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Article ; Online: Microbes mediated immunogenic cell death in cancer immunotherapy

Huang, Jumin / Duan, Fugang / Xie, Chun / Chui, Kawai / Zhang, Yizhong / Wang, Yuwei / Tang, Yu‐Ping / Leung, Elaine Lai‐Han

Immunological Reviews. 2024 Jan., v. 321, no. 1 p.128-142

2024

Abstract: Immunogenic cell death (ICD) is one of the 12 distinct cell death forms, which can trigger immune system to fight against cancer cells. During ICD, a number of cellular changes occur that can stimulate an immune response, including the release of ... ...

Abstract	Immunogenic cell death (ICD) is one of the 12 distinct cell death forms, which can trigger immune system to fight against cancer cells. During ICD, a number of cellular changes occur that can stimulate an immune response, including the release of molecules called damage‐associated molecular patterns (DAMPs), signaling to immune cells to recognize and attack cancer cells. By virtue of their pivotal role in immune surveillance, ICD‐based drug development has been a new approach to explore novel therapeutic combinations and personalized strategies in cancer therapy. Several small molecules and microbes can induce ICD‐relevant signals and cause cancer cell death. In this review, we highlighted the role of microbe‐mediate ICD in cancer immunotherapy and described the mechanisms through which microbes might serve as ICD inducers in cancer treatment. We also discussed current attempts to combine microbes with chemotherapy regimens or immune checkpoint inhibitors (ICIs) in the treatment of cancer patients. We surmise that manipulation of microbes may guide personalized therapeutic interventions to facilitate anticancer immune response.
Keywords	cancer therapy ; cell death ; drug development ; drug therapy ; immune response ; immune system ; immunotherapy ; monitoring ; neoplasm cells
Language	English
Dates of publication	2024-01
Size	p. 128-142.
Publishing place	John Wiley & Sons, Ltd
Document type	Article ; Online
Note	REVIEW
ZDB-ID	391796-4
ISSN	1600-065X ; 0105-2896
ISSN (online)	1600-065X
ISSN	0105-2896
DOI	10.1111/imr.13261
Database	NAL-Catalogue (AGRICOLA)

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Article ; Online: Microbes mediated immunogenic cell death in cancer immunotherapy.

Huang, Jumin / Duan, Fugang / Xie, Chun / Xu, Jiahui / Zhang, Yizhong / Wang, Yuwei / Tang, Yu-Ping / Leung, Elaine Lai-Han

Immunological reviews

2023 Volume 321, Issue 1, Page(s) 128–142

Abstract	Immunogenic cell death (ICD) is one of the 12 distinct cell death forms, which can trigger immune system to fight against cancer cells. During ICD, a number of cellular changes occur that can stimulate an immune response, including the release of molecules called damage-associated molecular patterns (DAMPs), signaling to immune cells to recognize and attack cancer cells. By virtue of their pivotal role in immune surveillance, ICD-based drug development has been a new approach to explore novel therapeutic combinations and personalized strategies in cancer therapy. Several small molecules and microbes can induce ICD-relevant signals and cause cancer cell death. In this review, we highlighted the role of microbe-mediate ICD in cancer immunotherapy and described the mechanisms through which microbes might serve as ICD inducers in cancer treatment. We also discussed current attempts to combine microbes with chemotherapy regimens or immune checkpoint inhibitors (ICIs) in the treatment of cancer patients. We surmise that manipulation of microbes may guide personalized therapeutic interventions to facilitate anticancer immune response.
MeSH term(s)	Humans ; Immunogenic Cell Death ; Antineoplastic Agents/therapeutic use ; Neoplasms ; Cell Death ; Immunotherapy
Chemical Substances	Antineoplastic Agents
Language	English
Publishing date	2023-08-08
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	391796-4
ISSN	1600-065X ; 0105-2896
ISSN (online)	1600-065X
ISSN	0105-2896
DOI	10.1111/imr.13261
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Article ; Online: A novel strategy for identifying biomarker in serum of patient with COVID-19 using immune complex.

