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  1. Book: Glucocorticoid signaling

    Wang, Jen-Chywan / Harris, Charles

    from molecules to mice to man

    (Advances in experimental medicine and biology ; 872)

    2015  

    Author's details Jen-Chywan Wang ; Charles Harris ed
    Series title Advances in experimental medicine and biology ; 872
    Collection
    Keywords Glucocorticoids
    Subject code 615.77
    Language English
    Size XI, 385 S. : Ill., graph. Darst.
    Publisher Springer
    Publishing place New York u.a.
    Publishing country United States
    Document type Book
    Note Includes bibliographical references and index
    HBZ-ID HT018731929
    ISBN 978-1-4939-2894-1 ; 9781493928958 ; 1-4939-2894-5 ; 1493928953
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    Zs A 3192 -872- not available for loan Zeitschriften-Lesesaal

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  2. Article: A Clinical Case.

    Harris, Charles T

    The Homoeopathic physician

    2023  Volume 4, Issue 9, Page(s) 264–265

    Language English
    Publishing date 2023-05-01
    Publishing country United States
    Document type Journal Article
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  3. Article: A New Method for the Management of Abortion.

    Harris, Charles H

    Atlanta medical and surgical journal (1884)

    2022  Volume 9, Issue 3, Page(s) 135–139

    Language English
    Publishing date 2022-07-11
    Publishing country United States
    Document type Journal Article
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article: Cold Dry Air as a Therapeutic Measure in Pulmonary Diseases.

    Harris, Charles H

    Atlanta medical and surgical journal

    2022  Volume 13, Issue 5, Page(s) 262–264

    Language English
    Publishing date 2022-07-11
    Publishing country United States
    Document type Journal Article
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  5. Article: Animal Models of Altered Glucocorticoid Signaling.

    Harris, Charles

    Advances in experimental medicine and biology

    2015  Volume 872, Page(s) 337–352

    Abstract: In this chapter we will review genetically engineered mice with alterations in glucocorticoid signaling. Most of the mice involve direct alterations to the glucocorticoid receptor locus, but we will touch briefly on other relevant models including 11-β- ... ...

    Abstract In this chapter we will review genetically engineered mice with alterations in glucocorticoid signaling. Most of the mice involve direct alterations to the glucocorticoid receptor locus, but we will touch briefly on other relevant models including 11-β-HSD transgenics which alter tissue levels of ligand as well as mice with glucocorticoid excess. Of course, the number of mice with mutations in genes such as GR targets and transcriptional coregulators is beyond the scope of this chapter.
    MeSH term(s) Animals ; Glucocorticoids/metabolism ; Mice ; Mice, Knockout ; Models, Animal ; Signal Transduction
    Chemical Substances Glucocorticoids
    Language English
    Publishing date 2015
    Publishing country United States
    Document type Journal Article
    ISSN 2214-8019 ; 0065-2598
    ISSN (online) 2214-8019
    ISSN 0065-2598
    DOI 10.1007/978-1-4939-2895-8_15
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  6. Article: Clinical Perspective: What Do Addison and Cushing Tell Us About Glucocorticoid Action?

    Harris, Charles

    Advances in experimental medicine and biology

    2015  Volume 872, Page(s) 83–96

    Abstract: This chapter is distinct from the others in its clinical subject matter. I will attempt to outline the major points of interest in glucocorticoids clinically. To aid the illustration in the evaluation of a patient with Cushing disease I have created a ... ...

    Abstract This chapter is distinct from the others in its clinical subject matter. I will attempt to outline the major points of interest in glucocorticoids clinically. To aid the illustration in the evaluation of a patient with Cushing disease I have created a case study.
    MeSH term(s) Addison Disease/physiopathology ; Adrenal Insufficiency/diagnosis ; Adrenal Insufficiency/therapy ; Cushing Syndrome/physiopathology ; Female ; Glucocorticoids/physiology ; Humans ; Middle Aged
    Chemical Substances Glucocorticoids
    Language English
    Publishing date 2015
    Publishing country United States
    Document type Journal Article ; Review
    ISSN 2214-8019 ; 0065-2598
    ISSN (online) 2214-8019
    ISSN 0065-2598
    DOI 10.1007/978-1-4939-2895-8_4
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  7. Article ; Online: Mutant prolactin receptor and familial hyperprolactinemia.

    Harris, Charles

    The New England journal of medicine

    2014  Volume 370, Issue 10, Page(s) 976

    MeSH term(s) Female ; Germ-Line Mutation ; Humans ; Hyperprolactinemia/genetics ; Male ; Receptors, Prolactin/genetics
    Chemical Substances Receptors, Prolactin
    Language English
    Publishing date 2014-03-05
    Publishing country United States
    Document type Letter ; Comment
    ZDB-ID 207154-x
    ISSN 1533-4406 ; 0028-4793
    ISSN (online) 1533-4406
    ISSN 0028-4793
    DOI 10.1056/NEJMc1315848
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Ua VI Zs.34: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
    bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
    ab Jg. 2022: Lesesaal (EG)

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  8. Article ; Online: Pathology in Practice.

