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Book ; Article ; Online: De plantas, sociedades y pandemias (antropología desde casa)

Jiménez-Escobar, David

2020

Abstract: Con ejemplos puntuales, David Jiménez-Escobar –Doctor en Ciencias Biológicas del Instituto de Antropología de Córdoba Consejo Nacional de Investigaciones Científicas y Técnicas de Argentina –Museo de Antropología de la Universidad Nacional de Córdoba –, ... ...

Abstract	Con ejemplos puntuales, David Jiménez-Escobar –Doctor en Ciencias Biológicas del Instituto de Antropología de Córdoba Consejo Nacional de Investigaciones Científicas y Técnicas de Argentina –Museo de Antropología de la Universidad Nacional de Córdoba –, destaca el papel que tienen las plantas en nuestras sociedades. Estas protagonistas, algunas veces olvidadas, han construido profundos vínculos a lo largo de miles de años, lazos de convivencia con la humanidad. Las pandemias traen reflexiones y en otras ocasiones las plantas las soluciones. Fil: Jiménez-Escobar, David. Universidad Nacional de Córdoba. Facultad de Filosofía y Humanidades. Museo de Antropología; Argentina. Fil: Jiménez-Escobar, David. Consejo Nacional de Investigaciones Científicas y Técnicas. Instituto de Antropología de Córdoba. Argentina.
Keywords	COVID-19 ; Coronavirus ; SARS-CoV-2 ; Pandemia ; Aislamiento social ; Cuarentena ; Plantas ; Curación ; Quina ; Cinchona spp ; Paludismo ; Malaria ; Amapola ; Adormidera ; Papaver somniferum ; Sustancias opiáceas ; Hambruna irlandesa ; Hambruna escocesa ; Década de 1840 ; Plaga de la papa ; Solanum tuberosum ; Hongo ; Phytophthora infestans ; covid19
Language	Spanish
Publishing date	2020-04-13
Publisher	Universidad Nacional de Córdoba. Facultad de Filosofía y Humanidades. Consejo Nacional de Investigaciones Científicas y Técnicas de Argentina. Instituto de Antropología de Córdoba
Publishing country	ar
Document type	Book ; Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article ; Online: High-Density IgG4+ Plasma Cells Infiltration Is Associated With Fibroplasia in Fibrostenotic Crohn's Disease.

Escobar, David / Bushara, Omar / Sun, Leyu / Liao, Jie / Yang, Guang-Yu

International journal of surgical pathology

2023 Volume 31, Issue 6, Page(s) 1085–1092

Abstract: Transmural fibrosis and stricture formation are key pathogenic processes for Crohn's disease that underlies clinical refractoriness, resulting in severe morbidity. The mechanisms for fibroplasia in Crohn's are not fully elucidated. In this study, we ... ...

Abstract	Transmural fibrosis and stricture formation are key pathogenic processes for Crohn's disease that underlies clinical refractoriness, resulting in severe morbidity. The mechanisms for fibroplasia in Crohn's are not fully elucidated. In this study, we identified a cohort of refractory Crohn's disease with surgically resected bowel specimens including cases with bowel stricture and age-/sex-matched refractory disease without bowel stricture. Via immunohistochemistry, density and distribution of IgG4+ plasma cells in resected cases were analyzed. The histologic severity of fibrosis and association with gross evidence of stricture formation and IgG4+ plasma cells were comprehensively analyzed. Our results showed that density of IgG4+ plasma cells/high-power field (IgG4+ PCs/HPF) was significantly associated with increasing histologic fibrosis score (15 IgG4+ PCs/HPF in specimens with fibrosis score 0 vs 31 IgG4+ PC/HPF in fibrosis score 2 and 3,
MeSH term(s)	Humans ; Crohn Disease/complications ; Crohn Disease/pathology ; Constriction, Pathologic/pathology ; Immunoglobulin G ; Plasma Cells/pathology ; Inflammation/pathology ; Intestinal Obstruction/etiology ; Fibrosis
Chemical Substances	Immunoglobulin G
Language	English
Publishing date	2023-03-03
Publishing country	United States
Document type	Journal Article
ZDB-ID	1336393-1
ISSN	1940-2465 ; 1066-8969
ISSN (online)	1940-2465
ISSN	1066-8969
DOI	10.1177/10668969231152242
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Article: Microcomputed tomography visualization and quantitation of the pulmonary arterial microvascular tree in mouse models of chronic lung disease.

