LIVIVO - Search results -

Search results

Result 1 - 10 of total 60

Search options

Article: Intrinsic feedback factors producing inertial compensation in muscle.

Partridge, L D

2008 Volume 7, Issue 6, Page(s) 853–863

Abstract: An attempt was made to determine the factors causing the load-inertia compensation that has been observed in skeletal muscle. Cat skeletal muscle force output was determined as a function of the two variables, length and stimulus pulse rate. The results ... ...

Abstract	An attempt was made to determine the factors causing the load-inertia compensation that has been observed in skeletal muscle. Cat skeletal muscle force output was determined as a function of the two variables, length and stimulus pulse rate. The results were represented in a system diagram from which it becomes apparent that: (a) the length-tension relationship in muscle forms a functional, non-neural servo feedback; (b) the force-velocity curve appears as an oscillation-damping, velocity feedback in the muscle servo; (c) the nonlinear action of pulse rate on response is, in effect, in the input element to the muscle servo system. For purpose of analysis of the motor system it appears that these signal handling characteristics of muscle make it more nearly a "position servo" than a "force motor."
Language	English
Publishing date	2008-12-31
Publishing country	United States
Document type	Journal Article
ZDB-ID	218078-9
ISSN	1542-0086 ; 0006-3495
ISSN (online)	1542-0086
ISSN	0006-3495
DOI	10.1016/S0006-3495(67)86625-6
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

In stock of ZB MED Cologne/Königswinter

Zs.A 235: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (1.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MO 2: Show issues

Order via subito

This service is chargeable due to the Delivery terms set by subito. Orders including an article and supplementary material will be classified as separate orders. In these cases, fees will be demanded for each order.

Details ▾

Article: A mechanism for spike frequency adaptation.

Partridge, L D / Stevens, C F

The Journal of physiology

2006 Volume 256, Issue 2, Page(s) 315–332

Abstract: 1. Spike frequency adaptation was studied in large neurones of the marine molluscs Archidoris montereyensis and Anisodoris nobilis. These cells respond to a current step with a rapid rise in spike frequency followed by a gradual decline to a new steady ... ...

Abstract	1. Spike frequency adaptation was studied in large neurones of the marine molluscs Archidoris montereyensis and Anisodoris nobilis. These cells respond to a current step with a rapid rise in spike frequency followed by a gradual decline to a new steady level.2. An exponentially declining current, I(s), was measured when the cell was voltage clamped following an adapting spike train. The initial amplitude of this current depended on the preceding number of spikes and on the voltage to which the cell was clamped. A reversal potential (V(s)) for this current was obtained by clamping to various potentials following a spike train. The time constant (tau(s)) of decay of the current was dependent upon the clamping potential.3. Clamping the membrane potential to a constant test level from various initial levels initiates an exponentially decaying current of similar time constant. The voltage dependence of the steady-state conductance (g(s)a(s)(V, infinity)) associated with this current was determined using this technique.4. Equations for neural repetitive firing (Connor & Stevens, 1971c) were modified by the addition of a term describing these slow membrane currents: [Formula: see text]. The solution to the modified equation was in good agreement with the spike frequency adaptation observed in these cells.
Language	English
Publishing date	2006-09-21
Publishing country	England
Document type	Journal Article
ZDB-ID	3115-x
ISSN	1469-7793 ; 0022-3751
ISSN (online)	1469-7793
ISSN	0022-3751
DOI	10.1113/jphysiol.1976.sp011327
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

In stock of ZB MED Cologne/Königswinter

Uc I Zs.65: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾

Article: Cytoplasmic Ca2+ activity regulation as measured by a calcium-activated current.

Partridge, L D

Brain research

1994 Volume 647, Issue 1, Page(s) 76–82

Abstract: Calcium-activated non-selective cation (CAN) currents were activated by quantitative injections of Ca2+ into voltage clamped bursting neurons of the snails Helix aspersa or Helix pomatia. Membrane potential was held at the potassium equilibrium potential ...

