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  1. Article ; Online: The application of the Kampala trauma Score for prehospital assessment of severity of injuries and prediction of outcome after severe trauma

    Milivojčević-Bevc Ivana / Tasić-Uroš Danijela / Anđelić Slađana / Čolaković Goran / Petrović Snežana / Stefanović Ivana / Vukićević Verica

    Halo 194, Vol 29, Iss 2-3, Pp 68-

    2023  Volume 73

    Abstract: Severe trauma is the main cause of mortality and disability in modern society. Emergency medical doctors are usually the first to establish contact with the injured person, and the extent of definitive care largely depends on their correct assessment of ... ...

    Abstract Severe trauma is the main cause of mortality and disability in modern society. Emergency medical doctors are usually the first to establish contact with the injured person, and the extent of definitive care largely depends on their correct assessment of the severity of the injury, using an adequate pre-hospital trauma score. Injury severity scores are used to numerically categorize the type and extent of the injury. They represent an important additional instrument, which is used to enable faster triage, the categorization of injury severity, adequate care, treatment, and transport of patients with multiple injuries to the appropriate hospital. They are also important in research. This paper aims to suggest, using several case reports, the possibility of pre-hospital use of the Kampala Trauma Score (KTS) as an easily applicable and very suitable system for monitoring the condition and predicting the outcome of seriously injured patients. The patients were primarily assessed at the pre-hospital level and assigned a certain injury severity score according to the KTS, which later proved to reflect their definitive outcome. It can be concluded that the KTS is an effective scoring system that can be used during initial triage of the seriously injured for categorization of the severity of the injury, prediction of mortality and necessity of hospitalization. The possibility of its potential application during emergency care of the seriously injured, both for differentiating the severity of injuries and for predicting the definitive outcome, is indicated. However, due to the limited number of patients, original research should be conducted on a larger sample.
    Keywords kampala trauma score ; trauma ; outcome ; Medicine (General) ; R5-920
    Language English
    Publishing date 2023-01-01T00:00:00Z
    Publisher City Medical emergency department, Belgrade
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  2. Article ; Online: The lexicon manuscript by Dimitrije Čemerikić as a source for ethnolinguistic and ethnologic research

    Petrović Snežana / Tasovac Toma

    Glasnik Etnografskog Instituta SANU, Vol 62, Iss 2, Pp 171-

    2014  Volume 180

    Abstract: This paper highlights the importance of Dimitrije Čemerikić’s handwritten collection of words from the historic city of Prizren as a valuable resource for the ethnolinguistic and ethnologic study of its colorful, multicultural past. The manuscript, which ...

    Abstract This paper highlights the importance of Dimitrije Čemerikić’s handwritten collection of words from the historic city of Prizren as a valuable resource for the ethnolinguistic and ethnologic study of its colorful, multicultural past. The manuscript, which contains around 16,000 lemmas with definitions and examples, was compiled in the middle of the 20th century. The original is nowadays archived at the Institute for the Serbian Language of the Serbian Academy of Arts and Sciences, while a digital edition has been available since 2013 via the Platform for Transcription of Serbian Handwritten Heritage, http://prepis.org. The paper shows how Čemerikić’s lexicographic treatment of the Prizren dialect reflects various aspects of popular culture (customs, superstitions, witchcraft) and urban life (guilds, social and ethnic relations, etc.) by analyzing exemplary lexemes such dečanska neprelja (lazy female, lit. ‘woman from Dečani who doesn’t know how to weave’), martifal (divination on St. George’s Day), dete (child), kuče (dog), magareći mozak (potion, lit. ‘donkey’s brain’), meso govecko (beef), esnaf (guild), Kaljaja (fortress), Latinka (Albanian Catholic female), kisela mrva (pejorative for Aromanian, lit. ‘sour crumb’), kožuvar (slang for Russian, lit. ‘leather-worker’), Karafera, mladoženja (groom), mladanevesta (bride), and dati (to give). [Projekat Ministarstva nauke Republike Srbije, br. 47016: Interdisciplinarno istraživanje kulturnog i jezičkog nasleđa Srbije i izrada multimedijalnog internet portala „Pojmovnik srpske kulture“]
    Keywords Prizren ; dialects ; Serbian language ; ethnolinguistic ; ethnology ; Dimitrije Čemerkić ; Anthropology ; GN1-890
    Language English
    Publishing date 2014-01-01T00:00:00Z
    Publisher Institute of Ethnography, SASA, Belgrade
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  3. Article ; Online: Tracing lexical borrowings in the Balkans Albanian loan-words in the Serbian speech of Prizren

    Petrović Snežana

    Balcanica, Vol 2003, Iss 34, Pp 157-

    2003  Volume 172

    Abstract: During the past centuries the city of Prizren has persisted as a unique multiethnic and multicultural environment atypical even for the Balkans, well-known for its ethnic, linguistic, cultural and religious diversity. Various nations used to live and ... ...

