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Article: An 'Exo-Tick' Problem: A Sudden Increase in Tick Bite - A Case Series.

Sumitra, Sathyavath / Nazeer, Fathima A / Bhaskaramenon, Sindhu C / Sivaprasad, R / Abdul Nazeer, Marjan M / George, Sandhya

Indian dermatology online journal

2023 Volume 15, Issue 1, Page(s) 78–81

Abstract: Ticks are blood-sucking arthropod ectoparasites of vertebrates, which are vectors of many diseases. They cause varied skin manifestations, which occur either due to the attachment of the tick to the host or due to the infections it spreads. Dermoscopy ... ...

Abstract	Ticks are blood-sucking arthropod ectoparasites of vertebrates, which are vectors of many diseases. They cause varied skin manifestations, which occur either due to the attachment of the tick to the host or due to the infections it spreads. Dermoscopy serves as a precise diagnostic tool for tick bites and also helps in ensuring complete removal of the tick. Prompt removal and identification of the tick, along with appropriate antibiotic therapy, are important aspects of the management of this condition. Herein, we present a case series of nine patients with tick bites, by ticks of similar morphology but at different body sites and with varied predisposing factors.
Language	English
Publishing date	2023-11-24
Publishing country	India
Document type	Case Reports
ZDB-ID	2585814-2
ISSN	2249-5673 ; 2229-5178
ISSN (online)	2249-5673
ISSN	2229-5178
DOI	10.4103/idoj.idoj_149_23
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Article ; Online: A case of human buffalopox in Malappuram, India: The role of mpox surveillance in 2022.

Patil, Deepak Y / George, Sandhya / Sahay, Rima R / Chenayil, Shubin / Shete, Anita M / Bhaskaramenon, Sindhu C / Anish, Thekkumkara Surendran / Jain, Rajlaxmi / Nazeer, Fathima Abdul / John, Reena / Joshi, Yash / Sivaprasad, R / Waghmare, Priyanka / Ray, Shilpa / Balakrishnan, Swapna / Kumari, Vaishnavi / Sathe, Shubhangi / Yadav, Pragya D

Journal of medical virology

2023 Volume 95, Issue 2, Page(s) e28580

MeSH term(s)	Humans ; Mpox (monkeypox) ; Vaccinia virus ; Vaccinia ; India/epidemiology
Language	English
Publishing date	2023-03-02
Publishing country	United States
Document type	Case Reports ; Letter ; Research Support, Non-U.S. Gov't
ZDB-ID	752392-0
ISSN	1096-9071 ; 0146-6615
ISSN (online)	1096-9071
ISSN	0146-6615
DOI	10.1002/jmv.28580
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Article: Combined efficacies of lipoic acid and 2,3-dimercaptosuccinic acid against lead-induced lipid peroxidation in rat liver

Sivaprasad, R / Nagaraj, M / Varalakshmi, P

Journal of nutritional biochemistry. 2004 Jan., v. 15, no. 1

2004

Abstract: Oxidative stress with subsequent lipid peroxidation has been postulated as one mechanism for lead toxicity. Hence in assessing the protective effects of lipoic acid (LA) and meso 2,3-dimercaptosuccinic acid (DMSA) on lead toxicity, they were tested ... ...

