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  1. Artikel ; Online: SARS-CoV-2 persistence on common food covering materials: plastic wrap, fruit wax, and cardboard takeout containers.

    Glasbrenner, David C / Choi, Young W / Middleton, Jason K

    Journal of applied microbiology

    2023  Band 134, Heft 2

    Abstract: Aims: Assess the persistence of infectious SARS-CoV-2 virus and virus genomic material on three common food coverings.: Methods and results: The stability of infectious virus and genomic material on plastic wrap, fruit wax, and cardboard takeout ... ...

    Abstract Aims: Assess the persistence of infectious SARS-CoV-2 virus and virus genomic material on three common food coverings.
    Methods and results: The stability of infectious virus and genomic material on plastic wrap, fruit wax, and cardboard takeout containers was measured. SARS-CoV-2 in simulated saliva was applied to the surface of these materials and allowed to dry. Samples were stored at 4°C or 20°C and a relative humidity of 30%, 50%, 65%, or 70% for up to 7 days. Viability was measured by TCID50 and the half-life for infectious virus was determined to be ~24 hours and ~8 hours at 4°C and 20°C, respectively, on all surfaces and RH tested. There was no loss of virus genomic material as measured by qRT-PCR at all conditions evaluated.
    Conclusions: SARS-CoV-2 virus remains infectious on food coverings for hours to days. It is estimated that a 99.9% reduction in titer requires 10 days at 4°C and 3 days at 20°C for all RH tested. SARS-CoV-2 genomic material showed no loss when assayed by qRT-PCR. Significance and Impact of Study: SARS-CoV-2 virus on food coverings loses infectivity over a certain period, but PCR assays can still detect virus genomic material throughout the same time. Thus, testing and controls may need to consider the fact that virus genomic material may still be detected when no infectious virus is present.
    Mesh-Begriff(e) SARS-CoV-2 ; COVID-19 ; Fruit ; Plastics
    Chemische Substanzen Plastics
    Sprache Englisch
    Erscheinungsdatum 2023-01-19
    Erscheinungsland England
    Dokumenttyp Journal Article
    ZDB-ID 1358023-1
    ISSN 1365-2672 ; 1364-5072
    ISSN (online) 1365-2672
    ISSN 1364-5072
    DOI 10.1093/jambio/lxac071
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  2. Artikel: Cancer-Causing Effects of Orthopaedic Metal Implants in Total Hip Arthroplasty.

    Sun, Cherry W Y / Lau, Lawrence C M / Cheung, Jason P Y / Choi, Siu-Wai

    Cancers

    2024  Band 16, Heft 7

    Abstract: Background: Metal implants have been preferentially used in THA due to its biocompatibility, mechanical stability and durability. Yet concerns have emerged regarding their potential to release metallic ions, leading to long-term adverse effects, ... ...

    Abstract Background: Metal implants have been preferentially used in THA due to its biocompatibility, mechanical stability and durability. Yet concerns have emerged regarding their potential to release metallic ions, leading to long-term adverse effects, including carcinogenicity. This study aimed to investigate the risk of cancer development in patients with orthopaedic metal implants in total hip arthroplasty (THA).
    Methods: Patients with THA conducted at a local tertiary implant centre from 2001-2008 were linked to the local cancer registry and followed up to the end of 2023. Standardized incidence ratios (SIRs) for cancer incidence and its confidence interval by Poisson distribution were calculated. Survival analysis was depicted using the Kaplan-Meier method, and the log-rank test was used to assess the differences across groups.
    Results: The study cohort included 388 patients and 53 cancers diagnosed during follow-up, at least 5 years post THA. All-site cancer risks were increased in patients with THA (SIR: 1.97; 95% CI: 1.48-2.46), validated with chi-square analysis (chi-square = 15.2551, N = 100,388,
    Conclusions: Patients with THA were found to have an increased risk for cancer compared to the general population during a mean follow-up of 16 years.
    Sprache Englisch
    Erscheinungsdatum 2024-03-29
    Erscheinungsland Switzerland
    Dokumenttyp Journal Article
    ZDB-ID 2527080-1
    ISSN 2072-6694
    ISSN 2072-6694
    DOI 10.3390/cancers16071339
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  3. Artikel ; Online: Perinuclear damage from nuclear envelope deterioration elicits stress responses that contribute to

