean, C. T. / DeBlasio, K. / Dehmelt, K. / Denisov, A. / Deshpande, A. / Desmond, E. J. / Dion, A. / Dixit, D.

2022

Abstract: Suppression of the $J/\psi$ nuclear-modification factor has been seen as a trademark signature of final-state effects in large collision systems for decades. In small systems, the nuclear modification was attributed to cold-nuclear-matter effects until ... ...

Abstract	Suppression of the $J/\psi$ nuclear-modification factor has been seen as a trademark signature of final-state effects in large collision systems for decades. In small systems, the nuclear modification was attributed to cold-nuclear-matter effects until the observation of strong differential suppression of the $\psi(2S)$ state in $p/d$$+$$A$ collisions suggested the presence of final-state effects. Results of $J/\psi$ and $\psi(2S)$ measurements in the dimuon decay channel are presented here for $p$$+$$p$, $p$$+$Al, and $p$$+$Au collision systems at $\sqrt{s_{_{NN}}}=200$ GeV. The results are predominantly shown in the form of the nuclear-modification factor, $R_{pA}$, the ratio of the $\psi(2S)$ invariant yield per nucleon-nucleon collision in collisions of proton on target nucleus to that in $p$$+$$p$ collisions. Measurements of the $J/\psi$ and $\psi(2S)$ nuclear-modification factor are compared with shadowing and transport-model predictions, as well as to complementary measurements at Large-Hadron-Collider energies. Comment: 315 authors from 69 institutions, 16 pages, 9 figures, 4 tables, 2015 data. v2 is version accepted for publication in Physical Review C. Plain text data tables for the points plotted in figures for this and previous PHENIX publications are (or will be) publicly available at http://www.phenix.bnl.gov/papers.html
Keywords	Nuclear Experiment ; High Energy Physics - Experiment
Subject code	306
Publishing date	2022-02-08
Publishing country	us
Document type	Book ; Online
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Article ; Online: Gene expression profiling in Paget's disease of bone: upregulation of interferon signaling pathways in pagetic monocytes and lymphocytes.

Nagy, Zsolt B / Gergely, Péter / Donáth, Judit / Borgulya, Gábor / Csanád, Mónika / Poór, Gyula

Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research

2008 Volume 23, Issue 2, Page(s) 253–259

Abstract: Unlabelled: We examined the gene expression profile of genes involved in bone metabolism in 23 patients with PD compared with 23 healthy controls. We found a significant overexpression of the genes of the IFN pathway along with a downregulation of tnf- ... ...

Abstract	Unlabelled: We examined the gene expression profile of genes involved in bone metabolism in 23 patients with PD compared with 23 healthy controls. We found a significant overexpression of the genes of the IFN pathway along with a downregulation of tnf-alpha. Our result suggest that IFN-mediated signaling may play important roles in aberrant osteoclastogenesis of PD. Introduction: Paget's disease of bone (PD) is characterized by focal regions of highly exaggerated bone remodeling and aberrant osteoclastogenesis. Under physiological conditions, circulating monocytes may serve as early progenitors of osteoclasts and along with peripheral blood lymphocytes produce a wide variety of factors important in bone metabolism. Nevertheless, little is known about the roles of circulating monocytes and lymphocytes in relation to the pathological bone turnover in PD. Materials and methods: In this study, we aimed at investigating the gene expression pattern of PD using quantitative real-time PCR in monocytes and lymphocytes isolated from peripheral blood mononuclear cells (PBMCs). Fifteen genes known to be involved in osteoclastogenesis were studied in cells from 23 patients with PD and in cells from 23 healthy controls. Eight human genes including ifn-alpha (3.48-fold, p < 0.001), ifn-beta (2.68-fold, p < 0.001), ifn-gamma (1.98-fold, p = 0.002), p38 beta2 mapk (2.47-fold, p = 0.002), ifn-gammar1 (2.03-fold, p = 0.01), ifn-gammar2 (1.81-fold, p = 0.02), stat1 (1.57-fold, p = 0.037), and tnf-alpha (-2.34, p < 0.001) were found to be significantly altered in pagetic monocytes compared with monocytes of healthy controls. Results: In pagetic lymphocytes, significant changes in the expression of ifn-alpha (2.17-fold, p < 0.001), ifn-beta (2.13-fold, p = 0.005), ifn-gamma (1.89-fold, p < 0.001), ifn-gammar1 (1.02-fold, p = 0.04), ifn-gammar2 (1.01-fold, p = 0.031), stat2 (1.79-fold, p < 0.001), and tnf-alpha (-1.49, p < 0.001) were found compared with lymphocytes of healthy controls. Furthermore, IFN-gamma protein was significantly elevated in the sera of PD patients (18.7 +/- 6.69 pg/ml) compared with healthy controls (3.87 +/- 6.48 pg/ml, p = 0.042). Conclusions: In conclusion, our data suggest that novel pathways mainly related to the IFN-mediated signaling may play important roles in the aberrant osteoclastogenesis of PD.
MeSH term(s)	Adult ; Aged ; Aged, 80 and over ; Base Sequence ; Female ; Gene Expression Profiling ; Humans ; Interferon-gamma/blood ; Interferon-gamma/genetics ; Interferon-gamma/metabolism ; Lymphocytes/pathology ; Male ; Middle Aged ; Molecular Sequence Data ; Monocytes/pathology ; Osteitis Deformans/genetics ; Signal Transduction/genetics ; Signal Transduction/immunology
Chemical Substances	Interferon-gamma (82115-62-6)
Language	English
Publishing date	2008-02
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	632783-7
ISSN	1523-4681 ; 0884-0431
ISSN (online)	1523-4681
ISSN	0884-0431
DOI	10.1359/jbmr.071021
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Article ; Online: Gene expression patterns of insulin-like growth factor 1, insulin-like growth factor 2 and insulin-like growth factor binding protein 3 in human placenta from pregnancies with intrauterine growth restriction.

