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  1. Article ; Online: Estrogen Receptor Bio-Activities Determine Clinical Endocrine Treatment Options in Estrogen Receptor-Positive Breast Cancer.

    Xia, Song / Lin, Qiong

    Technology in cancer research & treatment

    2022  Volume 21, Page(s) 15330338221090351

    Abstract: In estrogen receptor positive (ER+) breast cancer therapy, estrogen receptors (ERs) are the major targeting molecules. ER-targeted therapy has provided clinical benefits for approximately 70% of all breast cancer patients through targeting the ERα ... ...

    Abstract In estrogen receptor positive (ER+) breast cancer therapy, estrogen receptors (ERs) are the major targeting molecules. ER-targeted therapy has provided clinical benefits for approximately 70% of all breast cancer patients through targeting the ERα subtype. In recent years, mechanisms underlying breast cancer occurrence and progression have been extensively studied and largely clarified. The PI3K/AKT/mTOR pathway, microRNA regulation, and other ER downstream signaling pathways are found to be the effective therapeutic targets in ER+ BC therapy. A number of the ER+ (ER+) breast cancer biomarkers have been established for diagnosis and prognosis. The
    MeSH term(s) Biomarkers, Tumor ; Breast Neoplasms/diagnosis ; Breast Neoplasms/drug therapy ; Breast Neoplasms/genetics ; Drug Resistance, Neoplasm/genetics ; Estrogen Receptor alpha/genetics ; Estrogen Receptor alpha/metabolism ; Female ; Humans ; Phosphatidylinositol 3-Kinases/metabolism ; Receptors, Estrogen/genetics ; Receptors, Estrogen/metabolism
    Chemical Substances Biomarkers, Tumor ; Estrogen Receptor alpha ; Receptors, Estrogen
    Language English
    Publishing date 2022-04-08
    Publishing country United States
    Document type Journal Article ; Review ; Research Support, Non-U.S. Gov't
    ZDB-ID 2146365-7
    ISSN 1533-0338 ; 1533-0346
    ISSN (online) 1533-0338
    ISSN 1533-0346
    DOI 10.1177/15330338221090351
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    In stock of ZB MED Cologne/Königswinter

    Zs.A 5871: Show issues Location:
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  2. Article: The Role of HECT E3 Ubiquitin Ligases in Colorectal Cancer.

    Sun, Aiqin / Chen, Yifei / Tian, Xianyan / Lin, Qiong

    Biomedicines

    2023  Volume 11, Issue 2

    Abstract: Colorectal cancer (CRC) is estimated to rank as the second reason for cancer-related deaths, and the prognosis of CRC patients remains unsatisfactory. Numerous studies on gastrointestinal cell biology have shown that the E3 ligase-mediated ubiquitination ...

    Abstract Colorectal cancer (CRC) is estimated to rank as the second reason for cancer-related deaths, and the prognosis of CRC patients remains unsatisfactory. Numerous studies on gastrointestinal cell biology have shown that the E3 ligase-mediated ubiquitination exerts key functions in the pathogenesis of CRC. The homologous to E6-associated protein C-terminus (HECT) family E3 ligases are a major group of E3 enzymes, featured with the presence of a catalytic HECT domain, which participate in multiple cellular processes; thus, alterations in HECT E3 ligases in function or expression are closely related to the occurrence and development of many human malignancies, including-but not limited to-CRC. In this review, we summarize the potential role of HECT E3 ligases in colorectal carcinogenesis and the related underlying molecular mechanism to expand our understanding of their pathological functions. Exploiting specific inhibitors targeting HECT E3 ligases could be a potential therapeutic strategy for CRC therapy in the future.
    Language English
    Publishing date 2023-02-07
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 2720867-9
    ISSN 2227-9059
    ISSN 2227-9059
    DOI 10.3390/biomedicines11020478
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  3. Article ; Online: Non-hippo kinases: indispensable roles in YAP/TAZ signaling and implications in cancer therapy.

    Zhu, Jun / Wu, Tiantian / Lin, Qiong

    Molecular biology reports

    2023  Volume 50, Issue 5, Page(s) 4565–4578

    Abstract: The transcriptional co-activators Yes-associated protein (YAP) and PDZ-binding domain (TAZ) are the known downstream effectors of the Hippo kinase cascade. YAP/TAZ have been shown to play important roles in cellular growth and differentiation, tissue ... ...

