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Article ; Online: Effectiveness and prognosis of covered stents with different diameters in transjugular intrahepatic portosystemic shunt: a meta-analysis.

Gong, Jiahong / Xia, Ziqin / Zhou, Zhidai / Chen, Liping / Wang, Xiaobing / Zhou, Feng

European journal of gastroenterology & hepatology

2023 Volume 36, Issue 2, Page(s) 229–237

Abstract: Purpose: This meta-analysis was designed to evaluate the clinical outcomes of transjugular intrahepatic portosystemic shunt (TIPS) conducted utilizing stents of different diameters, thus providing recommendations for more logical selections of covered ... ...

Abstract	Purpose: This meta-analysis was designed to evaluate the clinical outcomes of transjugular intrahepatic portosystemic shunt (TIPS) conducted utilizing stents of different diameters, thus providing recommendations for more logical selections of covered stents for patients with portal hypertension, in particular for the Asian population. Materials and methods: Web of Science, PubMed, Embase, Cochrane Library, Chinese National Knowledge Infrastructure and Wan Fang were searched for randomized controlled trials and cohort studies from inception until February 2023. The meta-analysis was carried out using Revman 5.4 Software. Heterogeneity between researches was assessed by the χ2 test and I2 index. The outcomes evaluated were the incidence of post-TIPS hepatic encephalopathy (HE), variceal rebleeding, shunt dysfunction, 1-year overall survival and decrease in portal pressure gradient (PPG). Results: Eight appropriate clinical trials with 1246 patients were selected (638 and 608 patients in the experimental and control groups, respectively). In regards to preoperative PPG reduction, there was no discernible difference between the two groups [mean difference = 1.15, 95% confidence interval (CI) = -0.29-2.58, P = 0.12]. The rate of post-TIPS HE was significantly higher in patients in the 8 mm stent group than in the 6-7 mm stent group [odds ratio (OR) = 0.54, 95% CI = 0.42-0.70, P < 0.00001, I2 = 46%]. There were no significant differences in the rates of variceal rebleeding (OR = 1.05, 95% CI = 0.67-1.65, P = 0.84, I2 = 0%), shunt dysfunction (OR = 0.88, 95% CI = 0.53-1.47, P = 0.64, I2 = 0%) and 1-year overall survival (OR = 0.86, 95% CI = 0.50-1.50, P = 0.61, I2 = 0%). Conclusion: Asian populations with portal hypertension may benefit more from TIPS with 6-7 mm covered stents because they can reduce the risk of postoperative HE while offering similar efficacy when compared to 8 mm covered stents.
MeSH term(s)	Humans ; Portasystemic Shunt, Transjugular Intrahepatic/adverse effects ; Esophageal and Gastric Varices/therapy ; Esophageal and Gastric Varices/complications ; Gastrointestinal Hemorrhage ; Prognosis ; Hypertension, Portal/surgery ; Hypertension, Portal/complications ; Stents/adverse effects ; Hepatic Encephalopathy/etiology ; Hepatic Encephalopathy/epidemiology ; Treatment Outcome
Language	English
Publishing date	2023-12-10
Publishing country	England
Document type	Meta-Analysis ; Journal Article
ZDB-ID	1034239-4
ISSN	1473-5687 ; 0954-691X
ISSN (online)	1473-5687
ISSN	0954-691X
DOI	10.1097/MEG.0000000000002696
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Zs.A 2932: Show issues

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Article: Different therapeutic effects between diabetic and non-diabetic adipose stem cells in diabetic wound healing.

Gong, Jia-Hong / Dong, Jiao-Yun / Xie, Ting / Zhao, Qingnan / Lu, Shu-Liang

Journal of wound care

2021 Volume 30, Issue Sup4, Page(s) S14–S23

Abstract: Objective: This study aimed to investigate how adipose tissue-derived stem cells (ASCs) from diabetic and from non-diabetic rats affect wound healing in different microenvironments.: Method: The two types of ASC-rich cells were distinguished by ... ...