    Sikder, Kunal / Phillips, Elizabeth / Zhong, Zhijiu / Wang, Nadan / Saunders, Jasmine / Mothy, David / Kossenkov, Andrew / Schneider, Timothy / Nichtova, Zuzana / Csordas, Gyorgy / Margulies, Kenneth B / Choi, Jason C

    Science advances

    2024  Band 10, Heft 19, Seite(n) eadh0798

    Abstract: Mutations in ... ...

    Abstract Mutations in the
    Mesh-Begriff(e) Animals ; Lamin Type A/metabolism ; Lamin Type A/genetics ; Mice ; Nuclear Envelope/metabolism ; Cardiomyopathies/metabolism ; Cardiomyopathies/etiology ; Cardiomyopathies/pathology ; Cardiomyopathies/genetics ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/pathology ; Autophagy ; Stress, Physiological ; Disease Models, Animal ; Endoplasmic Reticulum Stress ; Golgi Apparatus/metabolism ; Mice, Knockout
    Chemische Substanzen Lamin Type A ; Lmna protein, mouse
    Sprache Englisch
    Erscheinungsdatum 2024-05-08
    Erscheinungsland United States
    Dokumenttyp Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 2810933-8
    ISSN 2375-2548 ; 2375-2548
    ISSN (online) 2375-2548
    ISSN 2375-2548
    DOI 10.1126/sciadv.adh0798
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  4. Artikel ; Online: Lowering P

    Jimenez, Jose Victor / Ackrivo, Jason / Hsu, Jesse Y / Wilson, Mathew W / Labaki, Wassim W / Hansen-Flaschen, John / Hyzy, Robert C / Choi, Philip J

    Respiratory care

    2023  Band 68, Heft 12, Seite(n) 1613–1622

    Abstract: Background: Chronic hypercapnic respiratory failure is associated with high mortality. Although previous work has demonstrated a mortality improvement with high-intensity noninvasive ventilation in COPD, it is unclear whether a P: Methods: The ... ...

    Abstract Background: Chronic hypercapnic respiratory failure is associated with high mortality. Although previous work has demonstrated a mortality improvement with high-intensity noninvasive ventilation in COPD, it is unclear whether a P
    Methods: The objective of this study was to investigate the association between P
    Results: The mean ± SD age of 337 subjects was 57 ± 16 years, 37% women, and 85% white. In a univariate analysis, survival probability increased with reductions in P
    Conclusions: Reduction in P
    Mesh-Begriff(e) Humans ; Female ; Adult ; Middle Aged ; Aged ; Male ; Noninvasive Ventilation/adverse effects ; Hypercapnia/therapy ; Hypercapnia/complications ; Carbon Dioxide ; Pulmonary Disease, Chronic Obstructive/therapy ; Cohort Studies ; Prospective Studies ; Respiratory Insufficiency/etiology ; Respiratory Insufficiency/therapy ; Respiratory Insufficiency/diagnosis
    Chemische Substanzen Carbon Dioxide (142M471B3J)
    Sprache Englisch
    Erscheinungsdatum 2023-11-25
    Erscheinungsland United States
    Dokumenttyp Journal Article
    ZDB-ID 603252-7
    ISSN 1943-3654 ; 0098-9142 ; 0020-1324
    ISSN (online) 1943-3654
    ISSN 0098-9142 ; 0020-1324
    DOI 10.4187/respcare.10813
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  5. Artikel ; Online: Med25 Limits Master Regulators That Govern Adipogenesis.

