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Article ; Online: The good and bad of adipose tissue macrophage exosomes in obesity.

Greenberg, Andrew S / Reeves, Andrew R

2021 Volume 33, Issue 4, Page(s) 700–702

Abstract: Adipose tissue macrophages regulate adipose tissue inflammation and systemic insulin-glucose homeostasis. In a recent study by Ying et al. (2021), M2 polarized bone marrow-derived macrophages secreted exosomes containing miR-690 that, when administered ... ...

Abstract	Adipose tissue macrophages regulate adipose tissue inflammation and systemic insulin-glucose homeostasis. In a recent study by Ying et al. (2021), M2 polarized bone marrow-derived macrophages secreted exosomes containing miR-690 that, when administered to obese mice, improved glucose-insulin homeostasis. miR-690 reduced expression of Nadk, which decreased inflammation and improved insulin signaling.
MeSH term(s)	Adipose Tissue ; Animals ; Exosomes ; Inflammation ; Insulin Resistance ; Macrophages ; Mice ; Mice, Inbred C57BL ; MicroRNAs/genetics ; Obesity
Chemical Substances	MicroRNAs
Language	English
Publishing date	2021-04-07
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Comment
ZDB-ID	2176834-1
ISSN	1932-7420 ; 1550-4131
ISSN (online)	1932-7420
ISSN	1550-4131
DOI	10.1016/j.cmet.2021.03.011
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Article ; Online: Intestinal Acyl-CoA synthetase 5 (ACSL5) deficiency potentiates postprandial GLP-1 & PYY secretion, reduces food intake, and protects against diet-induced obesity.

Griffin, John D / Zhu, Ying / Reeves, Andrew / Buhman, Kimberly K / Greenberg, Andrew S

Molecular metabolism

2024 Volume 83, Page(s) 101918

Abstract: Objective: In the small intestine, the products of digestion of dietary triacylglycerol (TAG), fatty acids (FA) and monoacylglycerol, are taken up by absorptive cells, enterocytes, for systemic energy delivery. These digestion products can also bind ... ...

Abstract	Objective: In the small intestine, the products of digestion of dietary triacylglycerol (TAG), fatty acids (FA) and monoacylglycerol, are taken up by absorptive cells, enterocytes, for systemic energy delivery. These digestion products can also bind receptors on endocrine cells to stimulate the release of hormones capable of influencing systemic energy metabolism. The initial phase of intestinal FA absorption involves the acylation of FAs to acyl-CoA by the acyl-CoA long chain synthetase (ACSL) enzymes. ACSL5 is abundantly expressed in the small intestinal epithelium where it is the major ACSL isoform, contributing approximately 80% of total ACSL activity. In mice with whole body deficiency of ACSL5, the rate of dietary fat absorption is reduced and energy expenditure is increased. However, the mechanisms by which intestinal ACSL5 contributes to intestinal FA metabolism, enteroendocrine signaling, and regulation of energy expenditure remain undefined. Here, we test the hypothesis that intestinal ACSL5 regulates energy metabolism by influencing dietary fat absorption and enteroendocrine signaling. Methods: To explore the role of intestinal ACSL5 in energy balance and intestinal dietary fat absorption, a novel mouse model of intestine specific ACSL5 deficiency (ACSL5 Results: We found that ACSL5 Conclusions: These data indicate that intestinal ACSL5 serves as a critical regulator of energy balance, protecting mice from diet-induced obesity exclusively by increasing satiety and reducing food intake during HFD feeding. The reduction in food intake observed in ACSL5
MeSH term(s)	Animals ; Coenzyme A Ligases/metabolism ; Coenzyme A Ligases/genetics ; Mice ; Obesity/metabolism ; Diet, High-Fat ; Male ; Glucagon-Like Peptide 1/metabolism ; Peptide YY/metabolism ; Mice, Inbred C57BL ; Eating ; Postprandial Period ; Energy Metabolism ; Mice, Knockout
Chemical Substances	Coenzyme A Ligases (EC 6.2.1.-) ; Glucagon-Like Peptide 1 (89750-14-1) ; Peptide YY (106388-42-5) ; acyl CoA synthetase 5 (EC 6.2.1.-)
Language	English
Publishing date	2024-03-16
Publishing country	Germany
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	2708735-9
ISSN	2212-8778 ; 2212-8778
ISSN (online)	2212-8778
ISSN	2212-8778
DOI	10.1016/j.molmet.2024.101918
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Article ; Online: Geriatric chronic recurrent multifocal osteomyelitis (CRMO) mimicking multifocal multiple myeloma: a first in an octogenarian.

