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  1. Article ; Online: COVID-19: How can a department of general surgery survive in a pandemic?

    Guerci, Claudio / Maffioli, Anna / Bondurri, Andrea A / Ferrario, Luca / Lazzarin, Francesco / Danelli, Piergiorgio

    Surgery

    2020  Volume 167, Issue 6, Page(s) 909–911

    MeSH term(s) Betacoronavirus ; COVID-19 ; Coronavirus Infections/epidemiology ; Coronavirus Infections/prevention & control ; Coronavirus Infections/transmission ; Coronavirus Infections/virology ; Disease Outbreaks ; Elective Surgical Procedures/standards ; General Surgery/standards ; Global Health ; Humans ; Pandemics/prevention & control ; Pneumonia, Viral/epidemiology ; Pneumonia, Viral/prevention & control ; Pneumonia, Viral/transmission ; Pneumonia, Viral/virology ; SARS-CoV-2
    Keywords covid19
    Language English
    Publishing date 2020-03-31
    Publishing country United States
    Document type Letter
    ZDB-ID 202467-6
    ISSN 1532-7361 ; 0039-6060
    ISSN (online) 1532-7361
    ISSN 0039-6060
    DOI 10.1016/j.surg.2020.03.012
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  2. Article ; Online: COVID-19 and surgical training in Italy: Residents and young consultants perspectives from the battlefield.

    Ferrario, Luca / Maffioli, Anna / Bondurri, Andrea A / Guerci, Claudio / Lazzarin, Francesco / Danelli, Piergiorgio

    American journal of surgery

    2020  Volume 220, Issue 4, Page(s) 850–852

    MeSH term(s) Betacoronavirus ; COVID-19 ; Consultants ; Coronavirus Infections/epidemiology ; Education, Medical, Graduate/methods ; Humans ; Internship and Residency/methods ; Italy ; Pandemics ; Pneumonia, Viral/epidemiology ; SARS-CoV-2
    Keywords covid19
    Language English
    Publishing date 2020-06-01
    Publishing country United States
    Document type Editorial
    ZDB-ID 2953-1
    ISSN 1879-1883 ; 0002-9610
    ISSN (online) 1879-1883
    ISSN 0002-9610
    DOI 10.1016/j.amjsurg.2020.05.036
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  3. Article ; Online: COVID-19: HOW CAN A DEPARTMENT OF GENERAL SURGERY SURVIVE TO A PANDEMIC?

    Guerci, Claudio / Maffioli, Anna / Bondurri, Andrea A. / Ferrario, Luca / Lazzarin, Francesco / Danelli, Piergiorgio

    Surgery

    Keywords covid19
    Publisher Elsevier
    Document type Article ; Online
    DOI 10.1016/j.surg.2020.03.012
    Database COVID19

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  4. Article: COVID-19 and surgical training in Italy: Residents and young consultants perspectives from the battlefield

    Ferrario, Luca / Maffioli, Anna / Bondurri, Andrea A / Guerci, Claudio / Lazzarin, Francesco / Danelli, Piergiorgio

    Am J Surg

    Keywords covid19
    Publisher WHO
    Document type Article
    Note WHO #Covidence: #457452
    Database COVID19

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  5. Article: COVID-19: How can a department of general surgery survive in a pandemic?

    Guerci, Claudio / Maffioli, Anna / Bondurri, Andrea A / Ferrario, Luca / Lazzarin, Francesco / Danelli, Piergiorgio

    Surgery

    Keywords covid19
    Publisher WHO
    Document type Article
    Note WHO #Covidence: #71618
    Database COVID19

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  6. Article ; Online: COVID-19

    Guerci, Claudio / Maffioli, Anna / Bondurri, Andrea A. / Ferrario, Luca / Lazzarin, Francesco / Danelli, Piergiorgio

    Surgery

    How can a department of general surgery survive in a pandemic?

    2020  Volume 167, Issue 6, Page(s) 909–911

    Keywords Surgery ; covid19
    Language English
    Publisher Elsevier BV
    Publishing country us
    Document type Article ; Online
    ZDB-ID 202467-6
    ISSN 1532-7361 ; 0039-6060
    ISSN (online) 1532-7361
    ISSN 0039-6060
    DOI 10.1016/j.surg.2020.03.012
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  7. Article ; Online: COVID-19 and surgical training in Italy

    Ferrario, Luca / Maffioli, Anna / Bondurri, Andrea A. / Guerci, Claudio / Lazzarin, Francesco / Danelli, Piergiorgio

    The American Journal of Surgery

    Residents and young consultants perspectives from the battlefield

    2020  Volume 220, Issue 4, Page(s) 850–852

    Keywords Surgery ; General Medicine ; covid19
    Language English
    Publisher Elsevier BV
    Publishing country us
    Document type Article ; Online
    ZDB-ID 2953-1
    ISSN 1879-1883 ; 0002-9610
    ISSN (online) 1879-1883
    ISSN 0002-9610
    DOI 10.1016/j.amjsurg.2020.05.036
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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    Uk I Zs.42: Show issues Location:
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  8. Article ; Online: Environment and T regulatory cells in allergy.

