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Article ; Online: Mechanism of muscle atrophy in a normal-weight rat model of type 2 diabetes established by using a soft-pellet diet.

Akieda-Asai, Sayaka / Ma, Hao / Han, Wanxin / Nagata, Junko / Yamaguchi, Fumitake / Date, Yukari

2024 Volume 14, Issue 1, Page(s) 7670

Abstract: Dietary factors such as food texture affect feeding behavior and energy metabolism, potentially causing obesity and type 2 diabetes. We previously found that rats fed soft pellets (SPs) were neither hyperphagic nor overweight but demonstrated glucose ... ...

Abstract	Dietary factors such as food texture affect feeding behavior and energy metabolism, potentially causing obesity and type 2 diabetes. We previously found that rats fed soft pellets (SPs) were neither hyperphagic nor overweight but demonstrated glucose intolerance, insulin resistance, and hyperplasia of pancreatic β-cells. In the present study, we investigated the mechanism of muscle atrophy in rats that had been fed SPs on a 3-h time-restricted feeding schedule for 24 weeks. As expected, the SP rats were normal weight; however, they developed insulin resistance, glucose intolerance, and fat accumulation. In addition, skeletal muscles of SP rats were histologically atrophic and demonstrated disrupted insulin signaling. Furthermore, we learned that the muscle atrophy of the SP rats developed via the IL-6-STAT3-SOCS3 and ubiquitin-proteasome pathways. Our data show that the dietary habit of consuming soft foods can lead to not only glucose intolerance or insulin resistance but also muscle atrophy.
MeSH term(s)	Rats ; Animals ; Insulin Resistance/physiology ; Glucose Intolerance/metabolism ; Diabetes Mellitus, Type 2/metabolism ; Insulin/metabolism ; Muscular Atrophy/etiology ; Muscular Atrophy/metabolism ; Muscle, Skeletal/metabolism ; Diet ; Diet, High-Fat
Chemical Substances	Insulin
Language	English
Publishing date	2024-04-01
Publishing country	England
Document type	Journal Article
ZDB-ID	2615211-3
ISSN	2045-2322 ; 2045-2322
ISSN (online)	2045-2322
ISSN	2045-2322
DOI	10.1038/s41598-024-57727-2
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article: d-Allulose Inhibits Ghrelin-Responsive, Glucose-Sensitive and Neuropeptide Y Neurons in the Arcuate Nucleus and Central Injection Suppresses Appetite-Associated Food Intake in Mice

Rakhat, Yermek / Kaneko, Kentaro / Wang, Lei / Han, Wanxin / Seino, Yutaka / Yabe, Daisuke / Yada, Toshihiko

Nutrients. 2022 July 29, v. 14, no. 15

2022

Abstract: d-allulose, a rare sugar, has sweetness with few calories. d-allulose regulates feeding and glycemia, and ameliorates hyperphagia, obesity and diabetes. All these functions involve the central nervous system. However, central mechanisms underlying these ... ...

Abstract	d-allulose, a rare sugar, has sweetness with few calories. d-allulose regulates feeding and glycemia, and ameliorates hyperphagia, obesity and diabetes. All these functions involve the central nervous system. However, central mechanisms underlying these effects of d-allulose remain unknown. We recently reported that d-allulose activates the anorexigenic neurons in the hypothalamic arcuate nucleus (ARC), the neurons that respond to glucagon-like peptide-1 and that express proopiomelanocortin. However, its action on the orexigenic neurons remains unknown. This study investigated the effects of d-allulose on the ARC neurons implicated in hunger, by measuring cytosolic Ca²⁺ concentration ([Ca²⁺]ᵢ) in single neurons. d-allulose depressed the increases in [Ca²⁺]ᵢ induced by ghrelin and by low glucose in ARC neurons and inhibited spontaneous oscillatory [Ca²⁺]ᵢ increases in neuropeptide Y (NPY) neurons. d-allulose inhibited 10 of 35 (28%) ghrelin-responsive, 18 of 60 (30%) glucose-sensitive and 3 of 8 (37.5%) NPY neurons in ARC. Intracerebroventricular injection of d-allulose inhibited food intake at 20:00 and 22:00, the early dark phase when hunger is promoted. These results indicate that d-allulose suppresses hunger-associated feeding and inhibits hunger-promoting neurons in ARC. These central actions of d-allulose represent the potential of d-allulose to inhibit the hyperphagia with excessive appetite, thereby counteracting obesity and diabetes.
Keywords	blood glucose ; calcium ; central nervous system ; diabetes ; food intake ; ghrelin ; glucagon-like peptide 1 ; glucose ; hunger ; neuropeptide Y ; obesity ; overeating ; pro-opiomelanocortin ; psicose ; sweetness
Language	English
Dates of publication	2022-0729
Publishing place	Multidisciplinary Digital Publishing Institute
Document type	Article
ZDB-ID	2518386-2
ISSN	2072-6643
ISSN	2072-6643
DOI	10.3390/nu14153117
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Article: First Report of Rhizome Rot Caused by Pectobacterium brasiliense on Ginger in China

