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Article ; Online: A Hybrid Scheme of MCS Selection and Spectrum Allocation for URLLC Traffic under Delay and Reliability Constraints.

Gao, Yuehong / Yang, Haotian / Hong, Xiao / Chen, Lu

2022 Volume 24, Issue 5

Abstract: The Ultra-Reliable Low-Latency Communication (URLLC) is expected to be an important feature of 5G and beyond networks. Supporting URLLC in a resource-efficient manner demands optimal Modulation and Coding Scheme (MCS) selection and spectrum allocation. ... ...

Abstract	The Ultra-Reliable Low-Latency Communication (URLLC) is expected to be an important feature of 5G and beyond networks. Supporting URLLC in a resource-efficient manner demands optimal Modulation and Coding Scheme (MCS) selection and spectrum allocation. This paper presents a study on MCS selection and spectrum allocation to support URLLC. The essential idea is to establish an analytical connection between the delay and reliability requirements of URLLC data transmission and the underlying MCS selection and spectrum allocation. In particular, the connection factors in fundamental aspects of wireless data communication include channel quality, coding and modulation, spectrum allocation and data traffic characteristics. With this connection, MCS selection and spectrum allocation can be efficiently performed based on the delay and reliability requirements of URLLC. Theoretical results in the scenario of a 5G New Radio system are presented, where the Signal-to-Noise Ratio (SNR) thresholds for adaptive MCS selection, data-transmission rate and delay, as well as spectrum allocation under different configurations, including data duplication, are discussed. Simulation results are also obtained and compared with the theoretical results, which validate the analysis and its efficiency.
Language	English
Publishing date	2022-05-20
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2014734-X
ISSN	1099-4300 ; 1099-4300
ISSN (online)	1099-4300
ISSN	1099-4300
DOI	10.3390/e24050727
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article ; Online: Postpartum stress in the first 6 months after delivery: a longitudinal study in Nantong, China.

Wang, Yanchi / Gu, Jian / Gao, Yuehong / Lu, Yi / Zhang, Feng / Xu, Xujuan

BMJ open

2023 Volume 13, Issue 10, Page(s) e073796

Abstract: Objectives: The objective is to to explore the longitudinal change trajectories of postpartum stress and its related factors.: Design: A longitudinal study with follow-ups from 42 days to 6 months after delivery.: Settings and participants: A ... ...

Abstract	Objectives: The objective is to to explore the longitudinal change trajectories of postpartum stress and its related factors. Design: A longitudinal study with follow-ups from 42 days to 6 months after delivery. Settings and participants: A total of 406 postpartum women were recruited at baseline (42 days after delivery) from 6 hospitals in Nantong, Jiangsu Province, China, and followed up at 3 and 6 months. After the follow-ups, 358 postpartum women were retained for further analysis. Methods: Postpartum stress was evaluated using the Maternal Postpartum Stress Scale (MPSS) at baseline (42 days) and 3 and 6 months after delivery. MPSS has three dimensions, such as: personal needs and fatigue, infant nurturing and body changes and sexuality. Postpartum depression and anxiety were measured using the Edinburgh Postnatal Depression Scale and the short-form Depression, Anxiety and Stress Scale, respectively. The MPSS scores were normalised using a rank-based inverse normal transformation. Results: Postpartum stress decreased significantly after 3 months, and postpartum stress reduced further after 6 months. Additionally, the scores for all three dimensions reduced after 6 months, while infant nurturing reduced after both 3 and 6 months. Older age (β=0.028, p=0.049), higher education level (β=0.153, p=0.005) and higher body mass index (BMI) (β=0.027, p=0.008) of the postpartum women were significantly associated with higher postpartum stress levels in corresponding dimensions at 42 days. Older age was also associated with higher postpartum stress at 3 (β=0.030, p=0.033) and 6 months (β=0.050, p<0.001) in the dimension of personal needs and fatigue. Postpartum stress levels were significantly higher in women with depression or anxiety symptoms. Conclusions: Postpartum stress continuously declined from 42 days to 6 months after delivery. Postpartum women with older age, higher education levels, higher BMI and anxiety or depression symptoms should be the target population for early intervention.
MeSH term(s)	Infant ; Female ; Humans ; Longitudinal Studies ; Surveys and Questionnaires ; Postpartum Period ; Depression, Postpartum/epidemiology ; Depression, Postpartum/diagnosis ; China/epidemiology
Language	English
Publishing date	2023-10-21
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2599832-8
ISSN	2044-6055 ; 2044-6055
ISSN (online)	2044-6055
ISSN	2044-6055
DOI	10.1136/bmjopen-2023-073796
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Article ; Online: Family intimacy and adaptability and non-suicidal self-injury: a mediation analysis.