Duan, Fugang / Wang, Yifan / Chen, Taoyu / Zhu, Zhu / Yu, Meng / Dai, Hui / Zheng, Shangen / Lu, Yinying / Li, Tingting / Qiu, Xiaoyan

Signal transduction and targeted therapy

2022 Volume 7, Issue 1, Page(s) 63

MeSH term(s)	Antibodies, Viral/blood ; Antigen-Antibody Complex/blood ; Antigens, Viral/blood ; Antigens, Viral/genetics ; Antigens, Viral/immunology ; Biomarkers/blood ; COVID-19/blood ; COVID-19/diagnosis ; COVID-19/immunology ; COVID-19/virology ; Case-Control Studies ; Chromatography, Affinity ; Glycoproteins/blood ; Glycoproteins/genetics ; Glycoproteins/immunology ; Humans ; Immune Sera/chemistry ; Immunoglobulin A/blood ; Immunoglobulin G/blood ; SARS-CoV-2/immunology ; Tandem Mass Spectrometry
Chemical Substances	Antibodies, Viral ; Antigen-Antibody Complex ; Antigens, Viral ; Biomarkers ; Glycoproteins ; Immune Sera ; Immunoglobulin A ; Immunoglobulin G ; LRG1 protein, human
Language	English
Publishing date	2022-02-28
Publishing country	England
Document type	Letter ; Research Support, Non-U.S. Gov't
ZDB-ID	2886872-9
ISSN	2059-3635 ; 2095-9907
ISSN (online)	2059-3635
ISSN	2095-9907
DOI	10.1038/s41392-022-00909-z
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Article ; Online: Analysis of TCR Repertoire by High-Throughput Sequencing Indicates the Feature of T Cell Immune Response after SARS-CoV-2 Infection.

Wang, Yifan / Duan, Fugang / Zhu, Zhu / Yu, Meng / Jia, Xiaodong / Dai, Hui / Wang, Pingzhang / Qiu, Xiaoyan / Lu, Yinying / Huang, Jing

Cells

2021 Volume 11, Issue 1

Abstract: Coronavirus disease 2019 (COVID-19) is a global infectious disease caused by the SARS-CoV-2 coronavirus. T cells play an essential role in the body's fighting against the virus invasion, and the T cell receptor (TCR) is crucial in T cell-mediated virus ... ...

Abstract	Coronavirus disease 2019 (COVID-19) is a global infectious disease caused by the SARS-CoV-2 coronavirus. T cells play an essential role in the body's fighting against the virus invasion, and the T cell receptor (TCR) is crucial in T cell-mediated virus recognition and clearance. However, little has been known about the features of T cell response in convalescent COVID-19 patients. In this study, using 5'RACE technology and PacBio sequencing, we analyzed the TCR repertoire of COVID-19 patients after recovery for 2 weeks and 6 months compared with the healthy donors. The TCR clustering and CDR3 annotation were exploited to discover groups of patient-specific TCR clonotypes with potential SARS-CoV-2 antigen specificities. We first identified CD4
MeSH term(s)	Aged ; Amino Acid Sequence ; CD4-Positive T-Lymphocytes/immunology ; CD4-Positive T-Lymphocytes/metabolism ; CD4-Positive T-Lymphocytes/virology ; CD8-Positive T-Lymphocytes/immunology ; CD8-Positive T-Lymphocytes/metabolism ; CD8-Positive T-Lymphocytes/virology ; COVID-19/immunology ; COVID-19/metabolism ; COVID-19/virology ; Cells, Cultured ; Complementarity Determining Regions/genetics ; Complementarity Determining Regions/immunology ; Convalescence ; Female ; High-Throughput Nucleotide Sequencing/methods ; Humans ; Immunity/immunology ; Male ; Middle Aged ; Patient Acuity ; Receptors, Antigen, T-Cell/genetics ; Receptors, Antigen, T-Cell/immunology ; Receptors, Antigen, T-Cell/metabolism ; Receptors, Antigen, T-Cell, alpha-beta/genetics ; Receptors, Antigen, T-Cell, alpha-beta/immunology ; Receptors, Antigen, T-Cell, alpha-beta/metabolism ; SARS-CoV-2/immunology ; SARS-CoV-2/physiology ; T-Lymphocytes/immunology ; T-Lymphocytes/metabolism ; T-Lymphocytes/virology
Chemical Substances	Complementarity Determining Regions ; Receptors, Antigen, T-Cell ; Receptors, Antigen, T-Cell, alpha-beta
Language	English
Publishing date	2021-12-27
Publishing country	Switzerland
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2661518-6
ISSN	2073-4409 ; 2073-4409
ISSN (online)	2073-4409
ISSN	2073-4409
DOI	10.3390/cells11010068
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Article ; Online: B-Cell Receptor Features and Database Establishment in Recovered COVID-19 Patients by Combining 5'-RACE with PacBio Sequencing.