    Abstract The transcriptional co-activators Yes-associated protein (YAP) and PDZ-binding domain (TAZ) are the known downstream effectors of the Hippo kinase cascade. YAP/TAZ have been shown to play important roles in cellular growth and differentiation, tissue development and carcinogenesis. Recent studies have found that, in addition to the Hippo kinase cascade, multiple non-Hippo kinases also regulate the YAP/TAZ cellular signaling and produce important effects on cellular functions, particularly on tumorigenesis and progression. In this article, we will review the multifaceted regulation of the YAP/TAZ signaling by the non-Hippo kinases and discuss the potential application of the non-Hippo kinase-regulated YAP/TAZ signaling for cancer therapy.
    MeSH term(s) Humans ; Adaptor Proteins, Signal Transducing/metabolism ; Protein Serine-Threonine Kinases/metabolism ; Intracellular Signaling Peptides and Proteins/metabolism ; Trans-Activators/metabolism ; YAP-Signaling Proteins ; Transcriptional Coactivator with PDZ-Binding Motif Proteins ; Hippo Signaling Pathway ; Phosphoproteins/metabolism ; Signal Transduction ; Transcription Factors/metabolism ; Carcinogenesis
    Chemical Substances Adaptor Proteins, Signal Transducing ; Protein Serine-Threonine Kinases (EC 2.7.11.1) ; Intracellular Signaling Peptides and Proteins ; Trans-Activators ; YAP-Signaling Proteins ; Transcriptional Coactivator with PDZ-Binding Motif Proteins ; Phosphoproteins ; Transcription Factors
    Language English
    Publishing date 2023-03-06
    Publishing country Netherlands
    Document type Journal Article ; Review
    ZDB-ID 186544-4
    ISSN 1573-4978 ; 0301-4851
    ISSN (online) 1573-4978
    ISSN 0301-4851
    DOI 10.1007/s11033-023-08329-0
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    In stock of ZB MED Cologne/Königswinter

    Zs.A 1140: Show issues Location:
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  4. Article ; Online: Promoting or inhibiting: the impact of China's urban-rural digital divide on regional environmental development.

    Yan, Yuecen / Cheng, Li / Lin, Qiong / He, Qiang

    Environmental science and pollution research international

    2023  Volume 30, Issue 52, Page(s) 112710–112724

    Abstract: Based on the panel data of 30 provinces in China from 2013 to 2020, this paper uses the panel fixed effect model and threshold regression method to systematically examine the impact of the urban-rural digital divide on regional environmental development ... ...

    Abstract Based on the panel data of 30 provinces in China from 2013 to 2020, this paper uses the panel fixed effect model and threshold regression method to systematically examine the impact of the urban-rural digital divide on regional environmental development from the perspective of technological innovation and human capital. The study found that the urban-rural digital divide significantly inhibited regional environmental development; however, this inhibitory effect will be substantially reduced in technological innovation and human capital adjustment. Furthermore, technological innovation and human capital have played a significant single threshold effect in the impact of the urban-rural digital divide on regional environmental development. When technological innovation and human capital cross the corresponding threshold value, the adverse effects of the urban-rural digital divide on the regional environment will be alleviated. Therefore, it is suggested to strengthen the construction of rural information infrastructure; research and develop key technologies for ecological protection and environmental governance; build an ecological environment science and technology innovation system; and promote the sharing of human resources in the region and the ability of rural residents to apply digital resources and digital technology.
    MeSH term(s) Humans ; Conservation of Natural Resources ; Digital Divide ; Environmental Policy ; China ; Drug-Related Side Effects and Adverse Reactions ; Economic Development
    Language English
    Publishing date 2023-10-14
    Publishing country Germany
    Document type Journal Article
    ZDB-ID 1178791-0
    ISSN 1614-7499 ; 0944-1344
    ISSN (online) 1614-7499
    ISSN 0944-1344
    DOI 10.1007/s11356-023-30346-6
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    Z 5915: Show issues

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  5. Article: Probiotics for the treatment of ulcerative colitis: a review of experimental research from 2018 to 2022.

