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Article: Hospitality's ethical values and unethical employee behaviour: The mediating roles of work values and the moderating role of perceived organisational support.

Chen, Qiuping / Liu, Zijuan

2022 Volume 13, Page(s) 1063797

Abstract: In recent years, hotels have occasionally engaged in unethical behaviour. This has become an urgent problem that requires a solution. Based on social exchange theory, this study constructs a theoretical model of the relationship between hospitality's ... ...

Abstract	In recent years, hotels have occasionally engaged in unethical behaviour. This has become an urgent problem that requires a solution. Based on social exchange theory, this study constructs a theoretical model of the relationship between hospitality's ethical values and unethical behaviour. According to 543 questionnaires, the findings indicate that hospitality's ethical values negatively affect the unethical behaviour of employees. Work values played a part in the intermediary role between the two, and perceived organisational support significantly positively moderated the relationship between hospitality's ethical values and unethical behaviour. By exploring the logical relationship between hotels' and employees' morality, this study expands the research content and theoretical framework of unethical employee behaviour and helps to bridge the work values of hotels and individuals. Furthermore, it helps to build a good hotel ethical value system, which can effectively reduce and suppress the emergence of unethical employee behaviour.
Language	English
Publishing date	2022-11-28
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2563826-9
ISSN	1664-1078
ISSN	1664-1078
DOI	10.3389/fpsyg.2022.1063797
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Article ; Online: Arsenic Metabolism, Toxicity and Accumulation in the White Button Mushroom

Dong, Owen / Powers, Michael / Liu, Zijuan / Yoshinaga, Masafumi

Toxics

2022 Volume 10, Issue 10

Abstract: Mushrooms have unique properties in arsenic metabolism. In many commercial and wild-grown mushrooms, arsenobetaine (AsB), a non-toxic arsenical, was found as the dominant arsenic species. The AsB biosynthesis remains unknown, so we designed experiments ... ...

Abstract	Mushrooms have unique properties in arsenic metabolism. In many commercial and wild-grown mushrooms, arsenobetaine (AsB), a non-toxic arsenical, was found as the dominant arsenic species. The AsB biosynthesis remains unknown, so we designed experiments to study conditions for AsB formation in the white button mushroom,
Language	English
Publishing date	2022-09-22
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2733883-6
ISSN	2305-6304 ; 2305-6304
ISSN (online)	2305-6304
ISSN	2305-6304
DOI	10.3390/toxics10100554
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Article ; Online: SLC39A8 gene encoding a metal ion transporter: discovery and bench to bedside.

Nebert, Daniel W / Liu, Zijuan

Human genomics

2019 Volume 13, Issue Suppl 1, Page(s) 51

Abstract: SLC39A8 is an evolutionarily highly conserved gene that encodes the ZIP8 metal cation transporter in all vertebrates. SLC39A8 is ubiquitously expressed, including pluripotent embryonic stem cells; SLC39A8 expression occurs in every cell type examined. ... ...

Abstract	SLC39A8 is an evolutionarily highly conserved gene that encodes the ZIP8 metal cation transporter in all vertebrates. SLC39A8 is ubiquitously expressed, including pluripotent embryonic stem cells; SLC39A8 expression occurs in every cell type examined. Uptake of ZIP8-mediated Mn
MeSH term(s)	Animals ; Cation Transport Proteins/genetics ; Evolution, Molecular ; Glycosylation ; Humans ; Ions ; Metals/metabolism ; Organ Specificity ; Translational Research, Biomedical
Chemical Substances	Cation Transport Proteins ; Ions ; Metals
Language	English
Publishing date	2019-09-14
Publishing country	England
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	2147618-4
ISSN	1479-7364 ; 1479-7364
ISSN (online)	1479-7364
ISSN	1479-7364
DOI	10.1186/s40246-019-0233-3
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Article: First Report of Macrophomina phaseolina Causing Stalk Rot of Sacha Inchi (Plukenetia volubilis) in China

Wang, Weiwei / Liu, Zijuan / Song, Xiqiang / Wang, Wei

Plant disease. 2020 Feb., v. 104, no. 2

2020

Abstract: Sacha inchi (Plukenetia volubilis L.), a perennial oilseed plant in the Euphorbiaceae family, was introduced into China in 2006. The seeds of sacha inchi are an excellent source of essential fatty acid, protein, and some bioactive compounds, and the oil ... ...