Zhu, Zhu / Wang, Pingzhang / Jia, Xiaodong / Yu, Meng / Yan, Huige / Liu, Lei / Liu, Wanbing / Zheng, Yaqiong / Kou, Guomei / Wang, Jie / Xu, Weiyan / Huang, Jing / Duan, Fugang / Lu, Fengmin / Fu, Ning / Zhang, Ning / Lu, Yingying / Dai, Hui / Zheng, Shangen /

Qiu, Xiaoyan

Frontiers in bioscience (Landmark edition)

2022 Volume 28, Issue 2, Page(s) 40

Abstract: Background: Antibodies induced by viral infection can not only prevent subsequent virus infection, but can also mediate pathological injury following infection. Therefore, understanding the B-cell receptor (BCR) repertoire of either specific ... ...

Abstract	Background: Antibodies induced by viral infection can not only prevent subsequent virus infection, but can also mediate pathological injury following infection. Therefore, understanding the B-cell receptor (BCR) repertoire of either specific neutralizing or pathological antibodies from patients convalescing from Coronavirus disease 2019 (COVID-19) infection is of benefit for the preparation of therapeutic or preventive antibodies, and may provide insight into the mechanisms of COVID-19 pathological injury. Methods: In this study, we used a molecular approach of combining 5' Rapid Amplification of cDNA Ends (5'-RACE) with PacBio sequencing to analyze the BCR repertoire of all 5 Results: We observed numerous BCR clonotypes within most COVID-19 patients, but not in healthy controls, which validates the association of the disease with a prototypical immune response. In addition, many clonotypes were found to be frequently shared between different patients or different classes of antibodies. Conclusions: These convergent clonotypes provide a resource to identify potential therapeutic/prophylactic antibodies, or identify antibodies associated with pathological effects following infection with SARS-CoV-2.
MeSH term(s)	Humans ; COVID-19 ; SARS-CoV-2 ; Receptors, Antigen, B-Cell/genetics ; Antibodies ; B-Lymphocytes
Chemical Substances	Receptors, Antigen, B-Cell ; Antibodies
Language	English
Publishing date	2022-11-06
Publishing country	Singapore
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2704569-9
ISSN	2768-6698 ; 2768-6698
ISSN (online)	2768-6698
ISSN	2768-6698
DOI	10.31083/j.fbl2802040
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Article: Krukovine Suppresses KRAS-Mutated Lung Cancer Cell Growth and Proliferation by Inhibiting the RAF-ERK Pathway and Inactivating AKT Pathway.

