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Article: Knowledge, attitude, perceived barriers of hard-to-healed wound care and the association with confidence: A cross-sectional study among community nurses.

Chuang, Shu-Ting / Lo, Shu-Fen / Liao, Pei-Lin / Lin, Pi-Yu / Tsay, Shwu-Feng

2023 Volume 32, Issue 4, Page(s) 487–492

Language	English
Publishing date	2023-08-28
Publishing country	England
Document type	Journal Article
ZDB-ID	1282604-2
ISSN	0965-206X
ISSN	0965-206X
DOI	10.1016/j.jtv.2023.08.003
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Article ; Online: 9-POHSA prevents NF-kB activation and ameliorates LPS-induced inflammation in rat hepatocytes.

Situmorang, Jiro Hasegawa / Chen, Ming-Cheng / Kuo, Wei-Wen / Lin, Shinn-Zong / Shih, Cheng-Yen / Lin, Pi-Yu / Loh, Ching-Hui / Huang, Chih-Yang

Lipids

2023 Volume 58, Issue 5, Page(s) 241–249

Abstract: Liver inflammation has become increasingly prevalent in recent years, leading to the development of diseases like hepatitis, alcoholic liver disease, and fatty liver disease. One factor that has been linked to liver inflammation is increased levels of ... ...

Abstract	Liver inflammation has become increasingly prevalent in recent years, leading to the development of diseases like hepatitis, alcoholic liver disease, and fatty liver disease. One factor that has been linked to liver inflammation is increased levels of lipopolysaccharides (LPS), which can be caused by poor diets and sedentary lifestyles that contribute to liver inflammation. There is promising research on a new class of lipids called fatty acid esters of hydroxy fatty acids (FAHFAs), which have been shown to potentiate insulin release and exert an anti-inflammatory effect. Specifically, one type of FAHFA called 9-POHSA (palmitoleic acid ester of 9-hydroxy stearic acid) has been studied for its potential to attenuate inflammation-related indexes induced by LPS in hepatocytes, which play a critical role in the progression of liver inflammation. This study found that following LPS treatment, tumor necrosis factor- α, interleukin-6, and connective tissue growth factor (CTGF) were upregulated and increased cell migration, but 9-POHSA pre-treatment attenuated the upregulation of these markers and prevented cell migration induced by LPS. Using flowcytometry analysis, intracellular reactive oxygen species (ROS) was found to be responsible for CTGF upregulation. In addition, the effects of 9-POHSA were likely associated with its inhibition of the activation of the NF-kB. These results suggest that 9-POHSA has potential as a therapy for liver inflammation and fibrosis by attenuating inflammation-related indexes induced by LPS in hepatocytes. This study provides important insight into the mechanisms of liver inflammation and the potential for new treatments to address liver diseases.
MeSH term(s)	Animals ; Rats ; Lipopolysaccharides/toxicity ; NF-kappa B ; Inflammation/chemically induced ; Inflammation/drug therapy ; Cell Movement ; Fatty Acids ; Hepatocytes ; Hydroxy Acids
Chemical Substances	Lipopolysaccharides ; NF-kappa B ; Fatty Acids ; Hydroxy Acids
Language	English
Publishing date	2023-08-21
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	241539-2
ISSN	1558-9307 ; 0024-4201
ISSN (online)	1558-9307
ISSN	0024-4201
DOI	10.1002/lipd.12380
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Zs.A 645: Show issues

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Article ; Online: PKC-δ-dependent mitochondrial ROS attenuation is involved as 9-OAHSA combats lipoapotosis in rat hepatocytes induced by palmitic acid and in Syrian hamsters induced by high-fat high-cholesterol high-fructose diet.

Loh, Ching-Hui / Kuo, Wei-Wen / Lin, Shinn-Zong / Shih, Cheng-Yen / Lin, Pi-Yu / Situmorang, Jiro Hasegawa / Huang, Chih-Yang

Toxicology and applied pharmacology

2023 Volume 470, Page(s) 116557

Abstract: Metabolic-associated fatty liver disease (MAFLD) is a global concern, often undetected until reaching an advanced stage. Palmitic acid (PA) is a type of fatty acid that increases and leads to liver apoptosis in MAFLD. However, there is currently no ... ...