Abstract	Sacha inchi (Plukenetia volubilis L.), a perennial oilseed plant in the Euphorbiaceae family, was introduced into China in 2006. The seeds of sacha inchi are an excellent source of essential fatty acid, protein, and some bioactive compounds, and the oil is one of the best edible oils in the world that can be used in disease prevention and cosmetics (Chirinos et al. 2013), so this plant is considered to be one of the most promising economic crops in China. In 2018, black and dusty speckles were observed on the stems of sacha inchi plants in the fields in Danzhou campus of Hainan University, and the disease incidence was about 3%. Naturally infected stem fragments (5 to 10 mm) were surface sterilized in 2% sodium hypochlorite (NaClO) for 2 min, rinsed with sterile double-distilled water five to seven times, plated on potato dextrose agar (PDA) medium, and incubated at 28°C. Three pure cultures were obtained for subsequent experiments. The isolates showed black colony color on PDA, and microscopically observed microsclerotia were 48.80 ± 5.85 μm in size. The molecular identification was done by amplifying the sequences of internal transcribed spacers (ITS) using primers ITS1 and ITS4, and EF-1α by using EF1-688F and EF1-1251R primers (Alves et al. 2008). The amplicons of ITS (MK922557) showed 100% similarity with M. phaseolina ITS of KR012878 and MH864176 in GenBank. The EF-1α (MK922558) revealed 100% and 99.77% identities with KF226710 and KX890093, respectively. In addition, MpKF1 and MpKR1 primers were used to amplify MpK, which is partial of ITS and specific for M. phaseolina (Babu et al. 2007), and a 287-bp sequence (MK922559) was obtained, which showed 100% and 99.7% similarity with M. phaseolina EU338443 and MK571619 accessions, respectively. Based on the morphological and molecular results, the isolates were identified as M. phaseolina (Tassi) Goid. For pathogenicity testing, the mycelium plugs from 4-day-old cultures growing on PDA were used to inoculate nine 1-month-old sacha inchi plants using the cut-stem inoculation technique, and uninoculated plants were used as a control (Twizeyimana et al. 2012). All plants were placed in the greenhouse at 28°C and 80% relative humidity. Black speckles appeared on the stems of inoculated plants in 18 days, and no symptoms were observed from the control plants. M. phaseolina was reisolated from inoculated plants, and the experiment was repeated with similar results. To our knowledge, this is the first report on M. phaseolina stalk rot of sacha inchi in China. Our findings enriched the pathogen library of sacha inchi and promoted the development of management strategies for the disease.
Keywords	bioactive compounds ; color ; cooking fats and oils ; cosmetics ; crops ; culture media ; disease incidence ; disease prevention ; DNA primers ; essential fatty acids ; greenhouses ; inoculation methods ; internal transcribed spacers ; Macrophomina phaseolina ; mycelium ; oilseed crops ; pathogenicity ; pathogens ; Plukenetia volubilis ; relative humidity ; sclerotia ; seeds ; sodium hypochlorite ; stems ; China
Language	English
Dates of publication	2020-02
Size	p. 570.
Publishing place	Plant Disease
Document type	Article
ZDB-ID	754182-x
ISSN	0191-2917
ISSN	0191-2917
DOI	10.1094/PDIS-06-19-1312-PDN
Database	NAL-Catalogue (AGRICOLA)

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Article ; Online: Endogenous FGF1 Deficiency Aggravates Doxorubicin-Induced Hepatotoxicity.

