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Article ; Online: Follow-up regimens for carriers of hereditary transthyretin variants.

Losada-López, Inés Asunción / Kapetanovic-García, Solange / Sevilla-Mantecón, Teresa / Muñoz-Beamud, Francisco

2022 Volume 160, Issue 5, Page(s) 213–217

MeSH term(s)	Humans ; Prealbumin/genetics ; Follow-Up Studies ; Mutation ; Amyloid Neuropathies, Familial/genetics ; Heterozygote
Chemical Substances	Prealbumin
Language	Spanish
Publishing date	2022-12-16
Publishing country	Spain
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	411607-0
ISSN	1578-8989 ; 0025-7753
ISSN (online)	1578-8989
ISSN	0025-7753
DOI	10.1016/j.medcli.2022.10.002
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Article ; Online: Biomarkers of axonal damage to favor early diagnosis in variant transthyretin amyloidosis (A-ATTRv).

González-Moreno, Juan / Gragera-Martínez, Álvaro / Rodríguez, Adrián / Borrachero-Garro, Cristina / García-Garrido, Sandra / Barceló, Carles / Manovel-Sánchez, Ana / Ribot-Sansó, Maria Antonia / Ibargüen-González, Lesly / Gomila, Rosa / Muñoz-Beamud, Francisco / Losada-López, Inés / Cisneros-Barroso, Eugenia

Scientific reports

2024 Volume 14, Issue 1, Page(s) 581

Abstract: Early identification of ATTRv amyloidosis disease onset is still often delayed due to the lack of validated biomarkers of this disease. Light chain neurofilament (NfL) have shown promising results in early diagnosis in this disease, but data is still ... ...

Abstract	Early identification of ATTRv amyloidosis disease onset is still often delayed due to the lack of validated biomarkers of this disease. Light chain neurofilament (NfL) have shown promising results in early diagnosis in this disease, but data is still needed, including with alternative measuring methods. Our aim was to study the levels of NfL measured by ELISA. Furthermore, interstitial matrix metalloproteinase type 1 (MMP-1) serum levels were measured as a potential new biomarker in ATTRv. Serum NfL and MMP-1 were measured using ELISA assays in 90 participants (29 ATTR-V30M patients, 31 asymptomatic V30M-TTR variant carriers and 30 healthy controls). Median NfL levels among ATTRv amyloidosis patients were significantly higher (116 pg/mL vs 0 pg/mL in both comparison groups). The AUC comparing ATTRv amyloidosis patients and asymptomatic carriers was 0.90 and the NfL concentration of 93.55 pg/mL yielded a sensitivity of 79% and a specificity of 87%. NfL levels had a significant positive correlation with NIS values among patients. We found a negative significant correlation between eGFR and NfL levels. Finally, MMP1 levels were not different between groups. Evidence of NfL use for early diagnosis of ATTR-PN amyloidosis is growing. ELISA seems a reliable and available technique for it quantification. Decreased GFR could influence NfL plasma levels.
MeSH term(s)	Humans ; Matrix Metalloproteinase 1 ; Amyloid Neuropathies, Familial/diagnosis ; Early Diagnosis ; Biomarkers
Chemical Substances	Matrix Metalloproteinase 1 (EC 3.4.24.7) ; Biomarkers
Language	English
Publishing date	2024-01-05
Publishing country	England
Document type	Journal Article
ZDB-ID	2615211-3
ISSN	2045-2322 ; 2045-2322
ISSN (online)	2045-2322
ISSN	2045-2322
DOI	10.1038/s41598-023-50212-2
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Article: A narrative review and expert recommendations on the assessment of the clinical manifestations, follow-up, and management of post-OLT patients with ATTRv amyloidosis.

Casasnovas, Carlos / Lladó, Laura / Borrachero, Cristina / Pérez-Santamaría, Patricia Valentina / Muñoz-Beamud, Francisco / Losada-López, Inés Asunción / Baliellas-Comellas, Maria Carme / González-Moreno, Juan

Therapeutic advances in neurological disorders

2023 Volume 16, Page(s) 17562864231191590

Abstract: Orthotopic liver transplantation (OLT) was the first treatment able to modify the natural course of hereditary transthyretin (ATTRv) amyloidosis, which is a rare and fatal disorder caused by the accumulation of misfolded transthyretin (TTR) variants in ... ...

