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  1. Article ; Online: Calprotectin, a misnomer for another player in vascular calcification.

    Drueke, Tilman B / Massy, Ziad A

    Kidney international

    2024  Volume 105, Issue 5, Page(s) 915–918

    MeSH term(s) Humans ; Leukocyte L1 Antigen Complex ; Vascular Calcification/diagnostic imaging ; Signal Transduction
    Chemical Substances Leukocyte L1 Antigen Complex
    Language English
    Publishing date 2024-02-22
    Publishing country United States
    Document type Journal Article
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2023.11.016
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 797: Show issues Location:
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  2. Article ; Online: Do macrophages inhibit or promote atheroma plaque calcification and stability?

    Drueke, Tilman B / Massy, Ziad A

    Kidney international

    2023  Volume 104, Issue 3, Page(s) 412–415

    MeSH term(s) Humans ; Plaque, Atherosclerotic ; Vascular Calcification/diagnostic imaging ; Calcification, Physiologic ; Macrophages
    Language English
    Publishing date 2023-06-17
    Publishing country United States
    Document type Journal Article ; Comment
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2023.06.005
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 797: Show issues Location:
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  3. Article ; Online: Vascular calcification is accelerated by interleukin-29.

    Drueke, Tilman B / Massy, Ziad A

    Kidney international

    2023  Volume 103, Issue 5, Page(s) 826–829

    MeSH term(s) Humans ; Vascular Calcification/diagnostic imaging ; Cytokines ; Interleukins ; Renal Insufficiency, Chronic ; Myocytes, Smooth Muscle
    Chemical Substances Cytokines ; Interleukins
    Language English
    Publishing date 2023-02-10
    Publishing country United States
    Document type Journal Article ; Comment
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2023.01.020
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article ; Online: Role of uremic toxins in vascular disease-the end of nihilism?

    Massy, Ziad A / Drueke, Tilman B

    Kidney international

    2022  Volume 101, Issue 6, Page(s) 1100–1102

    MeSH term(s) Humans ; Renal Insufficiency, Chronic ; Toxins, Biological ; Uremia/complications ; Uremia/therapy ; Uremic Toxins ; Vascular Diseases/etiology
    Chemical Substances Toxins, Biological ; Uremic Toxins
    Language English
    Publishing date 2022-02-12
    Publishing country United States
    Document type Journal Article
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2022.01.024
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  5. Article ; Online: Vascular calcification in chronic kidney disease: contribution of ferroptosis?

    Drüeke, Tilman B / Massy, Ziad A

    Kidney international

    2022  Volume 102, Issue 6, Page(s) 1209–1211

    Abstract: Vascular calcification associated with chronic kidney disease (CKD) is an active, regulated process. Apoptosis of vascular smooth muscle cells has long been known to play a major role in its pathogenesis, with apoptotic bodies derived from these cells ... ...

    Abstract Vascular calcification associated with chronic kidney disease (CKD) is an active, regulated process. Apoptosis of vascular smooth muscle cells has long been known to play a major role in its pathogenesis, with apoptotic bodies derived from these cells acting as nucleating structures for calcium crystal formation and deposition. Ye et al. now show in experimental models in vitro and in vivo that ferroptosis can also contribute to the development of vascular calcification in CKD.
    MeSH term(s) Humans ; Ferroptosis ; Vascular Calcification/complications ; Renal Insufficiency, Chronic/complications ; Renal Insufficiency, Chronic/pathology ; Myocytes, Smooth Muscle/pathology ; Apoptosis
    Language English
    Publishing date 2022-10-28
    Publishing country United States
    Document type Journal Article
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2022.08.031
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  6. Article ; Online: Soluble urokinase plasminogen activator receptor (suPAR) promotes atherosclerosis.

    Drueke, Tilman B / Massy, Ziad A

    Kidney international

    2022  Volume 103, Issue 3, Page(s) 451–454

    MeSH term(s) Humans ; Receptors, Urokinase Plasminogen Activator ; Atherosclerosis ; Urokinase-Type Plasminogen Activator ; Biomarkers
    Chemical Substances Receptors, Urokinase Plasminogen Activator ; Urokinase-Type Plasminogen Activator (EC 3.4.21.73) ; Biomarkers
    Language English
    Publishing date 2022-12-19
    Publishing country United States
    Document type Journal Article ; Comment
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2022.12.006
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  7. Article ; Online: Sortilin, carbamylation, and cardiovascular calcification in chronic kidney disease.

    Massy, Ziad A / Liabeuf, Sophie

    Kidney international

    2022  Volume 101, Issue 3, Page(s) 456–459

    Abstract: Sortilin appears to play important roles in the pathogenesis of vascular and metabolic diseases, such as cardiovascular calcification. Post-translational modifications of sortilin (i.e., phosphorylation or carbamylation) are among the key mechanisms of ... ...