    Saunders, Jasmine / Sikder, Kunal / Phillips, Elizabeth / Ishwar, Anurag / Mothy, David / Margulies, Kenneth B / Choi, Jason C

    International journal of molecular sciences

    2023  Band 24, Heft 7

    Abstract: Mediator 25 (Med25) is a member of the mediator complex that relays signals from transcription factors to the RNA polymerase II machinery. Multiple transcription factors, particularly those involved in lipid metabolism, utilize the mediator complex, but ... ...

    Abstract Mediator 25 (Med25) is a member of the mediator complex that relays signals from transcription factors to the RNA polymerase II machinery. Multiple transcription factors, particularly those involved in lipid metabolism, utilize the mediator complex, but how Med25 is involved in this context is unclear. We previously identified Med25 in a translatome screen of adult cardiomyocytes (CMs) in a novel cell type-specific model of
    Mesh-Begriff(e) Animals ; Mice ; 3T3-L1 Cells ; Adipogenesis/genetics ; Cell Differentiation ; Lamin Type A/genetics ; Lamin Type A/metabolism ; Lipids/pharmacology ; Mediator Complex/genetics ; Mediator Complex/metabolism ; PPAR gamma/metabolism ; Transcription Factors/metabolism
    Chemische Substanzen Lamin Type A ; Lipids ; Mediator Complex ; PPAR gamma ; Transcription Factors ; Med25 protein, mouse
    Sprache Englisch
    Erscheinungsdatum 2023-03-24
    Erscheinungsland Switzerland
    Dokumenttyp Journal Article
    ZDB-ID 2019364-6
    ISSN 1422-0067 ; 1422-0067 ; 1661-6596
    ISSN (online) 1422-0067
    ISSN 1422-0067 ; 1661-6596
    DOI 10.3390/ijms24076155
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  6. Artikel ; Online: Med25 Limits Master Regulators That Govern Adipogenesis

    Jasmine Saunders / Kunal Sikder / Elizabeth Phillips / Anurag Ishwar / David Mothy / Kenneth B. Margulies / Jason C. Choi

    International Journal of Molecular Sciences, Vol 24, Iss 6155, p

    2023  Band 6155

    Abstract: ... to recapitulate the in vivo phenotype by depleting lamins A and C (lamin A/C) in vitro. Although lamin A/C ... mediators driving adipogenesis revealed that C/EBPα and PPARγ are super-induced by Med25 silencing ...

    Abstract Mediator 25 (Med25) is a member of the mediator complex that relays signals from transcription factors to the RNA polymerase II machinery. Multiple transcription factors, particularly those involved in lipid metabolism, utilize the mediator complex, but how Med25 is involved in this context is unclear. We previously identified Med25 in a translatome screen of adult cardiomyocytes (CMs) in a novel cell type-specific model of LMNA cardiomyopathy. In this study, we show that Med25 upregulation is coincident with myocardial lipid accumulation. To ascertain the role of Med25 in lipid accumulation, we utilized iPSC-derived and neonatal CMs to recapitulate the in vivo phenotype by depleting lamins A and C (lamin A/C) in vitro. Although lamin A/C depletion elicits lipid accumulation, this effect appears to be mediated by divergent mechanisms dependent on the CM developmental state. To directly investigate Med25 in lipid accumulation, we induced adipogenesis in Med25 -silenced 3T3-L1 preadipocytes and detected enhanced lipid accumulation. Assessment of pertinent mediators driving adipogenesis revealed that C/EBPα and PPARγ are super-induced by Med25 silencing. Our results indicate that Med25 limits adipogenic potential by suppressing the levels of master regulators that govern adipogenesis. Furthermore, we caution the use of early-developmental-stage cardiomyocytes to model adult-stage cells, particularly for dissecting metabolic perturbations emanating from LMNA mutations.
    Schlagwörter LMNA ; mediator complex ; lipid accumulation ; adipogenesis ; Biology (General) ; QH301-705.5 ; Chemistry ; QD1-999
    Thema/Rubrik (Code) 570
    Sprache Englisch
    Erscheinungsdatum 2023-03-01T00:00:00Z
    Verlag MDPI AG
    Dokumenttyp Artikel ; Online
    Datenquelle BASE - Bielefeld Academic Search Engine (Lebenswissenschaftliche Auswahl)