    Abstract During the past centuries the city of Prizren has persisted as a unique multiethnic and multicultural environment atypical even for the Balkans, well-known for its ethnic, linguistic, cultural and religious diversity. Various nations used to live and pass through Prizren in the course of more than six centuries – the Serbs Turks, Albanians, Aromanians, Greeks and others – leaving their specific marks, visible on the city itself as well as the life and the mentality of its inhabitants. One of the most vivid testimonies to this long-term coexistence can be seen in vernaculars of the local population. This paper presents some Albanian loan-words from the Serbian speech of Prizren hitherto not registered (or not recognised as such) in the respective etymological dictionaries and linguistic studies. The aim of this work is to offer their etymological analysis, through an investigation of the ways and mechanisms of lexical borrowing in the limited area of this town and to observe this phenomenon in a broader Balkan perspective. Analyzed are the following words: bajmak m., adj. indecl. "with bow legs (of man or horse)", cub adj. indecl. "short; short-tailed", cub m., adj. indecl. "robber, outlaw", ćul adj. indecl. "soaking wet", điza f. "kind of crumbly cheese", glistra f. "worm", korse, korsem, krsem adv. "perhaps, as if", kulme n., kuljma f. "top of the roof", ljajka f. "lie", ljaper m. "idler", ljočka f. "darling, sweetheart", ljum adj. indecl. "dear", ljunga f. "boil; swelling", putarka f. "salted and dried caviar", ravš adv. "flat", roktar m. "servant, attendant", škret adj. "lonely, deserted, empty", škrum adj. indecl. "dry as ashes".
    Keywords History of Balkan Peninsula ; DR1-2285 ; History (General) and history of Europe ; D ; DOAJ:History ; DOAJ:History and Archaeology
    Subject code 900
    Language English
    Publishing date 2003-01-01T00:00:00Z
    Publisher Institute for Balkan Studies SASA
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  4. Article ; Online: Interleukin 6 in maternal serum as marker of bacterial infection and preterm delivery

    Erić Želimir / Patić Aleksandra / Bogavac Mirjana / Tepić-Petrović Snežana

    Scripta Medica, Vol 48, Iss 2, Pp 114-

    2017  Volume 119

    Abstract: Introduction: Preterm delivery remains a burning issue all over the world, especially in Serbia because of the rate of negative natural increase. IL-6 can stimulate the release of prostaglandins and cause premature contractions and premature labor. Aim ... ...

    Abstract Introduction: Preterm delivery remains a burning issue all over the world, especially in Serbia because of the rate of negative natural increase. IL-6 can stimulate the release of prostaglandins and cause premature contractions and premature labor. Aim of the Study: The aim of this research is to examine the importance of infection in the occurrence of premature contractions and to examine whether the preterm labor is associated with increased concentrations of IL-6 in patients with intact fetal membranes. Patients and Methods: We examined 83 pregnant women. The age range was between 15 and 43 years. The experimental group had 53 pregnant women and a control group was consisted of 30 pregnant women. All pregnant women had singleton pregnancies. The age of pregnancy in both groups was between the 21st and 35th week of gestation. The experimental group was divided into three groups according to the localization of infection: bacterial vaginosis, an infection of the cervix and urinary tract infections. The laboratory identification of IL-6 was performed as a double sandwich ELISA method. The reagents used for the identification of IL-6 were manufactured by Beckman-Coulter and were strictly intended for the research. Results: There were 34 pregnant women or 64% with infection of the cervix, 26 pregnant women or 49% had a positive urine culture, while bacterial vaginosis was present in 47 pregnant women or 89%. A positive finding on all localization was found in 14 pregnant women or 26%. In the experimental group, IL-6 was detected in 37 pregnant women or 70%. The mean value of IL-6 proven in the experimental group was 20.6 pg/ml (SD=18.2, n=53). Conclusion: This research demonstrated a direct link between a bacterial infection and preterm delivery. IL-6 can be used as a serological marker of bacterial infection and preterm delivery.
    Keywords interleukin-6 ; bacterial infections ; premature birth ; serologic tests ; Medicine ; R
    Subject code 610
    Language English
    Publishing date 2017-01-01T00:00:00Z
    Publisher Medical Society of the Republic of Srpska, Banja Luka, University of Banja Luka. Faculty of Medicine
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  5. Article ; Online: Missense mutations and proximal RTA. Have we reached a new threshold? Focus on "missense mutation T485S alters NBCe1-A electrogenicity causing proximal renal tubular acidosis".