Sgaglione, Jonathan / Muran, Andrew / Rhode, Matthew / Goodman, Howard J / Edelman, Morris C / Shah, Suhail Ahmed / Greenberg, Andrew S / Kenan, Shachar

Skeletal radiology

2024

Abstract: Chronic recurrent multifocal osteomyelitis (CRMO), an autoinflammatory bone disorder characterized by non-bacterial osteomyelitis causing recurrent multifocal bone lesions, is a well-known, yet uncommon pediatric condition that rarely affects adults; to ... ...

Abstract	Chronic recurrent multifocal osteomyelitis (CRMO), an autoinflammatory bone disorder characterized by non-bacterial osteomyelitis causing recurrent multifocal bone lesions, is a well-known, yet uncommon pediatric condition that rarely affects adults; to date, it has never been diagnosed over the age of 75. The following report will discuss the first octogenarian diagnosed with CRMO and therefore represents an exceptionally rare presentation of a rare disease. An 83-year-old woman presented with progressive right shoulder, forearm, and hip pain, with associated weight loss and global weakness, requiring a wheelchair for mobility. Imaging revealed a pathologic right ulna fracture in addition to lytic lesions of the right proximal humerus and proximal femur. The clinical picture was thus that of a patient with probable multiple myeloma versus metastatic disease. After an extensive workup, however, the lesions were not malignant; histologic findings were instead suggestive of chronic osteomyelitis with negative cultures. Given the multifocal nature of this condition, combined with a lack of clinical symptoms of infection, a diagnosis of CRMO was rendered. The patient underwent intramedullary nailing of the right femur and splinting of the ulna, with a subsequent remarkable recovery to painless ambulation, complete union of the right ulna fracture, and resolution of the lytic lesions without receiving any targeted medical treatment. This case highlights the importance of maintaining CRMO on the differential for multifocal skeletal lesions, regardless of age. Performing a thorough workup with necessary imaging, biopsy, and culture are critical to establishing this diagnosis, which can only made as a diagnosis of exclusion.
Language	English
Publishing date	2024-03-19
Publishing country	Germany
Document type	Journal Article
ZDB-ID	527592-1
ISSN	1432-2161 ; 0364-2348
ISSN (online)	1432-2161
ISSN	0364-2348
DOI	10.1007/s00256-024-04653-z
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Article ; Online: Integrated Action of Autophagy and Adipose Tissue Triglyceride Lipase Ameliorates Diet-Induced Hepatic Steatosis in Liver-Specific PLIN2 Knockout Mice.

Griffin, John D / Bejarano, Eloy / Wang, Xiang-Dong / Greenberg, Andrew S

Cells

2021 Volume 10, Issue 5

Abstract: An imbalance in the storage and breakdown of hepatic lipid droplet (LD) triglyceride (TAG) leads to hepatic steatosis, a defining feature of non-alcoholic fatty liver disease (NAFLD). The two primary cellular pathways regulating hepatic TAG catabolism ... ...

Abstract	An imbalance in the storage and breakdown of hepatic lipid droplet (LD) triglyceride (TAG) leads to hepatic steatosis, a defining feature of non-alcoholic fatty liver disease (NAFLD). The two primary cellular pathways regulating hepatic TAG catabolism are lipolysis, initiated by adipose triglyceride lipase (ATGL), and lipophagy. Each of these processes requires access to the LD surface to initiate LD TAG catabolism. Ablation of perilipin 2 (PLIN2), the most abundant lipid droplet-associated protein in steatotic liver, protects mice from diet-induced NAFLD. However, the mechanisms underlaying this protection are unclear. We tested the contributions of ATGL and lipophagy mediated lipolysis to reduced hepatic TAG in mice with liver-specific PLIN2 deficiency (PLIN2
MeSH term(s)	Adipose Tissue/enzymology ; Animals ; Autophagy ; Diet/adverse effects ; Lipase/genetics ; Lipase/metabolism ; Lipid Metabolism ; Liver/metabolism ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Non-alcoholic Fatty Liver Disease/etiology ; Non-alcoholic Fatty Liver Disease/metabolism ; Non-alcoholic Fatty Liver Disease/pathology ; Non-alcoholic Fatty Liver Disease/prevention & control ; Perilipin-2/physiology ; Triglycerides/metabolism
Chemical Substances	Perilipin-2 ; Plin2 protein, mouse ; Triglycerides ; Lipase (EC 3.1.1.3)
Language	English
Publishing date	2021-04-25
Publishing country	Switzerland
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, Non-P.H.S.
ZDB-ID	2661518-6
ISSN	2073-4409 ; 2073-4409
ISSN (online)	2073-4409
ISSN	2073-4409
DOI	10.3390/cells10051016
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Article ; Online: Knotless Suture Anchor Suspensionplasty for Basal Joint Arthritis A Prospective Case Series with Short-Term Outcomes.