Abstract	Calcium-activated non-selective cation (CAN) currents were activated by quantitative injections of Ca2+ into voltage clamped bursting neurons of the snails Helix aspersa or Helix pomatia. Membrane potential was held at the potassium equilibrium potential and CAN currents were fit with a rising and falling exponential function. Ca2+ transporters and pumps of the cell membrane, endoplasmic reticulum, and mitochondria were selectively blocked with pharmacological agents. Bath solutions containing 0 Na Ringers, chlorpromazine, Na3VO4, or thapsigargin did not significantly change the CAN current decay constants from those measured in Ringers. External 2,4-dinitrophenol or internal ruthenium red, however, significantly lengthened the CAN current decay constant. It is concluded that mitochondria are the most important sink for sub-membrane Ca2+ activity in the range necessary to effectively activate CAN currents.
MeSH term(s)	2,4-Dinitrophenol ; Animals ; Biological Transport/drug effects ; Calcium/metabolism ; Calcium/physiology ; Calcium Channel Blockers/pharmacology ; Chlorpromazine/pharmacology ; Cytoplasm/metabolism ; Dinitrophenols/pharmacology ; Electrophysiology ; Helix (Snails) ; Mitochondria/metabolism ; Neurons/metabolism ; Neurons/physiology ; Ruthenium Red/pharmacology
Chemical Substances	Calcium Channel Blockers ; Dinitrophenols ; Ruthenium Red (11103-72-3) ; 2,4-Dinitrophenol (Q13SKS21MN) ; Calcium (SY7Q814VUP) ; Chlorpromazine (U42B7VYA4P)
Language	English
Publishing date	1994-05-30
Publishing country	Netherlands
Document type	Journal Article ; Research Support, U.S. Gov't, Non-P.H.S.
ZDB-ID	1200-2
ISSN	1872-6240 ; 0006-8993
ISSN (online)	1872-6240
ISSN	0006-8993
DOI	10.1016/0006-8993(94)91400-1
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Zs.A 161: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
Jg. 1995 - 2021: Lesesall (1.OG)
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article: Neurosteroid-induced enhancement of glutamate transmission in rat hippocampal slices.

Partridge, L D / Valenzuela, C F

Neuroscience letters

2001 Volume 301, Issue 2, Page(s) 103–106

Abstract: Pregnenolone sulfate, one of the most abundantly produced neurosteroids in the hippocampus, has well characterized effects at postsynaptic receptors including the N-methyl-D-asparate type of glutamate receptor. Little is known, however, about the ... ...

Abstract	Pregnenolone sulfate, one of the most abundantly produced neurosteroids in the hippocampus, has well characterized effects at postsynaptic receptors including the N-methyl-D-asparate type of glutamate receptor. Little is known, however, about the mechanism of action of neurosteroids on the release of glutamate. In this study we describe a robust effect of pregnenolone sulfate at glutamatergic synapses in the CA1 region of the hippocampus. In particular, we found that pregnenolone sulfate enhances paired-pulse facilitation of EPSPs at the two major classes of ionotropic glutamate receptors with an EC(50)<1 microM. Thus, we propose a novel mechanism of action of neurosteroids in hippocampal neurons that involves the modulation of glutamate release.
MeSH term(s)	Animals ; Excitatory Postsynaptic Potentials/drug effects ; Glutamic Acid/metabolism ; Hippocampus/physiology ; Male ; Neuronal Plasticity/physiology ; Organ Culture Techniques ; Pregnenolone/pharmacology ; Rats ; Rats, Sprague-Dawley ; Receptors, AMPA/metabolism ; Receptors, N-Methyl-D-Aspartate/metabolism ; Synaptic Transmission/drug effects
Chemical Substances	Receptors, AMPA ; Receptors, N-Methyl-D-Aspartate ; pregnenolone sulfate (04Y4D91RG0) ; Glutamic Acid (3KX376GY7L) ; Pregnenolone (73R90F7MQ8)
Language	English
Publishing date	2001-03-30
Publishing country	Ireland
Document type	Journal Article
ZDB-ID	194929-9
ISSN	1872-7972 ; 0304-3940
ISSN (online)	1872-7972
ISSN	0304-3940
DOI	10.1016/s0304-3940(01)01613-5
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