Lai, Huanling / Wang, Yuwei / Duan, Fugang / Li, Ying / Jiang, Zebo / Luo, Lianxiang / Liu, Liang / Leung, Elaine L H / Yao, Xiaojun

Frontiers in pharmacology

2018 Volume 9, Page(s) 958

Abstract: Oncogenic activation of the KRAS gene via point mutations occurs in 20-30% of patients with non-small cell lung cancer (NSCLC). The RAS-RAF-ERK and RAS-PI3K-AKT pathways are the major hyper-activated downstream pathways in RAS mutation, which promotes ... ...

Abstract	Oncogenic activation of the KRAS gene via point mutations occurs in 20-30% of patients with non-small cell lung cancer (NSCLC). The RAS-RAF-ERK and RAS-PI3K-AKT pathways are the major hyper-activated downstream pathways in RAS mutation, which promotes the unlimited lifecycle of cancer cells and their metastasis in humans. However, the success of targeted therapy is restricted by many factors. Herein, we show a new pharmacological KRAS signaling inhibitor krukovine, which is a small molecular bisbenzylisoquinoline alkaloid, isolated from the bark of
Language	English
Publishing date	2018-08-22
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2587355-6
ISSN	1663-9812
ISSN	1663-9812
DOI	10.3389/fphar.2018.00958
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Article ; Online: miR‑20b promotes growth of non‑small cell lung cancer through a positive feedback loop of the Wnt/β‑catenin signaling pathway.

Ren, Tao / Fan, Xing-Xing / Wang, Mei-Fang / Duan, Fu-Gang / Wei, Chun-Li / Li, Run-Ze / Jiang, Ze-Bo / Wang, Yu-Wei / Yao, Xiao-Jun / Chen, Ming-Wei / Tang, Yi-Jun / Leung, Elaine Lai-Han

International journal of oncology

2019 Volume 56, Issue 2, Page(s) 470–479

Abstract: microRNAs (miRNAs or miRs) are endogenous noncoding single‑stranded RNA molecules that can regulate gene expression by targeting the 3'‑untranslated region and play an important role in many biological and pathological processes, such as inflammation and ...

Abstract	microRNAs (miRNAs or miRs) are endogenous noncoding single‑stranded RNA molecules that can regulate gene expression by targeting the 3'‑untranslated region and play an important role in many biological and pathological processes, such as inflammation and cancer. In this study, we found that miR‑20b was significantly increased in human non‑small cell lung cancer (NSCLC) cell lines and patient tissues, suggesting that it may possess a carcinogenic role in lung cancer. This miRNA promoted the proliferation, migration and invasion of NSCLC cells by targeting and downregulating the expression of adenomatous polyposis coli (APC), which is a negative regulator of the canonical Wnt signaling pathway. Wnt signaling activation may increase transcription of miR‑20b. Therefore, miR‑20b and canonical Wnt signaling were coupled through a feed‑forward positive feedback loop, forming a biological regulatory circuit. Finally, an in vivo investigation further demonstrated that an increase in miR‑20b promoted the growth of cancer cells. Overall, our findings offer evidence that miR‑20b may contribute to the development of NSCLC by inhibiting APC via the canonical Wnt signaling pathway.
MeSH term(s)	Adenomatous Polyposis Coli Protein/genetics ; Aged ; Animals ; Carcinoma, Non-Small-Cell Lung/blood ; Carcinoma, Non-Small-Cell Lung/genetics ; Carcinoma, Non-Small-Cell Lung/pathology ; Carcinoma, Non-Small-Cell Lung/surgery ; Case-Control Studies ; Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; Feedback, Physiological ; Female ; Gene Expression Regulation, Neoplastic ; Healthy Volunteers ; Humans ; Lung/pathology ; Lung/surgery ; Lung Neoplasms/blood ; Lung Neoplasms/genetics ; Lung Neoplasms/pathology ; Lung Neoplasms/surgery ; Male ; Mice ; MicroRNAs/metabolism ; Middle Aged ; Neoplasm Invasiveness/genetics ; Pneumonectomy ; Wnt Signaling Pathway/genetics ; Xenograft Model Antitumor Assays
Chemical Substances	APC protein, human ; Adenomatous Polyposis Coli Protein ; MIRN20b microRNA, human ; MicroRNAs
Language	English
Publishing date	2019-12-13
Publishing country	Greece
Document type	Journal Article
ZDB-ID	1154403-x
ISSN	1791-2423 ; 1019-6439
ISSN (online)	1791-2423
ISSN	1019-6439
DOI	10.3892/ijo.2019.4940
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Article ; Online: Prognostic significance of tumor poliovirus receptor and CTLA4 expression in patients with surgically resected non-small-cell lung cancer.