Abstract	Orthotopic liver transplantation (OLT) was the first treatment able to modify the natural course of hereditary transthyretin (ATTRv) amyloidosis, which is a rare and fatal disorder caused by the accumulation of misfolded transthyretin (TTR) variants in different organs and tissues and which leads to a progressive and multisystem dysfunction. Because the liver is the main source of TTR, OLT dramatically reduces the production of the pathogenic TTR variant, which should prevent amyloid formation and halt disease progression. However, amyloidosis progression may occur after OLT due to wild-type TTR deposition, especially in the nerves and heart. In this review, we discuss the disease features influencing OLT outcomes and the clinical manifestations of ATTRv amyloidosis progression post-OLT to improve our understanding of disease worsening after OLT and optimize the follow-up and clinical management of these patients. By conducting a literature review on the PubMed database, we identified patient characteristics that have been associated with worse post-OLT outcomes, including late-onset V50M and non-V50M variants, age >40 years, long disease duration, advanced neuropathy and autonomic dysfunction, and malnutrition. Regarding post-OLT mortality, deaths occurring within the first year after OLT were mainly associated with fatal graft complications and infectious diseases, whereas cardiovascular-related deaths usually occurred later. Considering the diverse clinical manifestations of ATTRv amyloidosis progression post-OLT, including worsening neuropathy and/or cardiomyopathy, autonomic dysfunction, and oculoleptomeningeal involvement, we present advice on the most relevant tests for assessing disease progression post-OLT. Finally, we discuss the use of new therapies based on TTR stabilizers and
Language	English
Publishing date	2023-08-24
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	2442245-9
ISSN	1756-2864 ; 1756-2856
ISSN (online)	1756-2864
ISSN	1756-2856
DOI	10.1177/17562864231191590
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Article ; Online: Recomendaciones para el diagnóstico y tratamiento de la polineuropatía amiloidótica familiar por transtiretina.

García-Pavía, Pablo / Muñoz-Beamud, Francisco / Casasnovas, Carlos

Medicina clinica

2015 Volume 145, Issue 5, Page(s) 211–217

Title translation	Recommendations regarding diagnosis and treatment of transthyretin familial amyloid polyneuropathy.Grupo de Estudio y Tratamiento de la Polineuropatía Amiloidótica Familiar por Transtiretina (GETPAF-TTR).
MeSH term(s)	Amyloid Neuropathies, Familial/diagnosis ; Amyloid Neuropathies, Familial/genetics ; Amyloid Neuropathies, Familial/therapy ; Cardiovascular Diseases/etiology ; Cardiovascular Diseases/therapy ; Central Nervous System Agents/therapeutic use ; Combined Modality Therapy ; Diagnosis, Differential ; Diagnostic Techniques, Neurological ; Eye Diseases/etiology ; Eye Diseases/therapy ; Gastrointestinal Diseases/etiology ; Gastrointestinal Diseases/therapy ; Heart Transplantation ; Humans ; Liver Transplantation ; Prealbumin/genetics ; Symptom Assessment ; Urologic Diseases/etiology ; Urologic Diseases/therapy
Chemical Substances	Central Nervous System Agents ; Prealbumin
Language	Spanish
Publishing date	2015-09-07
Publishing country	Spain
Document type	Journal Article
ZDB-ID	411607-0
ISSN	1578-8989 ; 0025-7753
ISSN (online)	1578-8989
ISSN	0025-7753
DOI	10.1016/j.medcli.2015.02.003
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Article ; Online: Patisiran treatment in patients with hereditary transthyretin-mediated amyloidosis with polyneuropathy after liver transplantation.

Schmidt, Hartmut H / Wixner, Jonas / Planté-Bordeneuve, Violaine / Muñoz-Beamud, Francisco / Lladó, Laura / Gillmore, Julian D / Mazzeo, Anna / Li, Xingyu / Arum, Seth / Jay, Patrick Y / Adams, David

American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons

2022 Volume 22, Issue 6, Page(s) 1646–1657

Abstract: Hereditary transthyretin-mediated (hATTR) amyloidosis, or ATTRv amyloidosis, is a progressive disease, for which liver transplantation (LT) has been a long-standing treatment. However, disease progression continues post-LT. This Phase 3b, open-label ... ...

