LIVIVO - Search results -

Search results

Result 1 - 10 of total 69

Search options

Article ; Online: Genetic screening for transthyretin familial amyloid polyneuropathy to avoid misdiagnosis in patients with polyneuropathy associated with high protein in the cerebrospinal fluid.

Lorenzoni, Paulo José / Giugno, Vinicius Riegel / Ducci, Renata Dal-Prá / Werneck, Lineu Cesar / Kay, Claudia Suemi Kamoi / Scola, Rosana Herminia

Acta neurologica Belgica

2023 Volume 123, Issue 4, Page(s) 1655–1657

MeSH term(s)	Humans ; Amyloid Neuropathies, Familial/diagnosis ; Amyloid Neuropathies, Familial/genetics ; Prealbumin/genetics ; Polyneuropathies/diagnosis ; Polyneuropathies/genetics ; Mutation ; Genetic Testing ; Diagnostic Errors ; Amyloid/genetics ; Amyloid/metabolism
Chemical Substances	Prealbumin ; Amyloid
Language	English
Publishing date	2023-02-25
Publishing country	Italy
Document type	Letter
ZDB-ID	127315-2
ISSN	2240-2993 ; 0300-9009
ISSN (online)	2240-2993
ISSN	0300-9009
DOI	10.1007/s13760-023-02215-z
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

In stock of ZB MED Cologne/Königswinter

Ui I Zs.57: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

This service is chargeable due to the Delivery terms set by subito. Orders including an article and supplementary material will be classified as separate orders. In these cases, fees will be demanded for each order.

Details ▾

Article ; Online: Triple-seronegative myasthenia gravis: clinical and epidemiological characteristics.

Rodrigues, Paula Raquel do Vale Pascoal / Kay, Cláudia Suemi Kamoi / Ducci, Renata Dal-Pra / Utiumi, Marco Antonio Takashi / Fustes, Otto Jesus Hernandez / Werneck, Lineu Cesar / Lorenzoni, Paulo José / Scola, Rosana Herminia

Arquivos de neuro-psiquiatria

2024 Volume 82, Issue 1, Page(s) 1–7

Abstract: Background: : Objective: This study aims to describe the frequency and clinical and epidemiological characteristics of patients with triple-SN MG.: Methods: This was a retrospective cross-sectional study carried out through the analysis of ... ...

Title translation	Miastenia gravis triplo-soronegativa: características clínicas e epidemiológicas.
Abstract	Background: Objective: This study aims to describe the frequency and clinical and epidemiological characteristics of patients with triple-SN MG. Methods: This was a retrospective cross-sectional study carried out through the analysis of medical records. Descriptive and analytical statistical analysis was performed comparing subgroups of myasthenic patients, classified according to serological profile. Results: The sample population consisted of 93 MG patients: 85 were positive for antibodies, 80 (86%) with AChR-Abs, 5 (5.4%) with MuSK-Abs, and no MG patients with LRP4-Abs. Eight patients (8.6%) had triple-SN MG; they had a median age at disease onset of 30 years (21-45). Their most common initial symptoms were ptosis, diplopia, and generalized weakness. Most patients presented with mild symptoms at their last visit, reflecting a median MG composite scale score of 4 (0-6), and 75% of patients had an adequate response to treatment. Conclusion: Our study showed a low frequency of triple-SN MG in Brazilian MG patients. Triple-SN MG was predominant in females, who presented with ptosis, diplopia, and generalized weakness, and most patients had an adequate response to immunosuppressive treatment. There was no significant difference between triple-SN MG and the other subgroups.
MeSH term(s)	Female ; Humans ; Young Adult ; Adult ; Middle Aged ; Retrospective Studies ; Diplopia ; Cross-Sectional Studies ; Autoantibodies ; Receptor Protein-Tyrosine Kinases ; LDL-Receptor Related Proteins ; Myasthenia Gravis/diagnosis ; Myasthenia Gravis/drug therapy ; Myasthenia Gravis/epidemiology
Chemical Substances	Autoantibodies ; Receptor Protein-Tyrosine Kinases (EC 2.7.10.1) ; LDL-Receptor Related Proteins
Language	English
Publishing date	2024-02-05
Publishing country	Germany
Document type	Journal Article
ZDB-ID	418916-4
ISSN	1678-4227 ; 0004-282X
ISSN (online)	1678-4227
ISSN	0004-282X
DOI	10.1055/s-0044-1779052
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Ui I Zs.239: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: Myopathy due to carnitine palmitoyltransferase II deficiency: updating genetic aspects of the first publication in Brazil.