Abstract	Oxidative stress with subsequent lipid peroxidation has been postulated as one mechanism for lead toxicity. Hence in assessing the protective effects of lipoic acid (LA) and meso 2,3-dimercaptosuccinic acid (DMSA) on lead toxicity, they were tested either separately or in combination for their effects on selected indices of hepatic oxidative stress. Elevated levels of lipid peroxides were accompanied by altered antioxidant defense systems. Lead acetate (Pb - 0.2%) was administered in drinking water for five weeks to induce toxicity. LA (25 mg kg-1 body wt. day-1 i.p) and DMSA (20 mg kg-1 body wt. day-1 i.p) were administered individually and also in combination during the sixth week. Lead damage to the liver was evident in the decreases in hepatic enzymes alanine transaminase (-38%), aspartate transaminase (-42%) and alkaline phosphatase (-43%); increases in lipid peroxidation (+38%); decreases in the antioxidant enzymes catalase (-45%), superoxide dismutase (-40%), glutathione peroxidase (-46%) and decreases in glutathione (-43%) and decreases in glutathione metabolizing enzymes, glutathione reductase (-59%), glucose-6-phosphate dehydrogenase (-27%) and glutathione-S-transferase (-42%). In combination LA and DMSA completely ameliorated the lead induced oxidative damage. Either compound alone was however only partially protective against lead damage.
Keywords	lead acetate ; hepatotoxicity ; lead poisoning ; oxidative stress ; reactive oxygen species ; rats ; animal models ; albino ; drinking water ; nutrient intake ; liver ; lipid peroxidation ; antidotes ; protective effect ; antioxidants ; glutathione peroxidase ; glutathione reductase (NADPH) ; superoxide dismutase ; catalase ; glutathione transferase ; enzyme activity ; liver function ; alanine transaminase ; aspartate transaminase ; alkaline phosphatase
Language	English
Dates of publication	2004-01
Size	p. 18-23.
Document type	Article
ZDB-ID	1014929-6
ISSN	1873-4847 ; 0955-2863
ISSN (online)	1873-4847
ISSN	0955-2863
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Article: Effect of lipoic acid on the oxidoreductive status of red blood cells in rats subject to oxidative stress by chronic administration of adriamycin.

Malarkodi, K P / Sivaprasad, R / Varalakshmi, P

Human & experimental toxicology

2004 Volume 23, Issue 3, Page(s) 129–135

Abstract: One of the most intriguing phenomena observed during adriamycin (ADR) toxicity has been attributed to ADR-induced oxidative stress. The study was aimed to assess the protective effect of lipoic acid (LA) against ADR-induced damage to erythrocytes. Male ... ...

Abstract	One of the most intriguing phenomena observed during adriamycin (ADR) toxicity has been attributed to ADR-induced oxidative stress. The study was aimed to assess the protective effect of lipoic acid (LA) against ADR-induced damage to erythrocytes. Male albino rats (Wistar strain) were subjected to ADR (1 mg/kg body weight/day i.v.) once a week for a period of 12 weeks. Haematological indices like haemoglobin levels (Hb) and haematocrit (Ht) were also lowered along with a marked increase in the activities of serum glutamate pyruvate transaminase (SGPT) and serum glutamate oxaloacetate transaminase (SGOT). These rats demonstrated enhanced erythrocyte membrane lipid peroxidation (LPO) and an onslaught in the antioxidant defence armoury, witnessed by lowered activities of superoxide dismutase (SOD), glutathione peroxidase (GPx), vitamin A, vitamin C and vitamin E. Rats administered with ADR showed a marked decline in the activities of membrane-bound ATPases. Abnormal LPO and decreased deformability led to increased osmotic fragility of the red blood cells. Pretreatment with LA (35 mg/kg body weight/day i.p.) 24 hours prior to the administration of ADR once a week for a period of 12 weeks was effective in counteracting these biochemical disturbances, thereby minimizing the toxic side effects of ADR.
MeSH term(s)	Animals ; Antibiotics, Antineoplastic/toxicity ; Antioxidants/metabolism ; Antioxidants/pharmacology ; Doxorubicin/toxicity ; Erythrocyte Membrane/drug effects ; Erythrocyte Membrane/enzymology ; Erythrocytes/drug effects ; Erythrocytes/metabolism ; Lipid Peroxidation/drug effects ; Male ; Osmotic Fragility/drug effects ; Oxidation-Reduction/drug effects ; Oxidative Stress ; Rats ; Rats, Wistar ; Thioctic Acid/pharmacology
Chemical Substances	Antibiotics, Antineoplastic ; Antioxidants ; Thioctic Acid (73Y7P0K73Y) ; Doxorubicin (80168379AG)
Language	English
Publishing date	2004-03
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1027454-6
ISSN	1477-0903 ; 0960-3271 ; 0144-5952
ISSN (online)	1477-0903
ISSN	0960-3271 ; 0144-5952
DOI	10.1191/0960327104ht428oa
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Article: Combined efficacies of lipoic acid and meso-2,3-dimercaptosuccinic acid on lead-induced erythrocyte membrane lipid peroxidation and antioxidant status in rats.

