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Article ; Online: Food, microbiome and colorectal cancer.

Niederreiter, Lukas / Adolph, Timon E / Tilg, Herbert

Digestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver

2018 Volume 50, Issue 7, Page(s) 647–652

Abstract: You are what you eat. This adage has been confirmed by many studies demonstrating the high impact of nutrition on risk of cardiovascular diseases, many malignancies and other diseases. Dietary factors are of major relevance in the evolution of colorectal ...

Abstract	You are what you eat. This adage has been confirmed by many studies demonstrating the high impact of nutrition on risk of cardiovascular diseases, many malignancies and other diseases. Dietary factors are of major relevance in the evolution of colorectal carcinoma. Various aspects are involved in colorectal carcinoma pathogenesis including genetics, lifestyle, age, chronic inflammation and others. It has only recently been recognized that the gut microbiota might reflect an important missing link in the interaction between diet and subsequent colorectal carcinoma development. Dietary factors are a major confounding factor affecting the composition of the intestinal microbiota. Several preclinical and clinical studies have recently suggested a role for the intestinal microbiota in potentially initiating and driving colorectal carcinoma. Therefore it is increasingly acknowledged that dietary factors might favor carcinogenesis via manipulation of the gut microbiota via potential outgrowth of certain bacterial populations, such as Fusobacterium nucleatum, Escherichia coli or Bacteroides fragilis. Excitingly, recent large clinical studies also highlighted a role for the gut microbiota and in particular Akkermansia muciniphila in tumor response toward chemotherapeutic agents and immune checkpoint inhibitors. This review will concentrate on the role of dietary factors in affecting the microbiota and implications in colorectal carcinoma.
MeSH term(s)	Colorectal Neoplasms/microbiology ; Diet ; Food ; Gastrointestinal Microbiome ; Humans
Language	English
Publishing date	2018-04-03
Publishing country	Netherlands
Document type	Journal Article ; Review
ZDB-ID	1459373-7
ISSN	1878-3562 ; 1125-8055
ISSN (online)	1878-3562
ISSN	1125-8055
DOI	10.1016/j.dld.2018.03.030
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Article ; Online: Decline in acute upper gastrointestinal bleeding during COVID-19 pandemic after initiation of lockdown in Austria.

Schmiderer, Andreas / Schwaighofer, Hubert / Niederreiter, Lukas / Profanter, Christoph / Steinle, Hartmut / Ziachehabi, Alexander / Tilg, Herbert

Endoscopy

2020 Volume 52, Issue 11, Page(s) 1036–1038

Abstract: Background: COVID-19 has spread rapidly around the world. The Austrian government implemented a lockdown on 16 March to contain further spread of the disease. We investigated the effects of lockdown on acute upper gastrointestinal (GI) bleeding in ... ...

Abstract	Background: COVID-19 has spread rapidly around the world. The Austrian government implemented a lockdown on 16 March to contain further spread of the disease. We investigated the effects of lockdown on acute upper gastrointestinal (GI) bleeding in Austria. Methods: We contacted 98 Austrian hospitals performing emergency endoscopies. The hospitals were asked to report upper GI endoscopies performed for recent hematemesis, melena, or both, and exhibiting endoscopically visible signs of bleeding. The study period was from 3 weeks before (calendar Week 9) to 3 weeks after (Week 14) initiation of the lockdown. Results: 61 % of Austrian hospitals, and importantly all major state hospitals, responded. A total of 575 upper GI bleedings occurred during the 3 weeks before and 341 during the 3 weeks after initiation of lockdown (40.7 % reduction). There was a 54.6 % decline in nonvariceal bleeding events at Week 14 compared with Week 9 (89 vs. 196), whereas rates of variceal hemorrhage did not change (15 vs. 17). Conclusions: National lockdown resulted in a dramatic decrease in upper GI bleeding events in Austrian hospitals.
MeSH term(s)	Austria ; Betacoronavirus ; COVID-19 ; Communicable Disease Control ; Coronavirus Infections/epidemiology ; Coronavirus Infections/prevention & control ; Coronavirus Infections/transmission ; Endoscopy ; Esophageal and Gastric Varices/diagnosis ; Esophageal and Gastric Varices/epidemiology ; Esophageal and Gastric Varices/therapy ; Gastrointestinal Hemorrhage/diagnosis ; Gastrointestinal Hemorrhage/epidemiology ; Gastrointestinal Hemorrhage/therapy ; Humans ; Pandemics/prevention & control ; Pneumonia, Viral/epidemiology ; Pneumonia, Viral/prevention & control ; Pneumonia, Viral/transmission ; SARS-CoV-2 ; Social Isolation
Keywords	covid19
Language	English
Publishing date	2020-07-09
Publishing country	Germany
Document type	Journal Article
ZDB-ID	80120-3
ISSN	1438-8812 ; 0013-726X
ISSN (online)	1438-8812
ISSN	0013-726X
DOI	10.1055/a-1178-4656
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Zs.A 669: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (1.OG) ab Jg. 2022: Lesesaal (EG)
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Article: Das duktale Adenokarzinom des Pankreas

