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  1. Article ; Online: Should PLEX Be Used for Severe AKI and/or Pulmonary Hemorrhage in ANCA-Associated Vasculitis (AAV)? PRO.

    Derebail, Vimal K

    Kidney360

    2021  Volume 2, Issue 5, Page(s) 776–778

    MeSH term(s) Acute Kidney Injury/etiology ; Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/complications ; Hemorrhage/etiology ; Humans ; Lung Diseases/etiology
    Language English
    Publishing date 2021-05-27
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural
    ISSN 2641-7650
    ISSN (online) 2641-7650
    DOI 10.34067/KID.0006762020
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  2. Article ; Online: Sparsentan and the Expanding Landscape of IgA Nephropathy Treatment.

    Derebail, Vimal K / Nachman, Patrick H

    Clinical journal of the American Society of Nephrology : CJASN

    2023  

    Language English
    Publishing date 2023-07-27
    Publishing country United States
    Document type Journal Article
    ZDB-ID 2226665-3
    ISSN 1555-905X ; 1555-9041
    ISSN (online) 1555-905X
    ISSN 1555-9041
    DOI 10.2215/CJN.0000000000000268
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  3. Article: Editorial: Pathogenesis and management of glomerular diseases.

    Lionaki, Sophia / Derebail, Vimal K

    Frontiers in medicine

    2022  Volume 9, Page(s) 1018776

    Language English
    Publishing date 2022-09-26
    Publishing country Switzerland
    Document type Editorial
    ZDB-ID 2775999-4
    ISSN 2296-858X
    ISSN 2296-858X
    DOI 10.3389/fmed.2022.1018776
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  4. Article ; Online: Sickle Cell Disease and the Kidney: Filters Gone Awry.

    Lebensburger, Jeffrey D / Derebail, Vimal K

    Hematology/oncology clinics of North America

    2022  Volume 36, Issue 6, Page(s) 1239–1254

    Abstract: Throughout the lifespan, the kidney of a person with sickle cell disease is at increased risk of injury to the glomeruli, tubules, and renal vasculature. This injury manifests as urine concentrating defects, enuresis, albuminuria, acute kidney injury, ... ...

    Abstract Throughout the lifespan, the kidney of a person with sickle cell disease is at increased risk of injury to the glomeruli, tubules, and renal vasculature. This injury manifests as urine concentrating defects, enuresis, albuminuria, acute kidney injury, chronic kidney disease, and end-stage kidney disease. The outcomes for patients who progress to end-stage kidney disease are poor and access to organ transplantation is limited. Therefore, identifying risk factors for progression, intervening with renoprotective agents early in life, and improving access to care are vital for sickle cell patients.
    MeSH term(s) Humans ; Kidney ; Anemia, Sickle Cell/complications ; Anemia, Sickle Cell/epidemiology ; Anemia, Sickle Cell/therapy ; Kidney Failure, Chronic/etiology ; Acute Kidney Injury/complications
    Language English
    Publishing date 2022-11-19
    Publishing country United States
    Document type Journal Article ; Review
    ZDB-ID 93115-9
    ISSN 1558-1977 ; 0889-8588
    ISSN (online) 1558-1977
    ISSN 0889-8588
    DOI 10.1016/j.hoc.2022.08.009
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  5. Article ; Online: Emerging Therapies and Advances in Sickle Cell Disease with a Focus on Renal Manifestations.

    Obadina, Mofiyin / Wilson, Sam / Derebail, Vimal K / Little, Jane

    Kidney360

    2023  Volume 4, Issue 7, Page(s) 997–1005

    Abstract: The underlying mechanisms of disease in sickle cell disease (SCD) contribute to a multifaceted nephropathy, commonly manifested as albuminuria. In severe SCD genotypes ( e.g. , Hemoglobin SS [HbSS]), albuminuria and CKD are major predictors of mortality ... ...

    Abstract The underlying mechanisms of disease in sickle cell disease (SCD) contribute to a multifaceted nephropathy, commonly manifested as albuminuria. In severe SCD genotypes ( e.g. , Hemoglobin SS [HbSS]), albuminuria and CKD are major predictors of mortality in this population. Therefore, the monitoring and management of renal function is an intrinsic part of comprehensive care in SCD. Management of nephropathy in SCD can be accomplished with SCD-directed therapies and/or CKD-directed therapies. In the past 5 years, novel disease-modifying and palliative therapies have been approved in SCD to target aspects of the disease, such as anemia, inflammation, and vasculopathy. Along with conventional hydroxyurea and chronic transfusion, l -glutamine, crizanlizumab, and voxelotor have all been shown to mitigate some adverse effect of SCD, and their effect on nephropathy is being investigated. CKD-directed therapies such as renin-angiotensin-aldosterone system blockers have long been used in SCD nephropathy; however, more complete long-term studies on benefits are needed. Given the effect of renal disease on survival, further assessment of the mechanisms and efficacy of these SCD-directed or CKD-directed therapeutic agents is essential.
    MeSH term(s) Humans ; Albuminuria/etiology ; Anemia, Sickle Cell/complications ; Anemia, Sickle Cell/drug therapy ; Kidney/physiology ; Hemoglobin, Sickle/therapeutic use ; Vascular Diseases ; Renal Insufficiency, Chronic/therapy
    Chemical Substances Hemoglobin, Sickle
    Language English
    Publishing date 2023-05-31
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, U.S. Gov't, P.H.S.
    ISSN 2641-7650
    ISSN (online) 2641-7650
    DOI 10.34067/KID.0000000000000162
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  6. Article ; Online: The nephropathy of sickle cell trait and sickle cell disease.

