LIVIVO - Search results -

Search results

Result 1 - 10 of total 28

Search options

Article: Nuclear control of the inflammatory response in mammals by peroxisome proliferator-activated receptors.

Mandard, Stéphane / Patsouris, David

2013 Volume 2013, Page(s) 613864

Abstract: Peroxisome proliferator-activated receptors (PPARs) are ligand-activated transcription factors that play pivotal roles in the regulation of a very large number of biological processes including inflammation. Using specific examples, this paper focuses on ...

Abstract	Peroxisome proliferator-activated receptors (PPARs) are ligand-activated transcription factors that play pivotal roles in the regulation of a very large number of biological processes including inflammation. Using specific examples, this paper focuses on the interplay between PPARs and innate immunity/inflammation and, when possible, compares it among species. We focus on recent discoveries establishing how inflammation and PPARs interact in the context of obesity-induced inflammation and type 2 diabetes, mostly in mouse and humans. We illustrate that PPAR γ ability to alleviate obesity-associated inflammation raises an interesting pharmacologic potential. In the light of recent findings, the protective role of PPAR α and PPAR β / δ against the hepatic inflammatory response is also addressed. While PPARs agonists are well-established agents that can treat numerous inflammatory issues in rodents and humans, surprisingly very little has been described in other species. We therefore also review the implication of PPARs in inflammatory bowel disease; acute-phase response; and central, cardiac, and endothelial inflammation and compare it along different species (mainly mouse, rat, human, and pig). In the light of the data available in the literature, there is no doubt that more studies concerning the impact of PPAR ligands in livestock should be undertaken because it may finally raise unconsidered health and sanitary benefits.
Language	English
Publishing date	2013-03-07
Publishing country	United States
Document type	Journal Article
ZDB-ID	2237981-2
ISSN	1687-4765 ; 1687-4757
ISSN (online)	1687-4765
ISSN	1687-4757
DOI	10.1155/2013/613864
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

This service is chargeable due to the Delivery terms set by subito. Orders including an article and supplementary material will be classified as separate orders. In these cases, fees will be demanded for each order.

Article: Immune response of BV-2 microglial cells is impacted by peroxisomal beta-oxidation.

Frontiers in molecular neuroscience

2023 Volume 16, Page(s) 1299314

Abstract: Microglia are crucial for brain homeostasis, and dysfunction of these cells is a key driver in most neurodegenerative diseases, including peroxisomal leukodystrophies. In X-linked adrenoleukodystrophy (X-ALD), a neuroinflammatory disorder, very long- ... ...

Abstract	Microglia are crucial for brain homeostasis, and dysfunction of these cells is a key driver in most neurodegenerative diseases, including peroxisomal leukodystrophies. In X-linked adrenoleukodystrophy (X-ALD), a neuroinflammatory disorder, very long-chain fatty acid (VLCFA) accumulation due to impaired degradation within peroxisomes results in microglial defects, but the underlying mechanisms remain unclear. Using CRISPR/Cas9 gene editing of key genes in peroxisomal VLCFA breakdown (
Language	English
Publishing date	2023-12-18
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2452967-9
ISSN	1662-5099
ISSN	1662-5099
DOI	10.3389/fnmol.2023.1299314
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

Article ; Online: Human cholesteryl ester transfer protein lacks lipopolysaccharide transfer activity, but worsens inflammation and sepsis outcomes in mice.

Dusuel, Aloïs / Deckert, Valérie / Pais de Barros, Jean-Paul / van Dongen, Kevin / Choubley, Hélène / Charron, Émilie / Le Guern, Naig / Labbé, Jérôme / Mandard, Stéphane / Grober, Jacques / Lagrost, Laurent / Gautier, Thomas

Journal of lipid research

2020 Volume 62, Page(s) 100011

Abstract: Bacterial lipopolysaccharides (LPSs or endotoxins) can bind most proteins of the lipid transfer/LPS-binding protein (LT/LBP) family in host organisms. The LPS-bound LT/LBP proteins then trigger either an LPS-induced proinflammatory cascade or LPS binding ...

