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Buch: Huan zhe dao de quan li bao hu yu he xie yi huan guan xi jian gou

Wang, Xiaobo

The protection of patients' moral rights & the building of harmonious doctor-patient relationship

2015

Titelvarianten	Protection of patients' moral rights & the building of harmonious doctor-patient relationship
Verfasserangabe	Wang Xiaobo zhu
Mesh-Begriff(e)	Patient Rights ; Physician-Patient Relations
Schlagwörter	China
Sprache	Chinesisch
Umfang	2, 4, 248 pages
Ausgabenhinweis	Di 1 ban.
Dokumenttyp	Buch
ISBN	9787010151342 ; 7010151342
Datenquelle	Katalog der US National Library of Medicine (NLM)

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Artikel ; Online: Reply to the Letter to the Editor of Y. Zhao, et al. concerning "Association between IGF1 gene single nucleotide polymorphism (rs5742612) and adolescent idiopathic scoliosis: a meta-analysis" by Ming Guan, Huan Wang et al. Eur Spine J (2016). doi:10.1007/s00586-016-4742-7.

Guan, Ming / Wang, Huan / Fang, Huang

European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society

2017 Band 26, Heft 4, Seite(n) 1314–1315

Sprache	Englisch
Erscheinungsdatum	2017-04
Erscheinungsland	Germany
Dokumenttyp	Letter
ZDB-ID	1115375-1
ISSN	1432-0932 ; 0940-6719
ISSN (online)	1432-0932
ISSN	0940-6719
DOI	10.1007/s00586-017-5007-9
Datenquelle	MEDical Literature Analysis and Retrieval System OnLINE

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Artikel ; Online: Bu Yang Huan Wu decoction prevents reperfusion injury following ischemic stroke in rats via inhibition of HIF-1 α, VEGF and promotion β-ENaC expression.

Chen, Zhen-Zhen / Gong, Xin / Guo, Qi / Zhao, Hui / Wang, Lei

Journal of ethnopharmacology

2018 Band 228, Seite(n) 70–81

Abstract: Ethnopharmacological relevance: Bu Yang Huan Wu Decoction (BYHW) is a famous ...

Abstract	Ethnopharmacological relevance: Bu Yang Huan Wu Decoction (BYHW) is a famous traditional Chinese medicine (TCM) formula used in China for the treatment of cerebral ischemic stroke. But the protective effects and underlining mechanisms of BYHW remain unclear. Aim of the study: This study was designed to investigate the protective effects and underlining signaling mechanisms of BYHW on brain tissues in a rat model of cerebral ischemic reperfusion (I/R) injury. Materials and methods: Liquid chromatography was used to verify the composition of BYHW. The cerebral edema and infarct volume were measured by magnetic resonance imaging (MRI). The morphology and ultrastructure of ischemic penumbra brain tissues were observed by hematoxylin-eosin (HE) and transmission electron microscopy (TEM). The expression levels of HIF-1 α, VEGF and β-ENaC were tested using immunohistochemistry technique, western blot and quantitative PCR analysis, respectively. Results: Administration of BYHW significantly decreased cerebral edema, rat neurological function scores, reduced brain infarct volume. At the same time, BYHW had protective effect on the blood-brain barrier (BBB), which improved the morphology and ultrastructure of ischemic penumbra brain tissues. BYHW treatment significantly decreased the protein and mRNA levels of HIF-1 α and VEGF compared with the model treatment. In addition, BYHW treatment significantly up-regulated the protein and mRNA levels of β-ENaC. Conclusions: BYHW protected against cerebral I/R injury in MCAO rats through inhibiting the activation of the HIF-1 α /VEGF pathway and stabilizing ion channel of β-ENaC in brain, indicating that BYHW shows potential for stroke treatment in acute stage.
Mesh-Begriff(e)	Animals ; Drugs, Chinese Herbal/pharmacology ; Drugs, Chinese Herbal/therapeutic use ; Epithelial Sodium Channels/metabolism ; Hypoxia-Inducible Factor 1, alpha Subunit/metabolism ; Infarction, Middle Cerebral Artery/drug therapy ; Infarction, Middle Cerebral Artery/metabolism ; Infarction, Middle Cerebral Artery/pathology ; Male ; Neuroprotective Agents/pharmacology ; Neuroprotective Agents/therapeutic use ; Rats, Sprague-Dawley ; Reperfusion Injury/drug therapy ; Reperfusion Injury/metabolism ; Reperfusion Injury/pathology ; Stroke/drug therapy ; Stroke/metabolism ; Stroke/pathology ; Vascular Endothelial Growth Factor A/metabolism
Chemische Substanzen	Drugs, Chinese Herbal ; Epithelial Sodium Channels ; Hypoxia-Inducible Factor 1, alpha Subunit ; Neuroprotective Agents ; Vascular Endothelial Growth Factor A ; buyang huanwu ; vascular endothelial growth factor A, rat
Sprache	Englisch
Erscheinungsdatum	2018-09-13
Erscheinungsland	Ireland
Dokumenttyp	Journal Article
ZDB-ID	134511-4
ISSN	1872-7573 ; 0378-8741
ISSN (online)	1872-7573
ISSN	0378-8741
DOI	10.1016/j.jep.2018.09.017
Datenquelle	MEDical Literature Analysis and Retrieval System OnLINE

