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Article ; Online: Spatial Human Development Index in China: Measurement and Interpretation Based on Bayesian Estimation.

Luo, Xiang / Qin, Jingjing / Wan, Qing / Jin, Gui

International journal of environmental research and public health

2023 Volume 20, Issue 1

Abstract: The development of urban agglomerations dominated by the service industry is an important driving force for further sustainable economic growth of China. Spatial analysis marked by population density and regional integration is an essential perspective ... ...

Abstract	The development of urban agglomerations dominated by the service industry is an important driving force for further sustainable economic growth of China. Spatial analysis marked by population density and regional integration is an essential perspective for studying the human development index (HDI) in China. Based on Bayesian estimation, this paper examines the influence of a spatial factor on HDI by using a spatial hierarchical factor model within the framework of Sen Capability Approach theory, overcoming the neglect of spatial factors and their equal weight in traditional measurement of HDI. On this basis, the HDI including the spatial factor was measured based on the panel data from 2000 to 2018. The results reveal that (1) provinces with high population densities and regional integration have higher rankings and low uncertainties of HDI, which can be attributed to the improvement of education weights; (2) HDI has a certain spatial spillover effect, and the spatial association increases year by year; (3) robust test by using nighttime lighting as an alternative indicator of GDP supports that the spatial correlation is positively related to HDI ranking. The policy recommendations of this paper are to remove the obstacles for cross-regional population mobility and adjust the direction and structure of public expenditure.
MeSH term(s)	Humans ; Bayes Theorem ; China ; Industry ; Population Density ; Spatial Analysis ; Economic Development
Language	English
Publishing date	2023-01-01
Publishing country	Switzerland
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2175195-X
ISSN	1660-4601 ; 1661-7827
ISSN (online)	1660-4601
ISSN	1661-7827
DOI	10.3390/ijerph20010818
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Article: The Influence of Reading Texts on L2 Reading-to-Write Argumentative Writing.

Qin, Jingjing / Liu, Yingliang

Frontiers in psychology

2021 Volume 12, Page(s) 655601

Abstract: Reading-to-write is an essential skill in academic writing, and reading-writing tasks have been widely adopted in standardized English tests. Much more recent literature on integrated reading-writing tasks has focused on writers' use of source texts or ... ...

Abstract	Reading-to-write is an essential skill in academic writing, and reading-writing tasks have been widely adopted in standardized English tests. Much more recent literature on integrated reading-writing tasks has focused on writers' use of source texts or the validity of integrated writing assessment, while little is known about whether the nature of the types of reading texts has any bearing on integrated reading-writing tasks. This study examines whether the types of reading texts (i.e., similar views or opposing views on a debatable issue) have any influence on second language (L2) students' argumentative writing in terms of the use of argument elements and its overall quality. Fifty-four Chinese second-year university students majoring in English language teaching were asked to write an argumentative essay after reading texts with either similar views or opposing views. Results show that students reading texts with opposing viewpoints presented more data and higher overall quality of argumentative essays than students reading texts with similar viewpoints, although the latter group presented more counterargument data. Pedagogical implications on teaching argumentative writing are discussed.
Language	English
Publishing date	2021-03-19
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2563826-9
ISSN	1664-1078
ISSN	1664-1078
DOI	10.3389/fpsyg.2021.655601
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Article ; Online: Dietary experiences after bariatric surgery in patients with obesity: A qualitative systematic review.

Li, Zhiwen / Pan, Yingli / Zhang, Yingchun / Qin, Jingjing / Lei, Xuejiao

Obesity surgery

2022 Volume 32, Issue 6, Page(s) 2023–2034

Abstract: This systematic review evaluated the dietary experience of patients with obesity post-bariatric surgery. Scopus, CINAHL, Medline, Psych INFO, and Embase databases were searched and JBI Critical Appraisal Tool was used for quality assessment. Thomas and ... ...

Abstract	This systematic review evaluated the dietary experience of patients with obesity post-bariatric surgery. Scopus, CINAHL, Medline, Psych INFO, and Embase databases were searched and JBI Critical Appraisal Tool was used for quality assessment. Thomas and Harden's three-stage thematic synthesis was undertaken using the Enhancing transparency in reporting the synthesis of qualitative research (ENTREQ) statement for reporting. Of the 24 studies extracted, we coded and developed 34 descriptive themes into 7 categories, which were then categorized to 3 analytical themes. The number of all the participants in the 24 articles is 383 people. The results revealed most patients can control their diet for a short period post-surgery. However, this was a matter of gradual self-consciousness as patients also required support and dietary management in postoperative recovery. CLINICAL TRIAL REGISTRATION NUMBER: The protocol for this qualitative systematic review has been registered with PROSPERO (registration number CRD42021229083).
MeSH term(s)	Bariatric Surgery ; Diet ; Humans ; Obesity, Morbid/surgery ; Qualitative Research
Language	English
Publishing date	2022-03-31
Publishing country	United States
Document type	Journal Article ; Review ; Systematic Review ; Research Support, Non-U.S. Gov't
ZDB-ID	1070827-3
ISSN	1708-0428 ; 0960-8923
ISSN (online)	1708-0428
ISSN	0960-8923
DOI	10.1007/s11695-022-06018-8
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Article ; Online: Activation of 5-Hydroxytryptamine 4 Receptor Improves Colonic Barrier Function by Triggering Mucin 2 Production in a Mouse Model of Type 1 Diabetes.

Han, Changhao / Geng, Qinghua / Qin, Jingjing / Li, Yulin / Yu, Huarong

The American journal of pathology

2022 Volume 192, Issue 6, Page(s) 876–886

Abstract: Diabetes leads to intestinal barrier dysfunction. 5-Hydroxytryptamine 4 receptor (5- ... ...

Abstract	Diabetes leads to intestinal barrier dysfunction. 5-Hydroxytryptamine 4 receptor (5-HT
MeSH term(s)	Animals ; Colon/metabolism ; Diabetes Mellitus, Experimental/metabolism ; Diabetes Mellitus, Type 1/metabolism ; Disease Models, Animal ; Intestinal Mucosa/metabolism ; Mice ; Mucin-2/metabolism ; Mucins/metabolism ; RNA, Ribosomal, 16S/metabolism ; Receptors, Serotonin, 5-HT4/metabolism ; Serotonin/metabolism ; Serotonin/pharmacology
Chemical Substances	Mucin-2 ; Mucins ; RNA, Ribosomal, 16S ; Receptors, Serotonin, 5-HT4 (158165-40-3) ; Serotonin (333DO1RDJY)
Language	English
Publishing date	2022-03-23
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2943-9
ISSN	1525-2191 ; 0002-9440
ISSN (online)	1525-2191
ISSN	0002-9440
DOI	10.1016/j.ajpath.2022.03.002
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Article ; Online: The crosslinks between ferroptosis and autophagy in asthma.

Lv, Xiaodi / Tang, Weifeng / Qin, Jingjing / Wang, Wenqian / Dong, Jingcheng / Wei, Ying

Frontiers in immunology

2023 Volume 14, Page(s) 1140791

Abstract: Autophagy is an evolutionarily conserved cellular process capable of degrading various biological molecules and ... ...

Abstract	Autophagy is an evolutionarily conserved cellular process capable of degrading various biological molecules and organelles
MeSH term(s)	Humans ; Ferroptosis ; Iron/metabolism ; Autophagy ; Asthma ; Inflammation
Chemical Substances	Iron (E1UOL152H7)
Language	English
Publishing date	2023-03-29
Publishing country	Switzerland
Document type	Journal Article ; Review ; Research Support, Non-U.S. Gov't
ZDB-ID	2606827-8
ISSN	1664-3224 ; 1664-3224
ISSN (online)	1664-3224
ISSN	1664-3224
DOI	10.3389/fimmu.2023.1140791
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Article: Intestinal organoid modeling: bridging the gap from experimental model to clinical translation.

Liu, Taotao / Li, Xiaoqi / Li, Hao / Qin, Jingjing / Xu, Hui / Wen, Jun / He, Yaqin / Zhang, Cao

Frontiers in oncology

2024 Volume 14, Page(s) 1334631

Abstract: The 3D culture of intestinal organoids entails embedding isolated intestinal crypts and bone marrow mesenchymal stem cells within a growth factor-enriched matrix gel. This process leads to the formation of hollow microspheres with structures resembling ... ...

Abstract	The 3D culture of intestinal organoids entails embedding isolated intestinal crypts and bone marrow mesenchymal stem cells within a growth factor-enriched matrix gel. This process leads to the formation of hollow microspheres with structures resembling intestinal epithelial cells, which are referred to as intestinal organoids. These structures encompass various functional epithelial cell types found in the small intestine and closely mimic the organizational patterns of the small intestine, earning them the name "mini-intestines". Intestinal tumors are prevalent within the digestive system and represent a significant menace to human health. Through the application of 3D culture technology, miniature colorectal organs can be cultivated to retain the genetic characteristics of the primary tumor. This innovation offers novel prospects for individualized treatments among patients with intestinal tumors. Presently established libraries of patient-derived organoids serve as potent tools for conducting comprehensive investigations into tissue functionality, developmental processes, tumorigenesis, and the pathobiology of cancer. This review explores the origins of intestinal organoids, their culturing environments, and their advancements in the realm of precision medicine. It also addresses the current challenges and outlines future prospects for development.
Language	English
Publishing date	2024-03-01
Publishing country	Switzerland
Document type	Journal Article ; Review
ZDB-ID	2649216-7
ISSN	2234-943X
ISSN	2234-943X
DOI	10.3389/fonc.2024.1334631
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Article ; Online: Suggestions and thoughts on the construction of “One Syndrome and One Product” nursing brand

CHU Yina / CAO Yanxia / GAO Xinxia / CUI Xuejiao / XIAO Xiong / CHEN Ying / QIN Jingjing

中西医结合护理, Vol 7, Iss 7, Pp 65-

2021 Volume 68

Abstract: This paper expounds the suggestions and thoughts on the construction of “One Syndrome and One Product” Chinese medicine nursing brand from four aspects: the hierarchical management mode of nursing system, the training of multi-disciplinary nursing ... ...

Abstract	This paper expounds the suggestions and thoughts on the construction of “One Syndrome and One Product” Chinese medicine nursing brand from four aspects: the hierarchical management mode of nursing system, the training of multi-disciplinary nursing professionals, the integration of Traditional Chinese Medicine nursing and rehabilitation, and the humanistic care of Traditional Chinese Medicine nursing, so as to optimize the nursing scheme of traditional Chinese medicine and create the nursing service brand of Traditional Chinese Medicine with different characteristics, And make nurses have evidence to rely on in nursing work, improve the patient's treatment experience.
Keywords	traditional chinese medicine nursing ; nursing based on syndrome differentiation ; continuing nursing ; nursing brand ; Nursing ; RT1-120
Language	Chinese
Publishing date	2021-07-01T00:00:00Z
Publisher	Association of Integrative Nursing
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article ; Online: The crosstalk between ferroptosis and autophagy in cancer.

Lv, Xiaodi / Wang, Bin / Dong, Ming / Wang, Wenqian / Tang, Weifeng / Qin, Jingjing / Gao, Yanglai / Wei, Ying

Autoimmunity

2023 Volume 56, Issue 1, Page(s) 2289362

Abstract: Background: In order to better understand the interplay between ferroptosis and autophagy, enhance the interpretation of the crosstalk between these two forms of regulated cell death, develop the effective pharmacological mechanisms for cancer treatment, ...

Abstract	Background: In order to better understand the interplay between ferroptosis and autophagy, enhance the interpretation of the crosstalk between these two forms of regulated cell death, develop the effective pharmacological mechanisms for cancer treatment, discover novel biomarkers for better diagnostic, and envisage the future hotspots of the research on ferroptosis and autophagy, we harnessed bibliometric tools to study the articles published from 2012 to 2022 on the relationship between ferroptosis and autophagy. Methods: Web of Science Core Collection (WOSCC) database was used to conduct a comprehensive search and analysis of articles in this field from January 1, 2012, to September 1, 2022. The Citespace 6.1.R2 software and VOS viewer 6.1.8 software were utilized to analyze the overall structure of the network, network clusters, links between clusters, key nodes or pivot points, and pathways. Results: A total of 756 articles associated with the crosstalk between ferroptosis and autophagy were published in 512 journals by 4183 authors in 980 organizations from 55 countries or regions. The distribution of countries and organizations was demonstrated using CiteSpace and VOS viewer. The top three countries with the most articles were China ( Conclusion: Our results provide insights into the development of recognition related to the crosstalk between ferroptosis and autophagy, and the current molecular crosslinked mechanisms in the context of common signal transduction pathways or affecting cellular environment to induce the adaptive stress response and to activate the particular form of regulated cell death (RCD), and the development of cancer treatment based on novel targets and signaling regulatory networks provided by ferroptosis and autophagy.
MeSH term(s)	Humans ; Ferroptosis ; Autophagy ; Cross Reactions ; Neoplasms ; Asthma
Language	English
Publishing date	2023-12-08
Publishing country	England
Document type	Journal Article ; Review ; Research Support, Non-U.S. Gov't
ZDB-ID	1025450-x
ISSN	1607-842X ; 0891-6934
ISSN (online)	1607-842X
ISSN	0891-6934
DOI	10.1080/08916934.2023.2289362
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Zs.A 2777: Show issues

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Article ; Online: TRAIL and Celastrol Combinational Treatment Suppresses Proliferation, Migration, and Invasion of Human Glioblastoma Cells via Targeting Wnt/β-catenin Signaling Pathway.

Qin, Jing-Jing / Niu, Meng-da / Cha, Zhe / Geng, Qing-Hua / Li, Yu-Lin / Ren, Chun-Guang / Molloy, David P / Yu, Hua-Rong

Chinese journal of integrative medicine

2023 Volume 30, Issue 4, Page(s) 322–329

Abstract: Objective: To investigate the mechanistic basis for the anti-proliferation and anti-invasion effect of tumor necrosis factor-related apoptosis-induced ligand (TRAIL) and celastrol combination treatment (TCCT) in glioblastoma cells.: Methods: Cell ... ...

Abstract	Objective: To investigate the mechanistic basis for the anti-proliferation and anti-invasion effect of tumor necrosis factor-related apoptosis-induced ligand (TRAIL) and celastrol combination treatment (TCCT) in glioblastoma cells. Methods: Cell counting kit-8 was used to detect the effects of different concentrations of celastrol (0-16 µmol/L) and TRAIL (0-500 ng/mL) on the cell viability of glioblastoma cells. U87 cells were randomly divided into 4 groups, namely control, TRAIL (TRAIL 100 ng/mL), Cel (celastrol 0.5 µmol/L) and TCCT (TRAIL 100 ng/mL+ celastrol 0.5 µmol/L). Cell proliferation, migration, and invasion were detected by colony formation, wound healing, and Transwell assays, respectively. Quantitative reverse transcription polymerase chain reaction and Western blotting were performed to assess the levels of epithelial-mesenchymal transition (EMT) markers (zona occludens, N-cadherin, vimentin, zinc finger E-box-binding homeobox, Slug, and β-catenin). Wnt pathway was activated by lithium chloride (LiCl, 20 mol/L) and the mechanism for action of TCCT was explored. Results: Celastrol and TRAIL synergistically inhibited the proliferation, migration, invasion, and EMT of U87 cells (P<0.01). TCCT up-regulated the expression of GSK-3β and down-regulated the expression of β-catenin and its associated proteins (P<0.05 or P<0.01), including c-Myc, Cyclin-D1, and matrix metalloproteinase (MMP)-2. In addition, LiCl, an activator of the Wnt signaling pathway, restored the inhibitory effects of TCCT on the expression of β-catenin and its downstream genes, as well as the migration and invasion of glioblastoma cells (P<0.05 or P<0.01). Conclusions: Celastrol and TRAIL can synergistically suppress glioblastoma cell migration, invasion, and EMT, potentially through inhibition of Wnt/β-catenin pathway. This underlies a novel mechanism of action for TCCT as an effective therapy for glioblastoma.
MeSH term(s)	Humans ; Wnt Signaling Pathway ; Glioblastoma/drug therapy ; Glioblastoma/pathology ; beta Catenin/metabolism ; Glycogen Synthase Kinase 3 beta/metabolism ; Ligands ; Cell Line, Tumor ; Apoptosis ; Tumor Necrosis Factors/pharmacology ; Cell Proliferation ; Cell Movement ; Epithelial-Mesenchymal Transition ; Pentacyclic Triterpenes
Chemical Substances	beta Catenin ; celastrol (L8GG98663L) ; Glycogen Synthase Kinase 3 beta (EC 2.7.11.1) ; Ligands ; Tumor Necrosis Factors ; Pentacyclic Triterpenes
Language	English
Publishing date	2023-10-20
Publishing country	China
Document type	Journal Article
ZDB-ID	2171254-2
ISSN	1993-0402 ; 1672-0415
ISSN (online)	1993-0402
ISSN	1672-0415
DOI	10.1007/s11655-023-3752-7
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Zs.A 5823: Show issues

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Article ; Online: Complement C3a promotes proliferation, migration and stemness in cutaneous squamous cell carcinoma.

Fan, Zhuo / Qin, Jingjing / Wang, Dandan / Geng, Songmei

Journal of cellular and molecular medicine

2019 Volume 23, Issue 5, Page(s) 3097–3107

Abstract: Background: Complement C3 has been shown to be highly expressed in cutaneous squamous cell carcinoma (cSCC) tumour tissues and is correlated with tumour cell growth. This study aimed to investigate the mechanism of C3 in cSCC malignant transformation.!## ...

Abstract	Background: Complement C3 has been shown to be highly expressed in cutaneous squamous cell carcinoma (cSCC) tumour tissues and is correlated with tumour cell growth. This study aimed to investigate the mechanism of C3 in cSCC malignant transformation. Methods: C3 expression was analysed in cSCC cell lines A431, Tca8113, SCC13, HSC-5 and HSC-1 and in immortalized HaCaT keratinocytes. Proliferation and migration of cSCC were determined after C3a exposure. Expression of cyclin D1, cyclin E, vascular endothelial growth factor (VEGF), pro-matrix metalloproteinase 1 (pro-MMP1), pro-matrix metalloproteinase 2 (pro-MMP2), stemness factors, GSK-3β, and β-catenin were analyzed. Tumour growth was examined in a murine xenograft model. Results: C3 expression was much more highly expressed in all cSCC cell lines than in HaCaT cells. C3a treatment significantly promoted cSCC cell proliferation and migration and upregulated cyclin D1, cyclin E, VEGF, pro-MMP1 and pro-MMP2 expression, which were impeded by the C3aR antagonist. Moreover, the expression of stemness factors Sox-2, Nanog, Oct-4, c-Myc and CD-44 was stimulated by C3a and slowed by C3aR disruption. Knockdown of Sox-2 by siRNA transfection suppressed cell proliferation and migration, constrained VEGF secretion and inhibited pro-MMP1 and pro-MMP2 expression. C3a also activated the Wnt and β-catenin pathway in cSCC cells. Disruption of C3aR expression dampened tumour growth and the expression of Wnt-1, β-catenin and Sox-2 in the xenograft model. Conclusions: C3a enhanced cell proliferation, migration and stemness in cSCC, and this activity was correlated with activation of the Wnt and β-catenin pathway.
MeSH term(s)	Animals ; Carcinoma, Squamous Cell/genetics ; Carcinoma, Squamous Cell/pathology ; Cell Line, Tumor ; Cell Movement/genetics ; Cell Proliferation/genetics ; Complement C3a/genetics ; Gene Expression Regulation, Neoplastic/genetics ; Heterografts ; Humans ; Mice ; Neoplasm Proteins/genetics ; Neoplastic Stem Cells/metabolism ; Neoplastic Stem Cells/pathology ; Signal Transduction ; Skin Neoplasms/genetics ; Skin Neoplasms/pathology ; Wnt1 Protein/genetics ; beta Catenin/genetics
Chemical Substances	CTNNB1 protein, human ; Neoplasm Proteins ; Wnt1 Protein ; beta Catenin ; Complement C3a (80295-42-7)
Language	English
Publishing date	2019-03-01
Publishing country	England
Document type	Journal Article
ZDB-ID	2074559-X
ISSN	1582-4934 ; 1582-4934 ; 1582-1838
ISSN (online)	1582-4934
ISSN	1582-4934 ; 1582-1838
DOI	10.1111/jcmm.13959
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