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Article ; Online: Roles of HIF-1α signaling in Mycobacterium tuberculosis infection: New targets for anti-TB therapeutics?

Li, Chaowei / Wang, Jiajun / Xu, Jun-Fa / Pi, Jiang / Zheng, Biying

Biochemical and biophysical research communications

2024 Volume 711, Page(s) 149920

Abstract: Tuberculosis (TB), a deadly infectious disease induced by Mycobacterium tuberculosis (Mtb), continues to be a global public health issue that kill millions of patents every year. Despite significant efforts have been paid to identify effective TB ... ...

Abstract	Tuberculosis (TB), a deadly infectious disease induced by Mycobacterium tuberculosis (Mtb), continues to be a global public health issue that kill millions of patents every year. Despite significant efforts have been paid to identify effective TB treatments, the emergence of drug-resistant strains of the disease and the presence of comorbidities in TB patients urges us to explore the detailed mechanisms involved in TB immunity and develop more effective innovative anti-TB strategies. HIF-1α, a protein involved in regulating cellular immune responses during TB infection, has been highlighted as a promising target for the development of novel strategies for TB treatment due to its critical roles in anti-TB host immunity. This review provides a summary of current research progress on the roles of HIF-1α in TB infection, highlighting its importance in regulating the host immune response upon Mtb infection and summarizing the influences and mechanisms of HIF-1α on anti-TB immunological responses of host cells. This review also discusses the various challenges associated with developing HIF-1α as a target for anti-TB therapies, including ensuring specificity and avoiding off-target effects on normal cell function, determining the regulation and expression of HIF-1α in TB patients, and developing drugs that can inhibit HIF-1α. More deep understanding of the molecular mechanisms involved in HIF-1α signaling, its impact on TB host status, and systematic animal testing and clinical trials may benefit the optimization of HIF-1α as a novel therapeutic target for TB.
MeSH term(s)	Humans ; Hypoxia-Inducible Factor 1, alpha Subunit/metabolism ; Mycobacterium tuberculosis/drug effects ; Mycobacterium tuberculosis/metabolism ; Mycobacterium tuberculosis/immunology ; Signal Transduction/drug effects ; Tuberculosis/drug therapy ; Tuberculosis/immunology ; Tuberculosis/metabolism ; Tuberculosis/microbiology ; Animals ; Antitubercular Agents/therapeutic use ; Antitubercular Agents/pharmacology ; Molecular Targeted Therapy/methods
Chemical Substances	Hypoxia-Inducible Factor 1, alpha Subunit ; Antitubercular Agents ; HIF1A protein, human
Language	English
Publishing date	2024-04-09
Publishing country	United States
Document type	Journal Article ; Review ; Research Support, Non-U.S. Gov't
ZDB-ID	205723-2
ISSN	1090-2104 ; 0006-291X ; 0006-291X
ISSN (online)	1090-2104 ; 0006-291X
ISSN	0006-291X
DOI	10.1016/j.bbrc.2024.149920
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Article ; Online: Essential contribution of the JAK/STAT pathway to carcinogenesis, lytic infection of herpesviruses and pathogenesis of COVID‑19 (Review).

Li, Wenkai / Zhuang, Yunjing / Shao, Song-Jun / Trivedi, Pankaj / Zheng, Biying / Huang, Guo-Liang / He, Zhiwei / Zhang, Xiangning

Molecular medicine reports

2024 Volume 29, Issue 3

Abstract: The intracellular pathway of Janus kinase/signal transducer and activator of transcription (JAK/STAT) and modification of nucleosome histone marks regulate the expression of proinflammatory mediators, playing an essential role in carcinogenesis, ... ...