Djanani, Angela / Niederreiter, Lukas / Niederreiter, Markus / Schmiderer, Andreas

Wiener medizinische Wochenschrift : Skriptum : Kongressjournal

2019 Volume 16, Issue 6, Page(s) 7

Language	German
Document type	Article
ZDB-ID	2142526-7
ISSN	1613-3803 ; 0043-5341
Database	Current Contents Medicine

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Zs.A 903 -Skriptum-: Show issues

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Article ; Online: Case report of a COVID-19-associated myocardial infarction with no obstructive coronary arteries: the mystery of the phantom embolus or local endothelitis.

Burkert, Francesco Robert / Niederreiter, Lukas / Dichtl, Wolfgang / Mayr, Agnes / Virgolini, Irene / Klauser, Andrea / Weiss, Günter / Bellmann-Weiler, Rosa

European heart journal. Case reports

2021 Volume 5, Issue 2, Page(s) ytaa521

Abstract: Background: Since the first documented outbreak of a novel severe acute respiratory syndrome inducing Coronavirus in China at the end of 2019 the virus has spread to all continents, leading the WHO to declare a pandemic in March 2020. While this virus ... ...

Abstract	Background: Since the first documented outbreak of a novel severe acute respiratory syndrome inducing Coronavirus in China at the end of 2019 the virus has spread to all continents, leading the WHO to declare a pandemic in March 2020. While this virus primarily targets the alveoli in the lungs, multiple authors have described an increased rate of thrombo-embolic events in affected patients. We present this case of a myocardial infarction with no obstructive coronary atherosclerosis in an otherwise healthy 48-year-old patient. Case summary: A 48-year-old female, presenting with chest pain radiating to her left shoulder with no cardiovascular risk factors other than genetic predisposition, was screened for Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) and tested positive. Although computed tomography angiography excluded obstructive coronary heart disease, cardiac magnetic resonance imaging showed an acute myocardial infarction with no obstructive coronary arteries of the inferior wall. The patient was treated with dual anti-platelet therapy, an angiotensin-converting-enzyme inhibitor and a statin, and assigned to a cardiac rehabilitation program. Conclusion: We report a serious thrombo-embolic event during an oligosymptomatic SARS-CoV-2 infection in a healthy, young patient. While these two diseases may have occurred simultaneously, by chance, it is possible that the pro-thrombotic effects of the SARS-CoV-2 infection facilitated the infarction. This case further demonstrates the significant cardiovascular morbidity potentially caused by SARS-CoV-2.
Language	English
Publishing date	2021-01-06
Publishing country	England
Document type	Journal Article
ISSN	2514-2119
ISSN (online)	2514-2119
DOI	10.1093/ehjcr/ytaa521
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Article: Endoplasmic reticulum stress and inflammatory bowel disease.

Niederreiter, Lukas / Kaser, Arthur

Acta gastro-enterologica Belgica

2011 Volume 74, Issue 2, Page(s) 330–333

Abstract: Endoplasmic reticulum (ER) stress arises from the accumulation of misfolded or unfolded proteins in the ER and elicits the unfolded protein response (UPR), an adaptive signalling pathway which aims at resolving ER stress. Genetic loci that confer risk ... ...