Abstract	Bacterial lipopolysaccharides (LPSs or endotoxins) can bind most proteins of the lipid transfer/LPS-binding protein (LT/LBP) family in host organisms. The LPS-bound LT/LBP proteins then trigger either an LPS-induced proinflammatory cascade or LPS binding to lipoproteins that are involved in endotoxin inactivation and detoxification. Cholesteryl ester transfer protein (CETP) is an LT/LBP member, but its impact on LPS metabolism and sepsis outcome is unclear. Here, we performed fluorescent LPS transfer assays to assess the ability of CETP to bind and transfer LPS. The effects of intravenous (iv) infusion of purified LPS or polymicrobial infection (cecal ligation and puncture [CLP]) were compared in transgenic mice expressing human CETP and wild-type mice naturally having no CETP activity. CETP displayed no LPS transfer activity in vitro, but it tended to reduce biliary excretion of LPS in vivo. The CETP expression in mice was associated with significantly lower basal plasma lipid levels and with higher mortality rates in both models of endotoxemia and sepsis. Furthermore, CETPTg plasma modified cytokine production of macrophages in vitro. In conclusion, despite having no direct LPS binding and transfer property, human CETP worsens sepsis outcomes in mice by altering the protective effects of plasma lipoproteins against endotoxemia, inflammation, and infection.
MeSH term(s)	Cholesterol Ester Transfer Proteins
Chemical Substances	CETP protein, human ; Cholesterol Ester Transfer Proteins
Language	English
Publishing date	2020-12-15
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	80154-9
ISSN	1539-7262 ; 0022-2275
ISSN (online)	1539-7262
ISSN	0022-2275
DOI	10.1194/jlr.RA120000704
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Zs.A 175: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
Jg. 1995 - 2021: Lesesall (1.OG)
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article: Physiological and Nutritional Roles of PPAR across Species.

Bionaz, Massimo / Hausman, Gary J / Loor, Juan J / Mandard, Stéphane

PPAR research

2013 Volume 2013, Page(s) 807156

Language	English
Publishing date	2013-05-15
Publishing country	United States
Document type	Journal Article
ZDB-ID	2237981-2
ISSN	1687-4765 ; 1687-4757
ISSN (online)	1687-4765
ISSN	1687-4757
DOI	10.1155/2013/807156
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

Article ; Online: Role of Diet and Nutrients in SARS-CoV-2 Infection: Incidence on Oxidative Stress, Inflammatory Status and Viral Production.

Nutrients

2022 Volume 14, Issue 11

Abstract: Coronavirus illness (COVID-19) is an infectious pathology generated by intense severe respiratory syndrome coronavirus 2 (SARS-CoV-2). This infectious disease has emerged in 2019. The COVID-19-associated pandemic has considerably affected the way of life ...

Abstract	Coronavirus illness (COVID-19) is an infectious pathology generated by intense severe respiratory syndrome coronavirus 2 (SARS-CoV-2). This infectious disease has emerged in 2019. The COVID-19-associated pandemic has considerably affected the way of life and the economy in the world. It is consequently crucial to find solutions allowing remedying or alleviating the effects of this infectious disease. Natural products have been in perpetual application from immemorial time given that they are attested to be efficient towards several illnesses without major side effects. Various studies have shown that plant extracts or purified molecules have a promising inhibiting impact towards coronavirus. In addition, it is substantial to understand the characteristics, susceptibility and impact of diet on patients infected with COVID-19. In this review, we recapitulate the influence of extracts or pure molecules from medicinal plants on COVID-19. We approach the possibilities of plant treatment/co-treatment and feeding applied to COVID-19. We also show coronavirus susceptibility and complications associated with nutrient deficiencies and then discuss the major food groups efficient on COVID-19 pathogenesis. Then, we covered emerging technologies using plant-based SARS-CoV-2 vaccine. We conclude by giving nutrient and plants curative therapy recommendations which are of potential interest in the COVID-19 infection and could pave the way for pharmacological treatments or co-treatments of COVID-19.
MeSH term(s)	Antiviral Agents/therapeutic use ; COVID-19 ; COVID-19 Vaccines ; Diet ; Humans ; Incidence ; Nutrients ; Oxidative Stress ; SARS-CoV-2
Chemical Substances	Antiviral Agents ; COVID-19 Vaccines
Language	English
Publishing date	2022-05-25
Publishing country	Switzerland
Document type	Journal Article ; Review
ZDB-ID	2518386-2
ISSN	2072-6643 ; 2072-6643
ISSN (online)	2072-6643
ISSN	2072-6643
DOI	10.3390/nu14112194
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

Article ; Online: Protective Effect of Cactus Cladode Extracts on Peroxisomal Functions in Microglial BV-2 Cells Activated by Different Lipopolysaccharides.