Sivaprasad, R / Nagaraj, M / Varalakshmi, P

Human & experimental toxicology

2003 Volume 22, Issue 4, Page(s) 183–192

Abstract: One of the most intriguing phenomenon observed during lead toxicity has been attributed to lead-induced oxidative stress. The combined effect of DL-alpha-lipoic acid (LA) and meso-2,3-dimercaptosuccinic acid (DMSA) on lead-induced alterations in selected ...

Abstract	One of the most intriguing phenomenon observed during lead toxicity has been attributed to lead-induced oxidative stress. The combined effect of DL-alpha-lipoic acid (LA) and meso-2,3-dimercaptosuccinic acid (DMSA) on lead-induced alterations in selected parameters, which are indicators of oxidative stress in erythrocytes, have been studied. Lead acetate (Pb, 0.2%) was administered in drinking water for 5 weeks to induce toxicity. LA (25 mg/ kg body weight per day i.p.) and DMSA (20 mg/kg body weight per day i.p.) were administered individually and also in combination during week 6. Clinical evidence of toxic exposure was evident from the elevated blood lead levels (BPb) along with lowered levels of haemoglobin (Hb) and haematocrit (Ht). Lead-exposed animals showed enhanced membrane lipid peroxidation (LPO) in the erythrocytes. Damage to the erythrocyte membrane was evident from the decline in the activities of the transmembrane enzymes, viz., Na+, K(+)-ATPase, Ca(2+)-ATPase and Mg(2+)-ATPase. Lead-exposed rats also suffered an onslaught on the antioxidant defence system witnessed by lowered activities of catalase (CAT), superoxide dismutase (SOD), glutathione peroxidase (GPx) and reduced glutathione (GSH). Serum glutamic-oxoloacetic transaminase (SGOT) and serum glutamic-pyruvic transaminase (SGPT) were also elevated in lead-exposed rats. Treatment with either LA or DMSA reversed the lead-induced biochemical disturbances encountered by the erythrocytes, but combined treatment with LA and DMSA was very effective in mitigating all the parameters indicative of oxidative stress.
MeSH term(s)	Alanine Transaminase/blood ; Animals ; Antidotes/pharmacology ; Antioxidants/metabolism ; Aspartate Aminotransferases/blood ; Erythrocyte Membrane/drug effects ; Erythrocyte Membrane/physiology ; Lead Poisoning/blood ; Lipid Peroxidation/drug effects ; Male ; Models, Animal ; Oxidative Stress/drug effects ; Rats ; Rats, Wistar ; Succimer/pharmacology ; Thioctic Acid/pharmacology
Chemical Substances	Antidotes ; Antioxidants ; Thioctic Acid (73Y7P0K73Y) ; Succimer (DX1U2629QE) ; Aspartate Aminotransferases (EC 2.6.1.1) ; Alanine Transaminase (EC 2.6.1.2)
Language	English
Publishing date	2003-04
Publishing country	England
Document type	Journal Article
ZDB-ID	1027454-6
ISSN	1477-0903 ; 0960-3271 ; 0144-5952
ISSN (online)	1477-0903
ISSN	0960-3271 ; 0144-5952
DOI	10.1191/0960327103ht335oa
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Article: Combined efficacies of lipoic acid and 2,3-dimercaptosuccinic acid against lead-induced lipid peroxidation in rat liver.