Abstract	The intracellular pathway of Janus kinase/signal transducer and activator of transcription (JAK/STAT) and modification of nucleosome histone marks regulate the expression of proinflammatory mediators, playing an essential role in carcinogenesis, antiviral immunity and the interaction of host proteins with Herpesviral particles. The pathway has also been suggested to play a vital role in the clinical course of the acute infection caused by severe acute respiratory syndrome coronavirus type 2 (SARS‑CoV‑2; known as coronavirus infection‑2019), a novel human coronavirus initially identified in the central Chinese city Wuhan towards the end of 2019, which evolved into a pandemic affecting nearly two million people worldwide. The infection mainly manifests as fever, cough, myalgia and pulmonary involvement, while it also attacks multiple viscera, such as the liver. The pathogenesis is characterized by a cytokine storm, with an overproduction of proinflammatory mediators. Innate and adaptive host immunity against the viral pathogen is exerted by various effectors and is regulated by different signaling pathways notably the JAK/STAT. The elucidation of the underlying mechanism of the regulation of mediating factors expressed in the viral infection would assist diagnosis and antiviral targeting therapy, which will help overcome the infection caused by SARS‑CoV‑2.
MeSH term(s)	Humans ; Carcinogenesis ; COVID-19 ; Herpesviridae/metabolism ; Janus Kinases/metabolism ; SARS-CoV-2/metabolism ; Signal Transduction/physiology ; STAT Transcription Factors/metabolism
Chemical Substances	Janus Kinases (EC 2.7.10.2) ; STAT Transcription Factors
Language	English
Publishing date	2024-01-19
Publishing country	Greece
Document type	Journal Article ; Review
ZDB-ID	2469505-1
ISSN	1791-3004 ; 1791-2997
ISSN (online)	1791-3004
ISSN	1791-2997
DOI	10.3892/mmr.2024.13163
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Article: Ferroptosis: A mixed blessing for infectious diseases.

Xiao, Leyao / Huang, Huanshao / Fan, Shuhao / Zheng, Biying / Wu, Jianguo / Zhang, Junai / Pi, Jiang / Xu, Jun-Fa

Frontiers in pharmacology

2022 Volume 13, Page(s) 992734

Abstract: To date, it has been confirmed that the occurrence and development of infectious diseases are tightly associated with regulatory cell death processes, such as apoptosis, autophagy, and necroptosis. Ferroptosis, as a newly discovered form of regulatory ... ...

Abstract	To date, it has been confirmed that the occurrence and development of infectious diseases are tightly associated with regulatory cell death processes, such as apoptosis, autophagy, and necroptosis. Ferroptosis, as a newly discovered form of regulatory cell death characterized by iron-dependent lipid peroxidation, is not only closely associated with tumor progression, but is also found to be tightly related to the regulation of infectious diseases, such as Tuberculosis, Cryptococcal meningitis, Malaria and COVID-2019. The emerging critical roles of ferroptosis that has been found in infectious disease highlight ferroptosis as a potential therapeutic target in this field, which is therefore widely expected to be developed into new therapy strategy against infectious diseases. Here, we summarized the underlying mechanisms of ferroptosis and highlighted the intersections between host immunity and ferroptosis. Moreover, we illuminated the roles of ferroptosis in the occurrence and progression of different infectious diseases, which might provide some unique inspiration and thought-provoking perspectives for the future research of these infectious diseases, especially for the development of ferroptosis-based therapy strategy against infectious diseases.
Language	English
Publishing date	2022-09-07
Publishing country	Switzerland
Document type	Journal Article ; Review
ZDB-ID	2587355-6
ISSN	1663-9812
ISSN	1663-9812
DOI	10.3389/fphar.2022.992734
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Article: Regulation of the expression of proinflammatory cytokines induced by SARS-CoV-2.

Zhang, Xiang-Ning / Wu, Long-Ji / Kong, Xia / Zheng, Bi-Ying / Zhang, Zhe / He, Zhi-Wei

World journal of clinical cases

2020 Volume 9, Issue 7, Page(s) 1513–1523

Abstract: An outbreak of a novel coronavirus was reported in Wuhan, China, in late 2019. It has spread rapidly through China and many other countries, causing a global pandemic. Since February 2020, over 28 countries/regions have reported confirmed cases. ... ...

