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Article ; Online: Gelsolin Inhibits the Proliferation of Colon Cancer Cells by Enhancing the Expression of TNFR2/CASP10 as a Death Receptor Pathway.

Wang, Zihang / Song, Wuqi

2023 Volume 30, Issue 3, Page(s) 214–220

Abstract: Background: Colon cancer has the second highest incidence rate of digestive system tumors. It relies on surgical treatment, radiotherapy and chemotherapy, and targeted drug therapy.: Objective: To study the mechanism of GSN in the proliferation of ... ...

Abstract	Background: Colon cancer has the second highest incidence rate of digestive system tumors. It relies on surgical treatment, radiotherapy and chemotherapy, and targeted drug therapy. Objective: To study the mechanism of GSN in the proliferation of colon cancer cells. Materials and methods: The expression of gelsolin (GSN) was analyzed with the data of colon cancer patients in the TCGA database. SW620 cells were treated by GSN in vitro and the gene expression was detected by immunoblotting and quantitative PCR. Results: The expression of GSN was found significantly low in colon cancer cells and correlated with the prognosis of patients. The SW620 cell line cultured in vitro was treated with exogenous GSN. SW620 can be significantly inhibited above the concentration of 250 μg/ml. The results of immunoblotting and quantitative PCR showed that exogenous GSN can effectively improve the transcription level of death receptor-related pathway genes such as TNFR2 and CASP10. Conclusion: This study found that GSN inhibited the proliferation of SW620 cells in vitro by upregulating the expression of death receptor pathway-related proteins.
MeSH term(s)	Humans ; Gelsolin/genetics ; Gelsolin/metabolism ; Receptors, Tumor Necrosis Factor, Type II/metabolism ; Colonic Neoplasms/drug therapy ; Colonic Neoplasms/genetics ; Cell Proliferation ; Receptors, Death Domain/metabolism ; Caspase 10/metabolism
Chemical Substances	Gelsolin ; Receptors, Tumor Necrosis Factor, Type II ; Receptors, Death Domain ; CASP10 protein, human (EC 3.4.22.63) ; Caspase 10 (EC 3.4.22.-)
Language	English
Publishing date	2023-02-01
Publishing country	Netherlands
Document type	Journal Article
ZDB-ID	1280776-x
ISSN	1875-5305 ; 0929-8665
ISSN (online)	1875-5305
ISSN	0929-8665
DOI	10.2174/0929866530666230124113639
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Zs.A 4452: Show issues

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Article ; Online: Integrated analysis of ferroptosis-related gene signature for overall survival prediction in Asian patients with hepatocellular carcinoma.

Wang, Zihang / Zhang, Lan / Xu, Jinyuan / Song, Wuqi

Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico

2022 Volume 25, Issue 3, Page(s) 721–730

Abstract: Purpose: Hepatocellular carcinoma (HCC) is one of the most prevalent types of cancers in Asia. Accumulating evidence suggests that ferroptosis is a non-apoptotic form of cell death, and has played an important role in cancer biology.: Methods: Based ... ...