Schneider, Ben / Kopf, Katrina W / Mason, Emma / Dawson, Maggie / Coronado Escobar, David / Majka, Susan M

Pulmonary circulation

2023 Volume 13, Issue 3, Page(s) e12279

Abstract: Pulmonary vascular dysfunction is characterized by remodeling and loss of microvessels in the lung and is a major manifestation of chronic lung diseases (CLD). In murine models of CLD, the small arterioles and capillaries are the first and most prevalent ...

Abstract	Pulmonary vascular dysfunction is characterized by remodeling and loss of microvessels in the lung and is a major manifestation of chronic lung diseases (CLD). In murine models of CLD, the small arterioles and capillaries are the first and most prevalent vessels that are affected by pruning and remodeling. Thus, visualization of the pulmonary arterial vasculature in three dimensions is essential to define pruning and remodeling both temporally and spatially and its role in the pathogenesis of CLD, aging, and tissue repair. To this end, we have developed a novel method to visualize and quantitate the murine pulmonary arterial circulation using microcomputed tomography (µCT) imaging. Using this perfusion technique, we can quantitate microvessels to approximately 6 µM in diameter. We hypothesize that bleomycin-induced injury would have a significant impact on the arterial vascular structure. As proof of principle, we demonstrated that as a result of bleomycin-induced injury at peak fibrosis, significant alterations in arterial vessel structure were visible in the three-dimensional models as well as quantification. Thus, we have successfully developed a perfusion methodology and complementary analysis techniques, which allows for the reconstruction, visualization, and quantitation of the mouse pulmonary arterial microvasculature in three-dimensions. This tool will further support the examination and understanding of angiogenesis during the development of CLD as well as repair following injury.
Language	English
Publishing date	2023-08-27
Publishing country	United States
Document type	Journal Article
ZDB-ID	2638089-4
ISSN	2045-8940 ; 2045-8932
ISSN (online)	2045-8940
ISSN	2045-8932
DOI	10.1002/pul2.12279
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Article ; Online: Influence of stroke rate on coordination and sprint performance in elite male and female swimmers.

Simbaña-Escobar, David / Hellard, Philippe / Seifert, Ludovic

Scandinavian journal of medicine & science in sports

2020 Volume 30, Issue 11, Page(s) 2078–2091

Abstract: This study investigated the effects of gender and the manipulation of the preferred stroke rate on swimming performance and arm coordination in elite front crawl swimmers. Nineteen swimmers performed a dual task, that is, imposed stroke rate and maximal ... ...

Abstract	This study investigated the effects of gender and the manipulation of the preferred stroke rate on swimming performance and arm coordination in elite front crawl swimmers. Nineteen swimmers performed a dual task, that is, imposed stroke rate and maximal speed. They swam nine 25-m trials at maximal speed twice: one trial at the preferential stroke rate, one trial at maximal stroke rate and seven trials at stroke rates between 41 and 59 cycles/min imposed by an Aquapacer. Stroke rate, arm stroke phases, and arm coordination were computed from an inertial measurement unit on each forearm and one on the sacrum. Time on the 25-m was recorded to assess swimming speed. Results indicated that the error between the imposed and performed stroke rates was lowest at the preferred stroke rate for women. An increase in stroke rate led to an increase in swimming speed and the index of coordination, but these changes could be influenced by the preferred stroke rate. Individual analysis revealed that some swimmers exhibited higher flexibility (larger range of stroke rate) around their preferred stroke rate. This stroke rate flexibility appeared more functional in swimmers who reached higher speeds when swimming at the maximal stroke rate than at the preferred stroke rate.
MeSH term(s)	Acceleration ; Arm/physiology ; Athletic Performance/physiology ; Biomechanical Phenomena ; Competitive Behavior/physiology ; Female ; Humans ; Linear Models ; Male ; Motor Skills/physiology ; Sex Factors ; Swimming/physiology ; Young Adult
Language	English
Publishing date	2020-08-13
Publishing country	Denmark
Document type	Journal Article
ZDB-ID	1077418-x
ISSN	1600-0838 ; 0905-7188
ISSN (online)	1600-0838
ISSN	0905-7188
DOI	10.1111/sms.13786
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Article ; Online: Race analysis of the men's 50 m events at the 2021 LEN European Championships.

