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  1. Article ; Online: miR-145 and incident thromboembolism.

    Zöller, Bengt

    Blood

    2024  Volume 143, Issue 17, Page(s) 1686–1687

    MeSH term(s) Female ; Humans ; Male ; Incidence ; MicroRNAs/genetics ; Thromboembolism/genetics ; Thromboembolism/etiology
    Chemical Substances MicroRNAs ; MIRN145 microRNA, human
    Language English
    Publishing date 2024-04-25
    Publishing country United States
    Document type Letter ; Research Support, Non-U.S. Gov't ; Research Support, N.I.H., Extramural ; Journal Article
    ZDB-ID 80069-7
    ISSN 1528-0020 ; 0006-4971
    ISSN (online) 1528-0020
    ISSN 0006-4971
    DOI 10.1182/blood.2023023798
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  2. Article ; Online: Genetics of venous thromboembolism revised.

    Zöller, Bengt

    Blood

    2019  Volume 134, Issue 19, Page(s) 1568–1570

    MeSH term(s) Factor V/genetics ; Genomics ; Humans ; Transcriptome ; Venous Thromboembolism
    Chemical Substances Factor V (9001-24-5)
    Language English
    Publishing date 2019-11-06
    Publishing country United States
    Document type Journal Article ; Comment
    ZDB-ID 80069-7
    ISSN 1528-0020 ; 0006-4971
    ISSN (online) 1528-0020
    ISSN 0006-4971
    DOI 10.1182/blood.2019002597
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  3. Article ; Online: Prevalence and in silico analysis of missense mutations in the PROS1 gene in the Swedish population: The SweGen dataset.

    Zöller, Bengt

    Thrombosis research

    2018  Volume 168, Page(s) 28–30

    MeSH term(s) Blood Proteins/genetics ; Humans ; Mutation, Missense ; Prevalence ; Protein S Deficiency/blood ; Protein S Deficiency/epidemiology ; Protein S Deficiency/genetics ; Sweden/epidemiology ; Venous Thromboembolism/blood ; Venous Thromboembolism/epidemiology ; Venous Thromboembolism/genetics
    Chemical Substances Blood Proteins ; PROS1 protein, human
    Language English
    Publishing date 2018-06-02
    Publishing country United States
    Document type Letter
    ZDB-ID 121852-9
    ISSN 1879-2472 ; 0049-3848
    ISSN (online) 1879-2472
    ISSN 0049-3848
    DOI 10.1016/j.thromres.2018.06.001
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    Zs.A 863: Show issues Location:
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  4. Article ; Online: The risk for venous thromboembolism and cardiometabolic disorders in offspring from thrombosis-prone pedigrees.

    Zöller, Bengt / Sundquist, Jan / Sundquist, Kristina / Ohlsson, Henrik

    Journal of thrombosis and haemostasis : JTH

    2023  Volume 22, Issue 3, Page(s) 775–784

    Abstract: Background: Most family studies on venous thromboembolism (VTE) have focused on first-degree relatives.: Objectives: We took a pedigree-based approach and examined the risk of VTE and cardiometabolic disorders in offspring from extended pedigrees ... ...