Abstract	Hereditary transthyretin-mediated (hATTR) amyloidosis, or ATTRv amyloidosis, is a progressive disease, for which liver transplantation (LT) has been a long-standing treatment. However, disease progression continues post-LT. This Phase 3b, open-label trial evaluated efficacy and safety of patisiran in patients with ATTRv amyloidosis with polyneuropathy progression post-LT. Primary endpoint was median transthyretin (TTR) reduction from baseline. Twenty-three patients received patisiran for 12 months alongside immunosuppression regimens. Patisiran elicited a rapid, sustained TTR reduction (median reduction [Months 6 and 12 average], 91.0%; 95% CI: 86.1%-92.3%); improved neuropathy, quality of life, and autonomic symptoms from baseline to Month 12 (mean change [SEM], Neuropathy Impairment Score, -3.7 [2.7]; Norfolk Quality of Life-Diabetic Neuropathy questionnaire, -6.5 [4.9]; least-squares mean [SEM], Composite Autonomic Symptom Score-31, -5.0 [2.6]); and stabilized disability (Rasch-built Overall Disability Scale) and nutritional status (modified body mass index). Adverse events were mild or moderate; five patients experienced ≥1 serious adverse event. Most patients had normal liver function tests. One patient experienced transplant rejection consistent with inadequate immunosuppression, remained on patisiran, and completed the study. In conclusion, patisiran reduced serum TTR, was well tolerated, and improved or stabilized key disease impairment measures in patients with ATTRv amyloidosis with polyneuropathy progression post-LT (www.clinicaltrials.gov NCT03862807).
MeSH term(s)	Amyloid Neuropathies, Familial/complications ; Amyloid Neuropathies, Familial/drug therapy ; Amyloid Neuropathies, Familial/surgery ; Humans ; Liver Transplantation ; Polyneuropathies/drug therapy ; Polyneuropathies/etiology ; Prealbumin/therapeutic use ; Quality of Life ; RNA, Small Interfering
Chemical Substances	Prealbumin ; RNA, Small Interfering ; patisiran (50FKX8CB2Y)
Language	English
Publishing date	2022-03-26
Publishing country	United States
Document type	Clinical Trial, Phase III ; Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2060594-8
ISSN	1600-6143 ; 1600-6135
ISSN (online)	1600-6143
ISSN	1600-6135
DOI	10.1111/ajt.17009
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Article: A Descriptive Analysis of ATTR Amyloidosis in Spain from the Transthyretin Amyloidosis Outcomes Survey.

González-Moreno, Juan / Losada-López, Inés / Cisneros-Barroso, Eugenia / Garcia-Pavia, Pablo / González-Costello, José / Muñoz-Beamud, Francisco / Campistol, Josep Maria / Fernandez-Torron, Roberto / Chapman, Doug / Amass, Leslie

Neurology and therapy

2021 Volume 10, Issue 2, Page(s) 833–845

Abstract: Introduction: Transthyretin amyloidosis (ATTR amyloidosis) is a clinically heterogeneous disease caused by mutations in the transthyretin (TTR) gene or aggregation of wild-type transthyretin (ATTRwt). In Spain, there are two large endemic foci of ATTR ... ...

