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  1. Book: Oxford handbook of nephrology and hypertension

    Steddon, Simon

    (Oxford medical publications)

    2006  

    Title variant Handbook of nephrology and hypertension ; Nephrology and hypertension
    Author's details ed. by Simon Steddon
    Series title Oxford medical publications
    Keywords Kidney Diseases
    Language English
    Size XIV, 657 S. : Ill., graph. Darst.
    Publisher Oxford Univ. Press
    Publishing place Oxford
    Publishing country Great Britain
    Document type Book
    Note Begleitbd. zu Oxford handbook of dialysis
    HBZ-ID HT014736087
    ISBN 0-19-852069-7 ; 978-0-19-852069-6
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2007 A 156 order for loan Magazin

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  2. Book: Oxford handbook of nephrology and hypertension

    Steddon, Simon

    (Oxford medical publications ; Oxford handbooks)

    2006  

    Title variant Handbook of nephrology and hypertension ; Nephrology and hypertension
    Author's details edited by Simon Steddon ... [et al.]
    Series title Oxford medical publications
    Oxford handbooks
    MeSH term(s) Kidney Diseases
    Language English
    Size xiv, 657 p. :, ill.
    Publisher Oxford University Press
    Publishing place Oxford ; New York
    Document type Book
    Note Includes index.
    ISBN 9780198520696 ; 0198520697
    Database Catalogue of the US National Library of Medicine (NLM)

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  3. Article ; Online: Renal Association Clinical Practice Guideline in mineral and bone disorders in CKD.

    Steddon, Simon / Sharples, Edward

    Nephron. Clinical practice

    2011  Volume 118 Suppl 1, Page(s) c145–52

    MeSH term(s) Bone Diseases/complications ; Bone Diseases/diagnosis ; Bone Diseases/therapy ; Calcification, Physiologic ; Chronic Kidney Disease-Mineral and Bone Disorder/complications ; Chronic Kidney Disease-Mineral and Bone Disorder/diagnosis ; Chronic Kidney Disease-Mineral and Bone Disorder/therapy ; Clinical Trials as Topic/standards ; Humans ; Kidney Failure, Chronic/complications ; Kidney Failure, Chronic/diagnosis ; Kidney Failure, Chronic/therapy ; National Health Programs/standards ; Practice Guidelines as Topic/standards ; Societies, Medical/standards ; United Kingdom
    Language English
    Publishing date 2011
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 207121-6
    ISSN 1660-2110 ; 1423-0186 ; 2235-3186 ; 1660-8151 ; 0028-2766
    ISSN (online) 1660-2110 ; 1423-0186 ; 2235-3186
    ISSN 1660-8151 ; 0028-2766
    DOI 10.1159/000328066
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article ; Online: Renal Association Clinical Practice Guideline in Mineral and Bone Disorders in CKD

    Steddon, Simon / Sharples, Edward

    Nephron Clinical Practice

    2011  Volume 118, Issue S1, Page(s) c145–c152

    Keywords mineral bone disorders ; chronic kidney disease ; parathyroid hormone ; calcium ; phosphate ; phosphate binder
    Language English
    Publisher S. Karger AG
    Publishing place Basel
    Publishing country Switzerland
    Document type Article ; Online
    ZDB-ID 207121-6
    ISSN 1660-2110 ; 1423-0186 ; 0028-2766 ; 1660-8151 ; 1660-2110 ; 0028-2766 ; 1660-8151
    ISSN (online) 1660-2110 ; 1423-0186
    ISSN 1660-2110 ; 0028-2766 ; 1660-8151
    DOI 10.1159/000328066
    Database Karger publisher's database

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  5. Article: Renal Association Clinical Practice Guideline in Mineral and Bone Disorders in CKD