Saih, Fatima-Ezzahra / Andreoletti, Pierre / Mandard, Stéphane / Latruffe, Norbert / El Kebbaj, M'Hammed Saïd / Lizard, Gérard / Nasser, Boubker / Cherkaoui-Malki, Mustapha

Molecules (Basel, Switzerland)

2017 Volume 22, Issue 1

Abstract: In this study, we aimed to evaluate the antioxidant and anti-inflammatory properties ... ...

Abstract	In this study, we aimed to evaluate the antioxidant and anti-inflammatory properties of
MeSH term(s)	Animals ; Anti-Inflammatory Agents/pharmacology ; Antioxidants/pharmacology ; Catalase/metabolism ; Cell Line ; Escherichia coli ; Fatty Acids/metabolism ; Lipopolysaccharides ; Mice ; Microglia/cytology ; Microglia/drug effects ; Microglia/metabolism ; Neuroprotective Agents/pharmacology ; Nitric Oxide/metabolism ; Opuntia/chemistry ; Oxidation-Reduction/drug effects ; Oxidative Stress/drug effects ; Peroxisomes/metabolism ; Plant Extracts/pharmacology ; Reactive Oxygen Species/metabolism ; Salmonella
Chemical Substances	Anti-Inflammatory Agents ; Antioxidants ; Fatty Acids ; Lipopolysaccharides ; Neuroprotective Agents ; Plant Extracts ; Reactive Oxygen Species ; Nitric Oxide (31C4KY9ESH) ; Catalase (EC 1.11.1.6)
Language	English
Publishing date	2017-01-07
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	1413402-0
ISSN	1420-3049 ; 1431-5165 ; 1420-3049
ISSN (online)	1420-3049
ISSN	1431-5165 ; 1420-3049
DOI	10.3390/molecules22010102
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Zs.MO 81: Show issues

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: Inhibition of mitophagy drives macrophage activation and antibacterial defense during sepsis.

Patoli, Danish / Mignotte, Franck / Deckert, Valérie / Dusuel, Alois / Dumont, Adélie / Rieu, Aurélie / Jalil, Antoine / Van Dongen, Kevin / Bourgeois, Thibaut / Gautier, Thomas / Magnani, Charlène / Le Guern, Naig / Mandard, Stéphane / Bastin, Jean / Djouadi, Fatima / Schaeffer, Christine / Guillaumot, Nina / Narce, Michel / Nguyen, Maxime /

Guy, Julien / Dargent, Auguste / Quenot, Jean-Pierre / Rialland, Mickaël / Masson, David / Auwerx, Johan / Lagrost, Laurent / Thomas, Charles

The Journal of clinical investigation

2020 Volume 130, Issue 11, Page(s) 5858–5874

Abstract: Mitochondria have emerged as key actors of innate and adaptive immunity. Mitophagy has a pivotal role in cell homeostasis, but its contribution to macrophage functions and host defense remains to be delineated. Here, we showed that lipopolysaccharide ( ... ...