Abstract	Introduction: Transthyretin amyloidosis (ATTR amyloidosis) is a clinically heterogeneous disease caused by mutations in the transthyretin (TTR) gene or aggregation of wild-type transthyretin (ATTRwt). In Spain, there are two large endemic foci of ATTR amyloidosis caused by the Val30Met variant, with additional cases across the country; however, these data may be incomplete, as there is no centralized patient registry. The Transthyretin Amyloidosis Outcomes Survey (THAOS) is an ongoing, global, longitudinal, observational survey of patients with ATTR amyloidosis, including both inherited and wild-type disease, and asymptomatic patients with TTR mutations. This analysis aimed to gain a deeper understanding of the clinical profile of patients with ATTR amyloidosis in Spain. Methods: This was a descriptive analysis of the demographic and clinical characteristics of symptomatic patients enrolled at six sites geographically dispersed throughout Spain (data cutoff: January 6, 2020). Patient data at enrollment, including genotype, demographics, and clinical presentation for symptomatic patients, were recorded. Patients were grouped by predominant phenotype based on clinical measures at enrollment: predominantly cardiac, predominantly neurologic, or mixed (cardiac and neurologic). Results: There were 379 patients (58.0% male; 63.3% symptomatic) enrolled in the six THAOS sites in Spain. Predominant genotypes were the Val30Met mutation (69.1%) or ATTRwt (15.6%). Predominant phenotype distribution was neurologic (50.4%), mixed (35.8%), and cardiac (13.8%) for all symptomatic patients (n = 240); neurologic (67.8%), mixed (21.2%), and cardiac (11.0%) for symptomatic Val30Met (n = 146); and mixed (64.9%), cardiac (22.8%), and neurologic (12.3%) for symptomatic ATTRwt (n = 57). Symptomatic patients reported a range of ATTR amyloidosis signs and symptoms at enrollment, with autonomic neuropathy and sensory neuropathy common in all phenotypes. Conclusions: These results from THAOS highlight the phenotypic heterogeneity associated with ATTR amyloidosis in Spain and the importance of comprehensive neurologic and cardiac evaluations in all patients with ATTR amyloidosis. Trial registration: ClinicalTrials.gov: NCT00628745.
Language	English
Publishing date	2021-07-30
Publishing country	New Zealand
Document type	Journal Article
ISSN	2193-8253
ISSN	2193-8253
DOI	10.1007/s40120-021-00267-y
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Article: Rituximab as an effective alternative therapy in refractory idiopathic inflammatory myopathies.

Muñoz-Beamud, Francisco / Isenberg, David A

Clinical and experimental rheumatology

2013 Volume 31, Issue 6, Page(s) 896–903

Abstract: Objectives: To assess the efficacy and safety of B-lymphocyte depletion therapy (BCDT) utilising rituximab in refractory idiopathic inflammatory myositis.: Methods: We retrospectively evaluated 16 adult patients with active dermatomyositis (DM) or ... ...

Abstract	Objectives: To assess the efficacy and safety of B-lymphocyte depletion therapy (BCDT) utilising rituximab in refractory idiopathic inflammatory myositis. Methods: We retrospectively evaluated 16 adult patients with active dermatomyositis (DM) or polymyositis (PM) who received 1 gram rituximab intravenous infusions two weeks apart after failing to respond to conventional therapy. The clinical and biochemical response were analysed by the Myositis Intention to Treat index (MITAX) and the serum creatine kinase (CK) levels at baseline and 6 and 12 months after treatment. The primary efficacy outcome was 20% improvement in the MITAX index and 30% reduction in CK. Results: Eight patients responded to treatment and achieved both the MITAX and CK levels objectives within 6 months of rituximab therapy. Five out of these 8 responders remained clinically stable at 12 months and CK levels were still reduced or normalised. Of note, 4 patients who did not respond were re-assessed and had their diagnoses corrected. All patients showed adequate B cell depletion (BCD) with re-population occurring for a 15.4 months average (range 3-42 months). Those simultaneously treated with cyclophosphamide achieved more long-lasting depletion (average 18.6 months). Conclusions: The heterogeneous clinical and serological characteristics of patients diagnosed with IIM probably explain why some, but not all patients respond to rituximab. Myositis overlap and anti-synthetase syndromes seem to respond better than other patient subsets.
MeSH term(s)	Adult ; Antibodies, Monoclonal, Murine-Derived/administration & dosage ; Antibodies, Monoclonal, Murine-Derived/adverse effects ; Antibodies, Monoclonal, Murine-Derived/therapeutic use ; B-Lymphocytes/drug effects ; B-Lymphocytes/immunology ; Biomarkers/blood ; Creatine Kinase/blood ; Drug Administration Schedule ; Female ; Humans ; Immunologic Factors/administration & dosage ; Immunologic Factors/adverse effects ; Immunologic Factors/therapeutic use ; Infusions, Intravenous ; Male ; Middle Aged ; Myositis/blood ; Myositis/diagnosis ; Myositis/drug therapy ; Myositis/immunology ; Remission Induction ; Retrospective Studies ; Rituximab ; Time Factors ; Treatment Outcome
Chemical Substances	Antibodies, Monoclonal, Murine-Derived ; Biomarkers ; Immunologic Factors ; Rituximab (4F4X42SYQ6) ; Creatine Kinase (EC 2.7.3.2)
Language	English
Publishing date	2013-11
Publishing country	Italy
Document type	Journal Article
ZDB-ID	605886-3
ISSN	1593-098X ; 0392-856X
ISSN (online)	1593-098X
ISSN	0392-856X
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Article ; Online: Estimating the annual economic burden for the management of patients with transthyretin amyloid polyneuropathy in Spain.