    Steddon, Simon / Sharples, Edward

    Nephron Clinical Practice

    2011  Volume 118, Issue 1, Page(s) c145–c152

    Institution Consultant Nephrologist, Guy’s and St Thomas’ NHS Foundation Trust, London Consultant Nephrologist, Churchill Hospital, Oxford
    Keywords mineral bone disorders ; chronic kidney disease ; parathyroid hormone ; calcium ; phosphate ; phosphate binder
    Language English
    Publishing date 2011-05-06
    Publisher S. Karger AG
    Publishing place Basel, Switzerland
    Document type Article
    Note Renal Association Clinical Practice Guidelines 5th Edition 2009–2012
    ZDB-ID 207121-6
    ISBN 978-3-8055-9724-1 ; 3-8055-9724-X
    ISSN 1660-2110 ; 1423-0186 ; 2235-3186 ; 1660-8151 ; 0028-2766
    ISSN (online) 1660-2110 ; 1423-0186 ; 2235-3186
    ISSN 1660-8151 ; 0028-2766
    DOI 10.1159/000328066
    Database Karger publisher's database

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  6. Article ; Online: Calcimimetics and calcilytics--fooling the calcium receptor.

    Steddon, Simon J / Cunningham, John

    Lancet (London, England)

    2005  Volume 365, Issue 9478, Page(s) 2237–2239

    Abstract: Context: Just a decade after the the calcium-sensing receptor (CaR) was identified, pharmacological manipulation of the CaR is about to enter routine practice. For hyperparathyroid states, calcimimetics, which increase activation of the CaR, have been ... ...

    Abstract Context: Just a decade after the the calcium-sensing receptor (CaR) was identified, pharmacological manipulation of the CaR is about to enter routine practice. For hyperparathyroid states, calcimimetics, which increase activation of the CaR, have been licensed in Europe and the USA. Calcilytics, which decrease CaR function and increase secretion of parathyroid hormone (PTH), might allow the anabolic effects of PTH on bone to be harnessed for the prevention and treatment of osteoporosis.
    Starting point: In a multicentre randomised double-blind placebo-controlled study, Munro Peacock and colleagues recently confirmed the efficacy of the oral calcimimetic cinacalcet for achieving long-term reductions in serum calcium and PTH concentrations in primary hyperparathyroidism (J Clin Endocrinol Metab 2005; 90: 135-41). The arrival of a non-surgical option for this common disorder is important. WHAT NEXT? Study in primary and uraemic secondary hyperparathyroidism will indicate whether the efficacy of calcimimetic agents extends into the longer term. The extracellular relation between the CaR and its ligands and the intracellular signalling cascades that modify PTH gene transcription and secretion need further study. Drugs acting on the CaR might treat other disorders of bone remodelling, including osteoporosis. CaR expression in tissues beyond those involved in mineral ion homoeostasis should remain an important focus of research.
    MeSH term(s) Bone Diseases, Metabolic/drug therapy ; Calcium/agonists ; Calcium/metabolism ; Cinacalcet Hydrochloride ; Humans ; Hypercalcemia/drug therapy ; Hyperparathyroidism/drug therapy ; Hyperparathyroidism/metabolism ; Hyperparathyroidism, Secondary/drug therapy ; Hyperparathyroidism, Secondary/metabolism ; Naphthalenes/therapeutic use ; Parathyroid Hormone/secretion ; Receptors, Calcium-Sensing/drug effects ; Receptors, Calcium-Sensing/physiology
    Chemical Substances Naphthalenes ; Parathyroid Hormone ; Receptors, Calcium-Sensing ; Cinacalcet Hydrochloride (1K860WSG25) ; Calcium (SY7Q814VUP)
    Language English
    Publishing date 2005-06
    Publishing country England
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 3306-6
    ISSN 1474-547X ; 0023-7507 ; 0140-6736
    ISSN (online) 1474-547X
    ISSN 0023-7507 ; 0140-6736
    DOI 10.1016/S0140-6736(05)66782-7
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.MG 94: Show issues

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  7. Article: QUALITY IMPROVEMENT: Supporting a hospital in difficulty: -experience of a '-buddying' agreement to implement a new medical pathway.

    Leach, Richard / Banerjee, Sandip / Beer, Gail / Tencheva, Svetka / Conn, Deidre / Waterman, Ashley / Parrott, Jackie / Gifford, Julie / Steddon, Simon / Abbs, Ian / Pritchard, Amanda / Kerr, Ron / Dwyer, Lesley / Hamilton-Fairley, Diana

    Future healthcare journal

    2019  Volume 6, Issue 1, Page(s) 67–75

    Abstract: Increased NHS regulation has identified many healthcare organisations with operational and/or financial difficulties. Although the causes are often complex, most cases are effectively managed internally with limited input from external agencies. How best ...