You, Hui / Zhang, Yi-Zhong / Lai, Huan-Ling / Li, Dan / Liu, Yu-Quan / Li, Run-Ze / Khan, Imran / Hsiao, Wendy Wen-Lun / Duan, Fu-Gang / Fan, Xing-Xing / Yao, Xiao-Jun / Cao, Ya-Bing / Wu, Qi-Biao / Leung, Elaine Lai-Han / Wang, Mei-Fang

Journal of cancer research and clinical oncology

2020 Volume 146, Issue 6, Page(s) 1441–1450

Abstract: Introduction: Poliovirus receptor (PVR) is a tumor promoter and a regulatory checkpoint that enhances immunosuppression. We investigated PVR expression by applying immunohistochemistry (IHC) staining. A positive association existed between PVR ... ...

Abstract	Introduction: Poliovirus receptor (PVR) is a tumor promoter and a regulatory checkpoint that enhances immunosuppression. We investigated PVR expression by applying immunohistochemistry (IHC) staining. A positive association existed between PVR expression and cytotoxic T lymphocyte-associated antigen 4 (CTLA4) expression in patients with surgically resected non-small-cell lung cancer (NSCLC). PVR expression is a prognosis predictor of lung adenocarcinoma. Purpose: To investigate the prognostic significance of PVR expression and CTLA4 expression for surgically resected NSCLC. Patients and methods: The medical records of 108 Chinese patients with primary NSCLC who underwent surgery were retrospectively reviewed. The expression of PVR and CTLA4 were measured through IHC. Clinical characteristics, the association between PVR and CTLA4, and the prognostic significance of PVR were analyzed. Results: A significant positive association was observed between PVR and CTLA4 expression in NSCLC (P = 0.016). PVR had a high positive rate among females, nonsmokers, and patients with adenocarcinoma and advanced lung cancer. The overall survival (OS) of patients with negative PVR expression was significantly longer than that of patients with positive PVR expression (P = 0.049), especially among females (P = 0.03) and nonsmokers (P = 0.025). Multivariate analysis results showed that advanced tumor stage and PVR expression were independent prognosis predictors of poor OS. Conclusion: PVR can potentially serve as a prognostic predictor and biomarker for NSCLC and cancer anti-CTLA4 immunotherapy response.
MeSH term(s)	Adult ; Aged ; Biomarkers, Tumor/metabolism ; CTLA-4 Antigen/metabolism ; Carcinoma, Non-Small-Cell Lung/metabolism ; Carcinoma, Non-Small-Cell Lung/pathology ; Carcinoma, Non-Small-Cell Lung/surgery ; Female ; Humans ; Lung Neoplasms/metabolism ; Lung Neoplasms/pathology ; Lung Neoplasms/surgery ; Male ; Middle Aged ; Prognosis ; Receptors, Virus/metabolism ; Retrospective Studies
Chemical Substances	Biomarkers, Tumor ; CTLA-4 Antigen ; CTLA4 protein, human ; Receptors, Virus ; poliovirus receptor
Language	English
Publishing date	2020-04-04
Publishing country	Germany
Document type	Journal Article
ZDB-ID	134792-5
ISSN	1432-1335 ; 0171-5216 ; 0084-5353 ; 0943-9382
ISSN (online)	1432-1335
ISSN	0171-5216 ; 0084-5353 ; 0943-9382
DOI	10.1007/s00432-020-03189-8
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Article: Gossypol Inhibits Non-small Cell Lung Cancer Cells Proliferation by Targeting EGFR

Wang, Yuwei / Lai, Huanling / Fan, Xingxing / Luo, Lianxiang / Duan, Fugang / Jiang, Zebo / Wang, Qianqian / Leung, Elaine Lai Han / Liu, Liang / Yao, Xiaojun

Frontiers in pharmacology

2018 Volume 9, Page(s) 728

Abstract: Background: ...