    Huang, Cuilan / Hao, Wujuan / Wang, Xuyang / Zhou, Renmin / Lin, Qiong

    Frontiers in microbiology

    2023  Volume 14, Page(s) 1211271

    Abstract: Ulcerative colitis (UC) has become a worldwide public health problem, and the prevalence of the disease among children has been increasing. The pathogenesis of UC has not been elucidated, but dysbiosis of the gut microbiota is considered the main cause ... ...

    Abstract Ulcerative colitis (UC) has become a worldwide public health problem, and the prevalence of the disease among children has been increasing. The pathogenesis of UC has not been elucidated, but dysbiosis of the gut microbiota is considered the main cause of chronic intestinal inflammation. This review focuses on the therapeutic effects of probiotics on UC and the potential mechanisms involved. In animal studies, probiotics have been shown to alleviate symptoms of UC, including weight loss, diarrhea, blood in the stool, and a shortened colon length, while also restoring intestinal microecological homeostasis, improving gut barrier function, modulating the intestinal immune response, and attenuating intestinal inflammation, thereby providing theoretical support for the development of probiotic-based microbial products as an adjunctive therapy for UC. However, the efficacy of probiotics is influenced by factors such as the bacterial strain, dose, and form. Hence, the mechanisms of action need to be investigated further. Relevant clinical trials are currently lacking, so the extension of animal experimental findings to clinical application requires a longer period of consideration for validation.
    Language English
    Publishing date 2023-07-06
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 2587354-4
    ISSN 1664-302X
    ISSN 1664-302X
    DOI 10.3389/fmicb.2023.1211271
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  6. Article ; Online: Emerging roles of the HECT E3 ubiquitin ligases in gastric cancer.

    Sun, Aiqin / Tian, Xianyan / Chen, Yifei / Yang, Wannian / Lin, Qiong

    Pathology oncology research : POR

    2023  Volume 29, Page(s) 1610931

    Abstract: Gastric cancer (GC) is one of the most pernicious gastrointestinal tumors with extraordinarily high incidence and mortality. Ubiquitination modification of cellular signaling proteins has been shown to play important roles in GC tumorigenesis, ... ...

    Abstract Gastric cancer (GC) is one of the most pernicious gastrointestinal tumors with extraordinarily high incidence and mortality. Ubiquitination modification of cellular signaling proteins has been shown to play important roles in GC tumorigenesis, progression, and prognosis. The E3 ubiquitin ligase is the crucial enzyme in the ubiquitination reaction and determines the specificity of ubiquitination substrates, and thus, the cellular effects. The HECT E3 ligases are the second largest E3 ubiquitin ligase family characterized by containing a HECT domain that has E3 ubiquitin ligase activity. The HECT E3 ubiquitin ligases have been found to engage in GC progression. However, whether HECT E3 ligases function as tumor promoters or tumor suppressors in GC remains controversial. In this review, we will focus on recent discoveries about the role of the HECT E3 ubiquitin ligases, especially members of the NEDD4 and other HECT E3 ligase subfamilies, in GC.
    MeSH term(s) Humans ; Ubiquitin-Protein Ligases/genetics ; Stomach Neoplasms ; Ubiquitination ; Carcinogenesis ; Ubiquitins ; Nedd4 Ubiquitin Protein Ligases/chemistry ; Nedd4 Ubiquitin Protein Ligases/genetics ; Nedd4 Ubiquitin Protein Ligases/metabolism
    Chemical Substances Ubiquitin-Protein Ligases (EC 2.3.2.27) ; Ubiquitins ; Nedd4 Ubiquitin Protein Ligases (EC 2.3.2.26)
    Language English
    Publishing date 2023-02-07
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 1375979-6
    ISSN 1532-2807 ; 1219-4956
    ISSN (online) 1532-2807
    ISSN 1219-4956
    DOI 10.3389/pore.2023.1610931
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    In stock of ZB MED Cologne/Königswinter

    Zs.A 4936: Show issues Location:
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    Jg. 1995 - 2021: Lesesall (2.OG)
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  7. Article ; Online: NEDD4 E3 ubiquitin ligases: Promising biomarkers and therapeutic targets for cancer.

    Tian, Xianyan / Chen, Yifei / Peng, Ziluo / Lin, Qiong / Sun, Aiqin

    Biochemical pharmacology

    2023  Volume 214, Page(s) 115641

    Abstract: Accumulating evidence has demonstrated that NEDD4 E3 ubiquitin ligase family plays a pivotal oncogenic role in a variety of malignancies via mediating ubiquitin dependent degradation processes. Moreover, aberrant expression of NEDD4 E3 ubiquitin ligases ... ...