Abstract	Mitochondria have emerged as key actors of innate and adaptive immunity. Mitophagy has a pivotal role in cell homeostasis, but its contribution to macrophage functions and host defense remains to be delineated. Here, we showed that lipopolysaccharide (LPS) in combination with IFN-γ inhibited PINK1-dependent mitophagy in macrophages through a STAT1-dependent activation of the inflammatory caspases 1 and 11. In addition, we demonstrated that the inhibition of mitophagy triggered classical macrophage activation in a mitochondrial ROS-dependent manner. In a murine model of polymicrobial infection (cecal ligature and puncture), adoptive transfer of Pink1-deficient bone marrow or pharmacological inhibition of mitophagy promoted macrophage activation, which favored bactericidal clearance and led to a better survival rate. Reciprocally, mitochondrial uncouplers that promote mitophagy reversed LPS/IFN-γ-mediated activation of macrophages and led to immunoparalysis with impaired bacterial clearance and lowered survival. In critically ill patients, we showed that mitophagy was inhibited in blood monocytes of patients with sepsis as compared with nonseptic patients. Overall, this work demonstrates that the inhibition of mitophagy is a physiological mechanism that contributes to the activation of myeloid cells and improves the outcome of sepsis.
MeSH term(s)	Animals ; Bacteria/immunology ; Female ; Humans ; Interferon-gamma/immunology ; Lipopolysaccharides/immunology ; Macrophage Activation ; Macrophages, Peritoneal/immunology ; Macrophages, Peritoneal/microbiology ; Macrophages, Peritoneal/pathology ; Male ; Mice ; Mitophagy/immunology ; Protein Kinases/immunology ; RAW 264.7 Cells ; Sepsis/immunology ; Sepsis/microbiology ; Sepsis/pathology
Chemical Substances	IFNG protein, human ; IFNG protein, mouse ; Lipopolysaccharides ; Interferon-gamma (82115-62-6) ; Protein Kinases (EC 2.7.-) ; PTEN-induced putative kinase (EC 2.7.11.1)
Language	English
Publishing date	2020-08-05
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	3067-3
ISSN	1558-8238 ; 0021-9738
ISSN (online)	1558-8238
ISSN	0021-9738
DOI	10.1172/JCI130996
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Ua VI Zs.184: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: A role for peroxisome proliferator-activated receptor gamma in resveratrol-induced colon cancer cell apoptosis.

Aires, Virginie / Brassart, Bertrand / Carlier, Annie / Scagliarini, Alessandra / Mandard, Stéphane / Limagne, Emeric / Solary, Eric / Martiny, Laurent / Tarpin, Michel / Delmas, Dominique

Molecular nutrition & food research

2014 Volume 58, Issue 9, Page(s) 1785–1794

Abstract: Scope: Resveratrol may function as a chemopreventive agent. A recent clinical study demonstrates a reduction in tumor cell proliferation in colorectal patients receiving repeated oral ingestion of resveratrol. However, gaps remain in our knowledge of ... ...

Abstract	Scope: Resveratrol may function as a chemopreventive agent. A recent clinical study demonstrates a reduction in tumor cell proliferation in colorectal patients receiving repeated oral ingestion of resveratrol. However, gaps remain in our knowledge of the molecular mechanisms by which resveratrol exerts its chemopreventive effect. We have previously demonstrated that resveratrol induces apoptosis in colon cancer cells and that resveratrol can sensitize chemoresistant colon cancer cells to various drugs. Based on its ability to activate peroxisome proliferator-activated receptor gamma (PPARγ) in colon cancer cells, we sought to determine the implication of this nuclear transcription factor in resveratrol-induced apoptosis. Methods and results: Transient transfection of cancer cells with a dominant-negative PPARγ mutant or treatment with a PPARγ antagonist (GW9662) reversed the inhibitory effect of resveratrol. Moreover, GW9662 prevented disruption of the cell cycle induced by resveratrol and consequently abrogated resveratrol-induced apoptosis. Tumor cell death was potentiated by combining resveratrol with rosiglitazone, a PPARγ agonist. Conclusion: The results show that PPARγ plays a role in resveratrol-induced apoptosis of colon carcinoma cells. The combination of resveratrol with a PPARγ agonist could be a promising pharmacological approach for treatment of colorectal cancer.
MeSH term(s)	Anilides/pharmacology ; Apoptosis/drug effects ; Cell Line, Tumor/drug effects ; Cell Proliferation/drug effects ; Colonic Neoplasms/drug therapy ; Colonic Neoplasms/pathology ; Humans ; PPAR gamma/agonists ; PPAR gamma/antagonists & inhibitors ; PPAR gamma/genetics ; PPAR gamma/metabolism ; S Phase Cell Cycle Checkpoints/drug effects ; Stilbenes/pharmacology ; Thiazolidinediones/pharmacology
Chemical Substances	2-chloro-5-nitrobenzanilide ; Anilides ; PPAR gamma ; Stilbenes ; Thiazolidinediones ; rosiglitazone (05V02F2KDG) ; resveratrol (Q369O8926L)
Language	English
Publishing date	2014-09
Publishing country	Germany
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2160372-8
ISSN	1613-4133 ; 1613-4125
ISSN (online)	1613-4133
ISSN	1613-4125
DOI	10.1002/mnfr.201300962
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