Abstract	Objective: This study aimed to investigate how adipose tissue-derived stem cells (ASCs) from diabetic and from non-diabetic rats affect wound healing in different microenvironments. Method: The two types of ASC-rich cells were distinguished by characteristic surface antigen detection. The ASC-rich cells were transplanted into the wounds of diabetic and non-diabetic rats. Wound healing rates were compared and the healing process in the wound margin sections was used to determine how ASC-rich cells affect wound healing in different microenvironments. Results: ASC density was decreased in diabetic rats. The generation time of ASC-rich cells from diabetic rats (d-ASC-rich cells) was longer than that of ASC-rich cells from non-diabetic rats. The number of pre-apoptotic cells in the third generation (passage 3) of d-ASC-rich cells was higher than that among the ASC-rich cells from non-diabetic rats. CD31 and CD34 expression was higher in d-ASC-rich cells than in ASC-rich cells from non-diabetic rats, whereas CD44 and CD105 expression was lower than that in ASC-rich cells from non-diabetic rats. Transplantation of ASC-rich cells from non-diabetic rats promoted wound healing in both non-diabetic and diabetic rats. In contrast, d-ASC-rich cells and enriched nuclear cells only promoted wound healing in non-diabetic rats. ASC-rich cell transplantation promoted greater tissue regeneration than d-ASC-rich cell transplantation. Conclusion: ASC-rich cells promoted wound healing in diabetic and non-diabetic rats. ASC density was lower in the adipose tissue of diabetic rats compared with non-diabetic rats. d-ASC-rich cells did not promote wound healing in diabetic rats, suggesting that caution is warranted regarding the clinical use of diabetic adipose stem cell transplantation for the treatment of diabetic wounds.
MeSH term(s)	Adipose Tissue/metabolism ; Animals ; Diabetes Mellitus, Experimental/pathology ; Diabetes Mellitus, Experimental/therapy ; Rats ; Stem Cell Transplantation ; Ulcer/pathology ; Ulcer/therapy ; Wound Healing
Language	English
Publishing date	2021-04-15
Publishing country	England
Document type	Journal Article
ZDB-ID	1353951-6
ISSN	0969-0700
ISSN	0969-0700
DOI	10.12968/jowc.2021.30.Sup4.S14
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Zs.A 4572: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (2.OG) ab Jg. 2022: Lesesaal (EG)
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Article: [Advances in the research of treating refractory diabetic wounds with stem cells].

Gong, Jiahong / Lu, Shuliang

Zhonghua shao shang za zhi = Zhonghua shaoshang zazhi = Chinese journal of burns

2014 Volume 30, Issue 6, Page(s) 518–520

Abstract: With the growth of aging society, China has become the country of population with the highest incidence of diabetes in the world. Diabetes leads to pathological changes in vascular and nervous system, rendering the diabetic skin fragile and hard to heal ... ...

Abstract	With the growth of aging society, China has become the country of population with the highest incidence of diabetes in the world. Diabetes leads to pathological changes in vascular and nervous system, rendering the diabetic skin fragile and hard to heal if wounded; in the end most diabetic wounds tend to become chronic skin ulcers. The refractory diabetic wound is the result of various endogenous and exogenous factors. It is a quite complicated pathophysiologic event which lacks an effective and specific therapeutic method in clinic. The use of stem cells could be a new approach of treating diabetic chronic wounds since they have a potential ability of self-renovation and multi-directional differentiation which will promote angiogenesis and wound healing process, thus be beneficial in the care of ischemia diseases of the lower limb. This article reviews basic theory of treating diabetic wound and the changes in microenvironment, and prompts many successful cases in curing refractory diabetic wounds.
MeSH term(s)	China ; Diabetes Mellitus ; Diabetic Foot/therapy ; Humans ; Skin ; Skin Ulcer/therapy ; Stem Cells/cytology ; Stem Cells/physiology ; Wound Healing/physiology
Language	Chinese
Publishing date	2014-12
Publishing country	China
Document type	English Abstract ; Journal Article
ZDB-ID	2229222-6
ISSN	1009-2587
ISSN	1009-2587
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Zs.A 6330: Show issues

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Article ; Online: Exercise in mice ameliorates high-fat diet-induced nonalcoholic fatty liver disease by lowering HMGCS2.