Abstract	Purpose: Hepatocellular carcinoma (HCC) is one of the most prevalent types of cancers in Asia. Accumulating evidence suggests that ferroptosis is a non-apoptotic form of cell death, and has played an important role in cancer biology. Methods: Based on the manually curated ferroptosis-related gene set and TCGA-LIHC dataset of Asian patients, we used DESeq2, Kaplan-Meier analysis, and univariate Cox regression to identify differentially expressed ferroptosis-related genes with significantly prognostic capacity. A risk signature was constructed based on the selected genes for predicting the survival of HCC patients in Asia. The survival prediction accuracy was confirmed by the time-dependent receiver operating characteristic (ROC) curve analysis. Gene set variation analysis (GSVA) was used to explore the functional associations of the signature. Ferroptosis potential index (FPI) and xCell algorithm was applied to quantify ferroptosis and immune cell infiltration, respectively. Two independent datasets from the GEO and the ICGC database were used for external validation. Results: The ferroptosis-related signature could accurately predict the survival outcomes of HCC patients in Asian (p value < 0.0001). We showed that the signature was an independent factor and was beneficial in elevating risk stratification of current clinicopathologic features, such as the amount of alpha-fetoprotein (AFP) and residual tumor classification. Functional characterization showed that critical processes in tumorigenesis belonged to the high-risk groups, for example inflammatory response, which may be the main driver of HCC. The high-risk group had higher FPIs and infiltrations of macrophages and T-helper cells than the low-risk group. Furthermore, two independent cohorts confirmed the prognostic value of our signature. Conclusion: Overall, our results demonstrated potential application of ferroptosis-related genes as independent biomarkers in Asian HCC patients. Targeting ferroptosis may be clinically useful beyond known clinicopathological factors and provide benefit in immunotherapy.
MeSH term(s)	Humans ; Carcinoma, Hepatocellular/genetics ; Ferroptosis/genetics ; Liver Neoplasms/genetics ; Algorithms ; Carcinogenesis ; Prognosis
Language	English
Publishing date	2022-11-01
Publishing country	Italy
Document type	Journal Article
ZDB-ID	2397359-6
ISSN	1699-3055 ; 1699-048X
ISSN (online)	1699-3055
ISSN	1699-048X
DOI	10.1007/s12094-022-02977-7
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Zs.A 6644: Show issues

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Article ; Online: Dihydroartemisinin Inhibits TGF-β-Induced Fibrosis in Human Tenon Fibroblasts via Inducing Autophagy.

Wang, Xiaoyuan / Song, Wuqi / Zhang, Fengmin / Huang, Renping

Drug design, development and therapy

2021 Volume 15, Page(s) 973–981

Abstract: Background: The formation of hypertrophic scars (HS) can result in the failure of glaucoma surgery, and fibrosis is known to be closely associated with the progression of HS. Dihydroartemisinin (DHA) has been reported to inhibit the progression of ... ...

Abstract	Background: The formation of hypertrophic scars (HS) can result in the failure of glaucoma surgery, and fibrosis is known to be closely associated with the progression of HS. Dihydroartemisinin (DHA) has been reported to inhibit the progression of fibrosis; however, whether DHA can alleviate the formation of HS remains unclear. Methods: In the present study, in order to examine the effects of DHA on the progression of HS, human Tenon's capsule fibroblasts (HTFs) were isolated from patients who underwent glaucoma surgery. In addition, Western blot analysis, microtubule associated protein 1 light chain 3 α staining and reverse transcription-quantitative PCR were performed to detect protein and mRNA expression levels in the HTFs, respectively. Cell proliferation was detected by Ki67 staining. Flow cytometry was used to examine apoptosis and reactive oxygen species (ROS) levels in the HTFs. Results: The results revealed that TGF-β promoted the proliferation and fibrosis of HTFs; however, DHA significantly reversed the effects of TGF-β by increasing cell autophagy. In addition, DHA notably induced the apoptosis of TGF-β-stimulated HTFs by increasing the ROS levels, while these increases were partially reversed by 3-methyladenine. Furthermore, DHA notably increased the expression of microRNA (miR)-145-5p in HTFs in a dose-dependent manner. Conclusion: The present study demonstrated that DHA inhibits the TGF-β-induced fibrosis of HTFs by inducing autophagy. These findings may aid in the development of novel agents for the prevention of the formation of HS following glaucoma surgery.
MeSH term(s)	Artemisinins/pharmacology ; Autophagy/drug effects ; Cell Proliferation/drug effects ; Cells, Cultured ; Dose-Response Relationship, Drug ; Fibroblasts/drug effects ; Fibrosis/drug therapy ; Fibrosis/metabolism ; Fibrosis/pathology ; Humans ; Molecular Structure ; Structure-Activity Relationship ; Tenon Capsule/drug effects ; Tenon Capsule/metabolism ; Tenon Capsule/pathology ; Transforming Growth Factor beta/antagonists & inhibitors ; Transforming Growth Factor beta/metabolism
Chemical Substances	Artemisinins ; Transforming Growth Factor beta ; artenimol (6A9O50735X)
Language	English
Publishing date	2021-03-03
Publishing country	New Zealand
Document type	Journal Article
ZDB-ID	2451346-5
ISSN	1177-8881 ; 1177-8881
ISSN (online)	1177-8881
ISSN	1177-8881
DOI	10.2147/DDDT.S280322
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Article: Psychophysical therapy and underlying neuroendocrine mechanisms for the rehabilitation of long COVID-19.

