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  1. Article ; Online: A mini review of Yu-Ping-Feng polysaccharides

    Yang Liu / Binbin Tong / Shuangxing Wang / Gen Li / Yuansheng Tan / Hui Yu / Kelin Yang / Yingying Yu

    Aquaculture Reports, Vol 20, Iss , Pp 100697- (2021)

    Their biological activities and potential applications in aquaculture

    2021  

    Abstract: Polysaccharides, one of the important active ingredients of traditional Chinese herbal medicine, become a focus of attention in many fields, particularly in medical research industry. Yu-Ping-Feng (YPF) is a primary component of many traditional Chinese ... ...

    Abstract Polysaccharides, one of the important active ingredients of traditional Chinese herbal medicine, become a focus of attention in many fields, particularly in medical research industry. Yu-Ping-Feng (YPF) is a primary component of many traditional Chinese medicine prescriptions, and it’s proven to be significantly beneficial to human health. In recent years, polysaccharides from YPF have been well documented after successful isolation and purification, but the polysaccharides from YPF post-processing wastes are not studied. Under the guidance of the Pharmacological Efficacy Index, we have extracted Yu-Ping-Feng polysaccharides (YPF-P) from the wastes of YPF in our previous work. Our previous findings showed that YPF-P promote the growth, enhance immunity and organ structure or function of cultured fish northern snakehead fish (Channa argus) and grass carp (Ctenopharyngodon idellus), and significantly improve the intestinal microflora of fish. This paper reviews the current status of YPF-P and their application in aquaculture, including the development of green feed for aquatic animals and the re-utilization of the YPF wastes. This series of work is a way to have the best of both worlds, not only to realize the rational treatment of medical wastes, but also to effectively use the wastes, to find a green raw material for aquatic feed.
    Keywords Traditional Chinese medicine ; Yu-Ping-Feng polysaccharides ; Feed additive ; Aquaculture. Fisheries. Angling ; SH1-691
    Subject code 540
    Language English
    Publishing date 2021-07-01T00:00:00Z
    Publisher Elsevier
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  2. Article: Lauric Acid Accelerates Glycolytic Muscle Fiber Formation through TLR4 Signaling

    Wang, Leshan / Gang Shu / Kelin Yang / Lina Wang / Lv Luo / Ping Gao / Qianyun Xi / Qingyan Jiang / Songbo Wang / Weijie Zhao / Xiaotong Zhu / Yongliang Zhang

    Journal of agricultural and food chemistry. 2018 June 07, v. 66, no. 25

    2018  

    Abstract: Lauric acid (LA), which is the primary fatty acid in coconut oil, was reported to have many metabolic benefits. TLR4 is a common receptor of lipopolysaccharides and involved mainly in inflammation responses. Here, we focused on the effects of LA on ... ...

    Abstract Lauric acid (LA), which is the primary fatty acid in coconut oil, was reported to have many metabolic benefits. TLR4 is a common receptor of lipopolysaccharides and involved mainly in inflammation responses. Here, we focused on the effects of LA on skeletal muscle fiber types and metabolism. We found that 200 μM LA treatment in C2C12 or dietary supplementation of 1% LA increased MHCIIb protein expression and the proportion of type IIb muscle fibers from 0.452 ± 0.0165 to 0.572 ± 0.0153, increasing the mRNA expression of genes involved in glycolysis, such as HK2 and LDH2 (from 1.00 ± 0.110 to 1.35 ± 0.0843 and from 1.00 ± 0.123 to 1.71 ± 0.302 in vivo, respectively), decreasing the catalytic activity of lactate dehydrogenase (LDH), and transforming lactic acid to pyruvic acid. Furthermore, LA activated TLR4 signaling, and TLR4 knockdown reversed the effect of LA on muscle fiber type and glycolysis. Thus, we inferred that LA promoted glycolytic fiber formation through TLR4 signaling.
    Keywords catalytic activity ; coconut oil ; dodecanoic acid ; gene expression ; glycolysis ; inflammation ; lactate dehydrogenase ; lactic acid ; lipopolysaccharides ; messenger RNA ; muscle fibers ; protein synthesis ; pyruvic acid ; Toll-like receptor 4
    Language English
    Dates of publication 2018-0607
    Size p. 6308-6316.
    Publishing place American Chemical Society
    Document type Article
    ZDB-ID 241619-0
    ISSN 1520-5118 ; 0021-8561
    ISSN (online) 1520-5118
    ISSN 0021-8561
    DOI 10.1021/acs.jafc.8b01753
    Database NAL-Catalogue (AGRICOLA)