Abstract	Endoplasmic reticulum (ER) stress arises from the accumulation of misfolded or unfolded proteins in the ER and elicits the unfolded protein response (UPR), an adaptive signalling pathway which aims at resolving ER stress. Genetic loci that confer risk for both forms of inflammatory bowel disease (IBD) include genes that are centrally involved in the UPR, including X-box binding protein-1 (XBP1), anterior gradient protein-2 (AGR2) and orosomucoid-1-like 3 (ORMDL3). The intestinal epithelium, in particular mucin-secreting goblet and antimicrobial peptide-secreting Paneth cells appear particularly sensitive towards disturbances of the UPR. Supportive of this view are mice with a genetic deletion of Xbp1 specifically in the intestinal epithelium, which develop spontaneous intestinal inflammation histologically remarkably similar to human IBD. Apart from such primary genetic factors that determine the threshold of tolerable ER stress within the epithelium, secondary factors emanating from the environment might intersect with the UPR as well. These secondary factors might include microbial products, inflammatory mediators per se, hypoxia and glucose deprivation, pharmacological agents, and many others. Interaction of such secondary factors in a genetically susceptible host might provide the basis for intestinal inflammation and might provide a framework to investigate gene-environment interactions in human IBD, whereby a normally homeostatic adaptive response (i.e. the UPR) transforms into a potent pathomechanism of intestinal inflammation in the context of unresolved (i.e. unresolvable) ER stress.
MeSH term(s)	Animals ; Endoplasmic Reticulum/physiology ; Gene Expression Regulation ; Humans ; Inflammatory Bowel Diseases/genetics ; Inflammatory Bowel Diseases/metabolism ; Proteins/genetics ; Proteins/metabolism ; Signal Transduction ; Stress, Physiological/physiology
Chemical Substances	Proteins
Language	English
Publishing date	2011-06
Publishing country	Belgium
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	127060-6
ISSN	1784-3227 ; 0001-5644
ISSN	1784-3227 ; 0001-5644
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Article: Decline in acute upper gastrointestinal bleeding during COVID-19 pandemic after initiation of lockdown in Austria

Schmiderer, Andreas / Schwaighofer, Hubert / Niederreiter, Lukas / Profanter, Christoph / Steinle, Hartmut / Ziachehabi, Alexander / Tilg, Herbert

Endoscopy

Abstract: BACKGROUND: COVID-19 has spread rapidly around the world. The Austrian government implemented a lockdown on 16 March to contain further spread of the disease. We investigated the effects of lockdown on acute upper gastrointestinal (GI) bleeding in ... ...

Abstract	BACKGROUND: COVID-19 has spread rapidly around the world. The Austrian government implemented a lockdown on 16 March to contain further spread of the disease. We investigated the effects of lockdown on acute upper gastrointestinal (GI) bleeding in Austria. METHODS: We contacted 98 Austrian hospitals performing emergency endoscopies. The hospitals were asked to report upper GI endoscopies performed for recent hematemesis, melena, or both, and exhibiting endoscopically visible signs of bleeding. The study period was from 3 weeks before (calendar Week 9) to 3 weeks after (Week 14) initiation of the lockdown. RESULTS: 61â% of Austrian hospitals, and importantly all major state hospitals, responded. A total of 575 upper GI bleedings occurred during the 3 weeks before and 341 during the 3 weeks after initiation of lockdown (40.7â% reduction). There was a 54.6â% decline in nonvariceal bleeding events at Week 14 compared with Week 9 (89 vs. 196), whereas rates of variceal hemorrhage did not change (15 vs. 17). CONCLUSIONS: National lockdown resulted in a dramatic decrease in upper GI bleeding events in Austrian hospitals.
Keywords	covid19
Publisher	WHO
Document type	Article
Note	WHO #Covidence: #264811
Database	COVID19

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Article: Decline in acute upper gastrointestinal bleeding during Covid-19 pandemic after lockdown in Austria

Schmiderer, Andreas / Schwaighofer, Hubert / Niederreiter, Lukas / Profanter, Christoph / Steinle, Hartmut / Ziachehabi, Alexander / Tilg, Herbert

Endoscopy

Abstract: Background and study aims COVID-19 disease has rapidly spread all over the world. The Austrian government has implemented a lockdown to contain further spread of this disease on March 16th. We investigated the effects of lockdown on acute upper ... ...