Gao, Yuehong / Wang, Yanchi / Wang, Zhiping / Ma, Mingzhen / Li, Hongjiao / Wang, Jinhong / Liu, Jianan / Qian, Huaying / Zhu, Ping / Xu, Xujuan

BMC psychiatry

2024 Volume 24, Issue 1, Page(s) 210

Abstract: Background: Current research has been focusing on non-suicidal self-injury (NSSI) behaviors among adolescents with depression. Although family intimacy and adaptability are considered protective factors for NSSI, evidence supporting this relationship is ...

Abstract	Background: Current research has been focusing on non-suicidal self-injury (NSSI) behaviors among adolescents with depression. Although family intimacy and adaptability are considered protective factors for NSSI, evidence supporting this relationship is lacking. Objective: This study aims to examine the mechanisms operating in the relationship between family intimacy and adaptability and NSSI behaviors among adolescents. Methods: A self-administered general demographic information questionnaire, the Behavioral Functional Assessment Scale for Non-Suicidal Self-Injury, the Family Intimacy and Adaptability Scale, the Connor-Davidson Resilience Scale, and the Self-Assessment of Depression Scale were distributed among adolescents with depression in three tertiary hospitals in Jiangsu Province. Results: The relationship between family intimacy and adaptability and NSSI was assessed among 596 adolescents with depression. The results revealed the following: (1) Family intimacy and adaptability were negatively correlated with NSSI behavior. (2) Psychological resilience and depression levels acted as chain mediators in the relationship between family intimacy and adaptability and NSSI behavior. Conclusions: Enhancing psychological resilience, controlling depressive symptoms, and reducing depression severity among adolescents by improving their family intimacy and adaptability are conducive to preventing and mitigating their NSSI behaviors.
MeSH term(s)	Adolescent ; Humans ; Mediation Analysis ; Self-Injurious Behavior/psychology ; Psychological Tests ; Resilience, Psychological
Language	English
Publishing date	2024-03-18
Publishing country	England
Document type	Journal Article
ZDB-ID	2050438-X
ISSN	1471-244X ; 1471-244X
ISSN (online)	1471-244X
ISSN	1471-244X
DOI	10.1186/s12888-024-05642-1
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Book ; Online: Reconfigurable Intelligent Surface & Edge -- An Introduction of an EM manipulation structure on obstacles' edge

Xiang, Tianqi / Jiang, Zhiwei / Hong, Weijun / Zhang, Xin / Gao, Yuehong

2023

Abstract: Reconfigurable Intelligent Surface (RIS) or metasurface is one of the important enabling technologies in mobile cellular networks that can effectively enhance the signal coverage performance in obstructed regions, and it is generally deployed on surfaces ...