Meng, Qing-Tai / Song, Wu-Qi / Churilov, Leonid P / Zhang, Feng-Min / Wang, Yu-Feng

Frontiers in endocrinology

2023 Volume 14, Page(s) 1120475

Abstract: With the global epidemic and prevention of the COVID-19, long COVID-19 sequelae and its comprehensive prevention have attracted widespread attention. Long COVID-19 sequelae refer to that three months after acute COVID-19, the test of SARS-CoV-2 is ... ...

Abstract	With the global epidemic and prevention of the COVID-19, long COVID-19 sequelae and its comprehensive prevention have attracted widespread attention. Long COVID-19 sequelae refer to that three months after acute COVID-19, the test of SARS-CoV-2 is negative, but some symptoms still exist, such as cough, prolonged dyspnea and fatigue, shortness of breath, palpitations and insomnia. Its pathological mechanism is related to direct viral damage, immunopathological response, endocrine and metabolism disorders. Although there are more effective methods for treating COVID-19, the treatment options available for patients with long COVID-19 remain quite limited. Psychophysical therapies, such as exercise, oxygen therapy, photobiomodulation, and meditation, have been attempted as treatment modalities for long COVID-19, which have the potential to promote recovery through immune regulation, antioxidant effects, and neuroendocrine regulation. Neuroendocrine regulation plays a significant role in repairing damage after viral infection, regulating immune homeostasis, and improving metabolic activity in patients with long COVID-19. This review uses oxytocin as an example to examine the neuroendocrine mechanisms involved in the psychophysical therapies of long COVID-19 syndrome and proposes a psychophysical strategy for the treatment of long COVID-19.
MeSH term(s)	Humans ; Post-Acute COVID-19 Syndrome ; COVID-19/therapy ; SARS-CoV-2 ; Neurosecretory Systems ; Disease Progression
Language	English
Publishing date	2023-09-29
Publishing country	Switzerland
Document type	Journal Article ; Review
ZDB-ID	2592084-4
ISSN	1664-2392
ISSN	1664-2392
DOI	10.3389/fendo.2023.1120475
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Article ; Online: Photobiomodulation with 630-nm LED Inhibits M1 Macrophage Polarization via STAT1 Pathway Against Sepsis-Induced Acute Lung Injury.

Pan, Yue / Zhang, Hanxu / Liu, Qiannan / Wu, Hao / Du, Siqi / Song, Wuqi / Zhang, Fengmin / Liu, Hailiang

Photobiomodulation, photomedicine, and laser surgery

2024 Volume 42, Issue 2, Page(s) 148–158

Abstract: Background: ...

Abstract	Background:
MeSH term(s)	Mice ; Animals ; Low-Level Light Therapy ; Mice, Inbred C57BL ; Acute Lung Injury/complications ; Acute Lung Injury/drug therapy ; Macrophages ; Sepsis/complications ; Sepsis/radiotherapy ; Sepsis/drug therapy ; RNA, Messenger
Chemical Substances	RNA, Messenger
Language	English
Publishing date	2024-02-02
Publishing country	United States
Document type	Journal Article
ISSN	2578-5478
ISSN (online)	2578-5478
DOI	10.1089/photob.2023.0034
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Article ; Online: LED irradiation at 630 nm alleviates collagen-induced arthritis in mice by inhibition of NF-κB-mediated MMPs production.