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  7. Artikel: Perinuclear damage from nuclear envelope deterioration elicits stress responses that contribute to

    Sikder, Kunal / Phillips, Elizabeth / Zhong, Zhijiu / Wang, Nadan / Saunders, Jasmine / Mothy, David / Kossenkov, Andrew / Schneider, Timothy / Nichtova, Zuzana / Csordas, Gyorgy / Margulies, Kenneth B / Choi, Jason C

    bioRxiv : the preprint server for biology

    2023  

    Abstract: Mutations in the : Teaser: Interplay of stress responses underlying the development ... ...

    Abstract Mutations in the
    Teaser: Interplay of stress responses underlying the development of
    Sprache Englisch
    Erscheinungsdatum 2023-06-07
    Erscheinungsland United States
    Dokumenttyp Preprint
    DOI 10.1101/2023.02.14.528563
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  8. Artikel ; Online: Patterns of interventions for central venous catheter-associated deep vein thrombosis and outcomes in cancer patients.

    Ngo, Dat / Chen, Jason / Nguyen, Chris / Choi, Kathy / Pullarkat, Vinod

    Journal of oncology pharmacy practice : official publication of the International Society of Oncology Pharmacy Practitioners

    2024  , Seite(n) 10781552231219995

    Abstract: Purpose: This letter evaluated the impact of different management strategies, specifically the presence or absence of therapeutic anticoagulation, on clinical outcomes for central venous catheter (CVC)-associated deep vein thrombosis (DVT) in cancer ... ...

    Abstract Purpose: This letter evaluated the impact of different management strategies, specifically the presence or absence of therapeutic anticoagulation, on clinical outcomes for central venous catheter (CVC)-associated deep vein thrombosis (DVT) in cancer patients.
    Methods: One-hundred ninety-eight adult cancer patients with a confirmed CVC-associated DVT diagnosis from February 2013 and February 2021 were included.
    Results: Incidence of symptomatic recurrent venous thromboembolism (VTE) was similar between patients who received therapeutic anticoagulation and those who did not (14% vs 16%,
    Conclusion and relevance: Therapeutic anticoagulation was not associated with a reduction in the incidence of recurrent VTE or increase the incidence of bleeding in adult cancer patients following a CVC-associated DVT diagnosis.
    Sprache Englisch
    Erscheinungsdatum 2024-01-02
    Erscheinungsland England
    Dokumenttyp Journal Article
    ZDB-ID 1330764-2
    ISSN 1477-092X ; 1078-1552
    ISSN (online) 1477-092X
    ISSN 1078-1552
    DOI 10.1177/10781552231219995
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  9. Artikel ; Online: Persistent ∆FosB expression limits recurrent seizure activity and provides neuroprotection in the dentate gyrus of APP mice.

    Stephens, Gabriel S / Park, Jin / Eagle, Andrew / You, Jason / Silva-Pérez, Manuel / Fu, Chia-Hsuan / Choi, Sumin / Romain, Corey P St / Sugimoto, Chiho / Buffington, Shelly A / Zheng, Yi / Costa-Mattioli, Mauro / Liu, Yin / Robison, A J / Chin, Jeannie

    Progress in neurobiology

    2024  Band 237, Seite(n) 102612

    Abstract: Recurrent seizures lead to accumulation of the activity-dependent transcription factor ∆FosB in hippocampal dentate granule cells in both mouse models of epilepsy and mouse models of Alzheimer's disease (AD), which is also associated with increased ... ...

