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  1. Article ; Online: Plasma cell-directed therapy for rituximab-refractory PLA2R+ membranous nephropathy.

    Salhi, Sofiane / Ribes, David / Fortenfant, Françoise / Faguer, Stanislas

    Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association

    2023  Volume 38, Issue 12, Page(s) 2851–2853

    MeSH term(s) Humans ; Rituximab/therapeutic use ; Glomerulonephritis, Membranous/drug therapy ; Plasma Cells ; Autoantibodies ; Receptors, Phospholipase A2
    Chemical Substances Rituximab (4F4X42SYQ6) ; Autoantibodies ; Receptors, Phospholipase A2
    Language English
    Publishing date 2023-07-03
    Publishing country England
    Document type Journal Article
    ZDB-ID 90594-x
    ISSN 1460-2385 ; 0931-0509
    ISSN (online) 1460-2385
    ISSN 0931-0509
    DOI 10.1093/ndt/gfad135
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 2198: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
    bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
    Jg. 1995 - 2021: Lesesall (1.OG)
    ab Jg. 2022: Lesesaal (EG)
    Zs.MO 329: Show issues

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  2. Article: Néphrotoxicité des anti-infectieux en médecine-intensive réanimation : où en sommes-nous? Nephrotoxicity of anti-infective drugs in intensive care unit: an update

    Salhi, Sofiane / Saint-Jacques, Camille / Rafat, Cédric

    Médecine intensive réanimation

    2023  Volume 32, Issue 2, Page(s) 209

    Language French
    Document type Article
    ZDB-ID 2870987-1
    ISSN 2496-6142
    Database Current Contents Medicine

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 4839: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
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    Jg. 1995 - 2021: Lesesall (2.OG)
    ab Jg. 2022: Lesesaal (EG)

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  3. Article ; Online: Utility of Routine Post Kidney Transplant Anti-HLA Antibody Screening.

    Salhi, Sofiane / Congy-Jolivet, Nicolas / Hebral, Anne-Laure / Esposito, Laure / Vieu, Guillaume / Milhès, Jean / Kamar, Nassim / Del Bello, Arnaud

    Kidney international reports

    2024  Volume 9, Issue 5, Page(s) 1343–1353

    Abstract: Introduction: De novo: Methods: To address this question, we retrospectively assessed the incidence of dnDSA according to the test justification (clinically indicated or systematic) in a cohort of low-immunological risk patients, defined by being ... ...

    Abstract Introduction: De novo
    Methods: To address this question, we retrospectively assessed the incidence of dnDSA according to the test justification (clinically indicated or systematic) in a cohort of low-immunological risk patients, defined by being nonhuman leukocyte antigen (non-HLA)-sensitized and having no previous kidney transplants.
    Results: A total of 1072 patients, for whom 4611 anti-HLA tests were performed, were included in the study. During the follow-up period of 8 (interquartile range, IQR: 5-11) years, 77 recipients developed dnDSA (prevalence of 7.2%). Thirty-five of these dnDSAs (45.5%) were detected during the first year posttransplantation. In 95% of patients with dnDSA, an immunizing event was identified in their medical records. dnDSA was detected in 46 of 4267 systematic screening tests (1.08%) performed. Active and chronic AMR were frequently observed in biopsies performed after systematic DSA testing (17.9% and 15.4%, respectively).
    Conclusion: Our results suggest that the detection by systematic screening of dnDSA in low-immunological risk kidney transplant patients without sensitizing events is a rare event, especially after 1 year. Moreover, in real life, systematic annual screening for dnDSA, seems having a limited impact to detect AMR at an earlier stage compared to patients in whom dnDSA was detected after a clinically indicated test.
    Language English
    Publishing date 2024-02-18
    Publishing country United States
    Document type Journal Article
    ISSN 2468-0249
    ISSN (online) 2468-0249
    DOI 10.1016/j.ekir.2024.02.1394
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article: Complement C5 inhibition reverses bleomycin-induced thrombotic microangiopathy.

    Salhi, Sofiane / Ribes, David / Faguer, Stanislas

    Clinical kidney journal

    2020  Volume 14, Issue 4, Page(s) 1275–1276

    Abstract: Whether C5 blocking may improve the outcomes of patients developing chemotherapy-induced thrombotic microangiopathy (TMA) remains elusive. Lung fibrosis is a well-known complication of bleomycin, whereas TMAs are very rare (<20 cases described). Here, we ...