    Braga, M / Schiavone, C / Di Gioacchino, G / De Angelis, I / Cavallucci, E / Lazzarin, F / Petrarca, C / Di Gioacchino, M

    The Science of the total environment

    2012  Volume 423, Page(s) 193–201

    Abstract: The central role of T regulatory cells in the responses against harmless environmental antigens has been confirmed by many studies. Impaired T regulatory cell function is implicated in many pathological conditions, particularly allergic diseases. The " ... ...

    Abstract The central role of T regulatory cells in the responses against harmless environmental antigens has been confirmed by many studies. Impaired T regulatory cell function is implicated in many pathological conditions, particularly allergic diseases. The "hygiene hypothesis" suggests that infections and infestations may play a protective role for allergy, whereas environmental pollutants favor the development of allergic diseases. Developing countries suffer from a variety of infections and are also facing an increasing diffusion of environmental pollutants. In these countries allergies increase in relation to the spreading use of xenobiotics (pesticides, herbicides, pollution, etc.) with a rate similar to those of developed countries, overcoming the protective effects of infections. We review here the main mechanisms of non-self tolerance, with particular regard to relations between T regulatory cell activity, infections and infestations such as helminthiasis, and exposure to environmental xenobiotics with relevant diffusion in developing countries.
    MeSH term(s) Communicable Diseases/immunology ; Environmental Exposure ; Environmental Pollutants/immunology ; Humans ; Hypersensitivity/etiology ; Hypersensitivity/immunology ; T-Lymphocytes, Regulatory/immunology ; Xenobiotics/immunology ; Xenobiotics/toxicity
    Chemical Substances Environmental Pollutants ; Xenobiotics
    Language English
    Publishing date 2012-04-15
    Publishing country Netherlands
    Document type Journal Article ; Review
    ZDB-ID 121506-1
    ISSN 1879-1026 ; 0048-9697
    ISSN (online) 1879-1026
    ISSN 0048-9697
    DOI 10.1016/j.scitotenv.2010.08.015
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 949: Show issues Location:
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  9. Article: Lactobacillus paracasei Lp6 favors immune modulation induced by allergoid treatment in ragweed sensitized mice.

    Petrarca, C / Lazzarin, F / Lanuti, P / Marchisio, M / Miscia, S / Rossi, C / Braga, M / Mistrello, G / Di Gioacchino, M

    International journal of immunopathology and pharmacology

    2011  Volume 24, Issue 4, Page(s) 881–893

    Abstract: It has been hypothesized that lactic acid bacteria (LAB) could be used as adjuvant for specific immunotherapy (SIT), as various studies conducted on humans and animals converge to define LAB as anti-Th2 modulators and Treg inducers. In the present study ...