    Abstract Sortilin appears to play important roles in the pathogenesis of vascular and metabolic diseases, such as cardiovascular calcification. Post-translational modifications of sortilin (i.e., phosphorylation or carbamylation) are among the key mechanisms of its actions. Recent data extended the role of carbamylated sortilin to the pathogenesis of cardiovascular calcifications in chronic kidney disease. Whether therapeutic tools potentially available to inhibit the carbamylation process in chronic kidney disease patients would allow preventing cardiovascular calcifications remains to be demonstrated.
    MeSH term(s) Adaptor Proteins, Vesicular Transport/metabolism ; Humans ; Protein Carbamylation ; Protein Processing, Post-Translational ; Renal Insufficiency, Chronic/complications
    Chemical Substances Adaptor Proteins, Vesicular Transport ; sortilin (Z020Y8WIJ4)
    Language English
    Publishing date 2022-02-01
    Publishing country United States
    Document type Journal Article ; Comment
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2021.11.027
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  8. Article: The evolving science of anemia management in chronic kidney disease.

    Massy, Ziad A / Drueke, Tilman B

    Kidney international supplements

    2021  Volume 11, Issue 1, Page(s) 1–2

    Language English
    Publishing date 2021-03-18
    Publishing country United States
    Document type Editorial
    ZDB-ID 193442-9
    ISSN 2157-1716 ; 2157-1724 ; 0098-6577
    ISSN (online) 2157-1716
    ISSN 2157-1724 ; 0098-6577
    DOI 10.1016/j.kisu.2020.11.002
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    Zs.A 797 -Suppl.-: Show issues Location:
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  9. Article ; Online: Role of proteinuria in the anemia of chronic kidney disease.

    Drüeke, Tilman B / Massy, Ziad A

    Kidney international

    2021  Volume 100, Issue 6, Page(s) 1160–1162

    Abstract: The most important contributors to the anemia of patients with chronic kidney disease are insufficient erythropoietin production and erythropoietin hyporesponsiveness, decreased red blood cell half-life, iron deficiency, and inflammation. However, in ... ...

    Abstract The most important contributors to the anemia of patients with chronic kidney disease are insufficient erythropoietin production and erythropoietin hyporesponsiveness, decreased red blood cell half-life, iron deficiency, and inflammation. However, in contrast to the role of kidney failure, that of proteinuria and nephrotic syndrome is less clear. Bissinger et al. now provide evidence in mouse models and patients with chronic kidney disease that heavy proteinuria alters erythrocyte metabolism and increases erythrocyte death.
    MeSH term(s) Anemia/etiology ; Animals ; Erythropoietin ; Humans ; Mice ; Nephrotic Syndrome ; Proteinuria/etiology ; Renal Insufficiency, Chronic/complications ; Renal Insufficiency, Chronic/diagnosis
    Chemical Substances Erythropoietin (11096-26-7)
    Language English
    Publishing date 2021-11-21
    Publishing country United States
    Document type Journal Article ; Comment
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2021.09.016
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  10. Article ; Online: Phosphate meeting cholesterol-consequences for cardiovascular disease in chronic kidney disease?

    Hénaut, Lucie / Massy, Ziad A

    Kidney international

    2021  Volume 99, Issue 6, Page(s) 1264–1267

    Abstract: Cardiovascular disease is highly prevalent in patients with chronic kidney disease. Hyperphosphatemia is associated with subclinical atheromatosis in chronic kidney disease. Phosphate-induced endothelial dysfunction and vascular calcification are thought ...

    Abstract Cardiovascular disease is highly prevalent in patients with chronic kidney disease. Hyperphosphatemia is associated with subclinical atheromatosis in chronic kidney disease. Phosphate-induced endothelial dysfunction and vascular calcification are thought to be key inducers of atherosclerosis in this condition. Zhou et al. now demonstrate that phosphate promotes de novo cholesterol synthesis in vascular smooth muscle and macrophages through increased 3-hydroxy-3-methylglutaryl coenzyme A reductase activation. This observation may change current concepts of atherosclerosis development and management in chronic kidney disease.
    MeSH term(s) Atherosclerosis ; Cardiovascular Diseases/epidemiology ; Cardiovascular Diseases/etiology ; Cholesterol ; Humans ; Hyperphosphatemia/etiology ; Mannosidases ; Phosphates ; Polysaccharides ; Renal Insufficiency, Chronic/complications ; Vascular Calcification
    Chemical Substances Phosphates ; Polysaccharides ; Cholesterol (97C5T2UQ7J) ; Mannosidases (EC 3.2.1.-)
    Language English
    Publishing date 2021-01-30
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Comment
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2021.02.022
    Database MEDical Literature Analysis and Retrieval System OnLINE

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