Galan, Lucia / Gonzalez-Moreno, Juan / Martínez-Sesmero, José Manuel / Muñoz-Beamud, Francisco / Santos-Rubio, Maria Dolores / Tran, Diana / Lebeau, Paul / Stewart, Michelle / Mallaina, Pablo / Tarilonte, Patricia / Peral, Carmen / Rozenbaum, Mark H

Expert review of pharmacoeconomics & outcomes research

2021 Volume 21, Issue 5, Page(s) 967–973

Abstract: ... ...

Abstract	Background
MeSH term(s)	Amyloid Neuropathies, Familial/economics ; Amyloid Neuropathies, Familial/therapy ; Benzoxazoles/administration & dosage ; Benzoxazoles/economics ; Cost of Illness ; Drug Costs/statistics & numerical data ; Health Care Costs/statistics & numerical data ; Hospitalization/economics ; Humans ; Oligonucleotides/administration & dosage ; Oligonucleotides/economics ; RNA, Small Interfering/administration & dosage ; RNA, Small Interfering/economics ; Spain
Chemical Substances	Benzoxazoles ; Oligonucleotides ; RNA, Small Interfering ; Inotersen (0IEO0F56LV) ; patisiran (50FKX8CB2Y) ; tafamidis (8FG9H9D31J)
Language	English
Publishing date	2021-03-24
Publishing country	England
Document type	Comparative Study ; Journal Article
ZDB-ID	2208481-2
ISSN	1744-8379 ; 1473-7167
ISSN (online)	1744-8379
ISSN	1473-7167
DOI	10.1080/14737167.2021.1900738
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Article ; Online: Extracellular Volume Detects Amyloidotic Cardiomyopathy and Correlates With Neurological Impairment in Transthyretin-familial Amyloidosis.

Gallego-Delgado, María / González-López, Esther / Muñoz-Beamud, Francisco / Buades, Juan / Galán, Lucía / Muñoz-Blanco, Jose Luis / Sánchez-González, Javier / Ibáñez, Borja / Mirelis, Jesus G / García-Pavía, Pablo

Revista espanola de cardiologia (English ed.)

2016 Volume 69, Issue 10, Page(s) 923–930

Abstract: Introduction and objectives: Cardiac involvement determines prognosis and treatment options in transthyretin-familial amyloidosis. Cardiac magnetic resonance T: Methods: A total of 31 transthyretin-familial amyloidosis patients (19 mean age, 49 ± 12 ... ...

Abstract	Introduction and objectives: Cardiac involvement determines prognosis and treatment options in transthyretin-familial amyloidosis. Cardiac magnetic resonance T Methods: A total of 31 transthyretin-familial amyloidosis patients (19 mean age, 49 ± 12 years; 26 with the Val30Met mutation) underwent a T Results: Five patients had cardiac amyloidosis (all confirmed by Conclusions: Extracellular volume quantification allows identification of cardiac amyloidosis and correlates with the degree of neurological impairment in transthyretin-familial amyloidosis. This noninvasive technique could be a useful tool for early diagnosis of cardiac amyloidosis and to track cardiac and extracardiac amyloid disease.
MeSH term(s)	Adult ; Aged ; Amyloid Neuropathies, Familial/complications ; Amyloid Neuropathies, Familial/diagnosis ; Amyloid Neuropathies, Familial/genetics ; Cardiac Volume/physiology ; Cardiomyopathies/complications ; Cardiomyopathies/diagnosis ; Cardiomyopathies/genetics ; Female ; Humans ; Magnetic Resonance Angiography ; Male ; Middle Aged ; Mutation/genetics ; Nervous System Diseases/complications ; Nervous System Diseases/genetics ; Observer Variation ; Risk Factors
Language	Spanish
Publishing date	2016-10
Publishing country	Spain
Document type	Journal Article
ISSN	1885-5857
ISSN (online)	1885-5857
DOI	10.1016/j.rec.2016.02.027
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Article ; Online: Treatment with tocilizumab or corticosteroids for COVID-19 patients with hyperinflammatory state