Gu, Chunjie / Liu, Zijuan / Li, Yingjian / Yi, Mei / Wang, Simeng / Fan, Xia / Sun, Da / Zhang, Chi / Yan, Xiaoqing / Wu, Guicheng

Toxics

2023 Volume 11, Issue 11

Abstract: Doxorubicin (DOX) is a broad-spectrum antineoplastic agent that widely used in clinic. However, its application is largely limited by its toxicity in multiple organs. Fibroblast growth factor 1 (FGF1) showed protective potential in various liver diseases, ...

Abstract	Doxorubicin (DOX) is a broad-spectrum antineoplastic agent that widely used in clinic. However, its application is largely limited by its toxicity in multiple organs. Fibroblast growth factor 1 (FGF1) showed protective potential in various liver diseases, but the role of endogenous FGF1 in DOX-induced liver damage is currently unknown. Both wild-type (WT) and FGF1 knockout (FGF1-KO) mice were treated with DOX. DOX induced loss of body weight and liver weight and elevation of ALT and AST in WT mice, which were aggravated by FGF1 deletion. FGF1 deletion exacerbated hepatic oxidative stress mirrored by further elevated 3-nitrosative modification of multiple proteins and malondialdehyde content. These were accompanied by blunted compensatively antioxidative responses indicated by impaired upregulation of nuclear factor erythroid 2-related factor 2 and its downstream antioxidant gene expression. The aggravated oxidative stress was coincided with exacerbated cell apoptosis in DOX-treated FGF1-KO mice reflected by further increased TUNEL positive cell staining and BCL-2-associated X expression and caspase 3 cleavage. These detrimental changes in DOX-treated FGF1-KO mice were associated with worsened intestinal fibrosis and increased upregulation fibrotic marker connective tissue growth factor and α-smooth muscle actin expression. However, DOX-induced hepatic inflammatory responses were not further affected by FGF1 deletion. These results demonstrate that endogenous FGF1 deficiency aggravates DOX-induced liver damage and FGF1 is a potential therapeutic target for treatment of DOX-associated hepatoxicity.
Language	English
Publishing date	2023-11-12
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2733883-6
ISSN	2305-6304 ; 2305-6304
ISSN (online)	2305-6304
ISSN	2305-6304
DOI	10.3390/toxics11110925
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Article ; Online: Loss of hepatic manganese transporter ZIP8 disrupts serum transferrin glycosylation and the glutamate-glutamine cycle.

Powers, Michael / Minchella, Dean / Gonzalez-Acevedo, Minelly / Escutia-Plaza, Daisy / Wu, Jiaqi / Heger, Chris / Milne, Ginger / Aschner, Michael / Liu, Zijuan

Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements (GMS)

2023 Volume 78, Page(s) 127184

Abstract: Background: ZIP8, encoded by SLC39A8, is a membrane transporter that facilitates the cellular uptake of divalent biometals including zinc (Zn), manganese (Mn), and iron (Fe). The hepatic system has long been accepted as the central modulator for whole- ... ...