In stock of ZB MED Cologne/Königswinter

Zs.A 1166: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
Jg. 1995 - 2021: Lesesall (1.OG)
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾

Article: Block of hippocampal CAN channels by flufenamate.

Partridge, L D / Valenzuela, C F

Brain research

2000 Volume 867, Issue 1-2, Page(s) 143–148

Abstract: Ca(2+)-activated non-selective cation (CAN) channels are activated by cytoplasmic Ca(2+) and I(CAN) underlies many slow depolarizing processes in neurons including a putative role in excitotoxicity. CAN channels in many non-neuronal cells are blocked by ... ...

Abstract	Ca(2+)-activated non-selective cation (CAN) channels are activated by cytoplasmic Ca(2+) and I(CAN) underlies many slow depolarizing processes in neurons including a putative role in excitotoxicity. CAN channels in many non-neuronal cells are blocked by non-steroidal antiinflammatory drugs that are derivatives of diphenylamine-2-carboxylate (DPC). The DPC derivative flufenamate (FFA) has a complex effect on certain neurons, whereby it blocks CAN channels and increases [Ca(2+)](i). We report here that FFA, but not the parent compound, DPC, blocks CAN channels in hippocampal CA1 neurons. As was the case in other neurons, the effects of FFA are complex and include a maintained rise in [Ca(2+)](i). Furthermore, the CAN channel blocking ability of FFA persists even when the channels have been potentiated by a Ca(2+)-dependent process. The use of a CAN channel-blocking drug is important for delineating CAN channel-dependent processes and may provide a basis for therapy for CAN channel-dependent events in ischemia.
MeSH term(s)	Action Potentials/drug effects ; Animals ; Anti-Inflammatory Agents, Non-Steroidal/pharmacology ; Calcium/metabolism ; Calcium Channels/physiology ; Cells, Cultured ; Excitatory Postsynaptic Potentials/drug effects ; Flufenamic Acid/pharmacology ; Hippocampus/cytology ; Hippocampus/physiology ; Ion Channel Gating/drug effects ; Neurons/chemistry ; Neurons/cytology ; Neurons/physiology ; Rats ; Rats, Sprague-Dawley
Chemical Substances	Anti-Inflammatory Agents, Non-Steroidal ; Calcium Channels ; Flufenamic Acid (60GCX7Y6BH) ; Calcium (SY7Q814VUP)
Language	English
Publishing date	2000-06-09
Publishing country	Netherlands
Document type	Journal Article ; Research Support, U.S. Gov't, P.H.S.
ZDB-ID	1200-2
ISSN	1872-6240 ; 0006-8993
ISSN (online)	1872-6240
ISSN	0006-8993
DOI	10.1016/s0006-8993(00)02275-7
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

In stock of ZB MED Cologne/Königswinter

Zs.A 161: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
Jg. 1995 - 2021: Lesesall (1.OG)
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾

Article: Ca2+ store-dependent potentiation of Ca2+-activated non-selective cation channels in rat hippocampal neurones in vitro.

Partridge, L D / Valenzuela, C F

The Journal of physiology

1999 Volume 521 Pt 3, Page(s) 617–627

Abstract: 1. Potentiation of calcium-activated non-selective cation (CAN) channels was studied in rat hippocampal neurones. CAN channels were activated by IP3-dependent Ca2+ release following metabotropic glutamate receptor (mGluR) stimulation either by Schaffer ... ...