Abstract	Background:
Language	English
Publishing date	2018-07-09
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2587355-6
ISSN	1663-9812
ISSN	1663-9812
DOI	10.3389/fphar.2018.00728
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Article ; Online: Proscillaridin A induces apoptosis and suppresses non-small-cell lung cancer tumor growth via calcium-induced DR4 upregulation.

Li, Run-Ze / Fan, Xing-Xing / Duan, Fu-Gang / Jiang, Ze-Bo / Pan, Hu-Dan / Luo, Lian-Xiang / Zhou, Yan-Ling / Li, Ying / Yao, Ying-Jia / Yao, Xiao-Jun / Leung, Elaine Lai-Han / Liu, Liang

Cell death & disease

2018 Volume 9, Issue 6, Page(s) 696

Abstract: Non-small-cell lung cancer (NSCLC) is the predominant histological type of lung cancer and is characterized by the highest mortality and incidence rates among these types of malignancies. Cardiac glycosides, a class of natural products, have been ... ...

Abstract	Non-small-cell lung cancer (NSCLC) is the predominant histological type of lung cancer and is characterized by the highest mortality and incidence rates among these types of malignancies. Cardiac glycosides, a class of natural products, have been identified as a potential type of chemotherapeutic agent. This study aims to investigate the anti-cancer effects and the mechanisms of action of Proscillaridin A (P.A) in NSCLC cells. In vitro sodium-potassium pump (Na
MeSH term(s)	Acetyl-CoA Carboxylase/metabolism ; Adenylate Kinase/metabolism ; Apoptosis/drug effects ; Calcium/pharmacology ; Carcinoma, Non-Small-Cell Lung/metabolism ; Carcinoma, Non-Small-Cell Lung/pathology ; Cell Line, Tumor ; Cell Proliferation/drug effects ; ErbB Receptors/genetics ; ErbB Receptors/metabolism ; Humans ; JNK Mitogen-Activated Protein Kinases/metabolism ; Lung Neoplasms/metabolism ; Lung Neoplasms/pathology ; Models, Biological ; Mutation/genetics ; NF-kappa B/metabolism ; Phosphorylation/drug effects ; Proscillaridin/chemistry ; Proscillaridin/pharmacology ; Receptors, TNF-Related Apoptosis-Inducing Ligand/metabolism ; Signal Transduction/drug effects ; Sodium-Potassium-Exchanging ATPase/metabolism ; Tumor Stem Cell Assay ; Up-Regulation/drug effects ; Xenograft Model Antitumor Assays
Chemical Substances	NF-kappa B ; Receptors, TNF-Related Apoptosis-Inducing Ligand ; EGFR protein, human (EC 2.7.10.1) ; ErbB Receptors (EC 2.7.10.1) ; JNK Mitogen-Activated Protein Kinases (EC 2.7.11.24) ; Adenylate Kinase (EC 2.7.4.3) ; Acetyl-CoA Carboxylase (EC 6.4.1.2) ; Sodium-Potassium-Exchanging ATPase (EC 7.2.2.13) ; Proscillaridin (KC6BL281EN) ; Calcium (SY7Q814VUP)
Language	English
Publishing date	2018-06-13
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2541626-1
ISSN	2041-4889 ; 2041-4889
ISSN (online)	2041-4889
ISSN	2041-4889
DOI	10.1038/s41419-018-0733-4
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