Abstract	Background and study aims COVID-19 disease has rapidly spread all over the world. The Austrian government has implemented a lockdown to contain further spread of this disease on March 16th. We investigated the effects of lockdown on acute upper gastrointestinal (GI) bleeding in Austria. Patients and methods We contacted 98 Austrian hospitals performing emergency endoscopies. The hospitals were asked to report upper GI endoscopies performed because of recent hematemesis, melena, or both and exhibiting endoscopically visible signs of bleeding. Investigated time points included three weeks before and after lockdown. Results 61% of Austrian hospitals and importantly all major state hospitals responded. 575 upper GI bleedings occurred in the three weeks before and 341 after lockdown (40.7% reduction). When comparing the first and last calendar week of investigation we observed an almost 55% decline in nonvariceal bleeding events (calendar week 9 versus 14: 196 versus 89) whereas rates of variceal hemorrhage did not change (calendar week 9 versus 14: 17 versus 15). Conclusions National lockdown resulted in a dramatic decrease in upper gastrointestinal bleeding events.
Keywords	covid19
Publisher	WHO
Document type	Article
Note	WHO #Covidence: #32408356
Database	COVID19

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Article: Decline in acute upper gastrointestinal bleeding during COVID-19 pandemic after initiation of lockdown in Austria

Schmiderer, Andreas / Schwaighofer, Hubert / Niederreiter, Lukas / Profanter, Christoph / Steinle, Hartmut / Ziachehabi, Alexander / Tilg, Herbert

Endoscopy

2020 Volume 52, Issue 11, Page(s) 1036–1038

Abstract	Background: COVID-19 has spread rapidly around the world. The Austrian government implemented a lockdown on 16 March to contain further spread of the disease. We investigated the effects of lockdown on acute upper gastrointestinal (GI) bleeding in Austria. Methods: We contacted 98 Austrian hospitals performing emergency endoscopies. The hospitals were asked to report upper GI endoscopies performed for recent hematemesis, melena, or both, and exhibiting endoscopically visible signs of bleeding. The study period was from 3 weeks before (calendar Week 9) to 3 weeks after (Week 14) initiation of the lockdown. Results: 61 % of Austrian hospitals, and importantly all major state hospitals, responded. A total of 575 upper GI bleedings occurred during the 3 weeks before and 341 during the 3 weeks after initiation of lockdown (40.7 % reduction). There was a 54.6 % decline in nonvariceal bleeding events at Week 14 compared with Week 9 (89 vs. 196), whereas rates of variceal hemorrhage did not change (15 vs. 17). Conclusions: National lockdown resulted in a dramatic decrease in upper GI bleeding events in Austrian hospitals.
Language	English
Publishing date	2020-05-14
Publisher	Georg Thieme Verlag KG
Publishing place	Stuttgart ; New York
Document type	Article
ZDB-ID	80120-3
ISSN	1438-8812 ; 0013-726X
ISSN (online)	1438-8812
ISSN	0013-726X
DOI	10.1055/a-1178-4656
Database	Thieme publisher's database

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Zs.A 669: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (1.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MO 162: Show issues

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Article ; Online: Decline in acute upper gastrointestinal bleeding during COVID-19 pandemic after initiation of lockdown in Austria

Schmiderer, Andreas / Schwaighofer, Hubert / Niederreiter, Lukas / Profanter, Christoph / Steinle, Hartmut / Ziachehabi, Alexander / Tilg, Herbert

Endoscopy

2020 Volume 52, Issue 11, Page(s) 1036–1038

Abstract: Background COVID-19 has spread rapidly around the world. The Austrian government implemented a lockdown on 16 March to contain further spread of the disease. We investigated the effects of lockdown on acute upper gastrointestinal (GI) bleeding in Austria. ...