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Buch: Wo guo he xie yi huan guan xi de jian gou

Wang, Xiaobo

2014

Verfasserangabe	Wang Xiaobo zhu
Mesh-Begriff(e)	Physician-Patient Relations ; Health Care Reform
Schlagwörter	China
Sprache	Chinesisch
Umfang	2, 3, 192 pages
Ausgabenhinweis	Di 1 ban.
Dokumenttyp	Buch
ISBN	9787564333836 ; 7564333839
Datenquelle	Katalog der US National Library of Medicine (NLM)

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Artikel: Bu Yang Huan Wu decoction prevents reperfusion injury following ischemic stroke in rats via inhibition of HIF-1 α, VEGF and promotion β-ENaC expression

Chen, Zhen-Zhen / Xin Gong / Qi Guo / Hui Zhao / Lei Wang

Journal of ethnopharmacology. 2019 Jan. 10, v. 228

2019

Abstract: Bu Yang Huan Wu Decoction (BYHW) is a famous traditional Chinese medicine (TCM) formula used ...

Abstract	Bu Yang Huan Wu Decoction (BYHW) is a famous traditional Chinese medicine (TCM) formula used in China for the treatment of cerebral ischemic stroke. But the protective effects and underlining mechanisms of BYHW remain unclear.This study was designed to investigate the protective effects and underlining signaling mechanisms of BYHW on brain tissues in a rat model of cerebral ischemic reperfusion (I/R) injury.Liquid chromatography was used to verify the composition of BYHW. The cerebral edema and infarct volume were measured by magnetic resonance imaging (MRI). The morphology and ultrastructure of ischemic penumbra brain tissues were observed by hematoxylin-eosin (HE) and transmission electron microscopy (TEM). The expression levels of HIF-1 α, VEGF and β-ENaC were tested using immunohistochemistry technique, western blot and quantitative PCR analysis, respectively.Administration of BYHW significantly decreased cerebral edema, rat neurological function scores, reduced brain infarct volume. At the same time, BYHW had protective effect on the blood-brain barrier (BBB), which improved the morphology and ultrastructure of ischemic penumbra brain tissues. BYHW treatment significantly decreased the protein and mRNA levels of HIF-1 α and VEGF compared with the model treatment. In addition, BYHW treatment significantly up-regulated the protein and mRNA levels of β-ENaC.BYHW protected against cerebral I/R injury in MCAO rats through inhibiting the activation of the HIF-1 α /VEGF pathway and stabilizing ion channel of β-ENaC in brain, indicating that BYHW shows potential for stroke treatment in acute stage.
Schlagwörter	Oriental traditional medicine ; Western blotting ; animal models ; blood-brain barrier ; brain ; chromatography ; edema ; gene expression regulation ; immunohistochemistry ; infarction ; ion channels ; magnetic resonance imaging ; messenger RNA ; protective effect ; quantitative polymerase chain reaction ; rats ; reperfusion injury ; stroke ; tissues ; transmission electron microscopy ; ultrastructure ; vascular endothelial growth factors ; China
Sprache	Englisch
Erscheinungsverlauf	2019-0110
Umfang	p. 70-81.
Erscheinungsort	Elsevier B.V.
Dokumenttyp	Artikel
ZDB-ID	134511-4
ISSN	1872-7573 ; 0378-8741
ISSN (online)	1872-7573
ISSN	0378-8741
DOI	10.1016/j.jep.2018.09.017
Datenquelle	NAL Katalog (AGRICOLA)