Abstract	An outbreak of a novel coronavirus was reported in Wuhan, China, in late 2019. It has spread rapidly through China and many other countries, causing a global pandemic. Since February 2020, over 28 countries/regions have reported confirmed cases. Individuals with the infection known as coronavirus disease-19 (COVID-19) have similar clinical features as severe acute respiratory syndrome first encountered 17 years ago, with fever, cough, and upper airway congestion, along with high production of proinflammatory cytokines (PICs), which form a cytokine storm. PICs induced by COVID-19 include interleukin (IL)-6, IL-17, and monocyte chemoattractant protein-1. The production of cytokines is regulated by activated nuclear factor-kB and involves downstream pathways such as Janus kinase/signal transducers and activators transcription. Protein expression is also regulated by post-translational modification of chromosomal markers. Lysine residues in the peptide tails stretching out from the core of histones bind the sequence upstream of the coding portion of genomic DNA. Covalent modification, particularly methylation, activates or represses gene transcription. PICs have been reported to be induced by histone modification and stimulate exudation of hyaluronic acid, which is implicated in the occurrence of COVID-19. These findings indicate the impact of the expression of PICs on the pathogenesis and therapeutic targeting of COVID-19.
Language	English
Publishing date	2020-11-10
Publishing country	United States
Document type	Journal Article ; Review
ISSN	2307-8960
ISSN	2307-8960
DOI	10.12998/wjcc.v9.i7.1513
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Article ; Online: Immunomodulatory activity of manganese dioxide nanoparticles: Promising for novel vaccines and immunotherapeutics.

Huang, Yuhe / Ruan, Yongdui / Ma, Yuhe / Chen, Dongsheng / Zhang, Tangxin / Fan, Shuhao / Lin, Wensen / Huang, Yifan / Lu, Hongmei / Xu, Jun-Fa / Pi, Jiang / Zheng, Biying

Frontiers in immunology

2023 Volume 14, Page(s) 1128840

Abstract: Manganese (Mn), a nutrient inorganic trace element, is necessary for a variety of physiological processes of animal body due to their important roles in oxidative regulation effects and other aspects of activities. Moreover, manganese ion ( ... ...

Abstract	Manganese (Mn), a nutrient inorganic trace element, is necessary for a variety of physiological processes of animal body due to their important roles in oxidative regulation effects and other aspects of activities. Moreover, manganese ion (Mn
MeSH term(s)	Animals ; Manganese Compounds/pharmacology ; Manganese Compounds/metabolism ; Manganese ; Oxides/pharmacology ; Hydrogen Peroxide/metabolism ; Nanoparticles/metabolism ; Oxygen ; Vaccines ; Immunotherapy
Chemical Substances	manganese dioxide (TF219GU161) ; Manganese Compounds ; Manganese (42Z2K6ZL8P) ; Oxides ; Hydrogen Peroxide (BBX060AN9V) ; Oxygen (S88TT14065) ; Vaccines
Language	English
Publishing date	2023-02-28
Publishing country	Switzerland
Document type	Journal Article ; Review ; Research Support, Non-U.S. Gov't
ZDB-ID	2606827-8
ISSN	1664-3224 ; 1664-3224
ISSN (online)	1664-3224
ISSN	1664-3224
DOI	10.3389/fimmu.2023.1128840
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Article: Implication of viral microRNAs in the genesis and diagnosis of Epstein-Barr virus-associated tumors.

Zhang, Xiangning / Ye, Yudong / Fu, Ming / Zheng, Biying / Qiu, Qianhui / Huang, Zunnan

Oncology letters

2019 Volume 18, Issue 4, Page(s) 3433–3442

Abstract: The Epstein-Barr virus (EBV) is tightly associated with a variety of human tumors, including Burkitt lymphoma and acquired immune deficiency syndrome-related lymphoma of B-cell origin, as well as nasopharyngeal carcinoma and gastric cancer of epithelial ... ...

Abstract	The Epstein-Barr virus (EBV) is tightly associated with a variety of human tumors, including Burkitt lymphoma and acquired immune deficiency syndrome-related lymphoma of B-cell origin, as well as nasopharyngeal carcinoma and gastric cancer of epithelial origin. The virus latently infects the host cells and expresses proteins and non-coding RNAs to achieve malignancy. MicroRNAs (miRNAs or miRs) are small RNAs consisting of 19-25 nucleotides, which directly bind to the 3'-untranslated region of mRNAs to promote degradation and inhibit translation of mRNAs. EBV-encoded miRs are generated from two regions of the viral genome, within the apoptosis regulator BHRF1 gene locus and near the
Language	English
Publishing date	2019-08-05
Publishing country	Greece
Document type	Journal Article ; Review
ZDB-ID	2573196-8
ISSN	1792-1082 ; 1792-1074
ISSN (online)	1792-1082
ISSN	1792-1074
DOI	10.3892/ol.2019.10713
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Article ; Online: Engineering zinc oxide hybrid selenium nanoparticles for synergetic anti-tuberculosis treatment by combining Mycobacterium tuberculosis killings and host cell immunological inhibition.

Lin, Wensen / Fan, Shuhao / Liao, Kangsheng / Huang, Yifan / Cong, Yanguang / Zhang, Junai / Jin, Hua / Zhao, Yi / Ruan, Yongdui / Lu, Hongmei / Yang, Fen / Wu, Changxian / Zhao, Daina / Fu, Zhendong / Zheng, Biying / Xu, Jun-Fa / Pi, Jiang

Frontiers in cellular and infection microbiology

2023 Volume 12, Page(s) 1074533

Abstract: Introduction: As a deadly disease induced by Mycobacterium tuberculosis (Mtb), tuberculosis remains one of the top killers among infectious diseases. The low intracellular Mtb killing efficiency of current antibiotics introduced the long duration anti- ... ...

Abstract	Introduction: As a deadly disease induced by Mycobacterium tuberculosis (Mtb), tuberculosis remains one of the top killers among infectious diseases. The low intracellular Mtb killing efficiency of current antibiotics introduced the long duration anti-TB therapy in clinic with strong side effects and increased drug-resistant mutants. Therefore, the exploration of novel anti-TB agents with potent anti-TB efficiency becomes one of the most urgent issues for TB therapies. Methods: Here, we firstly introduced a novel method for the preparation of zinc oxide-selenium nanoparticles (ZnO-Se NPs) by the hybridization of zinc oxide and selenium to combine the anti-TB activities of zinc oxide nanoparticles and selenium nanoparticles. We characterized the ZnO-Se NPs by dynamic laser light scattering and transmission electron microscopy, and then tested the inhibition effects of ZnO-Se NPs on extracellular Mtb by colony-forming units (CFU) counting, bacterial ATP analysis, bacterial membrane potential analysis and scanning electron microscopy imaging. We also analyzed the effects of ZnO-Se NPs on the ROS production, mitochondrial membrane potential, apoptosis, autophagy, polarization and PI3K/Akt/mTOR signaling pathway of Mtb infected THP-1 macrophages. At last, we also tested the effects of ZnO-Se NPs on intracellular Mtb in THP-1 cells by colony-forming units (CFU) counting. Results: The obtained spherical core-shell ZnO-Se NPs with average diameters of 90 nm showed strong killing effects against extracellular Mtb, including BCG and the virulent H37Rv, by disrupting the ATP production, increasing the intracellular ROS level and destroying the membrane structures. More importantly, ZnO-Se NPs could also inhibit intracellular Mtb growth by promoting M1 polarization to increase the production of antiseptic nitric oxide and also promote apoptosis and autophagy of Mtb infected macrophages by increasing the intracellular ROS, disrupting mitochondria membrane potential and inhibiting PI3K/Akt/mTOR signaling pathway. Discussion: These ZnO-Se NPs with synergetic anti-TB efficiency by combining the Mtb killing effects and host cell immunological inhibition effects were expected to serve as novel anti-TB agents for the development of more effective anti-TB strategy.
MeSH term(s)	Adenosine Triphosphate ; Antitubercular Agents/pharmacology ; Mycobacterium tuberculosis/drug effects ; Nanoparticles/chemistry ; Phosphatidylinositol 3-Kinases ; Proto-Oncogene Proteins c-akt ; Reactive Oxygen Species ; Selenium/pharmacology ; TOR Serine-Threonine Kinases ; Zinc Oxide/pharmacology ; Zinc Oxide/chemistry
Chemical Substances	Adenosine Triphosphate (8L70Q75FXE) ; Antitubercular Agents ; Phosphatidylinositol 3-Kinases (EC 2.7.1.-) ; Proto-Oncogene Proteins c-akt (EC 2.7.11.1) ; Reactive Oxygen Species ; Selenium (H6241UJ22B) ; TOR Serine-Threonine Kinases (EC 2.7.11.1) ; Zinc Oxide (SOI2LOH54Z)
Language	English
Publishing date	2023-01-26
Publishing country	Switzerland
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2619676-1
ISSN	2235-2988 ; 2235-2988
ISSN (online)	2235-2988
ISSN	2235-2988
DOI	10.3389/fcimb.2022.1074533
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Article ; Online: Elevated Interleukin-37 Associated with Dengue Viral Load in Patients with Dengue Fever.