Qian, Xiaoli / Wang, Ting / Gong, Jiahong / Wang, Li / Chen, Xuyan / Lin, Haiyan / Tu, Wenzhan / Jiang, Songhe / Li, Shengcun

Aging

2021 Volume 13, Issue 6, Page(s) 8960–8974

Abstract: Nonalcoholic fatty liver disease (NAFLD) is a common chronic liver disease worldwide. Exercise is a therapeutic strategy for preventing NAFLD. However, the underlying molecular mechanisms by which NAFLD can be ameliorated through exercise are still not ... ...

Abstract	Nonalcoholic fatty liver disease (NAFLD) is a common chronic liver disease worldwide. Exercise is a therapeutic strategy for preventing NAFLD. However, the underlying molecular mechanisms by which NAFLD can be ameliorated through exercise are still not clear. This study investigates the mechanisms by which exercise suppresses NAFLD development induced by a high-fat diet (HFD) in mice. Male 6-week-old C57BL/6J mice were fed a normal diet or HFD for 12 weeks and then induced to swim or remain sedentary for 8 weeks. Histomorphology, inflammatory factors, fat metabolizing enzymes, fibrosis, and steatosis were determined in HFD-fed mouse liver, and levels of hepatic enzymes and molecules in the related pathways were analyzed. NAFLD mice showed evident steatosis, fibrosis, and liver injury, and an increased expression of HMGCS2, Wnt3a/ β-catenin, and phosphorylated (p)-AMPK in the liver. Exercise significantly attenuated these symptoms and downregulated the level of Wnt3a/β-catenin in lipotoxic liver tissue. Inhibition of HMGCS2 expression decreased the activation of the Wnt3a/β-catenin pathway and lowered p-AMPK in palmitate-treated HepG2. Our results suggest that exercise prevents NAFLD-associated liver injury, steatosis, and fibrosis. Exercise-mediated hepatoprotection was achieved partly via the blocking of the upregulation of HMGCS2 and the attenuation of the Wnt3a/β-catenin pathway.
MeSH term(s)	Adenylate Kinase/metabolism ; Animals ; Diet, High-Fat/adverse effects ; Disease Models, Animal ; Hydroxymethylglutaryl-CoA Synthase/metabolism ; Lipid Metabolism/physiology ; Liver/metabolism ; Mice ; Non-alcoholic Fatty Liver Disease/etiology ; Non-alcoholic Fatty Liver Disease/metabolism ; Non-alcoholic Fatty Liver Disease/therapy ; Phosphorylation ; Physical Conditioning, Animal/physiology
Chemical Substances	HMGCS2 protein, mouse (EC 2.3.3.10) ; Hydroxymethylglutaryl-CoA Synthase (EC 2.3.3.10) ; Adenylate Kinase (EC 2.7.4.3)
Language	English
Publishing date	2021-03-01
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ISSN	1945-4589
ISSN (online)	1945-4589
DOI	10.18632/aging.202717
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Article ; Online: The Influence of AGEs Environment on Proliferation, Apoptosis, Homeostasis, and Endothelial Cell Differentiation of Human Adipose Stem Cells.

Gong, Jia-Hong / Dong, Jiao-Yun / Xie, Ting / Lu, Shu-Liang

The international journal of lower extremity wounds

2017 Volume 16, Issue 2, Page(s) 94–103

Abstract: The aim of this study was to evaluate the changes of proliferation, apoptosis, homeostasis, and differentiation of human adipose-derived stem cells (hASCs) in the simulated diabetic microenvironment and discuss the potential of the mesenchymal stem cell ... ...

Abstract	The aim of this study was to evaluate the changes of proliferation, apoptosis, homeostasis, and differentiation of human adipose-derived stem cells (hASCs) in the simulated diabetic microenvironment and discuss the potential of the mesenchymal stem cell in the treatment of chronic diabetic wound. We simulated diabetic microenvironment with glycation end products (AGEs) in vitro and studied the changes of hASCs in proliferation and apoptosis. We found that AGEs inhibited the proliferation and lead to hASCs apoptosis, and the endothelial cell directed differentiation was also inhibited. AGEs upregulated growth-related oncogene and monocyte chemoattractant protein-1 and downregulated urokinase-type plasminogen activator receptor, which may inhibit the proliferation and transference of endothelial cells. The simulated diabetic microenvironment affects the proliferation, apoptosis, and homeostasis of hASCs, the endothelial cell migration, and the synthesis of collagen protein, leading to delayed wound healing.
Language	English
Publishing date	2017-06
Publishing country	United States
Document type	Journal Article
ZDB-ID	2171119-7
ISSN	1552-6941 ; 1534-7346
ISSN (online)	1552-6941
ISSN	1534-7346
DOI	10.1177/1534734617701575
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Zs.A 6003: Show issues

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Article: Electroacupuncture pre-treatment alleviates sepsis-induced cardiac inflammation and dysfunction by inhibiting the calpain-2/STAT3 pathway.