    Abstract Background: Most family studies on venous thromboembolism (VTE) have focused on first-degree relatives.
    Objectives: We took a pedigree-based approach and examined the risk of VTE and cardiometabolic disorders in offspring from extended pedigrees according to the densities of VTE in pedigrees.
    Methods: From the Swedish population, we identified a total of 482 185 pedigrees containing a mean of 14.2 parents, aunts/uncles, grandparents, and cousins of a core full sibship that we termed the pedigree offspring (n = 751 060). We then derived 8 empirical classes of these pedigrees based on the density of cases of VTE. The risk was determined in offspring for VTE and cardiometabolic disorders as a function of VTE density in their pedigrees. Bonferroni correction for multiple comparisons was performed.
    Results: VTE was unevenly distributed in the population; the Gini coefficient was 0.59. Higher VTE density in pedigrees was associated in the offspring with a higher risk of different VTE manifestations (deep venous thrombosis, pulmonary embolism, pregnancy-related VTE, unusual thrombosis, and superficial thrombophlebitis), thrombophilia, and lower age of first VTE event. Moreover, VTE density in pedigrees was significantly associated in the offspring with obesity, diabetes, gout, varicose veins, and arterial embolism and thrombosis (excluding brain and heart). No significant associations were observed for retinal vein occlusion, hypercholesterolemia, hypertension, coronary heart disease, myocardial infarction, ischemic stroke, atrial fibrillation, heart failure, primary pulmonary hypertension, cerebral hemorrhage, aortic aneurysm, peripheral artery disease, and overall mortality.
    Conclusion: Offspring of pedigrees with a high density of VTE are disadvantaged regarding VTE manifestations and certain cardiometabolic disorders.
    MeSH term(s) Humans ; Venous Thromboembolism/diagnosis ; Venous Thromboembolism/epidemiology ; Venous Thromboembolism/genetics ; Pedigree ; Risk Factors ; Thrombophlebitis ; Pulmonary Embolism/epidemiology ; Pulmonary Embolism/genetics
    Language English
    Publishing date 2023-12-09
    Publishing country England
    Document type Journal Article
    ZDB-ID 2112661-6
    ISSN 1538-7836 ; 1538-7933
    ISSN (online) 1538-7836
    ISSN 1538-7933
    DOI 10.1016/j.jtha.2023.11.024
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    Zs.A 5805: Show issues Location:
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    Zs.MO 295: Show issues

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  5. Book ; Thesis: Familial thrombophilia

    Zöller, Bengt

    resistance to activated protein C and protein S deficiency

    1996  

    Author's details av Bengt Zöller
    Language English
    Size 141 S. : Ill., graph. Darst.
    Publishing place Malmö
    Publishing country Sweden
    Document type Book ; Thesis
    Thesis / German Habilitation thesis Lund, Univ., Diss., 1996
    HBZ-ID HT006843694
    Database Catalogue ZB MED Medicine, Health

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    Shelf markLoan statusLocation
    96 E 70 order for loan Magazin

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  6. Article ; Online: Heritability of Atrial Fibrillation Among Swedish Adoptees.

    Zöller, Bengt / Rosengren, Per / PirouziFard, MirNabi / Sundquist, Jan / Sundquist, Kristina

    Circulation. Genomic and precision medicine

    2024  , Page(s) e004563

    Language English
    Publishing date 2024-05-02
    Publishing country United States
    Document type Letter
    ISSN 2574-8300
    ISSN (online) 2574-8300
    DOI 10.1161/CIRCGEN.124.004563
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  7. Article ; Online: Multimorbidity disease clusters are associated with venous thromboembolism: an extended cross-sectional national study.

    Ahrén, Jonatan / Pirouzifard, MirNabi / Holmquist, Björn / Sundquist, Jan / Sundquist, Kristina / Zöller, Bengt

    Journal of thrombosis and thrombolysis

    2024  

    Abstract: Multimorbidity, i.e., two or more non-communicable diseases (NCDs), is an escalating challenge for society. Venous thromboembolism (VTE) is a common cardiovascular disease and it is unknown which multimorbidity clusters associates with VTE. Our aim was ... ...

    Abstract Multimorbidity, i.e., two or more non-communicable diseases (NCDs), is an escalating challenge for society. Venous thromboembolism (VTE) is a common cardiovascular disease and it is unknown which multimorbidity clusters associates with VTE. Our aim was to examine the association between different common disease clusters of multimorbidity and VTE. The study is an extended (1997-2015) cross-sectional Swedish study using the National Patient Register and the Multigeneration Register. A total of 2,694,442 Swedish-born individuals were included in the study. Multimorbidity was defined by 45 NCDs. A principal component analysis (PCA) identified multimorbidity disease clusters. Odds ratios (OR) for VTE were calculated for the different multimorbidity disease clusters. There were 16% (n = 440,742) of multimorbid individuals in the study population. Forty-four of the individual 45 NCDs were associated with VTE. The PCA analysis identified nine multimorbidity disease clusters, F1-F9. Seven of these multimorbidity clusters were associated with VTE. The adjusted OR for VTE in the multimorbid patients was for the first three clusters: F1 (cardiometabolic diseases) 3.44 (95%CI 3.24-3.65), F2 (mental disorders) 2.25 (95%CI 2.14-2.37) and F3 (digestive system diseases) 4.35 (95%CI 3.63-5.22). There was an association between multimorbidity severity and OR for VTE. For instance, the occurrence of at least five diseases was in F1 and F2 associated with ORs for VTE: 8.17 (95%CI 6.32-10.55) and 6.31 (95%CI 4.34-9.17), respectively. In this nationwide study we have shown a strong association between VTE and different multimorbidity disease clusters that might be useful for VTE prediction.
    Language English
    Publishing date 2024-04-27
    Publishing country Netherlands
    Document type Journal Article
    ZDB-ID 1230645-9
    ISSN 1573-742X ; 0929-5305
    ISSN (online) 1573-742X
    ISSN 0929-5305
    DOI 10.1007/s11239-024-02987-y
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    Zs.A 4352: Show issues Location:
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  8. Article ; Online: Rare-variant collapsing and bioinformatic analyses for different types of cardiac arrhythmias in the UK Biobank reveal novel susceptibility loci and candidate amyloid-forming proteins.