Abstract	Reconfigurable Intelligent Surface (RIS) or metasurface is one of the important enabling technologies in mobile cellular networks that can effectively enhance the signal coverage performance in obstructed regions, and it is generally deployed on surfaces different from obstacles to redirect electromagnetic (EM) waves by reflection, or covered on objects' surfaces to manipulate EM waves by refraction. In this paper, Reconfigurable Intelligent Surface & Edge (RISE) is proposed to extend RIS' abilities of reflection and refraction over surfaces to diffraction around obstacles' edge for better adaptation to specific coverage scenarios. Based on that, this paper analyzes the performance of several different deployment locations and EM manipulation structure designs for different coverage scenarios. Then a novel EM manipulation structure deployed at the obstacles' edge is proposed to achieve static EM environment modification. Simulations validate the preference of the schemes for different scenarios and the new structure achieves better coverage performance than other typical structures in the static scheme.
Keywords	Electrical Engineering and Systems Science - Signal Processing
Subject code	629
Publishing date	2023-11-03
Publishing country	us
Document type	Book ; Online
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Book ; Online: Study on MCS Selection and Spectrum Allocation for URLLC Traffic under Delay and Reliability Constraint in 5G Network

Gao, Yuehong / Sun, Changhao / Zhang, Xiaonan / Hong, Xiao

2021

Abstract: To support Ultra-Reliable and Low Latency Communications (URLLC) is an essential character of the 5th Generation (5G) communication system. Unlike the other two use cases defined in 5G, e.g. enhanced Mobile Broadband (eMBB) and massive Machine Type ... ...

Abstract	To support Ultra-Reliable and Low Latency Communications (URLLC) is an essential character of the 5th Generation (5G) communication system. Unlike the other two use cases defined in 5G, e.g. enhanced Mobile Broadband (eMBB) and massive Machine Type Communications (mMTC), URLLC traffic has strict delay and reliability requirement. In this paper, an analysis model for URLLC traffic is proposed from the generation of a URLLC traffic until its transmission over a wireless channel, where channel quality, coding scheme with finite coding length, modulation scheme and allocated spectrum resource are taken into consideration. Then, network calculus analysis is applied to derive the delay guarantee for periodical URLLC traffic. Based on the delay analysis, the admission region is found under certain delay and reliability requirement, which gives a lower bound on required spectrum resource. Theoretical results in the scenario of a 5G New Radio system are presented, where the SNR thresholds for adaptive modulation and coding scheme selection, transmission rate and delay, as well as admission region under different configurations are discussed. In addition, simulation results are obtained and compared with theoretical results, which validates that the admission region derived in this work provides a lower spectrum allocation bound.
Keywords	Electrical Engineering and Systems Science - Signal Processing ; Electrical Engineering and Systems Science - Systems and Control
Subject code	003
Publishing date	2021-01-05
Publishing country	us
Document type	Book ; Online
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Article ; Online: Correction to: SNX15 Regulates Cell Surface Recycling of APP and Aβ Generation.

Feng, Tuancheng / Niu, Mengmeng / Ji, Chengxiang / Gao, Yuehong / Wen, Jing / Bu, Guojun / Xu, Huaxi / Zhang, Yun-Wu

Molecular neurobiology

2022 Volume 60, Issue 1, Page(s) 428–429

Language	English
Publishing date	2022-11-02
Publishing country	United States
Document type	Published Erratum
ZDB-ID	645020-9
ISSN	1559-1182 ; 0893-7648
ISSN (online)	1559-1182
ISSN	0893-7648
DOI	10.1007/s12035-022-03097-7
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Zs.A 2411: Show issues

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Article ; Online: Age-related hyperinsulinemia leads to insulin resistance in neurons and cell-cycle-induced senescence.

Chow, Hei-Man / Shi, Meng / Cheng, Aifang / Gao, Yuehong / Chen, Guimiao / Song, Xuan / So, Raphaella Wai Lam / Zhang, Jie / Herrup, Karl

Nature neuroscience

2019 Volume 22, Issue 11, Page(s) 1806–1819

Abstract: Prediabetes and Alzheimer's disease both increase in prevalence with age. The former is a risk factor for the latter, but a mechanistic linkage between them remains elusive. We show that prediabetic serum hyperinsulinemia is reflected in the ... ...