Abstract	Metabolic-associated fatty liver disease (MAFLD) is a global concern, often undetected until reaching an advanced stage. Palmitic acid (PA) is a type of fatty acid that increases and leads to liver apoptosis in MAFLD. However, there is currently no approved therapy or compound for MAFLD. Recently, branched fatty acid esters of hydroxy fatty acids (FAHFAs), a group of bioactive lipids, have emerged as promising agents to treat associated metabolic diseases. This study utilizes one type of FAHFA, oleic acid ester of 9-hydroxystearic acid (9-OAHSA), to treat PA-induced lipoapoptosis in an in vitro MAFLD model using rat hepatocytes and a high-fat high-cholesterol high-fructose (HFHCHFruc) diet in Syrian hamsters. The results indicate that 9-OAHSA rescues hepatocytes from PA-induced apoptosis and attenuates lipoapoptosis and dyslipidemia in Syrian hamsters. Additionally, 9-OAHSA decreases the generation of mitochondrial reactive oxygen species (mito-ROS) and stabilizes the mitochondrial membrane potential in hepatocytes. The study also demonstrates that the effect of 9-OAHSA on mito-ROS generation is at least partially mediated by PKC-δ signaling. These findings suggest that 9-OAHSA shows promise as a therapy for MAFLD.
MeSH term(s)	Cricetinae ; Rats ; Animals ; Palmitic Acid/toxicity ; Reactive Oxygen Species/metabolism ; Mesocricetus ; Fructose/toxicity ; Hepatocytes ; Fatty Acids/metabolism ; Non-alcoholic Fatty Liver Disease/metabolism ; Cholesterol/metabolism ; Diet, High-Fat/adverse effects
Chemical Substances	Palmitic Acid (2V16EO95H1) ; Reactive Oxygen Species ; Fructose (30237-26-4) ; Fatty Acids ; Cholesterol (97C5T2UQ7J)
Language	English
Publishing date	2023-05-18
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	204477-8
ISSN	1096-0333 ; 0041-008X
ISSN (online)	1096-0333
ISSN	0041-008X
DOI	10.1016/j.taap.2023.116557
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Article ; Online: Corrigendum to "PKC-δ-dependent mitochondrial ROS attenuation is involved as 9-OAHSA combats lipoapotosis in rat hepatocytes induced by palmitic acid and in Syrian hamsters induced by high-fat high-cholesterol high-fructose diet" [Toxicology and Applied Pharmacology, 470, (2023), 116557].

Loh, Ching-Hui / Kuo, Wei-Wen / Lin, Shinn-Zong / Shih, Cheng-Yen / Lin, Pi-Yu / Situmorang, Jiro Hasegawa / Huang, Chih-Yang

Toxicology and applied pharmacology

2023 Volume 476, Page(s) 116658

Language	English
Publishing date	2023-08-19
Publishing country	United States
Document type	Published Erratum
ZDB-ID	204477-8
ISSN	1096-0333 ; 0041-008X
ISSN (online)	1096-0333
ISSN	0041-008X
DOI	10.1016/j.taap.2023.116658
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Article ; Online: A prospective review of the health-promoting potential of Jing Si Herbal Tea.

Ho, Tsung-Jung / Ahmed, Tanvir / Shibu, Marthandam Asokan / Lin, Yu-Jung / Shih, Cheng Yen / Lin, Pi-Yu / Ling, Shinn-Zong / Chiang, Chien-Yi / Kuo, Wei-Wen / Huang, Chih-Yang

Tzu chi medical journal

2024 Volume 36, Issue 1, Page(s) 1–22

Abstract: Traditional Chinese medicine (TCM) has gained considerable attention over the past few years for its multicomponent, multitarget, and multi-pathway approach to treating different diseases. Studies have shown that TCMs as adjuvant therapy along with ... ...

Abstract	Traditional Chinese medicine (TCM) has gained considerable attention over the past few years for its multicomponent, multitarget, and multi-pathway approach to treating different diseases. Studies have shown that TCMs as adjuvant therapy along with conventional treatment may benefit in safely treating various disorders. However, investigations on finding effective herbal combinations are ongoing. A novel TCM formula, "Jing Si Herbal Tea (JSHT)," has been reported recently for their health-promoting effects in improving overall body and mental health. JSHT is a combination of eight herbs recognized in Chinese herbal pharmacopoeia for their anti-viral, anti-aging, and anti-cancer properties as well as protective effects against cardiovascular, metabolic, neural, digestive, and genitourinary diseases. Thus, to better understand the beneficial effects of the ingredients of JSHT on health, this review intends to summarize the preclinical and clinical studies of the ingredients of JSHT on human health and diseases, and possible therapeutic effects with the related mode of actions and future prospects for their application in complementary therapies.
Language	English
Publishing date	2024-01-12
Publishing country	India
Document type	Journal Article ; Review
ZDB-ID	2452925-4
ISSN	2223-8956 ; 2223-8956
ISSN (online)	2223-8956
ISSN	2223-8956
DOI	10.4103/tcmj.tcmj_194_23
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article ; Online: 9‐POHSA prevents NF‐kB activation and ameliorates LPS‐induced inflammation in rat hepatocytes