Morais, Jorge E / Barbosa, Tiago M / Lopes, Tiago / Simbaña-Escobar, David / Marinho, Daniel A

Sports biomechanics

2022 , Page(s) 1–17

Abstract: The aim of this study was to: (i) characterise the stroke kinematics' stability of the male swimmers competing in the four 50 m events at the 2021 European Championships, and; (ii) understand the speed-time relationship in the four race events. All male ... ...

Abstract	The aim of this study was to: (i) characterise the stroke kinematics' stability of the male swimmers competing in the four 50 m events at the 2021 European Championships, and; (ii) understand the speed-time relationship in the four race events. All male swimmers who participated in the 50 m events (backstroke: 78 swimmers; breaststroke: 79 swimmers; butterfly: 89 swimmers; freestyle: 95 swimmers) were evaluated. In each swimming stroke swimmers were split in two groups (better and poorer performances). Significant variances (p < 0.05) were observed in both groups in all variables and for all swimming strokes. Swimming speed was the variable with the highest variance in both groups and strokes. Overall, better swimmers presented a low to high normative stability, and poorer swimmers a moderate-to-high. Speed-time curve fitting for all swimming strokes and groups suggested a cubic relationship. It can be considered that elite male swimmers racing 50 m sprint events at major competitions present an all-out trend. The present data provide coaches with substantial information about the main trend in the 50 m sprint events, specifically in each section of the race.
Language	English
Publishing date	2022-09-27
Publishing country	England
Document type	Journal Article
ISSN	1752-6116
ISSN (online)	1752-6116
DOI	10.1080/14763141.2022.2125430
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Article ; Online: The Possible Pathogenic Role of IgG4-Producing Plasmablasts in Stricturing Crohn's Disease.

Bushara, Omar / Escobar, David Joseph / Weinberg, Samuel Edward / Sun, Leyu / Liao, Jie / Yang, Guang-Yu

Pathobiology : journal of immunopathology, molecular and cellular biology

2022 Volume 89, Issue 4, Page(s) 187–197

Abstract: Background: Crohn's disease (CD) is a condition on the spectrum of inflammatory bowel disease that affects up to 20 people per 100,000 in the US annually, and with incidence increasing. One of the most significant sources of morbidity in CD is the ... ...

Abstract	Background: Crohn's disease (CD) is a condition on the spectrum of inflammatory bowel disease that affects up to 20 people per 100,000 in the US annually, and with incidence increasing. One of the most significant sources of morbidity in CD is the formation of strictures, with resultant intestinal blockage a common indication for hospitalization and surgical intervention in these patients. The pathophysiology of stricture formation is not fully understood. However, the fibroplasia that leads to fibrostenotic stricture formation may have shared pathophysiology with IgG4-related fibrosis. Summary: Initial intestinal inflammation recruits innate immune cells, such as neutrophils, that secrete IL-1β and IL-23, which induces a type 17 CD4+ T-helper T-cell (Th17)-mediated adaptive immune response. These CD4+ Th17 T cells also contribute to inflammation by secreting proinflammatory cytokines such as IL-17 and IL-21. IL-21 recruits and stimulates CD4+ T follicular helper (Tfh) cells, which secrete more IL-21. This causes ectopic germinal center formation, recruiting and stimulating naïve B cells. The IL-17 and IL-21 produced by Th17 cells and Tfh cells also induce IgG4 plasmablast differentiation. Finally, these IgG4-producing plasmablasts secrete platelet-derived growth factor (PDGF), which activates local PDGF-receptor expressing fibroblasts and myofibroblasts, resulting in uncontrolled fibroplasia.
MeSH term(s)	Constriction, Pathologic ; Crohn Disease ; Humans ; Immunoglobulin G ; Inflammation ; Plasma Cells/immunology ; Th17 Cells
Chemical Substances	Immunoglobulin G
Language	English
Publishing date	2022-01-13
Publishing country	Switzerland
Document type	Journal Article ; Review
ZDB-ID	1022703-9
ISSN	1423-0291 ; 1015-2008
ISSN (online)	1423-0291
ISSN	1015-2008
DOI	10.1159/000521259
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Article ; Online: Overexpression of programmed death ligand 1 in refractory inflammatory bowel disease.