    Abstract Whether C5 blocking may improve the outcomes of patients developing chemotherapy-induced thrombotic microangiopathy (TMA) remains elusive. Lung fibrosis is a well-known complication of bleomycin, whereas TMAs are very rare (<20 cases described). Here, we report an exceptional case of a male patient that developed acute respiratory distress syndrome and TMA following administration of bleomycin, cisplatin and etoposide . Refractoriness to plasma exchanges prompted us to use eculizumab as salvage therapy. Eculizumab led to complete remission of the TMA before Day 2. However, the patient progressed towards refractory respiratory failure, suggesting that pathophysiological mechanisms of bleomycin-induced lung fibrosis and TMA differ.
    Language English
    Publishing date 2020-07-09
    Publishing country England
    Document type Case Reports
    ZDB-ID 2655800-2
    ISSN 2048-8513 ; 2048-8505
    ISSN (online) 2048-8513
    ISSN 2048-8505
    DOI 10.1093/ckj/sfaa101
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 6587: Show issues Location:
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    ab Jg. 2022: Lesesaal (EG)

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  5. Article ; Online: Bortezomib plus dexamethasone for rituximab-resistant PLA2R

    Salhi, Sofiane / Ribes, David / Colombat, Magali / Fortenfant, Françoise / Faguer, Stanislas

    Kidney international

    2021  Volume 100, Issue 3, Page(s) 708–709

    MeSH term(s) Bortezomib ; Dexamethasone ; Glomerulonephritis, Membranous/drug therapy ; Humans ; Rituximab
    Chemical Substances Rituximab (4F4X42SYQ6) ; Bortezomib (69G8BD63PP) ; Dexamethasone (7S5I7G3JQL)
    Language English
    Publishing date 2021-07-23
    Publishing country United States
    Document type Letter ; Comment
    ZDB-ID 120573-0
    ISSN 1523-1755 ; 0085-2538
    ISSN (online) 1523-1755
    ISSN 0085-2538
    DOI 10.1016/j.kint.2021.04.011
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 797: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
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    Jg. 1995 - 2021: Lesesall (1.OG)
    ab Jg. 2022: Lesesaal (EG)

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  6. Article ; Online: Monoallelic Loss-of-Function IFT140 Pathogenic Variants Cause Autosomal Dominant Polycystic Kidney Disease: A Confirmatory Study With Suspicion of an Additional Cardiac Phenotype.

    Salhi, Sofiane / Doreille, Alice / Dancer, Marine Serveaux / Boueilh, Anna / Filipozzi, Pierre / El Karoui, Khalil / Ponce, Fanny / Lebre, Anne-Sophie / Raymond, Laure / Mesnard, Laurent

    American journal of kidney diseases : the official journal of the National Kidney Foundation

    2023  Volume 83, Issue 5, Page(s) 688–691

    Abstract: Autosomal dominant polycystic kidney disease (ADPKD) is the most common inherited kidney disease. While biallelic variants affecting IFT140 are responsible for Mainzer-Saldino syndrome (characterized by severe ciliopathy causing skeletal abnormalities, ... ...

    Abstract Autosomal dominant polycystic kidney disease (ADPKD) is the most common inherited kidney disease. While biallelic variants affecting IFT140 are responsible for Mainzer-Saldino syndrome (characterized by severe ciliopathy causing skeletal abnormalities, kidney disease, and cysts), monoallelic loss-of-function (LoF) variants have been recently reported as an important cause of ADPKD beyond PKD1/2 genes. Herein, we report 6 non-family-related cases of monoallelic IFT140 LoF variants, identified from 1,340 exomes sequenced for nephrological indications in our local database. Every patient presented with polycystic kidney disease. Furthermore, the mother of a boy diagnosed with Mainzer-Saldino syndrome with a biallelic variant affecting IFT140 presented with several bilateral cysts, revealed after kidney imaging, and was found to carry a pathologic frameshift IFT140 variation. As well as this particular Mainzer-Saldino case, our 6 additional patients confirm that heterozygous IFT140 frameshift variants are responsible for the cystic phenotype and kidney failure. Interestingly, of the 6 patients, 2 also exhibited dilated cardiomyopathy, which was of unknown origin, as no genetic cause was found after exome sequencing analysis, suggesting a potential connection between IFT140 and heart disease.
    MeSH term(s) Adolescent ; Adult ; Child ; Female ; Humans ; Male ; Alleles ; Cardiomyopathy, Dilated/genetics ; Carrier Proteins/genetics ; Frameshift Mutation ; Loss of Function Mutation ; Pedigree ; Phenotype ; Polycystic Kidney, Autosomal Dominant/genetics ; Polycystic Kidney, Autosomal Dominant/complications ; Polycystic Kidney, Autosomal Dominant/diagnosis
    Chemical Substances Carrier Proteins ; IFT140 protein, human
    Language English
    Publishing date 2023-10-14
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Case Reports
    ZDB-ID 604539-x
    ISSN 1523-6838 ; 0272-6386
    ISSN (online) 1523-6838
    ISSN 0272-6386
    DOI 10.1053/j.ajkd.2023.08.019
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 1669: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
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    Jg. 1995 - 2021: Lesesall (1.OG)
    ab Jg. 2022: Lesesaal (EG)
    Zs.MO 468: Show issues

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