Abstract	1. Potentiation of calcium-activated non-selective cation (CAN) channels was studied in rat hippocampal neurones. CAN channels were activated by IP3-dependent Ca2+ release following metabotropic glutamate receptor (mGluR) stimulation either by Schaffer collateral input to CA1 neurones in brain slices in which ionotropic glutamate and GABAA receptors, K+ channels, and the Na+-Ca2+ exchanger were blocked or by application of the mGluR antagonist ACPD in cultured hippocampal neurones. 2. The CAN channel-dependent depolarization (DeltaVCAN) was potentiated when [Ca2+]i was increased in neurones impaled with Ca2+-containing microelectrodes. 3. Fura-2 measurements revealed a biphasic increase in [Ca2+]i when 200 microM ACPD was bath applied to cultured hippocampal neurones. This increase was greatly attenuated in the presence of Cd2+. 4. Thapsigargin (1 microM) caused marked potentiation of DeltaVCAN in CA1 neurones in the slices and of the CAN current (ICAN) measured in whole cell-clamped cultured hippocampal neurones. 5. Ryanodine (20 microM) also led to a potentiation of DeltaVCAN while neurones pretreated with 100 microM dantrolene failed to show potentiation of DeltaVCAN when impaled with Ca2+-containing microelectrodes. 6. The mitochondrial oxidative phosphorylation uncoupler carbonyl cyanide m-chlorophenyl hydrazone (2 microM) also caused a potentiation of DeltaVCAN. 7. CAN channels are subject to considerable potentiation following an increase in [Ca2+]i due to Ca2+ release from IP3-sensitive, Ca2+-sensitive, or mitochondrial Ca2+ stores. This ICAN potentiation may play a crucial role in the 'amplification' phase of excitotoxicity.
MeSH term(s)	Animals ; Calcium/metabolism ; Calcium/physiology ; Cations/metabolism ; Cells, Cultured ; Cycloleucine/analogs & derivatives ; Cycloleucine/pharmacology ; Cytoplasm/metabolism ; Electrophysiology ; Excitatory Postsynaptic Potentials/drug effects ; Excitatory Postsynaptic Potentials/physiology ; Hippocampus/cytology ; Hippocampus/metabolism ; Image Processing, Computer-Assisted ; Inositol 1,4,5-Trisphosphate/pharmacology ; Ion Channels/metabolism ; Mitochondria/drug effects ; Mitochondria/metabolism ; Neurons/metabolism ; Patch-Clamp Techniques ; Pyramidal Cells/metabolism ; Rats ; Rats, Sprague-Dawley ; Receptors, Metabotropic Glutamate/agonists
Chemical Substances	Cations ; Ion Channels ; Receptors, Metabotropic Glutamate ; Cycloleucine (0TQU7668EI) ; 1-amino-1,3-dicarboxycyclopentane (111900-32-4) ; Inositol 1,4,5-Trisphosphate (85166-31-0) ; Calcium (SY7Q814VUP)
Language	English
Publishing date	1999-12-15
Publishing country	England
Document type	Journal Article ; Research Support, U.S. Gov't, P.H.S.
ZDB-ID	3115-x
ISSN	1469-7793 ; 0022-3751
ISSN (online)	1469-7793
ISSN	0022-3751
DOI	10.1111/j.1469-7793.1999.00617.x
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

In stock of ZB MED Cologne/Königswinter

Uc I Zs.65: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾

Article: Dissociated dopaminergic neurons from substantia nigra zona compacta in young rats lack functional NMDA receptors.

Wu, J / Partridge, L D

Pflugers Archiv : European journal of physiology

1998 Volume 435, Issue 5, Page(s) 699–704

Abstract: Glutamate-mediated excitotoxicity plays an important role in the degeneration of nigrostriatal dopamine (DA) neurons induced by 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), although the role of the N-methyl D-aspartate (NMDA) receptor subtype in ... ...