Lorenzoni, Paulo José / Kay, Cláudia Suemi Kamoi / Ducci, Renata Dal-Pra / Fustes, Otto Jesus Hernandez / Rodrigues, Paula Raquel do Vale Pascoal / Arndt, Raquel Cristina / Scola, Rosana Herminia / Werneck, Lineu Cesar

Arquivos de neuro-psiquiatria

2024 Volume 82, Issue 2, Page(s) 1–4

Abstract: Carnitine palmitoyltransferase II (CPT II) deficiency is an autosomal recessive inherited disorder related to lipid metabolism affecting skeletal muscle. The first cases of CPT II deficiency causing myopathy were reported in 1973. In 1983, Werneck et al ... ...

Title translation	Miopatia por deficiência de carnitina palmitoiltransferase II: atualizando os aspectos genéticos da primeira publicação no Brasil.
Abstract	Carnitine palmitoyltransferase II (CPT II) deficiency is an autosomal recessive inherited disorder related to lipid metabolism affecting skeletal muscle. The first cases of CPT II deficiency causing myopathy were reported in 1973. In 1983, Werneck et al published the first two Brazilian patients with myopathy due to CPT II deficiency, where the biochemical analysis confirmed deficient CPT activity in the muscle of both cases. Over the past 40 years since the pioneering publication, clinical phenotypes and genetic loci in the
MeSH term(s)	Humans ; Carnitine O-Palmitoyltransferase/genetics ; Carnitine O-Palmitoyltransferase/metabolism ; Carnitine O-Palmitoyltransferase/deficiency ; Brazil ; Muscular Diseases/genetics ; Muscular Diseases/pathology ; Metabolism, Inborn Errors/genetics ; Metabolism, Inborn Errors/metabolism ; Metabolism, Inborn Errors/pathology ; Mutation
Chemical Substances	Carnitine O-Palmitoyltransferase (EC 2.3.1.21)
Language	English
Publishing date	2024-02-23
Publishing country	Germany
Document type	Journal Article
ZDB-ID	418916-4
ISSN	1678-4227 ; 0004-282X
ISSN (online)	1678-4227
ISSN	0004-282X
DOI	10.1055/s-0044-1779508
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Ui I Zs.239: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: Pain-related nociceptive evoked potential and skin wrinkle test in small fiber neuropathy.

Hernández Fustes, Otto Jesus / Kay, Cláudia Suemi Kamoi / Lorenzoni, Paulo José / Ducci, Renata Dal-Prá / Ribas, Michelle Zonkowski / Werneck, Lineu Cesar / Scola, Rosana Herminia

Arquivos de neuro-psiquiatria

2022 Volume 80, Issue 5, Page(s) 545

MeSH term(s)	Evoked Potentials ; Humans ; Neuralgia/diagnosis ; Nociception ; Skin ; Small Fiber Neuropathy/diagnosis
Language	English
Publishing date	2022-04-26
Publishing country	Germany
Document type	Editorial
ZDB-ID	418916-4
ISSN	1678-4227 ; 0004-282X
ISSN (online)	1678-4227
ISSN	0004-282X
DOI	10.1590/0004-282X-ANP-2021-0327
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Ui I Zs.239: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: Single-centre experience with autosomal recessive limb-girdle muscular dystrophy: case series and literature review.

Lorenzoni, Paulo José / Kay, Cláudia Suemi Kamoi / Ducci, Renata Dal-Pra / Fustes, Otto Jesus Hernandez / Rodrigues, Paula Raquel do Vale Pascoal / Hrysay, Nyvia Milicio Coblinski / Arndt, Raquel Cristina / Werneck, Lineu Cesar / Scola, Rosana Herminia

Arquivos de neuro-psiquiatria

2023 Volume 81, Issue 10, Page(s) 922–933

Abstract: Limb-girdle muscular dystrophy (LGMD) is a group of myopathies that lead to progressive muscle weakness, predominantly involving the shoulder and pelvic girdles; it has a heterogeneous genetic etiology, with variation in the prevalence of subtypes ... ...

