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  1. Article ; Online: Overview of Image Denoising Methods

    LIU Liping, QIAO Lele, JIANG Liucheng

    Jisuanji kexue yu tansuo, Vol 15, Iss 8, Pp 1418-

    2021  Volume 1431

    Abstract: In real scenes, due to the imperfections of equipment and systems or the existence of low-light environments, the collected images are noisy. The images will also be affected by additional noise during the compression and transmission process, which will ...

    Abstract In real scenes, due to the imperfections of equipment and systems or the existence of low-light environments, the collected images are noisy. The images will also be affected by additional noise during the compression and transmission process, which will interfere with subsequent image segmentation and feature extraction processes. Traditional denoising methods use the non-local self-similarity (NLSS) characteristics of the image and the sparse representation in the transform domain, and the method based on block-matching and three-dimensional filtering (BM3D) shows a powerful image denoising performance. With the development of artificial intelligence, image denoising methods based on deep learning have achieved outstanding performance. But so far, there is almost no relevant research on the comprehensive comparison of image denoising methods. Aiming at the traditional image denoising methods and the image denoising methods based on deep neural networks that have emerged in recent years, this paper first introduces the basic framework of the classic traditional denoising and deep neural network denoising methods and classifies and summarizes the denoising methods. Then the existing denoising methods are analyzed and compared quantitatively and qualitatively on the public denoising data set. Finally, this paper points out some potential challenges and future research directions in the field of image denoising.
    Keywords non-local similarity ; transform domain ; block matching technology ; deep neural network ; Electronic computers. Computer science ; QA75.5-76.95
    Subject code 006
    Language Chinese
    Publishing date 2021-08-01T00:00:00Z
    Publisher Journal of Computer Engineering and Applications Beijing Co., Ltd., Science Press
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  2. Article ; Online: Stimulatory and inhibitory effects of phenanthrene on physiological performance of Chlorella vulgaris and Skeletonema costatum

    Lele Jiang / Yueqiang Pan / Shaoting Zhu / Jingmin Qiu / Yu Shang / Juntian Xu / Futian Li / Hongbin Wang

    Scientific Reports, Vol 12, Iss 1, Pp 1-

    2022  Volume 10

    Abstract: Abstract The effects of polycyclic aromatic hydrocarbons on phytoplankton have been extensively documented, but there is limited knowledge about the physiological responses of marine primary producers to phenanthrene at environmentally relevant levels. ... ...

    Abstract Abstract The effects of polycyclic aromatic hydrocarbons on phytoplankton have been extensively documented, but there is limited knowledge about the physiological responses of marine primary producers to phenanthrene at environmentally relevant levels. Here, we investigated the toxicity of phenanthrene (0, 1, and 5 or 10 μg L−1) to the physiological performance of two cosmopolitan phytoplankton species: the green alga Chlorella vulgaris and bloom-forming diatom Skeletonema costatum. The specific growth rates of both species were remarkably inhibited at both low (1 μg L−1) and high phenanthrene concentrations (5 or 10 μg L−1), while their tolerance to phenanthrene differed. At the highest phenanthrene concentration (10 μg L−1), the growth of C. vulgaris was inhibited by 69%, and no growth was observed for S. costatum cells. The superoxide dismutase activity of both species was enhanced at high phenanthrene concentration, and increased activity of catalase was only observed at high phenanthrene concentration in C. vulgaris. Interestingly, the low phenanthrene concentration stimulated the photosynthetic and relative electron transport rates of S. costatum, whereas hormetic effects were not found for growth. Based on our results, phenanthrene could be detrimental to these two species at a environmentally relevant level, while different tolerance levels were detected.
    Keywords Medicine ; R ; Science ; Q
    Subject code 590
    Language English
    Publishing date 2022-03-01T00:00:00Z
    Publisher Nature Portfolio
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  3. Article: An experimental study on carbon dioxide hydrate formation using a gas-inducing agitated reactor

    Li, Airong / Lele Jiang / Siyao Tang

    Energy. 2017 Sept. 01, v. 134

    2017  

    Abstract: The capture, storage and transportation technology of carbon dioxide based on hydrate formation is an innovative conception. This work presents an experimental investigation on CO2 hydrate formation as a function of rotation speed (0, 200, 400, 600, 800 ... ...

