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  1. Article ; Online: The ethics of peer review process.

    Remuzzi, Giuseppe

    Updates in surgery

    2023  Volume 75, Issue 6, Page(s) 1391–1392

    MeSH term(s) Humans ; Peer Review/ethics
    Language English
    Publishing date 2023-07-25
    Publishing country Italy
    Document type Editorial
    ZDB-ID 2572692-4
    ISSN 2038-3312 ; 2038-131X
    ISSN (online) 2038-3312
    ISSN 2038-131X
    DOI 10.1007/s13304-023-01602-8
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  2. Article ; Online: Stewart as I Knew Him.

    Remuzzi, Giuseppe

    Nephron

    2023  Volume 148, Issue 2, Page(s) 124–126

    Language English
    Publishing date 2023-10-26
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 207121-6
    ISSN 2235-3186 ; 1423-0186 ; 1660-8151 ; 0028-2766
    ISSN (online) 2235-3186 ; 1423-0186
    ISSN 1660-8151 ; 0028-2766
    DOI 10.1159/000534841
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  3. Book ; Online: Humoral immunity in kidney transplantation

    Remuzzi, Giuseppe

    what clinicians need to know ; an overview of recent developments

    2009  

    Author's details vol. eds.: Giuseppe Remuzzi
    Language English
    Size X + 146 S.
    Publisher Karger
    Publishing place Basel
    Publishing country Switzerland
    Document type Book ; Online
    HBZ-ID TT050388182
    ISBN 978-3-8055-8959-8 ; 3-8055-8959-X
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  4. Book ; Online ; E-Book: Nephrology and public health worldwide

    Silva Junior, Geraldo B. / Ferreiro Fuentes, Alejandro / Nangaku, Masaomi / Remuzzi, Giuseppe / Ronco, Claudio

    (Contributions to nephrology ; 199)

    2021  

    Author's details Editors: Silva Junior, Geraldo B. (Fortaleza) Ferreiro Fuentes, Alejandro (Montevideo) Nangaku, Masaomi (Tokyo ) Remuzzi, Giuseppe (Bergamo) Ronco, Claudio (Vicenza)
    Series title Contributions to nephrology ; 199
    Collection
    Keywords Nephrology
    Language English
    Size 1 Online-Ressource (viii, 359 Seiten), Illustrationen
    Edition 1. Auflage
    Publisher S. Karger
    Publishing place Basel
    Publishing country Switzerland
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    HBZ-ID HT021374733
    ISBN 978-3-318-06937-2 ; 9783318069365 ; 3-318-06937-X ; 3318069361
    DOI 10.1159/isbn.978-3-318-06937-2
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  5. Article ; Online: COVID-19 from a pharmacological perspective.

    Remuzzi, Giuseppe

    Advances in biological regulation

    2021  Volume 81, Page(s) 100821

    MeSH term(s) Antiviral Agents/therapeutic use ; COVID-19/epidemiology ; COVID-19/metabolism ; Humans ; SARS-CoV-2/metabolism ; COVID-19 Drug Treatment
    Chemical Substances Antiviral Agents
    Language English
    Publishing date 2021-08-16
    Publishing country England
    Document type Editorial
    ZDB-ID 2667413-0
    ISSN 2212-4934 ; 2212-4926
    ISSN (online) 2212-4934
    ISSN 2212-4926
    DOI 10.1016/j.jbior.2021.100821
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  6. Book: Humoral immunity in kidney transplantation

    Remuzzi, Giuseppe

    what clinicians need to know ; 12 tables

    (Contributions to nephrology ; 162)

    2009  

    Author's details vol. ed. Giuseppe Remuzzi
    Series title Contributions to nephrology ; 162
    Collection
    Keywords Kidney Transplantation / immunology ; Antibody Formation / immunology ; Histocompatibility Antigens / immunology ; Histocompatibility / immunology ; Nierentransplantation ; Immunologie
    Subject Klinische Immunologie ; Niere
    Language English
    Size IX, 145 S. : Ill., graph. Darst., 25 cm, 450 gr.
    Publisher Karger
    Publishing place Basel u.a.
    Publishing country Switzerland ; Germany
    Document type Book
    Note Literaturangaben
    HBZ-ID HT015744554
    ISBN 978-3-8055-8945-1 ; 3-8055-8945-X
    Database Catalogue ZB MED Medicine, Health

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    Shelf markLoan statusLocation
    2008 A 6035 order for loan Magazin

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  7. Article ; Online: C3G and Ig-MPGN-treatment standard.