    Abstract Increased NHS regulation has identified many healthcare organisations with operational and/or financial difficulties. Although the causes are often complex, most cases are effectively managed internally with limited input from external agencies. How best to support the few organisations needing additional support has not been established. 'Buddying', in which senior clinical and managerial teams from a well performing organisation work with colleagues from an organisation in difficulty has been proposed as a potential solution. Previous reports suggest that these partnerships are generally valued by the organisation in difficulty but there is a paucity of measured operational benefit. In this article we present our experience of a 'buddying agreement' and its impact on the introduction of a new 'whole system' medical pathway (ie rotas, staffing, process) at an organisation in difficulty. We describe the process, problems, effect on operational performance, staff survey feedback six months post-implementation and the lessons learned.  Factors critical to success were good communication; clear responsibilities, common values and strong governance; incorporation into an effective local improvement programme; targeting of specific issues; ability to influence people and foster relationships; adequate 'manpower' and gradual transition to local 'ownership'.
    Language English
    Publishing date 2019-05-15
    Publishing country England
    Document type Journal Article
    ZDB-ID 3016427-8
    ISSN 2514-6653 ; 2514-6645
    ISSN (online) 2514-6653
    ISSN 2514-6645
    DOI 10.7861/futurehosp.6-1-67
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  8. Article ; Online: Case report: making the CONN-ection: two cases of persistent hypertension and hypokalaemia following renal transplantation.

    Kumar, Atul / Hubbard, Johnathan / Moonim, Mufaddal / Steddon, Simon / Goldsmith, David

    International urology and nephrology

    2011  Volume 44, Issue 5, Page(s) 1577–1580

    Abstract: Conn's syndrome following renal transplantation is extremely rare. Here, we present two cases of persistent hypertension and hypokalemia, emerging after kidney transplantation, which proved resistant to medical treatment. In both patients, biochemical ... ...

    Abstract Conn's syndrome following renal transplantation is extremely rare. Here, we present two cases of persistent hypertension and hypokalemia, emerging after kidney transplantation, which proved resistant to medical treatment. In both patients, biochemical investigations elicited massively elevated plasma aldosterone levels and suppressed renin activity. Abdominal MRI demonstrated a 90- and 20-mm right adrenal tumour in cases 1 and 2, respectively. For both patients, curative treatment was achieved via laparoscopic right adrenalectomy.
    MeSH term(s) Adrenal Gland Neoplasms/complications ; Adrenal Gland Neoplasms/diagnosis ; Adrenal Gland Neoplasms/surgery ; Adult ; Aged ; Female ; Humans ; Hyperaldosteronism/etiology ; Kidney Transplantation/adverse effects ; Magnetic Resonance Imaging ; Male ; Tomography, X-Ray Computed
    Language English
    Publishing date 2011-03-04
    Publishing country Netherlands
    Document type Case Reports ; Journal Article
    ZDB-ID 204048-7
    ISSN 1573-2584 ; 0301-1623 ; 0042-1162
    ISSN (online) 1573-2584
    ISSN 0301-1623 ; 0042-1162
    DOI 10.1007/s11255-011-9924-y
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  9. Article ; Online: New prospects for the management of renal bone disease.

    Steddon, Simon J / Fan, Stanley L S / Cunningham, John

    Nephron. Clinical practice

    2005  Volume 99, Issue 1, Page(s) c1–7

    Abstract: The last decade has been a remarkably productive one in the field of bone biology. New insights into the maintenance of a normal bone microenvironment have led to significant advances in our understanding of many important skeletal disorders, including ... ...