Sivaprasad, R / Nagaraj, M / Varalakshmi, P

The Journal of nutritional biochemistry

2003 Volume 15, Issue 1, Page(s) 18–23

Abstract	Oxidative stress with subsequent lipid peroxidation has been postulated as one mechanism for lead toxicity. Hence in assessing the protective effects of lipoic acid (LA) and meso 2,3-dimercaptosuccinic acid (DMSA) on lead toxicity, they were tested either separately or in combination for their effects on selected indices of hepatic oxidative stress. Elevated levels of lipid peroxides were accompanied by altered antioxidant defense systems. Lead acetate (Pb - 0.2%) was administered in drinking water for five weeks to induce toxicity. LA (25 mg kg(-1) body wt. day(-1) i.p) and DMSA (20 mg kg(-1) body wt. day(-1) i.p) were administered individually and also in combination during the sixth week. Lead damage to the liver was evident in the decreases in hepatic enzymes alanine transaminase (-38%), aspartate transaminase (-42%) and alkaline phosphatase (-43%); increases in lipid peroxidation (+38%); decreases in the antioxidant enzymes catalase (-45%), superoxide dismutase (-40%), glutathione peroxidase (-46%) and decreases in glutathione (-43%) and decreases in glutathione metabolizing enzymes, glutathione reductase (-59%), glucose-6-phosphate dehydrogenase (-27%) and glutathione-S-transferase (-42%). In combination LA and DMSA completely ameliorated the lead induced oxidative damage. Either compound alone was however only partially protective against lead damage.
MeSH term(s)	Alanine Transaminase/metabolism ; Alkaline Phosphatase/metabolism ; Animals ; Antidotes ; Aspartate Aminotransferases/metabolism ; Catalase/metabolism ; Lead Poisoning/prevention & control ; Lipid Peroxidation/drug effects ; Liver/drug effects ; Liver/pathology ; Male ; Rats ; Rats, Wistar ; Succimer/pharmacology ; Superoxide Dismutase/metabolism ; Thioctic Acid/pharmacology
Chemical Substances	Antidotes ; Thioctic Acid (73Y7P0K73Y) ; Succimer (DX1U2629QE) ; Catalase (EC 1.11.1.6) ; Superoxide Dismutase (EC 1.15.1.1) ; Aspartate Aminotransferases (EC 2.6.1.1) ; Alanine Transaminase (EC 2.6.1.2) ; Alkaline Phosphatase (EC 3.1.3.1)
Language	English
Publishing date	2003-12-21
Publishing country	United States
Document type	Journal Article
ZDB-ID	1014929-6
ISSN	1873-4847 ; 0955-2863
ISSN (online)	1873-4847
ISSN	0955-2863
DOI	10.1016/j.jnutbio.2003.09.001
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Zs.A 2838: Show issues

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Article: The morning report: starting the day right at a teaching hospital.

Sivaprasad, R / Taguchi, J T

American College of Physicians observer

1982 Volume 2, Issue 10, Page(s) 1, 17

MeSH term(s)	Group Processes ; Hospital Bed Capacity, 500 and over ; Internship and Residency ; Medical Records ; Ohio
Language	English
Publishing date	1982-11
Publishing country	United States
Document type	Journal Article
ISSN	0279-9529
ISSN	0279-9529
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Article: Western diamondback rattlesnake (Crotalus atrox) poisoning.

Sivaprasad, R / Cantini, E M

Postgraduate medicine

1982 Volume 71, Issue 6, Page(s) 223–7, 230

MeSH term(s)	Adrenal Cortex Hormones/therapeutic use ; Adult ; Antivenins/therapeutic use ; Crotalid Venoms/poisoning ; Cryosurgery ; Fasciotomy ; First Aid ; Humans ; Male ; Snake Bites/therapy ; Suction ; Tourniquets
Chemical Substances	Adrenal Cortex Hormones ; Antivenins ; Crotalid Venoms
Language	English
Publishing date	1982-06
Publishing country	England
Document type	Case Reports ; Journal Article
ZDB-ID	410138-8
ISSN	1941-9260 ; 0032-5481
ISSN (online)	1941-9260
ISSN	0032-5481
DOI	10.1080/00325481.1982.11716105
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Article: Lipoic acid in combination with a chelator ameliorates lead-induced peroxidative damages in rat kidney.

Sivaprasad, Rajagopalan / Nagaraj, Manickam / Varalakshmi, Palaninathan

Archives of toxicology

2002 Volume 76, Issue 8, Page(s) 437–441

Abstract: The deleterious effect of lead has been attributed to lead-induced oxidative stress with the consequence of lipid peroxidation. The present study was designed to investigate the combined effect of DL-alpha-lipoic acid (LA) and meso-2,3-dimercaptosuccinic ...