Abstract	Background COVID-19 has spread rapidly around the world. The Austrian government implemented a lockdown on 16 March to contain further spread of the disease. We investigated the effects of lockdown on acute upper gastrointestinal (GI) bleeding in Austria. Methods We contacted 98 Austrian hospitals performing emergency endoscopies. The hospitals were asked to report upper GI endoscopies performed for recent hematemesis, melena, or both, and exhibiting endoscopically visible signs of bleeding. The study period was from 3 weeks before (calendar Week 9) to 3 weeks after (Week 14) initiation of the lockdown. Results 61 % of Austrian hospitals, and importantly all major state hospitals, responded. A total of 575 upper GI bleedings occurred during the 3 weeks before and 341 during the 3 weeks after initiation of lockdown (40.7 % reduction). There was a 54.6 % decline in nonvariceal bleeding events at Week 14 compared with Week 9 (89 vs. 196), whereas rates of variceal hemorrhage did not change (15 vs. 17). Conclusions National lockdown resulted in a dramatic decrease in upper GI bleeding events in Austrian hospitals.
Keywords	Gastroenterology ; covid19
Language	English
Publisher	Georg Thieme Verlag KG
Publishing country	de
Document type	Article ; Online
ZDB-ID	80120-3
ISSN	1438-8812 ; 0013-726X
ISSN (online)	1438-8812
ISSN	0013-726X
DOI	10.1055/a-1178-4656
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Zs.A 669: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (1.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MO 162: Show issues

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Article ; Online: Anti-IL-12/23 in Crohn's disease: bench and bedside.

Niederreiter, Lukas / Adolph, Timon Erik / Kaser, Arthur

Current drug targets

2013 Volume 14, Issue 12, Page(s) 1379–1384

Abstract: The heterodimeric cytokines IL-12 and IL-23 play a key role in T helper cell and innate lymphocyte cell differentiation and expansion. They are composed of a shared p40 chain, which pairs with a p35 or p19 chain to form IL-12 and IL-23, respectively. ... ...

Abstract	The heterodimeric cytokines IL-12 and IL-23 play a key role in T helper cell and innate lymphocyte cell differentiation and expansion. They are composed of a shared p40 chain, which pairs with a p35 or p19 chain to form IL-12 and IL-23, respectively. Preclinical model systems have predicted an important role of the p40 chain in intestinal inflammation. Moreover, genome-wide association studies have revealed that variants of the gene encoding the IL-23 receptor, as well as the locus harboring the gene encoding the p40 chain, confer genetic risk for developing Crohn's disease (CD) and ulcerative colitis (UC). Two monoclonal antibodies neutralizing the p40 chain (ustekinumab and briakinumab) and hence blocking both IL-12 and IL-23 activity, have been developed, which demonstrated clinical benefit in early phase trials, and hinted towards efficacy in a subpopulation of patients with CD who had failed prior anti-TNF antibody treatment. A dedicated phase 3 clinical trial of ustekinumab in patients suffering from moderate-to-severe CD who had previously failed anti-TNF antibody treatment indeed demonstrated a significant benefit over placebo for clinical response, but not remission, in this particularly difficult to treat patient population. Here we review the immunological and genetic background to anti-IL-12/IL-23-directed therapeutic strategies, and the lessons that can be learned from results of these and related clinical trials that tackle associated biological pathways.
MeSH term(s)	Animals ; Antibodies, Monoclonal/pharmacology ; Antibodies, Monoclonal/therapeutic use ; Antibodies, Monoclonal, Humanized/pharmacology ; Antibodies, Monoclonal, Humanized/therapeutic use ; Clinical Trials as Topic ; Crohn Disease/drug therapy ; Crohn Disease/genetics ; Crohn Disease/immunology ; Disease Models, Animal ; Humans ; Immunity, Innate ; Interleukin-12 Subunit p40/antagonists & inhibitors ; Interleukin-12 Subunit p40/immunology ; Interleukin-23 Subunit p19/antagonists & inhibitors ; Interleukin-23 Subunit p19/immunology ; Ustekinumab
Chemical Substances	Antibodies, Monoclonal ; Antibodies, Monoclonal, Humanized ; Interleukin-12 Subunit p40 ; Interleukin-23 Subunit p19 ; briakinumab (978I8M0P8X) ; Ustekinumab (FU77B4U5Z0)
Language	English
Publishing date	2013-10-14
Publishing country	United Arab Emirates
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	2064859-5
ISSN	1873-5592 ; 1389-4501
ISSN (online)	1873-5592
ISSN	1389-4501
DOI	10.2174/13894501113146660229
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