Rodríguez-Baño, Jesús / Pachón, Jerónimo / Carratalà, Jordi / Ryan, Pablo / Jarrín, Inmaculada / Yllescas, María / Arribas, José Ramón / Berenguer, Juan / Aznar Muñoz, Esther / Gil Divasson, Pedro / González Muñiz, Patricia / Muñoz Aguirre, Clara / Díaz Menéndez, Marta / de la Calle Prieto, Fernando / Arsuaga Vicente, Marta / Trigo Esteban, Elena / Pérez Valero, Ignacio / de Miguel Buckley, Rosa / Cadiñaños Loidi, Julen /

Diaz Pollan, Beatriz / Martín Carbonero, Luz / Ramos Ramos, Juan Carlos / Loeches Yagüe, Belén / Montejano Sánchez, Rocío / González García, Juan / García Rodríguez, Julio / Ramírez, Margarita / Gutiérrez, Isabel / Tejerina, Francisco / Aldámiz-Echevarría, Teresa / Díez, Cristina / Fanciulli, Chiara / Pérez-Latorre, Leire / Pinilla, Blanca / López, Juan Carlos / Such Diaz, Ana / Álvaro Alonso, Elena / Torres Macho, Juan / Cuevas Tascon, Guillermo / Jiménez González de Buitrago, Eva / Brañas Baztán, Fátima / Valencia de la Rosa, Jorge / Pérez Butragueño, Mario / Fernández Jiménez, Inés / Muñiz Nicolás, Gemma / Sepúlveda Berrocal, Antonia / Gato Díez, Alberto / Toledano Sierra, María Pilar / García Butenegro, María Paz / Peláez Ballesta, Ana Isabel / Morcillo Rodríguez, Elena / Fernández Romero, Isidoro / Peláez Ballesta, Cristina / Guirado Torrecillas, María Isabel / Goikoetxea Agirre, Josune / Bereciartua Bastarrica, Elena / Guio Carrión, Laura / Rodríguez Álvarez, Regino / Ibarrola Hierro, Marta / Pérez Hernández, Isabel A. / Pérez Zapata, Inés / Román Soto, Sergio / Kallouchi, Mohamed / Domínguez Vicent, Juan Ramón / Silvariño Fernández, Rafae / Ugalde Espiñeira, Jon / Sanjuan López, Ainhoa / García Martínez, Silvia / Temprano Gogenola, Mikel / Asensi, Víctor / Suárez, Silvia / Suárez, Lucia / Yllera, Carmen / Rivas-Carmenado, María / Romero-Palacios, Alberto / Ruiz Aragón, Jesús / Jiménez Aguilar, Patricia / Fernández Ávila, Ma Luisa / Castilla Ortiz, Rosario / Alende Castro, Vanesa / Pérez García, Cristina / Fernández Morales, Marta / Valle Feijoo Begoña Rodríguez Ferreira, María Lorena María / Gómez-Junyent, Joan / Villar-García, Judit / López-Montesinos, Inmaculada / Arrieta-Aldea, Itziar / Rial-Villavecchia, Abora / García Vázquez, Elisa / Roura Piloto, Aychel Elena / Moral Escudero, Encarnación / Hernández Torres, Alicia / Albendín Iglesias, Helena / Vinuesa García, David / Martínez Montes, Clara / De la Hera Fernández, Francisco Javier / Anguita Santos, Francisco / Ruiz Sancho, Andrés / Díaz de Brito Fernández, Vicens / Sanmarti Vilamala, Montserrat / España Cueto, Sergio / Molina Morant, Daniel / González-Cuevas, Araceli / Chara Cervantes, Joel Elías / Policarpo Torres, Guillem / Ortega Montoliu, Meritxell / Angerri Nadal, Mònica / De Genover Gil, Ariadna / Patera, Eleni / Godoy Lorenzo, Rita / Zioga, Evangelia Anna María / Isern Fernández, Virginia / Sabbagh Fajardo, Carlos Enrique / Ferrer Ribera, Ana / Bea Serrano, Carlos / Oltra Sempere, Rosa / Vela Bernal, Sara / Albiol Viñals, Paloma / Pedromingo Kus, Miguel / Garcinuño, María