Wang, Jinhui / Lu, Yuxiang / Han, Wanxin / Fu, Lijun / Han, Xiaoqing / Zhu, Jiehua / Zhang, Shangqing

Plant disease. 2022 July 01, v. 106, no. 7

2022

Abstract: In August 2020, ginger (Zingiber officinale) rhizomes (cv. Mianjiang) showing soft rot symptoms were observed in a field in Tayang Village, Fengrun District, Tangshan, Hebei Province (North China). The disease incidence in that field (15 ha in size) was ... ...

Abstract	In August 2020, ginger (Zingiber officinale) rhizomes (cv. Mianjiang) showing soft rot symptoms were observed in a field in Tayang Village, Fengrun District, Tangshan, Hebei Province (North China). The disease incidence in that field (15 ha in size) was more than 20%. Symptomatic rhizomes (brown and water-soaked) were surface-sterilized in 75% ethanol for 60 s, followed by three successive rinses with sterile distilled water. Rhizomes were cut into pieces ca. 0.5 cm in length that were soaked in 500 µl of 0.9% saline for 20 min. Aliquots (20 μl) of three tenfold dilutions of the tissue specimen soaking solution were plated onto lysogeny broth (LB) medium and incubated at 28°C for 24 h. Five single colonies were picked from each LB plate and restreaked three times for purity. Endophytic bacteria were also isolated from asymptomatic rhizomes as a control. The bacterial gDNA was extracted using the EasyPure Bacteria Genomic DNA Kit (TransGen Biotech, Beijing, China). The 16S rDNA region was amplified by PCR using the universal primer pair 27F/1492R (Weisburg et al. 1991) and sequenced. The results of BLASTn against NCBI nr of the 16S rDNA amplicons suggested that most isolates (8/10) obtained from the rotten rhizomes belonged to the genus Pectobacterium, and a few isolates (2/10) were Enterobacter spp. Only Enterobacter spp. were isolated from asymptomatic rhizomes. Since all Pectobacterium isolates showed identical 16S rDNA sequences, only two isolates were selected for further analysis. Pectobacterium isolates TS20HJ1 and TS20HJ2 (MZ853520, MZ853521) represent isolates from two plant individuals. To determine the species of the rhizome rot Pectobacterium isolates, multilocus sequence analysis (MLSA) was performed with five housekeeping genes: acnA, icdA, mdh, proA, and rpoS (MZ994717 to MZ994726) (Ma et al. 2007; Waleron et al. 2008), and a phylogenetic tree was reconstructed using RAxML v8.2.12 (github.com/stamatak/standard-RAxML). No sequence variation was observed at any MLSA locus between the two isolates. The results of phylogenetic analysis showed that the ginger rhizome isolates clustered with Pectobacterium brasiliense type strain IBSBF1692ᵀ (Duarte et al. 2004; Nabhan et al. 2012). Ginger seedlings (cv. Mianjiang) were inoculated with the isolate TS20HJ1 by injecting 10 µl of bacterial suspension (10⁸ CFU·ml⁻¹) into the rhizomes or 10 µl of 0.9% saline solution as a control. The seedlings were grown at 28°C and 50% relative humidity. Ten days after inoculation, only the bacteria-inoculated rhizomes showed disease symptoms resembling those observed in the field. Bacterial colonies were obtained from the infected rhizomes and were identified with MLSA gene sequencing, fulfilling Koch’s postulates. P. brasiliense causes soft rot of a wide range of economically important crops (Oulghazi et al. 2021). To our knowledge, this is the first report of P. brasiliense causing rhizome rot of ginger in China. The rhizome rot caused 20 to 25% yield loss on average in the Tangshan region in 2020, which poses a significant threat to local ginger farming. Further research on epidemiology and disease management options is needed.
Keywords	Enterobacter ; Pectobacterium ; Zingiber officinale ; disease control ; disease incidence ; endophytes ; ethanol ; ginger ; loci ; lysogeny ; multilocus sequence typing ; oligodeoxyribonucleotides ; phylogeny ; relative humidity ; rhizome rot ; rhizomes ; sequence diversity ; sodium chloride ; villages ; China
Language	English
Dates of publication	2022-0701
Publishing place	The American Phytopathological Society
Document type	Article
ZDB-ID	754182-x
ISSN	0191-2917
ISSN	0191-2917
DOI	10.1094/PDIS-10-21-2324-PDN
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Article: D-Allulose cooperates with glucagon-like peptide-1 and activates proopiomelanocortin neurons in the arcuate nucleus and central injection inhibits feeding in mice