    Petrovic, Snezana / Dubose, Thomas D

    American journal of physiology. Cell physiology

    2013  Volume 305, Issue 4, Page(s) C373–4

    MeSH term(s) Acidosis, Renal Tubular/metabolism ; Humans ; Kidney Tubules, Proximal/metabolism ; Mutation, Missense ; Sodium-Bicarbonate Symporters/metabolism
    Chemical Substances SLC4A4 protein, human ; Sodium-Bicarbonate Symporters
    Language English
    Publishing date 2013-05-29
    Publishing country United States
    Document type Editorial ; Comment
    ZDB-ID 392098-7
    ISSN 1522-1563 ; 0363-6143
    ISSN (online) 1522-1563
    ISSN 0363-6143
    DOI 10.1152/ajpcell.00152.2013
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  6. Article ; Online: An Acidic Environment Induces APOL1-Associated Mitochondrial Fragmentation.

    Li, DengFeng / Snipes, James A / Murea, Mariana / Molina, Anthony J A / Divers, Jasmin / Freedman, Barry I / Ma, Lijun / Petrovic, Snezana

    American journal of nephrology

    2020  Volume 51, Issue 9, Page(s) 695–704

    Abstract: Background: Apolipoprotein L1 gene (APOL1) G1 and G2 kidney-risk variants (KRVs) cause CKD in African Americans, inducing mitochondrial dysfunction. Modifying factors are required, because a minority of individuals with APOL1 high-risk genotypes develop ...

    Abstract Background: Apolipoprotein L1 gene (APOL1) G1 and G2 kidney-risk variants (KRVs) cause CKD in African Americans, inducing mitochondrial dysfunction. Modifying factors are required, because a minority of individuals with APOL1 high-risk genotypes develop nephropathy. Given that APOL1 function is pH-sensitive and the pH of the kidney interstitium is <7, we hypothesized the acidic kidney interstitium may facilitate APOL1 KRV-induced mitochondrial dysfunction.
    Methods: Human embryonic kidney (HEK293) cells conditionally expressing empty vector (EV), APOL1-reference G0, and G1 or G2 KRVs were incubated in media pH 6.8 or 7.4 for 4, 6, or 8 h. Genotype-specific pH effects on mitochondrial length (µm) were assessed using confocal microscopy in live cells and Fiji derivative of ImageJ software with MiNA plug-in. Lower mitochondrial length indicated fragmentation and early dysfunction.
    Results: After 6 h doxycycline (Dox) induction in pH 6.8 media, G2-expressing cells had shorter mitochondria (6.54 ± 0.40) than cells expressing EV (7.65 ± 0.72, p = 0.02) or G0 (7.46 ± 0.31, p = 0.003). After 8 h Dox induction in pH 6.8 media, both G1- (6.21 ± 0.26) and G2-expressing cells had shorter mitochondria (6.46 ± 0.34) than cells expressing EV (7.13 ± 0.32, p = 0.002 and p = 0.008, respectively) or G0 (7.22 ± 0.45, p = 0.003 and p = 0.01, respectively). Mitochondrial length in cells incubated in pH 7.4 media were comparable after 8 h Dox induction regardless of genotype. APOL1 mRNA expression and cell viability were comparable regardless of pH or genotype after 8 h Dox induction.
    Conclusion: Acidic pH facilitates early mitochondrial dysfunction induced by APOL1 G1 and G2 KRVs in HEK293 cells. We propose that the acidic kidney interstitium may play a role in APOL1-mediated mitochondrial pathophysiology and nephropathy.
    MeSH term(s) African Americans/genetics ; Apolipoprotein L1/genetics ; Apolipoprotein L1/metabolism ; Culture Media/chemistry ; Genetic Predisposition to Disease ; HEK293 Cells ; Humans ; Hydrogen-Ion Concentration ; Kidney/chemistry ; Kidney/cytology ; Kidney/pathology ; Mitochondria/pathology ; Recombinant Proteins/genetics ; Recombinant Proteins/metabolism ; Renal Insufficiency, Chronic/genetics ; Renal Insufficiency, Chronic/pathology
    Chemical Substances APOL1 protein, human ; Apolipoprotein L1 ; Culture Media ; Recombinant Proteins
    Language English
    Publishing date 2020-08-31
    Publishing country Switzerland
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 604540-6
    ISSN 1421-9670 ; 0250-8095
    ISSN (online) 1421-9670
    ISSN 0250-8095
    DOI 10.1159/000509989
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  7. Article ; Online: Adaptation by the collecting duct to an exogenous acid load is blunted by deletion of the proton-sensing receptor GPR4.