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Artikel ; Online: Metabolite identification of seven active components of Huan-Nao-Yi-Cong-Fang in rat plasma using high-performance liquid chromatography combined with hybrid ion trap/time-of-flight mass spectrometry.

Wang, Minchao / Lu, Yanzhen / Liu, Jiangang / Li, Hao / Wei, Yun

Biomedical chromatography : BMC

2016 Band 30, Heft 2, Seite(n) 269–279

Abstract: Huan-Nao-Yi-Cong-Fang (HNYCF) is a potential prescription in treating Alzheimer's disease. Seven ...

Abstract	Huan-Nao-Yi-Cong-Fang (HNYCF) is a potential prescription in treating Alzheimer's disease. Seven constituents [ferulic acid (FA), 2,3,5,4'-tetrahydroxystilbene-2-O-β-d-glucoside (THSG), berberine hydrochloride (BHCl), emodin, ginsenoside Rg1 (Rg1), ginsenoside Re (Re) and ginsenoside Rb1 (Rb1)] have been used as quality chemical markers of HNYCF owing to their biological significance and high contents in crude plant materials. This study explored the metabolites of the seven bioactive components in rat plasma to give useful data for further study of the action mechanism of HNYCF. LC/MS-IT-TOF was used to simultaneously characterize the metabolites of the seven components. Using the combination of MetID Solution 1.0 software and accurate mass measurements, the metabolites of HNYCF were reliably characterized. Their structures were elucidated based on the accurate MS(2) spectra and comparisons of their changes in accurate molecular masses and fragment ions with those of parent compounds. A total of five parent active compounds (BHCl, emodin, Rg1, Rb1 and Re) and 10 metabolites were found from the rat plasma 2 h after oral administration of HNYCF dosage, of which two metabolites of emodin were observed for the first time. The proposed metabolic pathways of the bioactive components in the rat plasma are helpful for further studies on the pharmacokinetics and real active compound forms of this drug.
Mesh-Begriff(e)	Administration, Oral ; Animals ; Berberine/blood ; Berberine/chemistry ; Berberine/pharmacokinetics ; Chromatography, High Pressure Liquid/methods ; Drugs, Chinese Herbal/administration & dosage ; Drugs, Chinese Herbal/chemistry ; Drugs, Chinese Herbal/pharmacokinetics ; Emodin/blood ; Emodin/chemistry ; Emodin/pharmacokinetics ; Ginsenosides/blood ; Ginsenosides/chemistry ; Ginsenosides/pharmacokinetics ; Male ; Mass Spectrometry/methods ; Rats ; Rats, Wistar
Chemische Substanzen	Drugs, Chinese Herbal ; Ginsenosides ; Berberine (0I8Y3P32UF) ; Emodin (KA46RNI6HN)
Sprache	Englisch
Erscheinungsdatum	2016-02
Erscheinungsland	England
Dokumenttyp	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	632848-9
ISSN	1099-0801 ; 0269-3879
ISSN (online)	1099-0801
ISSN	0269-3879
DOI	10.1002/bmc.3546
Datenquelle	MEDical Literature Analysis and Retrieval System OnLINE

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Artikel ; Online: Deciphering the neuroprotective mechanisms of Bu-yang Huan-wu decoction by an integrative neurofunctional and genomic approach in ischemic stroke mice.

Wang, Hsei-Wei / Liou, Kuo-Tong / Wang, Yea-Hwey / Lu, Chung-Kuang / Lin, Yun-Lian / Lee, I-Jung / Huang, Sheng-Teng / Tsai, Yuan-Hau / Cheng, Yi-Chieh / Lin, Hung-Jui / Shen, Yuh-Chiang

Journal of ethnopharmacology

2011 Band 138, Heft 1, Seite(n) 22–33

Abstract: Ethnopharmacological relevance: Bu-yang Huan-wu decoction (BHD) is a famous ...

Abstract	Ethnopharmacological relevance: Bu-yang Huan-wu decoction (BHD) is a famous traditional Chinese medicine formula that has been used clinically in Asia to treat stroke-induced disability for centuries, but the underlying neuroprotective mechanisms are not fully understood. Aim of the study: In this study, we aim to investigate the mechanisms of action using an integrative neurofunctional and broad genomics approach. Materials and methods: Male ICR mice were subjected to an acute ischemic stroke by inducing a middle cerebral ischemic/reperfusion (CI/R) injury. To examine whether BHD could extend the lifespan of mice with a stroke, we used oral administration of BHD (0.5 and 1.0g/kg) twice daily starting from 2h after ischemia and compared this with vehicle control treatments, recombinant tissue-type plasminogen activator (rt-PA, 10mg/kg, i.v.), and MK-801 (0.2mg/kg, i.p.). An integrative neurofunctional and genomic approach was performed to elucidate the underlying molecular mechanisms of BHD. Results: More than 80% of the mice died within 2 days after stroke induction in the vehicle control treatment group. However, the survival rates and life-spans of mice treated with BHD, rt-PA and MK-801 were significantly enhanced as compared to the vehicle-treated CI/R group in all three cases. Mice treated with BHD (1.0g/kg) showed the greatest protective effect across all groups. BHD successfully restored brain function, ameliorated the cerebral infarction, and significantly improved the neurological deficits of the mice with a stroke. BHD also reduced inflammation, oxidative stress, and apoptosis, as well as improved neurogenesis. The molecular impacts of BHD were assessed by genome-wide transcriptome analysis using brains from the CI/R mice. The results showed a total of 377 ischemia-induced probe-sets that were significantly influenced by BHD including 93 probe-sets that were commonly more abundant in BHD-treated and sham mice, and another 284 ischemia-induced probe sets that were suppressed by BHD. Mining the functional modules and genetic networks of these 377 genes revealed a significant upregulation of neuroprotective genes associated with neurogenesis (6 genes) and nervous system development (9 genes), and a significant down-regulation of destructive genes associated with the induction of inflammation (14 genes), apoptosis (15 genes), angiogenesis (11 genes) and blood coagulation (7 genes) by BHD. Conclusions: Our results suggested that BHD is able to protect mice against stroke and extend lifespan primarily through a significant down-regulation of genes involved in inflammation, apoptosis, angiogenesis and blood coagulation, as well as an up-regulation of genes mediating neurogenesis and nervous system development. The changes in expression after treatment with BHD are beneficial after ischemic stroke.
Mesh-Begriff(e)	Animals ; Anti-Inflammatory Agents/pharmacology ; Anti-Inflammatory Agents/therapeutic use ; Antioxidants/pharmacology ; Antioxidants/therapeutic use ; Apoptosis/drug effects ; Apoptosis/genetics ; Astragalus Plant ; Blood Coagulation/drug effects ; Blood Coagulation/genetics ; Brain/drug effects ; Brain/physiology ; Brain Ischemia/drug therapy ; Brain Ischemia/etiology ; Brain Ischemia/genetics ; Cerebral Infarction/prevention & control ; Drugs, Chinese Herbal/pharmacology ; Drugs, Chinese Herbal/therapeutic use ; Gene Expression/drug effects ; Inflammation/drug therapy ; Inflammation/genetics ; Male ; Mice ; Mice, Inbred ICR ; Neovascularization, Physiologic/drug effects ; Neovascularization, Physiologic/genetics ; Neurogenesis/drug effects ; Neurogenesis/genetics ; Neuroprotective Agents/pharmacology ; Neuroprotective Agents/therapeutic use ; Oxidative Stress/drug effects ; Oxidative Stress/genetics ; Phytotherapy ; Stroke/drug therapy ; Stroke/genetics ; Stroke/mortality ; Tissue Plasminogen Activator/pharmacology
Chemische Substanzen	Anti-Inflammatory Agents ; Antioxidants ; Drugs, Chinese Herbal ; Neuroprotective Agents ; buyang huanwu ; Tissue Plasminogen Activator (EC 3.4.21.68)
Sprache	Englisch
Erscheinungsdatum	2011-10-31
Erscheinungsland	Ireland
Dokumenttyp	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	134511-4
ISSN	1872-7573 ; 0378-8741
ISSN (online)	1872-7573
ISSN	0378-8741
DOI	10.1016/j.jep.2011.06.033
Datenquelle	MEDical Literature Analysis and Retrieval System OnLINE

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Buch: Zhong guo ren xi huan tiao wu - yi ji du ben

Wang, Ruifeng / Cui, Yonghua

chinese people like to dance

(Shi yong han yu fen ji yue du cong shu ; 乙级读本)

2010

Titelübersetzung	Chinese people like to dance <eng.>
Titelvarianten	Zhongguoren xihuan tiaowu ; Chung-kuo-jen-hsi-huan-t'iao-wu
Verfasserangabe	Ruifeng Wang
Serientitel	Shi yong han yu fen ji yue du cong shu 乙级读本
Sprache	Chinesisch ; Englisch
Umfang	128 S., ill
Verlag	Beijing yu yan da xue chu ban she
Erscheinungsort	Beijing
Dokumenttyp	Buch
Anmerkung	Teilw. in chines. Schr.
Begleitmaterial	1 CD
ISBN	9787561925225 ; 7561925220
Datenquelle	Ehemaliges Sondersammelgebiet Küsten- und Hochseefischerei

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Artikel: Deciphering the neuroprotective mechanisms of Bu-yang Huan-wu decoction by an integrative neurofunctional and genomic approach in ischemic stroke mice

Wang, Hsei-Wei / Liou, Kuo-Tong / Wang, Yea-Hwey / Lu, Chung-Kuang / Lin, Yun-Lian / Lee, I-Jung / Huang, Sheng-Teng / Tsai, Yuan-Hau / Cheng, Yi-Chieh / Lin, Hung-Jui / Shen, Yuh-Chiang

Journal of ethnopharmacology. 2011 Oct. 31, v. 138, no. 1

2011

Abstract: ETHNOPHARMACOLOGICAL RELEVANCE: Bu-yang Huan-wu decoction (BHD) is a famous ...

Abstract	ETHNOPHARMACOLOGICAL RELEVANCE: Bu-yang Huan-wu decoction (BHD) is a famous traditional Chinese medicine formula that has been used clinically in Asia to treat stroke-induced disability for centuries, but the underlying neuroprotective mechanisms are not fully understood. AIM OF THE STUDY: In this study, we aim to investigate the mechanisms of action using an integrative neurofunctional and broad genomics approach. MATERIALS AND METHODS: Male ICR mice were subjected to an acute ischemic stroke by inducing a middle cerebral ischemic/reperfusion (CI/R) injury. To examine whether BHD could extend the lifespan of mice with a stroke, we used oral administration of BHD (0.5 and 1.0g/kg) twice daily starting from 2h after ischemia and compared this with vehicle control treatments, recombinant tissue-type plasminogen activator (rt-PA, 10mg/kg, i.v.), and MK-801 (0.2mg/kg, i.p.). An integrative neurofunctional and genomic approach was performed to elucidate the underlying molecular mechanisms of BHD. RESULTS: More than 80% of the mice died within 2 days after stroke induction in the vehicle control treatment group. However, the survival rates and life-spans of mice treated with BHD, rt-PA and MK-801 were significantly enhanced as compared to the vehicle-treated CI/R group in all three cases. Mice treated with BHD (1.0g/kg) showed the greatest protective effect across all groups. BHD successfully restored brain function, ameliorated the cerebral infarction, and significantly improved the neurological deficits of the mice with a stroke. BHD also reduced inflammation, oxidative stress, and apoptosis, as well as improved neurogenesis. The molecular impacts of BHD were assessed by genome-wide transcriptome analysis using brains from the CI/R mice. The results showed a total of 377 ischemia-induced probe-sets that were significantly influenced by BHD including 93 probe-sets that were commonly more abundant in BHD-treated and sham mice, and another 284 ischemia-induced probe sets that were suppressed by BHD. Mining the functional modules and genetic networks of these 377 genes revealed a significant upregulation of neuroprotective genes associated with neurogenesis (6 genes) and nervous system development (9 genes), and a significant down-regulation of destructive genes associated with the induction of inflammation (14 genes), apoptosis (15 genes), angiogenesis (11 genes) and blood coagulation (7 genes) by BHD. CONCLUSIONS: Our results suggested that BHD is able to protect mice against stroke and extend lifespan primarily through a significant down-regulation of genes involved in inflammation, apoptosis, angiogenesis and blood coagulation, as well as an up-regulation of genes mediating neurogenesis and nervous system development. The changes in expression after treatment with BHD are beneficial after ischemic stroke.
Schlagwörter	angiogenesis ; apoptosis ; blood coagulation ; brain ; gene induction ; genes ; infarction ; inflammation ; longevity ; mechanism of action ; mice ; oral administration ; oxidative stress ; plasminogen activator ; protective effect ; stroke ; survival rate ; traditional medicine ; transcriptome
Sprache	Englisch
Erscheinungsverlauf	2011-1031
Umfang	p. 22-33.
Erscheinungsort	Elsevier Ireland Ltd
Dokumenttyp	Artikel
ZDB-ID	134511-4
ISSN	1872-7573 ; 0378-8741
ISSN (online)	1872-7573
ISSN	0378-8741
DOI	10.1016/j.jep.2011.06.033
Datenquelle	NAL Katalog (AGRICOLA)