Yermek, Rakhat / Wang, Lei / Kaneko, Kentaro / Han, Wanxin / Seino, Yutaka / Yabe, Daisuke / Yada, Toshihiko

Biochemical and biophysical research communications. 2022 July 12, v. 613

2022

Abstract: A rare sugar D-Allulose has sweetness without calorie. Previous studies have shown that D-Allulose improves glucose and energy metabolism and ameliorates obesity. However, underlying mechanisms remain elusive. This study explored the effect of central ... ...

Abstract	A rare sugar D-Allulose has sweetness without calorie. Previous studies have shown that D-Allulose improves glucose and energy metabolism and ameliorates obesity. However, underlying mechanisms remain elusive. This study explored the effect of central injection of D-Allulose on feeding behavior in mice. We also examined direct effects of D-Allulose on the neurons in the hypothalamic arcuate nucleus (ARC) that regulate feeding, including the anorexigenic glucagon-like peptide-1 (GLP-1)-responsive neurons and proopiomelanocortin (POMC) neurons. Single neurons were isolated from ARC and cytosolic Ca²⁺ concentration ([Ca²⁺]ᵢ) was measured by fura-2 microfluorometry. Administration of D-Allulose at 5.6, 16.7 and 56 mM concentration-dependently increased [Ca²⁺]ᵢ in ARC neurons. The [Ca²⁺]ᵢ increases took place similarly when the osmolarity of superfusion solution was kept constant. The majority (40%) of the D-Allulose-responsive neurons also responded to GLP-1 with [Ca²⁺]ᵢ increases. D-Allulose increased [Ca²⁺]ᵢ in 33% of POMC neurons in ARC. D-Allulose potentiated the GLP-1 action to increase [Ca²⁺]ᵢ in ARC neurons including POMC neurons. Intracerebroventricular injection of D-Allulose significantly decreased food intake at 1 and 2 h after injection. These results demonstrate that D-Allulose cooperates with glucagon-like peptide-1 and activates the ARC neurons including POMC neurons. Furthermore, central injection of D-Allulose inhibits feeding. These central actions of D-Allulose may underlie the ability of D-Allulose to counteract obesity and diabetes.
Keywords	calcium ; diabetes ; energy metabolism ; food intake ; glucagon-like peptide 1 ; glucose ; obesity ; osmolarity ; pro-opiomelanocortin ; psicose ; research ; sweetness
Language	English
Dates of publication	2022-0712
Size	p. 159-165.
Publishing place	Elsevier Inc.
Document type	Article
ZDB-ID	205723-2
ISSN	0006-291X ; 0006-291X
ISSN (online)	0006-291X
ISSN	0006-291X
DOI	10.1016/j.bbrc.2022.04.027
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Article ; Online: D-Allulose cooperates with glucagon-like peptide-1 and activates proopiomelanocortin neurons in the arcuate nucleus and central injection inhibits feeding in mice.