    Sun, Xuming / Stephens, Lisa / DuBose, Thomas D / Petrovic, Snezana

    American journal of physiology. Renal physiology

    2015  Volume 309, Issue 2, Page(s) F120–36

    Abstract: We previously reported that the deletion of the pH sensor GPR4 causes a non-gap metabolic acidosis and defective net acid excretion (NAE) in the GPR4 knockout mouse (GPR4-/-) (Sun X, Yang LV, Tiegs BC, Arend LJ, McGraw DW, Penn RB, and Petrovic S. J Am ... ...

    Abstract We previously reported that the deletion of the pH sensor GPR4 causes a non-gap metabolic acidosis and defective net acid excretion (NAE) in the GPR4 knockout mouse (GPR4-/-) (Sun X, Yang LV, Tiegs BC, Arend LJ, McGraw DW, Penn RB, and Petrovic S. J Am Soc Nephrol 21: 1745-1755, 2010). Since the major regulatory site of NAE in the kidney is the collecting duct (CD), we examined acid-base transport proteins in intercalated cells (ICs) of the CD and found comparable mRNA expression of kidney anion exchanger 1 (kAE1), pendrin, and the a4 subunit of H(+)-ATPase in GPR4-/- vs. +/+. However, NH4Cl loading elicited adaptive doubling of AE1 mRNA in GPR4+/+, but a 50% less pronounced response in GPR4-/-. In GPR4+/+, NH4Cl loading evoked a cellular response characterized by an increase in AE1-labeled and a decrease in pendrin-labeled ICs similar to what was reported in rabbits and rats. This response did not occur in GPR4-/-. Microperfusion experiments demonstrated that the activity of the basolateral Cl(-)/HCO3(-) exchanger, kAE1, in CDs isolated from GPR4-/- failed to increase with NH4Cl loading, in contrast to the increase observed in GPR4+/+. Therefore, the deficiency of GPR4 blunted, but did not eliminate the adaptive response to an acid load, suggesting a compensatory response from other pH/CO2/bicarbonate sensors. Indeed, the expression of the calcium-sensing receptor (CaSR) was nearly doubled in GPR4-/- kidneys, in the absence of apparent disturbances of Ca(2+) homeostasis. In summary, the expression and activity of the key transport proteins in GPR4-/- mice are consistent with spontaneous metabolic acidosis, but the adaptive response to a superimposed exogenous acid load is blunted and might be partially compensated for by CaSR.
    MeSH term(s) Acid-Base Equilibrium ; Acidosis, Renal Tubular/metabolism ; Adaptation, Physiological ; Animals ; Anion Exchange Protein 1, Erythrocyte/metabolism ; Anion Transport Proteins/metabolism ; Kidney Tubules, Collecting/metabolism ; Mice, Knockout ; Receptors, Calcium-Sensing/metabolism ; Receptors, G-Protein-Coupled/metabolism ; Sodium-Hydrogen Exchanger 3 ; Sodium-Hydrogen Exchangers/metabolism
    Chemical Substances Anion Exchange Protein 1, Erythrocyte ; Anion Transport Proteins ; GPR4 protein, mouse ; Receptors, Calcium-Sensing ; Receptors, G-Protein-Coupled ; Slc26a4 protein, mouse ; Slc4a1 protein, mouse ; Sodium-Hydrogen Exchanger 3 ; Sodium-Hydrogen Exchangers
    Language English
    Publishing date 2015-07-15
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 603837-2
    ISSN 1522-1466 ; 0363-6127
    ISSN (online) 1522-1466
    ISSN 0363-6127
    DOI 10.1152/ajprenal.00507.2014
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  8. Article ; Online: Increased acid load and deletion of AE1 increase Slc26a7 expression.