Order via subito

Details ▾
- Full text online
- Order with fees

Article ; Online: Lipopolysaccharides-mediated increase in glucose-stimulated insulin secretion: involvement of the GLP-1 pathway.

Nguyen, Anh Thoai / Mandard, Stéphane / Dray, Cédric / Deckert, Valérie / Valet, Philippe / Besnard, Philippe / Drucker, Daniel J / Lagrost, Laurent / Grober, Jacques

Diabetes

2014 Volume 63, Issue 2, Page(s) 471–482

Abstract: Lipopolysaccharides (LPS) of the cell wall of gram-negative bacteria trigger inflammation, which is associated with marked changes in glucose metabolism. Hyperglycemia is frequently observed during bacterial infection and it is a marker of a poor ... ...

Abstract	Lipopolysaccharides (LPS) of the cell wall of gram-negative bacteria trigger inflammation, which is associated with marked changes in glucose metabolism. Hyperglycemia is frequently observed during bacterial infection and it is a marker of a poor clinical outcome in critically ill patients. The aim of the current study was to investigate the effect of an acute injection or continuous infusion of LPS on experimentally induced hyperglycemia in wild-type and genetically engineered mice. The acute injection of a single dose of LPS produced an increase in glucose disposal and glucose-stimulated insulin secretion (GSIS). Continuous infusion of LPS through mini-osmotic pumps was also associated with increased GSIS. Finally, manipulation of LPS detoxification by knocking out the plasma phospholipid transfer protein (PLTP) led to increased glucose disposal and GSIS. Overall, glucose tolerance and GSIS tests supported the hypothesis that mice treated with LPS develop glucose-induced hyperinsulinemia. The effects of LPS on glucose metabolism were significantly altered as a result of either the accumulation or antagonism of glucagon-like peptide 1 (GLP-1). Complementary studies in wild-type and GLP-1 receptor knockout mice further implicated the GLP-1 receptor-dependent pathway in mediating the LPS-mediated changes in glucose metabolism. Hence, enhanced GLP-1 secretion and action underlies the development of glucose-mediated hyperinsulinemia associated with endotoxemia.
MeSH term(s)	Animals ; Blood Glucose ; Glucagon-Like Peptide 1/genetics ; Glucagon-Like Peptide 1/metabolism ; Glucagon-Like Peptide-1 Receptor ; Glucose/metabolism ; Insulin/metabolism ; Lipopolysaccharides/metabolism ; Lipopolysaccharides/toxicity ; Mice ; Mice, Knockout ; Phospholipid Transfer Proteins/genetics ; Phospholipid Transfer Proteins/metabolism ; Receptors, Glucagon/genetics ; Receptors, Glucagon/metabolism
Chemical Substances	Blood Glucose ; Glp1r protein, mouse ; Glucagon-Like Peptide-1 Receptor ; Insulin ; Lipopolysaccharides ; Phospholipid Transfer Proteins ; Receptors, Glucagon ; phospholipid transfer protein, mouse ; Glucagon-Like Peptide 1 (89750-14-1) ; Glucose (IY9XDZ35W2)
Language	English
Publishing date	2014-02
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	80085-5
ISSN	1939-327X ; 0012-1797
ISSN (online)	1939-327X
ISSN	0012-1797
DOI	10.2337/db13-0903
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Uh III Zs.175: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: Protective Effect of Argan and Olive Oils against LPS-Induced Oxidative Stress and Inflammation in Mice Livers.