Larsen, Christopher G / Doering, Travis A / Rinaldi, John / Greenberg, Andrew S / Birdsong, Edward / Tuckman, David V

Bulletin of the Hospital for Joint Disease (2013)

2022 Volume 80, Issue 2, Page(s) 129–136

Abstract: Background: The mainstay of surgical treatment for advanced basal joint arthritis is arthroplasty. Many differ- ent techniques of basal joint arthroplasty exist, but none has been determined to be superior to the others, and most methods used to ... ...

Abstract	Background: The mainstay of surgical treatment for advanced basal joint arthritis is arthroplasty. Many differ- ent techniques of basal joint arthroplasty exist, but none has been determined to be superior to the others, and most methods used to maintain the post-trapeziectomy space require postoperative immobilization or pin fixation. In this article, we describe a knotless suture anchor suspen- sionplasty (KSAS) technique and present a prospective case series with short-term outcomes. The KSAS technique utilizes a suspension construct to maintain the post-trapeziectomy space, allowing for early mobilization without the need for pin fixation or casting. Methods: Twenty-five patients underwent trapeziectomy with KSAS. Visual analog scale (VAS) for pain scores and Quick Disabilities of the Arm, Shoulder, and Hand (qDASH) scores were recorded preoperatively and at multiple post- operative points. Grip and pinch strengths were recorded. Maintenance of the post-trapeziectomy space and subsidence were determined by comparing preoperative and postopera- tive radiographs. Results: VAS pain scores were significantly reduced from baseline at all postoperative time points with a reduction from 6.54 to 1.47 at 20 to 24 weeks (p < 0.001). qDASH scores were also significantly decreased from baseline at all time points except for 1 week postoperatively with a re- duction from 57.71 to 12.27 at 20 to 24 weeks (p < 0.001). Grip strength improved from 80.43% compared to the non- operative side preoperatively to 90.36% at 6 months status post KSAS (p < 0.05). Radiographically, subsidence was 35.11% at final follow-up. Conclusions: Our data suggest that KSAS is a safe, effective, and reproducible basal joint arthroplasty tech- nique that allows for early mobilization while sufficiently maintaining the post-trapeziectomy space enough to prevent impingement of the first metacarpal on the scaphoid. Al- though there are limitations to this prospective case series, the data presented here warrant long-term outcome studies utilizing this technique.
MeSH term(s)	Arthritis, Gouty ; Carpometacarpal Joints/diagnostic imaging ; Carpometacarpal Joints/surgery ; Humans ; Osteoarthritis/diagnostic imaging ; Osteoarthritis/surgery ; Pain ; Suture Anchors ; Thumb/surgery
Language	English
Publishing date	2022-05-28
Publishing country	United States
Document type	Journal Article
ZDB-ID	390411-8
ISSN	2328-5273 ; 1936-9727 ; 1936-9719 ; 0018-5647 ; 0883-9344 ; 2328-4633
ISSN (online)	2328-5273 ; 1936-9727
ISSN	1936-9719 ; 0018-5647 ; 0883-9344 ; 2328-4633
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Article ; Online: Lipid Droplet Protein PLIN1 Regulates Inflammatory Polarity in Human Macrophages and is Involved in Atherosclerotic Plaque Development by Promoting Stable Lipid Storage.

Cho, Kyu Yong / Miyoshi, Hideaki / Nakamura, Akinobu / Greenberg, Andrew S / Atsumi, Tatsuya

Journal of atherosclerosis and thrombosis

2022 Volume 30, Issue 2, Page(s) 170–181

Abstract: Aim: Perilipins (PLINs), peripheral lipid droplet (LD) proteins, play important roles in lipid accumulation and maturation in adipocytes. The relationship between PLIN family proteins and macrophage polarization in atherosclerosis has not been ... ...