Yermek, Rakhat / Wang, Lei / Kaneko, Kentaro / Han, Wanxin / Seino, Yutaka / Yabe, Daisuke / Yada, Toshihiko

Biochemical and biophysical research communications

2022 Volume 613, Page(s) 159–165

Abstract	A rare sugar D-Allulose has sweetness without calorie. Previous studies have shown that D-Allulose improves glucose and energy metabolism and ameliorates obesity. However, underlying mechanisms remain elusive. This study explored the effect of central injection of D-Allulose on feeding behavior in mice. We also examined direct effects of D-Allulose on the neurons in the hypothalamic arcuate nucleus (ARC) that regulate feeding, including the anorexigenic glucagon-like peptide-1 (GLP-1)-responsive neurons and proopiomelanocortin (POMC) neurons. Single neurons were isolated from ARC and cytosolic Ca
MeSH term(s)	Animals ; Arcuate Nucleus of Hypothalamus/metabolism ; Fructose ; Glucagon-Like Peptide 1/metabolism ; Mice ; Neurons/metabolism ; Obesity/drug therapy ; Obesity/metabolism ; Pro-Opiomelanocortin/metabolism
Chemical Substances	psicose (23140-52-5) ; Fructose (30237-26-4) ; Pro-Opiomelanocortin (66796-54-1) ; Glucagon-Like Peptide 1 (89750-14-1)
Language	English
Publishing date	2022-04-27
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	205723-2
ISSN	1090-2104 ; 0006-291X ; 0006-291X
ISSN (online)	1090-2104 ; 0006-291X
ISSN	0006-291X
DOI	10.1016/j.bbrc.2022.04.027
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Article: First Report of Rhizome Rot Caused by

Wang, Jinhui / Lu, Yuxiang / Han, Wanxin / Fu, Lijun / Han, Xiaoqing / Zhu, Jiehua / Zhang, Shangqing

Plant disease

2022 , Page(s) PDIS10212324PDN

Language	English
Publishing date	2022-06-16
Publishing country	United States
Document type	Journal Article
ZDB-ID	754182-x
ISSN	0191-2917
ISSN	0191-2917
DOI	10.1094/PDIS-10-21-2324-PDN
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Article ; Online: d-Allulose Inhibits Ghrelin-Responsive, Glucose-Sensitive and Neuropeptide Y Neurons in the Arcuate Nucleus and Central Injection Suppresses Appetite-Associated Food Intake in Mice.

Rakhat, Yermek / Kaneko, Kentaro / Wang, Lei / Han, Wanxin / Seino, Yutaka / Yabe, Daisuke / Yada, Toshihiko

Nutrients

2022 Volume 14, Issue 15

Abstract	d-allulose, a rare sugar, has sweetness with few calories. d-allulose regulates feeding and glycemia, and ameliorates hyperphagia, obesity and diabetes. All these functions involve the central nervous system. However, central mechanisms underlying these effects of d-allulose remain unknown. We recently reported that d-allulose activates the anorexigenic neurons in the hypothalamic arcuate nucleus (ARC), the neurons that respond to glucagon-like peptide-1 and that express proopiomelanocortin. However, its action on the orexigenic neurons remains unknown. This study investigated the effects of d-allulose on the ARC neurons implicated in hunger, by measuring cytosolic Ca
MeSH term(s)	Animals ; Appetite ; Arcuate Nucleus of Hypothalamus/physiology ; Eating ; Fructose ; Ghrelin/pharmacology ; Glucose/pharmacology ; Hyperphagia/prevention & control ; Mice ; Neurons/metabolism ; Neuropeptide Y/metabolism ; Obesity/drug therapy ; Rats ; Rats, Sprague-Dawley
Chemical Substances	Ghrelin ; Neuropeptide Y ; psicose (23140-52-5) ; Fructose (30237-26-4) ; Glucose (IY9XDZ35W2)
Language	English
Publishing date	2022-07-29
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2518386-2
ISSN	2072-6643 ; 2072-6643
ISSN (online)	2072-6643
ISSN	2072-6643
DOI	10.3390/nu14153117
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Article: Identification and characterization of opportunistic pathogen