Börzsönyi, Balázs / Demendi, Csaba / Nagy, Zsolt / Tóth, Katalin / Csanád, Mónika / Pajor, Attila / Rig, János / Joó, József Gábor

Journal of perinatal medicine

2011 Volume 39, Issue 6, Page(s) 701–707

Abstract: Introduction: In this study, we compared insulin-like growth factor (IGF)-gene expression patterns and characteristics of glucose and insulin metabolism in human placenta from pregnancies with or without intrauterine growth restriction (IUGR).: ... ...

Abstract	Introduction: In this study, we compared insulin-like growth factor (IGF)-gene expression patterns and characteristics of glucose and insulin metabolism in human placenta from pregnancies with or without intrauterine growth restriction (IUGR). Materials and methods: We compared 101 human placentas from intrauterine growth restriction pregnancies to those of 140 normal pregnancies treated at our department in a one-year period. We have also assessed the serum glucose and insulin levels of the IUGR and control groups. Several possible predicting factors of IUGR were also investigated. Results: Risk for IUGR was suggested by gestational weight gain and gestational increase in maternal body mass index (BMI) as well as maternal birthweight. In pregnancies without IUGR, umbilical cord glucose and insulin levels were significantly higher than in pregnancies with IUGR. In placentas from pregnancies with IUGR an overexpression of the IGF-2 and the insulin-like growth factor binding protein (IGFBP)-3 genes was found. In placentas from pregnancies with male fetal gender we found a significant overexpression of the IGF-2 gene. Discussion: Gestational weight gain and BMI increase seem to predict the development of IUGR. Insulin and carbohydrate metabolism are also impaired in IUGR fetuses. In the placentas from pregnancies with IUGR, IGF-2 is overexpressed reflecting its physiological role in optimizing energy distribution in a low-energy environment.
MeSH term(s)	Adolescent ; Adult ; Base Sequence ; Birth Weight ; Blood Glucose/metabolism ; Case-Control Studies ; DNA Primers/genetics ; Female ; Fetal Blood/metabolism ; Fetal Growth Retardation/blood ; Fetal Growth Retardation/genetics ; Fetal Growth Retardation/pathology ; Gene Expression ; Humans ; Infant, Newborn ; Insulin/blood ; Insulin-Like Growth Factor Binding Protein 3/genetics ; Insulin-Like Growth Factor I/genetics ; Insulin-Like Growth Factor II/genetics ; Male ; Maternal Age ; Placenta/metabolism ; Pregnancy ; Risk Factors ; Sex Ratio ; Young Adult
Chemical Substances	Blood Glucose ; DNA Primers ; IGFBP3 protein, human ; Insulin ; Insulin-Like Growth Factor Binding Protein 3 ; Insulin-Like Growth Factor I (67763-96-6) ; Insulin-Like Growth Factor II (67763-97-7)
Language	English
Publishing date	2011-08-08
Publishing country	Germany
Document type	Journal Article
ZDB-ID	123512-6
ISSN	1619-3997 ; 0300-5577 ; 0936-174X
ISSN (online)	1619-3997
ISSN	0300-5577 ; 0936-174X
DOI	10.1515/jpm.2011.090
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Article ; Online: Current concepts in the genetic diagnostics of rheumatoid arthritis.