Abstract	The deleterious effect of lead has been attributed to lead-induced oxidative stress with the consequence of lipid peroxidation. The present study was designed to investigate the combined effect of DL-alpha-lipoic acid (LA) and meso-2,3-dimercaptosuccinic acid (DMSA) on lead-induced peroxidative damages in rat kidney. The increase in peroxidated lipids in lead-poisoned rats was accompanied by alterations in antioxidant defence systems. Lead acetate (Pb, 0.2%) was administered in drinking water for 5 weeks to induce lead toxicity. LA (25 mg/kg body weight per day i.p.) and DMSA (20 mg/kg body weight per day i.p.) were administered individually and also in combination during the sixth week. Nephrotoxic damage was evident from decreases in the activities of gamma-glutamyl transferase and N-acetyl beta- D-glucosaminidase, which were reversed upon combined treatment with LA and DMSA. Rats subjected to lead intoxication showed a decline in the thiol capacity of the cell, accompanied by high malondialdehyde levels along with lowered activities of catalase, superoxide dismutase, glutathione peroxidase and glutathione metabolizing enzymes (glutathione reductase, glucose-6-phosphate dehydrogenase, glutathione- S-transferase). Supplementation with LA as a sole agent showed considerable changes over oxidative stress parameters. The study has highlighted the combined effect of both drugs as being more effective in reversing oxidative damage by bringing about an improvement in the reductive status of the cell.
MeSH term(s)	Acetylglucosaminidase/metabolism ; Animals ; Catalase/metabolism ; Chelating Agents/pharmacology ; Chelating Agents/therapeutic use ; Disease Models, Animal ; Drug Therapy, Combination ; Glutathione/metabolism ; Glutathione Peroxidase/metabolism ; Kidney/drug effects ; Kidney/enzymology ; Kidney/metabolism ; Lead Poisoning/drug therapy ; Lead Poisoning/metabolism ; Lipid Peroxidation/drug effects ; Lipid Peroxides/metabolism ; Male ; Rats ; Rats, Wistar ; Succimer/pharmacology ; Succimer/therapeutic use ; Superoxide Dismutase/metabolism ; Thioctic Acid/pharmacology ; Thioctic Acid/therapeutic use ; gamma-Glutamyltransferase/metabolism
Chemical Substances	Chelating Agents ; Lipid Peroxides ; Thioctic Acid (73Y7P0K73Y) ; Succimer (DX1U2629QE) ; Catalase (EC 1.11.1.6) ; Glutathione Peroxidase (EC 1.11.1.9) ; Superoxide Dismutase (EC 1.15.1.1) ; gamma-Glutamyltransferase (EC 2.3.2.2) ; Acetylglucosaminidase (EC 3.2.1.52) ; Glutathione (GAN16C9B8O)
Language	English
Publishing date	2002-08
Publishing country	Germany
Document type	Journal Article
ZDB-ID	124992-7
ISSN	1432-0738 ; 0340-5761
ISSN (online)	1432-0738
ISSN	0340-5761
DOI	10.1007/s00204-002-0350-x
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Article: Jaundice: an internist's perspective.

Sivaprasad, R / Gopalaswamy, N

Radiologic clinics of North America

1980 Volume 18, Issue 2, Page(s) 179–185

Abstract: Jaundice may result from a pathophysiologic abnormality in the uptake, transport, conjugation, or excretion of bilirubin. The pathogenesis of this disease is always difficult to determine, but studies such as abdominal ultrasonography, percutaneous ... ...

Abstract	Jaundice may result from a pathophysiologic abnormality in the uptake, transport, conjugation, or excretion of bilirubin. The pathogenesis of this disease is always difficult to determine, but studies such as abdominal ultrasonography, percutaneous transhepatic cholangiography, and endoscopic retrograde cholangiopancreatography have facilitated diagnosis.
MeSH term(s)	Anemia, Hemolytic/etiology ; Bacterial Infections/complications ; Bilirubin/blood ; Biopsy, Needle ; Cholangiography ; Cholangiopancreatography, Endoscopic Retrograde ; Cholestasis/complications ; Diagnosis, Differential ; Humans ; Jaundice/diagnosis ; Jaundice/etiology ; Laparoscopy ; Liver/diagnostic imaging ; Liver/pathology ; Liver Cirrhosis, Alcoholic/etiology ; Radionuclide Imaging ; Tomography, X-Ray Computed ; Ultrasonography
Chemical Substances	Bilirubin (RFM9X3LJ49)
Language	English
Publishing date	1980-08
Publishing country	United States
Document type	Journal Article
ZDB-ID	215712-3
ISSN	1557-8275 ; 0033-8389
ISSN (online)	1557-8275
ISSN	0033-8389
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