Li, Xuqing / Wang, Li / Ying, Xinwang / Zheng, Yujun / Tan, Qianqian / Yu, Xiaolan / Gong, Jiahong / Li, Ming / Deng, Xiaofeng / Yang, Guanhu / Li, Shengcun / Jiang, Songhe

Frontiers in physiology

2022 Volume 13, Page(s) 961909

Abstract: Electroacupuncture (EA) has both anti-inflammatory and cardio-protective effects. Activation of calpain pathway is involved in several myocardiopathy. In sepsis, the role of calpain-2-regulated STAT3 in cardio-protective mechanism of electroacupuncture ... ...

Abstract	Electroacupuncture (EA) has both anti-inflammatory and cardio-protective effects. Activation of calpain pathway is involved in several myocardiopathy. In sepsis, the role of calpain-2-regulated STAT3 in cardio-protective mechanism of electroacupuncture remains unclear. In this study, we aimed to elucidate the mechanism by which electroacupuncture reduces cardiac inflammation and apoptosis and improves cardiac function during sepsis. Electroacupuncture pretreatment for 7 days was applied in septic cardiomyopathy model induced by lipopolysaccharide (LPS). lipopolysaccharide-induced sepsis was associated with a dramatically systemic inflammation and cardiac dysfunction, which was alleviated by electroacupuncture pre-treatment. Lipopolysaccharide resulted in increases of pro-inflammatory factors (TNF-α,IL1βand IL-6) and apoptosis (TUNEL staining and BAX/Bcl2) via activation of calpain-2/STAT3 pathway.Electroacupuncture pre-treatment inhibited LPS-induced activation of cardiac calpain-2/STAT3 signalling and ameliorated inflammatory and apoptosis. Additionally, inhibition of calpain-2 expression using the corresponding siRNA decreased the Phosphorylation of STAT3,pro-inflammatory factors and apoptosis in lipopolysaccharide- treated cardiomyocytes, confirming that calpain-2 activated p-STAT3 participate in septic cardiomyopathy. Furthermore, suppression of STAT3 by stattic enhanced anti-inflammatory and anti-apoptosis effects of electroacupuncture. These findings reveal mechanisms of electroacupuncture preconditioning protection against cardiac inflammation and apoptosis in sepsis mouse via calpain-2/STAT3 pathway and may provide novel targets for clinical treatments of the sepsis-induced cardiac dysfunction.
Language	English
Publishing date	2022-09-07
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2564217-0
ISSN	1664-042X
ISSN	1664-042X
DOI	10.3389/fphys.2022.961909
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Article ; Online: Exercise Training Reverses Lipotoxicity-induced Cardiomyopathy by Inhibiting HMGCS2.

Li, Shengcun / Qian, Xiaoli / Gong, Jiahong / Chen, Jie / Tu, Wenzhan / Chen, Xiaolong / Chu, Maoping / Yang, Guanhu / Li, Lei / Jiang, Songhe

Medicine and science in sports and exercise

2020 Volume 53, Issue 1, Page(s) 47–57

Abstract: Purpose: This study aimed to determine the effect of exercise training on preventing lipotoxic cardiomyopathy and to investigate the role of the 3-hydroxy-3-methylglutaryl-CoA synthase 2 (HMGCS2) and miR-344g-5p in cardiomyocytes.: Methods: Male ... ...