Zhang, Jun-Ai / Wang, Jia-Jun / Zhang, Wen-Ting / Zhang, Li / Zheng, Bi-Ying / Liu, Gan-Bin / Liang, Jing / Lu, Yuan-Bin / Wu, Xian-Jin / Yao, Shu-Ying / Chen, Guo-Ying / Xie, Yun-Qi / Wu, Jun-Yi / Shi, Jia-Hua / Pi, Jiang / Li, Si-Ping / Xu, Jun-Fa

Current microbiology

2023 Volume 80, Issue 5, Page(s) 171

Abstract: Dengue remains a public health issue worldwide. Similar to chronic infectious diseases, stimulation of cytokine production is not enough to drive immune effector cells for effective virus clearance. One possible mechanism is the virus induces a large ... ...

Abstract	Dengue remains a public health issue worldwide. Similar to chronic infectious diseases, stimulation of cytokine production is not enough to drive immune effector cells for effective virus clearance. One possible mechanism is the virus induces a large number of negative stimulatory cytokines inhibiting immune response. Interleukin 37 (IL-37) plays a crucial regulatory role in infection and immunity, inhibits innate and adaptive immunity as an anti-inflammatory cytokine by inhibiting proinflammatory mediators and pathways. To date, there are few studies reporting correlations between dengue fever (DF) and IL-37. In this study we found that the serum IL-37b and IL-37b-producing monocytes in patients were significantly increased in DF patients. A majority of the IL-37b produced by DF patients was produced by monocytes, not lymphocytes. Increased levels of IL-6, IL-10, and IFN-α were also found in DF patients. However, we failed to detect IL-1β, IL-17A and TNF-α in plasma, because of off-target. In our study, there was no relation between IL-6, IL-10, and IFN-α expressions and IL-37b in serum (P > 0.05). The IL-37b-producing monocytes were negatively correlated with the level of IFN-α in serum and platelet count, and positively correlated with lymphocytes percentage (P < 0.05, respectively). Additionally, serum DENV nonstructural protein 1 levels were positively correlated with monocytes percentages (P < 0.05). Our data represents findings for IL-37b expression and its potential mechanisms in DF patients' immune response.
MeSH term(s)	Humans ; Interleukin-10 ; Dengue ; Dengue Virus/physiology ; Interleukin-6 ; Viral Load ; Cytokines
Chemical Substances	Interleukin-10 (130068-27-8) ; Interleukin-6 ; Cytokines
Language	English
Publishing date	2023-04-06
Publishing country	United States
Document type	Journal Article
ZDB-ID	134238-1
ISSN	1432-0991 ; 0343-8651
ISSN (online)	1432-0991
ISSN	0343-8651
DOI	10.1007/s00284-023-03239-7
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Article ; Online: LincRNA-p21: Implications in Human Diseases.