Title translation	Experiência de um único centro em distrofia muscular de cinturas do tipo autossômica recessiva: série de casos e revisão de literatura.
Abstract	Limb-girdle muscular dystrophy (LGMD) is a group of myopathies that lead to progressive muscle weakness, predominantly involving the shoulder and pelvic girdles; it has a heterogeneous genetic etiology, with variation in the prevalence of subtypes according to the ethnic backgrounds and geographic origins of the populations. The aim of the present study was to analyze a series of patients with autosomal recessive LGMD (LGMD-R) to contribute to a better characterization of the disease and to find the relative proportion of the different subtypes in a Southern Brazil cohort. The sample population consisted of 36 patients with LGMD-R. A 9-gene targeted next-generation sequencing panel revealed variants in 23 patients with LGMD (64%), and it identified calpainopathy (LGMD-R1) in 26%, dysferlinopathy (LGMD-R2) in 26%, sarcoglycanopathies (LGMD-R3-R5) in 13%, telethoninopathy (LGMD-R7) in 18%, dystroglicanopathy (LGMD-R9) in 13%, and anoctaminopathy (LGMD-R12) in 4% of the patients. In these 23 patients with LGMD, there were 27 different disease-related variants in the
MeSH term(s)	Humans ; Anoctamins/genetics ; Brazil ; Muscle Weakness ; Muscular Dystrophies, Limb-Girdle/genetics ; Pentosyltransferases/genetics
Chemical Substances	2,2,2',4'-tetrachloroacetophenone (O43MMI49GE) ; ANO5 protein, human ; Anoctamins ; FKRP protein, human (EC 2.4.2.-) ; Pentosyltransferases (EC 2.4.2.-)
Language	English
Publishing date	2023-10-18
Publishing country	Germany
Document type	Case Reports ; Journal Article ; Review
ZDB-ID	418916-4
ISSN	1678-4227 ; 0004-282X
ISSN (online)	1678-4227
ISSN	0004-282X
DOI	10.1055/s-0043-1772833
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Ui I Zs.239: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: Spectrum of SPTLC1-related disorders: a novel case of 'Ser331 syndrome' that expand the phenotype of hereditary sensory and autonomic neuropathy type 1A and motor neuron diseases.

Lorenzoni, Paulo José / Bayer, Dafne Luana / Ducci, Renata Dal-Prá / Fustes, Otto Jesus Hernandez / do Vale Pascoal Rodrigues, Paula Raquel / Werneck, Lineu Cesar / Kay, Cláudia Suemi Kamoi / Scola, Rosana Herminia

Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology

2023 Volume 44, Issue 7, Page(s) 2551–2554

Abstract: We report a patient with early-onset hereditary sensory and autonomic neuropathy type 1A (HSAN-1A) who developed a distinct phenotype, with tongue fasciculation and atrophy, due to a mutation at serine 331 in the SPTLC1 gene. HSAN-1A manifestation ... ...

Abstract	We report a patient with early-onset hereditary sensory and autonomic neuropathy type 1A (HSAN-1A) who developed a distinct phenotype, with tongue fasciculation and atrophy, due to a mutation at serine 331 in the SPTLC1 gene. HSAN-1A manifestation causing tongue fasciculation and atrophy have been rarely found. Our report adds to the growing evidence of the existence of an overlap between hereditary neuropathy and motor neuron disease caused by pathogenic p.S331Y variant in SPTLC1 gene.
MeSH term(s)	Humans ; Serine C-Palmitoyltransferase/genetics ; Fasciculation ; Phenotype ; Hereditary Sensory and Autonomic Neuropathies/diagnosis ; Hereditary Sensory and Autonomic Neuropathies/genetics ; Mutation/genetics ; Motor Neuron Disease/complications ; Motor Neuron Disease/genetics ; Atrophy
Chemical Substances	Serine C-Palmitoyltransferase (EC 2.3.1.50) ; SPTLC1 protein, human (EC 2.3.1.50)
Language	English
Publishing date	2023-03-24
Publishing country	Italy
Document type	Case Reports ; Journal Article
ZDB-ID	2016546-8
ISSN	1590-3478 ; 1590-1874
ISSN (online)	1590-3478
ISSN	1590-1874
DOI	10.1007/s10072-023-06763-3
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

In stock of ZB MED Cologne/Königswinter

Zs.A 1877: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
Jg. 1995 - 2021: Lesesall (1.OG)
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾

Article: Myopathy due to carnitine palmitoyltransferase II deficiency: updating genetic aspects of the first publication in Brazil

Arquivos de Neuro-Psiquiatria

(February)