    Abstract It has been hypothesized that lactic acid bacteria (LAB) could be used as adjuvant for specific immunotherapy (SIT), as various studies conducted on humans and animals converge to define LAB as anti-Th2 modulators and Treg inducers. In the present study we evaluated the effects of LAB, in particular Lactobacillus paracasei Lp6 (Lp6), in a mouse model of ragweed (RW) allergy. Groups of Balb/c mice, experimentally sensitized towards ragweed, were treated by viable Lp6 or by RWallergoid with or without co-administration of Lp6. A control group was sham-sensitized with PBS and sham-treated with water and a group was sensitized with RW and treated with water. Serum IgE, RW-induced release of IFN-gamma, IL-4 and IL-10 from splenocytes and the frequency of CD4CD25 regulatory T cells (Tregs) expressing Foxp3 or IL-10 were evaluated in various groups. RW-allergoid treatment induced a reduction of serum IgE, with a decrease in RW-induced release of IL-4, and an increase in IL-10 and IFN-gamma, along with a significant change in the frequency of Tregs, both CD25+ and -. The joint RWallergoid+ Lp6 treatment induced the highest degree of suppression of allergen-driven IL-4, the greatest reduction of IL-4/IFN-gamma and IL-4/IL-10 ratios and the most significant increase of Foxp3 and IL-10 expressing Tregs. The study shows that Lp6 strengthens the immune modulation induced by allergoid-SIT in RW-sensitized mice, essentially characterized by a differential induction of Tregs associated to a reduction of IL-4; data converge to define a role of SIT adjuvant for Lp6.
    MeSH term(s) Adjuvants, Immunologic ; Animals ; Antigens, Plant/immunology ; Bacterial Outer Membrane Proteins/immunology ; Cells, Cultured ; Desensitization, Immunologic ; Disease Models, Animal ; Forkhead Transcription Factors/metabolism ; Hypersensitivity/immunology ; Hypersensitivity/therapy ; Interferon-gamma/metabolism ; Interleukin-10/metabolism ; Interleukin-2 Receptor alpha Subunit/metabolism ; Interleukin-4/metabolism ; Lactobacillus/immunology ; Lipoproteins/immunology ; Mice ; Mice, Inbred BALB C ; Plant Extracts/immunology ; Plant Proteins/immunology ; Spleen/immunology ; T-Lymphocytes, Regulatory/immunology
    Chemical Substances Adjuvants, Immunologic ; Allergoid ; Amb a I protein, Ambrosia artemisiifolia ; Antigens, Plant ; Bacterial Outer Membrane Proteins ; Forkhead Transcription Factors ; Foxp3 protein, mouse ; Il2ra protein, mouse ; Interleukin-2 Receptor alpha Subunit ; Lipoproteins ; Plant Extracts ; Plant Proteins ; Interleukin-10 (130068-27-8) ; Interleukin-4 (207137-56-2) ; Interferon-gamma (82115-62-6)
    Language English
    Publishing date 2011-10
    Publishing country England
    Document type Journal Article
    ZDB-ID 645171-8
    ISSN 2058-7384 ; 0394-6320
    ISSN (online) 2058-7384
    ISSN 0394-6320
    DOI 10.1177/039463201102400407
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 2537: Show issues Location:
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  10. Article: Immunotoxicity of nanoparticles.

    Di Gioacchino, M / Petrarca, C / Lazzarin, F / Di Giampaolo, L / Sabbioni, E / Boscolo, P / Mariani-Costantini, R / Bernardini, G

    International journal of immunopathology and pharmacology

    2011  Volume 24, Issue 1 Suppl, Page(s) 65S–71S

    Abstract: The interaction between NPs and immune system has been demonstrated, however, the data available are limited. Among all traits, i.s. hydrophilicity, lipophilicity, catalytic activity, composition, electronic structure, capacity to bind or coat surface ... ...

    Abstract The interaction between NPs and immune system has been demonstrated, however, the data available are limited. Among all traits, i.s. hydrophilicity, lipophilicity, catalytic activity, composition, electronic structure, capacity to bind or coat surface species and solubility, the dimension, and consequently the surface area, seems to be the main factor that contribute to the interactions of NPs with biological tissues and immune system in particular. Certain NPs accumulate to regional lymph nodes, where they can be taken up and processed by dendritic cells, interact with self-proteins and, hence, modify their antigenicity and elicit altered immune responses and even autoimmunity. Other NPs may induce allergic sensitization, i.e. allergic contact dermatitis to Pd. In vitro studies demonstrated that NPs can modulate cytokine production toward Th1 (Pl, Pd, Ni, Co) or Th2 (Ti, mw and sw Carbon) production patterns. Some NPs have been linked to allergic sensitization, however, It is unlikely that NPs can act as a hapten inducing a specific IgE production, likely they can act as adjuvant and induce a specific pattern of cytokines, antibody and cells that favor allergic sensitization to environmental allergens. Furthermore, NPs demonstrated pro-inflammatory effects in the lung in experimental animal with increased expression on IL-1beta, MIP-1alpha, MCP-1, MIP-2, keratinocyte chemoattractant, TARC, GM-CSF, MIP-1alpha and activation of the stress-activated MAPKs p38 and JNKs. All considered, the available data suggest that through the elicitation of an oxidative stress mechanism, engineered NPs may contribute to pro-inflammatory disease processes in the lung, particularly allergy.
    MeSH term(s) Animals ; Cytokines/biosynthesis ; Humans ; Hypersensitivity/etiology ; Immune System/drug effects ; Immune System/immunology ; Nanoparticles/toxicity ; Pneumonia/etiology
    Chemical Substances Cytokines
    Language English
    Publishing date 2011-01
    Publishing country England
    Document type Journal Article ; Review
    ZDB-ID 645171-8
    ISSN 2058-7384 ; 0394-6320
    ISSN (online) 2058-7384
    ISSN 0394-6320
    Database MEDical Literature Analysis and Retrieval System OnLINE

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