Abstract	Glutamate-mediated excitotoxicity plays an important role in the degeneration of nigrostriatal dopamine (DA) neurons induced by 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), although the role of the N-methyl D-aspartate (NMDA) receptor subtype in this process is still uncertain. We studied glutamate receptor subtype agonist-induced ionic currents in acutely dissociated DAergic neurons from the rat substantia nigra zona compacta (SNc) using the nystatin-perforated patch-clamp whole-cell recording technique. The results fall into four main categories. First, single neurons, freshly isolated from SNc, exhibited a large soma and multipolar morphology, responded to DA, and stained positively for tyrosine hydroxylase (TH). Second, rapid application of L-glutamate (> 10(-5) M) induced an inward current with minimal desensitization at a clamp voltage of -60 mV. Third, kainic acid (KA) or alpha-amino-3-hydroxy-5-methyl-isoxazole (AMPA) induced an inward current that was similar to the glutamate-induced current while, in the same neuron, NMDA (10(-4) M) failed to induce any current response in Mg2+-free solution that contained 10(-5) M glycine at a clamp voltage of -60 mV. Under the same experimental conditions, NMDA induced a clear current response in isolated substantia nigra reticulata (SNr) neurons. Fourth, the specific NMDA receptor antagonist DL-2-amino-5-phosphonovaleric acid (APV, 10(-4) M) failed to block 10(-4) M glutamate-induced inward current, while the specific KA/AMPA receptor antagonist 6-cyano-7-nitroguinoxaline-2, 3-dione (CNQX, 10(-5) M) completely blocked the glutamate-induced current. These results indicate that in single SNc DAergic neurons of 2-week-old rats, L-glutamate-induced inward current is mediated by non-NMDA receptors rather than by NMDA receptors.
MeSH term(s)	Animals ; Dopamine/metabolism ; Excitatory Amino Acid Antagonists/pharmacology ; Glutamic Acid/pharmacology ; In Vitro Techniques ; Ionophores ; Neurons/metabolism ; Neurons/physiology ; Nystatin ; Patch-Clamp Techniques ; Rats ; Rats, Sprague-Dawley ; Receptors, AMPA/drug effects ; Receptors, AMPA/physiology ; Receptors, Kainic Acid/drug effects ; Receptors, Kainic Acid/physiology ; Receptors, N-Methyl-D-Aspartate/drug effects ; Receptors, N-Methyl-D-Aspartate/physiology ; Substantia Nigra/cytology ; Substantia Nigra/metabolism ; Substantia Nigra/physiology
Chemical Substances	Excitatory Amino Acid Antagonists ; Ionophores ; Receptors, AMPA ; Receptors, Kainic Acid ; Receptors, N-Methyl-D-Aspartate ; Nystatin (1400-61-9) ; Glutamic Acid (3KX376GY7L) ; Dopamine (VTD58H1Z2X)
Language	English
Publishing date	1998-04
Publishing country	Germany
Document type	Journal Article
ZDB-ID	6380-0
ISSN	1432-2013 ; 0031-6768
ISSN (online)	1432-2013
ISSN	0031-6768
DOI	10.1007/s004240050571
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

In stock of ZB MED Cologne/Königswinter

Uc I Zs.35: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾

Article: The good enough calculi of evolving control systems: evolution is not engineering.

Partridge, L D

The American journal of physiology

1982 Volume 242, Issue 3, Page(s) R173–7

Abstract: In evolved aggregates of accidentally invented elements, retained when statistically good enough to identify limitations of antecedent systems, survival value might favor operators incorporating aspects of, while not identical with, feedback, feedforward, ...