Abstract	Background: ZIP8, encoded by SLC39A8, is a membrane transporter that facilitates the cellular uptake of divalent biometals including zinc (Zn), manganese (Mn), and iron (Fe). The hepatic system has long been accepted as the central modulator for whole-body biometal distribution. Earlier investigations suggest the propensity of ZIP8 to prioritize Mn influx, as opposed to Fe or Zn, in hepatocytes. Hepatic ZIP8 Mn transport is crucial for maintaining homeostasis of various Mn-dependent metalloenzymes and their associated pathways. Herein, we hypothesize that a drastic decrease in systemic Mn, via the loss of hepatic ZIP8, disrupts two unique cellular pathways, post-translational glycosylation and the glutamate-glutamine cycle. Methods: ZIP8 liver-specific knockout (LSKO) mice were chosen in an attempt to substantially decrease whole-body Mn levels. To further elucidate the role of Mn in serum glycosylation, a Mn-deficient diet was adopted in conjunction with the LSKO mice to model a near-complete loss of systemic Mn. After the treatment course, transferrin sialylation profiles were determined using imaged capillary isoelectric focusing (icIEF). We also investigated the role of Mn in the glutamate-glutamine cycle; the conversion of glutamate to glutamine in F/F and LSKO mice was assessed by the glutamine/glutamate ratio in cerebrospinal fluid (CSF) via HPLC-MS. An open-field study was ultimately conducted to check if these mice displayed atypical behavior. Results: Two major biological pathways were found to be significantly altered due to the loss of hepatic ZIP8. We identified a disparity between F/F and LSKO transferrin sialylation profiles that were exacerbated under a Mn-deficient diet. Additionally, we discovered a neurotransmitter imbalance between the levels of glutamine and glutamate, exclusive to LSKO mice. This was characterized by the decreased glutamine/glutamate ratio in CSF. Secondary to the neurotransmitter alteration, LSKO mice exhibited an increase in locomotor activity in an open-field. Conclusion: Our model successfully established a connection between the loss of hepatic ZIP8 and two Mn-dependent cellular pathways, namely, protein glycosylation and the glutamate-glutamine cycle.
MeSH term(s)	Mice ; Animals ; Manganese/metabolism ; Glycosylation ; Glutamine/metabolism ; Liver/metabolism ; Zinc/metabolism ; Mice, Knockout ; Transferrin/metabolism ; Membrane Transport Proteins/metabolism ; Glutamates/metabolism ; Cation Transport Proteins/genetics ; Cation Transport Proteins/metabolism
Chemical Substances	Manganese (42Z2K6ZL8P) ; Glutamine (0RH81L854J) ; Zinc (J41CSQ7QDS) ; Transferrin ; Membrane Transport Proteins ; Glutamates ; Slc39a8 protein, mouse ; Cation Transport Proteins
Language	English
Publishing date	2023-04-27
Publishing country	Germany
Document type	Journal Article
ZDB-ID	1236267-0
ISSN	1878-3252 ; 1611-602X ; 0946-672X
ISSN (online)	1878-3252 ; 1611-602X
ISSN	0946-672X
DOI	10.1016/j.jtemb.2023.127184
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Article: Roles of vertebrate aquaglyceroporins in arsenic transport and detoxification.

Liu, Zijuan

Advances in experimental medicine and biology

2009 Volume 679, Page(s) 71–81

Abstract: Aquaporins are important channel proteins that are responsible for the balance of cellular osmolarity and nutrient transport in vertebrates. Recently, new functions of these ancient channels have been found in the conduction of metalloid arsenic (As). ... ...

Abstract	Aquaporins are important channel proteins that are responsible for the balance of cellular osmolarity and nutrient transport in vertebrates. Recently, new functions of these ancient channels have been found in the conduction of metalloid arsenic (As). Chronic As exposure through contaminated water and food sources is associated with multiple human diseases and endangers millions of people's health worldwide. Therefore, identification of the As transport pathways is necessary to elucidate the mechanisms of As carcinogenesis. Arsenic detoxification systems have been studied in multiple vertebrates such as mammalian mouse, rat, humans and nonmammalian vertebrates. Multiple transporters and enzymes have been shown to be involved in As translocation and cellular transformation. In these vertebrates, members ofaquaglyceroporins, which include AQP7 in kidney and AQP9 in liver, catalyze uptake of inorganic trivalent arsenite [As(III)]. AQP9, the major liver aquaglyceroporin, conducts both inorganic As(III) and organic monomethylarsonous acid [MMA(III)], an intermediate that is generated during the cellular methylation. As a channel that facilitates a downhill movement of substances dependent on the concentration gradient, AQP9 may play an important role in the simultaneous influx of inorganic As(III) from blood to liver and efflux of As metabolite MMA(III) from liver to blood. In this chapter, we will discuss the function ofaquaglyceroporins ofvertebrates in uptake and detoxification of the metalloid As.
MeSH term(s)	Animals ; Aquaporins/metabolism ; Arsenic/chemistry ; Arsenicals/chemistry ; Biological Transport ; Carcinogens ; Humans ; Liver/drug effects ; Models, Biological ; Permeability ; Phylogeny ; Vertebrates/metabolism ; Water Pollutants, Chemical/analysis
Chemical Substances	Aquaporins ; Arsenicals ; Carcinogens ; Water Pollutants, Chemical ; Arsenic (N712M78A8G)
Language	English
Publishing date	2009-07-08
Publishing country	United States
Document type	Journal Article ; Review
ISSN	2214-8019 ; 0065-2598
ISSN (online)	2214-8019
ISSN	0065-2598
DOI	10.1007/978-1-4419-6315-4_6
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Article ; Online: Selenite Inhibits Notch Signaling in Cells and Mice.