Song, Wuqi / Zhang, Hanxu / Pan, Yue / Xia, Qing / Liu, Qiannan / Wu, Hao / Du, Siqi / Zhang, Fengmin / Liu, Hailiang

Photochemical & photobiological sciences : Official journal of the European Photochemistry Association and the European Society for Photobiology

2023 Volume 22, Issue 10, Page(s) 2271–2283

Abstract: Matrix metallopreteinase (MMP), a family of matrix degrading enzyme, plays a significant role in persistent and irreversible joint damage in rheumatoid arthritis (RA). Photobiomodulatory therapy (PBMT) has become an emerging adjunct therapy for RA. ... ...

Abstract	Matrix metallopreteinase (MMP), a family of matrix degrading enzyme, plays a significant role in persistent and irreversible joint damage in rheumatoid arthritis (RA). Photobiomodulatory therapy (PBMT) has become an emerging adjunct therapy for RA. However, the molecular mechanism of PBMT on RA remains unclear. The purpose of this study is to explore the effect of 630 nm light emitting diode (LED) irradiation on RA and its underly molecular mechanism. Arthritis clinic scores, histology analysis and micro-CT results show that 630 nm LED irradiation ameliorates collagen-induced arthritis (CIA) in mice with the reduction of the extents of paw swelling, inflammation and bone damage. 630 nm LED irradiation significantly reduces MMP-3 and MMP-9 levels and inhibits p65 phosphorylation level in the paws of CIA mice. Moreover, 630 nm LED irradiation significantly inhibits the mRNA and protein levels of MMP-3 and MMP-9 in TNF-α-treated MH7A cells, a human synovial cell line. Importantly, 630 nm LED irradiation reduces TNF-α-induced the phosphorylated level of p65 but not alters STAT1, STAT3, Erk1/2, JNK and p38 phosphorylation levels. Immunofluorescence result showed that 630 nm LED irradiation blocks p65 nuclear translocation in MH7A cells. In addition, other MMPs mRNA regulated by NF-κB were also significantly inhibited by LED irradiation in vivo and in vitro. These results indicates that 630 nm LED irradiation reduces the MMPs levels to ameliorate the development of RA by inhibiting the phosphorylation of p65 selectively, suggesting that 630 nm LED irradiation may be a beneficial adjunct therapy for RA.Graphical abstract.
MeSH term(s)	Animals ; Humans ; Mice ; Arthritis, Experimental/drug therapy ; Arthritis, Experimental/chemically induced ; Arthritis, Experimental/metabolism ; Arthritis, Rheumatoid/genetics ; Arthritis, Rheumatoid/metabolism ; Arthritis, Rheumatoid/pathology ; Matrix Metalloproteinase 3/genetics ; Matrix Metalloproteinase 9 ; NF-kappa B/metabolism ; Tumor Necrosis Factor-alpha/metabolism ; Tumor Necrosis Factor-alpha/pharmacology
Chemical Substances	Matrix Metalloproteinase 3 (EC 3.4.24.17) ; Matrix Metalloproteinase 9 (EC 3.4.24.35) ; NF-kappa B ; Tumor Necrosis Factor-alpha
Language	English
Publishing date	2023-07-02
Publishing country	England
Document type	Journal Article
ZDB-ID	2072584-X
ISSN	1474-9092 ; 1474-905X
ISSN (online)	1474-9092
ISSN	1474-905X
DOI	10.1007/s43630-023-00449-7
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Article ; Online: The rules and regulatory mechanisms of FOXO3 on inflammation, metabolism, cell death and aging in hosts.

Cao, Guoding / Lin, Monan / Gu, Wei / Su, Zaiyu / Duan, Yagan / Song, Wuqi / Liu, Hailiang / Zhang, Fengmin

Life sciences

2023 Volume 328, Page(s) 121877

Abstract: The FOX family of transcription factors was originally identified in 1989, comprising the FOXA to FOXS subfamilies. FOXO3, a well-known member of the FOXO subfamily, is widely expressed in various human organs and tissues, with higher expression levels ... ...