2024 Volume 82, Issue 02, Page(s) 1–4

Series title	February
Abstract	Carnitine palmitoyltransferase II (CPT II) deficiency is an autosomal recessive inherited disorder related to lipid metabolism affecting skeletal muscle. The first cases of CPT II deficiency causing myopathy were reported in 1973. In 1983, Werneck et al published the first two Brazilian patients with myopathy due to CPT II deficiency, where the biochemical analysis confirmed deficient CPT activity in the muscle of both cases. Over the past 40 years since the pioneering publication, clinical phenotypes and genetic loci in the CPT2 gene have been described, and pathogenic mechanisms have been better elucidated. Genetic analysis of one of the original cases disclosed compound heterozygous pathogenic variants (p.Ser113Leu/p.Pro50His) in the CPT2 gene. Our report highlights the historical aspects of the first Brazilian publication of the myopathic form of CPT II deficiency and updates the genetic background of this pioneering publication.
Keywords	Muscular Diseases ; Carnitine O-Palmitoyltransferase ; Lipids ; Genetics ; Doenças Musculares ; Carnitina O-Palmitoiltransferase ; Lipídeos ; Genética
Language	English
Publishing date	2024-02-01
Publisher	Thieme Revinter Publicações Ltda.
Publishing place	Stuttgart ; New York
Document type	Article
ZDB-ID	418916-4
ISSN	1678-4227 ; 0004-282X
ISSN (online)	1678-4227
ISSN	0004-282X
DOI	10.1055/s-0044-1779508
Database	Thieme publisher's database

In stock of ZB MED Cologne/Königswinter

Ui I Zs.239: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article: Triple-seronegative myasthenia gravis: clinical and epidemiological characteristics

Arquivos de Neuro-Psiquiatria

(January)

2024 Volume 82, Issue 01, Page(s) 1–7

Abstract: Background: Myasthenia gravis (MG) is an autoimmune disease usually caused by antibodies against the acetylcholine receptor (AChR-Abs), muscle-specific tyrosine kinase (MuSK-Abs), or low-density lipoprotein receptor-related protein 4 (LRP4-Abs). However, ...

Series title	January
Abstract	Background: Myasthenia gravis (MG) is an autoimmune disease usually caused by antibodies against the acetylcholine receptor (AChR-Abs), muscle-specific tyrosine kinase (MuSK-Abs), or low-density lipoprotein receptor-related protein 4 (LRP4-Abs). However, there are MG patients who do not have these antibodies and are thus said to have triple-seronegative (triple-SN) MG. Objective: This study aims to describe the frequency and clinical and epidemiological characteristics of patients with triple-SN MG. Methods: This was a retrospective cross-sectional study carried out through the analysis of medical records. Descriptive and analytical statistical analysis was performed comparing subgroups of myasthenic patients, classified according to serological profile. Results: The sample population consisted of 93 MG patients: 85 were positive for antibodies, 80 (86%) with AChR-Abs, 5 (5.4%) with MuSK-Abs, and no MG patients with LRP4-Abs. Eight patients (8.6%) had triple-SN MG; they had a median age at disease onset of 30 years (21-45). Their most common initial symptoms were ptosis, diplopia, and generalized weakness. Most patients presented with mild symptoms at their last visit, reflecting a median MG composite scale score of 4 (0-6), and 75% of patients had an adequate response to treatment. Conclusion: Our study showed a low frequency of triple-SN MG in Brazilian MG patients. Triple-SN MG was predominant in females, who presented with ptosis, diplopia, and generalized weakness, and most patients had an adequate response to immunosuppressive treatment. There was no significant difference between triple-SN MG and the other subgroups.
Keywords	Myasthenia Gravis ; Antibodies ; Receptors, Cholinergic ; Acetylcholine ; Miastenia Gravis ; Anticorpos ; Receptores Colinérgicos ; Acetilcolina
Language	English
Publishing date	2024-01-01
Publisher	Thieme Revinter Publicações Ltda.
Publishing place	Stuttgart ; New York
Document type	Article
ZDB-ID	418916-4
ISSN	1678-4227 ; 0004-282X
ISSN (online)	1678-4227
ISSN	0004-282X
DOI	10.1055/s-0044-1779052
Database	Thieme publisher's database

In stock of ZB MED Cologne/Königswinter

Ui I Zs.239: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: Peripheral polyneuropathy from electrodiagnostic tests: a 10-year etiology and neurophysiology overview.