    Noris, Marina / Remuzzi, Giuseppe

    Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association

    2023  Volume 39, Issue 2, Page(s) 202–214

    Abstract: Among the broad spectrum of membranoproliferative glomerulonephritis (MPGN), immunofluorescence distinguishes C3 glomerulopathy (C3G), with predominant C3 deposits, and immunoglobulin-associated MPGN (Ig-MPGN), with combined C3 and Ig. However, there are ...

    Abstract Among the broad spectrum of membranoproliferative glomerulonephritis (MPGN), immunofluorescence distinguishes C3 glomerulopathy (C3G), with predominant C3 deposits, and immunoglobulin-associated MPGN (Ig-MPGN), with combined C3 and Ig. However, there are several intersections between C3G and Ig-MPGN. Primary C3G and Ig-MPGN share the same prevalence of low serum C3 levels and of abnormalities of the alternative pathway of complement, and patients who present a bioptic pattern of Ig-MPGN at onset may show a C3G pattern in a subsequent biopsy. There is no specific therapy for primary C3G and Ig-MPGN and prognosis is unfavourable. The only recommended indications are inhibitors of the renin-angiotensin system, lipid-lowering agents and other renoprotective agents. The other drugs used currently, such as corticosteroids and mycophenolate mofetil, are often ineffective. The anti-C5 monoclonal antibody eculizumab has been tested in several patients, with mixed results. One reason for the uncertainty is the extremely variable clinical course, most likely reflecting a heterogeneous pathogenesis. An unsupervised clustering analysis that included histologic, biochemical, genetic and clinical data available at onset in patients with primary C3G and Ig-MPGN identified four clusters characterized by specific pathogenic mechanisms. This approach may facilitate accurate diagnosis and development of targeted therapies. Several trials are ongoing with drugs targeting different molecules of the complement cascade, however it is important to consider which component of the cascade may be the most appropriate for each patient. We review the current standards of treatment and discuss novel developments in the pathophysiology, diagnosis, outcome prediction and management of C3G and Ig-MPGN.
    MeSH term(s) Humans ; Glomerulonephritis, Membranoproliferative ; Complement C3 ; Immunoglobulins ; Complement Activation ; Fluorescent Antibody Technique
    Chemical Substances Complement C3 ; Immunoglobulins
    Language English
    Publishing date 2023-08-21
    Publishing country England
    Document type Review ; Journal Article
    ZDB-ID 90594-x
    ISSN 1460-2385 ; 0931-0509
    ISSN (online) 1460-2385
    ISSN 0931-0509
    DOI 10.1093/ndt/gfad182
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  8. Article ; Online: Shiga Toxin-Producing Escherichia coli and the Hemolytic-Uremic Syndrome.

    Noris, Marina / Remuzzi, Giuseppe

    The New England journal of medicine

    2023  Volume 389, Issue 26, Page(s) 2499

    MeSH term(s) Humans ; Shiga-Toxigenic Escherichia coli ; Hemolytic-Uremic Syndrome
    Language English
    Publishing date 2023-12-29
    Publishing country United States
    Document type Letter ; Comment
    ZDB-ID 207154-x
    ISSN 1533-4406 ; 0028-4793
    ISSN (online) 1533-4406
    ISSN 0028-4793
    DOI 10.1056/NEJMc2312844
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  9. Article ; Online: Tissue-Resident Macrophages in Solid Organ Transplantation: Harmful or Protective?