Nguyen, Jessica / Finkelman, Brian S / Escobar, David / Xue, Yue / Wolniak, Kristy / Pezhouh, Maryam

Human pathology

2022 Volume 126, Page(s) 19–27

Abstract: Programmed death ligand 1 (PD-L1) dysregulation has been implicated in chronic inflammatory diseases, but its role in regulating intestinal mucosa inflammation is still unclear. The aim of this study was to assess PD-L1 expression in the intestinal ... ...

Abstract	Programmed death ligand 1 (PD-L1) dysregulation has been implicated in chronic inflammatory diseases, but its role in regulating intestinal mucosa inflammation is still unclear. The aim of this study was to assess PD-L1 expression in the intestinal mucosa of patients with refractory inflammatory bowel disease (IBD) compared to controls. We evaluated PD-L1 expression by immunohistochemistry in colectomy specimens of patients with ulcerative colitis (UC) and Crohn disease (CD) compared to controls. PD-L1 expression was assessed in colonic epithelium and inflammatory cells, along with the location of the inflammatory cells expressing PD-L1. All cases were stained with CD3, CD4, CD8, FOXP3, CD20, CD68, and CD90 immunostains to determine the types of cells expressing PD-L1. The UC group showed significantly higher PD-L1 expression in the colonic epithelium than both CD and control groups (both P < 0.001), and CD was also significantly higher than the control group (P = 0.004). Both UC and CD groups showed similar PD-L1 expression in the inflammatory infiltrate but significantly higher than the control group (both P < 0.001). Among both IBD groups, higher IBD activity was associated with higher levels of PD-L1 expression in the colonic epithelium (P < 0.05) and inflammatory infiltrate (P < 0.001). When comparing PD-L1 expression to lineage-specific markers, CD3+, CD4+ T cells, CD68+ macrophages, and CD90+ colonic stromal cells appeared to be expressing PD-L1. These findings implicate a role for PD-L1 in the dysregulation of the immune response in refractory IBD. Further studies are warranted to better understand the role of the immune regulatory pathways in intestinal mucosa.
MeSH term(s)	B7-H1 Antigen/genetics ; B7-H1 Antigen/metabolism ; Colitis, Ulcerative/genetics ; Crohn Disease/genetics ; Humans ; Inflammatory Bowel Diseases ; Intestinal Mucosa
Chemical Substances	B7-H1 Antigen ; CD274 protein, human
Language	English
Publishing date	2022-04-28
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	207657-3
ISSN	1532-8392 ; 0046-8177
ISSN (online)	1532-8392
ISSN	0046-8177
DOI	10.1016/j.humpath.2022.04.011
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Article: A breast cancer classification and immune landscape analysis based on cancer stem-cell-related risk panel.

Hu, Haihong / Zou, Mingxiang / Hu, Hongjuan / Hu, Zecheng / Jiang, Lingxiang / Escobar, David / Zhu, Hongxia / Zhan, Wendi / Yan, Ting / Zhang, Taolan

NPJ precision oncology

2023 Volume 7, Issue 1, Page(s) 130

Abstract: This study sought to identify molecular subtypes of breast cancer (BC) and develop a breast cancer stem cells (BCSCs)-related gene risk score for predicting prognosis and assessing the potential for immunotherapy. Unsupervised clustering based on ... ...

Abstract	This study sought to identify molecular subtypes of breast cancer (BC) and develop a breast cancer stem cells (BCSCs)-related gene risk score for predicting prognosis and assessing the potential for immunotherapy. Unsupervised clustering based on prognostic BCSC genes was used to determine BC molecular subtypes. Core genes of BC subtypes identified by non-negative matrix factorization algorithm (NMF) were screened using weighted gene co-expression network analysis (WGCNA). A risk model based on prognostic BCSC genes was constructed using machine learning as well as LASSO regression and multivariate Cox regression. The tumor microenvironment and immune infiltration were analyzed using ESTIMATE and CIBERSORT, respectively. A CD79A
Language	English
Publishing date	2023-12-08
Publishing country	England
Document type	Journal Article
ISSN	2397-768X
ISSN	2397-768X
DOI	10.1038/s41698-023-00482-w
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Article ; Online: Tailored Coatings for Enhanced Performance of Zinc-Magnesium Alloys in Absorbable Implants.