    Abstract The last decade has been a remarkably productive one in the field of bone biology. New insights into the maintenance of a normal bone microenvironment have led to significant advances in our understanding of many important skeletal disorders, including renal osteodystrophy. Novel targets for therapeutic manipulation have been exposed and encouraging progress made towards new treatments. In addition, just as clinical studies have alerted us to the potential hazards of vascular calcification, basic science has unearthed the intimate nature of the relationship between the previously separate disciplines of bone and vascular biology. The clinical nephrologist, however, may be forgiven a little cynicism at this point. If such progress has been made, why do the same proverbial difficulties confront us in day-to-day practice? Control of phosphate remains inadequate, despite a literature which constantly reaffirms its crucial importance, and parathyroid hyperplasia seems inevitable in many patients. Furthermore, even the satisfaction of successful control of serum parathyroid hormone concentration must now be tempered by disquiet regarding the skeletal and cardiovascular consequences of oversuppression. This review aims to provide an update of the latest developments in relevant skeletal research and to assess how recently acquired knowledge may improve clinical nephrological practice over the next five years.
    MeSH term(s) Animals ; Bone Remodeling/physiology ; Calcium/blood ; Carrier Proteins/physiology ; Chronic Kidney Disease-Mineral and Bone Disorder/diagnostic imaging ; Chronic Kidney Disease-Mineral and Bone Disorder/physiopathology ; Chronic Kidney Disease-Mineral and Bone Disorder/therapy ; Cinacalcet Hydrochloride ; Humans ; Hyperparathyroidism/drug therapy ; Hyperplasia ; Membrane Glycoproteins/physiology ; Naphthalenes/therapeutic use ; Osteoclasts/physiology ; Parathyroid Glands/pathology ; Parathyroid Hormone/blood ; Parathyroidectomy ; RANK Ligand ; Radiography ; Receptor Activator of Nuclear Factor-kappa B ; Vitamin D/analogs & derivatives
    Chemical Substances Carrier Proteins ; Membrane Glycoproteins ; Naphthalenes ; Parathyroid Hormone ; RANK Ligand ; Receptor Activator of Nuclear Factor-kappa B ; TNFRSF11A protein, human ; TNFSF11 protein, human ; Vitamin D (1406-16-2) ; Cinacalcet Hydrochloride (1K860WSG25) ; Calcium (SY7Q814VUP)
    Language English
    Publishing date 2005
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 207121-6
    ISSN 1660-2110 ; 1423-0186 ; 2235-3186 ; 1660-8151 ; 0028-2766
    ISSN (online) 1660-2110 ; 1423-0186 ; 2235-3186
    ISSN 1660-8151 ; 0028-2766
    DOI 10.1159/000081787
    Database MEDical Literature Analysis and Retrieval System OnLINE

    Full text online

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  10. Article ; Online: New Prospects for the Management of Renal Bone Disease

    Steddon, Simon J. / Fan, Stanley L.S. / Cunningham, John

    Nephron Clinical Practice

    2005  Volume 99, Issue 1, Page(s) c1–c7

    Abstract: The last decade has been a remarkably productive one in the field of bone biology. New insights into the maintenance of a normal bone microenvironment have led to significant advances in our understanding of many important skeletal disorders, including ... ...

    Abstract The last decade has been a remarkably productive one in the field of bone biology. New insights into the maintenance of a normal bone microenvironment have led to significant advances in our understanding of many important skeletal disorders, including renal osteodystrophy. Novel targets for therapeutic manipulation have been exposed and encouraging progress made towards new treatments. In addition, just as clinical studies have alerted us to the potential hazards of vascular calcification, basic science has unearthed the intimate nature of the relationship between the previously separate disciplines of bone and vascular biology. The clinical nephrologist, however, may be forgiven a little cynicism at this point. If such progress has been made, why do the same proverbial difficulties confront us in day-to-day practice? Control of phosphate remains inadequate, despite a literature which constantly reaffirms its crucial importance, and parathyroid hyperplasia seems inevitable in many patients. Furthermore, even the satisfaction of successful control of serum parathyroid hormone concentration must now be tempered by disquiet regarding the skeletal and cardiovascular consequences of oversuppression. This review aims to provide an update of the latest developments in relevant skeletal research and to assess how recently acquired knowledge may improve clinical nephrological practice over the next five years.
    Keywords Osteodystrophy ; Adynamic bone disease ; Bone remodelling ; Calcium ; Phosphorus ; Vitamin D
    Language English
    Publisher S. Karger AG
    Publishing place Basel
    Publishing country Switzerland
    Document type Article ; Online
    ZDB-ID 207121-6
    ISSN 1660-2110 ; 1423-0186 ; 0028-2766 ; 1660-8151 ; 1660-2110 ; 0028-2766 ; 1660-8151
    ISSN (online) 1660-2110 ; 1423-0186
    ISSN 1660-2110 ; 0028-2766 ; 1660-8151
    DOI 10.1159/000081787
    Database Karger publisher's database

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