Powers, Michael / Liu, Liu / Deemer, Dane / Chen, Selina / Scholl, Aaron / Yoshinaga, Masafumi / Liu, Zijuan

International journal of molecular sciences

2021 Volume 22, Issue 5

Abstract: Selenium is an essential micronutrient with a wide range of biological effects in mammals. The inorganic form of selenium, selenite, is supplemented to relieve individuals with selenium deficiency and to alleviate associated symptoms. Additionally, ... ...

Abstract	Selenium is an essential micronutrient with a wide range of biological effects in mammals. The inorganic form of selenium, selenite, is supplemented to relieve individuals with selenium deficiency and to alleviate associated symptoms. Additionally, physiological and supranutritional selenite have shown selectively higher affinity and toxicity towards cancer cells, highlighting their potential to serve as chemotherapeutic agents or adjuvants. At varying doses, selenite extensively regulates cellular signaling and modulates many cellular processes. In this study, we report the identification of Delta-Notch signaling as a previously uncharacterized selenite inhibited target. Our transcriptomic results in selenite treated primary mouse hepatocytes revealed that the transcription of Notch1, Notch2, Hes1, Maml1, Furin and c-Myc were all decreased following selenite treatment. We further showed that selenite can inhibit Notch1 expression in cultured MCF7 breast adenocarcinoma cells and HEPG2 liver carcinoma cells. In mice acutely treated with 2.5 mg/kg selenite via intraperitoneal injection, we found that Notch1 expression was drastically lowered in liver and kidney tissues by 90% and 70%, respectively. Combined, these results support selenite as a novel inhibitor of Notch signaling, and a plausible mechanism of inhibition has been proposed. This discovery highlights the potential value of selenite applied in a pathological context where Notch is a key drug target in diseases such as cancer, fibrosis, and neurodegenerative disorders.
MeSH term(s)	Animals ; Breast Neoplasms/drug therapy ; Breast Neoplasms/metabolism ; Cell Line, Tumor ; Female ; Hep G2 Cells ; Hepatocytes/drug effects ; Hepatocytes/metabolism ; Humans ; Liver Neoplasms/drug therapy ; Liver Neoplasms/metabolism ; MCF-7 Cells ; Male ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Receptors, Notch/metabolism ; Selenious Acid/pharmacology ; Selenium/metabolism ; Signal Transduction/drug effects ; Transcriptome/drug effects
Chemical Substances	Receptors, Notch ; Selenious Acid (F6A27P4Q4R) ; Selenium (H6241UJ22B)
Language	English
Publishing date	2021-03-03
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2019364-6
ISSN	1422-0067 ; 1422-0067 ; 1661-6596
ISSN (online)	1422-0067
ISSN	1422-0067 ; 1661-6596
DOI	10.3390/ijms22052518
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Article ; Online: Potential role of astrocyte angiotensin converting enzyme 2 in the neural transmission of COVID-19 and a neuroinflammatory state induced by smoking and vaping.

Zhang, Yong / Archie, Sabrina Rahman / Ghanwatkar, Yashwardhan / Sharma, Sejal / Nozohouri, Saeideh / Burks, Elizabeth / Mdzinarishvili, Alexander / Liu, Zijuan / Abbruscato, Thomas J

Fluids and barriers of the CNS

2022 Volume 19, Issue 1, Page(s) 46

Abstract: Background: Knowledge of the entry receptors responsible for SARS-CoV-2 is key to understand the neural transmission and pathogenesis of COVID-19 characterized by a neuroinflammatory scenario. Understanding the brain distribution of angiotensin ... ...