Abstract	Aim: Perilipins (PLINs), peripheral lipid droplet (LD) proteins, play important roles in lipid accumulation and maturation in adipocytes. The relationship between PLIN family proteins and macrophage polarization in atherosclerosis has not been elucidated. Methods: The experiments used tissues from human arteries of 65 patients who had undergone a carotid endarterectomy, and cultured macrophages generated from healthy human peripheral blood mononuclear cells. Results: Plaque immunohistochemistry demonstrated co-expression of PLIN1 and PLIN2 in both symptomatic (n=31) and asymptomatic patients (n=34). PLIN2 mRNA expression increased 3.38-fold in the symptomatic group compared with those from asymptomatic. PLIN1 was not expressed on small LDs at a shorter incubation but was on large LDs at longer incubation with oxidized LDL and VLDL, while PLIN2 was observed after 24 h and increased with a longer incubation in cultured M1 macrophage. In M2 macrophages, PLIN1 was seen as early as 24 h following incubation with VLDL, and LD size increased with longer incubation. PLIN1 overexpression increased the size of LDs in M1 macrophages, even after a short incubation, and reduced the RNA expression of TNFA, MMP2, ABCA1, and ABCG1 versus the M1 control. Conversely, silencing of PLIN1 in M2 macrophages had the opposite effects on LD size and RNA expression. Conclusion: There was a relationship between macrophage polarity, cytosolic LD size, and PLIN1/PLIN2 expression levels. PLIN2 was mainly expressed in arterial plaques in symptomatic stroke patients, and associated with the inflammatory phenotype of human macrophages, while PLIN1 expression is closely associated with plaque stability and the anti-inflammatory phenotype.
MeSH term(s)	Humans ; Plaque, Atherosclerotic/metabolism ; Lipid Droplets/metabolism ; Leukocytes, Mononuclear/metabolism ; Macrophages/metabolism ; Perilipin-2/genetics ; Perilipin-2/metabolism ; Lipids ; RNA/metabolism ; Perilipin-1/genetics ; Perilipin-1/metabolism
Chemical Substances	Perilipin-2 ; Lipids ; RNA (63231-63-0) ; PLIN1 protein, human ; Perilipin-1
Language	English
Publishing date	2022-06-06
Publishing country	Japan
Document type	Journal Article
ZDB-ID	2011474-6
ISSN	1880-3873 ; 1340-3478
ISSN (online)	1880-3873
ISSN	1340-3478
DOI	10.5551/jat.63153
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Article ; Online: Correction: Myeloid-Specific Deficiency of

Reeves, Andrew R / Sansbury, Brian E / Pan, Meixia / Han, Xianlin / Spite, Matthew / Greenberg, Andrew S

Journal of immunology (Baltimore, Md. : 1950)

2022 Volume 208, Issue 11, Page(s) 2593

Language	English
Publishing date	2022-05-16
Publishing country	United States
Document type	Journal Article ; Published Erratum
ZDB-ID	3056-9
ISSN	1550-6606 ; 0022-1767 ; 1048-3233 ; 1047-7381
ISSN (online)	1550-6606
ISSN	0022-1767 ; 1048-3233 ; 1047-7381
DOI	10.4049/jimmunol.2200133
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Article: The Impacts of Slc19a3 Deletion and Intestinal SLC19A3 Insertion on Thiamine Distribution and Brain Metabolism in the Mouse.

Wen, Anita / Zhu, Ying / Yee, Sook Wah / Park, Brian I / Giacomini, Kathleen M / Greenberg, Andrew S / Newman, John W

Metabolites

2023 Volume 13, Issue 8

Abstract: The Thiamine Transporter 2 (THTR2) encoded ... ...

Abstract	The Thiamine Transporter 2 (THTR2) encoded by
Language	English
Publishing date	2023-07-26
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2662251-8
ISSN	2218-1989
ISSN	2218-1989
DOI	10.3390/metabo13080885
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Article ; Online: NAAA-regulated lipid signaling in monocytes controls the induction of hyperalgesic priming in mice.