Situmorang, Jiro Hasegawa / Chen, Ming‐Cheng / Kuo, Wei‐Wen / Lin, Shinn‐Zong / Shih, Cheng‐Yen / Lin, Pi‐Yu / Loh, Ching‐Hui / Huang, Chih‐Yang

Lipids. 2023 Sept., v. 58, no. 5 p.241-249

2023

Abstract	Liver inflammation has become increasingly prevalent in recent years, leading to the development of diseases like hepatitis, alcoholic liver disease, and fatty liver disease. One factor that has been linked to liver inflammation is increased levels of lipopolysaccharides (LPS), which can be caused by poor diets and sedentary lifestyles that contribute to liver inflammation. There is promising research on a new class of lipids called fatty acid esters of hydroxy fatty acids (FAHFAs), which have been shown to potentiate insulin release and exert an anti‐inflammatory effect. Specifically, one type of FAHFA called 9‐POHSA (palmitoleic acid ester of 9‐hydroxy stearic acid) has been studied for its potential to attenuate inflammation‐related indexes induced by LPS in hepatocytes, which play a critical role in the progression of liver inflammation. This study found that following LPS treatment, tumor necrosis factor‐ α, interleukin‐6, and connective tissue growth factor (CTGF) were upregulated and increased cell migration, but 9‐POHSA pre‐treatment attenuated the upregulation of these markers and prevented cell migration induced by LPS. Using flowcytometry analysis, intracellular reactive oxygen species (ROS) was found to be responsible for CTGF upregulation. In addition, the effects of 9‐POHSA were likely associated with its inhibition of the activation of the NF‐kB. These results suggest that 9‐POHSA has potential as a therapy for liver inflammation and fibrosis by attenuating inflammation‐related indexes induced by LPS in hepatocytes. This study provides important insight into the mechanisms of liver inflammation and the potential for new treatments to address liver diseases.
Keywords	anti-inflammatory activity ; cell movement ; fatty liver ; fibrosis ; hepatitis ; hepatocytes ; inflammation ; insulin ; interleukin-6 ; lipopolysaccharides ; liver ; necrosis ; neoplasms ; palmitoleic acid ; rats ; reactive oxygen species ; stearic acid ; therapeutics ; transcription factor NF-kappa B
Language	English
Dates of publication	2023-09
Size	p. 241-249.
Publishing place	John Wiley & Sons, Inc.
Document type	Article ; Online
Note	JOURNAL ARTICLE
ZDB-ID	241539-2
ISSN	1558-9307 ; 0024-4201
ISSN (online)	1558-9307
ISSN	0024-4201
DOI	10.1002/lipd.12380
Database	NAL-Catalogue (AGRICOLA)

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Zs.A 645: Show issues

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Article: Protective effects of

Lin, Heng-Dao / Lee, Yuan-Chieh / Chiang, Chien-Yi / Lin, Yu-Jung / Shih, Cheng Yen / Tsai, Rong-Kung / Lin, Pi-Yu / Lin, Shinn-Zong / Ho, Tsung-Jung / Huang, Chih-Yang

Frontiers in nutrition

2023 Volume 10, Page(s) 1085248

Abstract: Diabetic retinopathy (DR) is a major cause of vision loss in diabetic patients. Hyperglycemia-induced oxidative stress and the accumulation of inflammatory factors result in blood-retinal barrier dysfunction and the pathogenesis of DR. ...

Abstract	Diabetic retinopathy (DR) is a major cause of vision loss in diabetic patients. Hyperglycemia-induced oxidative stress and the accumulation of inflammatory factors result in blood-retinal barrier dysfunction and the pathogenesis of DR.
Language	English
Publishing date	2023-03-30
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2776676-7
ISSN	2296-861X
ISSN	2296-861X
DOI	10.3389/fnut.2023.1085248
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Article ; Online: Human adipose-derived stem cells preconditioned with a novel herbal formulation Jing Shi attenuate doxorubicin-induced cardiac damage.