Abstract	The FOX family of transcription factors was originally identified in 1989, comprising the FOXA to FOXS subfamilies. FOXO3, a well-known member of the FOXO subfamily, is widely expressed in various human organs and tissues, with higher expression levels in the ovary, skeletal muscle, heart, and spleen. The biological effects of FOXO3 are mostly determined by its phosphorylation, which occurs in the nucleus or cytoplasm. Phosphorylation of FOXO3 in the nucleus can promote its translocation into the cytoplasm and inhibit its transcriptional activity. In contrast, phosphorylation of FOXO3 in the cytoplasm leads to its translocation into the nucleus and exerts regulatory effects on biological processes, such as inflammation, aerobic glycolysis, autophagy, apoptosis, oxidative stress, cell cycle arrest and DNA damage repair. Additionally, FOXO3 isoform 2 acts as an important suppressor of osteoclast differentiation. FOXO3 can also interfere with the development of various diseases, including inhibiting the proliferation and invasion of tumor cells, blocking the production of inflammatory factors in autoimmune diseases, and inhibiting β-amyloid deposition in Alzheimer's disease. Furthermore, FOXO3 slows down the aging process and exerts anti-aging effects by delaying telomere attrition, promoting cell self-renewal, and maintaining genomic stability. This review suggests that changes in the levels and post-translational modifications of FOXO3 protein can maintain organismal homeostasis and improve age-related diseases, thus counteracting aging. Moreover, this may indicate that alterations in FOXO3 protein levels are also crucial for longevity, offering new perspectives for therapeutic strategies targeting FOXO3.
MeSH term(s)	Humans ; Forkhead Box Protein O3/genetics ; Forkhead Transcription Factors/genetics ; Aging ; Apoptosis/genetics ; Inflammation
Chemical Substances	Forkhead Box Protein O3 ; Forkhead Transcription Factors ; FOXO3 protein, human
Language	English
Publishing date	2023-06-22
Publishing country	Netherlands
Document type	Journal Article ; Review
ZDB-ID	3378-9
ISSN	1879-0631 ; 0024-3205
ISSN (online)	1879-0631
ISSN	0024-3205
DOI	10.1016/j.lfs.2023.121877
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Article ; Online: Web Crawling and mRNA Sequencing Analyze Mechanisms of Photobiomodulation.

Wu, Hao / Liu, Qiannan / Meng, Caiyun / Xia, Qing / Pan, Yue / Zhang, Hanxu / Zhang, Fengmin / Song, Wuqi / Liu, Hailiang

Photobiomodulation, photomedicine, and laser surgery

2022 Volume 40, Issue 4, Page(s) 252–260

Abstract: Background: ...

Abstract	Background:
MeSH term(s)	Anti-Inflammatory Agents ; Computational Biology ; Cytokines/genetics ; Humans ; Internet ; Low-Level Light Therapy ; RNA, Messenger/genetics
Chemical Substances	Anti-Inflammatory Agents ; Cytokines ; RNA, Messenger
Language	English
Publishing date	2022-04-22
Publishing country	United States
Document type	Journal Article
ISSN	2578-5478
ISSN (online)	2578-5478
DOI	10.1089/photob.2021.0142
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Article ; Online: Heat Shock Protein Is Associated with Inhibition of Inflammatory Cytokine Production by 630 nm Light-Emitting Diode Irradiation in Fibroblast-Like Synoviocytes Based on RNA Sequencing Analysis.

Liu, Qiannan / Wu, Hao / Zhang, Hanxu / Pan, Yue / Du, Siqi / Song, Wuqi / Zhang, Fengmin / Liu, Hailiang

Photobiomodulation, photomedicine, and laser surgery

2022 Volume 40, Issue 11, Page(s) 751–762

Abstract: Background: ...