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    Z 1185: Show issues

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  3. Article: Phytol increases adipocyte number and glucose tolerance through activation of PI3K/Akt signaling pathway in mice fed high-fat and high-fructose diet

    Wang, Jianbing / Xiaoquan Hu / Wei Ai / Fenglin Zhang / Kelin Yang / Lina Wang / Xiaotong Zhu / Ping Gao / Gang Shu / Qingyan Jiang / Songbo Wang

    Biochemical and biophysical research communications. 2017,

    2017  

    Abstract: It has been shown that adipose tissue hyperplasia (increased adipocyte number or adipogenesis) has beneficial effects on metabolic health. The aim of the present study was to determine whether phytol could modulate hyperplasia/adipogenesis and glucose ... ...

    Abstract It has been shown that adipose tissue hyperplasia (increased adipocyte number or adipogenesis) has beneficial effects on metabolic health. The aim of the present study was to determine whether phytol could modulate hyperplasia/adipogenesis and glucose homeostasis, and to explore the underlying mechanisms in mice fed high-fat and high fructose diet (HFFD). Our results demonstrated that phytol administration decreased body weight gain and inguinal subcutaneous white adipose tissue (iWAT) weight. However, phytol significantly increased the adipocyte number in iWAT, with the smaller average adipocyte diameter. Meanwhile, OGTT result showed that phytol improved glucose tolerance. In accord, phytol administration markedly increased expression of marker genes associated with adipogenesis (PPARγ and C/EBPα) and glucose uptake (AS160 and GLUT4) and activated PI3K/Akt signaling pathway in mice iWAT. In agreement with the in vivo findings, the in vitro results indicated that 100 μM phytol significantly enhanced 3T3-L1 adipogenesis and glucose uptake, and activated PI3K/Akt signaling pathway. However, phytol-induced enhancement of 3T3-L1 adipognesis and glucose uptake, activation of PI3K/Akt signaling pathway, elevation of marker genes involved in adipogensis and glucose uptake, as well as translocation of GLUT4 from cytoplasm to membrane were abolished by Wortmannin, a specific PI3K/Akt inhibitor. Taken together, phytol increased adipocyte number in iWAT and improved glucose tolerance in mice fed HFFD, which was coincident with the enhanced adipogenesis and glucose uptake in 3T3-L1, and was associated with activation of PI3K/Akt signaling pathway. These data suggested the application of phytol as a potential nutritional agent to combat obesity and type 2 diabetes.
    Keywords adipocytes ; adipogenesis ; body weight changes ; cytoplasm ; diet ; fructose ; genetic markers ; glucose ; glucose tolerance ; glucose transporters ; homeostasis ; hyperplasia ; mice ; noninsulin-dependent diabetes mellitus ; obesity ; signal transduction ; white adipose tissue
    Language English
    Size p. .
    Publishing place Elsevier Inc.
    Document type Article
    Note Pre-press version
    ZDB-ID 205723-2
    ISSN 0006-291X ; 0006-291X
    ISSN (online) 0006-291X
    ISSN 0006-291X
    DOI 10.1016/j.bbrc.2017.05.160
    Database NAL-Catalogue (AGRICOLA)

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    In stock of ZB MED Bonn / Germany

    Z 71/706: Show issues
    Z 3.8: Show issues

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