Abstract	Purpose: This study aimed to determine the effect of exercise training on preventing lipotoxic cardiomyopathy and to investigate the role of the 3-hydroxy-3-methylglutaryl-CoA synthase 2 (HMGCS2) and miR-344g-5p in cardiomyocytes. Methods: Male C57BL/6 mice were fed a 60% high-fat diet (HFD) for 12 wk then began swimming exercise or remained sedentary for 8 wk. Thereafter, cardiac function was assessed by echocardiography, and heart tissue and plasma were collected for further measurements. The molecular mechanism of exercise was investigated after treating Hmgcs2 siRNA in palmitate-induced neonatal mouse cardiomyocytes. Results: HFD induced myocardial hypertrophy and fibrosis and reduced coronary reserve and cardiac function. HMGCS2 levels increased, but junctophilin-2 (JPH2) levels decreased in HFD mice hearts. Such effects were attenuated by swimming exercise. Mechanistically, Hmgcs2 silencing prevented apoptosis and caspase-3 cleavage and elevated the expression of JPH2 in palmitate-stimulated cardiomyocytes. In addition, exercise promoted miR-344g-5p expression in HFD hearts. The overexpression of miR-344g-5p by chemical mimic reduced HMGCS2, apoptosis, and caspase-3 cleavage and elevated JPH2 expression in palmitate-induced cardiomyocytes. Conclusion: Our results suggest that exercise limits lipid metabolic disorder, cardiac hypertrophy, and fibrosis and aids in the prevention of lipotoxic cardiomyopathy. Exercise-mediated cardioprotection by upregulating miR-344g-5p, which targets Hmgcs2 mRNA, prohibits HMGCS2 upregulation and thus lipotoxicity.
MeSH term(s)	Animals ; Body Weight ; Calcium/metabolism ; Cardiomegaly/prevention & control ; Cardiomyopathies/diagnostic imaging ; Cardiomyopathies/metabolism ; Cardiomyopathies/pathology ; Cardiomyopathies/prevention & control ; Diet, High-Fat/adverse effects ; Disease Models, Animal ; Echocardiography, Doppler ; Fibrosis/prevention & control ; Hydroxymethylglutaryl-CoA Synthase/metabolism ; Ketones/metabolism ; Male ; Membrane Proteins/metabolism ; Mice, Inbred C57BL ; MicroRNAs/metabolism ; Muscle Proteins/metabolism ; Myocytes, Cardiac/metabolism ; Physical Conditioning, Animal ; Swimming/physiology ; Up-Regulation ; Mice
Chemical Substances	Ketones ; Membrane Proteins ; MicroRNAs ; Mirn344 microRNA, mouse ; Muscle Proteins ; junctophilin-2 protein, mouse ; HMGCS2 protein, mouse (EC 2.3.3.10) ; Hydroxymethylglutaryl-CoA Synthase (EC 2.3.3.10) ; Calcium (SY7Q814VUP)
Language	English
Publishing date	2020-08-20
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	603994-7
ISSN	1530-0315 ; 0195-9131 ; 0025-7990
ISSN (online)	1530-0315
ISSN	0195-9131 ; 0025-7990
DOI	10.1249/MSS.0000000000002453
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Zs.A 869: Show issues

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Article ; Online: Ischemic stroke induces cardiac dysfunction and alters transcriptome profile in mice.

Chen, Jie / Gong, Jiahong / Chen, Haili / Li, Xuqing / Wang, Li / Qian, Xiaoli / Zhou, Kecheng / Wang, Ting / Jiang, Songhe / Li, Lei / Li, Shengcun

BMC genomics

2021 Volume 22, Issue 1, Page(s) 641

Abstract: Background: Stroke can induce cardiac dysfunction in the absence of primary cardiac disease; however, the mechanisms underlying the interaction between the neurological deficits and the heart are poorly understood. The objective of this study was to ... ...