    Ataga, Kenneth I / Saraf, Santosh L / Derebail, Vimal K

    Nature reviews. Nephrology

    2022  Volume 18, Issue 6, Page(s) 361–377

    Abstract: Sickle cell syndromes, including sickle cell disease (SCD) and sickle cell trait, are associated with multiple kidney abnormalities. Young patients with SCD have elevated effective renal plasma flow and glomerular filtration rates, which decrease to ... ...

    Abstract Sickle cell syndromes, including sickle cell disease (SCD) and sickle cell trait, are associated with multiple kidney abnormalities. Young patients with SCD have elevated effective renal plasma flow and glomerular filtration rates, which decrease to normal ranges in young adulthood and subnormal levels with advancing age. The pathophysiology of SCD-related nephropathy is multifactorial - oxidative stress, hyperfiltration and glomerular hypertension are all contributing factors. Albuminuria, which is an early clinical manifestation of glomerular damage, is common in individuals with SCD. Kidney function declines more rapidly in individuals with SCD than in those with sickle cell trait or in healthy individuals. Multiple genetic modifiers, including APOL1, HMOX1, HBA1 and HBA2 variants are also implicated in the development and progression of SCD-related nephropathy. Chronic kidney disease and rapid decline in estimated glomerular filtration rate are associated with increased mortality in adults with SCD. Renin-angiotensin-aldosterone system inhibitors are the standard of care treatment for albuminuria in SCD, despite a lack of controlled studies demonstrating their long-term efficacy. Multiple studies of novel therapeutic agents are ongoing, and patients with SCD and kidney failure should be evaluated for kidney transplantation. Given the high prevalence and severe consequences of kidney disease, additional studies are needed to elucidate the pathophysiology, natural history and treatment of SCD-related nephropathy.
    MeSH term(s) Adult ; Albuminuria/etiology ; Anemia, Sickle Cell/complications ; Anemia, Sickle Cell/genetics ; Apolipoprotein L1/genetics ; Glomerular Filtration Rate ; Humans ; Kidney Diseases ; Renal Insufficiency, Chronic/therapy ; Sickle Cell Trait/complications ; Young Adult
    Chemical Substances APOL1 protein, human ; Apolipoprotein L1
    Language English
    Publishing date 2022-02-21
    Publishing country England
    Document type Journal Article ; Review ; Research Support, U.S. Gov't, P.H.S. ; Research Support, N.I.H., Extramural
    ZDB-ID 2490366-8
    ISSN 1759-507X ; 1759-5061
    ISSN (online) 1759-507X
    ISSN 1759-5061
    DOI 10.1038/s41581-022-00540-9
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  7. Article ; Online: ANCA-Associated Vasculitis - Refining Therapy with Plasma Exchange and Glucocorticoids.

    Derebail, Vimal K / Falk, Ronald J

    The New England journal of medicine

    2020  Volume 382, Issue 7, Page(s) 671–673

    MeSH term(s) Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis ; Glucocorticoids ; Humans ; Plasma Exchange ; Plasmapheresis
    Chemical Substances Glucocorticoids
    Language English
    Publishing date 2020-02-20
    Publishing country United States
    Document type Editorial ; Comment
    ZDB-ID 207154-x
    ISSN 1533-4406 ; 0028-4793
    ISSN (online) 1533-4406
    ISSN 0028-4793
    DOI 10.1056/NEJMe1917490
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  8. Article ; Online: The Authors Reply.

    Bomback, Andrew S / Derebail, Vimal K / Kshirsagar, Abhijit V

    Kidney international

    2021  Volume 78, Issue 7, Page(s) 708–709

    Language English
    Publishing date 2021-07-26
    Publishing country United States
    Document type Journal Article ; Comment
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1038/ki.2010.277
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  9. Article ; Online: GLP-1 agonists and SGLT-2 inhibitors in adults with sickle cell disease.

    Sun, Ryan / Srivastava, Anand / Derebail, Vimal K / Han, Jin / Molokie, Robert E / Gordeuk, Victor / Saraf, Santosh L

    American journal of hematology

    2024  

    Language English
    Publishing date 2024-04-24
    Publishing country United States
    Document type Letter
    ZDB-ID 196767-8
    ISSN 1096-8652 ; 0361-8609
    ISSN (online) 1096-8652
    ISSN 0361-8609
    DOI 10.1002/ajh.27348
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  10. Article ; Online: Polypharmacy in US Medicare beneficiaries with antineutrophil cytoplasmic antibody vasculitis.

    Cao, Binxin / Hogan, Susan L / Derebail, Vimal K / Ehlert, Alexa / Thorpe, Carolyn T

    Journal of managed care & specialty pharmacy

    2023  Volume 29, Issue 7, Page(s) 770–781

    Abstract: BACKGROUND: ...

    Abstract BACKGROUND:
    MeSH term(s) Humans ; Aged ; United States/epidemiology ; Medicare ; Antibodies, Antineutrophil Cytoplasmic ; Retrospective Studies ; Churg-Strauss Syndrome ; Granulomatosis with Polyangiitis
    Chemical Substances Antibodies, Antineutrophil Cytoplasmic
    Language English
    Publishing date 2023-06-21
    Publishing country United States
    Document type Journal Article
    ISSN 2376-1032
    ISSN (online) 2376-1032
    DOI 10.18553/jmcp.2023.29.7.770
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