Hsieh, Dennis Jine-Yuan / Tsai, Bruce Chi-Kang / Barik, Parthasarathi / Shibu, Marthandam Asokan / Kuo, Chia-Hua / Kuo, Wei-Wen / Lin, Pi-Yu / Shih, Cheng-Yen / Lin, Shinn-Zong / Ho, Tsung-Jung / Huang, Chih-Yang

Aging

2023 Volume 15, Issue 17, Page(s) 9167–9181

Abstract: Pathological cardiac hypertrophy is a considerable contributor to global disease burden. Chinese herbal medicine (CHM) has been used to treat cardiovascular diseases since antiquity. Enhancing stem cell-mediated recovery through CHM represents a ... ...

Abstract	Pathological cardiac hypertrophy is a considerable contributor to global disease burden. Chinese herbal medicine (CHM) has been used to treat cardiovascular diseases since antiquity. Enhancing stem cell-mediated recovery through CHM represents a promising approach for protection against doxorubicin (Dox)-induced cardiac hypertrophy. Herein, we investigated whether human adipose-derived stem cells (hADSCs) preconditioned with novel herbal formulation Jing Si (JS) improved protective ability of stem cells against doxorubicin-induced cardiac damage. The effect of JS on hADSC viability and migration capacity was determined via MTT and migration assays, respectively. Co-culture of hADSC or JS-preconditioned hADSCs with H9c2 cells was analyzed with immunoblot, flow cytometry, TUNEL staining, LC3B staining, F-actin staining, and MitoSOX staining. The
MeSH term(s)	Humans ; Animals ; Rats ; Rats, Sprague-Dawley ; Heart ; Stem Cells ; Doxorubicin/toxicity ; Cardiomegaly
Chemical Substances	Doxorubicin (80168379AG)
Language	English
Publishing date	2023-09-02
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ISSN	1945-4589
ISSN (online)	1945-4589
DOI	10.18632/aging.205026
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Article ; Online: A combination of isoliquiritigenin with Artemisia argyi and Ohwia caudata water extracts attenuates oxidative stress, inflammation, and apoptosis by modulating Nrf2/Ho-1 signaling pathways in SD rats with doxorubicin-induced acute cardiotoxicity.

Yuan Hsieh, Dennis Jine / Islam, Md Nazmul / Kuo, Wei-Wen / Shibu, Marthandam Asokan / Lai, Chin-Hu / Lin, Pi-Yu / Lin, Shinn-Zong / Chen, Michael Yu-Chih / Huang, Chih-Yang

Environmental toxicology

2023 Volume 38, Issue 12, Page(s) 3026–3042

Abstract: Ohwia caudata (Thunb.) H. Ohashi (Leguminosae) also called as "Evergreen shrub" and Artemisia argyi H.Lév. and Vaniot (Compositae) also named as "Chinese mugwort" those two-leaf extracts frequently used as herbal medicine, especially in south east Asia ... ...

Abstract	Ohwia caudata (Thunb.) H. Ohashi (Leguminosae) also called as "Evergreen shrub" and Artemisia argyi H.Lév. and Vaniot (Compositae) also named as "Chinese mugwort" those two-leaf extracts frequently used as herbal medicine, especially in south east Asia and eastern Asia. Anthracyclines such as doxorubicin (DOX) are commonly used as effective chemotherapeutic drugs in anticancer therapy around the world. However, chemotherapy-induced cardiotoxicity, dilated cardiomyopathy, and congestive heart failure are seen in patients who receive DOX therapy, with the mechanisms underlying DOX-induced cardiac toxicity remaining unclear. Mitochondrial dysfunction, oxidative stress, inflammatory response, and cardiomyocytes have been shown to play crucial roles in DOX-induced cardiotoxicity. Isoliquiritigenin (ISL, 10 mg/kg) is a bioactive flavonoid compound with protective effects against inflammation, neurodegeneration, cancer, and diabetes. Here, in this study, our aim is to find out the Artemisia argyi (AA) and Ohwia caudata (OC) leaf extract combination with Isoliquiritigenin in potentiating and complementing effect against chemo drug side effect to ameliorate cardiac damage and improve the cardiac function. In this study, we showed that a combination of low (AA 300 mg/kg; OC 100 mg/kg) and high-dose(AA 600 mg/kg; OC 300 mg/kg) AA and OC water extract with ISL activated the cell survival-related AKT/PI3K signaling pathway in DOX-treated cardiac tissue leading to the upregulation of the antioxidant markers SOD, HO-1, and Keap-1 and regulated mitochondrial dysfunction through the Nrf2 signaling pathway. Moreover, the water extract of AA and OC with ISL inhibited the inflammatory response genes IL-6 and IL-1β, possibly through the NFκB/AKT/PI3K/p38α/NRLP3 signaling pathways. The water extract of AA and OC with ISL could be a potential herbal drug treatment for cardiac hypertrophy, inflammatory disease, and apoptosis, which can lead to sudden heart failure.
MeSH term(s)	Animals ; Rats ; Apoptosis ; Artemisia/chemistry ; Cardiotoxicity/drug therapy ; Cardiotoxicity/metabolism ; Doxorubicin/toxicity ; Inflammation/chemically induced ; Inflammation/drug therapy ; Inflammation/metabolism ; Myocytes, Cardiac ; NF-E2-Related Factor 2/drug effects ; NF-E2-Related Factor 2/metabolism ; Oxidative Stress/drug effects ; Phosphatidylinositol 3-Kinases/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; Rats, Sprague-Dawley ; Signal Transduction ; Plant Extracts/pharmacology ; Plant Extracts/therapeutic use ; Heme Oxygenase-1/drug effects ; Heme Oxygenase-1/metabolism
Chemical Substances	Doxorubicin (80168379AG) ; isoliquiritigenin (B9CTI9GB8F) ; NF-E2-Related Factor 2 ; Phosphatidylinositol 3-Kinases (EC 2.7.1.-) ; Proto-Oncogene Proteins c-akt (EC 2.7.11.1) ; Plant Extracts ; Heme Oxygenase-1 (EC 1.14.14.18)
Language	English
Publishing date	2023-09-04
Publishing country	United States
Document type	Journal Article
ZDB-ID	1463449-1
ISSN	1522-7278 ; 1520-4081
ISSN (online)	1522-7278
ISSN	1520-4081
DOI	10.1002/tox.23936
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Article ; Online: Ohwia caudata aqueous extract attenuates doxorubicin-induced mitochondrial dysfunction in Wharton's jelly-derived mesenchymal stem cells.