    Aiello, Sistiana / Benigni, Ariela / Remuzzi, Giuseppe

    Journal of immunology (Baltimore, Md. : 1950)

    2024  Volume 212, Issue 7, Page(s) 1051–1061

    Abstract: Transplanted organs carry donor immune cells into the recipient, the majority of which are tissue-resident macrophages (TRMs). The role they play in guiding the fate of the transplanted organ toward acceptance or rejection remains elusive. TRMs originate ...

    Abstract Transplanted organs carry donor immune cells into the recipient, the majority of which are tissue-resident macrophages (TRMs). The role they play in guiding the fate of the transplanted organ toward acceptance or rejection remains elusive. TRMs originate from both embryonic and bone marrow-derived precursors. Embryo-derived TRMs retain the embryonic capability to proliferate, so they are able to self-renew and, theoretically, persist for extended periods of time after transplantation. Bone marrow-derived TRMs do not proliferate and must constantly be replenished by adult circulating monocytes. Recent studies have aimed to clarify the different roles and interactions between donor TRMs, recipient monocytes, and monocyte-derived macrophages (MFs) after organ transplantation. This review aims to shed light on how MFs affect the fate of a transplanted organ by differentiating between the role of donor TRMs and that of MFs derived from graft infiltrating monocytes.
    MeSH term(s) Macrophages ; Monocytes ; Organ Transplantation ; Bone Marrow ; Embryo, Mammalian
    Language English
    Publishing date 2024-03-18
    Publishing country United States
    Document type Review ; Journal Article
    ZDB-ID 3056-9
    ISSN 1550-6606 ; 0022-1767 ; 1048-3233 ; 1047-7381
    ISSN (online) 1550-6606
    ISSN 0022-1767 ; 1048-3233 ; 1047-7381
    DOI 10.4049/jimmunol.2300625
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  10. Article ; Online: Sirtuins in kidney health and disease.

    Perico, Luca / Remuzzi, Giuseppe / Benigni, Ariela

    Nature reviews. Nephrology

    2024  Volume 20, Issue 5, Page(s) 313–329

    Abstract: Sirtuins (SIRTs) are putative regulators of lifespan in model organisms. Since the initial discovery that SIRTs could promote longevity in nematodes and flies, the identification of additional properties of these proteins has led to understanding of ... ...

    Abstract Sirtuins (SIRTs) are putative regulators of lifespan in model organisms. Since the initial discovery that SIRTs could promote longevity in nematodes and flies, the identification of additional properties of these proteins has led to understanding of their roles as exquisite sensors that link metabolic activity to oxidative states. SIRTs have major roles in biological processes that are important in kidney development and physiological functions, including mitochondrial metabolism, oxidative stress, autophagy, DNA repair and inflammation. Furthermore, altered SIRT activity has been implicated in the pathophysiology and progression of acute and chronic kidney diseases, including acute kidney injury, diabetic kidney disease, chronic kidney disease, polycystic kidney disease, autoimmune diseases and renal ageing. The renoprotective roles of SIRTs in these diseases make them attractive therapeutic targets. A number of SIRT-activating compounds have shown beneficial effects in kidney disease models; however, further research is needed to identify novel SIRT-targeting strategies with the potential to treat and/or prevent the progression of kidney diseases and increase the average human healthspan.
    MeSH term(s) Sirtuins/metabolism ; Sirtuins/physiology ; Humans ; Kidney Diseases/metabolism ; Animals ; Kidney/metabolism ; Oxidative Stress ; Renal Insufficiency, Chronic/metabolism ; Mitochondria/metabolism ; Aging/physiology ; Aging/metabolism ; Autophagy/physiology
    Chemical Substances Sirtuins (EC 3.5.1.-)
    Language English
    Publishing date 2024-02-06
    Publishing country England
    Document type Journal Article ; Review ; Research Support, Non-U.S. Gov't ; Research Support, N.I.H., Extramural
    ZDB-ID 2490366-8
    ISSN 1759-507X ; 1759-5061
    ISSN (online) 1759-507X
    ISSN 1759-5061
    DOI 10.1038/s41581-024-00806-4
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