Ducci, Renata Dal-Prá / Tessaro, Camila Lorenzini / Kay, Cláudia Suemi Kamoi / Fustes, Otto Jesus Hernandez / Werneck, Lineu Cesar / Lorenzoni, Paulo José / Scola, Rosana Herminia

Arquivos de neuro-psiquiatria

2021 Volume 80, Issue 3, Page(s) 270–279

Abstract: Background: Polyneuropathies are characterized by a symmetrical impairment of the peripheral nervous system, resulting in sensory, motor and/or autonomic deficits. Due to the heterogeneity of causes, an etiological diagnosis for polyneuropathy is ... ...

Abstract	Background: Polyneuropathies are characterized by a symmetrical impairment of the peripheral nervous system, resulting in sensory, motor and/or autonomic deficits. Due to the heterogeneity of causes, an etiological diagnosis for polyneuropathy is challenging. Objective: The aim of this study was to determine the main causes of polyneuropathy confirmed by electrodiagnostic (EDX) tests in a tertiary service and its neurophysiological aspects. Methods: This observational cross-sectional study from a neuromuscular disorders center included individuals whose electrodiagnostic tests performed between 2008 and 2017 confirmed a diagnosis of polyneuropathy. Through analysis of medical records, polyneuropathies were classified according to etiology and neurophysiological aspect. Results: Of the 380 included patients, 59.5% were male, with a median age of 43 years. The main etiologies were: inflammatory (23.7%), hereditary (18.9%), idiopathic (13.7%), multifactorial (11.1%), and diabetes (10.8%). The main electrophysiological patterns were axonal sensorimotor polyneuropathy (36.1%) and "demyelinating and axonal" sensorimotor polyneuropathy (27.9%). Axonal patterns showed greater etiological heterogeneity, with a predominance of idiopathic and multifactorial polyneuropathy, while demyelinating and "demyelinating and axonal" polyneuropathies had a significantly fewer etiologies, with a predominance of hereditary and inflammatory polyneuropathies. Conclusion: The main causes of polyneuropathy confirmed by EDX test in this study were those that presented a severe, atypical and/or rapidly progressing pattern. Other causes were hereditary and those that defy clinical reasoning, such as multiple risk factors; some polyneuropathies did not have a specific etiology. EDX tests are useful for etiological diagnosis of rare polyneuropathies, because neurophysiological patterns are correlated with specific etiologies.
MeSH term(s)	Adult ; Axons ; Cross-Sectional Studies ; Electrodiagnosis/adverse effects ; Female ; Humans ; Male ; Neurophysiology ; Physical Examination ; Polyneuropathies/diagnosis ; Polyneuropathies/etiology
Language	English
Publishing date	2021-11-23
Publishing country	Germany
Document type	Journal Article ; Observational Study
ZDB-ID	418916-4
ISSN	1678-4227 ; 0004-282X
ISSN (online)	1678-4227
ISSN	0004-282X
DOI	10.1590/0004-282X-ANP-2020-0561
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Ui I Zs.239: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: Somatosensory evoked potentials in clinical practice: a review.

Fustes, Otto Jesus Hernández / Kay, Cláudia Suemi Kamoi / Lorenzoni, Paulo José / Ducci, Renata Dal-Prá / Werneck, Lineu Cesar / Scola, Rosana Herminia

Arquivos de neuro-psiquiatria

2021 Volume 79, Issue 9, Page(s) 824–831

Abstract: The authors present a review of the current use of somatosensory evoked potentials (SSEPs) in neurological practice as a non-invasive neurophysiological technique. For this purpose we have reviewed articles published in English or Portuguese in the ... ...

Abstract	The authors present a review of the current use of somatosensory evoked potentials (SSEPs) in neurological practice as a non-invasive neurophysiological technique. For this purpose we have reviewed articles published in English or Portuguese in the PubMed and LILACS databases. In this review, we address the role of SSEPs in neurological diseases that affect the central nervous system and the peripheral nervous system, especially in demyelinating diseases, for monitoring coma, trauma and the functioning of sensory pathways during surgical procedures. The latter, along with new areas of research, has become one of the most important applications of SSEPs.
MeSH term(s)	Evoked Potentials, Motor ; Evoked Potentials, Somatosensory ; Humans
Language	English
Publishing date	2021-11-08
Publishing country	Brazil
Document type	Journal Article ; Review
ZDB-ID	418916-4
ISSN	1678-4227 ; 0004-282X
ISSN (online)	1678-4227
ISSN	0004-282X
DOI	10.1590/0004-282X-ANP-2020-0427
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Ui I Zs.239: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

To top

Your last searches

Full text online

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

Full text online

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito