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  1. Artikel ; Online: Association between Inflammation and Function of Cell Adhesion Molecules Influence on Gastrointestinal Cancer Development.

    Huang, Hsiang-Wei / Chang, Cheng-Chih / Wang, Chia-Siu / Lin, Kwang-Huei

    Cells

    2021  Band 10, Heft 1

    Abstract: Gastrointestinal cancer is highly associated with inflammatory processes inducing the release of cytokines from cancer or immune cells, including interferons, interleukins, chemokines, colony-stimulating factors, and growth factors, which promote or ... ...

    Abstract Gastrointestinal cancer is highly associated with inflammatory processes inducing the release of cytokines from cancer or immune cells, including interferons, interleukins, chemokines, colony-stimulating factors, and growth factors, which promote or suppress tumor progression. Inflammatory cytokines within the tumor microenvironment promote immune cell infiltration. Infiltrating immune, and tumor-surrounding stromal cells support tumor growth, angiogenesis, metastasis, and immunosuppression through communication with inflammatory cytokines and cell adhesion molecules. Notably, infiltrating immune and tumor cells present immunosuppressive molecules, such as programmed death-ligand 1 (PD-L1) and CD80/CD86. Suppression of cytotoxic T cells promotes tumor avoidance of immune surveillance and greater malignancy. Moreover, glycosylation and sialylation of proteins hyperexpressed on the cancer cell surface have been shown to enhance immune escape and metastasis. Cytokine treatments and immune checkpoint inhibitors are widely used in clinical practice. However, the tumor microenvironment is a rapidly changing milieu involving several factors. In this review, we have provided a summary of the interactions of inflammation and cell adhesion molecules between cancer and other cell types, to improve understanding of the tumor microenvironment.
    Mesh-Begriff(e) Animals ; Carcinogenesis/metabolism ; Carcinogenesis/pathology ; Cell Adhesion Molecules/metabolism ; Cell Communication ; Gastrointestinal Neoplasms/metabolism ; Gastrointestinal Neoplasms/pathology ; Gastrointestinal Neoplasms/therapy ; Humans ; Inflammation/metabolism ; Tumor Microenvironment
    Chemische Substanzen Cell Adhesion Molecules
    Sprache Englisch
    Erscheinungsdatum 2021-01-04
    Erscheinungsland Switzerland
    Dokumenttyp Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 2661518-6
    ISSN 2073-4409 ; 2073-4409
    ISSN (online) 2073-4409
    ISSN 2073-4409
    DOI 10.3390/cells10010067
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  2. Artikel ; Online: Association between Inflammation and Function of Cell Adhesion Molecules Influence on Gastrointestinal Cancer Development

    Hsiang-Wei Huang / Cheng-Chih Chang / Chia-Siu Wang / Kwang-Huei Lin

    Cells, Vol 10, Iss 67, p

    2021  Band 67

    Abstract: Gastrointestinal cancer is highly associated with inflammatory processes inducing the release of cytokines from cancer or immune cells, including interferons, interleukins, chemokines, colony-stimulating factors, and growth factors, which promote or ... ...

    Abstract Gastrointestinal cancer is highly associated with inflammatory processes inducing the release of cytokines from cancer or immune cells, including interferons, interleukins, chemokines, colony-stimulating factors, and growth factors, which promote or suppress tumor progression. Inflammatory cytokines within the tumor microenvironment promote immune cell infiltration. Infiltrating immune, and tumor-surrounding stromal cells support tumor growth, angiogenesis, metastasis, and immunosuppression through communication with inflammatory cytokines and cell adhesion molecules. Notably, infiltrating immune and tumor cells present immunosuppressive molecules, such as programmed death-ligand 1 (PD-L1) and CD80/CD86. Suppression of cytotoxic T cells promotes tumor avoidance of immune surveillance and greater malignancy. Moreover, glycosylation and sialylation of proteins hyperexpressed on the cancer cell surface have been shown to enhance immune escape and metastasis. Cytokine treatments and immune checkpoint inhibitors are widely used in clinical practice. However, the tumor microenvironment is a rapidly changing milieu involving several factors. In this review, we have provided a summary of the interactions of inflammation and cell adhesion molecules between cancer and other cell types, to improve understanding of the tumor microenvironment.
    Schlagwörter gastrointestinal cancer ; inflammation ; tumor microenvironment ; cell adhesion molecule ; Biology (General) ; QH301-705.5
    Thema/Rubrik (Code) 610
    Sprache Englisch
    Erscheinungsdatum 2021-01-01T00:00:00Z
    Verlag MDPI AG
    Dokumenttyp Artikel ; Online
    Datenquelle BASE - Bielefeld Academic Search Engine (Lebenswissenschaftliche Auswahl)

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  3. Artikel ; Online: Functional roles of non-coding RNAs regulated by thyroid hormones in liver cancer

    Po-Shuan Huang / Cheng-Chih Chang / Chia-Siu Wang / Kwang-Huei Lin

    Biomedical Journal, Vol 44, Iss 3, Pp 272-

    2021  Band 284

    Abstract: Recent reports have shown the important role of the non-coding part of human genome RNA (ncRNA) in cancer formation and progression. Among several kinds of ncRNAs, microRNAs (miRNA) play a pivotal role in cancer biology. Accumulating researches have been ...

    Abstract Recent reports have shown the important role of the non-coding part of human genome RNA (ncRNA) in cancer formation and progression. Among several kinds of ncRNAs, microRNAs (miRNA) play a pivotal role in cancer biology. Accumulating researches have been focused on the importance of non-coding genes in various diseases. In addition to miRNAs, long non-coding RNAs (lncRNAs) have also been extensively documented. Recently, the study of human liver cancer has gradually shifted to these non-coding RNAs that were originally considered “junk”. Notably, dysregulated ncRNAs maybe influence on cell proliferation, angiogenesis, anti-apoptosis, and metastasis. Thyroid hormones play critical roles in human development and abnormalities in thyroid hormone levels are associated with various diseases, such as liver cancer. Thyroid hormone receptors (TR) act as ligand-activated nuclear transcription factors to affect multiple functions through the gene-level regulation in the cells and several studies have revealed that thyroid hormone associated with ncRNAs expression. TR actions are complex and tissue- and time-specific, aberrant expression of the various TR isoforms have different effects and are associated with different types of tumor or stages of development. In this review, we discuss various aspects of the research on the thyroid hormones modulated ncRNAs to affect the functions of human liver cells.
    Schlagwörter Non-coding RNA ; Thyroid ; Receptors ; Hepatocellular carcinoma ; Medicine (General) ; R5-920 ; Biology (General) ; QH301-705.5
    Thema/Rubrik (Code) 610
    Sprache Englisch
    Erscheinungsdatum 2021-06-01T00:00:00Z
    Verlag Elsevier
    Dokumenttyp Artikel ; Online
    Datenquelle BASE - Bielefeld Academic Search Engine (Lebenswissenschaftliche Auswahl)

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  4. Artikel ; Online: Functional roles of non-coding RNAs regulated by thyroid hormones in liver cancer.

    Huang, Po-Shuan / Chang, Cheng-Chih / Wang, Chia-Siu / Lin, Kwang-Huei

    Biomedical journal

    2020  Band 44, Heft 3, Seite(n) 272–284

    Abstract: Recent reports have shown the important role of the non-coding part of human genome RNA (ncRNA) in cancer formation and progression. Among several kinds of ncRNAs, microRNAs (miRNA) play a pivotal role in cancer biology. Accumulating researches have been ...

    Abstract Recent reports have shown the important role of the non-coding part of human genome RNA (ncRNA) in cancer formation and progression. Among several kinds of ncRNAs, microRNAs (miRNA) play a pivotal role in cancer biology. Accumulating researches have been focused on the importance of non-coding genes in various diseases. In addition to miRNAs, long non-coding RNAs (lncRNAs) have also been extensively documented. Recently, the study of human liver cancer has gradually shifted to these non-coding RNAs that were originally considered "junk". Notably, dysregulated ncRNAs maybe influence on cell proliferation, angiogenesis, anti-apoptosis, and metastasis. Thyroid hormones play critical roles in human development and abnormalities in thyroid hormone levels are associated with various diseases, such as liver cancer. Thyroid hormone receptors (TR) act as ligand-activated nuclear transcription factors to affect multiple functions through the gene-level regulation in the cells and several studies have revealed that thyroid hormone associated with ncRNAs expression. TR actions are complex and tissue- and time-specific, aberrant expression of the various TR isoforms have different effects and are associated with different types of tumor or stages of development. In this review, we discuss various aspects of the research on the thyroid hormones modulated ncRNAs to affect the functions of human liver cells.
    Mesh-Begriff(e) Humans ; Liver Neoplasms/genetics ; MicroRNAs/genetics ; RNA, Long Noncoding/genetics ; RNA, Untranslated/genetics ; Thyroid Hormones
    Chemische Substanzen MicroRNAs ; RNA, Long Noncoding ; RNA, Untranslated ; Thyroid Hormones
    Sprache Englisch
    Erscheinungsdatum 2020-08-27
    Erscheinungsland United States
    Dokumenttyp Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 2698541-X
    ISSN 2320-2890 ; 2320-2890
    ISSN (online) 2320-2890
    ISSN 2320-2890
    DOI 10.1016/j.bj.2020.08.009
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  5. Artikel ; Online: Roles of Thyroid Hormone-Associated microRNAs Affecting Oxidative Stress in Human Hepatocellular Carcinoma.

    Huang, Po-Shuan / Wang, Chia-Siu / Yeh, Chau-Ting / Lin, Kwang-Huei

    International journal of molecular sciences

    2019  Band 20, Heft 20

    Abstract: Oxidative stress occurs as a result of imbalance between the generation of reactive oxygen species (ROS) and antioxidant genes in cells, causing damage to lipids, proteins, and DNA. Accumulating damage of cellular components can trigger various diseases, ...

    Abstract Oxidative stress occurs as a result of imbalance between the generation of reactive oxygen species (ROS) and antioxidant genes in cells, causing damage to lipids, proteins, and DNA. Accumulating damage of cellular components can trigger various diseases, including metabolic syndrome and cancer. Over the past few years, the physiological significance of microRNAs (miRNA) in cancer has been a focus of comprehensive research. In view of the extensive level of miRNA interference in biological processes, the roles of miRNAs in oxidative stress and their relevance in physiological processes have recently become a subject of interest. In-depth research is underway to specifically address the direct or indirect relationships of oxidative stress-induced miRNAs in liver cancer and the potential involvement of the thyroid hormone in these processes. While studies on thyroid hormone in liver cancer are abundantly documented, no conclusive information on the potential relationships among thyroid hormone, specific miRNAs, and oxidative stress in liver cancer is available. In this review, we discuss the effects of thyroid hormone on oxidative stress-related miRNAs that potentially have a positive or negative impact on liver cancer. Additionally, supporting evidence from clinical and animal experiments is provided.
    Mesh-Begriff(e) Animals ; Carcinoma, Hepatocellular/genetics ; Carcinoma, Hepatocellular/metabolism ; Disease Progression ; Gene Expression Regulation, Neoplastic ; Humans ; Liver Neoplasms/genetics ; Liver Neoplasms/metabolism ; MicroRNAs/genetics ; Oxidative Stress ; Reactive Oxygen Species/metabolism ; Thyroid Hormones/metabolism
    Chemische Substanzen MicroRNAs ; Reactive Oxygen Species ; Thyroid Hormones
    Sprache Englisch
    Erscheinungsdatum 2019-10-21
    Erscheinungsland Switzerland
    Dokumenttyp Journal Article ; Review
    ZDB-ID 2019364-6
    ISSN 1422-0067 ; 1422-0067 ; 1661-6596
    ISSN (online) 1422-0067
    ISSN 1422-0067 ; 1661-6596
    DOI 10.3390/ijms20205220
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  6. Artikel ; Online: Roles of Thyroid Hormone-Associated microRNAs Affecting Oxidative Stress in Human Hepatocellular Carcinoma

    Po-Shuan Huang / Chia-Siu Wang / Chau-Ting Yeh / Kwang-Huei Lin

    International Journal of Molecular Sciences, Vol 20, Iss 20, p

    2019  Band 5220

    Abstract: Oxidative stress occurs as a result of imbalance between the generation of reactive oxygen species (ROS) and antioxidant genes in cells, causing damage to lipids, proteins, and DNA. Accumulating damage of cellular components can trigger various diseases, ...

    Abstract Oxidative stress occurs as a result of imbalance between the generation of reactive oxygen species (ROS) and antioxidant genes in cells, causing damage to lipids, proteins, and DNA. Accumulating damage of cellular components can trigger various diseases, including metabolic syndrome and cancer. Over the past few years, the physiological significance of microRNAs (miRNA) in cancer has been a focus of comprehensive research. In view of the extensive level of miRNA interference in biological processes, the roles of miRNAs in oxidative stress and their relevance in physiological processes have recently become a subject of interest. In-depth research is underway to specifically address the direct or indirect relationships of oxidative stress-induced miRNAs in liver cancer and the potential involvement of the thyroid hormone in these processes. While studies on thyroid hormone in liver cancer are abundantly documented, no conclusive information on the potential relationships among thyroid hormone, specific miRNAs, and oxidative stress in liver cancer is available. In this review, we discuss the effects of thyroid hormone on oxidative stress-related miRNAs that potentially have a positive or negative impact on liver cancer. Additionally, supporting evidence from clinical and animal experiments is provided.
    Schlagwörter oxidative stress ; microrna ; thyroid hormone ; liver cancer ; Biology (General) ; QH301-705.5 ; Chemistry ; QD1-999
    Thema/Rubrik (Code) 610 ; 500
    Sprache Englisch
    Erscheinungsdatum 2019-10-01T00:00:00Z
    Verlag MDPI AG
    Dokumenttyp Artikel ; Online
    Datenquelle BASE - Bielefeld Academic Search Engine (Lebenswissenschaftliche Auswahl)

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  7. Artikel ; Online: CMAHP promotes metastasis by reducing ubiquitination of Snail and inducing angiogenesis via GM-CSF overexpression in gastric cancer.

    Huang, Hsiang-Wei / Chen, Ching-Ying / Huang, Ya-Hui / Yeh, Chau-Ting / Wang, Chia-Siu / Chang, Cheng-Chih / Lin, Kwang-Huei

    Oncogene

    2021  Band 41, Heft 2, Seite(n) 159–172

    Abstract: Pseudogenes are generally considered "junk" DNA or "genomic fossils" generated during the evolution process that lack biological activity. However, accumulating reports indicate that pseudogenes have biological functions critical for cancer development. ... ...

    Abstract Pseudogenes are generally considered "junk" DNA or "genomic fossils" generated during the evolution process that lack biological activity. However, accumulating reports indicate that pseudogenes have biological functions critical for cancer development. Experiments from the current study showed marked overexpression of the cytidine monophospho-N-acetylneuraminic acid hydroxylase pseudogene (CMAHP) in gastric cancer, which was associated with poor overall survival. However, the mechanisms underlying the activity of CMAHP in tumor development are largely unknown. Gene Set Enrichment Analysis (GSEA) revealed that CMAHP-correlated genes are significantly involved in epithelial-mesenchymal transition (EMT) and angiogenesis. Functional studies further confirmed that CMAHP mediates metastasis and angiogenesis in vitro and in vivo. Furthermore, CMAHP promoted cancer cell migration, invasion, and metastasis through Snail overexpression, which decreased ubiquitination mediated by NF-κB signaling. Angiogenesis is known to be induced by granulocyte-macrophage colony-stimulating factor (GM-CSF) stimulation. CMAHP increased GM-CSF transactivation via promoting direct binding of c-Jun to the -1981/-1975 region of the GM-CSF promoter. Notably, CMAHP interacts with Histone H1.4 promoting histone acetylation to enhance c-Jun and RelA (p65) expression. Our collective findings provide novel evidence that CMAHP contributes to tumor progression and modulates metastasis and angiogenesis in gastric cancer.
    Mesh-Begriff(e) Angiogenesis Inducing Agents/pharmacology ; Angiogenesis Inducing Agents/therapeutic use ; Cell Line, Tumor ; Granulocyte-Macrophage Colony-Stimulating Factor/genetics ; Humans ; Stomach Neoplasms/genetics ; Stomach Neoplasms/mortality ; Stomach Neoplasms/pathology ; Survival Analysis ; Ubiquitination/drug effects
    Chemische Substanzen Angiogenesis Inducing Agents ; Granulocyte-Macrophage Colony-Stimulating Factor (83869-56-1)
    Sprache Englisch
    Erscheinungsdatum 2021-10-29
    Erscheinungsland England
    Dokumenttyp Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 639046-8
    ISSN 1476-5594 ; 0950-9232
    ISSN (online) 1476-5594
    ISSN 0950-9232
    DOI 10.1038/s41388-021-02087-8
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  8. Artikel ; Online: C-reactive protein and malignancy: clinico-pathological association and therapeutic implication.

    Wang, Chia-Siu / Sun, Chien-Feng

    Chang Gung medical journal

    2009  Band 32, Heft 5, Seite(n) 471–482

    Abstract: C-reactive protein (CRP) is a widely used systemic biomarker for diagnosing acute and chronic inflammation. During the past decade, serum CRP has been re-emphasized by extending its clinical use to the prediction or diagnosis of cardiovascular diseases ... ...

    Abstract C-reactive protein (CRP) is a widely used systemic biomarker for diagnosing acute and chronic inflammation. During the past decade, serum CRP has been re-emphasized by extending its clinical use to the prediction or diagnosis of cardiovascular diseases and other conditions, particularly malignancies. Serum CRP has also been found to be elevated in patients with many malignancies, implying a close linkage between inflammation and malignancy. Prospective studies have shown a higher risk of developing cancer in those with elevated serum CRP. CRP is produced by hepatocytes in response to inflammatory cytokines, particularly, interleukin-6 from the tumor microenvironment. Preoperative CRP levels are parallel to the progression or pathological stages of malignancies, including gastric cancer in patients in our series. Elevated CRP is a determinant predictor of lower survival rates in patients with several cancers, including esophageal, colorectal, hepatocellular, pancreatic, urinary bladder, renal,ovarian and cervical cancer, after surgical resection. The measurement of serum CRP is simple, cheap, and available in daily practice. It can serve as an additional prognostic predictor for survival and post-treatment monitoring in cancer patients. In the future, CRP-lowering agents might offer a promising benefit in the prevention and therapy of many different types of cancer.
    Mesh-Begriff(e) C-Reactive Protein/analysis ; C-Reactive Protein/physiology ; Cyclooxygenase 2/blood ; Humans ; Interleukin-6/blood ; Neoplasms/blood ; Neoplasms/drug therapy ; Neoplasms/etiology ; Serum Amyloid A Protein/analysis ; Stomach Neoplasms/blood
    Chemische Substanzen Interleukin-6 ; Serum Amyloid A Protein ; C-Reactive Protein (9007-41-4) ; Cyclooxygenase 2 (EC 1.14.99.1) ; PTGS2 protein, human (EC 1.14.99.1)
    Sprache Englisch
    Erscheinungsdatum 2009-09
    Erscheinungsland China (Republic : 1949- )
    Dokumenttyp Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 1156932-3
    ISSN 2309-835X ; 2072-0939 ; 0255-8270
    ISSN (online) 2309-835X
    ISSN 2072-0939 ; 0255-8270
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  9. Artikel ; Online: Author Correction

    Jrhau Lung / Ming‑Szu Hung / Yu‑Ching Lin / Yuan Yuan Jiang / Yu‑Hung Fang / Ming‑Shian Lu / Ching‑Chuan Hsieh / Chia‑Siu Wang / Feng‑Che Kuan / Chang‑Hsien Lu / Ping‑Tsung Chen / Chieh‑Mo Lin / Yen‑Li Chou / Chin‑Kuo Lin / Tsung‑Ming Yang / Fen Fen Chen / Paul Yann Lin / Meng‑Jer Hsieh / Ying Huang Tsai

    Scientific Reports, Vol 11, Iss 1, Pp 1-

    A highly sensitive and specific real-time quantitative PCR for BRAF V600E/K mutation screening

    2021  Band 1

    Abstract: An amendment to this paper has been published and can be accessed via a link at the top of the paper. ...

    Abstract An amendment to this paper has been published and can be accessed via a link at the top of the paper.
    Schlagwörter Medicine ; R ; Science ; Q
    Sprache Englisch
    Erscheinungsdatum 2021-06-01T00:00:00Z
    Verlag Nature Portfolio
    Dokumenttyp Artikel ; Online
    Datenquelle BASE - Bielefeld Academic Search Engine (Lebenswissenschaftliche Auswahl)

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  10. Artikel ; Online: Author Correction: A highly sensitive and specific real-time quantitative PCR for BRAF V600E/K mutation screening.

    Lung, Jrhau / Hung, Ming-Szu / Lin, Yu-Ching / Jiang, Yuan Yuan / Fang, Yu-Hung / Lu, Ming-Shian / Hsieh, Ching-Chuan / Wang, Chia-Siu / Kuan, Feng-Che / Lu, Chang-Hsien / Chen, Ping-Tsung / Lin, Chieh-Mo / Chou, Yen-Li / Lin, Chin-Kuo / Yang, Tsung-Ming / Chen, Fen Fen / Lin, Paul Yann / Hsieh, Meng-Jer / Tsai, Ying Huang

    Scientific reports

    2021  Band 11, Heft 1, Seite(n) 12952

    Sprache Englisch
    Erscheinungsdatum 2021-06-15
    Erscheinungsland England
    Dokumenttyp Published Erratum
    ZDB-ID 2615211-3
    ISSN 2045-2322 ; 2045-2322
    ISSN (online) 2045-2322
    ISSN 2045-2322
    DOI 10.1038/s41598-021-92318-5
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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