Abstract	Background: Knowledge of the entry receptors responsible for SARS-CoV-2 is key to understand the neural transmission and pathogenesis of COVID-19 characterized by a neuroinflammatory scenario. Understanding the brain distribution of angiotensin converting enzyme 2 (ACE2), the primary entry receptor for SARS-CoV-2, remains mixed. Smoking has been shown as a risk factor for COVID-19 severity and it is not clear how smoking exacerbates the neural pathogenesis in smokers. Methods: Immunohistochemistry, real-time PCR and western blot assays were used to systemically examine the spatial-, cell type- and isoform-specific expression of ACE2 in mouse brain and primary cultured brain cells. Experimental smoking exposure was conducted to evaluate the effect of smoking on brain expression. Results: We observed ubiquitous expression of ACE2 but uneven brain distribution, with high expression in the cerebral microvasculature, medulla oblongata, hypothalamus, subventricular zones, and meninges around medulla oblongata and hypothalamus. Co-staining with cell type-specific markers demonstrates ACE2 is primarily expressed in astrocytes around the microvasculature, medulla oblongata, hypothalamus, ventricular and subventricular zones of cerebral ventricles, and subependymal zones in rhinoceles and rostral migratory streams, radial glial cells in the lateral ventricular zones, tanycytes in the third ventricle, epithelial cells and stroma in the cerebral choroid plexus, as well as cerebral pericytes, but rarely detected in neurons and cerebral endothelial cells. ACE2 expression in astrocytes is further confirmed in primary cultured cells. Furthermore, isoform-specific analysis shows astrocyte ACE2 has the peptidase domain responsible for SARS-CoV-2 entry, indicating astrocytes are indeed vulnerable to SARS-CoV-2 infection. Finally, our data show experimental tobacco smoking and electronic nicotine vaping exposure increase proinflammatory and/or immunomodulatory cytokine IL-1a, IL-6 and IL-5 without significantly affecting ACE2 expression in the brain, suggesting smoking may pre-condition a neuroinflammatory state in the brain. Conclusions: The present study demonstrates a spatial- and cell type-specific expression of ACE2 in the brain, which might help to understand the acute and lasting post-infection neuropsychological manifestations in COVID-19 patients. Our data highlights a potential role of astrocyte ACE2 in the neural transmission and pathogenesis of COVID-19. This also suggests a pre-conditioned neuroinflammatory and immunocompromised scenario might attribute to exacerbated COVID-19 severity in the smokers.
MeSH term(s)	Angiotensin-Converting Enzyme 2 ; Animals ; Astrocytes ; COVID-19 ; Endothelial Cells ; Humans ; Mice ; SARS-CoV-2 ; Smoking/adverse effects ; Synaptic Transmission ; Tobacco Smoking ; Vaping
Chemical Substances	Angiotensin-Converting Enzyme 2 (EC 3.4.17.23)
Language	English
Publishing date	2022-06-07
Publishing country	England
Document type	Journal Article
ZDB-ID	2595406-4
ISSN	2045-8118 ; 2045-8118
ISSN (online)	2045-8118
ISSN	2045-8118
DOI	10.1186/s12987-022-00339-7
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Article ; Online: Correction: Potential role of astrocyte angiotensin converting enzyme 2 in the neural transmission of COVID-19 and a neuroinflammatory state induced by smoking and vaping.

Zhang, Yong / Archie, Sabrina Rahman / Ghanwatkar, Yashwardhan / Sharma, Sejal / Nozohouri, Saeideh / Burks, Elizabeth / Mdzinarishvili, Alexander / Liu, Zijuan / Abbruscato, Thomas J

Fluids and barriers of the CNS

2022 Volume 19, Issue 1, Page(s) 91

Language	English
Publishing date	2022-11-17
Publishing country	England
Document type	Published Erratum
ZDB-ID	2595406-4
ISSN	2045-8118 ; 2045-8118
ISSN (online)	2045-8118
ISSN	2045-8118
DOI	10.1186/s12987-022-00388-y
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