Artikel ; Online: M
2022 Band 15, Heft 760, Seite(n) eabm3720
Abstract: Many dementias are propagated through the spread of "prion-like" misfolded proteins. This includes prion diseases themselves (such as Creutzfeldt-Jakob disease) and Alzheimer's disease (AD), for which no treatments are available to slow or stop ... ...
Abstract | Many dementias are propagated through the spread of "prion-like" misfolded proteins. This includes prion diseases themselves (such as Creutzfeldt-Jakob disease) and Alzheimer's disease (AD), for which no treatments are available to slow or stop progression. The M |
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Mesh-Begriff(e) | Humans ; Animals ; Mice ; Prions/genetics ; Neurodegenerative Diseases/genetics ; Pathology, Molecular ; Proteomics ; Prion Diseases/genetics ; Alzheimer Disease/genetics ; Alzheimer Disease/pathology ; Receptor, Muscarinic M1/genetics ; Receptor, Muscarinic M1/metabolism |
Chemische Substanzen | Prions ; Receptor, Muscarinic M1 |
Sprache | Englisch |
Erscheinungsdatum | 2022-11-15 |
Erscheinungsland | United States |
Dokumenttyp | Journal Article ; Research Support, Non-U.S. Gov't |
ZDB-ID | 2417226-1 |
ISSN | 1937-9145 ; 1945-0877 |
ISSN (online) | 1937-9145 |
ISSN | 1945-0877 |
DOI | 10.1126/scisignal.abm3720 |
Datenquelle | MEDical Literature Analysis and Retrieval System OnLINE |
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