    Abstract Accumulating evidence has demonstrated that NEDD4 E3 ubiquitin ligase family plays a pivotal oncogenic role in a variety of malignancies via mediating ubiquitin dependent degradation processes. Moreover, aberrant expression of NEDD4 E3 ubiquitin ligases is often indicative of cancer progression and correlated with poor prognosis. In this review, we are going to address association of expression of NEDD4 E3 ubiquitin ligases with cancers, the signaling pathways and the molecular mechanisms by which the NEDD4 E3 ubiquitin ligases regulate oncogenesis and progression, and the therapies targeting the NEDD4 E3 ubiquitin ligases. This review provides the systematic and comprehensive summary of the latest research status of E3 ubiquitin ligases in the NEDD4 subfamily, and proposes that NEDD4 family E3 ubiquitin ligases are promising anti-cancer drug targets, aiming to provide research direction for clinical targeting of NEDD4 E3 ubiquitin ligase therapy.
    MeSH term(s) Humans ; Ubiquitination ; Endosomal Sorting Complexes Required for Transport ; Nedd4 Ubiquitin Protein Ligases/metabolism ; Ubiquitin-Protein Ligases/metabolism ; Neoplasms/drug therapy ; Ubiquitin/metabolism
    Chemical Substances Endosomal Sorting Complexes Required for Transport ; Nedd4 Ubiquitin Protein Ligases (EC 2.3.2.26) ; Ubiquitin-Protein Ligases (EC 2.3.2.27) ; Ubiquitin
    Language English
    Publishing date 2023-06-10
    Publishing country England
    Document type Journal Article ; Review ; Research Support, Non-U.S. Gov't
    ZDB-ID 208787-x
    ISSN 1873-2968 ; 0006-2952
    ISSN (online) 1873-2968
    ISSN 0006-2952
    DOI 10.1016/j.bcp.2023.115641
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    Zs.A 19: Show issues Location:
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  8. Article ; Online: Overexpression of SCYL1 Is Associated with Progression of Breast Cancer.

    Sun, Aiqin / Tian, Xianyan / Yang, Wannian / Lin, Qiong

    Current oncology (Toronto, Ont.)

    2022  Volume 29, Issue 10, Page(s) 6922–6932

    Abstract: SCYL1 is a pseudokinase and plays roles in cell division and gene transcription, nuclear/cytoplasmic shuttling of tRNA, protein glycosylation, and Golgi morphology. However, the role of SCYL1 in human breast cancer progression remains largely unknown. In ...

    Abstract SCYL1 is a pseudokinase and plays roles in cell division and gene transcription, nuclear/cytoplasmic shuttling of tRNA, protein glycosylation, and Golgi morphology. However, the role of SCYL1 in human breast cancer progression remains largely unknown. In this study, we determined expression of SCYL1 in breast cancer by searching the Cancer Genome Atlas (TCGA) and Tumor Immunoassay Resource (TIMER) databases. Meanwhile, we collected breast tumor tissue samples from 247 cases and detected expression of SCYL1 in the tumors using the tissue microarray assay (TMA). Association of SCYL1 with prognosis of breast cancer was determined based on the PrognoScan database. The results have shown that SCYL1 is overexpressed in breast cancer, and the expression of SCYL1 is associated with poor clinical outcomes of breast cancer patients. Furthermore, knockdown of SCYL1 by shRNAs significantly inhibited the proliferation and migration of breast cancer cells. Taken together, our data suggest that SCYL1 is a biomarker for poor prognosis of breast cancer, has a promoting role in breast cancer progression, and is a potential target for breast cancer therapy.
    MeSH term(s) Humans ; Female ; Breast Neoplasms/genetics ; Prognosis ; RNA, Transfer ; DNA-Binding Proteins ; Adaptor Proteins, Vesicular Transport/genetics ; Adaptor Proteins, Vesicular Transport/metabolism
    Chemical Substances RNA, Transfer (9014-25-9) ; SCYL1 protein, human ; DNA-Binding Proteins ; Adaptor Proteins, Vesicular Transport
    Language English
    Publishing date 2022-09-24
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 1236972-x
    ISSN 1718-7729 ; 1198-0052
    ISSN (online) 1718-7729
    ISSN 1198-0052
    DOI 10.3390/curroncol29100544
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    Zs.A 4305: Show issues Location:
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  9. Article ; Online: Frequent presence of major dust mite allergens in human digestive tissues of children with gastritis.

    Zhou, Ying / Yang, Shudong / Lin, Qiong / He, Qixin / Cui, Yubao

    Allergy

    2022  Volume 78, Issue 2, Page(s) 590–592

    MeSH term(s) Humans ; Child ; Animals ; Allergens ; Dust ; Gastritis/etiology ; Pyroglyphidae ; Antigens, Dermatophagoides
    Chemical Substances Allergens ; Dust ; Antigens, Dermatophagoides
    Language English
    Publishing date 2022-11-23
    Publishing country Denmark
    Document type Letter ; Research Support, Non-U.S. Gov't
    ZDB-ID 391933-x
    ISSN 1398-9995 ; 0105-4538
    ISSN (online) 1398-9995
    ISSN 0105-4538
    DOI 10.1111/all.15587
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    Uh III Zs.150: Show issues Location:
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  10. Article ; Online: Umbilical cord mesenchymal stem cells inhibited inflammation of bronchial epithelial cells by regulating Hedgehog pathway.

    Lin, Qiong / Yu, Tianxing / Li, Xiaohua / Lin, Xin / Fan, Yong / Xu, Liyu

    European journal of histochemistry : EJH

    2023  Volume 67, Issue 4

    Abstract: This study aimed to explore the role and mechanism of umbilical cord mesenchymal stem cells (UCMSCs) in regulating inflammation of bronchial epithelial cells. Transforming growth factor beta-1 (TGF-β1) was used to induce inflammation in human bronchial ... ...

    Abstract This study aimed to explore the role and mechanism of umbilical cord mesenchymal stem cells (UCMSCs) in regulating inflammation of bronchial epithelial cells. Transforming growth factor beta-1 (TGF-β1) was used to induce inflammation in human bronchial epithelial cells. Cell proliferation was detected through CCK8 and cell apoptosis was detected by Annexin V and propidium iodide double staining. E-cadherin and α-smooth muscle actin (α-SMA) were detected by immunofluorescence, and tumor necrosis factor alpha (TNF-α) and interleukin 6 (IL-6) in culture medium supernatant were detected by ELISA. The expression of E-cadherin, α-SMA, Sonic hedgehog (Shh), Gli1 and Snail was detected by Western blot analysis. Compared with the control group, bronchial epithelial cells treated with TGF-β1 showed significantly decreased proliferation, increased apoptosis, increased secretion of TNF-α and IL-6, increased expression of α-SMA, Shh, Gli1 and Snail and decreased E-cadherin expression. However, co-culture with UCMSCs inhibited TGF-β1-induced changes in human bronchial epithelial cell proliferation, apoptosis, secretion of TNF-α and IL-6 and activation of the Hedgehog pathway. In conclusion, UCMSCs have protective effects on TGF-β1-induced inflammation in human bronchial epithelial cells by regulating the Hedgehog pathway.
    MeSH term(s) Humans ; Transforming Growth Factor beta1/metabolism ; Hedgehog Proteins/metabolism ; Hedgehog Proteins/pharmacology ; Tumor Necrosis Factor-alpha/metabolism ; Interleukin-6/metabolism ; Zinc Finger Protein GLI1/metabolism ; Zinc Finger Protein GLI1/pharmacology ; Cadherins/metabolism ; Epithelial Cells/metabolism ; Mesenchymal Stem Cells/metabolism ; Umbilical Cord/metabolism
    Chemical Substances Transforming Growth Factor beta1 ; Hedgehog Proteins ; Tumor Necrosis Factor-alpha ; Interleukin-6 ; Zinc Finger Protein GLI1 ; Cadherins
    Language English
    Publishing date 2023-12-12
    Publishing country Italy
    Document type Journal Article
    ZDB-ID 1109511-8
    ISSN 2038-8306 ; 0391-7258 ; 1121-4201 ; 1121-760X
    ISSN (online) 2038-8306
    ISSN 0391-7258 ; 1121-4201 ; 1121-760X
    DOI 10.4081/ejh.2023.3908
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    Zs.A 739: Show issues Location:
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