Abstract	Prediabetes and Alzheimer's disease both increase in prevalence with age. The former is a risk factor for the latter, but a mechanistic linkage between them remains elusive. We show that prediabetic serum hyperinsulinemia is reflected in the cerebrospinal fluid and that this chronically elevated insulin renders neurons resistant to insulin. This leads to abnormal electrophysiological activity and other defects. In addition, neuronal insulin resistance reduces hexokinase 2, thus impairing glycolysis. This hampers the ubiquitination and degradation of p35, favoring its cleavage to p25, which hyperactivates CDK5 and interferes with the GSK3β-induced degradation of β-catenin. CDK5 contributes to neuronal cell death while β-catenin enters the neuronal nucleus and re-activates the cell cycle machinery. Unable to successfully divide, the neuron instead enters a senescent-like state. These findings offer a direct connection between peripheral hyperinsulinemia, as found in prediabetes, age-related neurodegeneration and cognitive decline. The implications for neurodegenerative conditions such as Alzheimer's disease are described.
MeSH term(s)	Aging/physiology ; Animals ; Cell Cycle/physiology ; Cell Death/physiology ; Cellular Senescence/drug effects ; Cellular Senescence/physiology ; Cyclin-Dependent Kinase 5/metabolism ; Excitatory Postsynaptic Potentials/physiology ; Gene Expression/drug effects ; Glycolysis/drug effects ; Hexokinase/metabolism ; Hyperinsulinism/cerebrospinal fluid ; Hyperinsulinism/physiopathology ; Inhibitory Postsynaptic Potentials/physiology ; Insulin/pharmacology ; Insulin Resistance/physiology ; Liraglutide/pharmacology ; Male ; Maze Learning/physiology ; Metformin/pharmacology ; Mice ; Neurons/metabolism ; Neurons/physiology ; Phosphotransferases/metabolism ; Primary Cell Culture ; Protein Serine-Threonine Kinases/metabolism ; Ubiquitination/physiology ; beta Catenin/metabolism
Chemical Substances	Cdk5r1 protein, mouse ; Insulin ; beta Catenin ; Liraglutide (839I73S42A) ; Metformin (9100L32L2N) ; Phosphotransferases (EC 2.7.-) ; salt-inducible kinase-2, mouse (EC 2.7.1.-) ; Hexokinase (EC 2.7.1.1) ; hexokinase 2, mouse (EC 2.7.1.1) ; Cyclin-Dependent Kinase 5 (EC 2.7.11.1) ; Protein Serine-Threonine Kinases (EC 2.7.11.1)
Language	English
Publishing date	2019-10-21
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1420596-8
ISSN	1546-1726 ; 1097-6256
ISSN (online)	1546-1726
ISSN	1097-6256
DOI	10.1038/s41593-019-0505-1
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Zs.A 4891: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (2.OG) ab Jg. 2022: Lesesaal (EG)
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Article ; Online: CDK5-mediated phosphorylation and stabilization of TPX2 promotes hepatocellular tumorigenesis.

Wang, Fuqiang / Zhao, Wenxing / Gao, Yuehong / Zhou, Jiechao / Li, Huifang / Zhang, Guanyun / Guo, Dong / Xie, Chengrong / Li, Jie / Yin, Zhenyu / Zhang, Jie

Journal of experimental & clinical cancer research : CR

2019 Volume 38, Issue 1, Page(s) 286

Abstract: Background: CDK5, an atypical member of the CDK family, play a significant role in the tumorigenesis of multiple organ, but CDK5 and its substrates in genesis and development of HCC is still unclear.: Methods: Expression of CDK5 in HCC tumor and ... ...