El Kamouni, Soufiane / El Kebbaj, Riad / Andreoletti, Pierre / El Ktaibi, Abderrahim / Rharrassi, Issam / Essamadi, Abdelkhalid / El Kebbaj, M'hammed Saïd / Mandard, Stéphane / Latruffe, Norbert / Vamecq, Joseph / Nasser, Boubker / Cherkaoui-Malki, Mustapha

International journal of molecular sciences

2017 Volume 18, Issue 10

Abstract: Sepsis causes severe dysregulation of organ functions, via the development of oxidative stress and inflammation. These pathophysiological mechanisms are mimicked in mice injected with bacterial lipopolysaccharide (LPS). Here, protective properties of ... ...

Abstract	Sepsis causes severe dysregulation of organ functions, via the development of oxidative stress and inflammation. These pathophysiological mechanisms are mimicked in mice injected with bacterial lipopolysaccharide (LPS). Here, protective properties of argan oil against LPS-induced oxidative stress and inflammation are explored in the murine model. Mice received standard chow, supplemented with argan oil (AO) or olive oil (OO) for 25 days, before septic shock was provoked with a single intraperitoneal injection of LPS, 16 hours prior to animal sacrifice. In addition to a rise in oxidative stress and inflammatory markers, injected LPS also caused hepatotoxicity, accompanied by hyperglycemia, hypercholesterolemia and hyperuremia. These LPS-associated toxic effects were blunted by AO pretreatment, as corroborated by normal plasma parameters and cell stress markers (glutathione: GSH) and antioxidant enzymology (catalase, CAT; superoxide dismutase, SOD and glutathione peroxidase, GPx). Hematoxylin-eosin staining revealed that AO can protect against acute liver injury, maintaining a normal status, which is pointed out by absent or reduced LPS-induced hepatic damage markers (i.e., alanine aminotransferase (ALT) and aspartate transaminase (AST)). Our work also indicated that AO displayed anti-inflammatory activity, due to down-regulations of genes encoding pro-inflammatory cytokines Interleukin-6 (IL-6) and Tumor Necrosis Factor-α (TNF-α) and in up-regulations of the expression of anti-inflammatory genes encoding Interleukin-4 (IL-4) and Interleukin-10 (IL-10). OO provided animals with similar, though less extensive, protective changes. Collectively our work adds compelling evidence to the protective mechanisms of AO against LPS-induced liver injury and hence therapeutic potentialities, in regard to the management of human sepsis. Activations of IL-4/Peroxisome Proliferator-Activated Receptors (IL-4/PPARs) signaling and, under LPS, an anti-inflammatory IL-10/Liver X Receptor (IL-10/LXR) route, obviously indicated the high potency and plasticity of the anti-inflammatory properties of argan oil.
MeSH term(s)	Animals ; Anti-Inflammatory Agents/administration & dosage ; Anti-Inflammatory Agents/pharmacology ; Anti-Inflammatory Agents/therapeutic use ; Antioxidants/administration & dosage ; Antioxidants/pharmacology ; Antioxidants/therapeutic use ; Dietary Supplements ; Lipopolysaccharides/toxicity ; Liver/drug effects ; Liver/metabolism ; Liver Diseases/drug therapy ; Liver Diseases/etiology ; Liver Diseases/prevention & control ; Mice ; Olive Oil/administration & dosage ; Olive Oil/pharmacology ; Olive Oil/therapeutic use ; Oxidative Stress ; Plant Oils/administration & dosage ; Plant Oils/pharmacology ; Plant Oils/therapeutic use
Chemical Substances	Anti-Inflammatory Agents ; Antioxidants ; Lipopolysaccharides ; Olive Oil ; Plant Oils ; argan oil (4V59G5UW9X)
Language	English
Publishing date	2017-10-19
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2019364-6
ISSN	1422-0067 ; 1422-0067 ; 1661-6596
ISSN (online)	1422-0067
ISSN	1422-0067 ; 1661-6596
DOI	10.3390/ijms18102181
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

To top

More links

Kategorien

Order via subito

More links

Kategorien

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

Order via subito

More links

Kategorien

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

Full text online

More links

Kategorien

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

Order via subito