Nagy, Zsolt B / Csanád, Mónika / Tóth, Katalin / Börzsönyi, Balázs / Demendi, Csaba / Rigó, János / Joó, József Gábor

Expert review of molecular diagnostics

2010 Volume 10, Issue 5, Page(s) 603–618

Abstract: Rheumatoid arthritis (RA) is a systemic, chronic and inflammatory disease of unknown etiology. HLA-DRB1 and PTPN22 1858T gene variants are risk factors of RA, clinical manifestations and rate of progression of joint destruction in this autoimmune disease. ...

Abstract	Rheumatoid arthritis (RA) is a systemic, chronic and inflammatory disease of unknown etiology. HLA-DRB1 and PTPN22 1858T gene variants are risk factors of RA, clinical manifestations and rate of progression of joint destruction in this autoimmune disease. Currently, several immunopathogenetic models of other genes (CTLA4, MIF, PADI4 and SLC22A4) are under debate. The clinical influence of some of the gene polymorphisms associated with RA and the principles of pharmacogenetics applied to different therapies, such as classical disease-modifying anti-rheumatic drugs and new biological agents. Pharmacogenetics is a rapidly advancing area of research that holds the promise that therapies will soon be tailored to an individual patient's genetic profile.
MeSH term(s)	Antirheumatic Agents/therapeutic use ; Arthritis, Rheumatoid/diagnosis ; Arthritis, Rheumatoid/drug therapy ; Arthritis, Rheumatoid/genetics ; Arthritis, Rheumatoid/immunology ; Azathioprine/metabolism ; Azathioprine/therapeutic use ; Genetic Predisposition to Disease ; HLA-DR Antigens/genetics ; HLA-DRB1 Chains ; Humans ; Methotrexate/metabolism ; Methotrexate/therapeutic use ; Pharmacogenetics ; Polymorphism, Single Nucleotide ; Protein Tyrosine Phosphatase, Non-Receptor Type 22/genetics ; Sulfasalazine/metabolism ; Sulfasalazine/therapeutic use
Chemical Substances	Antirheumatic Agents ; HLA-DR Antigens ; HLA-DRB1 Chains ; Sulfasalazine (3XC8GUZ6CB) ; Protein Tyrosine Phosphatase, Non-Receptor Type 22 (EC 3.1.3.48) ; Azathioprine (MRK240IY2L) ; Methotrexate (YL5FZ2Y5U1)
Language	English
Publishing date	2010-07
Publishing country	England
Document type	Journal Article
ZDB-ID	2112530-2
ISSN	1744-8352 ; 1473-7159
ISSN (online)	1744-8352
ISSN	1473-7159
DOI	10.1586/erm.10.36
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Article ; Online: Erratum: Global Polarization of Ξ and Ω Hyperons in Au+Au Collisions at sqrt[s_{NN}]=200 GeV [Phys. Rev. Lett. 126, 162301 (2021)].

Adam, J / Adamczyk, L / Adams, J R / Adkins, J K / Agakishiev, G / Aggarwal, M M / Ahammed, Z / Alekseev, I / Anderson, D M / Aparin, A / Aschenauer, E C / Ashraf, M U / Atetalla, F G / Attri, A / Averichev, G S / Bairathi, V / Barish, K / Behera, A / Bellwied, R /

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Physical review letters

2023 Volume 131, Issue 8, Page(s) 89901

Abstract: This corrects the article DOI: 10.1103/PhysRevLett.126.162301. ...

Abstract	This corrects the article DOI: 10.1103/PhysRevLett.126.162301.
Language	English
Publishing date	2023-09-04
Publishing country	United States
Document type	Published Erratum
ZDB-ID	208853-8
ISSN	1079-7114 ; 0031-9007
ISSN (online)	1079-7114
ISSN	0031-9007
DOI	10.1103/PhysRevLett.131.089901
Database	MEDical Literature Analysis and Retrieval System OnLINE

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