Lee, Pei-Ying / Tsai, Bruce Chi-Kang / Sitorus, Maria Angelina / Lin, Pi-Yu / Lin, Shinn-Zong / Shih, Cheng-Yen / Lu, Shang-Yeh / Lin, Yueh-Min / Ho, Tsung-Jung / Huang, Chih-Yang

Environmental toxicology

2023 Volume 38, Issue 10, Page(s) 2450–2461

Abstract: Mitochondrial dysfunction has been linked to many diseases, including organ degeneration and cancer. Wharton's jelly-derived mesenchymal stem cells provide a valuable source for stem cell-based therapy and represent an emerging therapeutic approach for ... ...

Abstract	Mitochondrial dysfunction has been linked to many diseases, including organ degeneration and cancer. Wharton's jelly-derived mesenchymal stem cells provide a valuable source for stem cell-based therapy and represent an emerging therapeutic approach for tissue regeneration. This study focused on screening the senomorphic properties of Ohwia caudata aqueous extract as an emerging strategy for preventing or treating mitochondrial dysfunction in stem cells. Wharton's jelly-derived mesenchymal stem cells were incubated with 0.1 μM doxorubicin, for 24 h to induce mitochondrial dysfunction. Next, the cells were treated with a series concentration of Ohwia caudata aqueous extract (25, 50, 100, and 200 μg/mL) for another 24 h. In addition, an untreated control group and a doxorubicin-induced mitochondrial dysfunction positive control group were maintained under the same conditions. Our data showed that Ohwia caudata aqueous extract markedly suppressed doxorubicin-induced mitochondrial dysfunction by increasing Tid1 and Tom20 expression, decreased reactive oxygen species production, and maintained mitochondrial membrane potential to promote mitochondrial stability. Ohwia caudata aqueous extract retained the stemness of Wharton's jelly-derived mesenchymal stem cells and reduced the apoptotic rate. These results indicate that Ohwia caudata aqueous extract protects Wharton's jelly-derived mesenchymal stem cells against doxorubicin-induced mitochondrial dysfunction and can potentially prevent mitochondrial dysfunction in other cells. This study provides new directions for the medical application of Ohwia caudata.
MeSH term(s)	Animals ; Wharton Jelly/metabolism ; Mesenchymal Stem Cells/metabolism ; Doxorubicin/toxicity ; Cells, Cultured ; Mitochondria/metabolism ; Urodela ; Cell Differentiation
Chemical Substances	Doxorubicin (80168379AG)
Language	English
Publishing date	2023-07-17
Publishing country	United States
Document type	Journal Article
ZDB-ID	1463449-1
ISSN	1522-7278 ; 1520-4081
ISSN (online)	1522-7278
ISSN	1520-4081
DOI	10.1002/tox.23880
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