Abstract	In evolved aggregates of accidentally invented elements, retained when statistically good enough to identify limitations of antecedent systems, survival value might favor operators incorporating aspects of, while not identical with, feedback, feedforward, state varible, and "homeostatic" control. Generally, simple organizational increments should predominate. After invention, an internal controller with readily modifiable rules could facilitate evolution of compound inventions, but criteria controlling rule changes would be only indirectly (probably imperfectly) survival referent. Consequent to combination of independent invention with indirect criteria and statistical acceptance, evolved control logic could be: both redundant and incomplete; good enough with malefic aspects; built of loosely linked or autonomous sublogics; and a source of good enough solutions from incomplete information. The partially explicit rules are defined more by rejections than by ratifications. Study of the result based on formal logic, engineering conventions, and familiar coordinate systems could conceive illegitimate illusions of understanding.
MeSH term(s)	Biological Evolution ; Engineering ; Feedback ; Homeostasis ; Logic ; Models, Biological
Language	English
Publishing date	1982-03
Publishing country	United States
Document type	Journal Article
ZDB-ID	2948-8
ISSN	0002-9513
ISSN	0002-9513
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Uc I Zs.89: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular ab Jg. 2022: Lesesaal (EG)
Zs.MA 19: Show issues

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article: Calcium independence of slow currents underlying spike frequency adaptation.

Partridge, L D

Journal of neurobiology

1980 Volume 11, Issue 6, Page(s) 613–622

Abstract: This study assessed the role of calcium in the activation of the slow potassium current responsible for spike frequency adaptation in molluscan neurons. Inward calcium currents were eliminated by using Co2+, Cd2+, or OCa2+ EGTA in the bathing solution. ... ...

Abstract	This study assessed the role of calcium in the activation of the slow potassium current responsible for spike frequency adaptation in molluscan neurons. Inward calcium currents were eliminated by using Co2+, Cd2+, or OCa2+ EGTA in the bathing solution. In each case adaptation was found to persist, as did the slow current believed to be responsible for adaptation. Injection of EGTA into neurons was also found not to block adaptation. This potassium current provides an example of a slow voltage-dependent potassium process which is independent of calcium influx.
MeSH term(s)	Action Potentials/drug effects ; Adaptation, Physiological ; Animals ; Cadmium/pharmacology ; Calcium/physiology ; Cell Membrane Permeability/drug effects ; Cobalt/pharmacology ; Egtazic Acid/pharmacology ; Ganglia/physiology ; Helix (Snails)/physiology ; Lymnaea/physiology ; Nickel/pharmacology
Chemical Substances	Cadmium (00BH33GNGH) ; Cobalt (3G0H8C9362) ; Egtazic Acid (526U7A2651) ; Nickel (7OV03QG267) ; Calcium (SY7Q814VUP)
Language	English
Publishing date	1980-11
Publishing country	United States
Document type	Comparative Study ; Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	300903-8
ISSN	1097-4695 ; 0022-3034
ISSN (online)	1097-4695
ISSN	0022-3034
DOI	10.1002/neu.480110610
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Zs.A 853: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
Jg. 1995 - 2021: Lesesall (1.OG)
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article: Control of cell function by neuronal calcium-activated nonselective (CAN) cation channels.

Partridge, L D / Swandulla, D

EXS

1993 Volume 66, Page(s) 175–183

MeSH term(s)	Action Potentials/drug effects ; Animals ; Calcium/metabolism ; Calcium/pharmacology ; Calcium/physiology ; Cell Membrane/physiology ; Ion Channels/drug effects ; Ion Channels/physiology ; Membrane Potentials/drug effects ; Neurons/physiology ; Second Messenger Systems ; Serotonin/pharmacology
Chemical Substances	Ion Channels ; Serotonin (333DO1RDJY) ; Calcium (SY7Q814VUP)
Language	English
Publishing date	1993
Publishing country	Switzerland
Document type	Journal Article ; Review
ISSN	1023-294X
ISSN	1023-294X
DOI	10.1007/978-3-0348-7327-7_13
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

Inter-library loan at ZB MED

Your chosen title can be delivered directly to ZB MED Cologne location if you are registered as a user at ZB MED Cologne.

To top

Full text online

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

Full text online

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

Full text online

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

Full text online

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

Full text online

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

Full text online

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

Order via subito

Inter-library loan at ZB MED