Abstract	Background:
MeSH term(s)	Humans ; Synoviocytes/chemistry ; Synoviocytes/metabolism ; Synoviocytes/pathology ; Synovial Membrane/chemistry ; Synovial Membrane/metabolism ; Synovial Membrane/pathology ; Tumor Necrosis Factor-alpha/metabolism ; Heat-Shock Proteins ; Cells, Cultured ; Fibroblasts/metabolism ; Arthritis, Rheumatoid/radiotherapy ; Arthritis, Rheumatoid/metabolism ; Arthritis, Rheumatoid/pathology ; Inflammation ; RNA, Messenger/genetics ; RNA, Messenger/analysis ; RNA, Messenger/metabolism ; Sequence Analysis, RNA
Chemical Substances	Tumor Necrosis Factor-alpha ; Heat-Shock Proteins ; RNA, Messenger
Language	English
Publishing date	2022-11-17
Publishing country	United States
Document type	Journal Article
ISSN	2578-5478
ISSN (online)	2578-5478
DOI	10.1089/photob.2022.0041
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Article ; Online: miR‑17‑5p regulates the proliferation and apoptosis of human trabecular meshwork cells by targeting phosphatase and tensin homolog.

Wang, Xiaoyuan / Li, Zhijian / Bai, Jie / Song, Wuqi / Zhang, Fengmin

Molecular medicine reports

2019 Volume 19, Issue 4, Page(s) 3132–3138

Abstract: Glaucoma is one of the leading causes of blindness. Previous studies have indicated that the oxidative stress‑induced apoptosis of trabecular meshwork cells (TMCs) may serve a key role in the pathogenesis of glaucoma, and that micro RNA(miR)‑17‑5p may be ...

Abstract	Glaucoma is one of the leading causes of blindness. Previous studies have indicated that the oxidative stress‑induced apoptosis of trabecular meshwork cells (TMCs) may serve a key role in the pathogenesis of glaucoma, and that micro RNA(miR)‑17‑5p may be involved in this process. However, the specific mechanisms require further investigation. The aim of the present study was to investigate the effects of miR‑17‑5p on the proliferation and apoptosis of human TMCs (HTMCs) in response to oxidative stress. It was observed that exposure to H2O2 induced a significant decrease in the proliferation and a marked increase in the apoptosis of HTMCs. H2O2 exposure also suppressed the expression of miR‑17‑5p and promoted the expression of phosphatase and tensin homolog (PTEN). Furthermore, transient overexpression of miR‑17‑5p induced a significant increase in the proliferation and a significant decrease in the apoptosis of HTMCs by affecting the expression of PTEN, and the apoptosis‑related proteins B‑cell lymphoma‑associated X protein (Bax), B‑cell lymphoma‑extra large (Bcl‑xL) and B‑cell lymphoma‑2 (Bcl‑2). However, knockdown of miR‑17‑5p demonstrated the opposite results. The results of a dual luciferase reporter assay demonstrated that PTEN may be a direct target of miR‑17‑5p. In conclusion, miR‑17‑5p was downregulated in HTMCs under oxidative conditions, and miR‑17‑5p may regulate the apoptosis of HTMCs by targeting PTEN. These results provide a novel theoretical basis and potential therapeutic target for the treatment of glaucoma.
MeSH term(s)	Apoptosis/genetics ; Cell Proliferation ; Gene Expression Regulation ; Humans ; MicroRNAs/genetics ; Oxidative Stress ; PTEN Phosphohydrolase/genetics ; PTEN Phosphohydrolase/metabolism ; RNA Interference ; Trabecular Meshwork/cytology ; Trabecular Meshwork/metabolism
Chemical Substances	MIRN17 microRNA, human ; MicroRNAs ; PTEN Phosphohydrolase (EC 3.1.3.67) ; PTEN protein, human (EC 3.1.3.67)
Language	English
Publishing date	2019-02-20
Publishing country	Greece
Document type	Journal Article
ISSN	1791-3004
ISSN (online)	1791-3004
DOI	10.3892/mmr.2019.9973
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