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Artikel: [Effects of bu yang huan wu decoction on astrocytes after cerebral ischemia and reperfusion].

Lai, Zhen / Wang, Sha-yan / Geng, Xiao-yin / Deng, Chang-qing / Zhang, Ruan-zhang

Zhongguo Zhong yao za zhi = Zhongguo zhongyao zazhi = China journal of Chinese materia medica

2002 Band 27, Heft 10, Seite(n) 763–765

Abstract: Objective: To study the effects of Bu Yang Huan Wu Decoction on astrocytes after cerebral ischemia ... a peak after cerebral ischemia for 15 min followed by reperfusion for 24 hours. Bu Yang Huan Wu Decoction ... by reperfusion for 48 hours, Bu Yang Huan Wu Decoction increased the expression.: Conclusion: The regulation ...

Abstract	Objective: To study the effects of Bu Yang Huan Wu Decoction on astrocytes after cerebral ischemia and reperfusion. Method: Cerebral ischemia model in gerbils was produced by ligating bilateral common carotid artery. The dynamic expressin of GFAP were determined by immunochemistry after cerebyal ischemia for 15 min followed by reperfusion for 24 hours and 48 hours. Result: GFAP positive reactions reached a peak after cerebral ischemia for 15 min followed by reperfusion for 24 hours. Bu Yang Huan Wu Decoction decreased the expression. GFAP positive reactions decreased after cerebral ischemia for 15 min followed by reperfusion for 48 hours, Bu Yang Huan Wu Decoction increased the expression. Conclusion: The regulation of Bu Yang Huan Wu Decoction on astrocytes after cerebral ischemia and reperfusion may be related to repairing process after cerebral ischemia.
Mesh-Begriff(e)	Animals ; Astrocytes/drug effects ; Brain Ischemia/complications ; Drugs, Chinese Herbal/isolation & purification ; Drugs, Chinese Herbal/pharmacology ; Female ; Glial Fibrillary Acidic Protein/metabolism ; Hippocampus/metabolism ; Male ; Plants, Medicinal/chemistry ; Reperfusion Injury/etiology ; Reperfusion Injury/metabolism ; Reperfusion Injury/pathology
Chemische Substanzen	Drugs, Chinese Herbal ; Glial Fibrillary Acidic Protein ; buyang huanwu
Sprache	Chinesisch
Erscheinungsdatum	2002-10
Erscheinungsland	China
Dokumenttyp	English Abstract ; Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1004649-5
ISSN	1001-5302 ; 0254-0029
ISSN	1001-5302 ; 0254-0029
Datenquelle	MEDical Literature Analysis and Retrieval System OnLINE

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