Han, Wanxin / Wang, Jinhui / Pirhonen, Minna / Pan, Yang / Qin, Jingxin / Zhang, Shangqing / Zhu, Jiehua / Yang, Zhihui

Frontiers in plant science

2023 Volume 14, Page(s) 1097741

Abstract: Blackleg and aerial stem rot of potato ( ...

Abstract	Blackleg and aerial stem rot of potato (
Language	English
Publishing date	2023-03-03
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2613694-6
ISSN	1664-462X
ISSN	1664-462X
DOI	10.3389/fpls.2023.1097741
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Article ; Online: Oral Semaglutide under Human Protocols and Doses Regulates Food Intake, Body Weight, and Glycemia in Diet-Induced Obese Mice.

Rakhat, Yermek / Wang, Lei / Han, Wanxin / Rustemova, Aktolkyn / Kulzhanova, Nazymgul / Yamada, Yuichiro / Yabe, Daisuke / Seino, Yutaka / Yada, Toshihiko

Nutrients

2023 Volume 15, Issue 17

Abstract: The first oral form of the glucagon-like peptide-1 receptor agonist, oral semaglutide, has recently been launched and potently controls glycemia and body weight in subjects with type 2 diabetes. This drug carries the absorption enhancer and requires ... ...

Abstract	The first oral form of the glucagon-like peptide-1 receptor agonist, oral semaglutide, has recently been launched and potently controls glycemia and body weight in subjects with type 2 diabetes. This drug carries the absorption enhancer and requires specific protocols of administration. The mechanism of action of oral semaglutide is not fully understood, for which an appropriate experimental model is required. This study explores the metabolic effects of oral semaglutide in mice under human protocols and doses. Oral semaglutide was bolus and once daily injected into high-fat diet-induced obese (DIO) mice under human protocols, followed by monitoring blood glucose, food intake, and body weight. Oral semaglutide 0.23 mg/kg, a comparable human dose (14 mg) in a small volume of water under human protocols rapidly decreased blood glucose and food intake and continuously reduced food intake and weight gain for 3 days in DIO mice. At 0.7 mg/kg (42 mg), this drug was somewhat more potent. Oral semaglutide with human protocols and doses rapidly reduces blood glucose and food intake and continuously suppresses feeding and weight in DIO mice. This study establishes mice as a model suitable for analyzing the mechanism of anti-obesity/diabetes actions of oral semaglutide.
MeSH term(s)	Glucagon-Like Peptides/administration & dosage ; Glucagon-Like Peptides/pharmacology ; Eating/drug effects ; Diet, High-Fat/adverse effects ; Mice, Obese ; Blood Glucose/drug effects ; Animals ; Mice
Chemical Substances	semaglutide (53AXN4NNHX) ; Glucagon-Like Peptides (62340-29-8) ; Blood Glucose
Language	English
Publishing date	2023-08-28
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2518386-2
ISSN	2072-6643 ; 2072-6643
ISSN (online)	2072-6643
ISSN	2072-6643
DOI	10.3390/nu15173765
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Article: First Report of Pectobacterium versatile Causing Aerial Stem Rot of Potato in China

Han, Wanxin / Wang, Jinhui / Li, Zheng / Pan, Yang / Zhang, Dai / Zhao, Dongmei / Li, Qian / Yang, Zhihui / Zhu, Jiehua

Plant disease. 2022 Feb. 28, v. 106, no. 2

2022

Abstract: Pectobacterium species cause blackleg, soft rot, and stem rot in potato and many other vegetable crops (Charkowski 2015). In July 2020, potato plants showing characteristic symptoms of aerial stem rot were observed in a field (cv. Xisen 6) in Fengning ... ...