    Sun, Xuming / Petrovic, Snezana

    Nephron. Physiology

    2008  Volume 109, Issue 3, Page(s) p29–35

    Abstract: Background/aims: Slc26a7 is a member of a family of anion transport proteins, Solute-Linked Carrier 26 (Slc26). Slc26a7, which can mediate Cl-/HCO3- exchange, is expressed in the acid-secreting, A-intercalated cells of the kidney collecting duct. On the ...

    Abstract Background/aims: Slc26a7 is a member of a family of anion transport proteins, Solute-Linked Carrier 26 (Slc26). Slc26a7, which can mediate Cl-/HCO3- exchange, is expressed in the acid-secreting, A-intercalated cells of the kidney collecting duct. On the basolateral side of the A-intercalated cells, Slc26a7 co-localizes with the anion exchanger 1 (AE1), a Cl-/HCO3- exchanger that mediates bicarbonate reabsorption in the collecting duct.
    Methods: To test if Slc26a7 is involved in acid-base regulation, as its localization and function suggest, we examined the effect of acid loading and deletion of AE1 on Slc26a7 expression with quantitative real-time RT-PCR and Western blotting.
    Results: Four days of acid loading increased Slc26a7 mRNA expression in the kidney inner medulla by 57% (n = 6 acid loaded vs. n = 6 control rats; p < 0.001), whereas mRNA expression in the outer medulla and the cortex did not change. Western blotting analysis demonstrated increased Slc26a7 protein expression in both outer (140%) and inner medulla (50%) in acid-loaded animals (n = 3) compared to controls (n = 3; p < 0.05). The expression of Slc26a7 mRNA was increased by 66% in the kidneys of AE1 knockout mice (n = 5) compared to the wild types (n = 5, p < 0.001). The increase in Slc26a7 mRNA correlated with a twofold increase in protein expression (p < 0.05).
    Conclusion: We suggest that the increase in Slc26a7 expression caused by acid challenge and deletion of AE1 represents an adaptive response, indicating that Slc26a7 contributes to the regulation of acid-base balance by the kidney.
    MeSH term(s) Acid-Base Equilibrium ; Acidosis/chemically induced ; Acidosis/metabolism ; Ammonium Chloride ; Animals ; Anion Exchange Protein 1, Erythrocyte/deficiency ; Anion Exchange Protein 1, Erythrocyte/genetics ; Anion Exchange Protein 1, Erythrocyte/metabolism ; Antiporters/genetics ; Antiporters/metabolism ; Blotting, Western ; Chloride-Bicarbonate Antiporters/genetics ; Chloride-Bicarbonate Antiporters/metabolism ; Disease Models, Animal ; Female ; Kidney Medulla/metabolism ; Mice ; Mice, Knockout ; RNA, Messenger/metabolism ; Rats ; Rats, Sprague-Dawley ; Reverse Transcriptase Polymerase Chain Reaction ; Up-Regulation
    Chemical Substances Anion Exchange Protein 1, Erythrocyte ; Antiporters ; Chloride-Bicarbonate Antiporters ; RNA, Messenger ; Slc26a7 protein, mouse ; Slc26a7 protein, rat ; Slc4a1 protein, mouse ; Ammonium Chloride (01Q9PC255D)
    Language English
    Publishing date 2008
    Publishing country Switzerland
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 207121-6
    ISSN 1660-2137 ; 1423-0186 ; 2235-3186 ; 1660-8151 ; 0028-2766
    ISSN (online) 1660-2137 ; 1423-0186 ; 2235-3186
    ISSN 1660-8151 ; 0028-2766
    DOI 10.1159/000145465
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  9. Article ; Online: Pyk2 regulates H+-ATPase-mediated proton secretion in the outer medullary collecting duct via an ERK1/2 signaling pathway.

    Fisher, Kimberly D / Codina, Juan / Petrovic, Snezana / DuBose, Thomas D

    American journal of physiology. Renal physiology

    2012  Volume 303, Issue 9, Page(s) F1353–62

    Abstract: Acid-secreting intercalated cells respond to changes in systemic pH through regulation of apical H(+) transporters. Little is known about the mechanism by which these cells sense changes in extracellular pH (pH(o)). Pyk2 is a nonreceptor tyrosine kinase ... ...

    Abstract Acid-secreting intercalated cells respond to changes in systemic pH through regulation of apical H(+) transporters. Little is known about the mechanism by which these cells sense changes in extracellular pH (pH(o)). Pyk2 is a nonreceptor tyrosine kinase activated by autophosphorylation at Tyr402 by cell-specific stimuli, including decreased pH, and is involved in the regulation of MAPK signaling pathways and transporter activity. We examined whether the Pyk2 and MAPK signaling pathway mediates the response of transport proteins to decreased pH in outer medullary collecting duct cells. Immunoblot analysis of phosphorylated Pyk2 (Tyr402), ERK1/2 (Thr202/Tyr204), and p38 (Thr180/Tyr182) was used to assay protein activation. To examine specificity of kinase activation and its effects, we used Pyk2 small interfering RNA to knockdown Pyk2 expression levels, the Src kinase inhibitor 4-amino-5-(4-methylphenyl)-7-(t-butyl)pyrazolo[3,4-d]-pyrimidine (PP 1) to inhibit Pyk2 phosphorylation, and the MEK inhibitor U0126 to inhibit ERK1/2 phosphorylation. The pH-sensitive fluorescent probe 2'-7'-bis(carboxyethyl)-5(6)-carboxyfluorescein-acetoxymethyl ester (BCECF-AM) was used to assay H(+) transporter activity. The activity of H(+) transporters was measured as the rate of intracellular pH (pH(i)) recovery after an NH(4)Cl prepulse. We show that Pyk2 is endogenously expressed and activated by acid pH in mouse-derived outer medullary collecting duct (mOMCD1) cells. Incubation of mOMCD1 cells in acid media [extracellular pH (pH(o)) 6.7] increased the phosphorylation of Pyk2, ERK1/2, and p38. Reduction in pH(i) induced by an NH(4)Cl prepulse also increased the phosphorylation of Pyk2, ERK1/2, and p38. Consistent with our previous studies, we found that mOMCD1 cells exhibit H(+)-ATPase and H(+),K(+)-ATPase activity. Pyk2 inhibition by Pyk2 siRNA and PP 1 prevented Pyk2 phosphorylation as well as H(+)-ATPase-mediated recovery in mOMCD1 cells. In addition, ERK1/2 inhibition by U0126 prevented acid-induced ERK1/2 phosphorylation and H(+)-ATPase-mediated pH(i) recovery but not phosphorylation of p38. We conclude that Pyk2 and ERK1/2 are required for increasing H(+)-ATPase, but not H(+),K(+)-ATPase, activity at decreased pH(i) in mOMCD1 cells.
    MeSH term(s) Ammonium Chloride/pharmacology ; Animals ; Butadienes/pharmacology ; Cell Line ; Enzyme Inhibitors/pharmacology ; Focal Adhesion Kinase 2/physiology ; Hydrogen-Ion Concentration ; Kidney Medulla/cytology ; Kidney Medulla/drug effects ; Kidney Medulla/metabolism ; Kidney Tubules, Collecting/cytology ; Kidney Tubules, Collecting/drug effects ; Kidney Tubules, Collecting/metabolism ; MAP Kinase Signaling System/drug effects ; MAP Kinase Signaling System/physiology ; Mice ; Mice, Transgenic ; Mitogen-Activated Protein Kinase Kinases/metabolism ; Models, Animal ; Nitriles/pharmacology ; Phosphorylation/drug effects ; Proton-Translocating ATPases/physiology ; Protons ; Pyrazoles/pharmacology ; Pyrimidines/pharmacology ; RNA, Small Interfering/pharmacology
    Chemical Substances 4-amino-5-(4-methylphenyl)-7-(tert-butyl)pyrazolo(3,4-d)pyrimidine ; Butadienes ; Enzyme Inhibitors ; Nitriles ; Protons ; Pyrazoles ; Pyrimidines ; RNA, Small Interfering ; U 0126 ; Ammonium Chloride (01Q9PC255D) ; Focal Adhesion Kinase 2 (EC 2.7.10.2) ; Mitogen-Activated Protein Kinase Kinases (EC 2.7.12.2) ; Proton-Translocating ATPases (EC 3.6.3.14)
    Language English
    Publishing date 2012-07-18
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 603837-2
    ISSN 1522-1466 ; 0363-6127
    ISSN (online) 1522-1466
    ISSN 0363-6127
    DOI 10.1152/ajprenal.00008.2012
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  10. Book: Evrokomunizam i partijski pluralizam

    Petrović, Snežana

    građenje socijalizma bez rušenja kapitalizma

    1997  

    Author's details Snežana Petrović
    Language Serbian
    Size 109 S
    Edition 3. izd
    Publisher Nomos
    Publishing place Beograd
    Document type Book
    Note In kyrill. Schr., serb.
    Database Former special subject collection: coastal and deep sea fishing

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