Fotio, Yannick / Mabou Tagne, Alex / Squire, Erica / Lee, Hye-Lim / Phillips, Connor M / Chang, Kayla / Ahmed, Faizy / Greenberg, Andrew S / Villalta, S Armando / Scarfone, Vanessa M / Spadoni, Gilberto / Mor, Marco / Piomelli, Daniele

Nature communications

2024 Volume 15, Issue 1, Page(s) 1705

Abstract: Circulating monocytes participate in pain chronification but the molecular events that cause their deployment are unclear. Using a mouse model of hyperalgesic priming (HP), we show that monocytes enable progression to pain chronicity through a mechanism ... ...

Abstract	Circulating monocytes participate in pain chronification but the molecular events that cause their deployment are unclear. Using a mouse model of hyperalgesic priming (HP), we show that monocytes enable progression to pain chronicity through a mechanism that requires transient activation of the hydrolase, N-acylethanolamine acid amidase (NAAA), and the consequent suppression of NAAA-regulated lipid signaling at peroxisome proliferator-activated receptor-α (PPAR-α). Inhibiting NAAA in the 72 hours following administration of a priming stimulus prevented HP. This effect was phenocopied by NAAA deletion and depended on PPAR-α recruitment. Mice lacking NAAA in CD11b
MeSH term(s)	Humans ; Amidohydrolases ; Enzyme Inhibitors/pharmacology ; Hyperalgesia/genetics ; Lipids ; Monocytes ; Pain ; PPAR alpha ; Animals ; Mice
Chemical Substances	Amidohydrolases (EC 3.5.-) ; Enzyme Inhibitors ; Lipids ; NAAA protein, human (EC 3.5.1.-) ; PPAR alpha ; NAAA protein, mouse (EC 3.5.1.-)
Language	English
Publishing date	2024-02-24
Publishing country	England
Document type	Journal Article
ZDB-ID	2553671-0
ISSN	2041-1723 ; 2041-1723
ISSN (online)	2041-1723
ISSN	2041-1723
DOI	10.1038/s41467-024-46139-5
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Article ; Online: The Effect of Trimethoprim on Thiamine Absorption: A Transporter‐Mediated Drug‐Nutrient Interaction

Vora, Bianca / Wen, Anita / Yee, Sook Wah / Trinh, Kim / Azimi, Mina / Green, Elizabeth A. E. / Sirota, Marina / Greenberg, Andrew S. / Newman, John W. / Giacomini, Kathleen M.

Clinical Pharmacology & Therapeutics. 2023 May 07, v. 114, no. 2 p.381-392

2023

Abstract: Trimethoprim is predicted to inhibit several thiamine transporters, including the primary thiamine intestinal absorptive transporter, ThTR-2, and the hepatic and renal organic cation transporters, OCT1, OCT2, and MATEs. To investigate the effect of ... ...

Abstract	Trimethoprim is predicted to inhibit several thiamine transporters, including the primary thiamine intestinal absorptive transporter, ThTR-2, and the hepatic and renal organic cation transporters, OCT1, OCT2, and MATEs. To investigate the effect of trimethoprim on thiamine absorption, studies were conducted in cells, mice, and healthy volunteers and supported by use of real-world data. In a randomized, crossover clinical study, seven healthy volunteers were given a single oral dose of thiamine or thiamine plus trimethoprim, followed by blood sampling. The thiamine area under the curve (AUC) increased with trimethoprim co-administration (P value = 0.031). Similar results were seen in mice. Trimethoprim appeared to act on thiamine absorption through inhibition of hepatic OCT1 as evidenced from its ability to modulate levels of isobutyrylcarnitine and propionylcarnitine, OCT1 biomarkers identified from metabolomic analyses. Real-world data further supported this finding, showing an association between trimethoprim use and higher lev
Keywords	absorption ; biomarkers ; blood ; cations ; intestines ; low density lipoprotein cholesterol ; metabolomics ; oral administration ; pharmacology ; thiamin ; trimethoprim
Language	English
Dates of publication	2023-0507
Size	p. 381-392.
Publishing place	Wiley
Document type	Article ; Online
Note	Resource is Open Access
ZDB-ID	123793-7
ISSN	1532-6535 ; 0009-9236
ISSN (online)	1532-6535
ISSN	0009-9236
DOI	10.1002/cpt.2932
Database	NAL-Catalogue (AGRICOLA)

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