    Zöller, Bengt / Manderstedt, Eric / Lind-Halldén, Christina / Halldén, Christer

    Cardiovascular digital health journal

    2023  Volume 5, Issue 1, Page(s) 15–18

    Abstract: Background: Cardiac arrhythmias are a common health problem. Both common and rare genetic risk factors exist for cardiac arrhythmias. Cardiac amyloidosis is a rare disease that may manifest various arrhythmias. Few large-scale whole exome sequencing ... ...

    Abstract Background: Cardiac arrhythmias are a common health problem. Both common and rare genetic risk factors exist for cardiac arrhythmias. Cardiac amyloidosis is a rare disease that may manifest various arrhythmias. Few large-scale whole exome sequencing studies elucidating the contribution of rare variations to arrhythmias have been published.
    Objective: To access gene collapsing analysis of rare variations for different types of cardiac arrhythmias in UK Biobank. Identified genes were analyzed
    Methods: We used 2 published UK Biobank portals (https://azphewas.com/ and https://app.genebass.org/) to access gene collapsing analysis of rare variations for different types of cardiac arrhythmias. Diagnosis of arrhythmia was based on the International Classification of Diseases, 10th Revision (ICD-10) codes: conduction disorders (I44, I45), paroxysmal tachycardia (I47), atrial fibrillation (I48), and other arrhythmias (I49).
    Results: Rare variations in 5 genes were linked to conduction disorders (
    Conclusion: Rare variations in 13 genes were associated with arrhythmias in the UK Biobank. Two of the heart conduction disorder-linked genes are potential amyloid-forming candidates. Amyloid formation may be an underestimated cause of heart conduction disorders.
    Language English
    Publishing date 2023-12-23
    Publishing country United States
    Document type Journal Article
    ISSN 2666-6936
    ISSN (online) 2666-6936
    DOI 10.1016/j.cvdhj.2023.12.001
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  9. Article ; Online: Rare-variant collapsing analyses of arterial hypertension in the UK biobank.

    Zöller, Bengt / Manderstedt, Eric / Lind-Halldén, Christina / Halldén, Christer

    Journal of human hypertension

    2023  Volume 37, Issue 11, Page(s) 1040–1042

    MeSH term(s) Humans ; Biological Specimen Banks ; Hypertension/diagnosis ; Hypertension/epidemiology ; United Kingdom/epidemiology
    Language English
    Publishing date 2023-04-14
    Publishing country England
    Document type Journal Article
    ZDB-ID 639472-3
    ISSN 1476-5527 ; 0950-9240
    ISSN (online) 1476-5527
    ISSN 0950-9240
    DOI 10.1038/s41371-023-00829-7
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  10. Article ; Online: Contribution of rare genetic variants to heart failure and cardiomyopathy in the UK Biobank.

    Zöller, Bengt / Manderstedt, Eric / Lind-Halldén, Christina / Halldén, Christer

    ESC heart failure

    2023  Volume 10, Issue 5, Page(s) 3234–3235

    Language English
    Publishing date 2023-09-04
    Publishing country England
    Document type Letter
    ZDB-ID 2814355-3
    ISSN 2055-5822 ; 2055-5822
    ISSN (online) 2055-5822
    ISSN 2055-5822
    DOI 10.1002/ehf2.14490
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