    Abstract The capture, storage and transportation technology of carbon dioxide based on hydrate formation is an innovative conception. This work presents an experimental investigation on CO2 hydrate formation as a function of rotation speed (0, 200, 400, 600, 800 rpm), temperatures (274.15–279.15 K) and initial pressures (2.09, 3.17, 4.03, 5.04, 6.02 MPa) by using a gas-inducing agitated reactor. It was found that stable CO2 hydrate formation was efficiently enhanced at the stage of CO2 dissolution and nucleation in the liquid phase by the gas-inducing agitated reactor through mechanical agitation and gas recycle. The induction time was shortened from 261 to 24 min with an increase of agitation speed. Reactor design is one of the important effects on CO2 hydrate formation. In addition, temperature and initial pressure also have strong effects on CO2 hydrate formation and storage capacity. The induction time of nucleation was greatly reduced with a decrease in cooling temperature and an increase in initial pressure. The amount of CO2 consumed and storage capacity were also increased.
    Keywords agitation ; carbon dioxide ; conception ; temperature ; transportation
    Language English
    Dates of publication 2017-0901
    Size p. 629-637.
    Publishing place Elsevier Ltd
    Document type Article
    ZDB-ID 2019804-8
    ISSN 0360-5442 ; 0360-5442
    ISSN (online) 0360-5442
    ISSN 0360-5442
    DOI 10.1016/j.energy.2017.06.023
    Database NAL-Catalogue (AGRICOLA)

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  4. Article ; Online: CLIC1 regulates dendritic cell antigen processing and presentation by modulating phagosome acidification and proteolysis

    Kanin Salao / Lele Jiang / Hui Li / Vicky W.-W. Tsai / Yasmin Husaini / Paul M. G. Curmi / Louise J. Brown / David A. Brown / Samuel N. Breit

    Biology Open, Vol 5, Iss 5, Pp 620-

    2016  Volume 630

    Abstract: Intracellular chloride channel protein 1 (CLIC1) participates in inflammatory processes by regulating macrophage phagosomal functions such as pH and proteolysis. Here, we sought to determine if CLIC1 can regulate adaptive immunity by actions on dendritic ...

    Abstract Intracellular chloride channel protein 1 (CLIC1) participates in inflammatory processes by regulating macrophage phagosomal functions such as pH and proteolysis. Here, we sought to determine if CLIC1 can regulate adaptive immunity by actions on dendritic cells (DCs), the key professional antigen presenting cells. To do this, we first generated bone marrow-derived DCs (BMDCs) from germline CLIC1 gene-deleted (CLIC1−/−) and wild-type (CLIC1+/+) mice, then studied them in vitro and in vivo. We found phagocytosis triggered cytoplasmic CLIC1 translocation to the phagosomal membrane where it regulated phagosomal pH and proteolysis. Phagosomes from CLIC1−/− BMDCs displayed impaired acidification and proteolysis, which could be reproduced if CLIC1+/+, but not CLIC1−/− cells, were treated with IAA94, a CLIC family ion channel blocker. CLIC1−/− BMDC displayed reduced in vitro antigen processing and presentation of full-length myelin oligodendrocyte glycoprotein (MOG) and reduced MOG-induced experimental autoimmune encephalomyelitis. These data suggest that CLIC1 regulates DC phagosomal pH to ensure optimal processing of antigen for presentation to antigen-specific T-cells. Further, they indicate that CLIC1 is a novel therapeutic target to help reduce the adaptive immune response in autoimmune diseases.
    Keywords CLIC1 ; Dendritic cells ; Phagosome ; Acidification ; Proteolysis ; Antigen presentation ; Science ; Q ; Biology (General) ; QH301-705.5
    Subject code 570
    Language English
    Publishing date 2016-05-01T00:00:00Z
    Publisher The Company of Biologists
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  5. Article ; Online: Macrophage inhibitory cytokine-1 (MIC-1/GDF15) slows cancer development but increases metastases in TRAMP prostate cancer prone mice.

    Yasmin Husaini / Min Ru Qiu / Glen P Lockwood / Xu Wei Luo / Ping Shang / Tamara Kuffner / Vicky Wang-Wei Tsai / Lele Jiang / Pamela J Russell / David A Brown / Samuel N Breit

    PLoS ONE, Vol 7, Iss 8, p e

    2012  Volume 43833

    Abstract: Macrophage inhibitory cytokine-1 (MIC-1/GDF15), a divergent member of the TGF-β superfamily, is over-expressed by many common cancers including those of the prostate (PCa) and its expression is linked to cancer outcome. We have evaluated the effect of ... ...