Ángeles / Fiorante, Silvana / Pérez Pinto, Sergio / de la Vega, Alexandra / Fariñas Álvarez, María Carmen / González Rico, Claudia / Arnaiz de las Revillas, Francisco / Giménez, Teresa / Calvo, Jorge / Meije Castillo, Yolanda / Duarte Borges, Alejandra / Pareja Coca, Júlia / Clemente Presas, Mercedes / Sanz Salvador, Xavier / Pérez Rodríguez, Ma Teresa / Sousa, Adrián / Pérez González, Alexandre / Longueira, Rebeca / Araujo, Alejandro / Alonso Martínez, Blanca / García Escudero, Laura / Lidia Kamel Rey, Sara / Roa Alonso, David / Avilés Parra, Juan Pablo / Pelegrín Senent, Iván / Rouco Esteves Marques, Rosana / Raich Montiu, Laia / Souto Higueras, Jessica / Gálvez Bobadilla, Manuel Alejandro / Parra Ruiz, Jorge / Ramos Sesma, Violeta / Velasco Fuentes, Sara / García Pereña, Laura / Lluna Carrascosa, Alfonso / Gilaberte Reyzábal, Sergio / Liébana Gómez, Mónica / Salillas Hernando, Juan / Serrano Martínez, Alberto / Torralba González de Suso, Miguel / Martínez Martín, Patricia / Rábago Lorite, Isabel / González-Ruano Pérez, Patricia / Pérez-Monte Mínguez, Beatriz / García Flores, Ángeles / Comas Casanova, Pere / Martín Plata, Andrea / Santana Báez, Sergio Manuel / Sanz Peláez, Oscar / Mohamed Ramírez, Karim / Robaina Bordón, José María / Vílchez Rueda, Helem Haydeé / Riera Jaume, Melchor / Mut Ramon, Gemma / Gavalda Manso, Meritxell / Planas Bibiloni, Lluis / Castelo Corral, Laura / Ramos Merino, Lucía / Sánchez Vidal, Efrén / Rodríguez Mayo, María / Míguez Rey, Enrique / García de Lomas Guerrero, José M. / De la Torre Lima, Javier / Correa Ruiz, Ana / Fernández Sánchez, Fernando / Jiménez-García, Nicolás / Sierra-Monzón, José Luis / Gracia-Tello, Borja / Hernández-Bonaga, María / Pellejero, Galadriel / Asín-Corrochano, Marta / Boix Palop, Lucia / Calbo, Esther / Badía, Cristina / Dietl, Beatriz / Lucía, Gómez / Domínguez-Castellano, Ángel / Ríos-Villegas, María José / del Toro, María D. / Palacios Baena, Zaira R. / Salamanca-Rivera, Elena / Marín, Elena / Almadana, Virginia / Pérez-Galera, Salvador / González-Iglesias, Luisa / Abelenda-Alonso, Gabriela / Álvarez-Pouso, Claudia / Escrihuela, Francesc / Gudiol, Carlota / Lorenzo-Esteller, Laia / Niubó, Jordi / Podzamczer, Daniel / Pujol, Miquel / Rombauts, Alexander / Salvert Lletí, Miguel / Gil Sánchez, Ricardo / Jiménez Escrig, Marta / Parra Gómez, Laura / Tasias Pitarch, Mariona / Navarro Vilasaró, Marta / Machado Sicilia, María Luisa / Gomila Grange, Aina / Calzado Isbert, Sonia / Carrasco Antón, Nerea / Petkova-Saiz, Elizabet / Cabello Úbeda, Alfonso / Górgolas Hernández-Mora, Miguel / Sánchez-Pernaute, Olga / Dueñas Gutiérrez, Carlos / Martin Guerra, Javier / Castrodeza Sanz, José Javier / Fernández Espinilla, Virginia / Rodríguez Fernández, Laura / González-Moreno, Juan / Villoslada Gelabert, Aroa / Ribot Sanso, María Antonia / Fernández-Baca, María Victoria / Hernández Milian, Almudena / Morán Rodríguez, Miguel Ángel / Ortiz de Zárate Ibarra, Zuriñe / Portu Zapirain, José Joaquin / Saez de Adana Arroniz, Ester / Gainzarain Arana, Juan Carlos / Meca Birlanga, Olga / del Amor Espín, Ma Jesús / Viqueira González, Montserrat / García García, Josefina / Martínez Madrid, Onofre / Bernal Morell, Enrique / Alcaraz, Antonia / Muñoz, Ángeles / Pina, Ignacio / de la Rosa, Vicente / Caínzos Romero, Tamara / Sánchez Trigo, Sabela / Mariño Callejo, Ana Isabel / Álvarez Díaz, Hortensia / Valcarce Pardeiro, Nieves / Sánchez Serrano, Adriana / Piñar Cabezos, Diana / García Villalba, Eva Pilar / Aguayo Jiménez, Carmen / Ruíz Campuzano, María / Naranjo Velasco, Virginia / Santos Peña, Marta / Mora Delgado, Juan / Sevilla Moreno, Israel / Lojo Cruz, Cristina / Kortajarena Urkola, Xabier / Iribarren Loyarte, José Antonio / Bustinduy Odriozola, María Jesús / Ibarguren Pinilla, Maialen / Álvarez Rodríguez, Ignacio / Martínez Marcos, Francisco Javier / Rodríguez Gómez, Francisco Javier / Asschert Agüero, Isabel / Muñoz Beamud, Francisco / Ruiz Reina, Antonio José / Llenas-García, Jara / González-Cuello, Inmaculada / Hellín-Valiente, Elena / Martínez Birlanga, Esther / Tafalla Torres, José Manuel / Calderón Parra, Jorge / Escudero López, Gabriela / Gutiérrez Martín, Isabel / Andrés Eisenhofer, Ane / García Prieto, Sonia / Álvarez Franco, Raquel / Roger Zapata, Daniel / Martínez Cifre, Blanca / Aranda Rife, Elena / Martín Rubio, Irene / Barbosa Ventura, André / Garrido, Javier / Gonzalo, Concepción / Piñero, Iván / de la Cruz Felipe, Nieves / Talavera García, Eva / Lamata Subero, Marta / Mendoza Roy, Paula / García de Carlos, María Soledad / Lajusticia Aisa, Justo / Arteche Eguizabal, Lorea / Urrutia Losada, Ainhoa / Domingo Echaburu, Saioa / Cuadros Tito, Pedro Ángel / Orbe Narváez, Gurutz / Liébana Martos, Ma del Carmen / Roldán Fontana, Carolina / Herrero Rodríguez, Carmen / Duro Ruiz, Gaspar / Pérez Parra, Santiago / Mera Fidalgo, Arantzazu / Hortos Alsina, Miquel / Alberich Conesa, Ana / Bladé Vidal, Lourdes / Merchante Gutiérrez, Nicolás / León Jiménez, Eva / Espíndola Gómez, Reinaldo / Erostarbe Gallardo, María / Martínez Pérez-Crespo, Pedro / Cisneros, José Miguel / Aguilar-Guisado, Manuela / Aldabó, Teresa / Bueno, Claudio / Cordero-Matía, Elisa / Escoresca, Ana / Infante, Carmen / Guillermo, Martín / Salto, Sonsoles / Gioia, Francesca / Vizcarra, Pilar / Fortún Abete, Jesús / Martín Dávila, Pilar / Moreno Guillén, Santiago / Oteo Revuelta, José A. / García-García, Concepción / Santibañez Sáenz, Paula / Cervera Acedo, Cristina / Azcona Gutiérrez, José M. / Reguera Iglesias, José María / Plata Ciezar, Antonio / Valiente de Santis, Lucia / Sobrino Diaz, Beatriz / Ruiz Mesa, Juan Diego

Clinical Microbiology and Infection ; ISSN 1198-743X

a multicentre cohort study (SAM-COVID-19)

2020

Keywords	Microbiology (medical) ; Infectious Diseases ; General Medicine ; covid19
Language	English
Publisher	Elsevier BV
Publishing country	us
Document type	Article ; Online
DOI	10.1016/j.cmi.2020.08.010
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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