    Abstract Recurrent seizures lead to accumulation of the activity-dependent transcription factor ∆FosB in hippocampal dentate granule cells in both mouse models of epilepsy and mouse models of Alzheimer's disease (AD), which is also associated with increased incidence of seizures. In patients with AD and related mouse models, the degree of ∆FosB accumulation corresponds with increasing severity of cognitive deficits. We previously found that ∆FosB impairs spatial memory in mice by epigenetically regulating expression of target genes such as calbindin that are involved in synaptic plasticity. However, the suppression of calbindin in conditions of neuronal hyperexcitability has been demonstrated to provide neuroprotection to dentate granule cells, indicating that ∆FosB may act over long timescales to coordinate neuroprotective pathways. To test this hypothesis, we used viral-mediated expression of ∆JunD to interfere with ∆FosB signaling over the course of several months in transgenic mice expressing mutant human amyloid precursor protein (APP), which exhibit spontaneous seizures and develop AD-related neuropathology and cognitive deficits. Our results demonstrate that persistent ∆FosB activity acts through discrete modes of hippocampal target gene regulation to modulate neuronal excitability, limit recurrent seizure activity, and provide neuroprotection to hippocampal dentate granule cells in APP mice.
    Mesh-Begriff(e) Animals ; Dentate Gyrus/metabolism ; Mice ; Seizures/metabolism ; Mice, Transgenic ; Proto-Oncogene Proteins c-fos/metabolism ; Amyloid beta-Protein Precursor/metabolism ; Amyloid beta-Protein Precursor/genetics ; Neuroprotection/physiology ; Disease Models, Animal ; Alzheimer Disease/metabolism ; Male ; Mice, Inbred C57BL ; Humans
    Chemische Substanzen Fosb protein, mouse
    Sprache Englisch
    Erscheinungsdatum 2024-04-19
    Erscheinungsland England
    Dokumenttyp Journal Article
    ZDB-ID 185535-9
    ISSN 1873-5118 ; 0301-0082
    ISSN (online) 1873-5118
    ISSN 0301-0082
    DOI 10.1016/j.pneurobio.2024.102612
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  10. Artikel ; Online: Genetic Disruption of System xc-Mediated Glutamate Release from Astrocytes Increases Negative-Outcome Behaviors While Preserving Basic Brain Function in Rat.

    Hess, Evan M / Kassel, Sara N / Simandl, Gregory / Raddatz, Nicholas / Maunze, Brian / Hurley, Matthew M / Grzybowski, Michael / Klotz, Jason / Geurts, Aron / Liu, Qing-Song / Choi, SuJean / Twining, Robert C / Baker, David A

    The Journal of neuroscience : the official journal of the Society for Neuroscience

    2023  Band 43, Heft 13, Seite(n) 2349–2361

    Abstract: The importance of neuronal glutamate to synaptic transmission throughout the brain illustrates the immense therapeutic potential and safety risks of targeting this system. Astrocytes also release glutamate, the clinical relevance of which is unknown as ... ...

    Abstract The importance of neuronal glutamate to synaptic transmission throughout the brain illustrates the immense therapeutic potential and safety risks of targeting this system. Astrocytes also release glutamate, the clinical relevance of which is unknown as the range of brain functions reliant on signaling from these cells hasn't been fully established. Here, we investigated system xc- (Sxc), which is a glutamate release mechanism with an
    Mesh-Begriff(e) Rats ; Male ; Animals ; Glutamic Acid/metabolism ; Astrocytes/metabolism ; Synaptic Transmission ; Brain/metabolism ; Neurons/metabolism
    Chemische Substanzen Glutamic Acid (3KX376GY7L)
    Sprache Englisch
    Erscheinungsdatum 2023-02-14
    Erscheinungsland United States
    Dokumenttyp Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 604637-x
    ISSN 1529-2401 ; 0270-6474
    ISSN (online) 1529-2401
    ISSN 0270-6474
    DOI 10.1523/JNEUROSCI.1525-22.2023
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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