Hernández-Escobar, David / Pajares-Chamorro, Natalia / Chatzistavrou, Xanthippi / Hankenson, Kurt D / Hammer, Neal D / Boehlert, Carl J

ACS biomaterials science & engineering

2023 Volume 10, Issue 1, Page(s) 338–354

Abstract: Absorbable metals exhibit potential for next-generation temporary medical implants, dissolving safely in the body during tissue healing and regeneration. Their commercial incorporation could substantially diminish the need for additional surgeries and ... ...

Abstract	Absorbable metals exhibit potential for next-generation temporary medical implants, dissolving safely in the body during tissue healing and regeneration. Their commercial incorporation could substantially diminish the need for additional surgeries and complications that are tied to permanent devices. Despite extensive research on magnesium (Mg) and iron (Fe), achieving the optimal combination of mechanical properties, biocompatibility, and controlled degradation rate for absorbable implants remains a challenge. Zinc (Zn) and Zn-based alloys emerged as an attractive alternative for absorbable implants, due to favorable combination of
MeSH term(s)	Humans ; Coated Materials, Biocompatible/pharmacology ; Coated Materials, Biocompatible/chemistry ; Magnesium/pharmacology ; Magnesium/chemistry ; Alloys/pharmacology ; Alloys/chemistry ; Zinc/pharmacology ; Absorbable Implants
Chemical Substances	Coated Materials, Biocompatible ; Magnesium (I38ZP9992A) ; Alloys ; Zinc (J41CSQ7QDS) ; pyrithione zinc (R953O2RHZ5) ; zinc phosphate (1E2MCT2M62)
Language	English
Publishing date	2023-12-18
Publishing country	United States
Document type	Journal Article
ISSN	2373-9878
ISSN (online)	2373-9878
DOI	10.1021/acsbiomaterials.3c01255
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Article ; Online: A novel immune checkpoint-related signature for prognosis and immune analysis in breast cancer.

Hu, Haihong / Yan, Ting / Zhu, Hongxia / Zhan, Wendi / Zhang, Jingdi / Wang, Siyu / Jiang, Lingxiang / Escobar, David / Zhang, Taolan

Clinical and experimental medicine

2023 Volume 23, Issue 8, Page(s) 5139–5159

Abstract: Breast cancer is one of the most prevailing forms of cancer globally. Immunotherapy has demonstrated efficacy in improving the overall survival of breast cancer. The aim of us was to formulate a novel signature predicated on immune checkpoint-related ... ...

Abstract	Breast cancer is one of the most prevailing forms of cancer globally. Immunotherapy has demonstrated efficacy in improving the overall survival of breast cancer. The aim of us was to formulate a novel signature predicated on immune checkpoint-related genes (ICGs) that could anticipate the prognosis and further analyze the immune status of patients with breast cancer. After acquiring data, we pinpointed the definitive ICGs for constructing the prognostic model of breast cancer. We constructed a novel prognostic model and created a fresh risk score called Immune Checkpoint-related Risk Score in breast cancer (ICRSBC). The nomogram was constructed to evaluate the accuracy of the model, and the new web-based tool was created to be more intuitive for predicting prognosis. We also investigated immunotherapy responsiveness and analyzed the tumor mutational burden (TMB) in ICRSBC subgroups. The ICRSBC was found to have significant correlations with the immune environment, immunotherapy responsiveness, and TMB. The expression levels of the 9 ICGs that construct the prognostic model and their promoter methylation levels are significantly different between breast cancer and normal tissues. Furthermore, the mutation profiles, the copy number alterations, and the levels of protein expression also exhibit marked disparities among the 9 ICGs. We have identified and validated a novel signature related to ICGs that is strongly associated with breast cancer progression. This signature enables us to create a risk score for prognosticating the survival and assessing the immune status of individuals affected by breast cancer.
MeSH term(s)	Humans ; Female ; Breast Neoplasms/genetics ; Prognosis ; Nomograms ; Immunotherapy ; Mutation
Language	English
Publishing date	2023-11-06
Publishing country	Italy
Document type	Journal Article
ZDB-ID	2053018-3
ISSN	1591-9528 ; 1591-8890
ISSN (online)	1591-9528
ISSN	1591-8890
DOI	10.1007/s10238-023-01247-2
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