    Abstract Macrophage inhibitory cytokine-1 (MIC-1/GDF15), a divergent member of the TGF-β superfamily, is over-expressed by many common cancers including those of the prostate (PCa) and its expression is linked to cancer outcome. We have evaluated the effect of MIC-1/GDF15 overexpression on PCa development and spread in the TRAMP transgenic model of spontaneous prostate cancer. TRAMP mice were crossed with MIC-1/GDF15 overexpressing mice (MIC-1(fms)) to produce syngeneic TRAMP(fmsmic-1) mice. Survival rate, prostate tumor size, histopathological grades and extent of distant organ metastases were compared. Metastasis of TC1-T5, an androgen independent TRAMP cell line that lacks MIC-1/GDF15 expression, was compared by injecting intravenously into MIC-1(fms) and syngeneic C57BL/6 mice. Whilst TRAMP(fmsmic-1) survived on average 7.4 weeks longer, had significantly smaller genitourinary (GU) tumors and lower PCa histopathological grades than TRAMP mice, more of these mice developed distant organ metastases. Additionally, a higher number of TC1-T5 lung tumor colonies were observed in MIC-1(fms) mice than syngeneic WT C57BL/6 mice. Our studies strongly suggest that MIC-1/GDF15 has complex actions on tumor behavior: it limits local tumor growth but may with advancing disease, promote metastases. As MIC-1/GDF15 is induced by all cancer treatments and metastasis is the major cause of cancer treatment failure and cancer deaths, these results, if applicable to humans, may have a direct impact on patient care.
    Keywords Medicine ; R ; Science ; Q
    Subject code 570
    Language English
    Publishing date 2012-01-01T00:00:00Z
    Publisher Public Library of Science (PLoS)
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  6. Article ; Online: The anorectic actions of the TGFβ cytokine MIC-1/GDF15 require an intact brainstem area postrema and nucleus of the solitary tract.

    Vicky Wang-Wei Tsai / Rakesh Manandhar / Sebastian Beck Jørgensen / Ka Ki Michelle Lee-Ng / Hong Ping Zhang / Christopher Peter Marquis / Lele Jiang / Yasmin Husaini / Shu Lin / Amanda Sainsbury / Paul E Sawchenko / David A Brown / Samuel N Breit

    PLoS ONE, Vol 9, Iss 6, p e

    2014  Volume 100370

    Abstract: Macrophage inhibitory cytokine-1 (MIC-1/GDF15) modulates food intake and body weight under physiological and pathological conditions by acting on the hypothalamus and brainstem. When overexpressed in disease, such as in advanced cancer, elevated serum ... ...

    Abstract Macrophage inhibitory cytokine-1 (MIC-1/GDF15) modulates food intake and body weight under physiological and pathological conditions by acting on the hypothalamus and brainstem. When overexpressed in disease, such as in advanced cancer, elevated serum MIC-1/GDF15 levels lead to an anorexia/cachexia syndrome. To gain a better understanding of its actions in the brainstem we studied MIC-1/GDF15 induced neuronal activation identified by induction of Fos protein. Intraperitoneal injection of human MIC-1/GDF15 in mice activated brainstem neurons in the area postrema (AP) and the medial (m) portion of the nucleus of the solitary tract (NTS), which did not stain with tyrosine hydroxylase (TH). To determine the importance of these brainstem nuclei in the anorexigenic effect of MIC-1/GDF15, we ablated the AP alone or the AP and the NTS. The latter combined lesion completely reversed the anorexigenic effects of MIC-1/GDF15. Altogether, this study identified neurons in the AP and/or NTS, as being critical for the regulation of food intake and body weight by MIC-1/GDF15.
    Keywords Medicine ; R ; Science ; Q
    Subject code 630
    Language English
    Publishing date 2014-01-01T00:00:00Z
    Publisher Public Library of Science (PLoS)
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  7. Article ; Online: TGF-b superfamily cytokine MIC-1/GDF15 is a physiological appetite and body weight regulator.

    Vicky Wang-Wei Tsai / Laurence Macia / Heiko Johnen / Tamara Kuffner / Rakesh Manadhar / Sebastian Beck Jørgensen / Ka Ki Michelle Lee-Ng / Hong Ping Zhang / Liyun Wu / Christopher Peter Marquis / Lele Jiang / Yasmin Husaini / Shu Lin / Herbert Herzog / David A Brown / Amanda Sainsbury / Samuel N Breit

    PLoS ONE, Vol 8, Iss 2, p e

    2013  Volume 55174

    Abstract: The TGF-b superfamily cytokine MIC-1/GDF15 circulates in all humans and when overproduced in cancer leads to anorexia/cachexia, by direct action on brain feeding centres. In these studies we have examined the role of physiologically relevant levels of ... ...

    Abstract The TGF-b superfamily cytokine MIC-1/GDF15 circulates in all humans and when overproduced in cancer leads to anorexia/cachexia, by direct action on brain feeding centres. In these studies we have examined the role of physiologically relevant levels of MIC-1/GDF15 in the regulation of appetite, body weight and basal metabolic rate. MIC-1/GDF15 gene knockout mice (MIC-1(-/-)) weighed more and had increased adiposity, which was associated with increased spontaneous food intake. Female MIC-1(-/-) mice exhibited some additional alterations in reduced basal energy expenditure and physical activity, possibly owing to the associated decrease in total lean mass. Further, infusion of human recombinant MIC-1/GDF15 sufficient to raise serum levels in MIC-1(-/-) mice to within the normal human range reduced body weight and food intake. Taken together, our findings suggest that MIC-1/GDF15 is involved in the physiological regulation of appetite and energy storage.
    Keywords Medicine ; R ; Science ; Q
    Language English
    Publishing date 2013-01-01T00:00:00Z
    Publisher Public Library of Science (PLoS)
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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