Abstract	Pectobacterium species cause blackleg, soft rot, and stem rot in potato and many other vegetable crops (Charkowski 2015). In July 2020, potato plants showing characteristic symptoms of aerial stem rot were observed in a field (cv. Xisen 6) in Fengning Manchu Autonomous County, Chengde, Hebei Province (North China). The disease incidence in that field (5 ha in size) was more than 50%. Putative pectolytic bacteria were obtained from symptomatic stem tissues (light brown and water-soaked stem sections) by culturing on crystal violet pectate (CVP) medium. Bacterial colonies producing pits were restreaked and purified on Luria-Bertani (LB) agar. The isolates causing stem rot were gram-negative and rod shaped, negative for oxidase, urease, indole production, gelatin liquefaction, and acid production from maltose and D-sorbitol. All isolates were catalase positive, produced acid from lactose, rhamnose, saccharose, raffinose, and D-arabinose, and were tolerant to 5% NaCl, and able to utilize citrate. The bacterial gDNA was extracted using the EasyPure Bacteria Genomic DNA Kit (TransGen Biotech, Beijing). The 16S rDNA region was amplified by PCR using the universal primer pair 27F/1492R and sequenced. Result of the BLASTn analysis of the 16S rDNA amplicons (MZ379788, MZ379789) suggested that the isolates FN20111 and FN20121 belonged to the genus Pectobacterium. To determine the species of the stem rot Pectobacterium isolates, multilocus sequence analysis (MLSA) was performed with six housekeeping genes acnA, gapA, icdA, mdh, proA, and rpoS (MZ403781–MZ403792), and the phylogenetic tree was reconstructed using RAxML v8.2.12 (https://github.com/stamatak/standard-RAxML). The result of phylogenetic analysis showed that the stem rot Pectobacterium isolates FN20111 and FN20121 clustered with P. versatile (syn. ‘Candidatus Pectobacterium maceratum’) strains CFBP6051ᵀ (Portier et al. 2019), SCC1 (Niemi et al. 2017), and F131 (Shirshikov et al. 2018). The isolates FN20111 and FN20121 were more closely related to the type strain CFBP6051ᵀ than to strains SCC1 and F131. Potato seedlings (cvs. Xisen 6 and Favorita) were inoculated with the isolates FN20111 and FN20121 by injecting 100 µl of bacterial suspensions (10⁸ CFU·ml⁻¹) into the upper parts of the stems of potato plants, or injected with 100 µl of 0.9% saline solution as control. The seedlings were grown at 28°C and 50% relative humidity. Three days postinoculation, only the bacteria-inoculated seedlings showed diseased symptoms resembling those observed in the field. Bacterial colonies were obtained from the infected stems and were identified using the same PCR primers of housekeeping genes as described above, fulfilling Koch’s postulates. Pectobacterium versatile causing soft rot and blackleg on potato plants has been reported in Finland (Niemi et al. 2017), Russia (Shirshikov et al. 2018), the Netherlands (Portier et al. 2019), Poland (Waleron et al. 2019), and in New York State (Ma et al. 2021). To our knowledge, this is the first report of P. versatile causing aerial stem rot of potato in China.
Keywords	Pectobacterium ; Russia ; agar ; arabinose ; catalase ; citrates ; disease incidence ; gelatin ; gentian violet ; indoles ; lactose ; liquefaction ; maltose ; multilocus sequence typing ; phylogeny ; potatoes ; raffinose ; relative humidity ; rhamnose ; sodium chloride ; sorbitol ; stem rot ; urease ; China ; Finland ; Netherlands ; New York ; Poland
Language	English
Dates of publication	2022-0228
Publishing place	The American Phytopathological Society
Document type	Article
ZDB-ID	754182-x
ISSN	0191-2917
ISSN	0191-2917
DOI	10.1094/PDIS-06-21-1264-PDN
Database	NAL-Catalogue (AGRICOLA)

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