Tang, Sai-Sai / Zheng, Bi-Ying / Xiong, Xing-Dong

International journal of molecular sciences

2015 Volume 16, Issue 8, Page(s) 18732–18740

Abstract: Long noncoding RNAs (lncRNAs), which lack significant protein-coding capacity, regulate various biological processes through diverse and as yet poorly understood molecular mechanisms. However, a number of studies in the past few years have documented ... ...

Abstract	Long noncoding RNAs (lncRNAs), which lack significant protein-coding capacity, regulate various biological processes through diverse and as yet poorly understood molecular mechanisms. However, a number of studies in the past few years have documented important functions for lncRNAs in human diseases. Among these lncRNAs, lincRNA-p21 has been proposed to be a novel regulator of cell proliferation, apoptosis and DNA damage response, and involved in the initiation and progression of human diseases. In this review, we summarize the current knowledge of lincRNA-p21, mainly focus on the known biological functions and its underlying mechanisms. Moreover, we highlight the growing body of evidences for the importance of lincRNA-p21 in diverse human diseases, which indicate lincRNA-p21 as a potential diagnostic marker and/or a valuable therapeutic target for these diseases.
MeSH term(s)	Animals ; Apoptosis/genetics ; Cellular Reprogramming/genetics ; DNA Damage ; Gene Expression Regulation ; Genetic Predisposition to Disease ; Humans ; RNA, Long Noncoding/genetics ; RNA, Long Noncoding/metabolism
Chemical Substances	RNA, Long Noncoding
Language	English
Publishing date	2015-08-11
Publishing country	Switzerland
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	2019364-6
ISSN	1422-0067 ; 1422-0067 ; 1661-6596
ISSN (online)	1422-0067
ISSN	1422-0067 ; 1661-6596
DOI	10.3390/ijms160818732
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Article: Role of B7-H4 in the Progression and Prognosis of Cervical Inflammation to Cancer After Human Papilloma Virus Infection.

Qiu, Feng / Yuan, Chunlei / Xu, Jianhua / Zhang, Junai / Chen, Fu / Liu, Dongdong / Zheng, Biying / Li, Ruixi / Huang, Xianzhang / Xu, Junfa

Journal of biomedical nanotechnology

2019 Volume 15, Issue 5, Page(s) 1043–1051

Abstract: The immune checkpoint molecule B7-H4 is highly expressed in various types of cancer, but little is known about its role in the development of cervical lesions associated with human papilloma virus (HPV). This study aimed to investigate the role of B7-H4 ... ...

Abstract	The immune checkpoint molecule B7-H4 is highly expressed in various types of cancer, but little is known about its role in the development of cervical lesions associated with human papilloma virus (HPV). This study aimed to investigate the role of B7-H4 in cervical lesion progression and the relationship between B7-H4 and HPV infection. This was a retrospective study of tissue samples from 30 patients with cervical cancer, 28 with cervical intraepithelial neoplasia (CIN), and 75 with cervical inflammatory lesions. Serum-soluble B7-H4 (sB7-H4) was assessed by ELISA. Tissue B7-H4 expression was measured by RT-PCR and immunochemistry. Expression of B7-H4 in dendritic cells was assessed by flow cytometry. sB7H4 increased gradually from the patients with cervical inflammation to patients with cervical cancer and decreased after treatment. sB7-H4 had a diagnostic value in cervical intraepithelial neoplasia (CIN (area under the curve (AUC) = 0.8929) and cervical cancer (AUC = 0.9545). B7-H4 expression in inflammatory cervical tissue and peripheral blood dendritic cells (DCs) increased gradually from inflammation to cancer (
MeSH term(s)	Female ; Humans ; Inflammation ; Papillomaviridae ; Papillomavirus Infections ; Prognosis ; Retrospective Studies ; Uterine Cervical Diseases ; V-Set Domain-Containing T-Cell Activation Inhibitor 1
Chemical Substances	V-Set Domain-Containing T-Cell Activation Inhibitor 1
Language	English
Publishing date	2019-04-02
Publishing country	United States
Document type	Journal Article
ISSN	1550-7033
ISSN	1550-7033
DOI	10.1166/jbn.2019.2741
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