    Morrison-Esmail, Krista / Harris, Charles / Perry, Amie

    Journal of the American Veterinary Medical Association

    2021  Volume 259, Issue 9, Page(s) 995–997

    MeSH term(s) Animal Diseases/diagnosis ; Animals
    Language English
    Publishing date 2021-10-14
    Publishing country United States
    Document type Journal Article
    ZDB-ID 390811-2
    ISSN 1943-569X ; 0003-1488
    ISSN (online) 1943-569X
    ISSN 0003-1488
    DOI 10.2460/javma.259.9.995
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 423: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
    bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
    Jg. 1995 - 2021: Lesesall (1.OG)
    ab Jg. 2022: Lesesaal (EG)

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  9. Article: Glucocorticoids and Diabetes.

    Bauerle, Kevin T / Harris, Charles

    Missouri medicine

    2018  Volume 113, Issue 5, Page(s) 378–383

    Abstract: Glucocorticoids (GCs), stress hormones produced by the adrenal gland, are involved in many pathways in physiology and metabolism including glucose homeostasis and inflammation. Excess GC signaling results in Cushing's syndrome and possibly metabolic ... ...

    Abstract Glucocorticoids (GCs), stress hormones produced by the adrenal gland, are involved in many pathways in physiology and metabolism including glucose homeostasis and inflammation. Excess GC signaling results in Cushing's syndrome and possibly metabolic syndrome. Diabetes, central adiposity, and hyperlipidemia are components of both syndromes. Here, we discuss the mechanisms of GC action, clinical syndromes of GC excess, modulation of glucose homeostasis by GCs, and future treatments for diabetes based on GC signaling.
    Language English
    Publishing date 2018-09-18
    Publishing country United States
    Document type Journal Article
    ZDB-ID 427362-x
    ISSN 0026-6620
    ISSN 0026-6620
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.B 182: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
    bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
    ab Jg. 2022: Lesesaal (EG)

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  10. Article: Epithelial-adipocyte interactions are required for mammary gland development, but not for milk production or fertility

    Brenot, Audrey / Hutson, Irina / Harris, Charles

    Developmental biology. 2020 Feb. 15, v. 458, no. 2

    2020  

    Abstract: To investigate the role of adipose tissue in reproductive function and mammary gland development and function, we have examined lipodystrophic (LD) mice. LD mice of both sexes are sterile, but fertility can be restored with leptin injections. Mammary ... ...

    Abstract To investigate the role of adipose tissue in reproductive function and mammary gland development and function, we have examined lipodystrophic (LD) mice. LD mice of both sexes are sterile, but fertility can be restored with leptin injections. Mammary glands from lipodystrophic mice were rudimentary and lacked terminal end buds. Leptin-injected LD mice were able to become pregnant, showed normal pregnancy-associated glandular proliferation despite a smaller glandular area, were able to produce a small amount of milk that had grossly normal content of milk proteins and neutral lipids, but could not sustain pups to weaning. In order to separate the individual requirements for 1) adipokines such as leptin, 2) estradiol, and 3) physical epithelial-adipocyte interactions, we performed a series of experiments with both lipodystrophic mice and ob (obese mice with a mutation in the lep gene encoding the adipokine leptin) mice that received either estradiol treatment or preadipocyte transplant. The resulting fat pad did not rescue the defect in mammary gland development in lipodystrophic mice. The defect also could not be rescued with estradiol pellets. Ob/ob mice, like LD mice, lack leptin and estradiol, but retain adipose tissue. Ob mice have defective mammary gland development. However, in striking contrast to what was observed in lipodystrophic mice, reconstitution of a WT fat pad in ob mice rescued the defect in mammary gland development. Estradiol treatment did not rescue mammary gland development in ob mice. Therefore direct interaction between mammary gland epithelia and adipocytes is a requirement for full invasion and expansion of the gland, but is not required for glandular proliferation during pregnancy and milk production.
    Keywords adipose tissue ; animal disease models ; epithelium ; estradiol ; genes ; leptin ; mammary development ; mammary glands ; mice ; milk ; milk production ; milk proteins ; mutation ; obesity ; pellets ; pregnancy ; pups ; triacylglycerols ; weaning
    Language English
    Dates of publication 2020-0215
    Size p. 153-163.
    Publishing place Elsevier Inc.
    Document type Article
    ZDB-ID 1114-9
    ISSN 1095-564X ; 0012-1606
    ISSN (online) 1095-564X
    ISSN 0012-1606
    DOI 10.1016/j.ydbio.2019.11.001
    Database NAL-Catalogue (AGRICOLA)

    Full text online

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    In stock of ZB MED Bonn / Germany

    Z 76/715: Show issues

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