Abstract	Background: CDK5, an atypical member of the CDK family, play a significant role in the tumorigenesis of multiple organ, but CDK5 and its substrates in genesis and development of HCC is still unclear. Methods: Expression of CDK5 in HCC tumor and paired adjacent noncancerous tissues from 90 patients were measured by Western blotting, immunohistochemistry, and real-time PCR. The role of CDK5 in cell function and tumorigenesis was explored in HCC cell lines, ex vivo xenografts and diethylnitrosamine induced HCC model. Furthermore, comparative phosphoproteomic screening identified the oncoprotein TPX2 as a new substrate of CDK5. We also identified the effect of CDK5/P25 interaction blocker tamoxifen on HCC cell growth and migration. Results: CDK5 was increased in HCC tisues and the level of CDK5 was correlated with the severity of HCC based on patient recurrence and 5-year fatality rate. Exogenously expressed CDK5 but not kinase-dead CDK5 promoted proliferation, migration, and invasion of HCC cells. Functional ablation of CDK5 significantly inhibited the exacerbation of HCC cells. Xenograft implantation of HCC cells overexpressing CDK5 promoted tumorigenesis, and genetic knockdown of CDK5 reduced HCC growth and metastasis in vivo. More importantly, heterozygous knockout CDK5 (Cdk5+/-) attenuated HCC tumorigenesis induced by diethylnitrosamine. CDK5-mediated phosphorylation of TPX2 at serine 486 promoted its protein stability. TPX2 silence could restore HCC cell migration capability with overexpression CDK5. Treatment with tamoxifen inhibited cell growth and migration of HCC, demonstrating the role of active CDK5 in HCC. Conclusions: Our results suggest activation of CDK5 is associated with HCC tumorigenesis. CDK5-mediated phosphorylation and stabilization of TPX2 promotes hepatocellular proliferation and tumorigenicity.
MeSH term(s)	Animals ; Carcinoma, Hepatocellular/genetics ; Carcinoma, Hepatocellular/metabolism ; Carcinoma, Hepatocellular/pathology ; Cell Cycle Proteins/metabolism ; Cell Line, Tumor ; Cyclin-Dependent Kinase 5/metabolism ; Hep G2 Cells ; Heterografts ; Humans ; Liver Neoplasms/genetics ; Liver Neoplasms/metabolism ; Liver Neoplasms/pathology ; Liver Neoplasms, Experimental/genetics ; Liver Neoplasms, Experimental/metabolism ; Liver Neoplasms, Experimental/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Mice, Nude ; Microtubule-Associated Proteins/metabolism ; Phosphorylation
Chemical Substances	Cell Cycle Proteins ; Microtubule-Associated Proteins ; TPX2 protein, human ; Cyclin-Dependent Kinase 5 (EC 2.7.11.1) ; CDK5 protein, human (EC 2.7.11.22)
Language	English
Publishing date	2019-07-04
Publishing country	England
Document type	Journal Article
ZDB-ID	803138-1
ISSN	1756-9966 ; 0392-9078
ISSN (online)	1756-9966
ISSN	0392-9078
DOI	10.1186/s13046-019-1297-6
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Zs.A 2065: Show issues

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Jg. 1995 - 2021: Lesesall (1.OG)
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Article: Bazhu Decoction, a Traditional Chinese Medical Formula, Ameliorates Cognitive Deficits in the 5xFAD Mouse Model of Alzheimer's Disease.

Peng, Axiang / Gao, Yuehong / Zhuang, Xiaomei / Lin, Yaoqi / He, Wencan / Wang, Yannan / Chen, Wenfan / Chen, Tingting / Huang, Xiaoqing / Yang, Renzhi / Huang, Yuanpeng / Xi, Shengyan / Zhang, Xian

Frontiers in pharmacology

2019 Volume 10, Page(s) 1391

Abstract: Alzheimer's disease (AD) is the most common neurodegenerative disorder associated with aging. There are currently no effective treatments for AD. Bazhu decoction (BZD), a traditional Chinese medicine (TCM) formula, has been employed clinically to ... ...