Abstract	Background: Stroke can induce cardiac dysfunction in the absence of primary cardiac disease; however, the mechanisms underlying the interaction between the neurological deficits and the heart are poorly understood. The objective of this study was to investigate the effects of stroke on cardiac function and to identify the transcriptome characteristics of the heart. Results: Stroke significantly decreased heart weight/tibia length ratio and cardiomyocyte cross-sectional areas and increased atrogin-1 and the E3 ubiquitin ligase MuRF-1, indicating myocardial atrophy in MCAO-induced mouse hearts. RNA sequencing of mRNA revealed 383 differentially expressed genes (DEGs) in MCAO myocardium, of which 221 were downregulated and 162 upregulated. Grouping of DEGs based on biological function and quantitative PCR validation indicated that suppressed immune response and collagen synthesis and altered activity of oxidoreductase, peptidase, and endopeptidase may be involved in MCAO-induced cardiomyopathy. The DEGs were mainly distributed in the membrane or extracellular region of cardiomyocytes and acted as potential mediators of stroke-induced cardiac dysregulation involved in cardiac atrophy. Conclusion: Stroke induced a unique transcriptome response in the myocardium and resulted in immediate cardiac atrophy and dysfunction.
MeSH term(s)	Animals ; Brain Ischemia/pathology ; Heart Diseases/pathology ; Ischemic Stroke ; Mice ; Muscular Atrophy ; Myocytes, Cardiac ; Stroke/genetics ; Stroke/pathology ; Transcriptome
Language	English
Publishing date	2021-09-04
Publishing country	England
Document type	Journal Article
ISSN	1471-2164
ISSN (online)	1471-2164
DOI	10.1186/s12864-021-07938-y
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Article: Catalpol as a Component of

Huang, Zhiyang / Gong, Jiahong / Lin, Wen / Feng, Zhiyi / Ma, Yirou / Tu, Yurong / Cai, Xiong / Liu, Jianhua / Lv, Chang / Lv, Xinru / Wu, Qiuji / Lu, Wenjie / Zhao, Juan / Ying, Yibo / Li, Shengcun / Ni, Wenfei / Chen, Haili

Frontiers in pharmacology

2022 Volume 13, Page(s) 860757

Abstract: Disturbance of the internal environment in the spinal cord after spinal cord injury (SCI) is an important cause of the massive death of neurons in the injury area and one of the major problems that lead to the difficult recovery of motor function in ... ...

Abstract	Disturbance of the internal environment in the spinal cord after spinal cord injury (SCI) is an important cause of the massive death of neurons in the injury area and one of the major problems that lead to the difficult recovery of motor function in patients.
Language	English
Publishing date	2022-07-08
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2587355-6
ISSN	1663-9812
ISSN	1663-9812
DOI	10.3389/fphar.2022.860757
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Article: [Research progress in treatment of bone defect with osteomyelitis by using drug-carried bionic nano-bone].

Gong, Jiahong / Wang, Jingcheng / Wang, Daxin

Zhongguo xiu fu chong jian wai ke za zhi = Zhongguo xiufu chongjian waike zazhi = Chinese journal of reparative and reconstructive surgery

2010 Volume 24, Issue 3, Page(s) 356–362

Abstract: Objective: To review the research progress in the treatment of bone defect or osteomyelitis with drug-carried bionic nano-bone so as to offer some theoretic supports and experimental references for those relative researchers.: Methods: The related ... ...

Abstract	Objective: To review the research progress in the treatment of bone defect or osteomyelitis with drug-carried bionic nano-bone so as to offer some theoretic supports and experimental references for those relative researchers. Methods: The related literature was reviewed. The research progress in the treatment of bone defect or osteomyelitis with drug-carried bionic nano-bone was analyzed, which included the designing and preparing of drug-carried nano composites, the way of making infected bone defect animal models, and the general steps, test indexes and results. Results: Traumatic osteomyelitis and bone defect were two kinds of disease to difficulty cure in nowadays clinical orthopaedics. The osteomyelitis, especially chronic one was quite troublesome during the treatments. Many researchers made artificial bone with nano-materials and added drugs into them by various ways for the purpose of controlling infection and repairing bone defection simultaneously. But these researches were still at the laboratory phase, lacking the criterions of making relative animal models, and needed to be improved in the way of drug slow-releasing and nano-scaffold producing. Conclusion: The drug-carried bionic nano-bone as a kind of newly emerging replacement material has splendid effects in the treatments of traumatic large area bone defect or osteomyelitis and will have a brilliant future.
MeSH term(s)	Animals ; Biomimetic Materials ; Bone Diseases/surgery ; Bone Substitutes ; Drug Carriers ; Nanostructures ; Osteomyelitis/surgery
Chemical Substances	Bone Substitutes ; Drug Carriers
Language	Chinese
Publishing date	2010-03
Publishing country	China
Document type	English Abstract ; Journal Article ; Review
ZDB-ID	1420212-8
ISSN	1002-1892
ISSN	1002-1892
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