Abstract	Alzheimer's disease (AD) is the most common neurodegenerative disorder associated with aging. There are currently no effective treatments for AD. Bazhu decoction (BZD), a traditional Chinese medicine (TCM) formula, has been employed clinically to alleviate AD. However, the underlying molecular mechanisms are still unclear. Here we found that middle- and high-doses of BZD ameliorated the behavioral aspects of 5xFAD transgenic mice in elevated plus maze, Y maze and Morris water maze tests. Moreover, BZD reduced the protein levels of BACE1 and PS1, resulting in a reduction of Aβ plaques. We also identified a beneficial effect of BZD on oxidative stress by attenuating MDA levels and SOD activity in the brains of 5xFAD mice. Together, these results indicate that BZD produces a dose-dependent positive effect on 5xFAD transgenic mouse model by decreasing APP processing and Aβ plaques, and by ameliorating oxidative damage. BZD may play a protective role in the cognitive and anxiety impairments and may be a complementary therapeutic option for AD.
Language	English
Publishing date	2019-11-27
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2587355-6
ISSN	1663-9812
ISSN	1663-9812
DOI	10.3389/fphar.2019.01391
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Article ; Online: Cyclin-dependent kinase 5-mediated phosphorylation of chloride intracellular channel 4 promotes oxidative stress-induced neuronal death.

Guo, Dong / Xie, Wenting / Xiong, Pan / Li, Huifang / Wang, Siqi / Chen, Guimiao / Gao, Yuehong / Zhou, Jiechao / Zhang, Ye / Bu, Guojun / Xue, Maoqiang / Zhang, Jie

Cell death & disease

2018 Volume 9, Issue 10, Page(s) 951

Abstract: Oxidative stress can cause apoptosis in neurons and may result in neurodegenerative diseases. However, the signaling mechanisms leading to oxidative stress-induced neuronal apoptosis are not fully understood. Oxidative stress stimulates aberrant ... ...

Abstract	Oxidative stress can cause apoptosis in neurons and may result in neurodegenerative diseases. However, the signaling mechanisms leading to oxidative stress-induced neuronal apoptosis are not fully understood. Oxidative stress stimulates aberrant activation of cyclin-dependent kinase 5 (CDK5), thought to promote neuronal apoptosis by phosphorylating many cell death-related substrates. Here, using protein pulldown methods, immunofluorescence experiments and in vitro kinase assays, we identified chloride intracellular channel 4 (CLIC4), the expression of which increases during neuronal apoptosis, as a CDK5 substrate. We found that activated CDK5 phosphorylated serine 108 in CLIC4, increasing CLIC4 protein stability, and accumulation. Pharmacological inhibition or shRNA-mediated silencing of CDK5 decreased CLIC4 levels in neurons. Moreover, CLIC4 overexpression led to neuronal apoptosis, whereas knockdown or pharmacological inhibition of CLIC4 attenuated H
MeSH term(s)	Animals ; Apoptosis/physiology ; Cell Death/genetics ; Cell Death/physiology ; Cell Line, Tumor ; Cell Survival/physiology ; Cells, Cultured ; Chloride Channels/metabolism ; Cyclin-Dependent Kinase 5 ; Electroporation ; Humans ; Immunohistochemistry ; Mice ; Mice, Inbred C57BL ; Mitochondrial Proteins/metabolism ; Neurons/cytology ; Neurons/metabolism ; Oxidative Stress/genetics ; Oxidative Stress/physiology ; Phosphorylation ; Signal Transduction/physiology
Chemical Substances	CLIC protein, mouse ; CLIC4 protein, human ; Chloride Channels ; Mitochondrial Proteins ; Cyclin-Dependent Kinase 5 (EC 2.7.11.1)
Language	English
Publishing date	2018-09-20
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2541626-1
ISSN	2041-4889 ; 2041-4889
ISSN (online)	2041-4889
ISSN	2041-4889
DOI	10.1038/s41419-018-0983-1
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